Master Techniques in Orthopaedic Surgery Knee11

Editors: Jackson, Douglas W.
Title: Master Techniques in Orthopaedic Surgery: Reconstructive Knee

Surgery, 3rd Edition
Copyright Â©2008 Lippincott Williams & Wilkins
> Front of Book > Editors
Editor
Douglas W. Jackson
P.xiii
Contributors
Frederick M. Azar MD
Professor
Director, Residency Program
Director, Sports Medicine
Fellowship, University of Tennesseeâ€”Campbell Clinic
Department of Orthopaedic Surgery
Memphis, Tennessee
Sue D. Barber-Westin BS
Director, Clinical and Applied Research
Cincinnati SportsMedicine Research and Education
Foundation
Cincinnati, Ohio
John A. Bergfeld MD
Director, Operating Room Management Team
Cleveland Clinic
Cleveland, Ohio
Kevin F. Bonner MD
Associate Professor
Eastern Virginia Medical School
Norfolk, Virginia
Petros J. Boscainos MD
Clinical Fellow, University of Toronto
Division of Orthopaedic Surgery
Toronto East General Hospital
Research Fellow, Division of Orthopaedic Surgery
Mount Sinai Hospital
Toronto, Ontario
William D. Bugbee MD
Associate Professor
Department of Orthopaedics
University of California
Attending Physician
Scripps Clinic
La Jolla, California
Anthony M. Buoncristiani MD
Sawtooth Orthopaedics and Sports Medicine
St. Luke's Hospital
Ketchum, Idaho
E. Lyle Cain Jr., MD

Fellowship Director
Orthopaedic Surgeon
American Sports Medicine Institute Center
Birmingham, Alabama
S. Terry Canale MD
Professor
Chairman
University of Tennesseeâ€”Campbell Clinic
Memphis Tennessee
Brett M. Cascio MD
LSU Health Sciences Center
School of Medicine at New Orleans
Department of Orthopaedics
New Orleans, Louisiana
Guglielmo Cerullo MD
Orthopaedic Surgeon
Clinica Valle Giulia
Roma, Italy
Edward Y. Cheng MD
Mairs Family Professor
University of Minnesota
Chief, Department of Orthopaedic Surgery
University of Minnesota Medical Center
Minneapolis, Minnesota
Massimo Cipolla MD
UniversitÃ di Roma La Sapienza
Roma, Italy
P.xiv
William G. Clancy Jr., MD, PhD
Orthopedic Associates of Aspen
Aspen Valley Hospital
Aspen, Colorado
Struan H. Coleman
Assistant Attending Orthopaedic Surgeon
Hospital for Special Surgery
Instructor in Orthopaedics
Weill Medical College of Cornell University
New York, New York
Andrew J. Cosgarea MD
Director, Sports Medicine and Shoulder Surgery
Associate Professor
Johns Hopkins University
Baltimore, Maryland
Brad A. Cucchetti DO
Florida Orthopaedic Institute
Tampa, Florida
ZsÃ³fia Duska
Physiotherapist, Human Kinesiologist
Orthopaedic and Trauma Department
Uzsoki Hospital
Budapest, Hungary
Craig J Edson MS, PT, ATC

Geisinger/Healthsouth Rehabilitation Hospital
Danville, Pennsylvania
Gianni Enrico MD
Roma, Italy
Gregory C Fanelli M.D.
Chief, Arthroscopic Surgery and Sports Medicine
Fanelli Sports Injury Clinic
Geisinger Clinic Medical Center
Danville, Pennsylvania
Mario Ferretti M.D.

Ferguson Laboratory for Orthopaedic Rearch
University of Pittsburgh
Pittsburgh, Pennsylvania
Vittorio Franco MD
Policlinico Umberto I
Roma, Italy
Freddie Fu MD, DSc, DPS

David Silver Professor and Chairman
University of Pittsburgh Medical Center
John P. Fulkerson M.D.

Orthopedic Associates of Hartford, P.C.
Clinical Professor and Sports Medicine Fellowship Director
University of Connecticut School of Medicine
Farmington, Connecticut
Seth Gasser MD
Florida Orthopaedic Institute
Tampa, Florida
Allen E. Gross MD, FRCSC, O.Ont.
Orthopaedic Surgeon
Professor of Surgery
Faculty of Medicine
University of Toronto
Toronto, Ontario
LÃ¡szlÃ³ Hangody MD, PhD, DSc

Clinical Professor
Uzsoki Hospital
Budapest, Hungary
Christopher D. Harner MD
University of Pittsburgh
Medical Director
UPMC Center for Sports Medicine
Stephen Hendricks
Alaska Orthopedic Labs
Anchorage, Alaska
Stephen M. Howell MD
Associate Professor
Department of Mechanical and Aeronautical Engineering
University of California-Davis
Davis, California
Douglas W. Jackson MD
President
Memorial Orthopaedic Surgical Group, Inc.
Southern California Center for Sports Medicine
Medical Director
Orthopaedic Research Institute
Long Beach, California
P.xv
ZoltÃ¡n KÃ¡rpÃ¡ti MD
Uzsoki Hospital
Budapest, Hungary
Catherine F. Kellett
Clinical Fellow
University of Toronto
Toronto, Ontario
Bryan T. Kelly
The Hospital for Special Surgery
New York, New York
Jason L. Koh MD
Fellow
Sports Medicine
Cleveland Clinic
Cleveland, Ohio
Peter R. Kurzweil MD
Fellowship Director
Roger V. Larson MD
Associate Professor
Orthopaedic and Sports Medicine
University of Washington
Seattle, Washington
Keith W. Lawhorn MD
Advanced Orthopaedics and Sports Medicine Institute
Inova Fair Oaks Hospital
Fairfax, Virginia
David A. McGuire MD
Affiliate Professor
University of Alaska, Anchorage
Healthsouth Alaska Surgery Center
Anchorage, Alaska
Frank R. Noyes MD
President and Medical Director
Cincinatti SportsMedicine and Orthopaedic Center
Clinical Professor
University of Cincinnati College of Medicine
Cincinnati, Ohio
Richard D. Parker MD
Professor of Surgery
Education Director
Cleveland Clinic Foundation
Cleveland, Ohio
Lars Peterson MD, PhD
Professor of Orthopaedics
University of GÃ¶teborg
Clinical Director
Gothenburg Medical Center
Gothenburg, Sweden
Christopher S. Proctor MD
Alta Orthopaedics
Santa Barbara, California
Giancarlo Puddu MD
Roma, Italy
Michael A. Rauh MD
State University of New York at Buffalo
Clinical Assistant Professor of Orthopaedic Surgery
University Sports Medicine
Buffalo, New York
Brant Richardson PAC

Alta Orthopaedics
Santa Barbara, California
William G. Rodkey DVM

Diplomat
American College of Veterinary Surgeons
Director
Basic Science Research and Senior Scientist
Steadman Hawkins Research Foundation
Vail Vally Medical Center
Vail, Colorado
Vineet Sharma
Fellow
Adult Reconstruction
Ranawat Orthopaedic Center
New York, New York
Wei Shen MD, PhD

Post-Doctoral Research Associate
Department of Orthopedic Surgery
P.xvi
Manno Steckel MD
University of Goettingen
Goettingen, Germany
Fotios Paul Tjoumakaris MD

Fellow
Center for Sports Medicine
Albert Mu-Hong Tsai MD

Memorial Orthopaedic Surgical Goup
William H. Warden III, MD

Blaine Warkentine MD
Russell F. Warren MD
Professor of Orthopaedics
Weill Cornell Medical College
Attending Surgeon-in-Chief Emeritus
Hospital for Special Surgery
New York, New York
William M. Wind Jr., MD

Clinical Assistant Professor
State University of New York at Buffalo
Buffalo, New York
> Front of Book > Dedication
Dedication
To My wife, Sandi, and the spouses of the other contributors, who have fostered
an environment that makes special projects possible.
> Front of Book > PREFACE
PREFACE
Symptoms and injuries related to the knee joint are responsible for more people
visiting an orthopaedic surgeon's office than any other joint. These patients have
high expectations for overcoming their knee complaints and returning to their
desired activity level. They have read about knee injuries in the sports page every
day and athletes returning to their sports following their injuries. As a result of lay
articles in the print and electronic media and marketing efforts to the public,
patients have come to expect less-invasive surgical approaches that preserve their
uninvolved anatomy and result in faster rehabilitation. The reconstructive knee
surgery procedures and techniques presented in this text focus on the latest
arthroscopic and less-invasive surgical applications. The procedures described in
this edition comprise a significant percentage of the total operative cases
performed by orthopaedic surgeons. These new and updated chapters focus on the
current state of surgical procedures and techniques for extensor mechanism and
patellofemoral disorders, for torn and damaged menisci, for articular cartilage
lesions, and for reconstructive ligament surgery of the knee. In addition, the
evolving role of allografts in knee surgery as well as computer-assisted applications
account for six of the new chapters in this edition.
Since the publication of the first edition of this book in 1995, there have been many
advances in the surgical treatment of the knee. This edition updates the important,
time-tested operations from the first and second editions and introduces many new
procedures that have emerged. There are 15 new chapters in this edition in
addition to the 20 other updated chapters. Experts share their approach to
meniscal repair and fixation; meniscal transplantation; cruciate ligament tunnel
placement and fixation; graft harvesting; the role of allografts; the latest
techniques for ACL, PCL, MCL and poterolateral corner reconstruction; opening-
wedge osteotomy; computer-assisted surgery; arthroscopic chondroplasty;
microfracture; osteochondral plugs; chondrocyte transplantation; and pigmented
villonodular synovitis resection.
The surgeons contributing to this volume were carefully chosen for their expertise
and experience in the clinical application of the latest techniques and technology
of specific procedures. They present surgical innovations, clinical judgment,
experience, operating room methodology, and decision making. They share their
indications and contraindications, as well as the pitfalls that may occur during
surgery. Each chapter includes systematic details with pictorial amplifications of
essential points designed to give the reader an operating room experience.
Because each chapter represents a selected individual author's approach to a

specific knee problem, the references at the end of each chapter are limited. It is
neither a review of the subject, nor a discussion of why the authors do not do it
differently. The chapters depict how carefully selected experts perform the
surgeries they find most beneficial in their experience.
My involvement in both selecting the authors and reviewing and editing these
chapters has improved the surgical care that I am able to offer my patients. I am
convinced that this book will be a benefit and will help the reader master these
surgical techniques.
Douglas W. Jackson MD
> Table of Contents > Part One - Extensor Mechanismâ€”Patellofemoral Problems > 1 -
Arthroscopic Lateral Release of the Patella with Radiofrequency Ablation
1
Arthroscopic Lateral Release of the Patella
with Radiofrequency Ablation
Seth I. Gasser
Brad A. Cucchetti
INDICATIONS/CONTRAINDICATIONS
Arthroscopic lateral release of the patella with electrocautery was first reported in the
literature in 1982 (10). Its prime advantage over standard lateral release procedures is
the potential to minimize postoperative bleeding and subsequent hemarthrosis. The
reported rate of significant postoperative hemarthrosis has decreased from more than
15% to less than 5% with the use of electrocautery (6). Other advantages of this
technique include improved arthroscopic visualization during transection of the lateral
retinaculum and a decrease in postoperative pain by minimizing the potential for
significant postoperative hemarthrosis. This allows the patient to participate in an
earlier, aggressive rehabilitation program that may improve the ultimate results of
surgery.
The indications for arthroscopic lateral release of the patella have changed over the
past decade and are much narrower today than in the past. The procedure has
significant potential complications and should be performed only in selected cases.
Our current indications for arthroscopic lateral release of
P.2
the patella include patients with recalcitrant anterior knee pain unresponsive to
conservative treatment with:
Tightness of the lateral retinaculum associated with lateral patellar tilt

(excessive lateral pressure syndrome)
Patellofemoral arthritis with lateral patellar and/or trochlear chondral damage

and associated lateral patellar tilt
Recurrent patellar subluxations/dislocations (typically performed in

conjunction with either reconstruction of the medial patellofemoral ligament
or a distal bony realignment)
Painful bipartite patella with associated lateral pressure syndrome
The exact mechanism whereby lateral release is effective in relieving pain is unknown.
Current theories include:
Decreased tension in the lateral retinaculum
Partial denervation of the patella
Correction of patellar tilt with decreased loading of the lateral

patella/trochlea
Arthroscopic lateral release of the patella is not indicated for the treatment of chronic
anterior knee pain in adolescents with normal patellar tracking.
Conservative therapy is successful in treating most cases of anterior knee pain

syndrome. This therapy ideally consists of 6 to 12 months of activity modification,
selective use of anti-inflammatory agents, and a carefully structured exercise and
stretching program. Patients are instructed to limit those activities that exacerbate
their symptoms. They are encouraged to substitute other pain-free activities to
maintain muscle strength, flexibility, and aerobic fitness. The therapeutic exercise
program begins with the stretching of tight muscle-tendon units (e.g., hamstrings,
triceps surae, iliotibial band, hip flexors) as well as the strengthening of weak muscles
[e.g., quadriceps, vastus medalis oblique (VMO), hip external rotators]. Initially,
isometric exercises are performed at or near full extension. Later, short-arc isotonic
exercises and closed-chain quadriceps exercises are added. Emphasis should be on
pain-free functional exercises for the entire lower extremity kinetic chain.
Various mechanical devices may also be used, including a Polumbo-style brace, a

neoprene patellar sleeve, or orthotics to prevent excessive foot pronation. McConnell
taping may be helpful during therapy sessions and when exercising to improve patellar
tracking and relieve lateral facet compression (5). Braces that attempt to simulate the
effects of McConnell taping are available. Additional braces have been introduced for
patients with recalcitrant anterior knee pain who demonstrate tightness of the
iliotibial band (Ober test) and hip flexors (Thomas test), with anterior pelvic tilt on
physical examination. These braces have provided mixed results in treating patients
with patellofemoral pain in our practice. Likewise, ultrasonography, phonophoresis,
and iontophoresis have not been very helpful in our therapeutic programs for anterior
knee pain. We have used them to treat localized soft-tissue areas that are
symptomatic.
The great majority of patients with anterior knee pain will respond to a nonsurgical
protocol (4). Once the symptoms have improved, we encourage a maintenance
exercise program done a minimum of three times per week, and a gradual return to
full activity. Surgery is offered only after attempts at nonsurgical treatment have
failed and the patient is unwilling to live with the persisting symptoms (8). It is
important that patients have realistic expectations of the success and failure rate of a
lateral release in relieving their anterior knee pain.
PREOPERATIVE PLANNING
Patient evaluation begins with a careful detailed history. It is important to note
whether the onset of the presenting pain was insidious or related to a specific or
repetitive trauma. Acute anterior knee pain associated with a large hemarthrosis can
be associated with a recent patellar subluxation or dislocation. These patients should
also be evaluated for injury to the anterior cruciate ligament, since both can occur
secondary to hyperextension of the knee. Rupture of the quadriceps or patellar tendon
is an infrequent finding but needs to be ruled out, particularly in older patients.
Localization of the type and character of pain is quite helpful, including location,
duration, frequency, exacerbating activities, alleviating maneuvers, and previous
treatments. Subtle malalignment may be associated with anterior knee pain that is
typically worse with squatting, stair climbing, and prolonged knee flexion. Teenage
females commonly experience self-limited anterior knee pain that resolves over a
period of time. A history of recurrent effusions may suggest articular cartilage
degeneration of the undersurface of the patella and/or trochlea.
P.3
The history usually provides a good indication of the diagnosis, which is then
confirmed by a thorough physical examination. Key points of the physical examination
include assessment of the sitting Q angle, patellar inhibition test, patellar
apprehension test, active and passive patellar tracking, and specific muscle tightness.
The Q angle has traditionally been measured with the knee in full extension. A sitting
Q angle (tubercle sulcus angle) is a better measure of the relationship of the two
vectors of the quadriceps and the patellar tendon. At 90 degrees of knee flexion, the
tibial tubercle should be directly under the center of the femoral sulcus, or at an
angle of 0 degrees. If it is lateral, or at a valgus angle, this indicates lateralization of
the tibial tubercle (3). In addition, the presence of VMO atrophy or hypoplasia should
be assessed. Comparison of the VMO musculature to the vastus lateralis should be
noted, as well as whether the contraction is concentric. The patella should track
smoothly through an active range of motion, without abrupt or sudden movements.
Crepitus with flexion and extension may be present but does not always correlate with
pain or the degree of chondromalacia. Facet tenderness and retinacular pain should
also be documented.
The evaluation for patellar tilt and glide is an important part of the physical
examination. Patellar tilt is performed with the patient in the supine position and the
knee in full extension. Normally, the lateral side of the patella can be elevated above
the horizontal. Inability to do this indicates tightness of lateral restraints, and
correlates with a higher success rate with surgery in symptomatic patients who
undergo lateral release. Testing of the patellar glide is performed with the knee in 30
degrees of flexion. A lateral glide of greater than 75% of the patellar width suggests
incompetent medial restraints, whereas a medial glide of less than 25% indicates
tightness of the lateral restraints (5).
Hamstring tightness may be associated with increased loads on the patellofemoral

joint. This can be assessed by measuring the popliteal angle with the patient supine
and the hip flexed to 90 degrees. With the patient supine, flex the hip and knee to 90
degrees. Then slowly extend the knee until muscle resistance is felt, keeping the low
back flat. The knee should be fully extended. If the hamstrings are tight, the knee will
remain flexed, and this angle short of full extension can be recorded. An Ober test
should be performed to rule out iliotibial band contracture. This is performed with the
patient on his side with the involved leg uppermost. The leg is abducted with the knee
flexed to 90 degrees while keeping the hip joint in neutral to slight extension. With
release of the abducted leg, the thigh should drop to an adducted position if the
iliotibial tract is normal. However, the thigh will remain abducted if there is a
contracture of the iliotibial band. Hip flexor tightness should be evaluated by
performing a Thomas test. The patient should be supine with his pelvis level. Both hips
are maximally flexed to the chest. Have the patient hold the uninvolved leg on the
chest while letting the involved leg down until it is as straight as possible. The extent
of a hip flexion contracture can be estimated by observing the angle between the leg
and the table. Recently, anterior pelvic tilt with hip muscular strength abnormalities
has been shown to be associated with patellofemoral pain. Reducing postural habits
that cause anterior pelvic tilt while strengthening hip flexors and abductors may be
effective in this group of patients. Weakness of the hip musculature can be evaluated
via a single leg squat test.
In general, both extremities should be examined for side-to-side differences, and

routine evaluation for meniscal and ligamentous pathology should be performed. Foot
pronation is associated with obligatory internal tibial rotation and may contribute to
anterior knee pain in patients with flatfoot deformity. These patients may benefit
from orthotics. Referred pain from the low back or hip should be excluded by
performing a straight-leg raise test and examining hip range of motion. In addition,
the patient should be checked for generalized ligamentous laxity, which may be
associated with patella alta and lateral subluxation or dislocation of the patella.
Radiographic evaluation consists of a standing PA view in 30 degrees of flexion, a

lateral view in 30 degrees of flexion, and a Merchant tangential view. These
radiographs should be evaluated for bipartite patella, occult fractures, tibiofemoral
arthritis, patella alta, and patellar malalignment. Abnormalities in patellar height can
be associated with symptomatic anterior knee pain. Insall and Salvati's ratio, normally
0.8 to 1.2, can be calculated from the lateral view in 30 degrees of flexion. It is
determined by the diagonal length of the patella divided by the length of the patellar
tendon (Fig. 1-1). A ratio of 0.8 or less demonstrates patella alta, whereas a ratio of
greater than 1.2 indicates patella baja. The lateral radiograph can also be used to
measure trochlear depth and dysplasia. Galland and coworkers defined three types of
trochlear dysplasia based on where the line of the intercondylar groove crossed the
shadow of the medial and lateral condyles (11).
The Merchant view is obtained with the patient supine and the knee flexed 45 degrees
in a Merchant frame (9). The x-ray beam is directed from cephalad to caudad, 30
degrees from the horizontal. Both patellofemoral joints are imaged on a single
cassette, allowing calculation of the sulcus
P.4
angle (normal: 130 degrees; range: 126 to 150 degrees; standard deviation: 6 degrees)
and the congruence angle (normal: -6 degrees; standard deviation: 11 degrees). The
congruence angle is determined by bisecting the sulcus angle to establish a reference
line. A second line is then drawn from the apex of the sulcus to the lowest point on
the articular ridge of the patella. A negative value is designated if the apex lies medial
to the reference line (Fig. 1-2). Any angle greater than +16 degrees is associated with
patellofemoral malalignment (9). All radiographic measurements have significant
shortcomings, and we do not routinely measure angles and draw lines on every
radiograph taken. Furthermore, abnormalities on radiographs do not necessarily
correlate with clinical symptoms and must be viewed in the context of the entire
history and physical examination.
FIGURE 1-1
Insall and Salvati's ratio (P/PT). Normal values with the knee in 30 degrees flexion
range from 0.8 to 1.2.
Computerized tomography (CT) has gained increasing popularity in evaluating patellar
alignment and tracking. CT cuts at 0, 15, 30, and 45 degrees allow the relationship of
the patella to the tibial tubercle to be established, as well as calculation of the
patellar tilt angle. Furthermore, CT scans of the hip, patella, and tibial tubercle can
be used to calculate the Q angle.
Magnetic resonance imaging (MRI) scans may show abnormalities of articular cartilage
and are helpful in evaluating other suspected intra-articular pathology. Sophisticated
imaging studies, including stress radiographs, bone scans, and kinematic MRI, are
available for more difficult diagnostic cases but are rarely needed.
SURGERY
Patient Positioning
The patient is placed on the operating table in the supine position. Under IV sedation,
the knee is sterilely prepped and injected with 50 mL of a 1:1 mixture of 1% lidocaine
with epinephrine and 0.25% Marcaine. An additional 10 mL is instilled into the region
of the anteromedial and anterolateral portals, as well as the soft tissue over the
lateral retinaculum. The surgeon then scrubs while the entire extremity is prepped
and draped, allowing the local anesthesia time to take full effect. Routine diagnostic
arthroscopy is performed using two standard portals. A commercially available pump
with appropriate cannulas allows inflow through the same portal as the arthroscope.
The anesthesiologist can titrate the amount of IV sedation needed during the
procedure so the patient does
P.5
not experience pain. The use of epidural or general anesthesia depends on patient and
surgeon preference.
FIGURE 1-2
Calculation of the congruence angle.
Since a leg holder may bind the quadriceps and alter patellofemoral mechanics, we
prefer a lateral post to assist in the arthroscopic assessment of the entire knee joint.
We do not use a tourniquet for several reasons. First, with meticulous hemostasis, we
have not found one to be necessary. Second, we believe our patients experience more
pain postoperatively when a tourniquet is used. Third, a tourniquet that is applied
tightly but not inflated may act to decrease venous blood flow, which can increase
bleeding and diminish visualization.
Technique
During the diagnostic arthroscopy, particular attention is paid to patellar tracking (tilt
and subluxation) and to articular cartilage wear. The patella should centralize in the
femoral groove at 30 to 45 degrees of knee flexion. Failure of the midpatellar ridge to
seat centrally in the femoral groove at 45 degrees of knee flexion suggests lateral
tracking (8) (Fig. 1-3). A separate superolateral viewing portal can also be used to
assess tracking, and may assist in the decision to proceed with arthroscopic
P.6
lateral release. However, we have not found this portal necessary and no longer use it.
Any of the currently available radiofrequency ablation devices are satisfactory for the
procedure.
FIGURE 1-3
A: Failure of the midpatellar ridge to seat centrally with the knee flexed 45
degrees. Note the cartilage fissures on the central ridge of the patella. B: Lateral
overhang of the lateral patellar facet on the femoral condyle.
FIGURE 1-4
An 18-gauge needle placed through the lateral patellar retinaculum, just proximal
to the level of the superior pole of the patella, denotes the upper level of
retinacular release.
An 18-gauge needle may be placed through the lateral patellar retinaculum just
proximal to the level of the superior pole of the patella for orientation. This denotes
the upper level of the retinacular release (Fig. 1-4). The arthroscopic and working
portals are then switched; the radiofrequency wand is inserted through the
anterolateral portal and the arthroscope anteromedially (Fig. 1-5). Using the ablation
mode on the lowest effective setting, the lateral retinaculum is released from a
proximal to distal direction, beginning at the level of the needle, 1 cm proximal and
lateral to the superior edge of the patella, with the knee in full extension.
The tissue is released in layers in a controlled and well-visualized manner. The vessels
can often be seen before their transection and are immediately coagulated. The
lateral superior geniculate artery must be cauterized if it is transected at the
superolateral border of the patella. The radiofrequency devices allow ablation and
coagulation with the same wand by simply stepping on the appropriate control pedal.
The proximal release is not extended into the muscle fibers of the vastus lateralis or
quadriceps tendon. The release is continued distally to just below the inferior pole of
the patella, effectively to the level of the tib/fib joint (Fig. 1-6). The capsular or
retinacular bands running
P.7
from the inferolateral pole to the lateral tibial plateau should be released (Fig. 1-7).
Removing a portion of the fat pad in this area with a full-radius resector enhances
visualization and helps ensure that the distal aspect of the release is complete. The
resection should proceed stepwise through the tissue layers until the superficial fat is
seen. This layer should be preserved. Upon completion of the release, the positive
inflow pressure is reduced and supplemented with gentle suction to allow
identification and coagulation of any additional source of bleeding. Patellar tracking
should be reassessed (Fig. 1-8). After a full release has been completed, there should
be strong outflow of irrigant from the inferior lateral portal once the wand has been
removed. Complete correction of patellar maltracking may not be obtained in all
patients. Those with more significant subluxation should be considered for a medial
plication/reconstruction and/or distal realignment. Adequate release is confirmed by
the ability to manually evert the patella in the femoral groove with the knee in full
extension (Fig. 1-9).
FIGURE 1-5
Portals used for arthroscopic lateral release of the patella with radiofrequency
ablation.
FIGURE 1-6
A: The radiofrequency wand is inserted through the anterolateral portal adjacent

to the lateral retinaculum. B: The retinaculum is released from proximal to
distal, beginning 1 cm proximal and lateral to the superior edge of the patella. C:
The tissue is released in layers down to the level of the superficial fat, which
should be preserved. D: The lateral patellotibial ligament is visualized just
proximal to the Arthrocare wand. E: The completed lateral release.
P.8
FIGURE 1-7
The lateral patellofemoral and patellotibial ligaments should be released.

FIGURE 1-8
Patellar tracking after lateral release. A: Central tracking. B: Correction of

lateral overhang.
P.9
FIGURE 1-9
The patella should be manually everted 90 degrees to confirm adequate lateral

release.
Pearls and Pitfalls
Lateral release should be performed only with the appropriate surgical

indications.
A slight bend made near the tip of the radiofrequency probe facilitates ease of
lateral release.
Avoid injury to the muscle fibers of the vastus lateralis proximally.
Mechanical debridement of the fat pad around the anterolateral portal

improves visualization and ensures an adequate distal release.
Decrease the pump pressure at the end of the procedure to help identify all
bleeding vessels. Meticulous hemostasis avoids the common complication of a
postoperative hemarthrosis and the associated pain that accompanies it.
POSTOPERATIVE MANAGEMENT
The immediate postoperative management and treatment is aimed at decreasing pain
and swelling with rest, ice, compression, and elevation. We feel the early initiation of
a specific rehabilitation program is an important part in achieving the best results
following an arthroscopic lateral release of the patella. Patients are encouraged to
start isometric exercises in the immediate postoperative course. They are allowed
range of motion and weight bearing as tolerated. We suggest using ice and crutches for
comfort and ambulation until muscle control of the extremity is re-established. Formal
physical therapy usually begins at the first postoperative visit 5 to 7 days after surgery.
The patient may shower within 2 days, but we recommend not immersing the knee in
water for 2 weeks postoperatively. Patients can generally return to school or a
sedentary job within 3 to 4 days after surgery. Crutches are recommended for several
weeks for walking any distance, but shorter distances may be negotiated without
crutches after only a few days. Limiting prolonged standing and walking for the first
few weeks after surgery helps prevent the development of a large effusion, which may
slow the recovery process. It takes 3 to 4 months for most patients to reach maximum
recovery.
Electrical stimulation of the VMO and biofeedback may be selectively employed if

progress is slow. Patient participation in passive patellar tilting and mobilization is
instituted at the first postoperative visit. A long-term exercise program that re-
establishes and maintains quadriceps strength ensures the best long-term results. The
regimen begins with isometric exercises at or near full extension
P.10
and progresses to short-arc isotonic quad sets, closed chain limited squats, and bicycle
riding. Both closed- and open-chain exercises may be employed. Progression is
individualized for each patient.
COMPLICATIONS
Complications of arthroscopic lateral release using radiofrequency ablation include:
Hemarthrosis
Thermal injury to the skin
Infection
Medial subluxation following excessive lateral release
Patellar hypermobility
Quadriceps weakness
Transection or rupture of the vastus lateralis insertion and/or quadriceps

tendon
Reflex sympathetic dystrophy
Persistent knee pain
The incidence of hemarthrosis can be reduced by careful attention to surgical detail,

obtaining adequate visualization, and meticulous cauterization of all bleeding vessels.
The lowest setting that allows adequate cutting and coagulation should be used to
decrease the incidence of thermal injury to the overlying skin. Insertion of an 18-
gauge needle at the superolateral border of the patella to delineate the starting point
of the lateral release should help prevent transection of the quadriceps tendon and
extension of the release too proximally into the vastus lateralis muscle fibers.
Postoperative medial subluxation and patellar hypermobility occur primarily in those
cases in which the lateral retinacular release is carried too proximally, or was
misdiagnosed and not appreciated preoperatively. This represents a formidable
treatment problem; if it exists but is not recognized preoperatively, it is often made
worse by a lateral retinacular release. Surgical reattachment of the vastus lateralis
with or without medial release provides unpredictable results in trying to salvage this
complication.
Superficial infections require special attention because of the potential for

subcutaneous accumulation of blood that communicates with an underlying
hemarthrosis. Intra-articular postoperative infections are rare. If there is any question
of deep infection, the patient should be returned to the operating room for formal
irrigation and debridement followed by appropriate antibiotics if indicated. We also
recommend restricting knee range of motion if there is any suggestion of drainage
from an intra-articular effusion.
Pre-existing complex regional pain syndrome should be recognized at the stage of

preoperative planning because it can be aggravated by surgery. An early rehabilitation
program is important to its postoperative prevention. Patients with unusual
postoperative pain and/or slow progress regaining knee motion deserve special
attention. We feel any signs of complex regional pain syndrome developing in the
postoperative period are best dealt with by a multidisciplinary approach.
Unrecognized complex regional pain syndrome or delayed treatment for this condition
can result in a postoperative disability that exceeds the patient's preoperative
symptoms.
RESULTS
As the indications for arthroscopic lateral release have become more specific,
outcomes have improved, but are still evolving. The literature currently reports
success rates in the range of 65% to 92% (1,2,6). These statistics bring to light the fact
that it is imperative to reserve lateral release for those cases with definitive clinical,
radiographic, and arthroscopic findings consistent with the surgical indications
previously outlined.
We believe that approximately 75% of our patients have achieved significant

improvement following surgery. The degree and timing of improvement vary a great
deal. Patients should have realistic expectations and understand that they may
continue to have some symptoms after surgery.
RECOMMENDED READING
Fanni AS, Tartarone M, Patricola A, et al. Long-term results of lateral retinacular
release. Arthroscopy. 2005;21(5): 526-531.
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REFERENCES
1. Aderinto J, Cobb AG. Lateral release for patellofemoral arthritis. Arthroscopy.
2002;18(4):399-403.
2. Carson WG, James SL, Larson RL, et al. Patellofemoral disorders: physical and
radiographic evaluation. Clin Orthop. 1984;185:165-186.
3. Friederichs MG, Burks RT. Patellofemoral disorders. In: Garrick JG, ed.
Orthopaedic knowledge update: sports medicine 3. Rosemont, IL: American
Academy of Orthopaedic Surgeons; 2004:213-221.
4. Fulkerson JP. Patellofemoral pain disorders: evaluation and management. J

AmAcad Orthop Surg. 1994;2:124-132.
5. Fulkerson JP, Kalenak A, Rosenberg TD, et al. Patellofemoral pain. In: Eilert RE,
ed. AAOS instructional course lectures, vol. 41. Rosemont, IL: American Academy
of Orthopaedic Surgeons; 1992:57-71.
6. Gallick GS, Brna JA, Fox JM. Electrosurgery in operative arthroscopy. Clin Sports
Med. 1987;6(3):607-618.
7. Kelly MA. Algorithm for anterior knee pain. In: Cannon WD, ed. AAOS
instructional course lectures, vol. 47. Rosemont, IL: American Academy of
Orthopaedic Surgeons; 1998:339-343.
8. Lombardo SJ, Bradley JP. Arthroscopic diagnosis and treatment of

patellofemoral disorders. In: Scott WN, ed. Arthroscopy of the knee: diagnosis and
treatment. Philadelphia: WB Saunders; 1990:155-173.
9. Merchant AC. Classification of patellofemoral disorders. Arthroscopy.

1988;4:235-240.
10. Miller GK, Dickason JM, Fox J, et al. The use of electrosurgery for arthroscopic
subcutaneous lateral release. Orthopaedics. 1982;5:309-314.
11. Walsh WM. Patellofemoral joint. In: DeLee JC, Drez D, eds. Orthopaedic sports
medicine: principles and practice, vol. 2. Philadelphia: WB Saunders; 1994;1163-
1248.
> Table of Contents > Part One - Extensor Mechanismâ€”Patellofemoral Problems > 2 - Medial
Patellofemoral Ligament Reconstruction and Repair
2
Medial Patellofemoral Ligament
Reconstruction and Repair
Andrew J. Cosgarea
Brett M. Cascio
INDICATIONS
Over the years, a large number of procedures have been described to treat patellar
instability. Recently there has been a great deal of interest in procedures that address
the medial patellofemoral ligament (MPFL), the primary soft tissue passive restraint to
pathologic lateral patellar displacement (2,4). The MPFL is torn when the patella
dislocates (10,15), and a variety of techniques have been described to repair (3,10,15)
or reconstruct (5,12,16) the ligament in an attempt to restore its function as a
checkrein.
History
The indication for MPFL surgery is patellar instability; therefore, the most important
clinical determination to make is whether anterior knee pain is associated with
instability. The much more common clinical entity of patellofemoral pain syndrome is
nearly universally treated nonoperatively. Among the spectrum of disorders causing
anterior knee pain, patellar instability represents a distinct clinical entity that is
usually amenable to surgical treatment. Instability represents a continuum ranging
from minor incidental subluxation episodes to traumatic dislocation events.
Dislocations can occur from an indirect twisting mechanism as the upper body rotates
while the foot remains planted on the ground. Less commonly, a direct blow along the
medial aspect of the patella during sports activity drives the patella laterally. Medial
dislocations are rare in patients who have not undergone previous lateral retinacular
release surgery. Sometimes the patella reduces spontaneously, although on occasion a
formal reduction maneuver may be necessary. With a first dislocation episode, the
patient usually experiences significant pain, and swelling is caused by soft tissue and
intra-articular damage. The resulting swelling and muscle weakness may take several
weeks to resolve. Patients who sustain multiple episodes tend to experience less
dramatic symptoms. Subluxation episodes also tend to be less dramatic. Patients may
describe a feeling of instability with or without pain. Instability usually occurs with
trunk rotation during physical activity, as the foot remains planted on the ground. The
discomfort is usually anterior, and may be bilateral, especially in patients with
malalignment or hyperlaxity.
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Physical Examination
Careful physical examination can confirm patellar instability. Alignment is evaluated
with the patient standing or walking. The knee is then observed for extra-articular
swelling or an effusion. The soft tissues are palpated, and the examiner should try to
identify an area of tenderness along the course of the MPFL, as this usually identifies
the location of the tear. A thorough cruciate and collateral ligament examination is
necessary to rule out concomitant pathology. Medial collateral ligament tears often
occur at the same time as a patellar dislocation. The quadriceps angle (Q angle) can
be measured as a gross assessment of the lateral force vector. Patellar tracking is
observed as the knee is extended from a flexed position. The patella has a tendency to
slip laterally as the knee approaches the last 20 degrees of extension and the patella is
no longer constrained by the lateral trochlear ridge (J sign). Patellar translation is
assessed by pushing on the medial side of the patella with the knee in full extension.
The amount of translation is quantified in quadrants and compared to the normal
contralateral knee. An indistinct endpoint suggests MPFL incompetence. A feeling of
apprehension (apprehension sign) supports the diagnosis of instability. Lateral
retinacular tightness is assessed by manually elevating the lateral edge of the patella
(tilt test).
The standard radiograph series includes anteroposterior, lateral (30 degrees flexion),
tunnel, and sunrise (30 to 45 degrees flexion) views. The tunnel view may
demonstratel osteochondritis dissecans lesions or loose bodies in the notch. The
sunrise view shows the degree of subluxation and tilt. Computed tomography (CT) scan
axial images (20 degrees flexion) are occasionally helpful to identify subluxation and
tilt. Magnetic resonance imaging (MRI) demonstrates the characteristic bone bruise
pattern involving the medial facet of the patella and the proximal lateral femoral
condyle in patients who have recently sustained a dislocation.
MPFL reconstruction is indicated in skeletally mature patients with symptomatic

recurrent lateral subluxation or dislocation. Patients have usually failed attempts at
nonoperative treatment including activity modification, physical therapy, and knee
bracing. Some authors recommend MPFL repair for patients following their first
patellar dislocation (15). We usually recommend treating an initial dislocation episode
nonoperatively, but will repair an acute MPFL tear when surgery is necessary for
concomitant intra-articular pathology (large loose body, meniscus tear). MPFL repair
techniques may also be used to treat recurrent instability. The MPFL becomes
attenuated and functionally incompetent with repeated instability episodes, so it is
tightened by cutting, shortening, and reattaching it at the patellar or femoral
insertion, or by midsubstance imbrication.
CONTRAINDICATIONS
MPFL repair and reconstruction are contraindicated in patients with medial instability
or isolated anterior knee pain. Great care should be taken not to overtighten the MPFL
or malposition the graft as this will result in excessive medial patellofemoral joint
pressures and is likely to exacerbate patellofemoral pain, particularly in patients with
medial patellofemoral chondral damage. The distal femoral growth plate is at risk for
injury in skeletally immature patients. The traditional MPFL reconstruction technique
can be modified so that the graft is passed around the proximal superficial medial
collateral ligament (MCL), or sutured directly to adjacent soft tissue.
Tibial tuberosity osteotomy procedures, such as the Elmslie-Trillat, have a theoretical

advantage in patients with greater degrees of malalignment. The Fulkerson
anteromedialization osteotomy is preferred in patients with malalignment and
degenerative changes involving the inferior pole of the patella or lateral facet (9).
MPFL reconstruction or repair can be combined with distal osteotomy if either alone is
insufficient to provide adequate stability.
SURGERY
Patient Positioning
The patient is placed supine on a standard operating room table. A vertical post is
used to facilitate arthroscopic evaluation and can be removed before starting the open
portion of the surgery. A tourniquet is used to facilitate hemostatis and visualization.
Surgery is performed on an outpatient basis using general or regional anesthesia.
Prophylactic intravenous antibiotics are administered before incision.
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Exam under Anesthesia

After induction of satisfactory anesthesia, a comprehensive examination is performed.
Patellar stability, translation, and tilt are usually easier to characterize when the
patient is anesthetized. With the knee extended, the position of the patella is
determined at rest, and with a lateral translation force applied. The amount of
translation on the symptomatic side is compared to the normal lateral patellar
translation in the contralateral knee (Fig. 2-1). The patella is pushed laterally, and the
amount of translation and the consistency of the endpoint are assessed. The examiner
everts the lateral edge of the patella to determine whether the lateral retinaculum is
tight. If the patient's symptoms and exam are consistent with excessive lateral
retinacular tightness, then consideration should be given to performing a concomitant
arthroscopic lateral retinacular release.
Diagnostic Arthroscopy
Arthroscopy is performed first using the standard superolateral, inferomedial, and
inferolateral portals. The suprapatellar pouch, the medial and lateral parapatellar
gutters, and the posteromedial and posterolateral compartments are carefully
assessed for loose bodies. The articular surfaces of the patella and trochlear are
thoroughly scrutinized for chondral lesions. The medial facet of the patella and the
proximal portion of the lateral femoral condyle are the areas most commonly injured
during a traumatic patellar dislocation. Hemorrhage may be noted in the soft tissue
adjacent to the medial edge of the patella in patients who have sustained a recent
avulsion of the MPFL insertion. Patellar tracking in the trochlear groove is visualized as
the knee is ranged. Any large chondral lesions are addressed surgically with
debridement, microfracture, or repair techniques as indicated. In some circumstances,
large defects should be unloaded using tibial tuberosity anteromedialization.
Arthroscopic lateral retinacular release may be performed at this point, although we
have not found that to be routinely necessary.
Surface Landmarks
After removing the arthroscopy equipment, a marking pen is used to identify the
important bony landmarks (Fig. 2-2). Marks are made over the patella, tibial
tuberosity, adductor tubercle, and medial femoral epicondyle. The pes anserine
tendon insertion site is palpated, and a 3- to 4-cm oblique mark is made over the
sartorial fascia insertion on the proximal medial tibia for the harvest site incision. A
second 3- to 4-cm line is drawn directly over the MPFL, halfway between the medial
border of the patella and the medial femoral epicondyle. The position is modified if
the location of the MPFL tear has been ascertained from the preoperative examination
or MRI, and an MPFL repair is planned.
The tourniquet is raised while the knee is in an extended position. A longitudinal

incision is made midway between the medial edge of the patella and the medial
epicondyle. Sharp and blunt dissection is used to expose the thick medial retinacular
layer contiguous with the inferior border of the vastus medialis obliquus muscle. With
gentle dissection, the superior and inferior borders of the MPFL are isolated (Fig. 2-3).
In patients who have sustained a recent dislocation, it may be possible
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to identify the site of the MPFL tear at the medial border of the patella, the medial
femoral epicondyle, or in midsubstance.
FIGURE 2-1
Comparison of patellar translation between symptomatic left and asymptomatic

right knees with equal lateral forces applied under general anesthesia. Note
lateral translation of the left patella is greater than two quadrants.
FIGURE 2-2
The circle is drawn over the patella. The proximal â€œXâ€• is over the adductor
tubercle; the distal â€œXâ€• overlies the medial epicondyle. The proximal line is
over the midportion of the MPFL and is where the incision is made to expose the
medial border of the patella and medial femoral epicondyle. The distal line
overlies the pes anserine insertion and is where the incision is made to harvest
the gracilis tendon.
Medial Patellofemoral Ligament Repair

The medial edge of the patella is brought into the operative field by applying a
medially directed force to the lateral border of the patella. If the MPFL is torn at this
location, a direct repair can be performed using bone tunnels or suture anchors. The
adductor tubercle and medial femoral epicondyle can be exposed through the same
incision with posterior retraction. The torn MPFL can be repaired in this location to a
stump of remaining tissue using #2 nonabsorbable sutures, or reattached directly to a
freshened bony surface using 2 or 3 suture anchors. With midsubstance ruptures it is
not always possible to identify the location of tissue failure. In chronic cases, it can be
especially difficult to determine how much to shorten the attenuated ligament.
Overtightening the ligament will cause repair failure or overconstrain the patella. We
usually perform MPFL repairs in patients who have experienced recent instability
episodes when acute surgical intervention is indicated for concomitant intra-articular
pathology.
Graft Harvest for Medial Patellofemoral Ligament

Reconstruction
A variety of different graft sources are available for reconstructing the MPFL. We
prefer to use the gracilis tendon because it is adjacent to the reconstruction site and
is relatively easy to harvest. An incision is made over the pes anserine insertion, and
the sartorial fascia is exposed. The superior edge of the sartorial fascia is identified
just anterior to the superficial medial collateral ligament. It is incised and then
everted to expose the underlying gracilis and semitendinosus tendons (Fig. 2-4). The
gracilis tendon is dissected free from the undersurface of the sartorial fascia. The free
end of the
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tendon is then tagged and harvested using a tendon stripper (Fig. 2-5). Although the
gracilis is smaller and shorter than the semitendinosus tendon, it is still significantly
stronger then the native MPFL (1,2,11) and long enough to construct an adequate
graft. The sartorial fascia is sutured back to its insertion on the proximal medial tibia.
FIGURE 2-3
The superior and inferior borders of the MPFL are identified and exposed.
FIGURE 2-4
The sartorial fascia is incised and inverted, exposing the gracilis tendon.
Muscle is removed from the surface of the gracilis tendon (Fig. 2-6). The length and
diameter are measured. A folded graft length of 9 to 10 cm is sufficient, as the length
will be increased by the addition of the Continuous Loop EndoButton (Acufex, Smith &
Nephew, Andover, MA) once the graft construct has been completed.
Patella Tunnel
The MPFL insertion on the medial edge of the patella is identified and a rongeur is
used to clear the soft tissue and expose cancellous bone (Fig. 2-7). The proximal and
distal poles of the patella are
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palpated, confirming that the patellar tunnel starting point is in the superior half of
the patella. A 2.5mm drill bit is advanced laterally in the midportion of the MPFL
insertion, which is usually just above the equator of the patella. The surgeon must be
careful while drilling not to violate either the anterior bony cortex or posterior
articular surface. Lateral fluoroscopy can be used to confirm appropriate positioning
(Fig. 2-8). The 2.5-mm drill bit is then replaced with a 2.0-mm eyelet K-wire, which is
then overdrilled with a 4.5-mm cannulated drill bit (Fig. 2-9). The length of the tunnel
is then measured with a depth gauge. The appropriate length Continuous Loop
EndoButton is chosen to complete the graft construct based on the length of the
tendon and the amount of graft that the surgeon wants in the patellar tunnel. The
gracilis tendon is passed through the loop of the EndoButton and sutured to itself using
a #2 nonabsorbable woven suture (Fig. 2-10). Sutures are woven through the femoral
end of the graft, which will be used later to pull the graft into the femoral tunnel. If
the graft diameter is greater than 4.5 mm, then the patellar tunnel is enlarged with
the appropriate-sized cannulated drill bit (Fig. 2-11).
FIGURE 2-5
The gracilis tendon is tagged with a #2 Ticron suture, then harvested with a
tendon striper.
FIGURE 2-6
Muscle is removed from the gracilis tendon.
The sutures from the EndoButton are loaded through the eyelet of the 2.0-mm K-wire,
which is then pulled out through the superolateral portal site (Fig. 2-12). With the
EndoButton positioned lengthwise, the graft is pulled through the patellar tunnel.
Once it clears the tunnel, tension is placed on the femoral end of the graft so that
EndoButton is brought flush to the lateral edge of the patella. The position of the
EndoButton can be manually manipulated so that it lies flush and lengthwise along the
lateral edge of the patella, which can be confirmed with fluoroscopy if desired (Fig. 2-
13).
Femoral Tunnel
A common error that occurs during MPFL reconstruction surgery is to place the femoral
tunnel too far proximal. Several studies have shown that the femoral attachment site
of the MPFL is at the medial epicondyle, which is approximately 1 cm distal to the
adductor tubercle (13,14,17). Therefore, it is crucial to distinguish between these 2
bony prominences (Fig. 2-14). A recent biomechanical study suggests that
malpositioning the femoral tunnel even 5 mm too far proximal results in increased
graft force and pressure applied to the cartilage of the medial patellofemoral cartilage
(6).
FIGURE 2-7
The attachment site of the MPFL at the medial edge of the patella is exposed in
preparation for drilling the patellar tunnel.
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FIGURE 2-8
A 2.5-mm drill bit passed transversely from medial to lateral. Fluoroscopy can be
used to confirm the appropriate position of the drill bit in the patella proximal to
the equator.
FIGURE 2-9
The 2.5-mm drill bit is replaced with a 2.0-mm eyelet K-wire, which is overdrilled
with a cannulated 4.5-mm drill bit.
FIGURE 2-10
The gracilis tendon is passed through the loop of the EndoButton and sewn back
to itself with #2 Ticron suture. A loop is sewn into the free end so that the pull-
through suture can be removed at the end of the case.
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FIGURE 2-11
The patellar tunnel size is increased to the appropriate depth based on the length
of the EndoButton loop and diameter of the completed graft.
FIGURE 2-12
The graft is brought through the patellar tunnel.

