Content-Length: 27717 | pFad | http://www.ncbi.nlm.nih.gov/pubmed/16874111

ear Autophagy in immune defense against Mycobacterium tuberculosis

Autophagy in immune defense against Mycobacterium tuberculosis

Autophagy. 2006 Jul-Sep;2(3):175-8. doi: 10.4161/auto.2830. Epub 2006 Jul 7.

Abstract

Autophagy is a newly recognized innate and adaptive immunity defense against intracellular pathogens, in keeping with its role as a cytoplasmic maintenance pathway. Induction of autophagy by physiological, pharmacological or immunological means can eliminate intracellular Mycobacterium tuberculosis, providing one of the first examples of the immunological role of autophagy. Under normal circumstances, M. Tuberculosis survives in macrophages by inhibiting phagolysosome biogenesis. Induction of autophagy overcomes the mycobacterial phagosome maturation block, and delivers the tubercle bacilli to degradative compartments where they are eliminated.

Publication types

  • Research Support, N.I.H., Extramural
  • Review

MeSH terms

  • Autophagy / physiology*
  • Cytokines / physiology
  • GTP Phosphohydrolases / physiology
  • Humans
  • Interferon-gamma / physiology
  • Lipids / physiology
  • Macrophages / microbiology
  • Microbial Viability
  • Models, Biological
  • Mycobacterium tuberculosis / immunology
  • Mycobacterium tuberculosis / pathogenicity*
  • Phagocytosis / physiology
  • Phagosomes / metabolism
  • Phosphatidylinositol Phosphates / biosynthesis
  • Th2 Cells / metabolism
  • Th2 Cells / physiology
  • Tuberculosis / immunology*

Substances

  • Cytokines
  • Lipids
  • Phosphatidylinositol Phosphates
  • phosphatidylinositol 3-phosphate
  • Interferon-gamma
  • GTP Phosphohydrolases








ApplySandwichStrip

pFad - (p)hone/(F)rame/(a)nonymizer/(d)eclutterfier!      Saves Data!


--- a PPN by Garber Painting Akron. With Image Size Reduction included!

Fetched URL: http://www.ncbi.nlm.nih.gov/pubmed/16874111

Alternative Proxies:

Alternative Proxy

pFad Proxy

pFad v3 Proxy

pFad v4 Proxy