FIGURE 2-13
The position of the tunnel and EndoButton may be confirmed with fluoroscopy.
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FIGURE 2-14
The anatomic insertion site of the MPFL just anterior to the medial femoral
epicondyle is exposed.
A 2.5-mm drill bit is placed just anterior to the medial femoral epicondyle. The graft is
then passed underneath the medial retinaculum and the remnant of the MPFL and then
wrapped around the drill bit, allowing for assessment of graft isometry as the knee is
ranged (Fig. 2-15). The position of the drill bit can be adjusted to allow for fine-tuning
of tunnel positioning. Once the femoral tunnel has been determined, the drill bit is
replaced with a 2.0-mm eyelet K-wire. The K-wire is then over drilled with a 6.0-mm
cannulated drill bit to the correct depth based on the remaining length of the graft.
The suture ends are then passed through the eyelet of the K-wire and pulled into the
femoral tunnel as the K-wire exits the lateral side of the knee.
Graft Tensioning and Fixation

With tension placed on the free suture ends, the surgeon moves the knee several times
through a full range of motion. As the knee is ranged, graft isometry can be
ascertained by feeling tension on the free suture ends. In addition, the graft is directly
palpated and visualized through the medial incision. It is crucial not to overconstrain
the graft. Next, the knee is placed in full extension, and the patella is pushed
laterally. An attempt is made to identify the graft length, which allows the same
amount of lateral translation noted on the normal contralateral side. Once the
appropriate graft length is identified, fixation is obtained using a 7.0-mm
bioabsorbable cannulated femoral interference screw (Fig. 2-16) and can be
augmented by suturing the graft to the adjacent soft tissue. The surgeon should then
confirm that the knee has full range of motion, the patella is no longer dislocatable,
and the graft provides a firm checkrein to pathologic lateral translation.
The wound is closed in layers. A subcuticular skin closure allows for excellent cosmesis
(Fig. 2-17). A cryotherapy unit is used to help control pain and swelling. A compressive
dressing is then applied, followed by a thigh-high compression stocking and a postop
brace locked in full extension.
The procedure's surgical steps are summarized in Table 2-1.
FIGURE 2-15
The femoral end of the EndoButton is wrapped around the K-wire and graft
tension is assessed as the knee is ranged. The femoral tunnel position may be
modified if necessary.
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FIGURE 2-16
Once the correct femoral tunnel position is determined, it is drilled with a 6.0-
mm cannulated drill bit to the appropriate depth, based on the remaining length
of the graft. Fixation is achieved using 7.0-mm bioabsorbable interference screw.
FIGURE 2-17
Subcuticular wound closure allows for excellent cosmesis.

TABLE 2-1. Surgical Steps for MPFL Reconstruction
â€¢ Exam under anesthesia â€¢ Soft tissue tunnel passage
â€¢ Arthroscopic evaluation â€¢ Femoral tunnel preparation
â€¢ Hamstring graft harvest â€¢ Femoral tunnel graft passage
â€¢ MPFL exposure â€¢ Graft tensioning
â€¢ Femoral interference screw

â€¢ Patellar tunnel preparation fixation
â€¢ Hamstring graft preparation â€¢ Wound closure
â€¢ Graft passage and EndoButton

fixation
P.23
Pearls and Pitfalls
An incision over the midportion of the MPFL allows exposure of both the medial
border of the patella and the medial femoral epicondyle.
The correct location of the patellar tunnel is on the medial border of the
patella proximal to the equator.
The patellar tunnel should be initially drilled with a 2.5-mm drill bit and the
position confirmed with fluoroscopy before overdrilling with a 4.5-mm drill bit
to minimize the risk of patellar fracture.
When the free ends of the hamstring graft are sewn together, a suture loop is
created so that the passing suture that is used to pull the graft into the
femoral tunnel can be completely removed where it exits percutaneously on
the lateral side of the knee.
The femoral tunnel should be distal to the adductor tubercle and anterior to
the medial epicondyle.
Final femoral fixation should occur at the knee flexion angle that causes the
greatest amount of tension in the graft with the goal of reproducing the same
amount of lateral patellar translation as was appreciated on the contralateral
normal side with the knee extended during the examination under anesthesia.
It is better to leave the reconstructed MPFL graft slightly loose than to

overconstrain the patellofemoral joint by making the graft too tight.
Patients are instructed in quadriceps sets and straight leg raises in the recovery room
and are allowed to ambulate with crutches and touchdown weight-bearing status. The
brace remains locked in full extension for 1 week, and then patients are encouraged to
begin knee range of motion and to progress as tolerated. Patients are instructed to
attend formal physical therapy 3 times per week, where knee range of motion and
quadriceps strengthening are emphasized. Weight bearing progresses as tolerated, and
the brace is unlocked for ambulation as soon as quadriceps strength is sufficient.
Patients are encouraged to reach 120 degrees of knee flexion by 4 weeks postop, and
the brace is generally discontinued by 6 weeks. Full knee range of motion should be
achieved by 8 weeks. Patients are allowed to progress to jogging and sports-specific
drills by 12 weeks, and most patients are able to return to sports by 4 to 5 months. A
rehabilitation plan is outlined in Table 2-2.
COMPLICATIONS
As with most patellofemoral reconstruction procedures, the most common
postoperative complication is loss of motion. Motion deficits may be secondary to
inadequate postoperative rehabilitation. Loss of flexion may also be secondary to
intraoperative technical errors such as malpositioning or overtensioning the graft.
These errors could also overload the medial patellofemoral joint articular surfaces and
result in arthrosis, especially if there is a pre-existing medial patellar chondral lesion.
The saphenous nerve is at risk for injury during exposure of the femoral tunnel. Other
potential complications include recurrent instability secondary to graft failure, painful
hardware, and patella fracture.
TABLE 2-2. Postoperative Rehabilitation
â€¢ Postop: Touchdown weight bearing in brace locked in extension,

quadriceps sets, and straight leg raises
â€¢ Week 1: Physical therapy referral, progress with knee range of motion,
weight bearing, and strengthening
â€¢ Week 6: Discontinue brace
â€¢ Week 8: Should have full range of motion
â€¢ Week 12: Jogging program
â€¢ Week 16: Sports-specific drills
â€¢ Week 20: Return to sports when strength and agility allow
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RESULTS
Outcome following MPFL repair or reconstruction has been generally favorable
(5,7,8,10,15). Although these studies generally have been limited by small numbers of
patients, retrospective design, and the lack of control groups, most report satisfactory
outcome (good and excellent) in the range of 86% to 96% (5,7,8,10).
REFERENCES
1. Amis AA, Firer P, Mountney J, et al. Anatomy and biomechanics of the medial
patellofemoral ligament. Knee. 2003;10:215-220.
2. Conlan T, Garth WP Jr, Lemons JE. Evaluation of the medial soft-tissue

restraints of the extensor mechanism of the knee. J Bone Joint Surg Am.
1993;75:682-693.
3. Davis DK, Fithian DC. Techniques of medial retinacular repair and

reconstruction. Clin Orthop Relat Res. 2002;402: 38-52.
4. Desio SM, Burks RT, Bachus KN. Soft tissue restraints to lateral patellar
translation in the human knee. Am J Sports Med. 1998;26:59-65.
5. Drez D Jr, Edwards TB, Williams CS. Results of medial patellofemoral ligament
reconstruction in the treatment of patellar dislocation. Arthroscopy. 2001;17:298-
306.
6. Elias JJ, Cosgarea AJ. Technical errors during medial patellofemoral ligament
reconstruction could overload medial patellofemoral cartilage. Am J Sports Med.
2006;34:1478-1485.
7. Ellera Gomes JL. Medial patellofemoral ligament reconstruction for recurrent

dislocation of the patella: a preliminary report. Arthroscopy. 1992;8:335-340.
8. Ellera Gomes JL, Stigler Marczyk LR, Cesar de Cesar P, et al. Medial
patellofemoral ligament reconstruction with semitendinosus autograft for chronic
patellar instability: a follow-up study. Arthroscopy. 2004;20:147-151.
9. Fulkerson JP, Becker GJ, Meaney JA, et al. Anteromedial tibial tubercle transfer
without bone graft. Am J Sports Med. 1990;18:490-496; disc 496-497.
10. Garth WP Jr, DiChristina DG, Holt G. Delayed proximal repair and distal
realignment after patellar dislocation. Clin Orthop Relat Res. 2000;377:132-144.
11. Hamner DL, Brown CH Jr, Steiner ME, Hecker AT, Hayes WC. Hamstring tendon
grafts for reconstruction of the anterior cruciate ligament: biomechanical
evaluation of the use of multiple strands and tensioning techniques. J Bone Joint
Surg. 1999;81A:549-557.
12. Muneta T, Sekiya I, Tsuchiya M, et al. A technique for reconstruction of the

medial patellofemoral ligament. Clin Orthop Relat Res. 1999;359:151-155.
13. Nomura E, Horiuchi Y, Kihara M. Medial patellofemoral ligament restraint in

lateral patellar translation and reconstruction. Knee. 2000;7:121-127.
14. Nomura E, Inoue M. Surgical technique and rationale for medial patellofemoral
ligament reconstruction for recurrent patellar dislocation. Arthroscopy.
2003;19:E47.
15. Sallay PI, Poggi J, Speer KP, et al. Acute dislocation of the patella. A
correlative pathoanatomic study. Am J Sports Med. 1996;24:52-60.
16. Schock E, Burks R. Medial patellofemoral ligament reconstruction using a

hamstring graft. Op Tech Sports Med. 2001; 9:169-175.
17. Smirk C, Morris H. The anatomy and reconstruction of the medial

patellofemoral ligament. Knee. 2003;10: 221-227.
Anteromedial Tibial Tubercle Transfer
3
Anteromedial Tibial Tubercle Transfer
John P. Fulkerson
The best candidate for anteromedial tibial tubercle transfer (2,5,6) (Fig. 3-1) is a
patient with lateral patellar tilt (and/or subluxation) associated with grade III or IV
articular degeneration localized on the lateral and/or distal medial patellar facets
following the failure of nonsurgical therapy. If there is no articular degeneration or
pain, there is no need to anteriorize the tibial tubercle, and a straight medial transfer
of the tibial tubercle as described by Trillat (11) and reviewed by Carney, Mologne,
Muldoon, and Cox (1) is more appropriate for correcting subluxation. However, some
patients have distal and/or lateral patella articular softening (grade I) or cartilage
breakdown (grade II-IV).
In such patients, the surgeon may wish to anteriorize the extensor mechanism to some
extent at the time of realignment. An oblique osteotomy will transfer load off an area
of articular degeneration, particularly when damage is noted on the distal aspect of
the patella (anteriorization â€œtipsâ€• up the distal patella, thereby unloading it).
Anteromedial tibial tubercle transfer is appropriate whenever the surgeon wishes to
shift contact stress on the patella from the lateral and distal aspects of the patella to
the more proximal and medial patellar articular cartilage. Most important is to
recognize that tibial tubercle anteriorization at the time of medialization gives the
added benefit of removing articulation with the distal patella, which is often a
fragmented or softened source of pain.
Before considering anteromedial tibial tubercle transfer, alternative nonsurgical

treatment methods must fail. In particular, a well-structured program of rehabilitation
should be tried involving mobilization of a lateralized and tight extensor mechanism
supplemented by patellar bracing (9). I use the TruPull braces (DJOrtho, Vista, CA). A
tight extensor mechanism and hamstrings should be mobilized through stretching and
strengthening. Prone quadriceps stretching has been very helpful and is easy for
patients to do at home. Lower extremity core stability including hip stabilization
exercises is important in these patients. Nonsteroidal anti-inflammatory medication is
often helpful for symptomatic treatment during the period of nonsurgical therapy.
Failed lateral release is another potential indication for anteromedial tibial tubercle
transfer. If a patient is left with residual articular pain or symptomatic lateral
malalignment of the patella following lateral release, anteromedial tibial tubercle
transfer may be helpful.
Contraindications to anteromedial tibial tubercle transfer include:
Diffuse patella articular breakdown. Particularly if the cartilage loss involves

the proximal medial patella (dashboard or crush injury), patients are less likely
to benefit from anteromedial tibial tubercle transfer.
Tilt alone and no significant subluxation or lateral facet collapse. Such patients
may benefit from simple lateral retinacular release; therefore anteromedial
tibial tubercle transfer is not recommended as a first procedure.
Reflex sympathetic dystrophy and chronic pain syndrome.
Patients with a bleeding tendency or history of deep venous thrombosis are less
desirable candidates for this type of surgery.
Poor healing capacity, diabetes, gross obesity, and smoking are relative
contraindications for tibial tubercle transfer.
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FIGURE 3-1
Anteromedial tibial tubercle transfer.
Clinical examination is important in preoperative planning for anteromedial tibial
tubercle transfer. The examination should include compression of the patella while
flexing and extending the knee to see if articular pain can be elicited (Fig. 3-2). The
examining surgeon should also rule out other nonarticular causes of pain such as
neuroma, isolated retinacular pain, patellar tendonitis, referred pain, plica, meniscus
derangement, synovitis, and osteochondritis dissecans. The physical examination
should be done with the patient in both standing and supine positions. The examiner
should look for evidence of lateralization of the extensor mechanism and tilt. Passive
patellar tilt and tightness of the lateral retinaculum should be noted (2). Pain on
compression of the patella with flexion and extension of the knee is likely to have an
articular source. If this is associated with lateralization and lateral tilt of the patella,
the patient may be a candidate for anteromedial tibial tubercle transfer, particularly
if the compression pain is prominent in early knee flexion, indicating a distal patella
articular lesion that will be completely unloaded on anteriorization of the tibial
tubercle. It is important to be certain that pain is not due to a retinacular neuroma,
painful scar, or intra-articular cause that might be corrected by a smaller surgical
procedure or injection.
Standard anteroposterior and lateral radiographs should be taken on all surgical

candidates and supplemented with an axial (tangential) view. A standard axial (10)
view (knee flexed 45 degrees and x-ray beam 30 degrees from the horizontal plane)
provides a good idea of patella orientation in the trochlea and is our view of choice. If
a specific malalignment pattern is identifiable on the axial radiograph, no further
radiographic evaluation is necessary. On the other hand, some patients with significant
malalignment of the extensor mechanism will demonstrate a normal axial radiograph
yet have significant lateral subluxation and/or tilt on computed tomography (CT) of
the patellofemoral joint.
A true lateral radiograph of the knee (posterior condyles superimposed) at 0 and 30

degrees knee flexion will help greatly in understanding trochlea morphology from top
to bottom.
CT of the patellofemoral joint should be done with normal standing alignment of the
patient reproduced in the scanner gantry. Midpatellar transverse tomographic images
should be made with the
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CT cut directed to include the posterior condyles of the femur (Fig. 3-3). These
tomographic slices should be taken with the knees flexed 15, 30, and 45 degrees (5).
The patella is significantly tilted if the patellar tilt angle (the angle created by lines
drawn along the lateral facet of the patella and the posterior femoral condyles) is less
than 12 degrees on the tomographic image with the knee flexed 15 degrees. The
patella should be centered in the trochlea on the tomographic image in which the
knee is flexed at 15 degrees. (In other words, the congruence angle [10] should be 0 or
slightly negative). Furthermore, CT provides an ideal opportunity to understand the
relationship of the tibial tubercle to the central trochlea (TT-TG index as described by
Goutallier [7]). As the TT-TG index rises, TT transfer becomes increasingly desirable,
particularly when it exceeds 20 mm.
FIGURE 3-2
Compression of the patella while passing the knee through a range of motion will
give an impression of the extent of articular damage and pain emanating from the
articular surfaces of the patellofemoral joint.
Following full clinical evaluation and radiographic study, the surgeon will know
whether the patella is malaligned and will have some idea of the degree of articular
degeneration, particularly as demonstrated by pain and crepitation on
flexion/extension of the knee while compressing the patella. If appropriate
nonsurgical treatment has failed, the patient may be a candidate for anteromedial
tibial tubercle transfer, given significant malalignment (tilt and/or subluxation) and
evidence of pain coming from the patellar articular surface (particularly if articular
degeneration is distal and/or lateral).
SURGERY
The patient is positioned supine on the operating room table, and the anesthesiologist
may apply a percutaneous nerve stimulator over the femoral nerve at the groin level.
A tourniquet is applied on the proximal thigh and the patient is prepped from the
tourniquet level down to and including the toes. Impermeable drapes are used.
Technique
An arthroscopy is routine at the outset of anteromedial tibial tubercle transfer. A
superomedial portal, two finger-breadths above the medial proximal pole of the
patella, allows an excellent view of the patellar articular surfaces (Fig. 3-4), but
standard arthroscopic evaluation using medial and lateral infrapatellar portals is often
sufficient. By distending the knee to 60 mm Hg pressure, the surgeon can determine
the extent of articular damage as well as the extent and location of articular
breakdown. Reducing the fluid inflow pressure gives an idea of the extent of patellar
subluxation and/or tilt with flexion and extension of the knee. The femoral nerve may
be stimulated (to brief tetany) with the knee in progressive flexion to help determine
the dynamic alignment/malalignment. The arthroscopy is completed through an
infrapatellarportal to evaluate the rest of the knee and to confirm the patellar
findings noted if a proximal approach was used.
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FIGURE 3-3
Computed tomographic imaging of the patellofemoral joint should include precise

midpatellar transverse cuts directed through the posterior femoral condyles.
FIGURE 3-4
Arthroscopy of the patellofemoral joint using the superomedial approach gives an

excellent view of the entire patellar articular surface as well as the medial and
lateral recesses.
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The surgeon must first characterize and document (by print or video) the nature and
location of any articular lesion. A precise description of all articular lesions and their
location should be dictated later in the operation report.
Loose flaps of articular cartilage or fibrillated cartilage can be removed most easily at
the time of arthroscopy. Abrasion or resection of exposed sclerotic bone can also be
done at the time of arthroscopy or with the knee open at the time of anteromedial
tibial tubercle transfer. Drilling of the patella is best accomplished after lateral
release. Debridement of inflamed synovium, preferably using cautery, and removal of
loose bodies are best accomplished arthroscopically.
Most important at the time of arthroscopy is a critical assessment of whether

anteromedial tibial tubercle transfer will remove the loading from soft or fragmented
articular surfaces and transfer it to better cartilage. Experience has shown that
patients in whom the proximal medial facet is preserved are those most likely to
benefit from anteromedial tibial tubercle transfer. The operation effectively transfers
load from the more lateral and distal aspects of the patella onto proximal medial
articular cartilage. Since most patients with chronic patellar malalignment have
breakdown of the lateral facet or distal patella, anteromedial tibial tubercle transfer
is helpful to many if not most patients with patellar articular degeneration related to
chronic malalignment. Patients with dashboard injuries, which occur with the knee
flexed, often have destruction of proximal patellar cartilage and may be less amenable
to anteromedial tibial tubercle transfer, since this operation transfers load onto
proximal and medial patellar cartilage. Failure of straight medial tibial tubercle
transfer may result from a failure to unload a distal patella articular pain source.
In performing an anteromedial tibial tubercle transfer, a longitudinal incision is made

immediately along the patellar tendon and extended to a point 6 cm distal to the
tibial tubercle at the anterior midline (Fig. 3-5). The patellar tendon is identified by
sharp dissection (Fig. 3-6) and a lateral retinacular release is accomplished, taking
care to dissect away the subcutaneous tissues (Fig. 3-7) bluntly and to release all
components of the lateral retinaculum (6) and distal vastus lateralis obliquus (9), but
not the main vastus lateralis tendon. In fact, it is best to do a limited lateral release,
only to the level of the top of the patella, in most patients, to minimize the risk of
creating a medial subluxation problem. At this time also release any indurated fat pad
that might tether the distal patella and limit free movement of the patella anteriorly
after the osteotomy is completed. Inspect the patellar surface and debride carefully to
remove loose cartilage flaps while abrading or drilling eburnated bone (Fig. 3-8). Also
at this time, determine the need for any autologous, allogeneic, or synthetic
osteochondral allograft resurfacing. Place Kelly clamp behind the patellar tendon (Fig.
3-9), and incise longitudinally into the periosteum parallel to the anterior crest of the
tibia, just medial to the crest and lateral at the muscle origin of the anterior
compartment, reflecting the periosteum and muscle belly to expose the entire lateral
tibia for 8 to 10 cm. Periosteum is elevated carefully, and a drill guide is used to
define the osteotomy plane, allowing a cut close to the anterior tibial crest medially
(Fig. 3-10). A special drill guide with a cutting slot, such as the Tracker guide (Mitek),
can make this process easier. The drill bits are pointed posterolaterally, with the most
proximal drill bit penetrating the lateral tibial cortex under direct visualization and
with careful retraction of the tibialis anterior (Fig. 3-11). Multiple parallel drill holes
are made (Figs. 3-12 and 3-13). The angle can be modified according to the desired
degree of anteriorization and medialization of the tibial tuberosity. The drill bits
should be aligned to limit the depth of the distal tibial pedicle tip to 2 mm, so that
fracture of the distal pedicle will be possible. Great care must be taken to avoid drill
penetration too far posteriorly into the posterior neurovascular structures. Although
the pedicle of bone deep to the tibial tubercle is fairly thick, it tapers anteriorly to
the tip of the pedicle distally.
FIGURE 3-5
The incision for anteromedial tibial tubercle transfer should extend from the
lateral patella to a point 6 cm distal to the tibial tubercle at the midline.
FIGURE 3-6
The patellar tendon is identified by sharp dissection.
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FIGURE 3-7
The lateral retinacular release should extend through the entire retinaculum,
with care taken to release any tight fibrous bands in the retropatellar fat-pad
area. The lateral release should extend only to the superior pole of the patella,
avoiding the main tendon of the vastus lateralis. Do not overrelease. Leaving
some of the proximal muscle/vastus lateralis obliquus attached to the main vastus
lateralis tendon is a good idea in most cases.
FIGURE 3-8
The patella can be inspected thoroughly following lateral release.

FIGURE 3-9
The patellar tendon insertion is carefully identified and elevated with a Kelly
clamp.
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FIGURE 3-10
A: A drill guide allowing parallel drill placement along the osteotomy plane allows
for precise definition of the osteotomy plane. It is most important to taper the
osteotomy distally toward the anterior cortex, leaving only 1 to 2 mm of cortical
bone at the distal anterior aspect of the bone pedicle. B: The Mitek Tracker guide
is ideal for this purpose.
An oscillating saw or osteotomes are used to carefully fashion a cut connecting the
drill holes (Figs. 3-14 and 3-15), adding an oblique cut from the most proximal drill
hole to a spot just proximal to the insertion of the lateral patellar tendon (Fig. 3-16).
This avoids cutting into the broad metaphyseal region of the tibia. The osteotomy
must be made in the defined plane only, and care should be taken to work at all times
in the osteotomy plane. The cut is completed at about 5 to 8 cm from the tibial
tuberosity. The length of this bone pedicle should provide good surface contact and
avoid tilting the fragment medially. With great care, the bone fragment is carefully
mobilized (Fig. 3-16) and displaced anteromedially along the osteotomy plane (Fig. 3-
17). If advancement of the tuberosity is desired, a segment of the distal pedicle may
be removed and the remaining fragment advanced slightly to compensate for patella
alta. This may also be desirable if significant laxity of the patellar tendon is noted
following anteromedialization or if there is patella alta. Once the best position for the
tibial tuberosity is determined, a drill hole is made through the pedicle and into the
posterior cortex of the tibia distal to the tuberosity, and a cortical lag screw is passed
into the posterior cortex of the tibia, applying only slight compression (Fig. 3-18). The
anterior tibial cortex must be overdrilled slightly to create a slight lag effect, and a
second screw is added for additional stability, also using lag technique (Fig. 3-19).
Anteriorization of the tibial tuberosity by about 15 mm is most desirable when there is
a primary patellofemoral arthritis problem. In some patients, if subluxation persists,
careful advancement of the medial patellofemoral ligament and vastus medialis
obliquus may be desirable to selectively, but not so much that the patella is pulled
posteriorly or a medial articular lesion is loaded.
FIGURE 3-11
Exiting of the drill bits at the lateral tibial cortex must be directly visualized by
carefully retracting the tibialis anterior muscle from the lateral tibia. The deep
peroneal nerve and anterior tibial artery are just behind the posterolateral corner
of the tibia.
FIGURE 3-12
The obliquity of the osteotomy plane is demonstrated.
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FIGURE 3-13
Great care must be taken to place the drill bits parallel to each other so that an
accurate osteotomy will be defined before cutting the anterior tibial bone
pedicle. The Mitek Tracker guide works very well for this purpose and has a
cutting slot for an oscillating saw.
FIGURE 3-14
The proximal and distal drill bits are left in place to help define the plane while
cutting the bone pedicle.
FIGURE 3-15
A sharp, broad osteotome works well for fashioning the osteotomy. The surgeon
may wish to use an oscillating saw.
FIGURE 3-16
An osteotome or oscillating saw is used to connect the proximal drill bit tip with
the tibial bone posterior and proximal to the patellar tendon insertion. Full
visualization of the cutting tool as it exits the lateral tibia is mandatory. Note
taper of the osteotomy toward the anterior cortex.
FIGURE 3-17
The bone pedicle will be displaced in an anteromedial direction, usually giving 12

to 17 mm of anteriorization as well as some medialization, depending on the
obliquity of the osteotomy.
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FIGURE 3-18
Cortical lag screws are used to lock the pedicle in the corrected position, placing
the screws into the posterior tibial cortex for secure fixation. Keep the proximal
screw at least 1 to 2 cm distal to the patella tendon insertion.
When careful surgical technique is used, technical problems are uncommon. The
surgeon must be careful to taper the distal osteotomy anteriorly so as not to remove
too much of the tibial diaphyseal bone at the distal extent of the osteotomy. The
osteotomy should be very flat, thereby maintaining excellent bone-to-bone contact on
transfer of the bone pedicle. All drill holes must be made very carefully, taking care to
avoid the deep peroneal nerve and anterior tibial artery just behind the proximal
P.34
posterolateral tibia. Once the bone pedicle has been shifted in an anteromedial
direction, it must be held securely in the corrected position both while making the
initial drill hole and subsequently. Shifting of the transferred pedicle may make
engagement of the posterior cortex difficult and necessitate extra drill holes, thereby
weakening the posterior tibia. Once the first screw is secured to the posterior cortex,
the pedicle is quite secure and the second screw is easy to place.
FIGURE 3-19
In most cases, two cortical lag screws are used to give firm fixation of the
transferred bone pedicle.
FIGURE 3-20
A local bone graft taken from the tibial metaphysis may be placed in the
osteotomy plane, thereby displacing the osteotomy in an anterolateral direction,
producing net anterior displacement of the tibial tubercle. Direct anteriorization
with less bone graft can be achieved using this technique. A sagittal cut in the
tibia as described in the text will also permit straight anteriorization.
The surgeon may choose to use an â€œoffset bone graftâ€• (Fig. 3-20) if straight
anterior displacement of the tibial tubercle is needed. By placing a 4- to 5-mm-thick
section of corticocancellous auto- or allograft bone in the oblique osteotomy plane,
any medial displacement of the tibial tubercle will be â€œoffsetâ€• and slight
additional anteriorization of the tubercle achieved. If the patella appears to be
located too medially following anteromedial tibial tubercle transfer, an offset bone
graft may be added. Also, because this gives additional anteriorization, the offset
bone graft may be used selectively if there is need to further unload the distal, medial
portion of the patella. Care must be used when an offset bone graft is added in the
osteotomy site. Because this adds to anteriorization, the surgeon must be careful not
to anteriorize excessively. In our experience, 2 cm of anteriorization is maximal to
avoid undue â€œtentingâ€• of the skin and risk of skin slough. In a consecutive series
of over 600 anteromedial tibial tubercle transfers, I have not had a skin slough.
Another option for achieving straight anteriorization is to make a sagittal plane cut
into the tibia, followed by a back cut from the lateral side, both tapered to an
anterior point distally, thereby permitting a straight anterior transfer of the tibial
tubercle along the osteotomy plane. This transferred bone pedicle can then be fixated
with medial-lateral cortical screws.
The skin should close without tension, provided that no bone graft is added. We use a
full subcutaneous closure with 2-0 absorbable suture, followed by skin clips or
subcuticular closure and the application of full-length 0.5-inch Steri-strips, followed by
infiltration with 0.5% Marcaine. A suction drain may be used but is not usually
necessary. If one anteriorizes more than 2 cm, one may wish to consult preoperatively
with a plastic surgeon.
Using careful surgical technique, a precise flat osteotomy, secure fixation of the
transferred pedicle with two screws, appropriate anteriorization, meticulous
hemostasis, appropriate patient selection, early range of motion, and good
postoperative rehabilitation, anteromedial tibial tubercle transfer will give uniformly
good results.
Assuming secure two-screw fixation of the transferred bone pedicle (as described),
patients are started on immediate, once-daily active and passive range-of-motion
exercises of the knee but are maintained in a knee immobilizer for 4 weeks on
crutches. Continuous passive motion has not been necessary except in unusual
circumstances, such as concomitant release of arthrofibrosis. Cryotherapy is helpful in
the immediate postoperative period. A cryocuff is applied immediately after surgery
(Fig. 3-21) and maintained for 3 to 4 days. This has helped diminish pain and facilitate
early mobilization by minimizing swelling and pain.
Elevation is encouraged for the first 72 hours, and patients are allowed to ambulate
with toe-touch weight bearing, using crutches. The drain, if used, is removed at 18 to
24 hours after surgery if bleeding has subsided. Most patients are given antibiotic
coverage preoperatively, and this may be continued
P.35
for 5 days selectively. We recommend postoperative aspirin daily for 6 to 8 weeks.
Most patients can be discharged the day of surgery as long as they are mobile, swelling
and pain are controlled, and there is no evidence of fever, calf tenderness, or any
complication. Most patients need only a light dressing and knee immobilizer (4 weeks)
over the wound as long as they are competent and appropriately cautious on crutches.
The immobilizer should be taken off once each day to permit knee flexion. After 4
weeks, the patient may switch to a Tru-pull brace. Eighty-nine to ninety percent of
patients have an objectively good or excellent result (4). Those patients with more
advanced articular breakdown, however, can expect a 75% chance of a good result.
Excellent results appear to be uncommon in patients with extensive articular
breakdown. With 5 years minimum follow-up, results are maintained in most patients.
Follow-up for 8 to 10 years has shown sustained improvement.
FIGURE 3-21
Cryotherapy will help reduce swelling and pain postoperatively. A Hemovac may
be left in the wound for 2 to 24 hours, as needed.
Some patients complain of prolonged discomfort along the anterior aspect of the tibia.
Generally, this resolves by 6 to 12 months from the time of surgery. Most patients have
some discomfort from the screws, and removal of the screws at 6 to 12 months after
surgery is common. Most patients benefit from physical therapy, but some can recover
on a home program, progressing to exercise bicycling, swimming, and progressive
physical activity as tolerated after healing of the transferred bone pedicle. Active and
passive motion are important in the first few weeks after surgery, and full range of
motion of the knee should be achieved by 4 to 8 weeks from the time of surgery.
Physical therapy may be very helpful to those patients with less motivation or who are
progressing slowly. Full active work on quadriceps strengthening will generally start at
8 to 12 weeks from the time of surgery, but light quadriceps exercise, leg lifts, and
range-of-motion exercises may be started immediately after surgery.
COMPLICATIONS, PEARLS, AND PITFALLS

Serious complications after properly performed anteromedial tibial tubercle transfer
have been very uncommon. The surgeon should watch carefully for evidence of
infection or thrombophlebitis. Although aspirin prophylaxis is routine, the threshold
for initiating coumadin should be low if there is any concern about thrombophlebitis.
Anterior compartment syndrome is a concern because of the subperiosteal elevation of
the tibialis anterior muscle from the lateral side of the tibia and open cancellous bone
from the osteotomy that can bleed into the anterior compartment. Although full
anterior or posterior fasciotomy has not been necessary in our experience, a low
threshold for performing fasciotomy must be maintained if a compartment syndrome is
suspected.
Technical complications include fracture through a screw hole, nonunion, tibial

fracture, neurovascular injury, deep venous thrombosis, overcorrection, skin slough,
undercorrection, and incisional neuroma. Another potential complication is failure to
achieve pain relief because of extensive articular degeneration.
P.36
Fracture of the tibia is avoidable by using careful technique but will require cast
immobilization if it does occur. The period of immobilization, however, should be kept
as short as possible.
In the event of undercorrection, the tubercle transfer procedure can be repeated at 6

to 12 months after the initial operation, with additional anteriorization and/or
medialization. After removing the screws, the operative technique is the same as that
used initially. This has been very uncommon.
In the event of overcorrection, careful lateral transfer of the healed tibial tubercle
may be necessary. This should not be undertaken until 6 to 12 months from the time of
the initial surgery, and it is best to watch the patellar tracking carefully, with femoral
nerve stimulation used at the time of surgery to balance the patella in the trochlea.
Medial subluxation, as a dynamic complication of patella realignment surgery, is

always possible. Such patients may not have an obvious medial alignment of the
patella. Instead, they express a devastating and very painful feeling of the patella
jumping out of place (from too far medial back into the trochlea) upon early knee
flexion in gait. The astute examiner will detect this by holding the patella medially
with the knee extended, then abruptly flexing the knee while releasing the patella
simultaneously. This reproduces the event that the afflicted patient experiences. This
complication can be controlled by using a Trupull brace (DJOtho, Vista, CA), holding
the patella from medial to lateral. If necessary, a repair of released lateral tissues,
with or without a tenodesis on the lateral side, can control this problem.
Nonunion of the transferred pedicle may be treated by removal of the fixation screws,
debridement of fibrous tissue in the osteotomy site, exposure of bleeding bone,
flattening of the osteotomy plane to allow improved apposition of the pedicle to
underlying bone, cancellous bone grafting, and refixation with two cortical lag screws.
Although stiffness is uncommon with the use of immediate postoperative range-of-

motion exercises and ambulation, it may be treated if necessary by postoperative
manipulation at 8 to 12 weeks after surgery, followed by physical therapy.
Avoiding Fracture
The key to avoiding fracture and non union after anteromedial tibial tubercle transfer
osteotomy is to taper a precise osteotomy cut, at its distal extent, towards the
anterior tibial cortex. Also, the surgeon should avoid the proximal posterior osteotomy
extending into the posterior tibia. In other words, do not let the osteotomy extend
into or past the posterior, lateral corner of the tibia. Excellent visualization and a
precise, flat, single plane osteotomy are of paramount importance in achieving the
optimal osteotomy. Furthermore, excellent bone-bone contact and secure cortical
screw fixation (2 screws), well seated into good bone, using excellent technique (lag
effect, countersink screw heads, precise drill holes with secure grasp of posterior
cortex) will improve the likelihood of the desired stable fixation. Proper technique
permits early motion, earlier full weight bearing, primary bone-bone healing quickly,
an excellent result, and a happy patient.
RECOMMENDED READING
Fulkerson J. Diagnosis and treatment of patients with patellofemoral pain. Am J Sports
Med. 2002;30(3):447-456. Saleh K, Arendt E, Eldridge J, et al. Operative treatment of
patellofemoral arthritis. J Bone Joint Surg 2005;87A;659-671.
REFERENCES
1. Carney J, Mologne T, Muldoon M, et al. Long-term evaluation of the Roux-
Elmslie-Trillat procedure for patellar instability. Am J Sports Med.
2005;33(8):1220-1223.
2. Fulkerson J. Awareness of the retinaculum in evaluating patellofemoral pain.

Am J Sports Med. 1982;10:147.
3. Fulkerson J. Diagnosis and treatment of patients with patellofemoral pain. Am J

Sports Med. 2002;30(3):447-456.
4. Fulkerson J. Disorders of the patellofemoral joint. Philadelphia: Lippincott

Williams & Wilkins; 2004.
5. Fulkerson J, Becker G, Meany J, et al. Anteromedial tibial tubercle transfer

without bone graft. Am J Sports Med. 1990;18:490-497.
6. Fulkerson J, Gossling H. Anatomy of the knee joint lateral retinaculum. Clin

Orthop. 1980;153:183-188.
7. Goutallier D, Bernageau J, Lecudonnec B. The measurement of the tibial
tuberosity patella groove distance: technique and results. Rev Chir Orthop
Reparatrice Appar Mot. 1978;64:423-428.
8. Hallisey M, Doherty N, Bennett W, et al. Anatomy of the junction of the vastus

lateralis tendon and the patella. J Bone Joint Surg. 1987;69A:545-549.
9. Powers CM, Ward SR, Chen YJ, et al. The effect of bracing on patellofemoral
joint stress during free and fast walking. Am J Sports Med. 2004;32(1):224-231.
10. Merchant A, Mercer R, Jacobsen R, et al. Roentgenographic analysis of

patellofemoral congruence. J Bone Joint Surg. 1974;56A:1391-1396.
11. Trillat A, Dejour H, Coutette A. Diagnostique et traitement des subluxations

recidivantes de la routle. Rev Chir Orthop. 1964;50:813-824.
Patellectomy
4
Patellectomy
Christopher S. Proctor
Douglas W. Jackson
The role of patellectomy in the treatment of patellofemoral disorders is a subject of
controversy. The popularity of this procedure was great initially when there was little
understanding of its biomechanics. Today, indications for patellectomy have narrowed
with the knowledge that the patella is vital to the normal function of the knee joint;
now more emphasis is placed on its preservation.
Major functions of the patella include lengthening the quadriceps lever arm, thereby
facilitating knee extension (8,10,11,14,15); decreasing the compressive stresses on the
patellofemoral joint; and providing a smooth, nearly frictionless surface that is
resistant to wear (16). Patellectomy has been shown to alter knee-joint mechanics.
Clinically, the most apparent effect of patellectomy is a decrease in the quadriceps
muscle lever arm, necessitating a 30% increase in force to maintain the same torque
during knee extension (10,11). We believe that every effort should be made to
preserve the patella and have found that alternatives to patellectomy exist (12,13).
Patellectomy has been described as the treatment for a variety of patellofemoral

disorders (7,9,13,15). Primary patellectomy is often the treatment of choice for cases
of trauma with severe comminution not amenable to osteosynthesis, and for rare
tumors of the patella. However, when evaluating patellar fractures consideration
should first be given to obtaining an adequate reduction with internal fixation. Partial
patellectomy is the surgical procedure chosen for comminuted fractures of the patella
that are not amenable to osteosynthesis but in which one pole of the patella can be
saved. In the debate over which pole of the patella to save, we recommend saving the
pole with the greatest amount of articular cartilage, irrespective of which pole this is.
Better results and postoperative function are obtained if more of the patella can be
saved (5,6,17).
Despite the limited indications as a primary procedure, patellectomy can be a useful

secondary or â€œsalvageâ€• procedure. Satisfactory results have been described for
the following disorders:
Chondromalacia patella
Recurrent patellar dislocations
Patellofemoral osteoarthritis
Patellar osteomyelitis
Failed prosthetic replacement of the patella
The indication for patellectomy in these conditions is limited to those cases with
persistent pain and disability not relieved by other avenues of treatment, including
vigorous rehabilitation, medical treatment, arthroscopic surgery, and surgery to
realign the extensor mechanism. If pain and disability continue despite these
measures, a patellectomy may be considered.
A complete history and physical examination are required as part of the preoperative
evaluation. Radiographs obtained should include anteroposterior, lateral, and
Merchant views of the knee. The diagnosis of patellar pain is made after other causes
of knee pain are ruled out. Other treatment options should be exhausted, including
rehabilitation, medical treatment, and limited surgical management. If pain and
disability persist, a patellectomy may be considered.
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SURGERY
Complete Patellectomy
Patient Positioning
The patient is in a supine position on the operating room table. A well-padded
tourniquet is applied on the proximal thigh, and the patient is prepped from the
tourniquet level down the entire lower extremity with a Betadine (povidone/iodine)
scrub. Drape the lower extremity free from the level of the mid thigh.
Surgieal Procedure
Make a midline anterior incision through the skin and subcutaneous tissue starting 5
cm above the superior pole of the patella and proceeding distally to below the level of
the tibial tubercle (Fig. 4-1). We favor a midline longitudinal incision, since it is not
limiting to future surgical procedures. Elevate medial and lateral full-thickness skin
flaps using blunt dissection to expose the quadriceps, patella, and patellar tendon.
Split the midportion of the quadriceps and patellar tendons and the expansion of the
extensor mechanism over the patella in line with their fibers (Fig. 4-2). Place
retractors proximal and distal into the joint to protect the trochlear articular surface,
and then split the patella longitudinally into equal halves with an osteotome or saw
(Fig. 4-3). Grasp each half of the patella in turn with a towel clip and enucleate it
from the quadriceps mechanism (Fig. 4-4). When enucleating the patella, use sharp
dissection starting from the under surface of the patellar and quadriceps tendons. This
will preserve the expansion of the quadriceps tendon over the patella and preserve the
continuity of the quadriceps and petellar tendons. Once the patella is removed,
inspect the anterior compartment of the knee joint; remove any loose bodies and
address any other intra-articular pathology at this time. If indicated, the patellar
tendon may be split to the tibial tubercle and the quadriceps tendon split 10 cm
proximal to the patella. Release the tourniquet and obtain hemostasis before closing
the rent in the tendon. Use nonabsorbable, interrupted figureeight sutures to close
one side of the tendon over the other in a pants-over-vest fashion so that a double row
of sutures is obtained (Fig. 4-5). Once the tendon is repaired, flex the knee to 90
degrees to ensure that there is no undue stress on the suture line and that the tendon
tracks in the midline of the trochlear groove. If lateral displacement of the patellar
tendon is noted, advance the vastus medialis obliquus laterally and distally over the
sutured defect. If the tendon is still not centralized, consider performing a limited
lateral release. Close the subcutaneous tissue with interrupted absorbable sutures and
the skin with absorbable suture material using a running subcuticular technique. Cover
the wound with a sterile dressing and apply a long leg-support hose. Place the lower
extremity in a knee immobilizer or a brace locked in extension.
FIGURE 4-1
Make a midline incision anteriorly starting 5 cm above the superior pole of the
patella and proceeding distally to below the level of the tibial tubercle.
FIGURE 4-2
Mark the midline of the quadriceps tendon, patellar tendon, and extensor
expansion over the patella and split longitudinally in line with the fibers.
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FIGURE 4-3
Place retractors underneath the patellar and quadriceps tendons to protect the
underlying articular surfaces. Then split the patella longitudinally with an
osteotome or saw into two equal halves.
FIGURE 4-4
Grasp the patella with a towel clip and sharply enucleate it, taking care to
preserve intact the surrounding soft tissues.
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FIGURE 4-5
A: Retract the lateral one half of the extensor mechanism and suture the medial
one half to its undersurface. B: Fold the lateral one half over the top of the
medial portion and suture in place in a pants-over-vest fashion.
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Partial Patellectomy
The surgical technique for the treatment of comminuted fractures of the patella by
partial patellectomy differs from the technique described earlier for total
patellectomy. Make an identical approach to expose the patella, but divide the
extensor apparatus over the patella transversely (Fig. 4-6). Retain the largest fragment
of the patella and subchondral surface, and remove all remaining fragments by sharp
dissection, taking care to cause as little injury to the soft tissues as possible (Fig. 4-7).
Debride and make smooth the edges of the remaining patellar fragment. Next, drill
parallel holes transversely across the patella such that they exit at the junction of the
anterior and middle thirds of the patella (Fig. 4-8). Suture the patellar or quadriceps
tendon to the anterior one third of the retained fragment using No. 1 nonabsorbable
suture and a horizontal mattress technique (Fig. 4-9). Although some authors
recommend suturing the tendon to the posterior one third of the patella at the
articular margin (6,13), we obtain superior results by attaching the tendon to the
anterior one third of the patella. Recent experimental data confirm that an anterior
reattachment minimizes the decrease in patellofemoral contact area and the
increased contact pressure associated with patellectomy (16). Next, carefully repair
the expansion of the extensor apparatus over the patella using 3-0 absorbable sutures.
The remainder of the surgical technique is as described earlier; postoperatively, place
the extremity in a knee immobilizer or locked knee brace for 6 weeks.
FIGURE 4-6
For a partial patellectomy use a midline incision, as for total patellectomy. Divide
the extensor apparatus transversely to expose the patellar fragments.
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FIGURE 4-7
Retain the larger fragment and remove the smaller fragments via sharp
dissection, causing as little soft-tissue damage as possible.
Pearls and Pitfalls
The trochlear cartilage can easily be damaged when splitting the patella
longitudinally into halves. It is, therefore, important to protect the underlying
cartilage with retractors.
Preserving the expansion of the quadriceps tendon over the patella is

important for successful reconstruction.
With partial patellectomy, the best results are obtained by anatomically

reconstructing the patellar or quadriceps tendon to the anterior edge of the
patella.
FIGURE 4-8
Drill holes transversely across the patella (A) such that they exit the patella at
the junction of its anterior and middle thirds (B); then suture the tendon to the
center of the patella using a horizontal mattress technique.
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FIGURE 4-9
The tendon is sutured to the anterior one third of the retained fragment. This
minimizes the decrease in patellofemoral contact area and the increase in
contact pressure associated with partial patellectomy.
Complete Patellectomy
Maintain the knee in a knee immobilizer or brace locked in extension. On
postoperative day 1, start quadriceps-strengthening exercises. Our preference is to
remove the immobilizer on postoperative day 5 and begin passive flexion and active
extension. Protective weight bearing is allowed in the immediate postoperative
period. Encourage progression to full, unprotected weight bearing when adequate
quadriceps strength is demonstrated. We expect that most patients will obtain at least
90 degrees of passive flexion by the second to third postoperative week, and full
active flexion by 8 to 10 weeks. Full extension with good muscle power is slower to
obtain and often requires as long as 5 to 6 months of an active, supervised exercise
program.
Partial Patellectomy
Maintain the knee in a knee immobilizer or locked knee brace for 3 weeks, allowing
weight bearing as tolerated. Begin straight leg-raising exercises during the first
postoperative week. Passive flexion exercises are started at 3 weeks. We expect a full
range of knee flexion in 8 to 10 weeks with full extension established by 3 to 4 months.
Loss of 10 to 20 degrees of symmetric flexion may still be associated with a functional
recovery.
P.44
COMPLICATIONS
Although patellectomy may be unavoidable in certain cases, the change in anatomy
has significant consequences.
Extensor lag/weak extensor power (20,21) is the most common complication.

The lever mechanism of the quad muscle becomes less efficient without the
patella, and it may result in failure to achieve full extension.
Decreased range of motion is often seen following long periods of

immobilization and with transverse repair of the quadriceps and patellar
tendons (2). If motion is not restored with aggressive rehabilitation, an
arthroscopic lysis of adhesions and re-establishment of the suprapatellar notch
should be coupled with a gentle manipulation under anesthesia. This should be
done as soon as it becomes apparent that no progress restoring motion is being
made over a 2- to 4-week period postoperatively.
Continued anterior knee pain is not uncommon in patients who have undergone
patellectomy. Treatment choices for patients are to have the patient live with
the pain or to try anteriorization of the tibial tubercle in selected cases.
One of the more serious complications following patellectomy is disruption of

tendon repair. This most commonly occurs with transverse repairs of the
tendon; it may also be seen in longitudinal repairs in association with
infection.
Subluxation of the extensor mechanism can arise with failure to recognize and
correct malalignment (3). The loss of the spacer effect of the patella leaves
the quadriceps tendon prone to dislocation. The increase in laxity leaves the
femoral joint surface vulnerable without the bony protection of the patella.
The femoral cartilage is easily damaged by knocks and falls.
Damage to the infrapatellar branch of the saphenous nerve may occur during
patellectomy. If the nerve is cut, the identified end should be buried in fat in a
nonprominent area to minimize the formation of a symptomatic neuroma.
Calcification at the patellectomy site has been described, but we have found it
to be of little consequence (3,6).
RESULTS
As stated previously, patellectomy has a deleterious effect on normal knee function
and may be unpredictable in providing pain relief in terms of the patient's underlying
disease. Recent studies have demonstrated good or excellent results in only about 53%
to 72% of patients (1,13,15,16). When evaluating the results of patellectomy, it is
important to remember that they may be different for different conditions (7).
Furthermore, it is important to note that better results are obtained with
patellectomy if the patellofemoral joint is the sole joint involved.
In our hands, the results of patellectomy for these indications have been variable.
Patients are not symptom-free, are seldom able to do running or jumping activities,
and often have difficulty with normal gait progression on stairs. In patients with
otherwise normal extensor mechanisms, patellectomy for severely comminuted
patellar fractures and for failed prosthetic replacements of the patella provides more
satisfactory short- and long-term results. Justifiably a terminal procedure, a well-
performed patellectomy is, for selected patients, a consideration in the management
of severe patellofemoral pain.
RECOMMENDED READING
Jensen DB, Hansen LB. Patellectomy for chondromalacia. Acta Orthop Scand.
1989;60:17-19.
Marder RA, Swanson TV, Sharkey NA, et al. Effects of partial patellectomy and
reattachment of the patellar tendon on patellofemoral contact areas and pressures. J
Bone Joint Surg. 1993;75:35-45.
REFERENCES
1. Ackroyd CE, Polyzoides AJ. Patellectomy for osteoarthritis. J Bone Joint Surg.
1978;60:353-357.
2. Baker CL, Hughston JC. Miyakawa patellectomy. J Bone Joint Surg.

1988;70:1489-1494.
3. Boucher HH. Results of excision of the patella. J Bone Joint Surg. 1952;34:516.
4. Boyd HB, Hawkins BL. Patellectomyâ€”a simplified technique. Surg Gynecol

Obstet. 1948;86:357-358.
5. BrÃ¶strom A. Fractures of the patella. Acta Orthop Scand. 1972;143(suppl):1.
6. Duthie HL, Hutchinson JR. The results of partial and total excision of the
patella. J Bone Joint Surg. 1975;40:75.
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7. Fulkerson JP, Hungerford DS. Disorders of the patellofemoral joint. 2nd ed.
Baltimore: Williams & Wilkins; 1990.
8. Haxton H. The function of the patella and the effects of its excision. Surg
Gynecol Obstet. 1945;80:389.
9. Ivey FM, Bazina ME, Fox JM, et al. Reoperation following patellectomy for
chondromalacia. Orthopaedics. 1979;2:136-137.
10. Kaufer H. Mechanical function of the patella. J Bone Joint Surg. 1971;53:1551-
1560.
11. Kaufer H. Patellar biomechanics. Clin Orthop. 1979;144:51-54.
12. Kelly MA, Insall JN. Patellectomy. Orthop Clin North Am. 1986;17:289-295.
13. Kelly MA, Proctor CS. Patellectomy. Semin Orthop. 1990;4:149-154.
14. Lewis MN, Fitzgerald PF, Jacobs B, et al. Patellectomy. An analysis of one
hundred cases. J Bone Joint Surg. 1976;58A:736.
15. Maquet PCJ. Biomechanics of the knee with application to the pathogenesis
and surgical treatment of osteoarthritis. Berlin; Springer-Verlag; 1976.
16. Mow VC, Proctor CS, Kelly MA. Biomechanics of articular cartilage. In: Nordin
M, Frankel, V, eds. Basic biomechanics of the musculoskeletal system. 2nd ed.
Philadelphia: Lea and Febiger, 1989:31-58.
17. Saltzman CL, Goulet JA, McClellan T, et al. Results of treatment of displaced
patellar fractures by partial patellectomy. J Bone Joint Surg. 1990;72A:1279-1285.
18. Sutton FS, Thompson CH, Lipke J, et al. The effect of patellectomy on knee
function. J Bone Joint Surg. 1976;58:537-540.
19. Watkins MP, Harris BS, Wender S, et al. Effect of patellectomy on the function
of the quadriceps and hamstrings. J Bone Joint Surg. 1983;65A:390-395.
> Table of Contents > Part One - Extensor Mechanismâ€”Patellofemoral Problems > 5 - Patella
and/or Extensor Mechanism Allograft Reconstruction
5
Patella and/or Extensor Mechanism Allograft
Reconstruction
William H. Warden III
Douglas W. Jackson
INDICATIONS
A chronic dysfunctional extensor mechanism with an associated symptomatic extensor
lag that persists after multiple repair or reconstruction attempts may be amenable to
allograft reconstruction. Potential allograft techniques for reconstructing a portion or
all of the extensor mechanism involve using an allograft that includes the quadriceps
tendon with or without a patella, patellar tendon, and tibial tubercle. The allograft is
used to fill defects and provide continuity by replacing or supplementing deficient
tissue. If the patient's patella is usable, we prefer using an Achilles tendon allograft
with attached bone block to replace the nonfunctional quadriceps and/or patellar
tendon.
Extensor mechanism disruption can occur as result of a traumatic event, tendon

degeneration, or as a complication of knee surgery. Primary and secondary techniques
for repair or reconstruction of specific portions of a deficient extensor mechanism
have been described and are often successful. If multiple attempts at direct repair and
reconstruction fail and the patient still has an extensor lag, allograft replacement of
the extensor mechanism becomes a consideration.
The main indication is a symptomatic extension lag related to a deficiency in the
extensor mechanism that has failed at least one primary repair and/or reconstruction
using autogenous tissue, for example, failed quadriceps and patellar tendon repairs
with large palpable tendon defects. Other causes of irreparable extensor deficits
include disruption of the extensor mechanism during or following procedures such as:
patellectomy
total knee arthroplasty
patellar realignment procedures
lysis of adhesions for arthrofibrosis
correction of patella infera or infrapatellar adhesions
P.48
CONTRAINDICATIONS
Current infection in the knee or underlying bone
An extensor mechanism that can be reconstructed using primary repair and/or

reconstruction with local autogenous tissue
A patient who does not wish to accept restricted knee flexion
A patient who is unwilling to accept to accept a minimum of 6 to 8 weeks or

longer of bracing and an involved postoperative rehabilitation
A patient who is averse to possibly a manipulation, arthroscopic lysis of

adhesions, and/or further open repair of portions of the graft
A patient who is not willing to accept the possibility of effusion and low-grade
inflammatory response that may persist for a long period after a large allograft
(entire extensor mechanism)
A patient who is unwilling to accept that an allograft may fail or need to be

removed
A history and physical examination of the knee and extremity includes a review of the
duration and severity of the patient's symptoms and extensor mechanism dysfunction
in relation to the patient's surgical history. Particular attention is given to prior
extensor mechanism procedures and includes the number performed and the success
or failure of each of those surgeries. Prior operative reports are reviewed; previous
history of infection is explored as well as associated potential risk factors (e.g.,
diabetes, renal failure, collagen diseases, immune deficiencies, chronic medications,
and smoking history). These and any factors that may delay wound healing are
discussed with the patient. The current symptoms related to the extensor lag,
episodes and frequency of giving-way, disability with stairs, and arising from the
seated position are documented.
The complete physical examination of the lower extremities includes documentation

of active and passive range of motion, evaluation of the use of assisted walking
devices, and gait pattern. Special attention is paid to the prior surgical incisions over
and around the knee. In addition, the degree of extensor lag and any restriction in
passive extension (fixed flexion contractures) are documented. The tracking of the
extensor mechanism during active and passive range-of-motion testing is assessed. The
ideal candidate for this reconstruction will have 120 degrees or more of active flexion.
Radiographs include four views of the knee with standing (anteroposterior and 30
degrees flexed), lateral, and Merchant views. These are evaluated for degenerative
arthritis in all three compartments, as well as for patella infera, patella alta, and the
presence or absence of a patella. They are also evaluated for hardware and
hetereotopic bone. If an associated knee replacement has been performed, it is
assessed for alignment, loosening, and the status of the patella, if present, and its
alignment. Further studies may be necessary to evaluate rotational alignment if
malrotation is a contributing factor to the extensor mechanism dysfunction.
Laboratory studies may include an erythrocyte sedimentation rate, serum C-reactive

protein level, and white blood cell count if there is a possibility of chronic infection. If
an effusion is present, a knee aspiration is performed and sent for cell count, crystal
analysis, and synovial fluid cultures. Radiographs are sent to the tissue bank for sizing
of the entire allograft if a patella allograft is to be part of the reconstructed extensor
mechanism.
SURGERY
Technique
Ancef 1 g (or alternative antibiotic) is given intravenously 1 hour before surgery if
there is no history of allergy or other contraindication. The knee to be reconstructed is
marked and observed by professional staff. This is done even when the patient has
multiple scars and a palpable defect at the surgical site. The patient is placed in the
supine position on the operating table with a pneumatic tourniquet high around the
thigh of the involved lower extremity that has been marked and again checked in the
operating room by the staff and physician. The extremity is then prepped and draped
separately in the usual orthopaedic manner.
The previous surgical incisions as well as a line for the incision are marked with
methylene blue before the application of the Steri-Drape. Once ready for the incision,
the tourniquet is inflated after extremity elevation and with the knee flexed 90
degrees. We prefer a midline skin incision if possible;
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however, we may use a previous incision and extend it as needed proximally and/or
distally to obtain adequate exposure. If there are multiple incisions present, it is our
preference to use the most lateral incision and not cross a previous surgical incision in
a manner that may compromise the blood supply to a section of skin. The dissection is
carried down in the midline elevating skin and subcutaneous flaps, with consideration
for blood supply preservation determining the extent of exposure. The medial and
lateral patellar retinaculum, quadriceps, and patellar tendon are exposed. The medial
and lateral gutters and suprapatellar pouch spaces are re-established. The defect(s) in
the extensor mechanism is defined. The midline incision is carried proximally into the
host quadriceps maintaining a medial and lateral margin of tendon to suture to in the
repair. This can be difficult if there is minimal host quadriceps tendon remaining
proximally. The incision is carried in the midline distally to expose the host tibial
tubercle. It is important to maintain as much of the host soft tissue medial and lateral
to the incision as possible.
The allograft specimen to be used is prepared on a side table (see Chapter 17,
Allograft Preparation for ACL Reconstruction) during the surgical exposure and
preparation for the eventual graft. The trough in the patient's tibial tubercle is not
prepared until the allograft tibial block has been shaped and sized. The allograft tibial
block shape is matched to a rectangular trough drawn out with methylene blue. It is
cut out using a micro-saw and will be approximately 6 cm long, 2 cm wide, and 2 cm
deep. The allograft block is further trimmed and downsized once the graft site is
prepared to facilitate a press-fit (Fig. 5-1). Two No. 2 Ethibond sutures as described by
Krackow et al are placed along the medial and lateral aspects of the allograft proximal
quadriceps tendon (5). These sutures are used to apply traction and test how well the
allograft bone is secured to the tibial bed. Once prepared, the graft is wrapped with a
sterile saline-soaked sponge and stored in a protected area until ready for
implantation.
The host proximal tibial trough is positioned to provide the desired height of the
patella in relation to the femoral sulcus and in comparison to the contralateral patella
height. Ideally, the tibial tubercle anatomy is duplicated by the new patella tendon
block at the original insertion and level. Care is taken to leave the proximal
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portion of the host tibia trough intact as it serves as the buttress against proximal
migration. The rectangular tibial trough is marked out in methylene blue and cut to a
length of 5 cm, a width of just less than 2 cm, and a depth of 2 cm. The final prepared
host site is slightly smaller than the allograft bone block, to allow a press-fit placed
with use of a bone punch. Once the allograft tibial bone is press fit, it is drilled and
tapped for a cortical screw that further fixes and compresses it into the host tibia (Fig.
5-2). Usually one well-placed screw gives good fixation. We do not use a washer and
slightly countersink the head so that it is flush. The traction placed on the graft is
perpendicular to the graft. The press-fit, well-shaped proximal tibial buttress and
screw fixation can tolerate greater forces than the graft will experience for several
weeks. The tibial bone graft is biologically fixed within 6 to 8 weeks and has not been
a problem in late displacement or migration. We confirm the screw length and tibial
position of the graft using a mini-C arm image intensifier at this point before
proceeding with the soft tissue fixation and tensioning (Fig. 5-3). Patients with at total
knee and tibial stems may require other fixation techniques. However, we have found
slightly angled screw fixation satisfactory in the limited cases we have done following
total knee replacement. In those patients who have had their extensor mechanisms
reconstructed before their total knee replacement, we have simply removed the screw
at the time of surgery. Alternate fixation of the tibial block should be considered if
the extensor mechanism surgery is done simultaneously with the total knee
replacement revision.
FIGURE 5-1
An artist's depiction of a tibial-patellar tendon-patella-quadriceps graft (A) and

an Achilles graft (B). The bone plugs are approximately 6Ã—2Ã—2 cm in size.
It is important to prepare and maintain medial and lateral soft-tissue sleeves along the
entire area the graft will touch. Our preference is to deflate the tourniquet before
soft tissue suturing. The sleeves (adjacent residual tendon, retinaculum, and soft
tissue) are sutured to and used to cover the allograft where possible (Fig. 5-4). The
side-to-side sutures provide additional fixation until the allografts are incorporated.
When they are approximated to the allograft, the quadriceps sutures are pulled
snuggly with the knee in full extension. It is important to place the sutures securely
into the remaining host quadriceps tendon and secure the proximal aspect of the
allograft. The medial and lateral side-to-side closure is then completed. When possible
the repair of the host retinaculum is brought over the top of the allograft. It is often
difficult to get complete coverage if an allograft patella is used in the reconstruction.
If sutures and bulky tissue are left on the surface of the patella, they can be a source
of constant irritation.
Prior to closure, we prefer to flex the knee at least 45 degrees (Fig. 5-5), directly
visualizing the repair, patella tracking, and bone block. This should occur without
sutures pulling out and without displacement of the distal bone block. Even in the
knee brace locked in extension, there will be up to 20 degrees of knee motion possible
during the healing phase. We want to make certain the graft repair will hold until
biologic fixation takes over (estimated 4 to 6 weeks). The subcutaneous tissues are
closed in routine fashion. The skin is closed with staples unless the skin is fragile and
then 3-0 nylon vertical mattress sutures are used.
FIGURE 5-2
Once the allograft tibial bone is press fit, it is drilled, tapped, and slightly
countersunk for a cortical screw that further fixes and compresses it into the host
tibia.
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FIGURE 5-3
We confirm the screw length and tibial position of the graft using a mini-C arm
image intensifier at this point before proceeding with the soft tissue fixation and
tensioning. This is a postoperative radiograph of an allograft patellar
reconstruction in a patient with a previous Maquet procedure.
FIGURE 5-4
Adjacent residual tendon, retinaculum, and soft tissue are sutured to and used to
cover the allograft. The side-to-side sutures provide additional fixation until the
allograft is incorporated.
FIGURE 5-5
Prior to closure, the knee is flexed at least 45 degrees, assessing fixation.
P.52
Pearls and Pitfalls
We prefer a fresh-frozen, nonirradiated allograft specimen from a well-

established tissue bank consisting of a quadriceps tendon, patella, patellar
tendon, and tibial bone. It is preferable to have the patella sized in
comparison to the contralateral patella and the recipient femoral groove.
Be sure to specify right or left in your allograft request and check that you
have an allograft for the appropriate side. Contralateral images or magnetic
resonance images used for sizing can lead to confusion! Furthermore, it can be
difficult to distinguish a right patella from a left.
Have at least 5 cm of quadriceps tendon allograft for suture repair into the
host quadriceps mechanism. This requires talking with the tissue bank or their
representative. We like to see a drawing and the graft dimensions.
If the patient's patella is acceptable and can be used, we prefer using an

Achilles tendon with attached bone block for the reconstruction.
If the native patella is present, we prefer to leave it and use soft tissue around
and over it. An effort should be made to maintain what blood supply there is to
the existing patella.
The most difficult reconstructions have been those that have had more than
one quadriceps repair attempted with very little or no functional host
quadriceps tendon to repair to. Preoperative magnetic resonance imaging of
the quadriceps tendon is helpful in determining whether this is a potential
problem.
It is important that the knee be able to be passively brought to full extension

preoperatively or at minimum at the time of surgery.
We prefer to press fit the allograft tibial bone into the recipient bone trough
and transfix it with at least one cortical screw (see Fig. 5-2). Care should be
taken to have an adequate bone block that will not split with the drilling,
tapping, and screw placement. We have not had difficulty with migration if
attention is given to placement and fixation.
The allograft is sutured in place tensioned with the knee in extension.
As much of the patient's adjacent soft tissue (retinaculum and tendon) as

possible is sutured to the graft to obtain additional fixation.
During the repair and at the completion of placing the allograft, we prefer to
flex the knee under direct visualization of the repair. We want at least 45
degrees of flexion with the fixation and sutures holding well.
The knee is immobilized in a brace in extension and weight bearing is

progressed as tolerated in extension.
The knee is placed in a hinged brace locked in full extension. We place it over a long
leg support stocking that holds the dressings in place. We maintain the knee in full
extension within the brace, realizing there is some passive motion in the brace. The
patient is started the following day on toe-touch weight bearing and allowed to
progress to full weight bearing as tolerated in the brace with the knee in extension.
Staples are removed 2 weeks after surgery assuming the wounds are closed and healing
as desired. We encourage isometric static quadriceps contractions. It is usually 2 to 3
weeks or longer before the patient is comfortable bearing full weight in the brace
around the house. We encourage crutches when outside for 4 to 6 weeks. After 6
weeks 30 degrees of active flexion is permitted, under the supervision of a physical
therapist, with the patient wearing a hinged knee brace with a lockout against further
flexion. During weight bearing, we lock the brace in full extension. The brace is worn
for 8 weeks when ambulating and sleeping. It can be off during the day for periods of
time. At 12 weeks, we allow further active flexion up to a maximum of 90 degrees,
and gentle quadriceps strengthening exercises are initiated. Passive flexion is limited
to minimize the chance of graft failure or early attenuation. If there is concern over
the tissue quality or fixation at the quadriceps, we are more conservative with our
rehabilitation.
P.53
COMPLICATIONS
Extensor mechanism allografts are a large transplant of bone (patella and tibia) and
soft tissue to the front of the knee. Although fresh-frozen allografts do not have living
cells within the transplanted tissue, they do contain cellular remains including residual
nuclear material. These donor cellular residuals have to be degraded further and
removed by the host. This occurs as the tissue is repopulated by recipient cells.
Certain individuals may have significantly more inflammatory reactions, with chronic
effusions and malaise for several weeks. It is possible that the inflammatory and/or
low-grade immune reaction would be so significant the graft would need to be
removed.
If the host does not have sufficient residual proximal quadriceps tendon to hold
sutures, the allograft quadriceps tendon can pull away from the quadriceps muscle
resulting in an extension lag. There is often loss of some knee flexion as a result of
prolonged immobilization associated with the secondary reconstruction of the
quadriceps tendon portion of the allograft. Knee flexion may not be completely re-
established even after rigorous physical therapy, surgical lysis of adhesions, and/or
manipulation under anesthesia.
RESULTS
The patients that may benefit from an allograft reconstruction of their extensor
mechanism represent a varied population. There are those with a deficient quadriceps
tendon and patella tendon that have failed recurrent attempts at primary repair and
reconstruction using autogenous tissue. The repairs may require replacement of tissue
that both covers and allows fixation to the existing patella. The patella tendon may
benefit from fixation into the tibial bone at its attachment site. Allograft extensor
mechanism reconstructions are usually reasonable in re-establishing full extension but
may limit the patient's flexion range. Patients are usually willing to accept this if they
have extension power back. These patients often have had multiple previous surgeries
and have underlying degenerative articular cartilage disease. They do not end up with
a normal knee but a more functional knee.
Reconstruction with the patella, quadriceps, and patellar tendon is a much more
extensive procedure than a soft tissue reconstruction. The patella takes a long time to
revascularize, if ever (Fig. 5-6). The underlying articular cartilage will degenerate with
time. The graft requires matching to the underlying femoral sulcus for tracking and
sizing. This is a salvage procedure. Once again the goals are eliminating the extension
lag enabling the patient to be more functional. It also prepares the patient for a
future knee replacement or helps salvage the current or revised one.
FIGURE 5-6
A biopsy specimen of a patellar allograft with no viable cells in the bone.
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RECOMMENDED READING
Burnett RS, Fornasier VL, Haydon CM, et al. Retrieval of a well-functioning extensor
mechanism allograft from a total knee arthroplasty. Clinical and histological findings. J
Bone Joint Surg Br. 2004;86:986-990.
Burnett RS, Berger RA, Paprosky WG, et al. Extensor mechanism allograft
reconstruction after total knee arthroplasty. A comparison of two techniques. J Bone
Joint Surg Am. 2004;86:2694-2699.
Emerson RH Jr, Head WC, Malinin TI. Extensor mechanism reconstruction with an
allograft after total knee arthroplasty. Clin Orthop Relat Res. 1994;303:79-85.
Jackson DW, Simon TM. Donor cell survival and repopulation after intraarticular
transplantation of tendon and ligament allografts. Microscopy Research and
Techniques. 2002;58(1):25-33.
Krackow KA, Thomas SC, Jones LC. Ligament-tendon fixation analysis of a new stitch
and comparison with standard techniques. Orthopedics. 1988;11:909-917.
> Table of Contents > Part One - Extensor Mechanismâ€”Patellofemoral Problems > 6 - Acute
Quadriceps Tendon Repair
6
Acute Quadriceps Tendon Repair
Christopher S. Proctor
Brant L. Richardson
Quadriceps tendon rupture is uncommon, occurring much more frequently in males
than in females (3,4,6) and more often in patients over 40 years of age. Rupture of
the quadriceps tendon is a serious injury that we believe should be surgically
repaired within 2 weeks of injury. Beyond 2 weeks, quadriceps contracture and
scarring can begin to make the repair more difficult, and postoperative strength
and patient satisfaction are adversely affected (4). Some studies have found a loss
of range of motion (ROM) with delayed repair as well (5,6). Complete ruptures that
go unrepaired produce a lower extremity with a poorly performing extensor
mechanism; however, even chronic tears stand to make significant gains through
repair (4). We feel that repair is indicated in all acute cases of complete rupture
other than the medically unstable patient and those patients who were not
ambulatory prior to the injury.
The first step in treating a quadriceps rupture is a prompt and accurate diagnosis.
Unfortunately, quadriceps tendon rupture often goes unrecognized in the family
practice and emergency setting, leading to delayed diagnoses and repair. Patients
with a quadriceps rupture will generally present with suprapatellar gap,
suprapatellar pain, and an inability to extend the knee. Diagnosis is complicated by
hematoma filling the suprapatellar gap and an unwillingness by some evaluators to
thoroughly palpate the tender area. Medical providers often rely on radiography,
which can show patella baja if bilateral radiographs are obtained, but more often
the case is a normal appearing unilateral series. In some patients, systemic medical
issues lead to tendon degeneration and relatively little pain on rupture with a low-
energy mechanism of injury. Bilateral ruptures are more likely to have a medical
predisposition for tears, such as autoimmune diseases, gout, diabetes mellitus,
rheumatoid arthritis, hyperparathyroidism, long-term steroid use, and end-stage
renal disease (1,2,3). Further complicating diagnosis is the case of tendon rupture
with an intact retinaculum, which allows weak knee extension to be maintained. If
the retinaculum is torn at all the patient will lose some ROM (6). Prompt diagnosis
and surgical repair will afford the best opportunity for a successful recovery after
quadriceps rupture.
A full history and physical examination are required as part of the preoperative
evaluation. Anteroposterior and lateral radiographs of the knee should also be
obtained. Magnetic resonance imaging (MRI) is helpful to determine the extent of
the tear and to prepare the surgeon for other injuries that may need to be
addressed; however, surgery should not be delayed to obtain an MRI.
SURGERY
Patient Positioning
We position the patient supine on the operating table with a well-padded
tourniquet fitted snugly high on the thigh. The entire lower extremity is then
prepared with Hibiclens solution, and the lower extremity is draped free from the
level of the mid thigh.
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Technique
A midline longitudinal incision is made starting 15 cm above the proximal patella
and is extended to the tibial tubercle. Medial and lateral full-thickness skin flaps
are elevated using blunt dissection to expose the extensor mechanism. If the
retinaculum is intact, it is split longitudinally. Irrigation is used to dislodge any
hematoma, allowing the tear to be assessed. The osteotendinous junction at the
patella is the most common site of the rupture. The tendon is then debrided of all
necrotic and frayed tissues. The quadriceps tendon is carefully mobilized from any
surrounding adhesions. Once tendon length has been maximized, we place sutures
in the tendon as follows: starting at the tear, No. 5 nonabsorbable sutures are
placed in a running Bunnel configuration along the edges of the tendon to 3 cm
proximal of the tear, then run back down closer to the midline (Fig. 6-1). The
anterior proximal pole of the patella is then debrided of soft tissue and lightly
roughened with a bur to provide a bed for the tendon. A 2-mm hole is drilled
longitudinally down the midline of the patella and out the distal pole. Two parallel
holes are drilled medially and laterally, in a similar fashion, approximately 1 cm
from the midline. A suture passer is then used to pass the two central sutures down
the central drill hole and the medial and lateral sutures through the medial and
lateral drill holes, respectively (Fig. 6-2). We then tie the sutures over the bone
bridge, securing the tendon to the patella. As much of the retinaculum as possible
is then repaired with No. 2-0 absorbable sutures.
More recently we have also been using suture anchors to secure the tendon to the
patella rather than drill holes. Using this technique, the tear is exposed and the
proximal pole of the patella is prepared as previously discussed. Two suture anchors
double loaded with No. 2 FiberWire sutures (Arthrex, Inc., Naples, FL) are then
inserted into the patella dividing it into approximately equal thirds (Fig. 6-3). Each
of the four sutures is then secured to the tendon using a modified Mason-Allen
technique. The remainder of the repair then proceeds as previously described. To
aid our postoperative planning, we then range the knee to determine how much the
knee can be flexed without overly stressing the repair. At this time, patellar
alignment is also assessed and lateral release performed if necessary.
FIGURE 6-1
Running sutures placed in a running Bunnel configuration, producing four free
strands.
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FIGURE 6-2
Sutures placed through three longitudinal drill holes in the patella are
tightened, pulling the quadriceps to the patella.
FIGURE 6-3
Suture anchors buried in the patella are made visible in this figure for clarity.
Each anchor is loaded with 2 pairs of FiberWire sutures; the sutures are tied to
the tendon using a modified Mason-Allen technique.
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Pearls and Pitfalls
Tension across the suture line is minimized during repair by extending the knee
completely, deflating the tourniquet, and pulling the tendon distally. Careful
attention must be paid to anatomic alignment of the quadriceps tendon and patella
to help prevent future patellofemoral complications. Repair of the lateral
retinaculum can be forgone and a more extensive lateral release performed if
patellar alignment demands it.
We believe it is important for patients to take an early and active role in their
recovery to give them a sense of control while they are disabled and to promote
the mind-set that their full recovery relies on their own hard work in physical
therapy. Postoperatively, we keep the leg locked in extension with a dial lock brace
and keep the patient non-weight bearing until they are examined at 1 week. After
the first week, the patient is allowed to partially weight bear with crutches.
Patients are encouraged to do quad sets and straight leg raises at home and are
started in physical therapy for gentle, passive ROM at 2 weeks. At 3 weeks, patients
are made full weight bearing but continue to be locked in extension. At rest, they
are encouraged to unlock their brace and work on passive ROM. At 6 weeks, active
ROM is begun. Patients are allowed to ambulate with the brace unlocked up to
their active ROM. Progressive increases of 10 to 15 degrees per week should yield
full ROM by 12 to 16 weeks. Patients begin full resistance exercise, move on to light
jogging, and by 20 weeks can start full running.
COMPLICATIONS
Although most patients regain a functional ROM, loss of motion is the most common
problem after quadriceps repair. Patients must work diligently in physical therapy
for a prolonged period to regain full function, and many find it easier to live with a
less-than-perfect lower extremity. Atrophy of the extensor mechanism is also not
uncommon but is usually not associated with overt weakness compared to the
uninjured leg (7). Delayed surgery increases the likelihood of extensor lag, need for
ambulatory aids, and poor stair-climbing ability (7).
RESULTS
Quadriceps tendon rupture is an infrequent but serious injury most often occurring
in men over 40 years of age. Most patients recover full ROM, near full strength, and
are able to return to their former occupation. If the quadriceps rupture is
diagnosed and repaired promptly, most patients can expect a satisfactory return of
extensor mechanism function. Rigorous, extended physical therapy is necessary for
complete recovery and a return to sports, although 50% of patients perform sports
at a decreased level. It is not surprising that any delay in surgical repair can lead to
increased weakness and disability postoperatively.
RECOMMENDED READING
Gelberman R, Woo SL, Buckwalter JA. Injury and repair of the musculoskeletal soft
tissues. Amer Acad Ortho Surg. 1988;21-23.
Ramseier LE, Werner CML, Heinzelmann M. Quadriceps and patellar tendon rupture.
Int J Care Injured. 2006;37:516-519.
Richards DP, Barber AF. Repair of quadriceps tendon ruptures using suture anchors.
J Arthro Rel Surg. 2002;18:556-559.
Woo SL, Vogrin TM, Abramowitch SD. Healing and repair of ligament injuries in the
knee. J Am Acad Orthop Surg. 2000;8:364-372.
REFERENCES
1. Ilan DI, Tejwani N, Keschner M, et al. Quadriceps tendon rupture. J Am Acad
Orthop Surg. 2003;11(3):192-200.
2. Konrath AG, Chen D, Lock T, et al. Outcomes following repair of quadriceps

tendon rupture. J Orthop Trauma. 1998;12:273-279.
3. Puranik GS, Faraj A. Outcomes of quadriceps tendon repair. Acta Orthop

Belg. 2006;72:176-178.
4. Rougraff BT, Reek CC, Essenmacher J. Complete quadriceps tendon ruptures.

Orthopedics. 1996;19:509-514.
5. Scuderi G. Ruptures of the quadriceps tendon: study of 20 tendon ruptures.

Am J Surg. 1958;91;626-635.
6. Siwek CW, Rao JP. Ruptures of the extensor mechanism of the knee joint. J
Bone Joint Surg. 1981;63:932-937.
Patellar Tendon Repair
7
Patellar Tendon Repair
Albert M. Tsai
Isolated rupture of the patellar tendon is a rare injury occurring primarily in patients
less than 40 years old. Most ruptures occur as the result of a rapid forceful contraction
of the quadriceps muscle with a flexed knee, and a history of stumbling or giving way
of the knee can often be elicited along with a sudden pop and acute onset of pain. It
has been demonstrated that normal tendons usually do not rupture without significant
trauma (2). A certain degree of tendon degeneration is often present, the result of
cumulative microtrauma or the degenerative changes associated with aging. The injury
can also be associated with systemic diseases, such as chronic renal failure,
rheumatoid arthritis, systemic lupus erythematosus, or diabetes mellitus, and occurs
with little or no trauma in these patients. Rarely, iatrogenic patellar tendon rupture
has been reported after harvesting the middle third of the tendon for anterior cruciate
ligament reconstruction (1,3).
Surgical repair of an acutely ruptured patellar tendon is the standard of care and is
necessary to re-establish continuity of the extensor mechanism of the knee. Neglected
injuries can lead to proximal retraction of the quadriceps and patella with resultant
adhesions and extensor mechanism insufficiency. Contraindications to acute surgical
repair include acute infection, medical comorbidities that make the surgical risk
prohibitive, or soft tissue loss that makes primary repair impossible.
The first step in assessing the injured knee is a thorough history and physical
examination. There is generally swelling and ecchymosis about the knee as well as
tenderness along the patellar tendon or at the inferior aspect of the patella. There
may also be a palpable defect in the tendon. Occasionally the patient will be able to
perform a straight leg raise maneuver through an intact extensor retinaculum.
However, the diagnosis should still be apparent as there will usually be an extensor lag
and weakness when compared to the uninjured extremity.
Imaging of the knee should include routine radiographs to rule out fractures. On a
lateral view of the knee, patella alta can be seen (Fig. 7-1). Magnetic resonance
imaging (MRI) is not usually necessary in acute injuries but may be helpful in chronic
neglected ruptures, in those few acute cases where the diagnosis is unclear, or to
diagnose suspected concomitant intra-articular pathology (Fig. 7-2).
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FIGURE 7-1
Lateral radiograph of patellar tendon rupture demonstrating patella alta.

FIGURE 7-2
Magnetic resonance imaging clearly shows disruption of the patellar tendon. MRI
was performed in this patient because of associated injuries to the anterior
cruciate ligament, posterior cruciate ligament, and posterolateral corner.
SURGERY
Patient Positioning
The patient is placed in the supine position, and a small bump is placed under the
ipsilateral hip to keep the patella pointing toward the ceiling. A tourniquet is placed
on the proximal thigh but is usually not inflated during the procedure. The entire
extremity is then sterilely prepped and draped free.
Technique
A midline skin incision is made centered over the defect in the patellar tendon. If the
disruption is an avulsion off the inferior pole of the patella, the incision may extend
more proximally. In the case of a midsubstance tear or tibial tubercle avulsion, the
incision may extend more distally. Routine dissection is carried sharply down through
skin and subcutaneous tissue. At this point, the knee joint is frequently visualized
through the ruptured tendon. The joint is thoroughly irrigated to remove old
hematoma and soft tissue debris, and the articular surfaces can be examined for any
injury. If possible, the paratenon should be identified and incised to further dissect
out the patellar tendon in preparation for repair.
Most commonly, the patellar tendon is avulsed from the inferior aspect of the patella,
along with tears of the medial and lateral retinaculum (Fig. 7-3). The frayed, tattered
stump of patellar tendon is sharply debrided with dissecting scissors or a sharp scalpel
to freshen up the tendon and remove any nonviable tissue. Next, a Rongeur is used to
debride the inferior pole of the patella, removing soft tissue remnants and providing a
healthy bleeding bone surface for the repaired tendon. A total of two No. 2 FiberWire
(Arthrex, Inc., Naples, FL) sutures are woven into the end of the tendon using a
Krackow-type locking stitch. One suture is woven into the medial half of the tendon,
working
P.61
from proximal to distal and then back up in the medial central portion of the tendon.
The second suture is similarly woven down the lateral half of the tendon and back up
the lateral central portion of the tendon, leaving four suture strands exiting the
tendon (Fig. 7-4). These sutures are marked with hemostats so that they can later be
passed through drill holes in the patella. For ease of identification, the central sutures
can be cut shorter than the peripheral ones and clamped together (Fig. 7-5).
FIGURE 7-3
In this patient there is a near complete avulsion of the patellar tendon from the
inferior aspect of the patella. The clamp lies behind the proximal patellar
tendon.
Next, a total of three parallel drill holes are made in the patella from distal to
proximal, using a 2mm drill bit. When drilling, the flat end of an Army-Navy retractor
can be used to locate the drill bit as it exits the superior border of the patella and the
quadriceps tendon (Fig. 7-6). After withdrawing the drill bit, often it is difficult to
locate the hole in the superior aspect of the patella to place a suture passer from
proximal to distal. Instead, a suture passer is used from distal to proximal to pass a
loop of 0 PDS suture. This loop of PDS can then be used to pass the FiberWire sutures
through their appropriate drill holes (Fig. 7-7). The lateral suture is passed through the
lateral drill hole, the medial suture through the medial drill hole, and the two shorter
central sutures through the central drill hole. After passing these sutures, they can be
tied with the knee in extension, the medial suture to the medial central suture and
the lateral suture to the lateral central suture (Fig. 7-8). The tendon should contact
the previously prepared inferior pole of the patella, but care should be taken not to
overtighten the repair and cause patella baja. If there is any question, an
intraoperative lateral radiograph can be taken and compared to the contralateral
knee.
The medial and lateral retinacular defects are then closed with No. 1 absorbable
suture. A No. 2-0 absorbable suture is used for the paratenon, if possible, as well as
the subcutaneous layer, and surgical staples or a running subcuticular stitch are used
to approximate the skin. A routine sterile dressing is placed and the patient is placed
into a range of motion brace, which is locked in extension.
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FIGURE 7-4
A,B: No. 2 FiberWire suture is woven into the stump of patellar tendon using
medial and lateral Krackow locking stitches. C: Cadaver specimen demonstrating
medial and lateral Krackow locking stitches for patellar tendon rupture.
P.63
FIGURE 7-5
The central strands of the medial and lateral Krackow stitches can be cut short
and clamped together for ease of identification.
A similar surgical technique is used for patellar tendon disruption from the tibial
tubercle, except the sutures are passed through transosseous tunnels in the tubercle
and tied over medial and lateral bony bridges. For a midsubstance tendon rupture,
direct tendon-to-tendon repair is performed with a total of four No. 2 FiberWire
sutures-medial and lateral Krackow-type locking stitches in both the proximal and
distal tendon stumps (Fig. 7-9). The patellar tendon suture line is oversewn with
interrupted No. 2-0 absorbable suture. Retinacular repair and closure are the same as
for proximal avulsion injuries.
FIGURE 7-6
An Army-Navy retractor helps locate the exiting drill bit.
P.64
FIGURE 7-7
A: Hewson suture passer used from distal to proximal. B,C,D: Loop of 0 PDS suture
passed from proximal to distal. E,F: FiberWire suture passed through drill hole
using PDS loop.
P.65
FIGURE 7-8
A,B: Cadaver specimen demonstrating patellar tendon repair with sutures passed
through patellar drill holes. C: Schematic illustrating patellar tendon repair.
P.66
FIGURE 7-9
Midsubstance patellar tendon repair with sutures.
Pearls and Pitfalls

When drilling the three parallel drill holes, take care to start the holes closer
to the articular surface rather than the dorsal surface of the patella. This will
prevent inward tilting of the distal pole of the patella when the repair is
tightened, which could lead to increased contact pressure in the
patellofemoral joint.
After drilling holes through the patella, it is difficult to pass a suture passer
from proximal to distal. Use a suture passer from distal to proximal to pass a
loop of 0 PDS. The loop can then be used to pass the FiberWire from distal to
proximal.
Overtightening the repair can lead to patella baja and a poor result. If in
doubt, obtain an intraoperative lateral radiograph and compare it to the
contralateral knee.
A good repair of the medial and lateral retinacular tissue is a key portion of the
procedure. This relieves much of the stress on the central repair and allows for
aggressive postoperative range of motion and strengthening.
Immediate full weight bearing is initiated after surgery with crutches and the range of
motion brace locked in extension. Isometric quadriceps and hamstring exercises are
also initiated on the first day after surgery. Surgical staples are removed in the office
at 10 to 14 days after surgery, at which time supervised physical therapy is begun with
active flexion and passive extension of the knee. The range of motion is started at 0 to
30 degrees and advanced 30 degrees every 2 weeks with the goal of 90 degrees at 6
weeks. The range of motion brace is gradually unlocked to coincide with the increases
in flexion allowed and is discontinued when 90 degrees of flexion is achieved at 6
weeks. A supervised strengthening program is then started, with full return to sports
activity prohibited until 6 months after surgery or until the patient demonstrates full
range of motion of the knee and at least 80% of the strength of the contralateral knee
with isokinetic testing.
P.67
COMPLICATIONS
Decreased Motion
Decreased range of motion is most likely a result of the initial injury itself, rather than
any technical problems related to the surgical procedure. To overcome this, a secure
repair allows an aggressive postoperative therapy program emphasizing early range of
motion and strengthening. Significant lack of motion at 2 to 3 months is uncommon
with acute primary repair but can be treated with manipulation under anesthesia to
improve flexion or arthroscopic lysis of adhesions for loss of passive extension.
Wound Problems
Wound dehiscence or infection is uncommon, but may occur distally, where skin
coverage is the thinnest. Local wound care and appropriate antibiotics if necessary are
usually sufficient to resolve these problems.
Hemarthrosis
Postoperative bleeding may cause an uncomfortable hemarthrosis, which usually
resolves spontaneously but can limit mobility and progress with postoperative physical
therapy. Meticulous hemostasis during surgery can help prevent this problem. If a
tourniquet is used, this should be deflated and hemostasis achieved prior to wound
closure.
Missed Diagnosis
Delayed treatment of a neglected patellar tendon rupture will often necessitate
reconstruction of the extensor mechanism. Lack of adequate soft tissue and native
tendon often precludes primary repair, and quadriceps muscle contracture and
adhesions often prevent the patella from being brought distally to an acceptable
position. During the preoperative evaluation, a lateral radiograph should be obtained
of the contralateral knee to be used in estimating patellar height during the
reconstruction.
Patient positioning is the same as for acute repair cases. A generous midline incision is
used to fully delineate the anatomy and address the quadriceps contracture. If
significant patella alta is present, the quadriceps mechanism may need fractional
lengthening with a pie-crusting technique or formal V-Y advancement. Either Achilles
or patellar tendon allograft can be used to reconstruct the extensor mechanism in the
absence of acceptable native tissue.
When using Achilles tendon allograft, a rectangular plug of calcaneus with attached
Achilles tendon is press fit into a matching rectangular trough made in the tibial
tubercle. This bone plug is fixed with 4.5-mm bicortical screws. The soft tissue of the
Achilles tendon is then split into two grafts and brought through two transosseous
patellar tunnels and sewn into the quadriceps mechanism (Fig. 7-10).
Alternatively, patellar tendon allograft allows bony fixation on both the tibial and
patellar sides. A large rectangular plug from the allograft tibia is inserted into a
matching trough in the patient's tibial tubercle and fixed with 4.5-mm bicortical
screws. On the patellar side, the inferior pole of the patient's patella is removed to
match a large portion of the inferior pole of the allograft patella that has been left
with the graft. Naturally, the appropriate size and length patellar tendon allograft
must be selected during the preoperative planning, which can be facilitated with a
lateral radiograph of the contralateral knee. Patellar fixation is achieved with
Kirschner wires or cannulated 4.0-mm screws and a figure-of-eight tension band wire
or No. 5 nonabsorbable suture (Fig. 7-11).
With reconstruction of chronic tears, a No. 5 nonabsorbable suture, or 5-mm Mersilene

tape placed through a transosseous drill hole in the tibial tubercle and then up and
over the patella through the quadriceps mechanism will help relieve stress on the
repair. A cerclage wire accomplishes the same task but frequently breaks when range
of motion is initiated, occasionally necessitating removal.
P.68
FIGURE 7-10
Achilles tendon allograft for patellar tendon reconstruction. The calcaneal bone
plug is fixed with 4.5-mm screws. The soft tissue is passed through 7-mm
transosseous tunnels and sutured to the quadriceps mechanism.
FIGURE 7-11
Patellar tendon reconstruction using patellar tendon allograft.
P.69
RESULTS
Results after patellar tendon repair are most closely correlated with the amount of
time that has passed between injury and surgical treatment. The overwhelming
majority of patients that undergo timely primary repair of an acute patellar tendon
rupture will have excellent results, with full range of motion and normal quadriceps
strength. Mild quadriceps atrophy may persist but should not affect the return of
strength. The results for patients that undergo delayed reconstruction are less
satisfactory, with loss of full knee flexion and a decrease in quadriceps strength more
likely. However, a functional extensor mechanism can still be re-established.
RECOMMENDED READINGS
Greis PE, Holmstrom MC, Lahav A. Surgical treatment options for patella tendon
rupture, Part I: acute. Orthopedics. 2005;28(7):672-679.
Greis PE, Lahav A, Holmstrom MC. Surgical treatment options for patella tendon
rupture, Part II: chronic. Orthopedics. 2005;28(8):765-769.
Kuechle DK, Stuart MJ. Isolated rupture of the patellar tendon in athletes. Am J Sports
Med. 1994;22(5):692-695.
Matava MJ. Patellar tendon ruptures. J Am Acad Orthop Surg. 1996;4(6):287-296.
REFERENCES
1. Busfield BT, Safran MR, Cannon WD. Extensor mechanism disruption after
contralateral middle-third patellar tendon harvest for anterior cruciate ligament
revision reconstruction. Arthroscopy. 2005;21(10):1268.e1-1268.e6.
2. Kannus P, Josza L. Histopathological changes preceding spontaneous rupture of

a tendon. A controlled study of 891 patients. J Bone Joint Surg Am. 1991;73:1507-
1525.
3. Marumoto JM, Mitsunaga MM, Richardson AB, et al. Late patellar tendon
ruptures after removal of the central third for anterior cruciate ligament
reconstruction. A report of two cases. Am J Sports Med. 1996:24(5):698-701.
> Table of Contents > Part Two - Meniscus Surgery > 8 - Meniscus Repair: Inside-Out Suture
Technique
8
Meniscus Repair: Inside-Out Suture Technique
Kevin F. Bonner
In an effort to preserve meniscal function, meniscus repair should be performed when
there is a reasonably good chance the meniscus will heal and maintain its function.
Patient age, activity level, knee stability, type, location, and chronicity of the tear
are important factors when considering meniscal repair versus resection. In general,
active patients under the age of 45 years with a longitudinal tear within 3 to 5 mm of
the meniscocapsular junction are good candidates for repair. Radial tears going to the
meniscocapsular junction are also typically repaired in a young patient (more common
lateral). The younger the patient, the more likely I am to attempt repairing a tear
considered to have marginal healing capacity. Degenerative type tears or tears with
minimal healing capacity are treated with partial meniscectomy. Anterior cruciate
ligament (ACL) reconstruction is strongly recommended in the setting of a repairable
meniscus tear and instability.
P.72
Although all-inside meniscus repair devices are becoming much more popular as the
result of their ease of use, the inside-out technique is the most versatile and still
considered the â€œgold standard.â€• Although I have also used suture-based all-inside
meniscus repair devices for several years, I still often use the inside-out repair
technique.
The diagnosis of a meniscus tear can be made with reasonable certainty with a
thorough history, physical examination, and radiographs. Magnetic resonance imaging
(MRI) is not required but is often helpful in differentiating between meniscal versus
articular cartilage pathology and in the assessment of associated injuries in the acute
setting. We can also get a fairly good idea whether a tear may be repairable based on
signal patterns; but certainly the final decision between meniscectomy and repair is
not made until the time of surgery (Fig. 8-1).
If a patient has a potentially repairable meniscus it is important to have all of the

equipment and personnel available. A skilled assistant is required to perform an inside-
out repair. Even for surgeons who prefer an all-inside repair technique, it is important
to be proficient and ready with the alternative technique of inside-out repair, since
some tears are not as amenable to all-inside repair. It is important to discuss with the
patient the risks and benefits, as well as the difference in the rehabilitation protocol,
for a meniscus repair versus a meniscectomy.
SURGERY
Preoperatively in the holding area, the correct side is confirmed and the patient's knee
is signed. Within 1 hour prior to surgery, the anesthesiologist intravenously administers
1 g (2 g if patient >80 kg) of a first-generation cephalosporin (Cefazolin) if the patient
is not allergic. Alternatively, the patient is given Clindamycin 600 to 900 mg
intravenously. Regional versus general anesthesia is decided by the patient and the
anesthesiologist.
Patient Positioning
The patient is placed in a supine position with the leg resting straight on the operating
room table. A tourniquet is placed on the proximal thigh, and lateral thigh post is used
to allow a valgus stress to improve visualization of the medial compartment. A pillow
is placed under the contralateral knee. The knee is positioned distal to the break in
the table, allowing posteromedial access if the foot of the table is flexed during the
procedure. After adequate anesthesia, an examination under anesthesia is performed
comparing both knees.
FIGURE 8-1
MRI of a potentially repairable medial meniscus tear in a 22-year-old woman.
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The portal sites and any additional incision sites are infiltrated with 0.25% Marcaine
with 1:200,000 epinephrine. The meniscus repair (with or without ACL reconstruction)
may be performed with or without a tourniquet. If a tourniquet is used, the leg is
exsanguinated with an esmark wrap, and the tourniquet is inflated to 300 mm Hg. If an
ACL reconstruction is to be performed in the same setting, I will harvest the graft prior
to performing knee arthroscopy. A diagnostic arthroscopy is performed first if there is
any uncertainty regarding the status of the ACL. Allografts are also used fairly
frequently in my practice. All of these grafts will be prepared at the back table by the
assistant. The assistant will work on the graft until he or she is needed for the
meniscus repair.
The diagnostic arthroscopy is started with placement of the anterolateral portal
adjacent to the patellar tendon 1 cm below the inferior pole of the patella. The
arthroscope is gently inserted into the joint through the anterolateral portal with the
knee in 70 to 90 degrees of flexion. With the arthroscope focused in the medial
compartment, the anteromedial portal is created with the aid of an 18-gauge spinal
needle. The anteromedial portal should be just to the medial side of the patellar
tendon. Depending on the side of the meniscus tear, the medial portal can be adjusted
appropriately. For a medial sided tear, the spinal needle is placed adjacent to the
patellar tendon just superior to the anterior horn of the medial meniscus confirming
optimal access to the posterior horn. If preoperatively it is determined that the lateral
meniscus is most likely going to require repair, I may delay making the anteromedial
portal until the knee is in the figure-of-4 position. In this setting, use the spinal needle
as a probe to help determine if a medial tear is present. Optimal placement of the
anteromedial portal for a lateral meniscus repair is typically more superior than for a
medial meniscus repair, since the suture cannula will need to avoid the tibial
eminence.
I will initially assess the tear and determine the feasibility of a successful repair. The
younger the patient the more prudent to attempt repair. A probe is used to
characterize the size of the tear, degree of instability, quality of the tissue, and zone
of the tear (i.e., red-red, red-white, white-white). The width and integrity of the
meniscal rim is evaluated in a far peripheral tear to determine if an all-inside device
may be an option. Far posterior tears may need to be assessed with a 70-degree scope
placed posterior through the notch. Once the diagnostic arthroscopy is complete, the
meniscus repair will proceed in a step-wise fashion.
Tear Preparation
A meniscal rasp (Acufex, Andover, MA) is used to debride granulation tissue on both
sides of the tear (Fig. 8-2). Alternatively, a 3.5-mm shaver may be used to debride the
edges of the tear site. It is important to take quite a bit of time in preparing the edges
to healthy tissue since this step is crucial in optimizing the healing potential. Rasp the
perimeniscal synovium both superior and inferior to the tear site. In the case of a
displaced repairable bucket handle tear, it is often useful to debride the edges of the
tear prior to reduction (Fig. 8-3). This will give you direct visualization and access to
both sides of the tear. Once the tear edges are abraded back to healthy tissue, the
meniscus is reduced into an anatomic position. After the meniscus site is prepared, the
posteromedial or posterolateral incision is created.
Preparation of a peripheral, far posterior tear may require access through a

posteromedial or posterolateral portal. These portals are made approximately 1 cm
above the joint line with the knee at 90 degrees of flexion. The 70 scope is placed
posterior through the notch, and a spinal needle is used to direct posterior portal
placement. Alternatively, I prefer to make a small portal through the capsule once the
posteromedial or posterolateral approach has been made. A rasp or shaver is used
through the posterior portal to access the tear site.
Technique for Medial Meniscus Repair

Optimal positioning of the skin incision can be enhanced with translumination of the
posteromedial joint line by placing the arthroscope in the medial compartment with
the room lights dimmed (Fig. 8-4). The knee should be flexed between 60 and 90
degrees for the exposure. A 3- to 4-cm vertical incision is created along the
posteromedial joint line posterior to the medial collateral ligament (Fig. 8-5).
Approximately two thirds of the incision should be distal to the joint line because the
needles tend to angle downward as they exit the meniscus and capsule. The saphenous
vein should be posterior to the incision and can often be seen by translumination
through the joint.
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FIGURE 8-2
A-C: Evaluation and preparation of a lateral meniscus tear using a meniscal rasp
(Acufex, Andover, MA) (D).
FIGURE 8-3
A: Displaced bucket handle medial meniscus tear in a 15-year-old. B: Tear edge

of the displaced fragment is prepared with a shaver prior to reduction. C:
Posterior rim is prepared with the fragment displaced anterior allowing optimal
visualization.
FIGURE 8-4
Translumination of the medial or lateral compartment may assist in planning

posterior incision site.
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FIGURE 8-5
A 3- to 4-cm incision is made posterior to the MCL. Two thirds of the incision is
below the joint line.
On dissection through the subcutaneous tissue, the oblique pes fascia is identified. An
incision is made anterior within the fascia, which should be split proximal and distal
using Metzenbaum scissors (Fig. 8-6). Care is taken to retract the sartorius, gracilis,
and semitendinosis tendons along with the saphenous nerve posteromedial (Fig. 8-7).
The saphenous nerve will lie within the posterior border of the sartorius and with
retraction of the tendons should be protected. Knee flexion will relax the pes tendons
and aid in retraction. If a hamstring autograft will be used for an ACL reconstruction,
it is very helpful to harvest the grafts first, allowing for much easier dissection and
retraction.
The deep dissection is continued in the interval between the medial head of the
gastrocnemius and the joint capsule. Keep the knee in flexion to relax the hamstrings
and the medial head of the gastrocnemius. Separating the muscle off the capsule is
facilitated by starting distal. Start with a pair of Metzenbaum scissors, followed by
placing an index finger to help expand this interval. By dorsiflexing and plantar flexing
the ankle, the surgeon should be able to palpate the gastrocnemius on the posterior
side by his or her finger (Fig. 8-8). This will assist in verifying the proper plane. Once
developed, a posterior retractor is positioned anterior to the medial gastrocnemius
muscle (Fig. 8-9). By retracting the medial head of the gastrocnemius lateral and
posterior, the neurovascular structures will be protected (Fig. 8-10). Various retractors
may be used; I prefer a Henning retractor or half of a pediatric speculum, although a
bent spoon can be used as a retractor as well (Fig. 8-11). The retractor needs to be
angled so the needles piercing through the posterior capsule are deflected medial
toward the assistant.
FIGURE 8-6
The pes fascia is identified, incised, and split proximal and distal.
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FIGURE 8-7
The saphenous nerve should be protected with posterior retraction of the pes
hamstring tendons.
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FIGURE 8-8
Confirmation of the proper interval anterior to the gastrocnemius can be made by

dorsiflexion and plantarflexion of the ankle. Palpate the motion of the
gastrocnemius on the posterior side of your finger.
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FIGURE 8-9
The posterior retractor is positioned revealing the posteromedial joint capsule.

The hamstrings and medial head of the gastrocnemius are posterior to the
retractor.
Once the retractor is placed, the knee is kept between 20 and 60 degrees of flexion.
Knee extension will enhance arthroscopic visualization of the posterior horn of the
meniscus, but consequently makes posterior retraction and visualization more
difficult. Increasing knee flexion relaxes the hamstrings and gastrocnemius, thus
improving posterior retraction and visualization of exiting needles.
I always prefer to place sutures into the medial meniscus through a cannula entering
the anterolateral portal. This angulation tends to improve the direction of exiting
needles toward the medial exposure. However, far posterior tears near the meniscal
root may require use of a curved cannula entering through the anteromedial portal
with the curve directed medial (away from midline). Sutures are typically placed from
posterior to anterior. Even if you begin with the suture cannula in the medial portal,
as the repair proceeds anterior it is easier to switch portals and place the majority of
sutures with the cannula entering from the anterolateral portal.
FIGURE 8-10
Retracting the medial head of the gastrocnemius lateral and posterior will protect
the neurovascular structures.
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FIGURE 8-11
Half of a pediatric speculum may be used as a posterior retractor.
Single- or double-lumen zone-specific suture cannulas are commercially available (Fig.

8-12). These come straight and with varying curvatures to address different zones of
the meniscus. I prefer single-lumen cannulas since they allow greater mobility and
independence when placing sutures. However, this does increase the risk of damage to
the suture as the second needle is being placed adjacent to the suture. Ten-inch
flexible needles with attached 2.0 Ethibond suture are used for the repair (Ethicon,
Inc., Somerville, NJ) (Fig. 8-13). I prefer nonabsorbable sutures, since there are data
to suggest these sutures may improve clinical healing rates.
Flexing the foot of the table will allow your assistant improved access to the
posteromedial exposure (Fig. 8-14). The assistant should â€œtoe inâ€• on the
retractor and be prepared to retrieve with a needle driver. With the surgeon applying
a valgus force to the knee and directing the cannula under arthroscopic visualization,
the inside-out sutures can be placed (Fig. 8-15).
Under direct visualization, place the suture cannula against the meniscus in the
desired location. I use a second assistant to help pass the needles through the cannula.
Pass the needle to the end of the cannula which is against the meniscus. Visualize the
tip of the needle by slightly retracting (3 to 5 mm) the cannula within the joint. Pierce
the unstable meniscus fragment with the needle tip. The needle can then be used to
aid in the tear reduction. Confirming the tear is anatomically reduced; proceed with
delivering the needle into the peripheral rim fragment and through the capsule. Place
the needles through the meniscus with the knee flexed approximately 10 to 20
degrees. Once the needle is through the meniscus, it is helpful to flex the knee to 45
to 60 degrees, which will facilitate posterior retrieval.
FIGURE 8-12
Commercially available single- and double-lumen suture cannulas (Acufex,

Andover, MA).
FIGURE 8-13
Ten-inch flexible needles with attached 2.0 Ethibond suture are used for the
repair (Ethicon, Inc., Somerville, NJ).
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FIGURE 8-14
The foot of the table is flexed to allow the assistant improved access to receive
needles through the medial approach.
Slowly advance the needles, paying strict attention to how far they are inserted. If the
needle is inserted approximately 1 to 1.5 cm and the needle tip cannot be visualized,
the needle is retracted and retractor placement is reassessed (look further inferior
and lateral). Sutures are typically placed from posterior to anterior. Alternatively, an
anterior suture may be placed giving the retrieving assistant a reference point for
placement of additional posterior sutures. Once the first needle tip is visualized, it is
grasped with the needle driver by the assistant and pulled out of the posterior capsule
(often bending the needles) (Fig. 8-16).
Once the first needle is delivered out of the incision, the single-barrel cannula is
redirected. To help avoid laceration to the suture, slight tension is applied to the
suture and the second needle is delivered down the cannula adjacent to the suture
(Fig. 8-17). In a similar fashion, the second needle is placed, thus creating either a
vertical, oblique, or horizontal stitch. Care is taken to ensure the sutures will
anatomically reduce the tear. Multiple sutures are passed in a similar fashion (Fig. 8-
18).
The assistant sequentially cuts off the needles and tags the sutures with a hemostat.
You can either tie as you go or clamp several sutures before tying. If greater than 3 or
4 sutures are placed, it is helpful to tie this group and then proceed, so as to avoid
tangling (Fig. 8-19). When tying medial sutures, the knee should be held in full
extension with the meniscus reduced under direct visualization. This will avoid
imbrication of the posteromedial capsule which may cause a postoperative flexion
contracture. Be careful not to overtighten the sutures.
FIGURE 8-15
A valgus stress is applied to the knee by the surgeon while holding the scope in
the medial portal and directing the suture cannula from the lateral portal.
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FIGURE 8-16
A: The first needle pierces through the posterior capsule. B: The 10-inch needle
is pulled through with a needle driver.
A vertical suture pattern is the preferred technique due to increased capture of the
strong circumferential fibers of the meniscus. However, it is very useful to often use
an oblique or horizontal pattern and to place these sutures both on the femoral and
tibial side of the meniscus (Fig. 8-20). Sutures should be placed approximately 3 to 5
mm apart. The number of sutures will depend on the character and stability of the
repair.
Biologic Augmentation
There are different methods to potentially improve the biologic healing capacity of a
meniscus repair (Table 8-1). In the setting of marginal vascularity, I will create
vascular access channels using an 18-gauge spinal needle. The needle (bend to help
maneuver) is used from inside-out through the meniscus after the repair has been
performed. Perforate into the meniscocapsular junction to promote bleeding and
vascular inflow. Be careful not to damage sutures and do not go through the capsule
behind the posterior horn of the lateral meniscus (popliteal vessels). If the repair is
not combined with an ACL reconstruction, I will create a postoperative hemarthrosis
by performing a limited notchplasty of the lateral femoral condyle. A 5.5-mm full
radius shaver is used to create a bleeding cancellous bone bed being careful not to
injure the ACL (Fig. 8-21). I have evolved to using this technique instead of placing a
fibrin clot.
FIGURE 8-17
The second needle is delivered down the single-lumen cannula adjacent to the
suture, which is placed on slight tension.
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FIGURE 8-18
A: The single-lumen suture cannula is used to place a vertical stitch. B: The loop
of Ethibond suture is pulled through the cannula.
FIGURE 8-19
A: Sutures are delivered and tagged through the posteromedial incision. B: With
the knee in full extension, sutures are tied over the posteromedial joint capsule.
FIGURE 8-20
Sutures are placed on both the femoral and tibial sides of the meniscus.
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TABLE 8-1. Biologic Augmentation Techniques for
Meniscus Repair
â€¢ Vascular access channels
â€¢ Fibrin clot*
â€¢ Limited notchplasty (creation of hemarthrosis)*
â€¢ Platelet-rich plasma*
* Option if not combined with ACL reconstruction.
The subcutaneous tissue and skin are closed in separate layers. Drains are not used in
the joint or posterior incisions. The joint and incisions are injected with 20 to 30 ml of
0.5% plain Ropivacaine combined with 5 mg of Duramorph. Alternatively, 0.5% plain
Marcaine may be substituted for Ropivacaine. After the dressing is applied, a cold
therapy device and postoperative brace locked in full extension are placed in the
operating room.
Technique for Lateral Meniscus Repair

The general principles for a lateral meniscal repair are the same as for a medial
repair. The anteromedial portal often needs to be slightly superior to avoid
obstruction from the tibial eminence when placing suture cannulas to the posterior
horn of the lateral meniscus. Use a spinal needle to plan this portal with the leg in the
figure-of-4 position. If necessary, you can always make a second anteromedial portal
more superior. Once the meniscal edges are prepared, the posterolateral approach is
made. The knee is placed into a figure-of-4 position, which places the knee in
approximately 70 to 90 degrees of flexion. This will take tension off the peroneal
nerve, biceps tendon, and lateral head of the gastrocnemius and place a varus stress
on the joint. Alternatively, it is often helpful to place the leg over the side of the
table, flexing the knee to 90 degrees. In this position, the surgeon can perform the
posterolateral dissection in a less awkward position. Once the dissection is carried out
and the retractor is placed, the leg can be repositioned into the figure-of-4 with the
foot on the table.
A vertical incision is placed just posterior to the lateral collateral ligament (LCL).
Translumination through the joint may aid in identifying the proper plane and level.
With the knee in a figure-of-4 position, the LCL can typically be palpated from the
fibular styloid to the lateral epicondyle. The incision is 3 to 4 cm with two thirds of
the incision below the joint line. The interval between the iliotibial band and biceps
tendon is incised. The biceps tendon is a valuable landmark since the peroneal nerve,
which is at risk during this procedure, is located posteromedial to the biceps tendon.
By staying anterior to the biceps tendon and lateral head of the gastrocnemius, the
nerve should be protected. Carrying the dissection deeper, identify the interval
between the lateral head of the gastrocnemius and the posterolateral joint capsule.
Once the interval is started with Metzenbaum scissors, the muscle can be bluntly
dissected off the capsule using either a blunt key elevator or your finger. The interval
development is facilitated by starting distal to the joint line. Similar to the medial
side, you may use your index finger to confirm you are anterior to the lateral head of
the gastrocnemius by plantar flexing and dorsiflexing the foot. Once in the proper
interval directly against the posterolateral joint capsule, the popliteal retractor should
be placed to protect the neurovascular structures (Fig. 8-22).The suture cannulas are
placed through the anteromedial portal. Debridement of the fat pad will facilitate
passage of the cannula. Suture cannulas are not used from the anterolateral portal due
to the potential risk to the popliteal vessels, which are just posterior to the posterior
horn of the lateral meniscus. The assistant receiving needles sits on a stool. It is often
helpful to elevate the table and remove the lateral post to improve the assistant's
comfort and visualization. Sutures are placed in a similar fashion to the medial side.
Do not to advance the needle beyond 1 to 1.5 cm before visualizing the needle coming
through the posteromedial capsule or reflecting into the surgical field off the posterior
retractor. Once retrieved, sutures can either be clamped or tied individually. Capture
of the posterolateral capsule is typically not an issue as on the medial side and
therefore the sutures are tied with the knee in 70 to 90 degrees of flexion. Although
some authors have advocated tying the sutures after the ACL reconstruction is
performed, I have found it helpful to tie them at the time of the meniscus surgery.
This tends to save time by avoiding the need to re-expose and replace retractors later
in the case. One difference between the lateral side and the medial side is dealing
with the popliteus tendon. I place sutures adjacent to the popliteus tendon, but not
through the tendon. However, other authors have not reported problems with placing
sutures through the tendon.
FIGURE 8-21
Postoperative hemarthrosis is created by performing a limited notchplasty.
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FIGURE 8-22
A: The proper interval dissection for the posterolateral approach. Note the
location of the peroneal nerve, which is at risk during this approach. B: Retractor
placement in relation to the neurovascular structures.
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FIGURE 8-22
(Continued)
Closure is similar to the medial side except that the interval between the iliotibial
band and biceps is closed using vicryl sutures.
Pearls and Pitfalls
Debride the edges of a bucket handle tear with the tear displaced anterior
(allows direct visualization).
P.86
Perform the posterolateral approach with the knee flexed over the side of the
table. Once the retractor is placed, place the leg into the figure-of-4 position.
Dissecting gastrocnemius off the posterior joint capsule is facilitated by

starting distal.
Always place suture cannulas through the anteromedial portal for a lateral
repair.
Pass the needles through the medial meniscus in greater extension (10 to 20
degrees) and once through the meniscus, flex the knee to 60 degrees to
retrieve the needles posterior.
If you have difficulty visualizing needles coming through the posterior capsule,
place a more anterior suture (easier to locate) and use this suture as a guide
for posterior sutures.
A limited notchplasty is an easy way to create a postoperative hemarthrosis

(similar to ACL reconstruction)
A postoperative long leg hinged knee brace locked in full extension is worn at night
and when ambulating for the first 6 weeks. The brace should be removed by the
patient, as well as the therapist, for range of motion exercises up to 90 degrees for
the first 6 weeks. The brace is discontinued at 6 weeks.
The patient is allowed immediate weight bearing as tolerated with the knee locked in
full extension. Weight bearing with flexion is not allowed during the first 6 weeks.
Radial repairs are kept non-weight bearing for the first 6 weeks followed by
progressive weight bearing. Range of motion exercises between 0 and 90 degrees are
allowed in the first 6 weeks. For tenuous repairs, motion is limited from 0 to 60
degrees for the first 4 weeks (isolated repair only). Between 6 and 12 weeks, range of
motion is progressively increased. Closed kinetic chain quadriceps exercises and
hamstring exercises are started at 4 to 6 weeks, but no resistance exercises beyond 80
degrees of flexion are allowed for 6 months. Squatting is not allowed for 6 months
postoperatively. Straight line running is allowed at 4 to 4-1/2 months. Return to full
sports for an isolated repair is 6 months as long as strength is adequate. Repairs
performed in conjunction with an ACL reconstruction follow the ACL protocol with the
addition of no squatting for 6 months.
COMPLICATIONS
Injury to the saphenous nerve can be avoided by careful dissection and retraction of
the nerve with the sartorius muscle.
Injury to the peroneal nerve can occur when inadvertently placing needles without
adequate retraction. This should be avoided by taking tension off the nerve with knee
flexion and by adequately protecting the nerve behind the biceps and lateral head of
the gastrocnemius with the popliteal retractor.
To avoid popliteal vessel injury, needles should always be directed away from the
midline toward the posterior exposure. Be extremely careful when placing needles
into the posterior horn of the lateral meniscus. Always bring suture cannulas from the
anteromedial portal across for a lateral repair.
To prevent limitation of motion, tie the posteromedial sutures with the knee in
extension, avoiding imbrication of the posterior capsule, which may subsequently
cause a flexion contracture. Immediate range of motion should be instituted when
combined with an ACL reconstruction, since this group is at higher risk for this
complication.
Infection rates are relatively low (0% to 2%). The hospital staff should wash down the
suture cannulas to properly sterilize.
RESULTS
Studies have reported successful meniscus repair in more than 90% of patients who
have inside-out repairs performed in conjunction with ACL reconstruction. In contrast,
a 40% to 50% failure rate is expected when performed in the setting of persistent
functional ACL instability. Isolated repairs (intact ACL) have an approximately 70% to
85% clinical success rate. In general, lateral meniscal repairs have improved healing
rates when compared with medial repairs. Nonabsorbable sutures have been reported
to have lower clinical failure rates compared with absorbable sutures. Recent reports
show that even in patients over the age 40, over 85% of repairs were asymptomatic at
the latest follow-up (most combined with ACL reconstruction).
P.87
RECOMMENDED READING
Baratz ME, Fu FH, Mengato R. Meniscal tears: the effect of meniscectomy and of repair
on intra-articular contact areas and stress in the human knee. Am J Sports Med.
1986;14:270-275.
Cannon WD, Vittori JM. The incidence of healing in arthroscopic meniscal repairs in
anterior cruciate ligament reconstructed knees versus stable knees. Am J Sports Med.
1992;20:176-181.
Lindenfeld T. Inside-out meniscus repair. Instr Course Lect. 2005;54:331-336.
Morgan CD, Wojtys EM, Casscells CD, et al. Arthroscopic meniscal repair evaluated by
second-look arthroscopy. Am J Sports Med. 1991;17:632-638.
O'Shea JJ, Shelbourne KD. Repair of locked bucket handle meniscus tears in knees with
chronic anterior cruciate ligament deficiency. Am J Sports Med. 2003;31:216-220.
Rosenberg TD, Scott SM, Coward DP, et al. Arthroscopic meniscal repair evaluated with
repeat arthroscopy. Arthroscopy. 1984;2:14-20.
Rubman MH, Noyes FR, Barber-Westin SD. Arthroscopic repair of meniscal tears that
extend into the avascular zone. A review of 198 single and complex tears. Am J Sports
Med. 1998;26:1987-1995.
Tenuta JJ, Arciero RA. Arthroscopic evaluation of meniscal repairs. Factors that affect
healing. Am J Sports Med. 1994;22:797-802.
Watson FJ, Arciero RA. Inside-out meniscus repair. Oper Tech Sports Med.
2003;11(2):104-126.
Wolf BR, Rodeo SA. Arthroscopic meniscus repair with suture: inside-out with fibrin
clot. Sports Med Arthrosc. 2004;12:15-24.
> Table of Contents > Part Two - Meniscus Surgery > 9 - Meniscal Repair with Meniscal Fixators
9
Meniscal Repair with Meniscal Fixators
Peter R. Kurzweil
The fixation of meniscal tears has undergone a revolution since the introduction of
fixators into arthroscopic surgery a decade ago. The simplicity and ease of insertion of
these devices led to their popularity for the repair of torn menisci. The only
alternative was the traditional suture technique, which required additional incisions,
prolonged operative time, and the risk of neurovascular injury.
The first fixators on the market were somewhat rigid devices made of a variety of
bioabsorbable materials, with varying resorption times. They are supplied in varying
lengths, with the appropriate size chosen depending on the location of the tear. They
were designed with barbs, heads, or screw threads to engage meniscal tissue, and
therefore are not well suited for the most peripheral tears or capsular detachments.
Compression at the tear site is often difficult to achieve and nearly impossible to
adjust after insertion. Among the most popular first-generation fixators are the Arrow,
the Hornet, the Clearfix Screw, and the Dart.
With increasing reports of unsatisfactory long-term results and the inability to repair
peripheral tears to the capsule, a second generation of meniscal fixators was
developed to more closely mimic suture repairs. The two suture-based implants
currently available are the Fast-Fix (Smith & Nephew, Endoscopy Division, Andover,
MA) and the RAPIDLOC (Mitek Surgical Products, Westwood, MA) (Fig. 9-1 A,B). Both of
these implants consist of two small polymer T-bar anchors connected by a pre-tied,
locking, sliding knot, which has eliminated the need for arthroscopic knot tying. Both
Fast-Fix anchors and one of the RAPIDLOC anchors are designed to be deployed across
the capsule, then rotate to engage the capsule when the connecting suture is pulled,
analogous to EndoButton (Smith & Nephew, Andover, MA) fixation. Since the suture
length between the two anchors adjusts during implantation to allow compression of
the tear, these fixators come in only one size. The only variability is the angle of the
needle delivery system. But, like other fixators, the anchors deployed across the
capsule cannot be removed arthroscopically once inserted.
The Fast-Fix Delivery Needle is supplied with three angles: 0, +22, and -22 degrees
(Fig. 9-1D). The -22-degree needle allows suture placement on the undersurface of the
meniscus. The device requires that each anchor be deployed across the capsule, and
at least one also pass through the tear. This allows for a vertical or horizontal suture
orientation. The Rapid-Loc comes with needle tips of 0, 12, and 27 degrees (Fig. 9-1C).
It is designed to be placed on the superior surface of the meniscus. Its insertion
requires only one pass of the â€œbackstopâ€• anchor across the tear and through the
capsule. Both fixators come with anchors and sutures that are permanent or
bioabsorbable. The choice depends on surgeon preference. I prefer permanent
material, as is my choice when doing the traditional inside-out or outside-in suture
repair techniques.
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FIGURE 9-1
A: The Rapid-Loc is made of two anchors called the â€œtop-hatâ€• and

â€œbackstop,â€• which are connected by a pre-tied, sliding suture of No. 2
nonabsorbable Ethibond (Ethicon, Somerville, NJ). Note that it is also available
with absorbable components using a PDS â€œtop-hatâ€• and Panacryl suture. B:
The Fast-Fix is made of two 5-mm anchors also connected by a pre-tied, sliding
No. 0 suture of nonabsorbable, braided polyester. C: The angles for the insertion
needles for the Rapid-Loc are 0, 12, and 27 degrees. D: The Fast-Fix is supplied
with three insertion angles of 0, +22, and -22 degrees.
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The indication for meniscal repair should remain the same regardless of technique.
Fixators are not ideal to use for every tear location and orientation. My preference is
to use outside-in suture repairs for anterior third tears, and once started, I will often
stretch this to the anterior two thirds of the meniscus. Fixators are most ideal for the
posterior third, where the traditional inside-out technique would have required a
posterior incision to protect neurovascular structures. For tears entirely in the
posterior half of the meniscus, I will generally use suture-based fixators only.
However, there is no reason to commit to just one method of fixation. I frequently use
hybrid fixation, with outside-in sutures anteriorly and fixators posteriorly. Cost is an
issue, and these second-generation fixators are far more expensive than using sutures.
In my experience, unstable bucket-handle tears, which are found displaced in the

notch, have sometimes failed when fixators alone were used. Although these failures
were seen with first-generation fixators, this led me to favor the traditional suture
repair techniques for this scenario. Recently, I have been more confident with hybrid
repairs using suture-based fixators and outside-in sutures for these displaced bucket-
handle tears.
Tear orientation is also important. Ideally, fixators (and sutures) should be inserted
into the meniscus so that they are perpendicular to the tear, making vertically
oriented tears ideal. Horizontal cleavage tears and radial tears are less suited for
fixators, and sutures are preferred if a repair is indicated. Some surgeons are
attempting to use the Fast-Fix in these situations.
SURGERY
Patient Positioning
We prefer to have the patient supine on the operating room table, allowing the knees
to rest in extension. A tourniquet is applied to the proximal thigh but is not routinely
inflated. A lateral thigh post clamped to the table allows application of a valgus stress
to the knee, opening the medial compartment for visualization. For viewing the lateral
compartment, the leg is placed in the figure-4 position. We have also found that
general anesthesia affords a little extra room in the compartments, since valgus or
varus stressing to open the knee is sometimes too painful with only local anesthesia.
Meniscal Preparation
Although fixators are simple to use, they should not be inserted immediately on
visualization of the tear. The tear site needs to be prepared just as it would be for any
repair technique. The edges on both sides of the tear are debrided of fibrous tissue
with a shaver or rasp. The capsular attachment at the meniscal-synovial border is
abraded with a rasp or motorized shaver to stimulate a healing response (bleeding).
The meniscus rim may also be trephinated to produce vascular access channels. This
can be quickly done using an 18- or 20-gauge needle from outside in or a meniscal
repair needle through a cannula introduced through a portal.
The best results are obtained when the meniscus tear is optimally reduced. Surgeons
strive for an anatomic reduction with fractures, and should do the same with torn
menisci. With very unstable tears, we have found it convenient to hold the reduction
with temporary fixation, much like using a K-wire to for provisional fracture fixation.
Use a probe under arthroscopic visualization to help reduce the tear and give counter-
pressure as a small needle is inserted from outside-in across the tear (Fig. 9-2). At this
point the fixators can be inserted.
Fixator Insertion
The needle angle and portal of entry are selected to provide the most perpendicular
approach to the tear. This is usually best accomplished through the contralateral
portal. Once again fixation of a fracture is a well-suited anaology, as screws are
inserted perpendicular to the orientation of the break. Sometimes with posterior tears
near the root, the best approach is through the ipsilateral portal. In these situations,
particular attention needs to be given to the neurovascular bundles.
The fixator insertion needle first engages the central portion of the tear, which is
manipulated to achieve the best anatomic reduction before passing the needle across
the tear, through the peripheral portion of the menicus and the capsule. If the
reduction of the tear is difficult to achieve with the fixator insertion device, the
outside-in needle â€œtrickâ€• described previously can be used.
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FIGURE 9-2
With unstable large meniscus tears, it is sometimes convenient to hold the

reduction with temporary fixation, much like using a K-wire for provisional
fracture fixation. A probe is used to manipulate the tear and give counterpressure
as a small needle is inserted from outside-in across the tear.
The number of fixators used depends on the length and stability of the tear. They are
generally spaced 5 to 8 mm apart. Once the repair has been completed, the knee is
ranged (with the arthroscope removed) and then the tear is probed to assess stability
and gapping. If the repair site gaps, supplemental fixation should be considered. In
cases requiring concomitant anterior cruciate ligament (ACL) reconstruction, the
meniscal pathology is addressed first.
Pearls and Pitfalls

Although the Rapid-Loc is supplied with three different angles, we almost always use
the 12-degree angle. It allows the best angle of approach to the tears, whether
directing the insertion needle vertically or horizontally. Also, for stocking purposes, it
simplifies having to have more than one. Before inserting the fixator into the knee,
the top-hat anchor is slid halfway down the loop, closer to the tip. The metal skid
supplied by Mitek is introduced into the portal and carefully directed toward the tear
with caution to avoid scuffing the articular cartilage. The fixator is inserted through
the portal such that the curve of the needle and the top-hat anchor are angled toward
the metal skid to avoid dragging the fat pad into the knee. The trailing suture is held
under some tension, keeping the top-hat anchor flat against the â€œmeniscal
applierâ€• deployment device during this manuever. The meniscus should be
penetrated at least 3 mm central to the tear (Fig. 9-3A,B). After inserting the needle
across the tear, with the depth of penetration limited by a built-in stop, the trigger is
pressed to deploy the backstop anchor (Fig. 9-3C). The trigger is held depressed as the
insertion needle is withdrawn for the knee joint, leaving the suture trailing out of the
portal. This suture is loaded through the eyelet of the tensioning-cutting instrument,
which is used to bring the top-hat anchor down onto the superior surface of the
meniscus, just under the pre-tied, sliding knot. Compression is adjusted with a push-
pull manuever. Slight dimpling of the meniscus is preferred before cutting the suture
(Fig. 9-3D).
The Fast-Fix anchor, like the Rapid-Loc, is generally inserted in the portal
contralateral to the tear. Occasionally, posterior horn tears are best approached
through the ispilateral portal. A curved meniscal depth probe is inserted to simulate
the angle of repair before selecting fixator needle angle and insertion portal. The
selected portal is spread with a snap to avoid fat-pad entrapment. This device comes
with a blue plastic cannula to help ensure a smooth entry into the joint, although the
metal skid from the Rapid-Loc works well here, too. Once again, the straight (0-
degree) device is rarely used and is generally not carried in our surgery center to
facilitate stocking. Most of the time the 122-degree fixator is used. The -22-degree
fixator works well to keep the meniscus from flouncing or puckering in the superior
direction by placing an occasional horizontal mattress suture on the undersurface of
the meniscus. When creating vertical sutures, the first pass is superior, often over
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the meniscus directly into the capsule. Placing the second anchor through the tear,
more inferiorly, facilitates cutting and tensioning the knot, which tends to slide
toward the second anchor. With horizontal suture placment, the first anchor is usually
placed more anteriorly.
FIGURE 9-3
Rapid-Loc insertion. A: The delivery needle is placed 3 mm central to the tear. B:

The insertion needle penetrates across the tear and through the capsule. C: The
trigger is depressed to deploy the backstop anchor behind the capsule. D: The
top-hat anchor is brought down onto the meniscus and tensioned with the pusher-
cutter instrument.
After the first pass, the needle is slowly retracted, leaving the first anchor behind the
capsule (Fig. 9-4A,B). The pre-tied knot is usually a good reference for depth of
insertion. While still in the joint and under arthroscopic visualization, the second
anchor is â€œclickedâ€• into position by sliding the gold trigger forward on the handle
of insertion device (Fig. 9-4C). After its deployment the insertion device is removed,
leaving only a single suture trailing out the portal (Fig. 9-5A). This suture is loaded on
the â€œsuture cutter and knot pusher,â€• and the suture is pulled, advancing the
sliding knot and reducing the tear (Fig. 9-5B). The knot is cinched down directly onto
the meniscus and tension of the construct adjusted with a push-pull action (Fig. 9-5C).
The suture/knot is appropriately tensioned before it is cut (Fig. 9-5D). The white
â€œdepth penetration limiterâ€• is helpful in the learning phase of using this device
to avoid plunging in the back of the knee. With experience, the surgeon gains
confidence with the feel of the needle penetrating the capsule. I no longer use this
protection sheath except when working near the root of the meniscus, or using the
device through the portal ispilateral to the tear.
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FIGURE 9-4
Fast-Fix insertion. A: The needle penetrates the meniscus 3 mm central and

passes through the capsule. B: The insertion device is slowly withdrawn, leaving
the first anchor behind the capsule. C: The gold trigger is pushed forward to
advance the second anchor into the ready position. D: The second anchor is
inserted approximately 5 mm away and deployed across the capsule.
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FIGURE 9-5
A: The insertion device is withdrawn from the joint, leaving a suture trailing out
of the portal. B: The suture is loaded on the â€œsuture cutter and knot
pusherâ€• device, although the knot is initially advanced and the meniscus
reduced by just pulling on the suture. C: The instrument helps set the tension on
the knot with a push-pull action. D: The suture is cut.
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We do not follow an â€œaggressiveâ€• postoperative protocol. For the first 2 weeks
postsurgery, patients are instructed to use a knee immobilizer and crutches for toe-
touch weight bearing. Progressive weight bearing in the knee immobilizer is allowed
during weeks 3 and 4. Patients are encouraged to range the knee only when seated or
supine beginning the week 3. Unrestricted walking occurs at 1 month, and use of the
stationary bike is recommended. Squatting is avoided for 4 months, and return to
cutting and pivoting sports is delayed 6 months. When concomitant ACL reconstruction
is performed, the postoperative regimen of the meniscus repair takes precedence.
COMPLICATIONS
There are numerous reports of complications with the first-generation fixators,
particularly the Arrow, as noted previously. (The Arrow has since been redesigned to
have a lower profile.) Complications with the suture-based fixators are much less
frequent, and often appear to be related to deployment or insertion problems. There
is only one report of the top-hat anchor of the Rapid-Loc scoring the femoral articular
cartilage.
RESULTS
Although short-term studies with first-generation fixators were initially favorable, the
results decreased over time and were associated with various complications. Reports
described scoring the overlying articular cartilage of the femoral condyle, implant
breakage, and protrusion of the tip of the device at the joint line. My experience with
the Arrow became quite unsatisfactory with longer follow-up. In 60 repairs followed an
average of 4.5 years, we experienced failure in 28% of our cases. The failure rate rose
to 42% with isolated repairs, not accompanied by an ACL reconstruction. Similar results
have have been reported by others, which has led us to abandone the use of first-
generation fixators.
The results of suture-based fixators appear to be more promising. One recent report
showed a success rate of 91% in 54 repairs after 35 months with the Rapid-Loc. Several
published studies on the Fast-Fix have also shown promising results. The success rate
in these reports ranged from 86% to 91% with follow-up periods varying from 18 to 32
months.
RECOMMENDED READING
Barber FA, Herbert MA, Richards DP. Load to failure testing of new meniscal repair
devices. Arthroscopy. 2004; 20(1):45-50.
Barber FA, Johnson DH, Halbrecht JL. Arthroscopic meniscal repair using the
BioStinger. Arthroscopy. 2005; 21(6):744-750.
Buchalter DP, Karzel R, Friedman M, et al. Evaluation of all-inside arthroscopic

meniscal repair using the FasT-Fix System (SS-35). Arthroscopy. 2006;22;6(suppl):e18-
e19.
Haas AL, Schepsis AA, Hornstein J, et al. Meniscal repair using the FasT-Fix all-inside
meniscal repair device. Arthroscopy. 2005;21(2):167-175.
Kotsovolos ES, Hantes ME, Mastrokalos DS, et al. Results of all-inside meniscal repair
with the FasT-Fix Meniscal Repair System. Arthroscopy. 2006;22(1):3-9.
Kurzweil PR, Lemos MJ. Meniscal repair with first generation fixators. Sports Med
Arthrosc Rev. 2004;12:37-43.
Kurzweil PR, Tifford CD, Ignacio EM. Unsatisfactory clinical results of meniscal repair
using the meniscus arrow. Arthroscopy. 2005;21(8):905.e1-905.e7.
Quinby JS, Golish SR, Hart J, et al. All-inside meniscal repair using a new flexible,
tensionable device (SS-37). Arthroscopy. 2006;22;6(suppl):e19-e20.
Sgaglione NA. Meniscus repair update: current concepts and new techniques.
Orthopedics. 2005;28(3):280-286.
Sgaglione NA. New generation meniscus fixator devices. Sports Med Arthrosc Rev.
2004;12:44-59.
Sgaglione NA, Steadman JR, Shaffer B, et al. Current concepts in meniscus surgery:
resection to replacement. Arthroscopy. 2003;19;Dec(suppl):161-188.
> Table of Contents > Part Two - Meniscus Surgery > 10 - Meniscal Transplantation without
Bone Plugs
10
Meniscal Transplantation without Bone Plugs
Fotios P. Tjoumakaris
Anthony M. Buoncristiani
Christopher D. Harner
The meniscus plays a vital role in the successful performance and well-being of the
knee. Loss of the meniscus, in part or in total, significantly alters joint functions and
predisposes the articular cartilage to degenerative change. Meniscal transplantation
has been developed in an attempt to alter the natural history of the
postmeniscectomy knee. The procedure is performed in hopes of forestalling
progressive joint deterioration by theoretically restoring part of the meniscal function.
Surgical techniques for meniscal transplantation have evolved over the last decade
from mainly open arthrotomies to arthroscopically assisted techniques, with or without
the use of bone plugs (or blocks) at the meniscal horn attachments. Proper patient
selection as well as realistic patient goals and expectations are vital to avoid
unnecessary failures. Results from long-term outcome studies should help to further
refine meniscal allograft techniques as well as its indications and improve our
understanding of where this procedure belongs in the treatment algorithm of the
postmeniscectomy knee.
The ideal candidate for a meniscal allograft transplant has joint line pain secondary to
meniscal deficiency and fits the following criteria: (a) documented evidence of a
previous subtotal or total meniscectomy or its biomechanical equivalent; (b)
ligamentously stable or concurrently performed ligament reconstruction; (c) early
grade I or II articular cartilage changes as determined by the use of a 45 degrees
posteroanterior (PA) flexion weight-bearing radiograph; (d) no evidence of
malalignment as determined on standing long cassette views; and (e) no evidence of
articular incongruity on diagnostic arthroscopy. All patients are informed during
preoperative counseling that the ultimate decision to proceed with the transplant is
made in the operating room. Contraindications to transplantation include grade III or
IV changes in the involved compartment, ligamentous instability, malalignment that
would place significant stress on the allograft, and a patient with unrealistic
expectations regarding outcome.
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Candidates being considered for meniscal transplant surgery require a comprehensive
examination of both lower extremities. Height and weight should be recorded as an
indication of body habitus as well as the standing alignment of the lower extremities.
The patient's ability to squat and any limitations in movement and associated
discomfort are noted. It is important to observe the gait of the patient for any
abnormalities. Both knees are examined for the presence of an effusion.
The patella must be examined and a thorough ligamentous examination performed

with special attention placed on the anterior cruciate ligament (ACL) status. This is
done through the Lachman test, pivot shift, and anterior drawer in neutral, internal,
and external rotation. The posterior cruciate ligament (PCL) is evaluated with the
posterior drawer and posterior sag with the hip and knee flexed to 90 degrees.
Posterolateral rotatory instability is ruled out through the dial test, reverse pivot shift,
and the posterolateral drawer. The collaterals are tested with varus and valgus stress
at 0 and 30 degrees of knee flexion. It is important to elicit the presence or absence of
joint line tenderness over the involved and the uninvolved compartments as well as
the presence of hyperflexion signs.
Imaging
We routinely obtain four views of the knee in meniscal transplant candidates. This
includes a PA 45degree flexion weight-bearing view, Merchant, lateral, and long
cassette from the hips to the ankles to evaluate the mechanical axis. From these
studies the alignment and joint space of the involved knee is calculated and compared
with the opposite asymptomatic knee. Patients who have a mechanical axis deviation
toward the involved compartment require a staged or combined osteotomy in addition
to transplantation. We routinely obtain a magnetic resonance image (MRI) of the knee
to assess the quality and amount of remaining meniscal tissue. In patients whose pain
is nonspecific, a three-phase 99Tc bone scan is obtained. Increased uptake in the
compartment indicates early degenerative changes.
Before the definitive procedure, the amount of remaining meniscus, the quality of the
articular surface, and any associated ligamentous injuries must be verified so they may
be addressed at the time of surgery. If necessary, this can involve an examination
under anesthesia and a diagnostic arthroscopy staged before transplantation. The plain
radiographs are also used to determine the appropriate size for the allograft tissue as
required for the tissue bank.
SURGERY
Positioning, Draping, and Operating Room
Organization
The patient is positioned supine on a regular operating room table with a sandbag
taped to the table at the foot of the bed to hold the leg at 90 degrees of knee flexion.
A standard side post is used at the level of the greater trochanter to provide a fulcrum
for valgus stress and to provide lateral support with the knee at 90 degrees of flexion.
A tourniquet is not routinely used for the procedure. The extremity is prepped from
the tourniquet distally with alcohol and betadine and draped with a nonimpervious
stockinette, extremity drape, and a half sheet to cover the post. A bolster is placed
between the lateral thigh and the post (Fig. 10-1).
The operating room should be organized to provide a controlled environment where

traffic and the potential for contamination are kept to a minimum. It is necessary to
have a separate table for graft preparation that has an oscillating saw, drill, and
instruments.
Specific Approaches
All incision sites as well as the joint are marked before prepping and draping, prepped
out with a betadine stick, and injected with 1% lidocaine with epinephrine. A standard
arthroscopy is performed using an anterolateral-viewing portal just adjacent to the
patellar tendon and above the joint line and an anteromedial working portal 1 cm
medial to the medial edge of the patellar tendon. Two main approaches are used in
both the medial and lateral transplants: the anteromedial or lateral and the
posteromedial or lateral approaches. The anterior approach involves an extension of
the arthroscopy skin incision superiorly along the patellar tendon. A medial or lateral
parapatellar arthrotomy is
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made. Often it is necessary to remove a small amount of the fat pad to visualize the
joint. The anterior horn of the remaining meniscus should be well visualized. Three
No. 2 braided nonabsorbable sutures are passed around the remaining anterior horn,
leaving the needles attached for later fixation to the allograft.
FIGURE 10-1
The extremity is prepped, draped, and positioned so that it can rest at 90 degrees
of flexion.
The posteromedial approach involves a skin incision 3 cm long just anterior to the
posterior margin of the medial femoral condyle. One third of the incision should be
above the joint line. Careful dissection is used to divide the subcutaneous fat, and
care is taken to look for and protect the infrapatellar branch of the saphenous nerve,
which typically crosses just above the joint line. The sartorial fascia (layer 1) is then
divided in line with the skin incision and a posterior capsulotomy is made between the
posterior border of the medial collateral ligament and the posterior oblique ligament
(layer 2). Three No. 2 nonabsorbable sutures are passed around the posterior oblique
ligament for later closure. The posterolateral approach involves a 3-cm skin incision
just posterior to the lateral collateral ligament, two thirds below and one third above
the joint line. The interval between the iliotibial band and the biceps femoris is
developed using a combination of blunt and sharp dissection. The plane between the
lateral head of the gastrocnemius and the posterolateral capsule is developed and a
popliteal retractor is placed.
Details of Procedure
A standard arthroscopy is initially performed to evaluate the condition of the articular
surface as well as the amount of remaining meniscal tissue. The graft can be thawed
after this information is obtained and it is determined that the patient is a suitable
candidate. The remaining edge of the meniscus is trimmed using a square-tipped biter
to provide an edge to which the transplant is sutured. This is easiest to perform
arthroscopically in a lateral meniscal transplant and open in the medial transplant.
Care is taken to preserve a portion of the remaining meniscus for fixation to the
allograft. The graft is then prepared on a separate table. We prefer to begin preparing
the graft before performing the arthrotomies and trough preparation because there
have been cases in which the incorrect graft has been sent. The graft preparation,
trough formation, and arthrotomies can then be performed simultaneously.
Lateral Meniscal Transplant
For lateral meniscal transplants, we use a bone block technique where a block of bone
is left joining the anterior and posterior insertions of the meniscus in anatomic
position. A soft tissue technique without allograft bone can be used as well; however,
we prefer to keep the meniscal attachments to the allgoraft for the lateral meniscus
due to their proximity, and it does not significantly add to the complexity of the
procedure. The bone block is trimmed of excess bone and contoured to fit into the
appropriate slot of the sizing block (Fig. 10-2). Two drill holes are placed in the center
of the bone block and a No. 2 braided nonabsorbable suture is passed. A second No. 2
suture is placed at the posterolateral corner of the graft to help guide the posterior
horn past the lateral femoral condyle (Fig. 10-3).
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FIGURE 10-2
A: Preparation of the lateral meniscus with a small bone block that retains the
meniscal root attachments. B: Finalized lateral meniscus allograft ready for
suture placement and ultimate passage.
The arthroscopy and meniscal debridement are performed as described earlier. With
the arthroscope in the lateral portal, a trough is prepared through the medial portal. A
0.25-inch curved osteotome is used to begin cutting the trough. The trough is started
by using the osteotome with the curve toward the notch and immediately adjacent to
the ACL (Fig. 10-4). The medial wall, lateral wall, and floor of the trough are
developed to the width of the osteotome, which is approximately 7 mm. Care is taken
to avoid penetration of the posterior cortex because of risk of injury to the
neurovascular structures and to provide a backstop for the allograft. The trough is
then progressively deepened with the quarter round and large cutting gouges to
correspond to the contour, depth, and thickness of the bone plug (Fig. 10-5). The
round rasp included in the set can then be used to smooth the edges of the trough.
Metal templates are manufactured for each width and are placed into the trough to
verify the size. The anterolateral and posterolateral open approaches are then
performed as described earlier.
FIGURE 10-3
A prepared lateral meniscus transplant with suture placed in the posterolateral
horn and through drill holes in the bone block.
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FIGURE 10-4
A: A quarter-inch curved osteotome is used through the medial portal to begin

the walls and floor of the trough. B: Arthroscopic view from the lateral portal.
FIGURE 10-5
A,B: A large gouge deepening the trough to match the bone block size.
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Transosseous holes in the tibia that are used to secure the bone block are drilled using
an ACL guide. An ACL tip aimed guide is used to target the center of the trough 1 cm
apart and to exit on the anteromedial tibia (Fig. 10-6). An incision is made in Langer's
lines corresponding to the location of exit of the holes, and 3/32-inch K-wires are
drilled. Care is taken to leave at least a 1-cm bone bridge between the exiting sutures
so there is an adequate distance between them when they are tied. The K-wires are
exchanged for Hewson suture passers, which are pulled into the joint and out of the
medial portal.
A tonsil clamp is passed through the medial portal, punched through the posterolateral
capsule, and pulled out through the posterolateral incision. This is exchanged for a
suture passer, and the posterolateral suture in the graft is pulled through. The sutures
in the bone block are pulled through the tibia (Fig. 10-7). Care must be taken to avoid
entangling the sutures. The graft is passed into the joint through the anterolateral
incision by first gently guiding the bone block into place, then pulling the meniscus
into the compartment by the posterolateral suture (Fig. 10-8).
The sutures that were previously passed through the anterior horn of the native lateral
meniscus are then passed in a simple fashion around the anterior horn of the
transplanted meniscus and tied. Zone-specific cannulas are then used to pass suture in
an inside-out technique to repair the posterior horn and body of the transplanted
meniscus to the native meniscus and capsule (Fig. 10-9).
Medial Meniscus
For the medial meniscus, we use a soft-tissue technique without bone plugs. The graft
is removed from the allograft tibial plateau and secured at each attachment with No.
2 bradied nonabsorbable suture in a whip stitch configuration (Fig. 10-10). Three No. 2
sutures, spaced 1 cm apart, are passed in a simple fashion through the posterior horn
of the graft. The top of the graft is marked TOP so that there is no confusion when the
graft is placed into the joint.
FIGURE 10-6
An ACL tip guide is used to drill two 3/32 K-wires from the anteromedial tibia to
the center of the trough 1 cm apart.
FIGURE 10-7
The K-wires are exchanged for suture passers, which are used to pull the bone
block sutures through the tibia. A tonsil clamp is used to pass a suture passer,
which is used to pull the posterolateral suture into the joint and out the
posterolateral capsule.
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FIGURE 10-8
The passage of the lateral meniscus allograft through the anterior incision.
FIGURE 10-9
The reduced bone block and meniscus after inside-out repair of the posterior horn
to the capsule.
FIGURE 10-10
Preparation of the medial meniscus graft is performed without the use of bone
plugs. The top portion of the graft is marked prior to removal from the plateau,
and care is taken to preserve the meniscal roots in their entirety.
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The arthroscopy, meniscal debridement, and open approaches are performed as
described earlier. Three nonabsorbable sutures are passed around the anterior capsule
and meniscus and clamped for later passage around the allograft. The ACL tip guide
set at 47.5 degrees is used to pass two 3/32 K-wires from the anterolateral tibia to the
anterior and posterior meniscal insertions (Fig. 10-11). The posterior insertion should
be just anterior to the PCL and is easier to visualize through the posteromedial incision
while the guide is placed through the anterolateral portal. An oblique incision in
Langer' s lines is made in the area in which the K-wires will exit. There should be a 7-
to 10-mm cortical bridge between the exit of the anterior and posterior sutures. Both
K-wires are replaced with suture passers that are pulled through the anterolateral
incision and clamped (Fig. 10-12).
The graft is passed from the anterior incision into the joint with the assistance of a
tonsil that helps to shuttle the rim sutures through the posteromedial incision. The
posterior horn suture is passed through the posterior suture passer and is pulled
through the tibia. The anterior suture is then shuttled in similar fashion for the
anterior attachment. Tension is placed on the sutures while the fit of the meniscus is
checked with the arthroscope. The sutures are then tied over the anterolateral tibial
cortex. A free needle is used to pass the previously placed suture in the posterior horn
of the graft around the native meniscal remnant and capsule (Fig. 10-13). The anterior
horn is sutured with the previously placed sutures in the capsule/remnant. Using zone-
specific cannulas, an inside-out repair is carried out for the body of the transplant. All
incisions are closed in layers.
FIGURE 10-11
With the assistance of an ACL guide, a K-wire is drilled through the posterior
medial meniscal root attachment for later passage of the allograft suture.
FIGURE 10-12
The meniscus (prepared without bone plugs) and suture placement for fixation to
the joint capsule.
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FIGURE 10-13
The reduced meniscus after the anterior bone plug has been pulled into place and
the posterior horn is sutured to the capsule open.
Pearls and Pitfalls

Reconfirmation of the indication for meniscal transplantation is performed
with arthroscopy before thawing the graft. Subsequently, confirmation of the
correct meniscus (right vs. left, medial vs. lateral) is performed before making
the arthrotomies in case the incorrect meniscus has been sent and the
procedure needs to be terminated.
Portal placement is extremely important. The anteromedial portal should be

just adjacent to the medial border of the patellar tendon to allow the bone
trough to be created in the correct position next to the ACL. A portal that is
created too medial will cause the surgeon to fight the soft tissues to prevent
the osteotome from migrating into the lateral plateau.
Adequate arthroscopic debridement of the remaining meniscus to stable, well-

vascularized tissue ensures that the transplant will have the greatest potential
for cell ingrowth. In the medial transplant it is simpler to perform after the
arthrotomies are created using an arthroscopic punch. For the most part, the
bed for the lateral transplant can be prepared arthroscopically.
During introduction of the lateral meniscus into the joint, it is important to

reduce the bone block into the trough before pulling the meniscus to the
capsule and reducing it into the joint. Slack must be left on the posterior
suture until the bone block is in place.
Because the medial compartment is much tighter than the lateral

compartment, the graft may be difficult to get into the compartment. Some
authors have removed the medial tibial spine, partially released the medial
collateral ligament, or placed an external fixator to distract the compartment
to facilitate graft passage.
In the immediate postoperative period up until 4 weeks, a hinged postoperative brace
is kept locked in extension at all times except during range of motion (ROM) exercises.
Twenty percent weight bearing is permitted with crutches. A continuous passive
motion (CPM) machine is used from 0 to 45 degrees and progressed 10 to 15 degrees
per week until full flexion is reached. Quadriceps rehabilitation is started using
quadriceps sets and straight leg raises. Active assisted ROM using heel slides and calf
pumps is also begun.
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At 4 to 8 weeks postsurgery, the brace is unlocked for walking and sleeping. Weight
bearing as tolerated is permitted with crutches. The CPM machine is discontinued.
Isometric quadriceps rehabilitation and active assisted ROM are continued. The knee
brace is removed at 6 weeks. Patients may return to sedentary work at 4 weeks.
At 8 to 12 weeks postsurgery, stationary bike and short-arc quadriceps strengthening

are added. Walking with one crutch or a cane and driving are permitted.
At 3 to 6 months postsurgery, patients progress to walking without a crutch or cane.

Rehabilitation progresses to include stepper exercises, treadmill walking, swimming
(no breast stroke), and jogging in a pool. Closed chain and eccentric strengthening are
added.
Patients who are 6 to 9 months postsurgery may begin open-chain, proprioception, and
activityspecific conditioning. When patients are 9 months postsurgery, they may return
to light sports and heavy labor. In general, return to high-demand, impact sports is not
recommended.
COMPLICATIONS
Meniscal allograft transplantation sensitizes both the humeral and cell-mediated
immune responses to create local and systemic responses to the graft. Lyophilizing and
deep-freezing the grafts decreases the antigenicity, whereas cryopreservation
maintains HLA antigens and host sensitization. However, based on the current studies
comparing clinical success and immunogenicity, there does not appear to be any
correlation between sensitization and outcome.
Complications include continued or increased compartment pain, full or partial loss of

graft function, failure of peripheral healing of the meniscus, progression of
degenerative arthrosis, and infection. In general, continued or increased pain,
complete failure to heal, and infection require complete graft removal. Partial
meniscectomy is the treatment of choice for partial tears of the graft. Significant
advancement of osteoarthrosis is treated with total knee arthroplasty in suitable
candidates.
RESULTS
Surgeon expectations from meniscal allograft transplantation are for greater than 90%
subjective improvement in pain, instability, and function. However, objectively 20% to
40% of patients are rated as clinical failures by having repeat surgery for either partial
or total graft removal. As for the goal of preventing the progression of degenerative
changes of the articular surface, studies to date support this only in compartments
with grade I or II changes.
RECOMMENDED READING
Cameron JC, Saha S. Meniscal allograft transplantation for unicompartmental arthritis
of the knee. Clin Orthop Rel Res. 1997;337:164-171.
Cole BJ, Dennis MG, Lee SJ, et al. Prospective evaluation of allograft meniscus
transplantation: a minimum 2-year follow-up. Am J Sports Med. 2006;34:919-27.
Garrett JC. Meniscal allograft transplantation. Sports Med Arthrosc Rev. 1993;2:164-
167.
Johnson DL, Swenson TM, Harner CD. Meniscal reconstruction using allograft tissue: an
arthroscopic technique. Oper Tech Sports Med. 1994;2:223-231.
Johnson DL, Swenson TM, Livesay GA, et al. Insertion-site anatomy of the human
menisci: gross, arthroscopic, and topographical anatomy as a basis for meniscal
transplantation. Arthroscopy. 1995;11:386-394.
Kuhn JE, Wojtys EM. Allograft meniscus transplantation. Clin Sports Med. 1996;15:537-
556.
Milachowski KA, Weismeier K, Worth CJ, et al. Homologous meniscus transplantation:

experimental and clinical results. Int Orthop. 1989;13:1-11.
Sekiya JK, Ellingson CI. Meniscal allograft transplantation. J Am Acad Orthop Surg.
2006;14:164-174.
Verdonk PC, Verstraete KL, Almqvist KF, et al. Meniscal allograft transplantation: long-
term clinical results with radiological and magnetic resonance imaging correlations.
Knee Surg Sports Traumatol Arthrosc. 2006;7:1-13.
Wirth CJ, Peters G, Milachowski KA, et al. Long-term results of meniscal allograft
transplantation. Am J Sports Med. 2002;30:174-181.
Yoldas EA, Sekiya JK, Irrgang JJ, et al. Arthroscopically assisted meniscal allograft
transplantation with and without combined anterior cruciate ligament reconstruction.
Knee Surg Sports Traumatol Arthrosc. 2003; 11:173-182.
> Table of Contents > Part Two - Meniscus Surgery > 11 - Medial and Lateral Meniscus
Transplantation: Arthroscopic-Assisted Techniques and Clinical Outcomes
11
Medial and Lateral Meniscus Transplantation:
Arthroscopic-Assisted Techniques and Clinical
Outcomes
Frank R. Noyes
Sue D. Barber-Westin
T he meniscus provides important functions in the human knee, including load bearing, shock
absorption, stability, and joint nutrition, that are vital for the integrity of the articular
cartilage. While many meniscus tears can be successfully repaired, including complex tears
that extend into the central avascular region (23 ), not all are salvageable, especially if
considerable tissue damage has occurred. Transplantation of human menisci is hypothesized to
restore partial load-bearing meniscus function, decrease patient symptoms, and provide
chondroprotective effects (7 ,15 ,27 ). However, the procedure remains in an evolving state,
as investigations of tissue-processing, secondary sterilization, and long-term function continue
to evaluate its effectiveness. The optimal candidate is a young patient who has had a total
meniscectomy and has pain and mild articular cartilage deterioration in the involved
tibiofemoral compartment. There are few treatment options for these individuals and the goal
of meniscal transplantation in the short term is to decrease pain, increase knee function,
allow pain-free activities of daily living, and delay the onset of tibiofemoral arthrosis.
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The indications for a meniscus allograft are:
Prior meniscectomy
Age of 50 years or less
Pain in the meniscectomized tibiofemoral compartment
No radiographic evidence of advanced joint deterioration (â‰¥2 mm of tibiofemoral

joint space demonstrated on 45-degree weight-bearing posteroanterior radiographs [22
] and no or only minimal bone exposed on tibiofemoral surfaces)
Normal axial alignment and a stable joint are required, as untreated abnormal limb alignment
and anterior cruciate ligament (ACL) deficiency have correlated with a high rate of meniscus
allograft failure in several studies. Axial correction is recommended in knees in which the
weight-bearing line is less than 45% (varus) or greater than 55% (valgus), representing a 2- to
3-degree change from normal alignment. An osteochondral autograft transfer or autologous
chondrocyte implantation may be required in knees with full-thickness articular cartilage
defects in the meniscectomized tibiofemoral compartment, which may be done either during
or before the meniscus transplant procedure. These cartilage procedures expand the
indications for meniscus transplantation in knees with femoral articular cartilage defects.
Contraindications for a meniscus allograft are:
Advanced knee joint arthrosis with flattening of the femoral condyle, concavity of the
tibial plateau, and osteophytes that prevent anatomic seating of the meniscus allograft
(14 )
Varus or valgus axial malalignment
Knee joint instability
Knee arthrofibrosis
Muscular atrophy
Prior joint infection
Symptomatic patellofemoral articular cartilage deterioration

Obesity (>30 body mass index)
We do not recommend a prophylactic meniscus transplantation after total meniscectomy in

asymptomatic patients who do not have articular cartilage deterioration, as long-term
predictable success rates are not available. In addition, the operative procedure does carry a
slight risk for complications that could make the patient's condition worse. There are
sophisticated magnetic resonance imaging (MRI) techniques that are useful in determining the
integrity of the articular cartilage and detect early deterioration prior to the onset of
symptoms (20 ). A relative indication of meniscus transplantation is early (mild) arthritic
deterioration in the involved tibiofemoral compartment in an asymptomatic patient.
A thorough history is taken that includes assessment of prior operative records and current
symptoms and functional limitations. The validated Cincinnati Knee Rating System (3 ) is used
to rate pain, swelling, and giving-way of the knee as well as limitations with daily, athletic,
and occupational activities. A comprehensive evaluation of the knee is conducted, which
documents range of knee motion, joint effusion, tibiofemoral joint pain on palpation and
during joint motion, and tibiofemoral joint crepitus. We also evaluate the patellofemoral
joint, all knee ligaments, and gait abnormalities (17 ).
Diagnostic imaging includes bilateral anteroposterior radiographs in full extension, a 45-degree

weight-bearing posteroanterior radiograph (22 ), and an axial view of the patellofemoral joint.
The anteroposterior and lateral radiographs are taken with a magnification marker to obtain
width and length measurements for the meniscus transplant (18 ). Axial lower limb alignment
is measured using full standing hip-knee-ankle weight-bearing radiographs in knees that
demonstrate varus or valgus alignment. MRI using fast-spin-echo techniques (19 ,20 ) are used
in select cases to evaluate the condition of the articular cartilage and subchondral bone.
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Selection of Meniscus Implant

A variety of sterilization techniques have been described for meniscus allografts, including
none (fresh-frozen), irradiated, and cryopreserved, and no scientific data exist to select one
type of graft processing method over another. Some authors advocate secondary sterilization
using low-dose irradiation (1-2 Mrads) for purposes of bacterial sterilization (6 ). We
recommend that allografts be obtained from tissue banks that have been accredited by the
American Association of Tissue Banking and inspected by the Food and Drug Administration,
where serological testing meets or exceeds the standards of these organizations (26 ).
Importantly, donor selection criteria may vary between tissue banks, and the surgeon should
understand the specific criteria used by the bank chosen to supply the transplant. Surgeons
should also be aware that even though allograft tissues are generally processed aseptically,
this may not prevent contamination and does not guarantee a sterile graft. We use
prophylactic antibiotics intravenously before surgery and carefully monitor patients
postoperatively for any signs of infection. The implications of different processing techniques
on graft sterility is beyond the scope of this chapter, but have been discussed in detail by
others (4 ,7 ,26 ).
We advise the surgeon to request that the tissue bank provide a photograph of the transplant
that has been selected for the patient before surgery. A metric ruler should be placed
adjacent to the transplant in the photograph to ensure that the allograft is of adequate size
and width. Medial menisci may have a hypoplastic anterior horn that is narrow, inserting distal
to the medial tibial surface (type III)(5 ); these menisci are not acceptable for implantation.
The middle one third of a medial or lateral meniscus may be 8 to 10 mm in width and only
suitable for small patients. The lateral meniscus may have reduced anteroposterior length,
less than that calculated on the sagittal radiograph, and therefore not suitable for
implantation.
SURGICAL TECHNIQUE
Transplant Preparation
The meniscus is thawed, inspected, and prepared before administration of patient anesthesia
because it is difficult to detect implant defects through the plastic packaging, and the
meniscus must be thoroughly evaluated. For a lateral meniscus allograft, implant preparation
is accomplished first to determine the depth and width required for the tibial slot for the
central bone bridge technique. The central bone portion of the lateral meniscus transplant
incorporates the anterior and posterior meniscal attachments and usually measures 8 mm wide
in smaller patients and 9 mm in larger patients. The length of the bone attachment is usually
35 mm, but this can be altered as required. The posterior 8 to 10 mm of tibia bone that
protrudes beyond the posterior horn attachment is removed to later produce a buttress
against the bone slot in the host knee. Commercially available (Stryker Endoscopy Co.,
Kalamazoo, MI, and Cryolife Inc., Kennesaw, GA) sizing blocks and channel cutters are helpful
for appropriate sizing. There is also a dove-tail technique that the surgeon may elect, which
has the advantage of providing added stability to the fixation at the tibial bone portion of the
transplant (Fig. 11-1 ). This procedure entails cutting a trapezoidal bone block which includes
a narrow, 7-mm bone bridge that preserves the ACL tibial attachment site.
Medial meniscus transplants are not prepared until it is determined if the central bone bridge
technique (which is preferred) or the two-tunnel technique (separate anterior and posterior
bone attachments and tunnels) will be performed, as we will describe in a following section.
It should be noted that the normal ACL tibia attachment site, or a planned ACL tibial graft
tunnel, may compromise lateral or medial meniscus transplant tibial fixation in smaller
patients who do not have sufficient width of the central tibial region to accommodate both
the transplant and ACL tibial attachment or tunnel. Since it is not possible in these knees to
use the central slot technique when an ACL reconstruction is to be performed concurrently,
the two-tunnel procedure is selected for a medial meniscus transplant. More commonly, the
two operations are staged by performing the ACL reconstruction first and the meniscus
transplant procedure several months later.
Lateral Meniscus Transplantation

The patient is placed in a supine position on the operating room table with a tourniquet
applied with a leg holder, and the table adjusted to allow 90 degrees of knee flexion. The
opposite lower extremity
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is placed in a thigh-high elastic stocking and is padded to maintain mild hip flexion to decrease
tension on the femoral nerve. After examination under anesthesia, diagnostic arthroscopy is
done to confirm the preoperative diagnosis and assess articular cartilage changes. A meniscus
bed of 3 mm is retained when possible, except at the popliteal tendon region. In knees that
require a cruciate ligment reconstruction, an arthroscopically assisted approach is used (11 ).
The femoral and tibial tunnels are drilled, and the ligament graft is passed through the tunnels
with femoral fixation done first, followed by the meniscal transplantation, and then tibial
cruciate graft fixation. Performing ligament graft fixation at the tibia as the final step allows
for maximum separation of the tibiofemoral joint during meniscal transplantation. This also
prevents potential ligament fixation problems during the operation.
FIGURE 11-1
Dovetail meniscus allograft technique. A: An outline of the dovetail bone block is drawn on the
end of the bone plug. An implant jig system is used to hold the meniscus implant and produce
the desired bone cuts. B: Final appearance showing typical dimensions for the trapezoidal
bone block prior to transplant insertion. (Reprinted with permission, Arthrex Meniscal Allograft
Transplantation Featuring Dovetail Meniscal Allograft Technique. Carter TR. Arthrex, Naples,
Florida, 2004.)
The tourniquet is inflated only for the two operative approaches. A limited 3-cm lateral
arthrotomy is made just adjacent to the patellar tendon. Although arthroscopic techniques are
available to prepare the tibial slot, we believe the limited arthrotomy provides superior
visualization and avoids incising into the patellar tendon, which must be displaced medially to
properly place the tibial slot. A common mistake involves placing the central tibial slot lateral
to the normal attachment of the anterior horn of the lateral meniscus. A second 3-cm
posterolateral incision is made just behind the lateral collateral ligament (Fig. 11-2 ) (10 ,24 ).
The interval between the short head of the biceps muscle and the iliotibial band is identified
and incised. The lateral head of the gastrocnemius is gently dissected with Metzenbaum
scissors off the posterior capsule at the joint line just above the fibular head. Care is taken at
this point, as dissection that extends too far proximal to the joint line at the posterolateral
aspect would enter the joint capsule. If this occurs, a capsular repair is required to maintain
joint integrity during the inside-out meniscal repair procedure. The inferior lateral geniculate
artery, also in close proximity, is identified and preserved. The space between the
posterolateral capsule and the lateral head of the gastrocnemius is further developed bluntly.
An appropriately sized popliteal retractor (Stryker Co., Kalamazoo, MI) is placed directly
behind the lateral meniscus bed and anterior to the lateral gastrocnemius muscle.
The width of the transplant is determined, and a template made out of aluminum foil of the
same width and length is cut and inserted into the lateral compartment to determine the
proper placement of the bone slot. This sizing step is important to ensure there is no lateral
overhang of the meniscal body produced by not placing the bone far enough medially. A
rectangular bone slot is prepared at the anterior and posterior meniscus tibial attachment
sites to match the dimensions of the prepared transplant. The sequence of steps to prepare
the lateral tibial slot is shown in Figure 11-3 . The tibial
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bone slot is 1 to 2 mm wider than the transplant to facilitate implantation. The anterior and
posterior horns of the implant are placed into their normal attachment locations, adjacent to
the ACL.
FIGURE 11-2
A: Site of the posterolateral incision for a lateral meniscus transplant. B: Incision site in the
interval between the posterior edge of the iliotibial band and the anterior edge of the biceps
tendon. C: The interval between the lateral gastrocnemius and posterolateral capsule is
opened bluntly, just proximal to the fibular head avoiding entering the joint capsule
proximally. (Redrawn after Noyes FR, Barber-Westin SD, Rankin M. Meniscal transplantation in
symptomatic patients less than fifty years old. Surgical technique. J Bone Joint Surg Am
2005;87(suppl 1):149-165, with permission.)
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FIGURE 11-3
Tibial slot technique for lateral meniscus transplantation. The technique is shown for the
lateral meniscus. An arthroscopic-assisted technique or mini-lateral arthrotomy may be used in
this procedure. The authors prefer the mini-arthrotomy as it offers superior visualization and
avoids incising the patellar tendon. A: Establish a line connecting the center of the anterior
and posterior horn attachments using an electrocautery device. In the mini-open arthrotomy,
a template of the meniscus coronal width is used to verify the medial-to-lateral width of the
transplant to move the slot appropriately to prevent tibial overhang of the implant. B: A burr
removes the tibial spine and creates a 4-mm straight anterior-to-posterior reference slot along
the plane of the tibial slope. This calibrated guide pin sits flush with the articular cartilage. C:
The drill guide uses a guide pin that has been marked with a laser to set the depth of a second
guide pin, allowing a drill to ream 5 mm less to retain the posterior portion of the tibial slot.
D: The 8-mm drill bit with a collar at the defined depth is used, followed by use of a box
cutter to create a rectangular slot of the desired depth and width. (â€œSlot Instruments for
Meniscal Transplantation: Surgical Technique.â€• Farr J and Cole B. Stryker Endoscopy, San
Jose, CA.) (Adapted from Noyes FR, Barber-Westin SD, Rankin M. Meniscal transplantation in
symptomatic patients less than fifty years old. Surgical technique. J Bone Joint Surg Am.
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FIGURE 11-3
(Continued )
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FIGURE 11-3
(Continued ) D: The 8-mm drill bit with a collar at the defined depth is used, followed by use
of a box cutter to create a rectangular slot of the desired depth and width. E: The lateral
meniscus implant with the central bone bridge technique is ready to be placed into the tibial
slot. A detailed description of the surgical steps and operative instruments is available.
(â€œSlot Instruments for Meniscal Transplantation: Surgical Technique.â€• Farr J and Cole B.
Stryker Endoscopy, San Jose, CA.) (Adapted from Noyes FR, Barber-Westin SD, Rankin M.
Meniscal transplantation in symptomatic patients less than fifty years old. Surgical technique.
J Bone Joint Surg Am. 2005;87(suppl 1):149-165, with permission.)
An alternative technique uses a starter chisel and finishing chisels to fashion the tibial slot to
its final depth and width (Fig. 11-4A ). A tibial slot sizing guide is used to check the length and
depth (Fig. 11-4B ). The allograft sizing block (Fig. 11-4C ) confirms that the allograft bone
bridge is of the correct width and depth.
The implant is inserted into the slot (Fig. 11-3E ), and the bone portion of the graft is seated
against the posterior bone buttress to achieve correct anterior-to-posterior placement of the
attachment sites. A vertical suture in the posterior meniscus body is passed posteriorly to
provide tension and facilitate implant placement and is later tied. The knee is flexed,
extended, and rotated to confirm correct placement of the transplant has been obtained.
Sutures are placed into the anterior one third of the meniscus, attaching it to the prepared
meniscus rim under direct visualization.
Two fixation methods are available for the central bone attachment. Two 2-0 nonabsorbable
sutures (Ticron, Davis and Beck Co., Wayne, NJ, or Ethibond, Ethicon Inc., Somerville, NJ) may
be placed over the central bone bridge, brought through a drill hole, and tied to a tibial post.
We prefer to place an interference screw (7 Ã— 25 mm, comprised of an absorbable composite
material) adjacent medially to the bone bridge (8 ). A tap is inserted over the guidewire to
create a path for the interference screw with the bone bridge held in place manually. The
arthrotomy is closed, and the inside-out meniscal repair is completed with multiple vertical
divergent sutures, which are placed first superiorly to reduce the meniscus (Fig. 11-5 ), and
then inferiorly in the outer one third of the implant. Sutures are not placed in the middle and
inner thirds of the meniscus to avoid weakening the implant due to its limited healing
capability in these regions (Fig. 11-6 ).
Medial Meniscus Transplantation

The patient is placed in a supine position on the operating room table with a tourniquet
applied with a leg holder, and the table adjusted to allow 90 degrees of knee flexion. The
opposite lower extremity
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is placed in a thigh-high elastic stocking and is padded to maintain mild hip flexion (to
decrease tension on the femoral nerve). After examination with the patient under anesthesia,
diagnostic arthroscopy is done to confirm the preoperative diagnosis and assess articular
cartilage changes.
FIGURE 11-4
An alternative technique uses a starter chisel and finishing chisels to fashion the tibial slot to
its final depth and width (A) . A tibial slot sizing guide is used to check the length and depth
(B) . The allograft sizing block (C) confirms that the allograft bone bridge is of the desired
width and depth. A detailed description of the surgical steps and operative instruments is
available. (â€œMeniscus Reconstruction Trough Surgical Technique.â€• Halbrecht JL. Cryolife,
Inc., Kennesaw, GA) (Reprinted with permission from Noyes FR, Barber-Westin SD, Rankin M.
Meniscal transplantation in symptomatic patients less than fifty years old. Surgical technique.
J Bone Joint Surg Am. 2005; Suppl 1; 149-165.
FIGURE 11-5
Cross-section showing spoon (a popliteal retractor may also be used) between the lateral
gastrocnemius and the posterior capsule. A single cannula is introduced from the adjacent
portal to facilitate placement of the vertical sutures into the periphery of the meniscus
implant. (Adapted from Noyes FR, Barber-Westin SD, Rankin M. Meniscal transplantation in
symptomatic patients less than fifty years old. Surgical technique. J Bone Joint Surg Am.
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FIGURE 11-6
Lateral meniscus graft in place and sutured. (Adapted from Noyes FR, Barber-Westin SD,
Rankin M. Meniscal transplantation in symptomatic patients less than fifty years old. Surgical
technique. J Bone Joint Surg Am. 2005;87(suppl 1):149-165, with permission.)
The tourniquet is inflated only for the anteromedial and posteromedial surgical approaches. A
4-cm skin anteromedial incision is made adjacent to the tibial tubercle and patellar tendon. A
second 3-cm vertical posteromedial incision is made, similar to that described for inside-out
meniscus repairs (10 ) (Fig. 11-7 ). The fascia is incised anterior to the sartorius muscle, and
the pes anserine muscle groups are retracted posteriorly. The interval between the
semimembranosus tendon and the capsule is sharply dissected. The tendon sheath of the
semimembranosus is incised to facilitate exposure. The layer between the medial
gastrocnemius tendon and the posterior capsule is separated with blunt dissection. Great care
is taken to identify and avoid injury to the infrapatellar branches of the saphenous nerve. The
two approaches are performed with the tourniquet inflated to 275 mm and usually require 15
minutes; otherwise, the tourniquet is not used.
FIGURE 11-7
The accessory posteromedial approach is shown for a medial meniscus allograft. A: Site of the
posteromedial skin incision.
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FIGURE 11-7
(Continued) B: The incision is shown through the anterior portion of the sartorius fascia. C:
The interval is opened between the posteromedial capsule and the gastrocnemius tendon, just
proximal to the semimembranosus tendon. The fascia over the tendon is excised to its tibial
attachment to facilitate retrieval of the meniscus sutures. (Adapted from Noyes FR, Barber-
Westin SD, Rankin M. Meniscal transplantation in symptomatic patients less than fifty years
old. Surgical technique. J Bone Joint Surg Am. 2005;87(suppl 1):149-165, with permission.)
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The goal of the operative procedure is to transplant the medial meniscus and bone
attachments into the normal anterior and posterior attachments and to suture the transplant
to maintain the desired position in the knee joint. An aluminum foil template of the medial
meniscus transplant is measured according to its anterior-posterior and medial-lateral
dimensions and is inserted through the anterior arthrotomy to measure the medial tibial
plateau. This allows the position of the central bone slot to be marked and to determine if the
meniscus implant will be properly positioned adjacent to the ACL tibial attachment without
medial tibial overhang. The anterior and posterior meniscus attachment locations are verified
to be at the anatomically correct sites. The central bone bridge technique removes 4 to 6 mm
of the medial tibial eminence. If the implant is suitable and no medial tibial overhang is
present, then the central bone bridge technique may be used. If the implant needs to be
adjusted to fit to the medial tibial plateau by attaching the anterior horn placement further
laterally, then the two-tunnel technique is selected. This sizing step is critical to obtain
proper placement of the medial meniscus implant into the host tibia. In many knees, the
central slot technique will not be possible due to a sizing problem that results in excessive
medial displacement of the meniscus body or that would compromise the ACL tibial
attachment. Once the appropriate technique has been determined, the meniscus allograft is
prepared.
Medial Meniscus Central Bone Bridge Technique

The technique is similar as that already described for lateral meniscus transplantation. A
reference slot is first made on the tibial plateau in the anteroposterior direction. A guide pin
is positioned in the slot, inferiorly on the tibia, and a cannulated drill bit is placed over the
pin to drill a tunnel. The final tibial slot is 8 to 9 mm wide and 10 mm deep. A rasp is used to
smooth the slot to allow insertion of the allograft central bone bridge.
Alternatively, the dove-tail technique may be used, as previously discussed for a lateral
meniscus transplant. The central bone bridge of the allograft is sized to a width of 7 mm (1
mm less than the dimension at the tibial site) and a depth of 10 mm (8 ). This allows
adjustment of the central bone bridge position in the anterior-posterior direction while the
meniscus is positioned to fit in the anatomically correct position relative to the femoral
condyle.
A vertical suture is placed through the posterior meniscus horn and advanced through the
capsule to exit through the posteromedial incision. The meniscus is passed through the
arthrotomy into the knee with tension placed on the posterior suture to facilitate proper
positioning in the knee joint. Care is taken to align the bone bridge with the recipient tibial
slot. The knee is taken through flexion and extension and tibial rotation to align the implant.
Once the appropriate anterior-posterior central bone bridge position is achieved, a guidewire
is inserted between the bone bridge and the lateral side of the slot. A tap is inserted over the
guidewire to create a path for an interference screw with the bone bridge held in place
manually. An absorbable bone interference screw is inserted adjacent to the bone bridge.
Occasionally, there will be an osteophyte on the anterior portion of the medial tibial plateau
that must be resected to avoid meniscal implant compression. The anterior horn of the
meniscus is sutured with vertical divergent sutures (2-0 Ethibond) under direct visualization
(Fig. 11-8 ). The anterior arthrotomy is closed and the inside-out vertical divergent sutures are
placed, as described, to suture the meniscus to the meniscus bed, removing any implant
undulations and restoring circumferential meniscal tension. The central bone bridge of the
implant provides for fixation of the anterior and posterior portions of the implant and
subsequent healing to the host tibia (Fig. 11-9 ).
Medial Meniscus Two-Tunnel Technique

If it is determined that the central bone bridge technique is not acceptable, the surgeon must
prepare separate anterior and posterior bone attachments for the meniscus transplant that
will be secured to the normal anatomic attachment sites (Fig. 11-10 ). The transplant is
prepared with a posterior bone plug 8 mm in diameter and 12 mm in length. Although
techniques have been described that use only a soft-tissue fixation of the posterior horn by
removing the bone portion of the meniscus graft to allow easier graft passage, we believe that
this provides less secure fixation and leads to a higher failure rate. The anterior bone
attachment is 12 mm in width, length, and depth. Two 2-0 nonabsorbable Ethibond sutures
(Ethicon, Somerville, NJ) are passed retrograde through each bone attachment, with two
additional locking sutures placed in the meniscus adjacent to the bone attachment for
subsequent secure fixation.
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FIGURE 11-8
Cross-section showing the arthroscope, needle cannula, and spoon in place. A popliteal
retractor may also be used, which the authors prefer. The meniscus is passed through the
arthrotomy into the knee with tension placed on the sutures to facilitate proper positioning in
the knee joint. Using a single-barrel cannula, the suture is advanced through the capsule at
the corresponding attachment site of the meniscus and exits through an accessory incision.
(Adapted from Noyes FR, Barber-Westin SD, Rankin M. Meniscal transplantation in symptomatic
patients less than fifty years old. Surgical technique. J Bone Joint Surg Am. 2005;87(suppl
1):149-165, with permission.)
A guide pin is placed adjacent to the tibia tubercle and is directed to the anatomic posterior
meniscus attachment. A tibial tunnel is drilled over the guidewire to a diameter of 8 mm. The
bone tunnel edges are champhered and slightly enlarged with a curette to allow easier passage
of the graft into the tibial tunnel. A limited medial femoral condyle notchplasty is usually
required. At least 8 mm of opening adjacent to the posterior cruciate ligament (PCL) and
medial femoral condyle is required to pass the posterior bone attachment of the graft. On
occasion, a subperiosteal release of the long fibers of the tibial attachment of the medial
collateral ligament (with later suture anchor repair) is required to open the medial
tibiofemoral joint sufficiently. The meniscus bed is prepared by removing any remaining
meniscus tissue, while preserving a 3-mm rim when possible. The meniscus bed is rasped for
revascularization of the graft.
The graft is passed through the anteromedial arthrotomy. The surgeon is seated with a
headlight in place, and the knee is flexed to 90 degrees. A guidewire is passed retrograde
through the tibial tunnel, and the sutures attached to the posterior bone are retrieved. A
second suture is placed into the posterior horn and is passed inside-out through the
posteromedial approach to guide the meniscus.
The knee is flexed to 20 degrees under a maximum valgus load to pass the posterior bone
portions of the graft, with the secondary meniscus body suture being held by an assistant. A
nerve hook is used to gently assist the passage of the graft. With direct visualization, it is
possible to confirm appropriate meniscus graft passage and positioning into the medial
tibiofemoral compartment. Care is taken not to advance the posterior meniscus body too far
into the tibial tunnel, but only to seat the bone portion of the graft in order to not shorten the
overall circumferential length of the meniscus graft. The posterior meniscus bone attachment
sutures are tied over the tibial post to provide tension to the posterior bone attachment. One
or two sutures are passed to secure the posterior horn. The knee is flexed and extended to
assess meniscus fit and displacement. The optimal location for the anterior meniscal bone
attachment is identified with appropriate lateral placement to restore proper meniscus
position and prevent medial transplant overhang. The knee is placed in full extension, and the
position of the transplant is verified to be correct. A 12-mm rectangular bone attachment is
fashioned in the tibia to correspond to the anterior bone attachment of the meniscus graft. A
4-mm bone tunnel is placed at the base of this bone slot and exits at the anterior tibia just
proximal to the posterior bone tunnel. The sutures are passed through the bone tunnel, and
the anterior horn is seated. Full knee flexion and extension are again performed to determine
proper graft placement and fit. Tension is applied to the anterior bone sutures, which are not
tied at this point but are used to maintain tension in the graft during the inside-out suture
repair. This meticulous seating of the meniscus transplant under circumferential tension with
bone attachment of both the anterior and posterior horns is believed to be crucial for
meniscus weight-bearing position and function.
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FIGURE 11-9
Weight-bearing posteroanterior radiograph of a 36-year-old woman 6 years after medial
meniscus central bone bridge implantation shows incorporation of the bone bridge into the
host, with preservation of the medial joint space. (Adapted from Noyes FR, Barber-Westin SD,
Rankin M. Meniscal transplantation in symptomatic patients less than fifty years old. Surgical
technique. J Bone Joint Surg Am. 2005;87(suppl 1):149-165, with permission.)
FIGURE 11-10
Two-tunnel technique for medial meniscus allografts. The illustration shows insertion of
transplant, including posteromedial suture placed to facilitate meniscus reduction. The
anterior and posterior bone attachments of the medial meniscus transplant are fixed into
separate tibial tunnels. (Adapted from Noyes FR, Barber-Westin SD, Rankin M. Meniscal
transplantation in symptomatic patients less than fifty years old. Surgical technique. J Bone
Joint Surg Am. 87 2005; Suppl 1: 149-165, with permission.)
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The anterior arthrotomy is closed, and the arthroscope is inserted into the anterolateral portal
for the posterior meniscal repair and into the anteromedial portal for the middle and anterior
one-third repairs, with the single needle cannula inserted in the other anterior portal. The
meniscal repair is performed in an inside-out fashion, starting with the posterior horn, with
use of multiple vertical divergent sutures of 2-0 nonabsorbable Ethibond both superiorly and
inferiorly, constantly tensioning the meniscus from posterior to anterior to establish
circumferential tension. The assistant is seated with a headlight and retrieves the suture
needles through the posteromedial approach. Each suture is placed and tied bringing the
meniscus directly to the meniscus bed with observation that correct meniscus placement,
fixation, and tension exist. We do not consider an alternative technique of using meniscus
fixators, as this lessens the ability to precisely secure and restore tension to the meniscus
transplant. The anterior arthrotomy is opened and final tensioning and fixation of the anterior
horn bone attachment is performed. Occasionally, additional sutures are required to secure
the most anterior one third of the meniscus to the capsular attachments. This step is
performed under direct vision (Fig. 11-11 ). An alternative method of fixation is to use a 3.5-
mm cancellous screw with a washer for fixation of the anterior horn meniscus-bone
attachment.
FIGURE 11-11
Final anterior and posterior tunnel fixation appearance of medial meniscus transplant and
vertical divergent sutures. (Adapted from Noyes FR, Barber-Westin SD, Rankin M. Meniscal
transplantation in symptomatic patients less than fifty years old. Surgical technique. J Bone
Joint Surg Am. 2005;87(suppl 1):149-165, with permission.)
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After final inspection of the graft with knee flexion and extension and tibial rotation, the
operative wounds are closed in a routine fashion.
Pearls and Pitfalls

We advise patients that the transplant will be inspected in the operating room just
before the administration of anesthesia. At this time, the decision will be made to
proceed if the graft is deemed suitable. There is also the remote possibility that during
the operation, either the final preparation or the implantation of the transplant may
not be possible due to problems with graft size or the ability to obtain correct
positioning in the joint. Although these problems are rare, the patient should be
advised.
The preparation of the meniscus template is critical to obtain successful placement of

the final tibial slot and ensure correct positioning of the transplant. The template is
made of aluminum foil to represent the size of the implant and is inserted through the
limited anterior arthrotomy incision.
The slot placement for the transplant must be exact. Otherwise, the meniscus may be
displaced at its midportion outside the joint, or positioned too far inside the joint and
subsequently incur excessive compression and tearing. It is possible to realign the bony
slot a few millimeters medially or laterally in the coronal plane and use an absorbable
interference screw for fixation in the final coronal adjustment of the implant.
The use of multiple vertical divergent sutures is required to position the transplant in
the anatomically correct manner. There are wavy areas in the implant with loss of
circumferential tension that can be successfully removed by correct placement of
these sutures.
We prefer the inside-out meniscus repair technique, which is viewed as the most
precise suturing method.
We avoid meniscal fixators, as it is not possible to provide the same secure fit and
exact placement of the implant as that obtained with sutures.
The sutures should not be placed in the middle and inner thirds of the meniscus, as
this may weaken the transplant.
A total of 12 to 15 sutures are placed both superiorly and inferiorly, in a vertical

direction. Horizontal sutures have poor holding ability and are therefore not used
during meniscal transplantation.
Care is taken to avoid damaging the articular cartilage. This technique requires two
surgical assistants, one dedicated to holding the lower limb to open the medial or
lateral tibiofemoral compartment for visualization of the implant, and another seated
to retrieve and tie the sutures at the posterior aspect of the joint.
The suturing of the lateral posterior horn adjacent to the posterior attachment
requires angulation of the suture needle away from the neurovascular structures.
In order for the meniscus transplant to function, it must be placed at the normal
anatomic insertion sites. The posterior horn attachment of the lateral meniscus, if
placed too far posteriorly, will not promote load sharing (1 ). Alternatively, too
anterior of a position of a medial meniscus transplant will produce excessive
compressive forces and damage the meniscus.
During medial meniscus transplantation, the template may indicate that the transplant
is excessively wide in the medial-to-lateral direction. If this occurs, the middle one
third of the transplant would rest outside of the medial tibial plateau to avoid
compromising the ACL attachment. In this case, the two-tunnel technique is selected
to obtain correct anatomic positioning and the desired subsequent circumferential
hoop stress.
We disagree with published techniques on medial meniscus transplantation in which

the posterior bone portion of a medial meniscus implant is not retained and the
fibrocartilaginous posterior horn is placed in a posterior tibial attachment tunnel.
Although far easier to prepare and implant surgically, there are inadequate scientific
data to support that the soft tissue ends of the meniscus implant (without the bone
attachment) will heal and provide the circumferential tension in the meniscus that is
required for function.
We disagree with published reports in which meniscus transplants are used in knees
with advanced tibiofemoral compartment arthrosis (25 ). In our experience, the
transplant extrudes from the joint in these knees, and its ability to decrease symptoms
or have a chondroprotective effect is lost (14 ).
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For medial meniscus transplants, the central bone slot is limited in its lateral-most
placement by the ACL tibial attachment. The anterior horn of the medial meniscus
must not be of a type III configuration (5 ), inserting too far distally on the anterior
tibial margin. If there is a medial-to-lateral size mismatch for a medial meniscus
transplant, then separate anterior and posterior bone attachments and tunnels are
required. The posterior bone-meniscus transplant is placed at the normal attachment,
and the anterior horn is placed in a medial-to-lateral direction to restore correct
tensioning and position in the joint.
For tight knees in which there are only a few millimeters of medial joint opening, the
central bone bridge technique avoids performing a partial detachment of the distal
medial collateral ligament which would otherwise be required to gain access to the
joint for suturing and avoid damage to the articular cartilage.
The initial goal of the postoperative rehabilitation program is to prevent excessive weight-
bearing and joint compressive forces that could disrupt the healing meniscus allograft (Table
11-1 ). Patients are placed in a long-leg postoperative brace immediately after surgery, and
the brace is worn for approximately 6 weeks. Knee range-of-motion exercises from 0 to 90
degrees are begun the first day. Knee flexion is increased 10 degrees each week to allow 135
degrees after 4 weeks. Patients are allowed only toe-touch (5 lb) weight bearing during the
first 2 weeks, and then are slowly progressed to bear 50% of body weight at 4 weeks and full
weight bearing at 6 weeks.
Flexibility and quadriceps strengthening exercises are begun immediately postoperatively.

Balance, proprioception, and closed-kinetic chain exercises are implemented when patients
achieve full weight bearing. Stationary bicycling with low resistance is begun at 8 weeks, and
swimming and walking programs are initiated between 9 and 12 weeks. Return to light
recreational sports is delayed for at least 12 months. Patients are advised not to return to
high-impact, strenuous athletics due to the joint damage present and questionable ability of
the meniscus transplant to restore normal load-bearing function.
COMPLICATIONS
We have not encountered knee arthrofibrosis or joint infection problems following meniscus
transplantation, although this is always a possibility after any major knee operation. Any
patient whose knee fails to achieve the extension and flexion goals is placed into an
intervention program previously described in detail (16 ). In our series of 40 consecutive
meniscus transplants, four knees required a gentle manipulation under anesthesia performed 4
to 6 weeks postoperative for a limitation of knee flexion. These patients had a concomitant
ligament reconstruction or osteochondral autograft transfer procedure.
The patient is observed for any loss of meniscus repair and stabilization in the initial
postoperative period. Rarely, a resuture and repeat fixation are required. Postoperative
radiographs are taken to assess bony incorporation of the transplant.
RESULTS
We have previously described the clinical outcome of 40 consecutive cryopreserved and 96
fresh-frozen irradiated medial and lateral meniscus transplants in detail (12 ,14 ,15 ,21 ).
Forty cryopreserved meniscus allografts were implanted into 38 patients from November 1995
to March 2000. Four of the meniscus transplants failed and were removed before the minimum
2-year follow-up study period; these cases were included in the overall failure rate. We
followed 35 patients (36 meniscus transplants; 18 lateral, 16 medial, 1 bilateral) a mean of 40
months (range, 24 to 69 months) postoperatively. There were 18 males and 17 females whose
mean age at surgery was 30 years (range, 14 to 49 years). A mean of 139 months (range, 12 to
372 months) had elapsed between the knee injury and the meniscus allograft procedure. A
total of 128 prior operative procedures had been done, including 61 partial or total
meniscectomies, 13 ACL reconstructions, four PCL reconstructions, one lateral collateral
ligament reconstruction, five high tibial osteotomies, and three osteochondral autograft
transfer procedures.
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Brace: long-leg postoperative
ROM minimum goals:
0-90 degrees
X
0-120 degrees
0-135 degrees
Weight bearing:
Toe touchâ€”1/4 body weight
1/2 to 3/4 body weight
Full
Patellar mobilization
Modalities:
Electrical muscle stimulation
Pain/edema cryotherapy
Flexibility: hamstring,
X
X
gastrocsoleus, iliotibial
band, quadriceps
Strengthening:
Quad iso, SLR, active knee exten
Closed-chain
Knee flexion hamstring curls (90 degrees)

X
Knee extension quads (90-30 degrees)
Hip abduction-adduction, multi-hip
X
X
Leg press (70-10 degrees)
Balance/proprioceptive training
Conditioning:
Upper body ergometer
X
X
Bike (stationary)
Aquatic program
Swimming (kicking)
X
X
Walking
Stair climbing machine
Ski machine
X
Light recreational sports
Iso, isometrics; ROM, range of motion; SLR, straight leg raises.
Closed chain: gait retraining, toe raises, wall sits, mini-squats
Balance/proprioceptive training: weight-shifting, mini-trampoline, Biomechanical Ankle

Platform System (BAPS, Camp, Jackson, MI), Kinesthetic Awareness Trainer (KAT, Breg, Inc.,
Vista, CA), plyometrics.
(Reprinted with permission from Noyes FR, Barber-Westin SD. Meniscus transplantation:
indications, techniques, clinical outcomes.
In: Pellegrini VD, ed: Instructional course lectures , vol. 54. Rosemont, IL: American Academy
of Orthopaedic Surgeons; 2005:341-353.)
Postop Weeks Postop Months
1-2 3-4 5-6 7-8 9-12 4 5 6 7-12
TABLE 11-1. Rehabilitation after Meniscus Transplant

At the time of the lateral meniscus transplant, a concurrent osteochondral autograft transfer
of the lateral femoral condyle was done in 13 knees for full-thickness articular cartilage
defects. Knee ligament reconstructions were done before the meniscus allograft in four knees
and at the same time as the transplant in four knees. In six knees, ACL reconstructions were
done using either bone-patellar tendon-bone or semitendinosus-gracilis autografts. A PCL 2-
strand quadriceps tendon autograft reconstruction (13 ) was done in one knee. Both the PCL
and ACL were reconstructed in one knee.
Abnormal articular cartilage surfaces (lesion >15 mm in diameter with fissuring and
fragmentation extending greater than one half of the depth of the cartilage, or subchondral
bone exposed) were detected in the meniscectomized tibiofemoral compartment in 31 (88%)
knees at the time of meniscus transplantation. Subchondral bone exposure was found in 18
knees and extensive fissuring and fragmentation was noted in 13 other knees.
Twenty-nine (73%) meniscus allografts were analyzed with MRI using our research protocol
previously described in detail (15 ,21 ) an average of 35 months (range, 12 to 27 months)
postoperatively. The radiographs were reviewed and measured by independent orthopaedic
surgeons blinded to patient information. The height, width, and displacement of the
transplant were determined during full or partial weight-bearing (loaded) conditions.
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The subjective and functional results were assessed with the Cincinnati Knee Rating System. A
classification system of meniscus transplant characteristics was developed on the basis of MRI,
clinical examination, follow-up arthroscopy (when performed), and tibiofemoral symptoms
(Table 11-2 ). The International Knee Documentations Committee (IKDC) classification system
was used to determine knee ligament graft function.
Before surgery, 27 patients (77%) had moderate to severe pain with daily activities; but at
follow-up, only two (6%) had pain with daily activities (Fig. 11-12 , p <0.0001). All patients had
pain in the meniscectomized tibiofemoral compartment preoperatively; but at follow-up, only
10 (29%) had some component of tibiofemoral pain. Thirty-three of 35 (94%) patients stated
their knee condition had improved on self-assessment ratings (Fig. 11-13 ). The mean
preoperative patient perception score (scale, 1 to 10) of 3.1 points (range, 1 to 6 points)
improved to a mean of 6.2 points (range, 1 to 9 points) at follow-up (p <0.0001).
Preoperatively, only one patient was able to participate in sports without problems. At follow-
up, 27 patients (77%) were participating in light low-impact sports without problems, and one
patient was participating with symptoms against advice.
One patient had signs of a meniscus tear at follow-up. One patient had tibiofemoral joint line
pain and increased palpable crepitation compared with the preoperative examination. All
patients had a normal range of knee motion. All knees that had an ACL reconstruction had
normal or nearly normal anterior stability restored except one in which the reconstruction
failed. The PCL reconstructions restored nearly normal function in both knees at 20 and 90
degrees of flexion, except for one knee in which partial function was restored at 90 degrees of
flexion.
Five patients had follow-up arthroscopy for allograft-related symptoms. In three patients,
tears in the periphery of the meniscus allograft at the capsular junction were successfully
repaired. In two patients, small tears in the allograft were resected. None of these patients
had further complaints. One other patient had a total knee replacement 35 months following
the meniscus allograft for unresolved knee pain and a failed meniscus allograft. With the six
patients described previously, and the four meniscus transplants that required removal early
postoperatively, the reoperation rate for meniscal allograft symptoms was 25% (10 of 40
meniscus allografts).
The mean displacement of the 29 meniscus allografts examined with MRI was 2.2 Â± 1.5 mm
(range, 0 to 5 mm) in the coronal plane. Seventeen allografts (59%) had no displacement, 11
had minor displacement, and one could not be evaluated due to artifacts from other operative
procedures.
In the sagittal plane, the mean displacement of the posterior horn of the allografts was 1.1 Â±
2.0 mm (range, 0 to 9 mm). Twenty-five allografts (86%) had no displacement of the posterior
horn, three had minor displacement, and one had major displacement (9 mm). The mean
displacement of
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the anterior horn of the allografts was 1.2 Â± 1.7 mm (range, 0 to 6 mm). Twenty-five
allografts had no displacement of the anterior horn, three had minor displacement, and one
had major displacement (6 mm). Intrameniscal signal intensity was normal in one, grade 1 in
13, grade 2 in 11, grade 3 in three, and could not be evaluated in one.
Normal
Healed
Normal (0%-25% of meniscus width)
None, Grade 1,2
None
None
None
Altered
Displacement (26%-50% of meniscus width)
Mild pain, improved over preop
Partial meniscectomy (less than 1/3 removed)
Failed
Not Healed
Major displacement (>50% of meniscus width)
Grade 3 (tearing, signal intensity extended to articular surface)
Obvious signs meniscus tear
Definite return pain, not improved over preop
Partial or total meniscectomy (more than 1/3 removed)

* One abnormal result = failed.
â€ Coronal and sagittal planes, percent displacement posterior or medial plateau margin
perpendicular to the joint line.
N = number of meniscus allografts.
(Adpated from Noyes FR, Barber-Westin SD, Rankin M. Meniscal transplantation in symptomatic
patients less than fifty years old. J Bone Joint Surg 2004;86A:1392-1404.)
Follow-up
Clinical Exam: Arthroscopy
MRI Evaluation (N = 17) Tibiofemoral (N = 28) (N = 8)
Allograft Peripheral Position Signal Meniscus Pain, Meniscus

Classification Attachment in Intensity Signs Clinical Tears
Jointâ€ Symptoms
TABLE 11-2. Meniscus Allograft Classification*
FIGURE 11-12
The pain scale shows the highest level of activity possible without the patient experiencing
knee pain. The difference between preoperative and follow-up was statistically significant (p
<0.0001). Mod = moderate; Sev = severe. (Adapted from Noyes FR, Barber-Westin SD, Rankin,
M. Meniscus transplantation in symptomatic patients less than fifty years old. J Bone Joint
Surg Am. 2004;86:1392-1404, with permission.)
Seventeen (42.5%) of the meniscus transplants had normal characteristics (Fig. 11-14 , Table
11-2 ), 12 (30%) had altered characteristics, and 11 (27.5%) failed. Of the 20 lateral menisci
transplants, nine had normal characteristics, seven had altered characteristics, and four
failed. Of the 20 medial meniscus allografts, eight had normal characteristics, five had altered
characteristics, and seven failed. There was a correlation between the arthrosis rating on MRI
and the allograft characteristics (p = 0.01). Of the 16 allografts in knees with mild arthrosis, 10
had normal characteristics and six had altered characteristics. Of the 12 allografts in knees
with moderate arthrosis, three had normal characteristics, four had altered characteristics,
and five failed.
All patients had a normal range of knee motion. All seven knees that had an ACL
reconstruction had normal or nearly normal anterior stability restored, except one in which
the reconstruction failed. The PCL reconstructions restored nearly normal stability in the two
knees that had this operation at 20 and 90 degrees of flexion, except for one knee in which
partial stability was restored at 90 degrees of flexion. A comparison of preoperative and
follow-up weight-bearing posteroanterior radiographs revealed that three knees had further
deterioration and narrowing of the tibiofemoral joint space in the involved compartment.
We previously described the results of 96 consecutive irradiated meniscus allografts implanted

into 82 patients (14 ). Twenty-nine menisci in 28 patients were removed prior to the minimum
2-year follow-up; this left 67 meniscus allografts that were followed 22 to 58 months
postoperatively with MRI and clinical examination. The overall rating of all 96 meniscus
allografts was nine grafts (9%), normal; 19 grafts (20%), altered; 67 grafts (70%), failed; and
one, unknown. Of the 67 meniscal allografts that survived at least 2 years postoperatively, the
rating was nine grafts (13%), normal; 19 grafts (29%), altered; 38 grafts (57%), failed; and one,
unknown.
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FIGURE 11-13
Distribution of patient perception of the overall knee condition. The difference between
preoperative and follow-up was statistically significant (p <0.0001). (Adapted from Noyes FR,
Barber-Westin SD, Rankin M. Meniscus transplantation in symptomatic patients under fifty
years of age. J Bone Joint Surg Am. 2004;86: 392-1404, with permission.)
In this study, the meniscus transplant failure rate was 6% (1 of 18 knees) in knees with normal
or only mild arthrosis on MRI, 45% (14 of 31 knees) in knees with moderate arthrosis, and 80%
(12 of 15 knees) in knees with advanced arthrosis. The relationship between the meniscus
allograft failure rate and increasing severity of joint arthrosis was significant (p <0.001). The
role of low-dose irradiation
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(2.0-2.5 Mrads) in terms of increasing the failure rate in any soft tissue graft is not
scientifically known at this time.
FIGURE 11-14
Sagittal MRI obtained 42 months postoperatively demonstrates a meniscus transplant with

normal characteristics.
The biology of meniscus healing and remodeling after implantation determines the final
outcome regarding potential load-bearing function and chondroprotective effects. The
meniscus transplant heals to the peripheral allograft-host rim junction in our experience
within a short period of time, usually 4 to 6 weeks, providing initial stability of the transplant
in the knee joint. In animal studies (2 ) and our analysis of failed meniscus transplants (14 ),
there is an incomplete and disordered remodeling process including the minimal cellular
repopulation of the allograft, disorganized collagen tissue lacking the normal collagen
orientation required for load sharing, and a predominant fibrocyte cellular structure. Authors
have reported an increase in water content and decrease in proteoglycan concentration (9 ).
In our experience, MRI done immediately after meniscus transplantation demonstrates uniform
low signal intensity throughout the transplant tissue. Increased signal intensity at the capsular
attachments of the transplant to the host meniscus rim is believed to be due to progressive
healing. The low signal intensity within the body of the meniscus remains until remodeling of
the transplant occurs. It is possible to see retention of the native meniscus low intensity signal
for up to 2 to 3 years after implantation, indicating a delayed remodeling response. When
remodeling occurs, MRI demonstrates an increased signal intensity that represents an ingrowth
of cells into the transplant, removal of portions of the dense well-formed collagen framework,
and replacement with randomized and disorganized collagenous tissues. During this stage,
alterations in mechanical properties and decreased load-sharing capabilities of the transplant
occur. With further remodeling, grade 3 signal intensity commonly appears, indicating early
degeneration and tears in the transplant. We hypothesize that all meniscus allografts will
undergo this deleterious remodeling process at different time periods postimplantation,
resulting in altered mechanical properties and potential for tearing, fragmentation, and
degeneration under joint-loading conditions.
CONCLUSIONS
On the basis of our investigations, we believe that the results of meniscus transplantation are
more favorable when the operation is done before the onset of moderate tibiofemoral joint
arthrosis. Several studies have documented a correlation between the final rating of the
meniscus transplant and knee joint arthrosis, as success rate increases in knees with only mild
arthrosis. This procedure is acceptable for younger patients, especially those who have
symptoms with daily activities, as there are few if any other available treatment options. The
short-term results are encouraging, as the majority of patients demonstrate improvement in
knee function and pain in the affected compartment. However, no clinical study to date has
determined if meniscus transplantation provides a chondroprotective effect.
Abnormal limb alignment and ligament instability require simultaneous or staged corrections
to warrant meniscus transplantation. The long-term function of meniscus transplants remains
questionable. The transplant undergoes limited remodeling, which results in alterations in
meniscus collagen fiber micro-architecture, matrix, and cellular function required for load
sharing and long-term survival. Patients are advised that the procedure is not curative in the
long-term and additional surgery will probably be required. We believe the meniscus implant
will eventually fail, requiring replacement in younger patients.
There are several important research areas that may improve the success rates of meniscus
transplantation. These include issues related to transplant remodeling; collagen fiber
restoration of micro-architecture to resist tensile, compressive, and shear forces; implant
collagen matrix changes with altered material and structural properties; cellular repopulation
and function in maintaining implant homeostasis; role of fresh transplants with viable cells;
and the role of meniscus scaffolds and tissue engineering.
REFERENCES
1. Alhalki MM, Hull ML, Howell SM. Contact mechanics of the medial tibial plateau after
implantation of a medial meniscal allograft. A human cadaveric study. Am J Sports Med.
2000;28(3):370-376.
2. Arnoczky SP, DiCarlo EF, O'Brien SJ, et al. Cellular repopulation of deep-frozen meniscal
autografts: an experimental study in the dog. Arthroscopy. 1992;8(4):428-436.
P.129
3. Barber-Westin SD, Noyes FR, McCloskey JW. Rigorous statistical reliability, validity, and
responsiveness testing of the Cincinnati knee rating system in 350 subjects with uninjured,
injured, or anterior cruciate ligament-reconstructed knees. Am J Sports Med.
1999;27(4):402-416.
4. Barbour SA, King W. The safe and effective use of allograft tissue-an update. Am J
Sports Med. 2003;31(5):791-797.
5. Berlet GC, Fowler PJ. The anterior horn of the medial meniscus. An anatomic study of
its insertion. Am J Sports Med. 1998;26(4):540-543.
6. Cameron JC, Saha S. Meniscal allograft transplantation for unicompartmental arthritis

of the knee. Clin Orthop Relat Res. 1997;337:164-171.
7. Cole BJ, Carter TR, Rodeo SA. Allograft meniscal transplantation. Background,
techniques, and results. J Bone Joint Surg Am. 2002;84-A(7):1236-1250.
8. Farr J, Meneghini RM, Cole BJ. Allograft interference screw fixation in meniscus
transplantation. Arthroscopy. 2004;20(3):322-327.
9. Jackson DW, Simon TM. Biology of meniscal allograft. In: Mow VC, Arnoczky SP, Jackson
DW, eds. Knee meniscus: basic and clinical foundations. New York: Raven; 1992:141-152.
10. McLaughlin JR, Noyes FR. Arthroscopic meniscus repair: recommended surgical
techniques for complex meniscal tears. Techniques in Orthopaedics. 1993;8(2):129-136.
11. Noyes FR, Barber-Westin SD. A comparison of results in acute and chronic anterior
cruciate ligament ruptures of arthroscopically assisted autogenous patellar tendon
reconstruction. Am J Sports Med. 1997;25(4):460-471.
12. Noyes FR, Barber-Westin SD. Meniscus transplantation: indications, techniques, clinical
outcomes. Instr Course Lect. 2005;54:341-353.
13. Noyes FR, Barber-Westin SD. Posterior cruciate ligament replacement with a two-
strand quadriceps tendon-patellar bone autograft and a tibial inlay technique. J Bone
Joint Surg Am. 2005;87(6):1241-1252.
14. Noyes FR, Barber-Westin SD, Butler DL, et al. The role of allografts in repair and
reconstruction of knee joint ligaments and menisci. Instr Course Lect. 1998;47:379-396.
15. Noyes FR, Barber-Westin SD, Rankin M. Meniscal transplantation in symptomatic

patients less than fifty years old. J Bone Joint Surg Am. 2004;86-A(7):1392-1404.
16. Noyes FR, Berrios-Torres S, Barber-Westin SD, et al. Prevention of permanent

arthrofibrosis after anterior cruciate ligament reconstruction alone or combined with
associated procedures: a prospective study in 443 knees. Knee Surg Sports Traumatol
Arthrosc. 2000;8(4):196-206.
17. Noyes FR, Mooar PA, Matthews DS, et al. The symptomatic anterior cruciate-deficient
knee. Part I: the long-term functional disability in athletically active individuals. J Bone
Joint Surg Am. 1983;65(2):154-162.
18. Pollard ME, Kang Q, Berg EE. Radiographic sizing for meniscal transplantation.
Arthroscopy. 1995;11(6):684-687.
19. Potter HG, Foo LF. Magnetic resonance imaging of articular cartilage: trauma,
degeneration, and repair. Am J Sports Med. 2006;34(4):661-677.
20. Potter HG, Linklater JM, Allen AA, et al. Magnetic resonance imaging of articular
cartilage in the knee. An evaluation with use of fast-spin-echo imaging. J Bone Joint Surg
Am. 1998;80(9):1276-1284.
21. Rankin M, Noyes FR, Barber-Westin SD, et al. Human meniscus allografts' in vivo size
and motion characteristics: magnetic resonance imaging assessment under weightbearing
conditions. Am J Sports Med. 2006;34(1):98-107.
22. Rosenberg TD, Paulos LE, Parker RD, et al. The forty-five-degree posteroanterior
flexion weight-bearing radiograph of the knee. J Bone Joint Surg. 1988;70A:1479-1483.
23. Rubman MH, Noyes FR, Barber-Westin SD. Arthroscopic repair of meniscal tears that
extend into the avascular zone. A review of 198 single and complex tears. Am J Sports
Med. 1998;26(1):87-95.
24. Rubman MH, Noyes FR, Barber-Westin SD. Technical considerations in the management
of complex meniscus tears. Clin Sports Med. 1996;15(3):511-530.
25. Stone KR, Walgenbach AW, Turek TJ, et al. Meniscus allograft survival in patients with
moderate to severe unicompartmental arthritis: a 2- to 7-year follow-up. Arthroscopy.
2006;22(5):469-478.
26. Vangsness CT Jr, Garcia IA, Mills CR, et al. Allograft transplantation in the knee: tissue
regulation, procurement, processing, and sterilization. Am J Sports Med. 2003;31(3):474-
481.
27. Verdonk PC, Demurie A, Almqvist KF, et al. Transplantation of viable meniscal
allograft. Survivorship analysis and clinical outcome of one hundred cases. J Bone Joint
Surg Am. 2005;87-A(4):715-724.
> Table of Contents > Part Three - Ligament Injuries and Instability > 12 - Anterior Cruciate
Ligament Reconstruction
12
Anterior Cruciate Ligament Reconstruction
Douglas W. Jackson
Peter R. Kurzweil
INDICATIONS
The treatment for a patient with an acutely torn anterior cruciate ligament (ACL) is
individualized by factors such as age, occupation, level of sports participation, and
associated intra-articular and ligament injuries. An important consideration in the
discussion with the patient is the patient's willingness to modify high-risk activities in
the future. We usually recommend anterior ligament reconstruction in the more
physically active individuals of all ages who want to pursue activities that involve
lateral movement, sudden stopping, and change of direction. We also suggest
reconstruction of the ruptured ACL when there are significant associated meniscus
tear(s) and/or a concomitant ligament injury. The decision to perform reconstructive
surgery in someone with symptomatic knee instability due to chronic ACL deficiency is
less controversial. Patients with an anterior cruciate deficient knee have had a chance
to experience the limitations it imposes on their lifestyle. Usually the reason they have
made the appointment with the surgeon is because they no longer want to live with
the limitations if something can be done to correct the knee.
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CONTRAINDICATIONS
The contraindications to ACL reconstruction have diminished over the past decade.
The procedure is now done on an outpatient basis, with an increasing number of
surgeons doing it technically well. The time taken from everyday activities for
recovery following an acute ACL rupture can take a few weeks up to 2 months. The
recovery time from the surgery to return to light work and/or school is about the
same. There are several considerations that should be discussed with the patient who
is considering a reconstruction:
Not every ACL rupture needs to be reconstructed. Many individuals live with an
ACL-deficient knee quite successfully.
There is no urgency to reconstruct an ACL immediately after the injury. The

patient can recover from the acute phase and determine if he or she wants to
live with the deficient knee or have it reconstructed at a later date.
The results from delayed reconstructions (>6 weeks or even months and years
later) appear to be almost equivalent to those reconstructions done shortly
after the acute injury. This is true as long as the patient does not sustain
repeated subluxations with effusions, additional meniscal tears, additional
bone contusions, and associated articular cartilage damage.
PREOPERATIVE PREPARATION
The first step in assessing the pathology of the injured knee with an ACL rupture is to
perform a thorough history and physical examination. The history is often diagnostic in
most acute ACL ruptures. The patient often describes hearing or feeling a pop within
the knee which was associated with a cutting motion or sudden change in direction.
The individual may report that the injury occurred as the result of a noncontact or
contact injury. In many instances, the patient was unable to continue participating in
that particular activity. The injured knee usually develops an effusion within a few
hours following the injury.
Imaging of the knee includes routine radiographs to rule out fractures, evaluate pre-
existing degenerative changes, and assess the overall alignment of the lower
extremity. A magnetic resonance imaging (MRI) scan is not always necessary, but is an
excellent independent method to confirm a torn ACL. In addition, it is helpful in
documenting meniscal tears, the extent of bone contusions (Fig. 12-1), and other
ligament injuries. However, other than bone contusions, the MRI may not offer much
more information than an experienced clinician will find with a thorough preoperative
evaluation, examination under anesthesia, and arthroscopic assessment at the time of
reconstruction. If a decision is made to treat the knee operatively, the associated
intra-articular pathology can be evaluated, documented, and treated during the
surgical procedure.
When the patient with an acute injury arrives for evaluation, the knee is usually
swollen (hemarthrosis) with restricted range of motion. If surgery is recommended, we
prefer waiting for the acute inflammatory phase, with its associated pain and swelling,
to subside. Waiting for the motion to improve has been associated with less difficulty
re-establishing the range of motion of the knee after surgery. Patients typically take 2
or 3 weeks to re-establish their knee motion following an ACL tear. A displaced bucket
handle tear causing a locked knee may be a consideration for shortening the waiting
period for surgical reconstruction.
The planned incisions and graft options are discussed with each patient. During the
1990s, we prefered to use a patellar tendon autograft almost exclusively; and although
it is still one of our primary choices, hamstrings and allografts are now used more
commonly. This is particularly true if the patient has a pre-existing patellofemoral
disorder, a history of anterior knee pain, or is particularly concerned about the
cosmetics of an incision. We prefer to avoid incisions around the patellar tendon in
workers who kneel and crawl as part of their occupation. With improved soft tissue
graft aperture fixation and reported results, our use of hamstring grafts has increased
significantly. We have used allografts in ACL reconstructions in a larger number of
cases over a 20-year period and prefer them in certain revisions, older and less active
patients, and those patients in whom the knee requires multiligament reconstruction.
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FIGURE 12-1
If the patient has had an MRI before surgery, we have the important films on the
view box at the time of surgery. The MRI following an acute ACL rupture confirms
the disrupted ACL, and quite accurately demonstrates associated torn menisci.
The typical anterolateral femoral and posterolateral tibial kissing bone contusions
are demonstrated in this figure.
SURGERY
The patient is placed in the supine position with both legs resting straight on the
operating table. Our patients and anesthesiologists usually elect general anesthesia,
although some choose an epidural block. A tourniquet is placed on the proximal thigh
but is infrequently inflated during the procedure. A lateral thigh post clamped to the
table allows a valgus stress to be applied to the knee, enhancing visualization and
access to the medial meniscus.
Most of our patients receive a single-injection femoral nerve block. We believe this has
signficantly reduced the level of pain and need for narcotic analgesics in the
immediate postoperative period. Most patients who have the block go home with very
little discomfort. The femoral nerve block does shut down the quadriceps muscle
temporarily, so all weight bearing on the operated knee is delayed for at least 1 day.
Prophylactic antiobioticsâ€”typically 1 g of a first-generation cephalosporin
(Cefazolin)â€”are given 30 to 60 minutes prior to surgey.
After adequate anesthesia, we perform a ligament examination and document the

preoperative arthrometric evaluation of both knees with the KT-1000. We look at the
manual maximum displacement numbers and do not use the test at 20 and 30 pounds
of force. A KT-1000 evaluation is also performed immediately following surgery, while
the patient is still under anesthesia. These values are used to help confirm that the
graft tensioning and fixation are satisfactory. The vast majority (98%) of the knees we
reconstruct have a preoperative side-to-side difference of 5 mm or more under
anesthesia and a positive pivot shift. If it is obvious that the ACL is nonfunctional, the
autograft is routinely harvested before the arthroscopic portion of the procedure.
When an allograft is used, it is not opened until we are certain that we will proceed
with the ACL reconstruction. The graft is then prepared at a side table during the
arthroscopy. However, when there is an equivocal pivot shift or a side-to-side
difference of less than 5 mm with the KT-1000, the knee is first evaluated
arthroscopically.
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We make a concerted effort not to inflate a tourniquet during the procedure. Three
techniques help control hemostasis and maintain our visual field. First, the pressure of
the irrigation solution from gravity is maintained by the height of the irrigation bags.
We have chosen not to use a pressurized inflow system (a pump) and prefer gravity
irrigation. Second, the joint is injected with 25 mL of 0.50% Marcaine with 1:200,000
epinephrine immediately after draping the knee. Third, we selectively use intra-
articular hemostasis with an insulated electrocuatery tip to control localized bleeding.
If the patellar tendon is being used as the graft, the skin over the patellar tendon is
injected with 10 mL of the same anesthetic solution before harvesting the autograft.
We believe this also helps reduce postoperative pain.
The surgery is performed in a stepwise fashion as follows: (a) evaluation under

anesthesia, (b) graft harvesting and preparation, (c) notch preparation, (d) positioning
and preparing the osseous tunnels, (e) graft implantation and fixation, (f) closure and
postoperative assessment of stability, and (g) postoperative management and
rehabilitation.
Technique
While under direct observation of the nurses in the preoperative area before sedation
is given, the patient writes a â€œyesâ€• on the ACL deficient knee. Once in the
operating room, a â€œtime outâ€• is called to confirm the correct patient, procedure,
and site. This â€œtime outâ€• requires the agreement of the circulator, scrub
assistant, surgeon, and anesthesiologist. Our method of harvesting the patellar tendon
is described later in this chapter when we elect to use it as our graft. Chapter 15
demonstrates another technique for patellar tendon bone-bone harvesting. When a
patella tendon autograft is chosen, we prefer a vertical incision along the medial
border of the patellar tendon. The incision starts at the level of the inferior pole of
the patella and extends to the level of the tibial tubercle (Fig. 12-2). The peritenon is
carefully dissected away from the tendon. We do not harvest more than one third of
the total width of the tendon. If the width is at least 30 mm, a double-bladed scalpel
(Fig. 12-3) with the blades set 10 mm apart is used to cut a central strip of tendon. In
smaller individuals, a 9-mm strip of tendon is harvested.
FIGURE 12-2
The skin incision to harvest the patella tendon autograft is vertical as depicted in
this right knee. It extends from the inferior pole of the patella to a point
approximately 1 cm medial to the tibial tubercle. Through this incision the
anteromedial arthroscopy portal is placed as is the accessory anteromedial portal
for the passage of the femoral interference screw. This incision allows the
medially located tibial tunnel to be drilled with less skin retraction.
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FIGURE 12-3
A double-handle scalpel (either 9 or 10 mm between the blades) ensures a

parallel cut when harvesting the central third of the patellar tendon.
The tendon's bony attachments are outlined with electrocautery (Fig. 12-4A). The
dimension of the patellar bone plug is typically 10 Ã— 25 mm. The bone plug from the
tibial side usually measures 10 Ã— 25 mm. We use circular oscillating saws (Stryker,
Kalamazoo, MI) with diameters of 9, 10, or 11 mm to harvest the bone plugs (Fig. 12-
4B). Leaving a smooth, semicylindrical defect in the patella reduces stress risers and
the potential for patella fracture postoperatively. This technique also allows for
simple bone grafting of the created defects by using the cylindrical core of bone
obtained from creating the the tibial tunnel. When split in half, the semicylindrical
pieces of bone sit perfectly in the defects in the patella and tibial.
FIGURE 12-4
A: The tendon's bony attachments are outlined with electrocautery. B: The

circular oscillating (Stryker, Kalamazoo, Ml) saw used to harvest the bone plugs is
smooth and recessed on top to prevent soft-tissue laceration when harvesting the
bone plug. C: By pulling the mobile extensor mechanism distally and retracting
the skin proximally, the incision can be shortened by 2 or 3 cm. The saw cuts
better when the bone is kept moist, which also helps minimize thermal necrosis.
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FIGURE 12-5
The tibial bone plug was harvested with a 10-mm circular saw and slides through
an 11-mm spacer with minimal trimming. The patellar end of the graft was taken
with a 10-mm harvester and easily slides through a 10-mm spacer. The spacers
represent the diameter of the bone tunnels to be drilled.
The tibial plug is harvested first, typically with a 10-mm circular saw blade. Once the
tibial bone plug is harvested, a towel clip is attached and used to pull the extensor
mechanism distally, drawing the patella into the wound. Retracting the loose tissue
over the patella proximally permits a shorter skin incision to be used. The patellar
bone plug is then harvested with the same blade (Fig. 12-4C). To avoid burning the
bone, saline drops are added to help dissipate the heat generated by the oscillating
harvesting blade.
The graft is taken to a side table and prepared for implantation by the assistant
surgeon. With minimal trimming, each bone plug should easily slide through a spacer
matching the diameter of the osseous tunnel through which it will eventually pass (Fig.
12-5). The tibial plug, intended for the femoral side, is trimmed to slide through a 9-
mm tunnel spacer (Fig. 12-6). The patellar plug, which will be fixed in an 10-mm
diameter tibial tunnel, usually requires only minimal trimming. Three drill holes (using
a 3/32-inch bit) are made in the patella bone plug and threaded with No. 5 Tevdek or
Ticron sutures. The holes are oriented at right angles to each other to minimize the
chance of suture laceration by the interference screw, and hence loosing tension on
the graft during tibial screw insertion. A single drill hole is made in the tibial bone
plug, which is threaded with a 30-inch No. 2 nylon suture. The graft is mounted on a
tension board (8-lb load), which also facilitates final soft-tissue trimming. The bone-
tendon junction is marked with a sterile pen. A line is drawn longitudinally
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along the center of the femoral plug to help orient its rotation as it is pulled into the
femoral tunnel (Fig. 12-7). Once preparation of the graft is complete, it is left under
tension and covered with an antibiotic-soaked sponge (Fig. 12-8). Using an assistant to
prepare the graft while the surgeon works on the knee can save 15 to 30 minutes per
case and can keep the surgical time for an isolated ACL reconstruction to less than 1
hour.
FIGURE 12-6
A: One surgeon shapes, sizes, and prepares the graft at a side table for passage
through the knee and into the desired positions in the bone tunnels. Two surgeons
working simultaneously shorten the operative time. B: A typical harvested graft
before shaping, sizing, and preparation for passage. Note the different collagen
insertions into the tibia and patella bone plugs. These different orientations to
the collagen insertions can be used to position the collagen in the desired portion
of the osseous tunnels.
Regardless of graft choice, the intra-articular portion of the procedure begins by

introducing the arthroscope into the anterolateral portal. When a patellar tendon
autograft is harvested, the anteromedial portal is made by retracting the incision to
expose the medial border of the patellar tendon (Fig. 12-9). A thorough diagnostic
arthroscopy is performed through a separate anterolateral portal to confirm the torn
ACL (Fig. 12-10) and determine the status of the menisci and articular cartilage. We
prefer to debride the soft tissue from the lateral wall of the notch with a full-radius
resector. Our goal is to visualize the â€œover-the-topâ€• position. The center of the
femoral tunnel is selected after preparing the notch. An endoscopic â€œover-the-
topâ€• guide is used to mark a point that will leave a thin shell of cortical bone
posteriorly after the femoral tunnel is drilled. Guides are available for creating tunnel
diameters from 6 to 11 mm (Fig. 12-11). When a 10-mm tunnel is planned with the
typical patellar tendon autograft, we use a guide (Stryker, Kalamazoo, MI) that is
designed to mark a point 6 to 7.0 mm anterior to the over-the-top position (Fig. 12-
12). A 10-mm tunnel (5-mm radius) with 7-mm placement will leave a 2.0-mm
posterior rim. The guide is inserted through an accessory anteromedial portal made
just over the joint line and along the medial border of the patellar tendon. The
desired site in the notch is drilled through the cannulated guide. The location of the
femoral tunnel has changed slightly to avoid a more vertical graft. We prefer placing
the center of the femoral tunnel at the 10:00 AM or 2:00 PM site. (See Chapter 14 for
more in-depth discussion on tunnel placement.)
FIGURE 12-7
A: Three drill holes (using a 3/32-inch bit) are made in the harvested patella bone
plug and threaded with No. 5 Tevdek sutures. The holes are oriented at right
angles to each other to minimize the chance of suture laceration by the
interference screw. We pass the sutures through the bone plugs with a Hewson
suture passer (B). There are no needles on the back table. This eliminates the
possibility of a needle puncture wound to the surgeon preparing the graft. A
single drill hole is made in the harvested tibial plug, which is threaded with a 30-
inch No. 2 nylon suture. The graft is mounted on a tension board (8-lb load),
which also facilitates final soft-tissue trimming (C). The bone-tendon junction is
marked with a sterile pen. A line is drawn longitudinally along the center of the
femoral plug to help orient its rotation as it is pulled into the femoral tunnel. The
prepared graft is mounted on a tensioning board, which facilitates final trimming.
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FIGURE 12-8
The larger bone plug has three No. 5 Tevdek sutures oriented at right angles,
while the smaller bone plug has a single long (30 inches) suture passed through
the cortical surface. The bone-tendon junction is marked with a sterile pen for
orientation during passage.
FIGURE 12-9
Because of the medially placed skin incision, a separate stab wound is used for
the anterolateral arthroscopic portal. We place it close to the lateral border of
the patellar tendon and approximately 1.5 cm above the tibial plateau as
depicted in this figure. The extra-articular â€œstarting pointâ€• determines the
length and obliquity of the tibial tunnel. To facilitate placing the femoral tunnel
toward the 10 o'clock postion in a right knee, the guide used for drilling is
positioned at a point about 2 cm medial to the tibial tubercle. The distance of
the starting position distal to the joint line determines the angle of the graft in
the saggital plane. Starting too distal may create a tibial tunnel that is too steep.
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FIGURE 12-10
An acutely torn ACL. It has been disrupted near its femoral insertion.
Attention is turned to preparation of the tibial tunnel. The length of the tunnel varies
with the graft length. Although the intra-articular opening for the osseous tunnel is
fixed, the position of the extra-articular opening of the tunnel can be varied. The
preferred length of the tibial tunnel is calculated as explained in Figures 12-13 and 12-
14. The total graft length (in millimeters) minus 50 mm estimates
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the desired tibial tunnel length. This will prevent the tunnel from being too short,
avoiding graft-tunnel mismatch. The tibial tunnel needs to be started medially to
allow access to the lower femoral tunnel site. (See Chapter 14 for more detail on
tunnel site selection.)
FIGURE 12-11
Over-the-top endoscopic guides for marking the site of the center of the femoral
tunnel. Each is designed to leave a 2-mm rim of posterior cortical bone when
drilling a 9-, 10-, or 11-mm femoral tunnel.
FIGURE 12-12
Intra-articular view of the femoral guide. The â€œprongâ€• has hooked over the
top. The guides are cannulated so that a drill will mark the desired location, in
this case 6.5 mm anterior to the over-the-top position.
FIGURE 12-13
The calculation of tibial tunnel length. A typical graft has bone plugs of 20 and 25
mm, and is 100 mm long. Assuming a femoral tunnel of 25 mm and an intra-
articular distance of 30 mm (the average length of an ACL), the tibial tunnel must
be at least 45 mm long to accommodate the graft. Another 5 mm should be added
to the calculated length, since drilling a 10- or 11-mm-diameter tibial tunnel
effectively shortens the tunnel length by its radius.
The intra-articular point for the tunnel opening is referenced from the posterior
cruciate ligament (PCL), lateral meniscus, and tibial spines. The arc made by the
posterior rim of the anterior horn of the lateral meniscus is followed to the medial
tibial spine. This is about 7 mm anterior to the PCL (Figs. 12-15 and 12-16). The tip of
the tibial drill guide is inserted through the anteromedial portal and placed on this
spot. The angle of this calibrated guide is adjusted until the desired tunnel length is
achieved (Fig. 12-17). The angle of the tunnel should place the graft in appropriate
relationship to the slope of the intercondylar notch. The guide pin is then inserted
(Fig. 12-18), with attention given to prevent it from â€œwalking upâ€• the tibia and
shortening the tibial tunnel.
When a patella tendon bone autograft is harvested, we use a cannulated core drill,
which removes a cylinder of bone, to create the tibial tunnel. This allows a clean
removal of the tibial ACL stump (Fig. 12-19) and the acquisition of a solid piece of
bone for grafting into the defects in the patella and tibial tubercle, as described
previously (Fig. 12-20).
FIGURE 12-14
The probe is seated in the debrided stump of the ACL. This position is parallel
with the posterior rim of the anterior horn of the lateral meniscus and 7 mm
anterior to the PCL. It is at the level of the midportion of the medial tibial spine.
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FIGURE 12-15
The length of the tibial tunnel is calculated by subtracting the total graft length
from the sum of the length of the femoral tunnel (25 mm) and the intra-articular
distance between the tunnel openings (30 mm). Another 5 mm is added to the
difference, as described in Figure 12-13.
FIGURE 12-16
Magnetic resonance image of the tibial plateau taken after reconstruction. This
demonstrates the posterior location of the tibial tunnel (thick arrowhead) and its
relationship to the lateral meniscus (thin arrows).
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FIGURE 12-17
Drilling the tibial tunnel. The tibial guide is set to drill a tunnel of a
predetermined length, in this case 45 mm. The length should generally not
exceed 55 mm, or the obliquity of the tibial tunnel will be too steep. Note the
starting position of the tibial tunnel is halfway between the tubercle and medial
border of the tibia.
FIGURE 12-18
The guide pin has emerged in the joint in the desired location (as described in
Fig. 12-16). The lateral meniscus can be seen in the lower-right-hand corner.
FIGURE 12-19
Removing a core of bone, rather than simply drilling the tibial tunnel, leaves an
intra-articular opening with smooth margins. The articular cartilage and ligament
stump have been cleanly removed rather than pushed aside.
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FIGURE 12-20
A core of bone is obtained when making the tibial tunnel. It is split in half and
trimmed to fit the patellar defect at the time of closure.
The femoral tunnel is drilled through the tibial tunnel. The guide pin is retrograded
through the tibial tunnel to the previously selected point in the notch. We recommend
that this be done with at least 70 degrees of knee flexion, which reduces the chances
of blowing out the posterior wall of the femoral tunnel and potential for vascular
and/or nerve injury. Once the pin is inserted, the position of the knee should not vary,
or the pin may bend or break during drilling. Sometimes the guide pin retrograded
through the tibial tunnel cannot â€œhitâ€• the previously marked center of the
femoral tunnel. In these occasions, we will drill the tunnel through the anteriormedial
poprtal in 120Â° of flexion.
A cannulated, acorn-shaped drill bit (usually 10-mm diameter) is manually passed over
the guide pin and through the tibial tunnel. Care is taken in passing the tip past the
PCL. When the bit touches the femoral wall, it is drilled to a depth of 25 to 30 mm
(Fig. 12-21). The calibrated measurement at the distal opening of the tibial tunnel
should be the same or slightly greater than the total graft length (Fig. 12-22), or the
graft will protrude. The tibial tunnel is plugged and a suction device is passed into the
notch area and femoral tunnel to remove extraneous morsels of bone from the joint.
The integrity of the posterior cortical rim of the tunnel is arthroscopically confirmed
(Fig. 12-23).
A small notch is made at the aperture of the femoral tunnel at 2 o'clock (in a left
knee) into which the interference screw guidewire will later be placed. This helps
prevent the screw from rotating around the plug as it is inserted in the tunnel. The
knee is now ready for graft implantation. A Beath pin with an eyelet proximally is
retrograded through the tibial and femoral tunnels and out the anterolateral thigh,
with the knee flexed approximately 90 degrees. The graft is removed from the tension
board and the nylon suture is threaded through the eyelet. The pin is grasped and
pulled out
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proximally, bringing with it the suture, which is then used to pull the graft into the
joint. The bone plug is oriented so that the tendon is located posterior. The pen
markings on the plug are helpful in achieving proper rotation. A clamp inserted
through the anteromedial portal is often helpful in controlling the orientation of the
plug as it is seated into the femoral tunnel (Fig. 12-24).
FIGURE 12-21
A calibrated drill allows the depth of the femoral tunnel to be visualized. We

prefer 25 to 30 mm in length for the femoral osseous tunnel. These tunnel lengths
are modified if the length of the graft bone plugs is shorter than usual.
FIGURE 12-22
The calibrated drill used for making the femoral tunnel also provides the overall
end-to-end length between the tunnels. The measurement at the distal opening
of the tibial tunnel should match (or be slightly greater than) the length of the
graft.
The bone plug in the femoral tunnel is fixed first. A guidewire for the cannulated
interference screw is inserted through the accessory medial portal. It is placed into
the notch of the femoral tunnel so that it is between the cancellous surface of the
bone plug and the wall of the tunnel. Ideally, it is parallel to the longitudinal pen
marking on the plug (Fig. 12-25). With the knee flexed 80 to 90 degrees, we prefer to
use a 7-mm tap in the site for the interference screw. We then place a cannulated 8
Ã— 23-mm interference screw over the wire (Fig. 12-26). It is advanced, giving secure
interference fixation. We have been using bioabsorbable screws routinely since 1998.
The screw is inserted flush with the marked bone-tendon junction, which is also flush
with the aperture of the femoral tunnel (Fig. 12-27).
FIGURE 12-23
After drilling the femoral tunnel, the integrity of the posterior cortical rim should
be assessed. If the drill has blown out the back wall, a second incision over the
distal femur can be used to drill another tunnel from outside into the other
tunnel, and interference fixation can still be used.
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FIGURE 12-24 A-C: The opening to the femoral tunnel is notched for placement
of the guide pin for the interference screw. As the graft is pulled into the joint,
the bone plug rotation is controlled and oriented in relation to the notch and
markings on the bone plug. The marked graft aids the surgeon in obtaining the
desired rotation depth for seating the graft.
FIGURE 12-25
The guidewire for the cannulated femoral interference screw should be directed
between the cancellous surface of the bone plug and the tunnel wall. We then
use a cannulated pretap 7 mm in diameter passed over the guide pin. The tap is
removed and an 8-mm-diameter bioabsorbable screw is passed over the guide pin.
FIGURE 12-26
The screw is inserted into the joint over the guidewire.
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The femoral fixation of the graft is tested by firmly pulling on the Tevdek sutures in
the tibial bone plug. The surgeon should be able to pull hard enough to gently rock the
patient without the fixation failing. It is important to keep the knee in flexion when
assessing the fixation so that the pull is directly parallel to the femoral tunnel and
screw. The knee is cycled several times while palpating for movement of the bone plug
in the tibial tunnel. Lack of movement does not necessarily mean that the graft is
perfectly isometric. Rather, it may indicate that the tibial bone plug is catching in the
tunnel, in which case pulling on the sutures may not allow the graft to be properly
tensioned. More than a few millimeters of bone-plug excursion is also undesirable and
may indicate improperly placed tunnels. As the knee is ranged, the position at which
the tibial plug is most distal in the tunnel is noted. This is usually close to full
extension. The graft is tensioned in this position while pulling on the Tevdek sutures. A
9 or 10 Ã— 28-mm bioabsorbable interference screw is then inserted for tibial fixation.
A posteriorly applied force on the tibia helps ensure joint reduction during screw
fixation. After the graft has been fixed, the knee is placed through a full range of
motion. If a audible loud click or pop is heard at this time, it may indicate failure of
fixation (usually at the femur). The pivot shift and Lachman test are performed, and if
they are unacceptable, the graft is retensioned. We find ourselves retensioning less
than 5% of reconstructions each year. The patient should not leave the operating room
until we have achieved the desired stability.
The core of bone from the tibial tunnel is split in half and trimmed to fit the bony
defect in the patella and tibial tubercle. The overlying tissue is sutured to hold the
graft in place. The patellar tendon is loosely reapproximated to prevent any palpable
gaps without shortening the patella tendon. The peritenon, subcutaneous tissue, and
skin are closed in separate layers. Steri-strips are applied. The joint is injected with 25
mL of 0.50% Marcaine with 1:200,000 epinephrine and 5 mg of Duramorph.
After the wound is closed, sterile gauze and steri-drape are used to temporarily cover
the wound to allow evaluation with KT-1000 (Fig. 12-28A). A postoperative side-to-side
comparison is made in each patient (Fig. 12-28B). If the difference in the
measurements is equal to or less than 2 mm in anterior translation, this is acceptable.
A fresh sterile dressing is placed over the incision, and a TEDS stocking and knee
immobilizer are applied (Fig. 12-29A-C). The patient is then taken to the recovery
room. The reconstruction is performed as an outpatient procedure and patients are
discharged home with oral pain medication.
FIGURE 12-27
Advancement of the screw should be visualized to ensure the screw does not
rotate around the graft as it is placed in its interference position. The screw is
inserted to a depth that makes it flush with the end of the bone plug, which was
highlighted with a marking pen. The location of the screw superolaterally pushes
the collagen posteriorly and toward the PCL. It also minimizes contact with the
collagen to prevent tendon abrasion.
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FIGURE 12-28
A,B: Manual maximum displacement KT-1000 readings are taken to confirm
fixation. A temporary dressing is applied to maintain wound sterility. The
uninvolved knee is also retested at the conclusion of the case. If there is more
than 2 mm of side-to-side laxity in the reconstructed knee, the graft is
retensioned. The ideal is to have symmetry side to side.
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FIGURE 12-29
A-D: The sterile dressing is held in place with a thigh-high TEDS stocking rather
than tape. Our nurses like to use a plastic bag over the foot to help slide on the
stocking.
Pearls and Pitfalls

Graft Protrusion
Excessive tibial plug protrusion from the tibial tunnel precludes interference fixation.
Graft tunnel mismatch (as described previously) is avoided by not drilling a tibial
tunnel that is too short (see Figs. 12-13 and 12-14). Protrusions of a few millimeters
can be handled with minor adjustments. The simplest is to shorten the protruding end
of the bone plugs by 3 to 5 mm with a bone rongeur. If this is not enough, the femoral
tunnel can be deepened by 5 mm. These two measures should compensate for 10 mm
of graft protrusion. If necessary, distal fixation can be accomplished by tying the
sutures in the tibial plug around a screw (post) and washer.
Problems with the Iibial Interference Screw

Two problems with the tibial interference screw may result in graft laxity. First, the
screw can push the tibial plug into the tunnel when inserted, resulting in loss of
tension on the graft. This is best avoided by maintaining a firm pull on the Tevdek
sutures and watching the position of the plug as the screw is inserted. Second, the soft
medullary bone and space of the tibia may result in inadequate interference fixation.
A feeling for adequate interference fixation can be ascertained by noting the
resistance when turning the screwdriver and by carrying out a full range of motion,
Lachman, and pivot shift testing once fixation is felt to be adequate. If back-up tibial
fixation is desired, we tie the traction sutures through small tunnels in the adjacent
bony defect where the tibial bone was harvested.
Problems with the Interference Screw Guidewire

If the guidewire for the femoral or tibial screw is inserted too deeply, it can be
difficult to extract after screw insertion. If the guide pin bends distal to the screw,
there is the potential that it may break off. This should be suspected if the guidewire
requires much force to extract. Be certain to examine the tip when the pin is
extracted to be certain some has not broken off and remains in the osseous tunnel or
knee. The chance of this problem occurring can be reduced by placing the guidewire
directly between the bone plug and osseous tunnel wall and not advancing it too far
beyond the place where the tip of the cannulated interference screw will be.
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Difficulties in Harvesting the Graft
In attempting to harvest the patellar tendon graft, the circular saw may advance too
deeply and lacerate the tendon. If this is recognized, it can be corrected by gently
redirecting the saw. However, sudden changes in direction can fracture the plug.
Switching to a larger-diameter saw blade or using an osteotome to complete the
harvest may be helpful if this problem is recognized.
Blowout of the Posterior Wall of the Femoral Tunnel

It is important to check for blowout of the posterior wall of the femoral tunnel, since
it would preclude endoscopic femoral interference fixation. If blowout does occur, the
procedure could be salvaged by using an Endobutton or converting to the two-incision
technique. Of course, prevention is the best way of avoiding this situation. The risk of
blowout can be minimized by flexing the knee 70 degrees or more while drilling the
femoral tunnel.
It is important in preparing the femoral tunnel that the knee be flexed at 70 degrees
or greater. If the knee is in more extension during the drilling of the femora tunnel
through the tibia tunnel and the drill goes out the back of the posterior cortex, there
is the potential for nerve and/or vascular injury.
We routinely perform ACL reconstructions on an outpatient basis. The femoral nerve
block has helped with pain control. Patients are sent home with Norco 7.5 mg as an
oral analgesic. They are allowed to shower the day after surgery with the wound
covered. Treatment with ice is continued intermittently for 3 or 4 days. A knee
immobilizer is worn for approximately 2 to 4 weeks during ambulation and sleeping. It
is removed for therapy and when the patient is sitting. Weight bearing in the knee
immobilizer is allowed as tolerated and dictated by swelling. Crutches are
recommended outside the home for 2 to 4 weeks or until adequate muscular control of
the lower extremity is regained. Patients are told to emphasize knee extension and
muscle strengthening on their own and in therapy.
Rehabilitation
Exercises are started the day after surgery. We prefer the patient be involved in their
recovery and re-etablishment of knee motion and strengthening. Many patients prefer
a close working relationship with a physical therapist. The therapist should
communicate and alert the surgeon if problems are developing. The surgeon and
therapists guide the return to various activities. Table 12-1 shows our standard
rehabilitation protocol. Our goal is to re-establish full knee range of motion and
eliminate effusions by 2 months postoperatively. Some patients meet these goals
before 2 months. In the uncomplicated recovery, cycling can usually begin by 1 month,
and jogging can be initiatd by 3 months. Unrestricted activity is usually delayed until 6
to 12 months following ACL reconstruction and needs to be individualized for each
patient.
COMPLICATIONS
Decreased Motion
Failure to regain motion is usually associated with (a) technical problems related to
the surgery, (b) cyclops formation and adhesions, or (c) inadequate postoperative
exercising and initiative.
Failure to regain knee extension is the most common range-of-motion complication.

Loss of extension may be due to graft impingement. It is usually from anterior
placement of the tibial tunnel. (See Chapter 14 for more details.) We prefer our
patients to have full extension within a few weeks of their ACL reconstruction.
Attention to extension is stressed and occasionally specific braces are prescribed if
flexion contractures are not improving. Failure to regain full motion by 2 months may
need to be addressed with aggressive treatment, such as arthroscopic lysis of
adhesions, debridement of soft tissue anterior to the graft, roof notchplasty, and/or
manipulation. Figure 12-30 demonstrates a well-placed ACL graft in relationship to the
intercondylar notch. Note the extension of the knee in this MRI and the uniform signal
within the graft. Graft impingement against the intercondylar roof most commonly
results from anterior placement of the tibial tunnel. (See Chapter 14, where graft
impingement in flexion and extension are discussed further.)
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TABLE 12-1. ACL Reconstruction and Rehabilitation
Protocol
Phase Iâ€”The Acute Phase (1-10 days)
â€¢ ROM
â—‹ 0-90 degrees; emphasize full extension with prone hangs or

pillow under ankle
â—‹ Active ROM exercises 5/day
â—‹ Heel slides, assisted with belt as needed
â—‹ Hip and ankle ROM
â€¢ Strength/Cardio
â—‹ Straight leg raise cocontractions 5/day (10 reps)
â—‹ Hamstring sets with good leg
â€¢ Gait
â—‹ PWB with crutches
â—‹ Knee immobilizer for ambulation and sleep

â€¢ Remove for ROM exercises and periodically
â€¢ Other
â—‹ Minimize swelling
â—‹ Emphasize patellar mobilization for B-PT-B autografts
Phase II (10 days-3 weeks)
â€¢ ROM
â—‹ 0-120 degrees; continue ROM exercises
â—‹ Begin quadriceps and hamstring sets
â—‹ Emphasize VMO
â—‹ Continue ankle and knee exercises
â—‹ Stationary bike: 1/2 to full revolutions
â—‹ Pool exercises optionalâ€”when wounds healed
â€¢ Gait
â—‹ Braced discontinued once quadriceps control regained
â—‹ Wean off crutches
â€¢ Other
â—‹ Balance and proprioception exercises
Phase III (3-6 weeks)
â€¢ ROM
â—‹ Push full PROM
â—‹ Stationary bike as tolerated
â—‹ Stair Master
â—‹ Hamstring curlsâ€”concentric and eccentric
â€¢ Gait
â—‹ Emphasize normal gait without brace or crutches
Phase IVâ€”The Normalization phase (6-12 weeks)

â€¢ ROMâ€”Full active and PROM
â—‹ Stair Master
â—‹ Partial squats
â—‹ Single leg squats
â—‹ Wall sits
â—‹ Open-and closed-chain exercises
â€¢ Gaitâ€”Normal
â€¢ Other
â—‹ Most can return to jobs that require prolonged standing
â—‹ Increase intensity of exercises as tolerated
Phase Vâ€”The Cardio phase (3-6 months)
â€¢ ROMâ€”should be normal
â—‹ Full, unrestricted low-impact workouts with Stair Master,
elliptical trainer
â—‹ Quadriceps sets, low weights for leg presses and hamstring
curls
â—‹ Strengthen hips, calves, ankles
â—‹ Jump rope
â€¢ Gait
â—‹ No limp
â—‹ Increase walking distances (can use treadmill)
â—‹ Begin jogging straight ahead and increase as tolerated
â€¢ Other
â—‹ Can begin sport-specific training
â—‹ Box drill
â—‹ Gentle figure-8s
Phase VIâ€”The Sport-Specific Phase (6-9 months)
â—‹ Increase weights and intensity from last phase
â—‹ Progress from jogging to running and sprinting
â—‹ Continue figure-8s and box drills
â—‹ Jump and hop drills
â€¢ Other
â—‹ Return to sports when strength is within 90% of good leg,

and ROM is normal
â—‹ Completed sport-specific training
â—‹ Intensity and duration of sports participation increases as

tolerated
PROM, passive range of motion; PWB, partial weight bearing; ROM, range of
motion; VMO, vastus medialis obliquus.
Neurologic Complications
The most commonly injured nerve in harvesting the patellar tendon graft is the
infrapatellar branch of the saphenous nerve. It frequently produces a small area of
numbness along the anterolateral aspect of the knee. This is usually not a significant
problem unless a painful neuroma develops.
Hemarthrosis
Hemarthrosis results in an uncomfortable swelling immediately after surgery. It usually
resolves spontaneously but may increase postoperative pain, decrease mobility, slow
progress in physical therapy, and increase the risk of infection. Meticulous hemostasis
during surgery, with use of the intra-articular electrocautery, can be helpful in
controlling persistent bleeding. We have not found it necessary or helpful to insert a
drain postoperatively in our ACL reconstructions.
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FIGURE 12-30
This MRI obtained 2 months after the ACL reconstruction demonstrates extension
of the knee with no graft impingement against the intercondylar notch and
uniform signal throughout the graft.
Effusions
Effusions can be a problem in the first few weeks after surgery and can retard progress
in rehabilitation, particularly flexion and quadriceps control. The management of
effusions includes aspiration (when necessary), anti-inflammatory medication, and
decreased activity. Persistent effusions after 2 months are indicative of an intra-
articular problem (i.e., articular cartilage surface particulate shedding) or a technical
aspect related to tunnel placement and graft position from the surgery.
Postoperative Infections
Intra-articular infections following ACL reconstruction are a quite infrequent
occurrence. Those that do occur have the best chance of successful treatment if
recognized early. We would prefer, if recognized early, to immediately perform an
arthroscopic lavage, debridement with graft retention, and to obtain cultures. Further
treatment continues with the desired administration and duration of appropriate
antibiotics. Those that respond as expected can be followed until all the infection is
eliminated. If the infection does not respond in a reasonable time, the graft and
fixation may need to be removed.
RESULTS
It has been our experience that 90% of our patients are pleased with their stabilty
following ACL reconstruction. Our results in patients over age 40 are comparable to
those in younger patients. Although stablization of the ACL deficient knee is quite
successful, it is the associated injuries and pathology that often limit outcomes. Those
who do not regain full symmetrical knee motion or have meniscal pathology,
underlying degenerative articular disease, associated ligament injury, and
malalignment of the lower extremity may not have as good long-term functional
outcomes.
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RECOMMENDED READING
Chhabra A, Starman JS, Ferretti M, et al. Anatomic, radiographic, biomechanical, and
kinematic evaluation of the ACL and its two functional bundles. J Bone Joint Surg,
2006;88:2-10.
Frank CB, Jackson DW. Current concepts review. The science of reconstruction of the
anterior cruciate ligament. J Bone Joint Surg. 1997;79A(10):1556-1576.
Jackson DW, ed. The anterior cruciate ligament: current and future concepts. New
York: Raven Press; 1993.
Matsumoto A, Howell SM. Avoiding posterior cruciate ligament and roof impingement
with transtibial anterior cruciate ligament reconstruction: keys to correct tunnel
placement. Techniques in Orthopaedics. 2005;20(3):211-217.
Schaefer RK, Jackson DW. Cyclops syndrome: loss of extension following intra-articular
ACL reconstruction. Arthroscopy 1990;6:171-178.
Scranton PE, Beynnon BD, Johnson RJ, et al. Outcomes of different autografts and
grafting techniques in anterior cruciate ligament replacement. J Bone Joint Surg Am.
2003;85(7):1397-1399.
> Table of Contents > Part Three - Ligament Injuries and Instability > 13 - Anatomic Anterior
Cruciate Ligament Double-Bundle Reconstruction
13
Anatomic Anterior Cruciate Ligament Double-
Bundle Reconstruction
Mario Ferretti
Wei Shen
Hanno Steckel
Freddie H. Fu
Anterior cruciate ligament (ACL) tear is a common injury in orthopaedic sports
medicine. Surgical reconstruction is the preferred choice in management to restore
knee stability and prevent intra-articular injuries. Current techniques in ACL surgery
have been associated with satisfactory long-term results in the majority of patients;
however, there remains a considerable subset of patients with unsatisfactory
outcomes. Specifically, patients report difficulties relating to rotational instability and
return to previous level of activity. The anatomic ACL double-bundle (DB)
reconstruction aims to restore the original anatomy and footprints of the ACL by
reconstructing the anteromedial (AM) and posterolateral (PL) bundle to achieve a
better functional outcome, which is in contrast to single-bundle (SB) reconstruction,
which attempts to restore the fibers of the AM bundle. The indications for a
reconstruction of a torn ACL applying the anatomic ACL DB reconstruction are like the
traditional SB reconstruction and are influenced by several factors. Universally
accepted indications for ACL reconstruction are heavy work occupation, high-risk
lifestyle, demanding level of sports activity, instability in spite of rehabilitation, and
associated injuries like meniscal tears or severe injuries to other ligament structures in
the knee. Age is also an important parameter to take into account; however, it is not
so much the biologic age, but the status of the knee and the individual lifestyle which
have to be considered.
Contraindications for the anatomic ACL DB reconstruction include advanced

osteoarthritis of the knee, patients who are unable to comply with the postoperative
rehabilitation protocol, and acute intra-articular sepsis. Open physes are not an
absolute contraindication for the DB reconstruction, since several authors have
described satisfactory results with reconstruction of the ACL using soft tissue grafts
passed through the transphysial drill holes. However, extra attention should be paid to
patients with large open physes or patients with potential to grow.
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Planning the surgical procedure includes taking the patient's history as well as a
physical examination. For outcome evaluation, clinical parameters such as range of
motion (ROM), Lachman grade, anterior drawer grade, pivot-shift grade, objective
measurements of knee laxity as obtained with KT-1000 arthrometer, and scores such as
the International Knee Documentation Committee Subjective Knee Form (IKDC) and
Lysholm are important and are routinely applied. Especially validated outcome
instruments like 36-Item Short Form Health Survey (SF-36), Activities of Daily Living
(ADLS), Knee Injury and Osteoarthritis Outcome Score (KOOS), and Sports Activities
Scales (SAS) are also needed for future evaluation of the DB reconstruction. Imaging of
the knee is performed with routine radiographs to determine fractures, osteoarthritis,
and the alignment of the lower extremity (Fig. 13-1). Magnetic resonance imaging is
always done to assess further intra- and extra-articular injury and to confirm the
rupture of the ACL (Fig. 13-2). The time interval from injury to reconstruction is not as
important as the condition of the knee at the time of surgery. The knee should have
minimal effusion and full range of motion. The patient must be physically and mentally
prepared for surgery and free of pain when determining the date of reconstruction.
SURGERY
The patient and the limb to be operated on are identified by the surgeon in the
preoperative holding area. In the operating room, the patient is placed in a supine
position. Either general or spinal anesthesia is performed according to the preference
of the patient and anesthesiologist. Examination under anesthesia is executed with the
Lachman test and pivot shift. Special attention is given to the laxity of the knee joint.
Under anesthesia, when the pivot shift is grade 1 or graded as a gliding pivot and the
side-to-side difference using KT 1000 is less than 5 mm, a possible partial tear of the
ACL is considered as a differential diagnosis and further arthroscopic evaluation will
elucidate the correct diagnosis. It is important to note that in the case of a complete
ACL tear, an anatomic ACL DB reconstruction will be performed. Rarely, when a partial
tear of the ACL is diagnosed, an augmentation of the torn bundle, AM or PL bundle,
may be a logical approach.
FIGURE 13-1
Anteroposterior (A) and lateral (B) views of radiographs are taken at the patient's
initial visit. Possible injures like fractures and osteoarthritis, and the alignment of
the lower extremity, are examined.
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FIGURE 13-2
A sagittal view of MRI confirms a torn ACL.
The procedure is performed with the patient in supine position with the nonoperative
leg placed in a well-leg holder in the abducted position. The operative leg is
exsanguinated by elevation for 3 minutes; a pneumatic tourniquet is applied around
the upper thigh and insufflated to 350 or 400 mmHg depending on the patient's size.
Finally, the operative leg is placed in an arthroscopic leg holder, which allows good
range of motion (Fig. 13-3), sterile preparation, and draping. The surgery is performed
in a stepwise fashion as follows: (a) evaluation under anesthesia, (b) patient set-up,
(c) graft preparation, (d) evaluation of the ACL rupture pattern and marking of the
footprints of the ACL, (e) positioning and preparation of the tunnels, (f) graft passage
and fixation, (g) closure and postoperative assessment of stability, and (h)
postoperative management and rehabilitation.
Technique
Arthroscopy is performed for diagnosis and treatment of associated injuries. The
portals are critical to obtain optimal intra-articular visualization and management of
the arthroscopic instruments. We use the standard anterolateral (AL) and
anteromedial (AM) portal; an accessory anteromedial portal (AAM) is also used (Fig. 13-
4). To establish the AAM portal, the arthroscope is placed into the standard AL or AM
portal and an 18-gauge spinal needle is inserted medially and distally to this portal just
above the meniscus to reach the center of the femoral footprint of the PL bundle (Fig.
13-5). Once the needle is placed correctly, the AAM portal is performed with a #11
blade. While the arthroscopy is being performed, the grafts are being prepared in a
back table by another surgeon. We prefer the use of two separate tibialis anterior or
tibialis posterior tendon allografts. These allografts are usually 24 to 30 cm in length,
and we fold each tendon graft to obtain 12 to 15 cm double-stranded grafts (Fig. 13-
6). First, the tendon allografts are trimmed and the diameters of the double-stranded
grafts are adjusted. Usually, the AM tendon graft is trimmed such that the diameter of
the double-stranded graft is 8 mm; the PL graft is trimmed such that the diameter of
the double-stranded graft is 7 mm. However, the diameters of the graft should be
smaller in a small patient. The ends of the grafts are sutured using a baseball stitch
fashion with 2-0 Ticron sutures. An EndoButton CL (Smith & Nephew, Andover, MA) is
used to loop each graft and obtain a double-stranded graft. The length of the
EndoButton loop is chosen according to the measured length of the femoral tunnels.
During the arthroscopy, it is important to observe the rupture pattern of the ACL (Fig.
13-7). This step definitively diagnoses the total tear of the ACL and shows us the
original footprint of the AM and PL bundles on the lateral wall of the intercondylar
notch and on the tibial side (Fig. 13-8). When identified, the footprints of each bundle
are marked by a thermal device (Artrocare Corporation, Sunnyvale, CA). A small hole
in the center of the femoral AM and PL footprint is created by Steadman awl to
facilitate further guidewire placement to create the femoral tunnels (Fig. 13-9). After
marked by a thermal device, the remaining tibial footprint fibers are left intact due to
their proprioceptive and vascular contributions. It is important to note that in acute
cases, the rupture pattern and original anatomy are more easily observed than in
chronic cases, in which you may not find the ACL footprints as well. A good
understanding of the double-bundle ACL anatomy is essential to identify and mark the
correct footprints.
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FIGURE 13-3
A knee holder is used to keep the surgery knee stable during the surgery (A). It
also allows a good range of motion, both extension (B) and flexion (C), during the
surgery.
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FIGURE 13-4
Standard anterolateral and anteromedial portal are used. In addition, an

accessory anteromedial portal is used to provide better view of the lateral wall of
the femoral notch. Tibial incision is made at the medial aspect of tibia, and it is
anterior to the tibial attachment of MCL.
FIGURE 13-5
A spinal needle is inserted to decide the position of the accessory anteromedial

portal. A: Outside and (B) inside.
FIGURE 13-6
A,B: The graft is prepared on a back table by an assistant surgeon. The length of
the femoral tunnels is measured, and the length of the grafts is adjusted
accordingly.
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FIGURE 13-7
A thermal device is used to investigate the injury pattern of ACL. One example
shows that the AM bundle is intact, whereas the PL bundle is stretched.
The PL femoral tunnel is the first tunnel to be drilled. The PL femoral tunnel is drilled
through the AAM portal, and a 3.2-mm guidewire is inserted through the portal. The
tip of the guidewire is placed on the small hole created previously by Steadman awl on
the center of the femoral footprint of the PL bundle. Once the tip of the guidewire is
malleted in the correct position, the femoral PL tunnel is drilled with an acorn drill
that is inserted over the guidewire. The PL femoral tunnel is drilled to a depth of 25 to
30 mm (Fig. 13-10). The far cortex is breached with a 4.5-mm EndoButton drill (Smith
& Nephew, Andover, MA) and the depth gauge is used to measure the distance to the
far cortex. It is important to note that during the drilling of the PL bundle tunnel, the
arthroscopy is placed through the anteromedial portal and the tunnel is drilled through
the accessory anteromedial portal (Fig. 13-11). It is also important to note that during
the entire procedure to create a PL femoral tunnel the knee is positioned at 110
degrees of flexion, which brings the PL bundle footprint anteriorly.
FIGURE 13-8
The insertion sites of AM and PL on both tibial side (A) and femoral side (B) are
marked by a thermal device.
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FIGURE 13-9
A Steadman awl is used to mark the desired femoral AM and PL positions for guide
pins. It also makes it easier for the guide pins to stick to the chosen spot.
FIGURE 13-10
A guide pin is used first (A), followed by an acorn drill (B). The EndoButton drill is
then used to drill a narrower tunnel for the EndoButton (C). The finished PL
femoral tunnel is shown (D).
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FIGURE 13-11
The PL femoral tunnel is drilled through the accessory anteromedial portal.

To create the two tibial tunnels, a 4-cm skin incision is made over the anteromedial
surface of the tibia at the level of the tibial tubercle. The PL tibial tunnel is the first
one to be drilled. The elbow ACL tibial drill guide is set up at 45 degrees and the tip of
the drill guide is placed intra-articularly on the tibial footprints of the PL bundle.
Another way to locate the tip of the tibial guide intra-articularly, is to place it just in
front of the posterior root of the lateral menisci, and posterolateral to the AM bundle
of the ACL (Fig. 13-12). On the tibial cortex, the tibial drill starts just anterior to the
superficial medial collateral ligament fibers. Once the tibial drill guide is set up intra-
articularly and on the tibial cortex, a 3.2-mm guidewire is passed into the stump of
the PL tibial footprint (Fig. 13-13). The AM tibial tunnel is drilled with the elbow ACL
tibial drill guide set at 45 degrees, and the
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tip of the drill guide is placed on the tibial footprint of the AM tunnel already marked
with the thermal device, and anteromedial to the PL guidewire. On the tibial cortex
the starting point for the AM bundle is placed anterior, central, and proximal than the
PL starting point. Once the elbow ACL tibial drill guide is placed in the desired
position, a 3.2-mm guidewire is passed into the stump of the AM tibial footprint (Fig.
13-14). After passing the tibial guidewire into an adequate position, the two tibial
tunnels are drilled using a cannulated drill. A curette is used for protection (Fig. 13-
15). Finally, a dilator is used to enlarge the tunnels to the desired diameters (Fig. 13-
16).
FIGURE 13-12
The tibial PL insertion site is located posterolaterally to the tibial AM insertion

site. It is also located 2 to 4 mm in front of the posterior root of lateral meniscus.
The PL triangle is formed by PCL, posterior root of lateral meniscus, and the AM
insertion site.
FIGURE 13-13
The tibial insertion sites of AM and PL bundles are shown. The ACUFEX aiming
elbow is used to guide the direction of the guide pin.
The femoral AM tunnel is the last tunnel to be drilled. We use three methods to drill
the AM femoral tunnel. A transtibial AM technique in a similar fashion for a single-
bundle ACL reconstruction is our first choice to create the AM femoral tunnel.
However, in some cases, the transtibial AM technique cannot reach the center of the
AM bundle marked previously. Then, we can try to reach the center of the AM femoral
footprint through the PL tibial tunnel. If the center of the AM femoral footprint is also
not reached through the PL tibial tunnel, we use the AAM portal to reach the center of
the AM bundle. The AM tunnel is drilled in a similar fashion that the PL femoral tunnel
was drilled (Fig. 13-17). Once the tip of the guidewire is placed in a correct position,
the guidewire is malleted, an acorn drill is inserted over the guidewire, and the AM
femoral tunnel is drilled to a depth of 35 to 40 mm when it is done transtibially either
through AM tibial tunnel or PL tibial tunnel. If the AM femoral tunnel is performed
through the AAM portal, it is drilled to a depth of 30 mm to avoid breaking cortex. The
far cortex of the AM femoral tunnel is breached with a 4.5 mm EndoButton drill, and
the depth gauge is used to measure the distance to the far cortex. The first graft to be
passed is the PL graft. A Beath pin with a long looped suture attached to the eyelet is
passed through the AAM portal and out through the PL femoral tunnel. The looped
suture is visualized within the joint and retrieved with an arthroscopic suture grasper
through the PL tibial tunnel (Fig. 13-18). A crossing pattern of the AM and PL graft is
clearly seen after passing both looped sutures through the tunnels (Fig. 13-19). The
graft is passed and the EndoButton is flipped to establish cortex fixation of the PL
bundle graft (Fig. 13-20). The AM is passed using the transtibial technique when the
tunnel was created transtibially or through the AAM portal when the tunnel was
created through the AAM portal, using the same technique as in the PL bundle graft
passage. The EndoButton is flipped in a standard fashion for AM bundle graft to
establish AM bundle femoral cortex fixation (Fig. 13-21). Preconditioning of the grafts
is performed by flexing and extending the knee through a range of motion from 0 to
120 degrees approximately 20 to 30 times (Fig. 13-22). On the tibial side, we prefer to
use bioabsorbable screws (Fig. 13-23). The graft fixation is important for the
tensioning of the grafts. The tensioning is performed manually by the surgeon and
fixed during the tibial fixation. The PL graft is fixed at 0 to 10 degrees of flexion. The
AM graft is fixed at 60 degrees of flexion. After tibial fixation, a final arthroscopic
inspection is performed to confirm the status of the graft, the absence of anterior
impingement, and posterior cruciate ligament (PCL) impingement (Fig. 13-24).
Subcutaneous tissue and skin are closed in a standard fashion. A Cryo Cuff (Aircast,
Vista, CA) is used for cold therapy and compression. A brace is used to keep the knee
locked in full extension (Fig. 13-25). A radiograph is taken to make sure the hardware
is in place (Fig 13-26). A magnetic resonance image is not routinely taken. However, it
can show the anatomic double-bundle graft (Fig. 13-27).
FIGURE 13-14
The AM and PL guide pins are inserted at 45 and 45 degrees, respectively (A). A
scope picture shows that AM and PL guide pins are inserted, and PL insertion site
is posterolateral to AM insertion site (B).
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FIGURE 13-15
A curette is used for protection when a cannulated drill is used to enlarge the
tibial tunnels.
FIGURE 13-16
Dilators are used to enlarge the tibial tunnels.

FIGURE 13-17
The femoral AM insertion site can be reached by different routes. By transtibial

AM tunnel (A), transtibial PL tunnel (B), or accessory anteromedial portal (C), the
femoral AM tunnel can be drilled. The finished femoral AM and PL tunnels are
shown (D).
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FIGURE 13-18
The looped suture is visualized and retrieved with an arthroscopic suture grasper
through the PL tibial tunnel.
FIGURE 13-19
When in flexion, the AM and PL graft sutures show a crossing pattern, which is
also true in intact ACL.
FIGURE 13-20
The PL graft is passed first. An EndoButton fixation is used to fix on the femoral
side (A). The graft will be pulled through (B) and the EndoButton will be flipped
on the femoral cortex.
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FIGURE 13-21
The AM graft is pulled through after PL graft. The reconstruction product restores
the normal anatomy of ACL very closely.
FIGURE 13-22
The grafts are conditioned by repetitive flexion and extension motion. This figure
also shows the exits of AM and PL sutures on the femoral side.
FIGURE 13-23
On the tibial side, the AM and PL grafts are fixed by biodegradable screws.
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FIGURE 13-24
By anatomic double-bundle reconstruction, anterior (A) and PCL (B)

impingements are usually avoided. The arrow shows that there are still spaces.
Pearls and Pitfalls
The accessory AM portal is established using an 18-gauge needle under direct

visualization. The portal location is medial and inferior to the standard AM
portal (see Fig. 13-4). Placement is critical in obtaining the correct trajectory
and entry point for the PL femoral tunnel, and to avoid injury to the medial
femoral condyle surface and medial meniscus during drilling. The accessory
anteromedial portal offers a complete view of both the AM and PL femoral
origins.
It is essential to position the leg to allow for a range of motion between full
extension and 120 degrees of flexion intraoperatively. This position is needed
to fully appreciate the anatomy of the ACL since the relative positions of the
femoral insertions of the AM and PL bundles change with the flexion angle.
Anatomic landmarks for the PL femoral tunnel pin are as follows: 5 to 7 mm

from the anterior lateral femoral condyle (LFC) cartilage, and 3 mm from the
inferior LFC cartilage. The knee is flexed to 110 degrees to protect the
peroneal nerve and to ensure adequate EndoButton tunnel length.
The PL tibial insertion is located anterior and medial to the posterior root of
the lateral meniscus. It is lateral and anterior to the PCL, and posterolateral to
the AM bundle insertion site. Thus, the PL tibial insertion site is located within
the triangle formed by the AM, the PCL, and the posterior root of the lateral
meniscus. A proximal medial tibial incision is made. A tibial drill guide is set at
45 and 45 degrees for the PL and AM bundles, respectively, and placed on the
tibial PL and AM insertion marked previously.
The AM femoral tunnel placement is referenced 2 to 3 mm posterior to the PL

tunnel posterior rim, with the knee held at 90 degrees. The guidewire may be
placed using either transtibial approaches or accessory anteromedial portal
approach, to achieve correct position relative to the PL tunnel. Transtibial is
preferred to achieve more divergent tunnels; however, accurate tunnel
position is the most important factor.
Prior to fixation, each graft is cycled through a full range of motion 25 to30
times using maximal manual tension.The AM graft is tensioned and fixed in 60
degrees of flexion.The PL graft is tensioned and fixed in 0 to 10 degrees of
flexion.
Caution is warranted when using the AAM portal to drill the femoral tunnel.
The tunnel tends to be short, and the femoral cortex may be more easily
broken for this reason. If the cortex is broken, the EndoButton fixation is not
feasible. A lateral incision is made to change the EndoButton femoral fixation
to a femoral post fixation.
By using an allograft, we can decide the exact diameter of the tunnels and
grafts. It is important to note that our standard tunnels sizes are 8 mm for the
AM bundle and 7 mm for the PL bundle. However, in smaller patients the
standard tunnels may be too large and may cause problems such as blowout of
the posterior wall of the femoral tunnel as well as blowout of the bridge
between AM and PL femoral tunnels. In addition, excessive grafts may cause
anterior impingement. Thus, the tunnel and graft size should be adjusted for
small patients.
The cortex of the lateral femoral condyle becomes thinner as it comes distally.
For this reason we should be careful when placing the PL tunnel and sometimes
even the AM tunnel, especially when it is drilled through the AAM portal. When
using EndoButton fixation, the EndoButton is flipped and fixed in the distal
part of the thin cortex. When pulling the graft for tensioning and confirming
the fixation, a large traction force is applied to the graft and the EndoButton
puts a large pressure on the thin cortex. The cortex may be broken by the
hardware and the EndoButton is pulled into the tunnel, leading to failure of
the fixation.
FIGURE 13-25
In addition to standard dressing, the knee is wrapped up by cryocuff for cold

therapy and compression (A). A knee brace is used to lock and protect the knee in
extension (B).
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FIGURE 13-26
Anteroposterior (A) and lateral (B) views of radiographs are taken after the
surgery to check the position of hardwares.
FIGURE 13-27
Coronal (A) and sagittal (B) views of MRI show the AM and PL grafts.
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The double-bundle ACL reconstruction is performed on an outpatient basis. Patients
will be taken to the recovery room when the operation is over and will remain there
for up to 2 hours. Their vital signs will be monitored, and they are usually sent to
home when they are fully awake and have recovered from the anesthesia, unless they
have significant discomfort from anesthesia or pain. The patient is prescribed pain
medication that is taken every 4 hours during the first 2 postoperative days, and as
needed after that. In addition, we recommend that patients take pain medication 1/2
hour before exercise or using the continuous passive motion (CPM) machine. Ice or
cryocuff is suggested, as well as elevation of the knee, especially 20 to 30 minutes
after physical therapy or other activities. The patients are taught to change their
dressing 2 days after surgery and keep their incision site clean and dry. A brace is
prescribed after the surgery. During the first week the brace should be locked straight
except while performing exercises or using a CPM machine. Crutches are used for 4 to
6 weeks after surgery, depending on the recovery of the patient. Weight bearing is
permitted as long as the patient tolerates it.
Rehabilitation
Rehabilitative therapy is begun the day after surgery. The important issues in
rehabilitation of double-bundle ACL reconstruction include minimizing inflammation,
restoring motion and muscle strength, enhancing proprioception and dynamic stability,
returning to function, and preventing recurrence. In the immediate postoperative
period, our goal is to reduce pain and swelling of the knee, increase range of motion,
and strengthen the quadriceps and hamstring muscles. The weight bearing should
progress from a partial level to full. At 1 to 3 months postoperatively, the work on
range
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of motion and muscle strength should be continued. Closed chain and functional
exercises should be incorporated, as well as activities to enhance balance and
neuromuscular control. After 3 months postoperatively, rehabilitation should continue
to improve strength and neuromuscular control. At this time plyometric and sports-
specific exercises should be added. In terms of returning to sports, we believe that the
individual has to have no pain or swelling, 80% to 90% quadriceps strength, a normal
hop test, and adequate proprioception and neuromuscular control. Our rehabilitation
protocol is outlined in Table 13-1.
TABLE 13-1. ACL Reconstruction and Rehabilitation

Protocol
Stage 1: Begins immediately postoperatively through approximately 6 weeks
Goals:
1. Protect graft fixation

2. Minimize effects of immobilization
3. Control inflammation
4. Full extension and flexion range of motion
5. Educate patient on rehabilitation progression
Therapeutic exercises:
1. Heel slides
2. Quadriceps sets
3. Patellar immobilization
4. Non-weight-bearing gastro/soleus stretches, begin hamstring

stretches at 4 weeks
5. Straight leg raises in all planes with brace in full extension until
quadriceps strength is sufficient to prevent extension lag
6. Quadriceps isometrics at 60 and 90 degrees
Stage 2: Begins immediately 6 weeks postoperatively and extends to

approximately 8 weeks
Goals:
1. Restore normal gait
2. Maintain full extension, progress flexion range of motion
3. Protect graft fixation
Therapeutic exercise
1. Wall slides 0 to 45 degrees
2. 4-way hip machine
3. Stationary bike
4. Closed-chain terminal extension with resistance tubing or weight

machine
5. Toe raises
6. Balance exercises
7. Hamstring curls
8. Aquatic therapy with emphasis on normalization of gait
9. Continue hamstring stretches, progress to weight-bearing

gastro/soleus stretches
Stage 3: Begins approximately 8 weeks and extends through approximately 6
months
Goals:
1. Full range of motion
2. Improve strength, endurance, and proprioception of the lower

extremity to prepare for functional activities
1. Continue flexibility exercises as appropriate for patient
2. Stair Master (begin with short steps, avoid hyperextension)
3. NordicTrack
4. Advanced closed-chain strengthening (one-leg squats, leg press 0

to 50 degrees, step-ups, etc.)
5. Progress proprioception activities (slide board, use of ball with

balance activities, etc.)
6. Progress aquatic therapy to include pool running, swimming (no

breaststroke)
Stage 4: Begins approximately 6 months and extends through approximately

9 months
Goals:
1. Progress strength, power, proprioception to prepare for return to

functional activities
1. Continue to progress flexibility and strengthening program
2. Initiate plyometric program as appropriate for patient's functional

goals
3. Functional progression including, but not limited to, walk/jog

progression, forward/backward running at 1/2, 3/4, full speed,
cutting, crossover, carioca, etc.
4. Initiate sport-specific drills as appropriate for patient
Stage 5: Begins approximately 9 months postoperatively
Goals:
1. Safe return to athletics
2. Maintenance of strength, endurance, proprioception
3. Patient education with regard to any possible limitations
1. Gradual return to sports participation
2. Maintenance program for strength and endurance
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COMPLICATIONS
When considering double-bundle reconstruction, it is important to address potential
complications. General complications after knee surgery include hemarthrosis,
effusions, and wound infection, which are treated according to the common
postoperative procedures. Tunnel widening and sclerosis, tibial and femoral fractures,
and notch or posterior cruciate ligament impingement following ACL reconstruction
are known to occur in a single-bundle reconstruction, but can also occur when applying
the anatomic ACL DB reconstruction. When performing the tibial incision, the
infrapatellar branch of the saphenous nerve can be damaged, leaving a small numb
area along the anterolateral aspect of the knee and, rarely, a painful neuroma.
RESULTS
From November 2003 to September 2006, the senior author has performed 340
anatomic ACL reconstructions. In a total of 275 primary cases, 261 patients (95%) had
primary double-bundle reconstruction and 14 patients (5%) had primary one-bundle
augmentation. In a total of 65 revision cases, 44 patients (68%) had double-bundle
revision and 21 patients (32%) had secondary one-bundle augmentation. In the primary
augmentation patients, we found in surgery that either the AM or the PL bundle was
still functional, so that only the torn bundle was reconstructed and the functional
bundle was left intact. In the 21 patients who underwent secondary augmentation, 19
patients had previous single-bundle reconstruction. They were unstable and had
positive pivot shift test, despite good range of motion and minimal side-to-side
difference of KT-2000 score. The original graft was found functionally intact or savable
during the surgery, and only PL bundle reconstruction was performed. The other two
secondary augmentation patients had previous double-bundle reconstruction. They
were found to have a failed AM graft and a functional PL graft. Only the AM graft was
reconstructed, and the PL graft was left intact. With primary or secondary one-bundle
augmentation, the double-bundle anatomy of ACL and the stability of the knee were
restored.
A prospective study was designed to evaluate the outcomes of all these patients.
Outcome measurements included the Lachman and pivot shift tests, KT-2000, range of
motion, and overall IKDC rating. Short-term results demonstrated that the range of
motion after primary double-bundle ACL reconstruction is better than primary single-
bundle ACL reconstruction at 1, 4, and 12 weeks postoperatively following the same
rehabilitation protocol. To determine if this technique offers advantages over single-
bundle reconstruction in the long term, these patients are being continuously
followed. The initial 100 consecutive patients who underwent primary double-bundle
ACL reconstruction have been followed for an average of nearly 2 years. Lachman test,
pivot shift test, and KT-2000 results demonstrated that anteroposterior stability was
effectively restored by double-bundle ACL reconstruction. Approximately 94% of
patients had an excellent or good IKDC rating. There were four failures in our initial
100 patients, three of whom required double-bundle revision surgery. For the
augmentation and the double-bundle revision cases, preliminary results are currently
being reviewed and appear promising.
Rotational stability should be an important portion of outcome evaluation after ACL

reconstruction. In an in vivo laboratory study, Tashman et al. have suggested the
superiority of double-bundle ACL reconstruction in restoring the rotational stability
when compared to single-bundle ACL reconstruction. In the near future, an ACL
surgery navigation system may also be used as a tool for clinical measurement of
rotational stability in our study.
RECOMMENDED READING
Amis AA, Dawkins GPC. Functional anatomy of the anterior cruciate ligament. Fiber
bundle actions and related to ligament replacement and injuries. J Bone Joint Surg Br.
1991;73:260-267.
Girgis FG, Marshall JL, Monajem A. The cruciate ligaments of the knee joint.
Anatomical, functional, and experimental analysis. Clin Orthop. 1975;106:216-231.
Tashman S, Collon D, Anderson K, et al. Abnormal rotational knee motion during

running after anterior cruciate ligament reconstruction. Am J Sports Med.
2004;32:975-983.
Yagi M, Wong EK, Kanamori A, et al. Biomechanical analysis of an anatomic anterior

cruciate ligament reconstruction. Am J Sports Med. 2002;30:660-666.
> Table of Contents > Part Three - Ligament Injuries and Instability > 14 - Avoiding ACL Graft
Impingement: Principles for Tunnel Placement Using the Transtibial Tunnel Technique
14
Avoiding ACL Graft Impingement: Principles
for Tunnel Placement Using the Transtibial
Tunnel Technique
Keith W. Lawhorn
Stephen M. Howell
The transtibial tunnel technique in which the femoral tunnel is drilled through the
tibial tunnel is a common technique for tunnel placement. This technique can be used
regardless of the type of graft material and fixation. The transtibial technique can be
used in all patients requiring a primary anterior cruciate ligament (ACL) reconstruction
both acute and chronic, revision ACL surgery, and ACL reconstruction in the setting of
the multiligament knee injury. There are no absolute contraindications to use of the
transtibial tunnel technique for ACL reconstruction. Correct tunnel placement for ACL
reconstruction is imperative to the success of the procedure. Graft sources, fixation
methods, and rehabilitation cannot overcome the adverse consequences of poor tunnel
placement.
There are three criteria of correctly placed tibial and femoral tunnels for ACL
reconstruction: (a) the avoidance of roof impingement, (b) avoidance of posterior
cruciate ligament (PCL) impingement, and (c) the establishment of the tensile
behavior in the ACL graft similar to the native ACL. All three criteria are required for a
successful ACL reconstruction.
Roof impingement occurs when the ACL graft prematurely makes contact with the
intercondylar roof of the notch before the knee reaches terminal extension, which
causes loss of extension and anterior laxity. The cause of roof impingement is
positioning the tibial tunnel too anterior in the sagittal plane.
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PCL impingement occurs when the ACL graft makes contact with the leading edge of
the PCL before the knee reaches terminal flexion, which causes loss of flexion and
anterior laxity. The cause of PCL impingement in the transtibial technique is
positioning the tibial tunnel too vertical and medial in the coronal plane.
Finally, correct femoral tunnel position in both the sagittal and coronal planes is
required to establish the tensile behavior in the ACL graft similar to the native ACL.
Because the femoral tunnel is drilled through the tibial tunnel using size-specific
aimers, the position of the tibial tunnel determines the position of the femoral tunnel
with the transtibial technique. Therefore, the tibial tunnel sets up the position of the
femoral tunnel, which means that the key tunnel in the transtibial tunnel technique is
the tibial tunnel.
Once the decision has been made by the patient to undergo surgical treatment of the
ACL deficient knee, the surgeon must decide on the technique for placing the tunnels,
graft choice, and fixation. All tunnel placement techniques including the transtibial,
transportal, and two-incision technique can be used with any type of graft and
fixation. Scientific studies by a variety of authors support the use of the transtibial
tunnel technique and the use of the 65-degree tibial guide (Arthrotek, Warsaw, IN)
that references the intercondylar roof with the knee in full extension for placing the
tibial tunnel (Fig. 14-1).
FIGURE 14-1
A: Lateral view of 65-degree tibial guide with coronal alignment rod. B: Superior
view of 65-degree tibial guide with coronal alignment rod. C: Axial view of 65-
degree tibial guide demonstrating bullet with multiple holes for guidewire
placement.
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An advantage of the transtibial technique with the 65-degree tibial guide is that the
tunnel placement is accurate and customized without the need for tedious
preoperative planning or time-consuming intraoperative imaging. Customization begins
with the use of the 9.5-mm wide tip of the 65-degree tibial guide to gauge the width
of the space between the lateral edge of the PCL and the lateral femoral condyle.
Generally, the space between the PCL and the lateral femoral condyle is substantially
smaller than the width of the ACL graft. The portion of the notch allocated to the ACL
graft varies among patients as evidenced by some notches being narrower than others,
and by the cross-sectional area of some notches being dominated by the PCL, leaving
little room for the ACL graft. We advocate widening the notch by removing bone from
the medial edge of the lateral femoral condyle (i.e., wallplasty) until the space
between the PCL and lateral femoral condyle exceeds the width of the ACL graft by 1
mm. Since most grafts range from 8 to 10 mm, the free insertion of the 9.5-mm wide
tip of the guide between the PCL and lateral femoral condyle indicates that the notch
is sufficiently wide. Customizing the width of the notch to accommodate the width of
the ACL minimizes PCL impingement by moving the tibial tunnel, femoral tunnel, and
ACL graft lateral away from the PCL, which minimizes PCL impingement improving
flexion and anterior stability.
A second step of customization is that the 65-degree tibial guide accounts for the
anatomic variability in the angle of the intercondylar roof and knee extension that
exists between patients and consistently positions the tibial tunnel in the posterior
half of the native ACL tibial footprint and avoids roof impingement without a
roofplasty. The roof angle (23 to 60 degrees) and knee extension (- to 30 degrees) both
vary widely among patients. The correlation between the two is weak, so that patients
with the same roof angle often have a different knee extension and patients with the
same knee extension often have a different roof angle. Surgeons need to account for
these two independent variables simultaneously. By drilling the tibial tunnel with the
knee in full extension while referencing the intercondylar roof with the 65-degree
tibial guide, the sagittal placement of the tibial tunnel has simultaneously accounted
for the patient's unique combination of roof angle and knee extension. Magnetic
resonance imaging studies have demonstrated the native ACL is located posterior and
parallel to the intercondylar roof with the knee in terminal extension. The tibial
tunnel is positioned posterior and parallel to the intercondylar roof and prevents roof
impingement of the ACL graft without performing a roofplasty, which improves knee
extension and anterior stability.
A third step of customization is that the 65-degree tibial guide incorporates the use of
a coronal alignment rod, which improves the accuracy of setting the angle of the tibial
tunnel in the coronal plane, further minimizing the risk of PCL impingement. Setting
the angle of the tibial tunnel at 65 Â± 5 degrees in the coronal plane avoids PCL
impingement. The use of the transtibial technique and a tibial tunnel at 65 Â± 5
degrees in the coronal plane positions the femoral tunnel so that the tensile behavior
of the ACL graft is similar to the native ACL while improving knee flexion and anterior
stability.
The last step in customization is the use of size-specific femoral aimers through a
tibial tunnel placed to create a femoral tunnel with a 1-mm back-wall for any
diameter ACL graft. The use of the size-specific femoral aimers in a knee in which the
notch has been sufficiently widened, without roof impingement and without PCL
impingement, ensures correct placement of the femoral tunnel. The remnants of the
ACL origin in the over-the-top position must be removed so that the femoral aimer
rests on bone. Resting the femoral aimer directly on bone creates a femoral tunnel
with a thin 1-mm back-wall and eliminates â€œblowingoutâ€• the posterior wall of the
femoral tunnel. Therefore, the advantage of the transtibial technique is that the
surgeon needs to focus on the meticulous placement of only one tunnel, the tibial
tunnel, reducing the error associated by placing the femoral and tibial tunnels
independently.
For surgeons, choosing to use a point-and-shoot guide, preoperative lateral

radiographs taken with the knee in full extension, helps determine the intercondylar
roof angles and knee extension and aids them intraoperatively when positioning the
tibial tunnel in the sagittal plane. Surgeons must estimate coronal plane positioning
with these guides since there are no coronal alignment devices on these guides. The
standard is to use intraoperative fluoroscopy to check the position of the tibial
guidewire in both the sagittal and coronal planes before drilling the tibial tunnel.
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SURGERY
Patient Positioning
Position the patient supine on the operating table. After induction of anesthesia,
perform an examination under anesthesia. Place a tourniquet around the proximal
thigh of the operative leg. Position the operative leg in a standard knee arthroscopy
leg holder with the foot of the operating table flexed completely. Alternatively,
surgeons may decide to use a lateral post instead of a leg holder. Position the
contralateral leg in a gynecologic leg holder with the hip flexed and abducted with
mild external rotation. Ensure there is no pressure on the peroneal nerve and calf (Fig.
14-2). Alternatively, surgeons can position the operative leg flexed over the side of the
table using a lateral post and maintaining the contralateral leg extended on the
operating table.
Technique
After sterile prep and drape, exsanguinate the leg and inflate the tourniquet. Establish
inferolateral and inferomedial portals touching the edges of the patella tendon
starting 1 cm distal to the inferior pole of the patella. Alternatively, a transpatellar
inferolateral portal can be used with a medical portal placed along the medial border
of the patella tendon (Fig. 14-3). The medial portal must touch the edge of the patella
tendon because if it is placed more medial, the tibial guide may not stay seated in the
intercondylar notch with the knee in full extension. An optional outflow portal can be
established superiorly.
Perform a diagnostic arthroscopy. Treat meniscal or articular cartilage injuries.

Identify and remove the torn remnant ACL stump (Fig. 14-4). It is not necessary to
denude the tibial insertion of the native ACL tissue. In fact, retaining the insertion of
the native ACL helps seal the edges of the ACL graft at the joint line and does not
result in roof impingement if the tibial tunnel has been appropriately positioned.
Remove the synovium and soft tissue in the notch to expose the lateral edge of the
PCL (Fig. 14-5). Remove any of the ACL origin from the over-the-top position using an
angled curette and shaver (Fig. 14-6).
FIGURE 14-2
Patient set-up.
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FIGURE 14-3
Portal placement for use of 65-degree tibial guide.
Insert the tibial guide through the medial portal. Advance the guide into the
intercondylar notch (Fig. 14-7). The tip of the guide is 9.5 mm wide. If the guide
makes contact and deforms the PCL as it enters the intercondylar notch, perform a
lateral wallplasty. Remove bone in 1- to 2-mm-wide slivers from the lateral wall until
the tip of the guide passes into the notch without deforming the PCL, which creates a
wide enough area for an 8- to 10-mm-wide graft (Fig. 14-8). Do not remove any bone
from the intercondylar roof since the roof anatomy is crucial for proper positioning of
the tibial guide-pin in the sagittal plane using the 65-degree tibial guide (Fig. 14-9).
Remove the lateral wallplasty fragments (Fig. 14-10).
Insert the 65-degree tibial guide through the anteromedial portal that touches the
medial edge of the patella tendon into the intercondylar notch between the PCL and
lateral femoral condyle to ensure adequate width of the notch for the ACL graft (Fig.
14-11). Fully extend the knee. Visualize that the tip of the guide is captured inside the
notch and that the arm of the 65-degree tibial guide contacts the trochlea groove (Fig.
14-12). Place the heel of the patient on a Mayo stand to maintain the knee in
maximum hyperextension. Stand on the lateral side of the leg and insert the coronal
alignment rod through the proximal hole in the guide. Rotate the 65-degree guide in
varus and valgus until the coronal alignment rod is parallel to the joint and
perpendicular to the long axis of the tibia (Fig. 14-13). Insert the combination bullet
guide/hole changer into the 65-degree guide and advance the bullet until seated
against the anteromedial cortex of the tibia. Drill the tibial guide-pin through the
lateral hole in the bullet until it strikes the guide intra-articularly (see Fig. 14-13).
Remove the bullet from the tibial guide and remove the guide from the notch. Tap the
guide-pin into the notch and assess its position.
FIGURE 14-4
Torn ACL stump.

FIGURE 14-5
Identify the leading superior border of the PCL.
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FIGURE 14-6
Clean the over-the-top position using an angled curette.

FIGURE 14-7
The 9.5-mm-wide tip of the 65-degree tibial guide demonstrates notch stenosis
with deformation of PCL.
FIGURE 14-8
A-C: Lateral wallplasty performed using angled osteotome.

FIGURE 14-9
Angled osteotome prevents extension of wallplasty into roof of intercondylar

notch.
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FIGURE 14-10
Removal of lateral wallplasty fragments.

FIGURE 14-11
Sixty-five-degree tibial guide tip ensures adequate notch width.
FIGURE 14-12
A: Sixty-five-degree tibial guide tip positioned in the intercondylar notch. B:

Extend the knee maintaining 65-degree tibial guide tip in the intercondylar notch.
C: Position of the 65-degree tibial guide arm against the trochlea groove with the
knee in terminal extension.
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FIGURE 14-13
A: Tibial guidewire drilled through lateral hole in bullet with the knee in full
extension and coronal alignment rod positioned parallel to the knee joint. B:
Lateral view.
The tibial guide-pin is properly positioned in the coronal plane when it enters the
notch midway between the lateral edge of the PCL and the lateral femoral condyle.
The guide-pin should not touch the PCL (Fig. 14-14). The tibial guide-pin is properly
positioned in the sagittal plane when there is 2 to 3 mm of space between the guide-
pin and intercondylar roof with the knee in full extension. The space can be assessed
by manipulating a 2-mm-wide nerve hook probe between the between the guide-pin
and intercondylar roof in the fully extended knee.
Prepare the tibial tunnel. Ream the tibial cortex with a reamer with the same
diameter as the prepared ACL graft. Harvest a bone dowel from the tibial tunnel by
inserting an 8-mm-in-diameter bone dowel harvester and centering rod over the tibial
guide-pin. Use a mallet and drive the bone dowel harvester until it reaches the
subchondral bone (Fig. 14-15). Remove the dowel harvester with cancellous bone
dowel (Fig. 14-16). If the tibial guide-pin is removed with the bone dowel, then
replace it by inserting it through an 8-mm reamer that has been reinserted into the
tunnel created by the bone dowel harvester. Ream the remainder of the tibial tunnel
with the appropriate diameter reamer.
Check for PCL impingement by placing the knee in 90 degrees of flexion and inserting
the impingement rod into the notch. A triangular space at the apex of the notch and
no contact at the base of the notch between the PCL and impingement rod confirms
the absence of PCL impingement (Fig. 14-17). Check for roof impingement by placing
the knee in full extension and inserting an impingement
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rod the same diameter as the tibial tunnel into the intercondylar notch (Fig. 14-18).
Free pistoning of the impingement rod in and out of the notch with the knee in full
extension confirms the absence of roof impingement.
FIGURE 14-14
Guide-pin position in the intercondylar notch.

FIGURE 14-15
Impact bone dowel harvester over guide-pin.
Place the femoral tunnel using the transtibial technique. Insert the size-specific
femoral aimer through the tibial tunnel with the knee in flexion. The size of the
â€œoff-setâ€• of the femoral aimer is based on the diameter of the ACL graft and is
designed to create a femoral tunnel with a 1-mm back-wall. Extend the knee and hook
the tip of the femoral aimer in the over-the-top position. Allow gravity to flex the
knee until the femoral guide seats on the femur. Rotate the femoral aimer a quarter
turn lateral away from the PCL, which positions the femoral guide-pin farther down
the lateral wall of the notch minimizing PCL impingement (Fig. 14-19). Drill a pilot
hole in the femur through the aimer and remove both the guide-pin and femoral
aimer.
Redirect the femoral guide-pin to shorten the femoral tunnel from 35 to 50 mm in

length with use of the following technique. Reinsert the femoral guide-pin into the
pilot hole and flex the knee to 90 to 100 degrees. Drill the guide-pin through the
anterolateral femoral cortex. Pass a cannulated 1-in reamer the same diameter as the
ACL graft over the guide-pin. Ream the femoral tunnel. Confirm the back wall of the
femoral aimer is only 1 mm thick (Fig. 14-20). Confirm the center of the femoral
tunnel is midway between the apex and base of the lateral half of the notch (Fig. 14-
21). A femoral tunnel placed correctly down the sidewall does not allow room for a
second posterolateral tunnel.
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FIGURE 14-16
Remove harvester with cancellous graft inside of harvester tube.
FIGURE 14-17
Impingement rod in notch.
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FIGURE 14-18
A: Extend knee and assess for roof impingement. B: Roof impingement not seen
with knee in full extension.
FIGURE 14-19
Femoral guide-pin position using femoral aimer through tibial tunnel.
FIGURE 14-20
Back wall thickness of femoral tunnel.

FIGURE 14-21
Femoral tunnel position assessed through lateral portal.
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Pearls and Pitfalls

Proper use of the 65-degree tibial guide minimizes the complications of impingement
associated with tunnel placement for ACL reconstruction surgery. Pitfalls typically
stem from improper use of the 65-degree tibial guide. First, it is important not to alter
the anatomy of the intercondylar roof when performing the lateral wallplasty should a
wallplasty be required. A significant roofplasty at the time of wallplasty can change
the sagittal plane position of the tibial tunnel. In revision cases where the
intercondylar notch anatomy may be distorted, the use of intraoperative fluoroscopy
or radiography may help to further ensure satisfactory tunnel position (Fig. 14-22). A
second pitfall is improper position of the medial portal. The 65-degree tibial guide
requires medial portal placement adjacent to the medial border of the patella tendon
to ensure the 65-degree tibial guide seats centrally in the intercondylar notch with the
knee in full extension. Too medial placement of the medial portal will position the 65-
degree tibial guide medial in the notch. Too medial placement of the tibial tunnel in
the notch using the transtibial tunnel technique will result in PCL impingement of the
graft. Finally, it is important to clean the over-the-top position using the angled
curette. Because the sizedspecific femoral aimers position the femoral tunnel with
only a 1- to 2-mm-thick posterior wall, any soft tissue remaining in the over-the-top
position will lead to further posterior positioning of the femoral tunnel with possible
blowout of the posterior wall.
Postoperative management of ACL reconstruction depends on (a) the use of slippage
resistance, stiffness, and strength of the femoral and tibial fixation devices; (b)
placement of the tibial and femoral tunnels so that roof impingement is prevented,
PCL impingement is prevented, and the tension pattern in the ACL graft matches the
tension of the native ACL; and (c) the rapidity of tendontunnel healing. Brace-free,
self-administered, aggressive rehabilitation of an impingement-free graft is safe when
slippage-resistant, high-strength, and high-stiffness fixation devices are used. Placing
the ACL graft without roof and PCL impingement eliminates the concern that the graft
might be injured by knee extension and flexion exercises. The addition of the bone
dowel in the tibial tunnel eliminates tunnel widening, increases stiffness, and snugs
the fit in the tunnel, which speeds tendontunnel healing.
COMPLICATIONS
Poor tibial and femoral tunnel placement causes catastrophic complications, including
loss of extension and anterior instability from roof impingement and loss of flexion and
anterior instability from PCL impingement. Because there are few salvage options
available to improve the success of ACL reconstruction in the setting of poorly placed
tibial and femoral tunnels, prevention is the best treatment for avoiding complications
associated with poor ACL tunnel placement. In the case of loss of extension in a stable
knee caused by mild roof impingement, a roofplasty may help regain extension and
reduce injury to the ACL graft from abrasion against the roof. Loss of flexion due to
PCL impingement is not treatable unless the ACL graft is removed. The use of cross-pin
fixation devices, not widening the notch, and the drilling of the femoral tunnel
through the anteromedial portal, all tend to place the femoral tunnel too vertical
causing PCL impingement and a loss of knee flexion and increased instability.
When using a transtibial tunnel technique, the 65-degree tibial guide prevents
impingement complications associated with poor tunnel placement by allowing
surgeons to consistently customize sagittal plane position of the tibial tunnel for all
patients regardless of the differences in roof angle anatomy and knee extension. The
use of a coronal alignment rod with the 65-degree tibial guide improves the accuracy
of tibial tunnel position in the coronal plane, thereby effectively avoiding PCL
impingement and improving rotational stability of the knee. The femoral tunnel
position is automatic with the transtibial tunnel technique. Therefore, consistent
anatomic placement of tibial tunnel in both the sagittal and coronal planes avoiding
both roof and PCL impingement results in proper femoral tunnel placement that
establishes graft tension behavior similar to the native ACL and increases the long-
term success of ACL reconstruction (Fig. 14-23).
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FIGURE 14-22
Lateral radiograph with knee in full extension demonstrating knee joint and
guide-pin position with the 65-degree tibial guide in place.
FIGURE 14-23
A: ACL graft position without PCL impingement. B: ACL graft position without
roof impingement near terminal position. C: ACL graft position with knee in
terminal extension.
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RESULTS
The use of the 65-degree tibial guide with coronal alignment rod increases the
accuracy of tibial tunnel placement in both the sagittal and coronal planes. The
increased accuracy of tibial tunnel placement avoids the complications of graft
impingement leading to better knee range of motion and stability. In addition,
because the 65-degree tibial guide was designed to reference the patient's anatomy, a
roofplasty is not required when the guide is properly used. Minimizing the notchplasty
required to position an impingement-free graft decreases the amount of graft
pretensioning required to stabilize the ACL-deficient knee and is favorable for graft
remodeling and long-term function.
RECOMMENDED READING
Cuomo P, Edwards A, Giron F, et al. Validation of the 65 degrees Howell guide for
anterior cruciate ligament reconstruction. Arthroscopy. 2006;22:70-75.
Giron F, Cuomo P, Aglietti P, et al. Femoral attachment of the anterior cruciate

ligament. Knee Surg Sports Traumatol Arthrosc. 2006;14:250-256.
Hame SL, Markolf KL, Hunter DM, et al. Effects of notchplasty and femoral tunnel
position on excursion patterns of an anterior cruciate ligament graft. Arthroscopy.
2003;19:340-345.
Howell SM. Principles for placing the tibial tunnel and avoiding roof impingement
during reconstruction of a torn anterior cruciate ligament. Knee Surg Sports Traumatol
Arthrosc. 1998;6(suppl 1):S49-S55.
Howell SM, Barad SJ. Knee extension and its relationship to the slope of the
intercondylar roof. Implications for positioning the tibial tunnel in anterior cruciate
ligament reconstructions. Am J Sports Med. 1995;23:288-294.
Howell SM, Berns GS, Farley TE. Unimpinged and impinged anterior cruciate ligament
grafts: MR signal intensity measurements. Radiology. 1991a;179:639-643.
Howell SM, Clark JA. Tibial tunnel placement in anterior cruciate ligament
reconstructions and graft impingement. Clin Orthop. 1992:187-195.
Howell SM, Clark JA, Farley TE. A rationale for predicting anterior cruciate graft
impingement by the intercondylar roof. A magnetic resonance imaging study. Am J
Sports Med. 1991b;19:276-282.
Howell SM, Gittins ME, Gottlieb JE, et al. The relationship between the angle of the
tibial tunnel in the coronal plane and loss of flexion and anterior laxity after anterior
Howell SM, Lawhorn KW. Gravity Reduces the Tibia When Using a Tibial Guide that
Targets the Intercondylar Roof. Am J Sports Med. 2004;32:1702-1710.
Howell SM, Roos P, Hull ML. Compaction of a bone dowel in the tibial tunnel improves
the fixation stiffness of a soft tissue anterior cruciate ligament graft: an in vitro study
in calf tibia. Am J Sports Med. 2005;33:719-725.
Karchin A, Hull ML, Howell SM. Initial tension and anterior load-displacement behavior
of high-stiffness anterior cruciate ligament graft constructs. J Bone Joint Surg Am.
2004;86-A:1675-1683.
Khalfayan EE, Sharkey PF, Alexander AH, et al. The relationship between tunnel
placement and clinical results after anterior cruciate ligament reconstruction. Am J
Sports Med. 1996;24:335-341.
Lawhorn KW, Howell SM. Scientific justification and technique for anterior cruciate
ligament reconstruction using autogenous and allogeneic soft-tissue grafts. Orthop Clin
North Am. 2003;34:19-30
Loh JC, Fukuda Y, Tsuda E, et al. Knee stability and graft function following anterior
cruciate ligament reconstruction: comparison between 11 o'clock and 10 o'clock
femoral tunnel placement. 2002 Richard O'Connor Award paper. Arthroscopy.
2003;19:297-304.
Matsumoto A, Howell SM, Liu-Barba D. Time-related changes in the cross-sectional

area of the tibial tunnel after compaction of an autograft bone dowel alongside a
hamstring graft. Arthroscopy. 2006;22:855-860.
Simmons R, Howell SM, Hull ML. Effect of the angle of the femoral and tibial tunnels in
the coronal plane and incremental excision of the posterior cruciate ligament on
tension of an anterior cruciate ligament graft: an in vitro study. J Bone Joint Surg Am.
2003;85-A:1018-1029.
Singhatat W, Lawhorn KW, Howell SM, et al. How four weeks of implantation affect the
strength and stiffness of a tendon graft in a bone tunnel: a study of two fixation
devices in an extra-articular model in ovine. Am J Sports Med. 2002;30:506-513.
> Table of Contents > Part Three - Ligament Injuries and Instability > 15 - Patellar Tendon
Graft Harvesting
15
Patellar Tendon Graft Harvesting
David A. McGuire
Stephen D. Hendricks
The patellar tendon graft is indicated in most patients undergoing reconstruction of a
torn anterior cruciate ligament (ACL). Situations in which an autogenous patellar
tendon may be contraindicated include the multiply operated knee where reharvest of
previously harvested patellar tendon would be needed, patients with an arthritic
patellofemoral joint, or patients with previous patellar trauma such as a fracture. In
addition, individuals whose occupation requires repetitive kneeling or working on their
hands and knees may pose a relative contraindication. In these patients, other graft
sources such as hamstring tendons or allografts should be considered.
Certain preoperative steps should be taken when an autogenous patellar tendon graft
is considered. Radiologic imaging will help identify those patients with a degenerative
patellofemoral joint or significant patellar baja or alta. If patella alta is identified,
there are several techniques available to deal with a graft that is longer than
required. The knee also should be examined for any signs of patella maltracking or
instability. A suitable graft table and required instrumentation should be available to
prepare the graft efficiently for fit and insertion ease.
SURGERY
Patient Positioning
The patient is positioned supine with the leg hung free. A standard arthroscopy prep
and drape of the knee is used. A tourniquet is placed around the thigh, and a leg
holder is placed at the level of the tourniquet. Anesthesia is typically general,
although an epidural is an alternative.
Technique
The bone patellar tendon bone (BTB) graft can be harvested through two small vertical
incisions (Fig. 15-1). The vertical incisions are cosmetic, and may lead to less
morbidity and pain than a single long incision. Because the tibial drill guide will be
positioned through the tibial harvest incision, the location of this incision is important
to the placement of the tibial tunnel. With this in mind, the tibial incision begins 2 cm
distal to the joint line and 1 cm medial to the tibial tubercle and extends 2.5 cm
distally. The patellar incision begins at the distal pole of the patella and extends 2.5
cm proximally over the midline of the patella. Although we prefer the two-incision
technique, a single skin incision can be used.
The tibial bone plug is harvested first. Lateral retraction of the skin will allow clear
visualization of the tendon attachment site. The central 10 mm of the patellar tendon
attachment is measured, and the medial/lateral borders are marked with
electrocautery (Fig. 15-2). Using a stoppered plunge blade (Plunge Blade, Linvatec,
Largo, FL), the tibial bone plug is created (Fig. 15-3). With the saw blade angled to
approximately 45 degrees, the cuts are made to a depth of 8 mm (Fig. 15-4). The
stoppered plunge blade will limit the cut depth to 8 mm and therefore eliminate the
risk of overpenetration. Using a side-to-side rocking motion, the side cuts are carried
distally for 2.5 cm. A horizontal saw cut is made to connect the distal ends of the
parallel side cuts.
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FIGURE 15-1
A: The BTB tendon can be harvested through two 1-in incisions. The two small
incisions are more cosmetic than one large one. B: Incisions closed and healed.
FIGURE 15-2
The medial and lateral borders of the patellar tendon tibial attachment are
measured and marked with electrocautery. We prefer electrocautery over a pen
because pen marks can become smeared and difficult to read.
FIGURE 15-3
The plunge blade has a stopper 8 mm from the saw blade teeth, preventing
penetration of the saw blade beyond that depth.
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FIGURE 15-4
Angling the saw blade during harvest will produce a trapezoidal bone plug.
Beginning distally at the tibial bone plug and working proximally toward the patella, a
double-bladed tendon harvester (Parasmillie meniscotome, Linvatec, Largo, FL) is used
to incise both sides of the graft simultaneously (Fig. 15-5). The fixed width of the
blades (10 mm) helps maintain a constant graft width throughout tendon harvest. The
tendon harvester is advanced proximally until the harvester tips make contact with the
distal pole of the patella. The tips of the harvester should be visible through the
patellar incision.
Using the tips of the harvester as a guide, the medial and lateral margins of the
patellar bone plug are marked with electrocautery (Fig. 15-6), generating a patellar
bone plug width of 11 mm. Beginning distally at the marked sites, use the stoppered
plunge blade to create the side cuts for the patellar bone plug. With the blade angled
to 45 to 60 degrees, the saw cuts are made to a depth of 8 mm (Fig. 15-7). Advance
the cuts proximally until a bone plug length of 25 mm is obtained. Using a horizontal
saw cut, connect the proximal termination of the two side cuts (Fig. 15-8).
Attention is directed to the tibial bone plug. Using a Â¼-inch osteotome, remove the
tibial bone plug from the tibia (Fig. 15-9). Scissors may be needed to cut any
remaining soft tissue connections.
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Grasp the tibial bone plug with a Kocher, and advance the bone plug proximally
through the tibial incision and out the patellar incision (Fig. 15-10). Using an
osteotome, remove the patellar bone plug. Again, scissors may be needed to remove
any remaining soft tissue connections and completely free the graft. Carefully transfer
the graft to a back table for preparation.
FIGURE 15-5
The Parasmillie meniscotome cleaves both the medial and lateral borders of the
patellar tendon simultaneously, ensuring a constant graft width along the entire
length of the harvest.
FIGURE 15-6
The medial and lateral borders of the patellar bone plug are measured from the
tips of the harvester. Marking bone plug borders in this manner ensures that all
tendon fibers attached to the tibial bone plug are also attached to the patellar
bone plug.
FIGURE 15-7
When in use, the stopper prevents the blade from producing cuts with variable
depths. A bone plug defect with varying cut depths may lead to stress risers and
patellar fracture.
FIGURE 15-8
The proximal ends of medial and lateral saw cuts are connected with a short
horizontal saw cut. To avoid creating stress risers, do not allow the horizontal cut
to extend beyond and therefore intersect either of the two side cuts.
Using a motorized burr, contour the corners of the patellar defect (Fig. 15-11). The
bone plug is trimmed to approximate a trapezoid shape and sharp edges are contoured
(Fig. 15-12). Save the bone chips created during sizing. The bone plugs are sized with
the aid of a sizing tube to ensure passage within the tunnels (Fig. 15-13). Typically,
the bone plugs are sized to 10 and 11 mm. A single hole is drilled in each bone plug. A
size 0 Ethibond suture is placed in the smaller bone plug (Fig. 15-14), which will
eventually be positioned within the femur. A 24-gauge flexible wire suture is placed in
the larger bone plug. Fill the patellar defect with the bone chips saved during graft
preparation. The patella defect is closed.
FIGURE 15-9
The tibial bone plug is pried loose with a Â¼-in osteotome. Pry the bone plug
loose by hand; do not use a mallet.
FIGURE 15-10
Once removed, the patellar bone plug is grasped with a Kocher and advanced
proximally.
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FIGURE 15-11
The sharp corners of the patella defect should be contoured with the aid of a
motorized burr. Failure to contour the defect corners can increase the risk of
postoperative patella fracture.
FIGURE 15-12
The bone plugs are trimmed into a trapezoidal shape. When inserted into the
tunnel, a trapezoidal plug leaves space for the advancing interference screw.
FIGURE 15-13
A graft table is useful when preparing the graft. Using the table, the graft can be
sized, the length measured, and holes drilled for the sutures used during graft
insertion and tensioning.
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FIGURE 15-14
A size 0 Ethibond suture is placed in the smaller bone plug, and a wire suture is
placed in the larger bone plug. The larger of the two bone plugs will be oriented
within the tibial tunnel. The wire suture cannot be lacerated by the passing screw
during fixation.
Pearls and Pitfalls

Pearls and pitfalls are outlined in Table 15-1.
TABLE 15-1. Pearls and Pitfalls of Patellar Tendon

Graft Harvesting
Pitfall Problem(s) Significance Tips

Single long Less cosmetic, increased Use two small incisions.
incision difficulty healing, The proximal one at the
increased risk of distal portion of the patella
infection, increased and the distal one medial
proprioception loss. to the tibial tuberosity.
Excessive bone Increased risk of patellar Use 25 mm maximum

plug length fracture. length; shorter lengths may
be used and matched with
shorter length interference
screws.
Stress risers Increased risk of patellar Use plunge saw blade with
during bone fracture caused by saw depth stop and rocking
plug creation cuts (kerfs) beyond the technique to prevent stress
corners of the removed riser creation.
plug or kerfs of uneven
depth.
Square corners Increased risk of patellar Round corners with rotary

in bone plug fracture. shaver.
defects
Empty patellar Prolonged regrowth and Bone graft defect with
defect associated pain and tibial tuberosity chips or
patellar weakness beyond from cored bone removed
the ACL remodeling from tibial tunnel with
interval. coring reamer (Trephine).
Tendon fibers Cross-cut or unattached Use a Parasmillie

to bone plug fibers create reduction in meniscotome that, once
mismatch connected width of fibers the distal plug is created,
resulting in weaker graft cleaves fibers and
with increased risk or identifies connected region
graft failure. of patellar tendon bone
plug harvest site.
Bone plug graft Increased difficulty Round ends with rongeur

tunnel inserting graft to desired and confirm graft size with
mismatch location. tunnel diameter-matched
sizing tube.
Bone plug Increased difficulty Use graft table with drill

trapping from inserting graft to desired guide to place to pull hole
tilted insertion location because suture at Â¼ to 1/8 distal to the
hole is too distal in the proximal end of the
femoral plug. femoral plug (Fig. 15-15).
Interference Increased risk of fracture Shape bone in trapezoid
screw insertion potential of bone plug or configuration by angling
torque is too bioabsorbable screw and the plunge blade during
high metal screw auguring of insertion to 60 degrees
cancellous material from vertical during plug
during initial insertion. harvest. Notch the tunnel
wall for screw insertion or
tap it.
Interference Increased risk of fixation Use next larger screw size

screw insertion failure. or use a second screw of
torque is too same size.
low
Patella baja Distal bone plug hangs Keep T tunnel between 55

out of the tibial tunnel and 60 degrees. Drill the F
precluding secure tunnel 5 mm deeper to 30
interference screw mm for its 25mm plug or
fixation. fold the distal plug over
onto itself to shorten the
LOA (Fig. 15-16).
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FIGURE 15-15
The proximal end of the bone plug should be rounded to facilitate ease of
insertion and the draw tunnel placed less than one quarter the length of the plug
toward the same end to prevent tilting-produced trapping at the femoral tunnel
distal entrance during graft insertion.
The patellar tendon harvest described here is part of an overall ACL reconstruction
and, therefore, the postoperative management and rehabilitation program are
primarily dictated by the circumstances of that procedure. However, some special
management and rehabilitation considerations should be taken into account when
using a patellar tendon autograft. Exercises that include kneeling, squatting, jumping,
or extension against resistance should be avoided. The increased stress to the patella
caused by these exercises may lead to patellar fracture.
COMPLICATIONS
There are several notable complications associated with BTB autograft harvest:
infrapatellar contracture syndrome (2,4), patella infera, patellar tendon rupture (1),
quadriceps insertion rupture, and patella fracture (2,3). Although these complications
are significant and may be debilitating to the patients in the relatively few incidences
reported, their occurrence can usually be avoided.
Patella fracture, although uncommon, can occur following autogenous patellar tendon
harvest (Fig. 15-17). Fracture is typically the result of stress risers caused by saw cuts
beyond the corners of the removed plug (Fig. 15-18), cuts made too deeply and
unevenly into the patella (Fig. 15-19), or the sharp corners of the trapezoid-shaped
bone plug defect. A lack of bone grafting also prolongs the risk of patellar fracture
beyond the rehabilitation and remodeling period for the ACL graft.
FIGURE 15-16
The distal plug of the BTB graft may be rolled over on itself to shorten the length
between the plugs if it protrudes significantly distally. If this procedure is used,
the distal bone plug may need resizing for it to fit properly in the tibial tunnel.
Once sized, suturing the configuration together will ease graft insertion,
tensioning, and fixation.
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FIGURE 15-17
A: Patellar fracture postoperatively. B: Patellar fracture reduced with ASIF

screw.
FIGURE 15-18
A: Failure to eliminate stress risers can increase the incidence of postoperative

stress risers. Intersection of the saw kerfs is a common stress riser. B: If
produced, these intersections should be rounded with the aid of a motorized
burr.
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FIGURE 15-19
Cuts made too deeply risk damage to retropatellar surface and longitudinal
fractures.
To reduce the risk of cutting too deeply into the patella, a stoppered plunge saw blade
(Linvatec, Largo, FL) should be used. With such a stopper, cuts are limited to a depth
of 8 mm and, as a result, stress risers from this source are moderated. In addition,
kerfs are of uniform depth and stress risers from this source are eliminated. The kerfs
must not extend beyond the ends of the graft, also helping avoid injury to the
retropatellar surface. The use of these measures also avoids injury to the retropatellar
surface and potential interruption of the quadriceps tendon insertion.
Another patellar tendon harvest pitfall arises when the tibial and patellar bone plugs
are offset and as a result not all tendon fibers are attached to both bone fragments
(Fig. 15-20). The width of the BTB graft is usually 10 mm, although 9 or 11 mm widths
are feasible alternatives depending on patient size. With a 10-mm width, the
corresponding bone plugs are 10 mm from the tibial tuberosity and 11 mm from the
patella. This is a technical variation that simplifies insertion. The 10-mm
â€œfemoralâ€• end of the graft slides easily through the 11-mm tibial tunnel. The
graft is weaker if the fibers are offset and all fibers are not attached to both bone
fragments (see Fig. 15-20A). It is possible to have a mismatch, even with the central
one third of the patellar tendon completely exposed by
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a 4-inch incision. The mismatch can also occur if both of the bone plugs are harvested
first and either of the plugs is offset medial or lateral to one another (see Fig. 15-20B).
The solution to this problem is to harvest the tibial bone plug first, the tendon fibers
second, and the patellar bone plug last. As long as the tibial bone plug is located
centrally on the tibial tuberosity and the graft fibers are cleaved vertically, then
simply matching the distal portion of the patellar tendon bone plug with the separated
fibers will eliminate a mismatch. A double-bladed harvester (Parasmillie meniscotome,
Linvatec, Largo, FL) that cleaves both sides of the patellar tendon simultaneously
helps maintain evenly spaced and parallel cuts, which lead to better and more
consistent grafts.
FIGURE 15-20
A: If the bone plugs are harvested prior to cleaving the tendon, any
medial/lateral offset in the plugs will result in a portion of the graft fibers only
being attached to one bone plug. B: This illustration of a cross-cut mismatch
depicts the relative fiber width which is defined by those fibers attached to both
plugs and in this case it is significantly less than the width of the bone plugs. To
avoid these complications, the tibial bone plug should be harvested first, the
graft second, and the patellar bone plug last.
FIGURE 15-21
Bone chips removed from the tibial defect with a rongeur.

FIGURE 15-22
Bone grafting with chips inserted into the patellar defect.
Bone grafting can be accomplished by filling the patellar defect with bone chips
removed from the bone plugs on the end of the graft during the shaping and sizing or
from the tibial defect area (Figs. 15-21 and 15-22). Since we have begun using a coring
reamer (Trephine, Smith & Nephew, Andover, MA) to make the tibial tunnel, we
extract a core of the patient's bone in every case (Fig. 15-23). The cored bone plug is
an excellent source of bone graft material that may be shaped to fit the patellar and
tibial defects exactly. These solid plugs seem to reduce the long-term pain in our
recent patient population when compared with patients with no grafting or chip bone
grafting.
FIGURE 15-23
Bone plug removed with coring reamer ready to be shaped into graft for patellar
and tibial graft harvest defects.
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REFERENCES
1. Bonamo JJ, Krinick RM, Sporn RM. Rupture of the patellar ligament after use of
its central third for anterior cruciate reconstruction: a report of two cases. J Bone
Joint Surg. 1984;66-A:1294-1297.
2. Graf B, Uhr F. Complications of intra-articular anterior cruciate reconstruction.

Clin Sports Med. 1988;7(4):835-848.
3. McCarroll JR. Fracture of the patella during a golf swing following

reconstruction of the anterior cruciate ligament: a case report. Am J Sports Med.
1983;11(1):26-27.
4. Rosenberg TD, Franklin JL, Baldwin GN, et al. Extensor mechanism function
after patellar tendon graft harvest for anterior cruciate ligament reconstruction.
Am J Sports Med. 1992;20(5):519-526.
> Table of Contents > Part Three - Ligament Injuries and Instability > 16 - Anterior Cruciate
Ligament Reconstruction with Autogenous Hamstring Tendon Autografts and EndoButton
Femoral Fixation
16
Anterior Cruciate Ligament Reconstruction
with Autogenous Hamstring Tendon
Autografts and EndoButton Femoral Fixation
Roger V. Larson
Injuries to the anterior cruciate ligament (ACL) are common and appear to be
increasing. The goal of treating an ACL-insufficient knee is to provide functional
stability to prevent recurrent injuries that may lead to subsequent damage to the
menisci and articular surfaces. Individuals of any age who wish to remain active in
jumping and pivoting sports can become candidates for ACL reconstructive surgery.
The success of ACL reconstruction depends on strict adherence to surgical principles,
including the selection of a graft of adequate strength and stiffness, graft placement
that avoids overstraining and bony impingement, and graft fixation that provides
initial strength and stiffness to allow early rehabilitation (5).
Historically, many tissues have been used to serve as ACL substitutes, including
autografts, allografts, and synthetic materials. The most common grafts currently used
for ACL reconstruction include autogenous bone patellar tendon bone grafts as well as
those of quadrupled hamstring tendons. Allografts are being used with increasing
frequency. Both patellar tendon and hamstring autogenous grafts
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have been shown to be effective in restoring normal anterior knee laxity when used
correctly. The use of hamstring tendons offers the advantage of avoiding the extensor
mechanism for graft harvest. This is of particular importance in patients with pre-
existing patellofemoral disease as well as in situations of revision surgery where the
patellar tendon has been used previously. Hamstring tendon grafts are also indicated
when cosmesis is of more than a casual concern. There are no absolute
contraindications to the use of hamstring tendon autografts for ACL reconstructive
surgery with the exception of their unavailability due to previous harvest.
The use of semitendinosus and gracilis tendon autografts for reconstruction of the ACL
has been well established (3,8,19). When both the semitendinosus and gracilis tendons
are harvested and doubled, the combined four-stranded graft offers several potential
advantages over other commonly used autografts such as patellar tendons. The
potential advantages of hamstring tendon autografts include increased strength,
stiffness characteristics more similar to a normal ACL, a large surface area for graft
revascularization or nutrient diffusion, a predictable means of tensioning, and
adaptability to precise positioning (15).
When the semitendinosus and gracilis tendons are both doubled and used as an ACL
graft, they provide a large-diameter, strong ACL substitute with a significantly greater
cross-sectional area of collagen than a 10-mm-wide patellar tendon autograft. The
ability to place a hamstring tendon graft precisely is enhanced over that of a patellar
tendon, since bone tunnels can be created in the desired locations and then
completely filled with collagen tissue. When using patellar tendon grafts with bone
blocks on each end, it is necessary to overdrill bone tunnels to fit the bone block.
Interference fit screws are frequently used for fixing patellar tendon grafts, and the
use of interference fit screws can distort the position of the collagen portion of the
graft. The ability of a multistranded graft to obtain nutrition by either diffusion or
vascular ingrowth is enhanced over that of a solid graft due to the increased surface
area and smaller depth of penetration needed for either revascularization or nutrient
diffusion.
Another potential advantage of using hamstring tendons for ACL reconstruction is that
the resulting graft tissue has stiffness characteristics that are more similar to a normal
ACL, particularly when compared with a patellar tendon autograft (20). The stiffness
characteristics at the time of implantation, however, depend on the type of fixation
used (9,27,29). Newer techniques of fixing hamstring tendon autografts have
alleviated what was once considered a weak link providing initial fixation comparable
to or superior to the fixation of bone tendon bone grafts (17,20,32). Soft-tissue grafts
such as hamstring tendons also allow for more precise tensioning of the grafts, making
overconstraint of the joint less likely. It has also been shown that the process of
tendon healing in a bone tunnel occurs relatively early, and thus with improved initial
fixation rehabilitation protocols do not need to be significantly different from those
used with bone tendon bone grafts (25). It also has been shown by histology and
biochemical analysis that post implantation, semitendinosus grafts undergo changes in
collagen crimp pattern, cell type, glycosaminoglycan composition, and collagen
crosslinking, which suggest a structural change toward that of a normal ACL (14).
The most compelling reason to consider hamstring tendon autografts for ACL
reconstruction, however, is the decreased surgical morbidity associated with this
procedure. With the use of hamstring tendon autografts, problems of motion and
patellofemoral arthrosis are rare (26,28). Several investigators have demonstrated
equal success rates when using this technique compared with the use of patellar
tendon autografts (1,18,21). These studies, when taken in combination, tend to show
increased patient satisfaction with the use of hamstring tendons and slightly, although
not significantly, tighter knees when reconstructed with patellar tendon grafts
(4,6,7,13,22,24).
Before embarking on surgical treatment of ACL insufficiency, it is imperative to
document the abnormal laxity and to obtain radiographs that clearly demonstrate the
bony anatomy. The diagnosis of ACL insufficiency can usually be suspected by taking a
careful history of the injury and the subsequent symptomatology of the knee. The
diagnosis can usually be confirmed by physical examination. In the acute situation,
physical examination may be difficult and subsequent examinations to confirm the
diagnosis must be planned. The most sensitive test for determining ACL laxity is the
Lachman test, in which an anterior force is applied to the tibia with the knee flexed
approxiately 25 degrees. This examination when compared with the normal knee can
be diagnostic in up to 95% of cases, including acute injuries. Tests that elicit the
â€œpivot shift phenomenonâ€• are helpful in grading the ACL laxity, but can seldom
be performed adequately in the acute situation.
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An arthrometer can be helpful in demonstrating ACL laxity and documenting the
extent of abnormal laxity. It has been shown that in normal individuals there is a 95%
chance that there will be a side-to-side difference of less than 2 mm in ACL laxity. If 3
mm or greater of side-to-side difference can be demonstrated with an arthrometer, it
can be assumed that the ACL has been injured.
Magnetic resonance imaging (MRI) when performed in the proper plane can be very
accurate in diagnosing an injury of the ACL. With careful study of MRI images, partial
tears of the ACL can also be identified when either the posterolateral or anteromedial
band of the ACL may remain intact. The MRI may also show a typical bone bruising
pattern involving the posterior aspect of the lateral tibial plateau and the mid anterior
portion of the lateral femoral condyle. This bruising pattern in the â€œimpact zoneâ€•
is highly suggestive of a torn ACL. The MRI is also important in delineating associated
pathology which may influence whether surgical intervention is undertaken or the
timing of such intervention.
It is also necessary before embarking on surgical intervention to review the technical

points that are important to a successful outcome. As with any surgical procedure,
several technical points must be respected to predictably obtain a successful outcome
when using hamstring tendon grafts. These include the successful harvest and
preparation of the grafts, correct tunnel placement, secure graft fixation, and
rehabilitation appropriate for the procedure performed.
SURGERY
Principles
Graft Harvest and Preparation: Technical Points
The potential difficulties in harvesting the hamstring tendons and the variability in
tissue obtained were once major detriments to the routine use of these grafts. It was
also once necessary to obtain considerable graft length to directly fix grafts
extraosseously. With newer endoscopic techniques, it is usually only necessary to
harvest 22 to 24 cm of tendon, and this amount of tendon can be consistently
harvested. The quadrupled graft is generally from 7 to 9 mm in diameter, which is
adequate for ACL substitution. It is important to be aware of the anatomy of the
semitendinosus and gracilis tendons, particularly of the routinely encountered bands
of tissue that extend from the inferior surfaces of each tendon to the medial head of
the gastrocnemius (16, 23) (Fig. 16-1). These bands must be identified and sectioned
before passing a tendon stripper to ensure that the stripper follows a direct course to
the muscle
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belly. In cases in which the semitendinosus tendon is of exceptionally high quality and
length or where a gracilis tendon is inadequate, a quadrupled semitendinosus tendon
graft can be used with some modification of fixation technique (2).
FIGURE 16-1
The semitendinosus and gracilis tendons must be isolated and separated before
harvesting. Extraneous bands of tissue that often connect to the medial head of
the gastrocnemius must be identified and released before passing a tendon
stripper to avoid deflecting the stripper and cutting the tendon short of its
maximal length.
Once the tendons have been harvested and sized, it is important to remove muscle
tissue from each graft and secure each end of each graft with a nonabsorbable suture.
The use of a supplemental suture technique allows for the fixation of every fiber of
the graft and for important secondary fixation. It has been shown that slippage under
washers, or against interference fit screws, can be considerably reduced by adding a
suture technique to the primary source of fixation.
Tunnel Placement
For a successful ACL reconstruction with hamstring tendons or any other graft, the
graft must be placed so that it does not overstrain as the knee passes through a full
range of motion and so that it does not impinge against the bony anatomy of the
intercondylar notch, particularly in terminal extension. This can usually be
accomplished by placing the tibial tunnel in a central position and the femoral tunnel
at the extreme back of the intercondylar notch at approximately the 10:30 or 1:30
position (30) (Fig. 16-2A). This is at the point where the sidewall and roof of the notch
meet. When properly positioned, this provides for a graft that deviates from the
vertical position of the posterior cruciate ligament (PCL) at an angle of approximately
25 to 30 degrees. This placement requires careful attention to the start point of the
tibial tunnel, particularly when the femoral tunnel is to be created through the tibial
tunnel (12). When ideally placed, the ACL graft will avoid contact with the lateral wall
of the intercondylar notch and will barely touch the roof of the intercondylar notch at
full terminal extension. The proper placement will also avoid excessive impingement
with the PCL, particularly in hyperflexion. The femoral tunnel should extend to or near
the â€œover-the-topâ€• area. It is acceptable to blow out the back wall of the notch
when creating the femoral tunnel, and in fact this would ensure that the tunnel is far
enough to the back of the notch.
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With EndoButton (EndoButtonTM, Smith and Nephew, Corp., Boston, MA) fixation on
the femur, blowing out of the back wall of the tunnel does not compromise fixation
strength. The leading edge of the femoral tunnel should be chamfered to avoid a sharp
corner that could traumatize the ACL substitute (21). The proper position of the
femoral tunnel is demonstrated in Figure 16-2B.
FIGURE 16-2
The position of a femoral tunnel that corresponds to a central tibial tunnel at

10:30 or 1:30 o'clock and to the extreme back of the intercondylar notch. The
cross-hatched area noted in (A) shows the area that strains less than 5% through a
full range of motion. It is located at the extreme back of the intercondylar notch.
This area can be accessed through a tibial tunnel if the start point of the tibial
tunnel is correctly positioned (B).
The tibial tunnel should be created far enough posterior in the notch so that at full
terminal extension the graft will barely graze the superior notch but will not be
impinged or â€œguillotinedâ€• by it. It has been shown that impinged grafts remain
inflamed and their MRI signals do not return to normal (10). With extreme
impingement knee extension can be blocked and anterior graft fibers can be broken,
creating a â€œcyclopsâ€• lesion (11). The tibial tunnel should also be placed in a
position that avoids lateral notch impingement. If the femoral tunnel is to be drilled
through the tibial tunnel, the start point must be medial enough to allow drilling of
the femoral tunnel at the appropriate position. This usually requires starting the tibial
tunnel within the fibers of the medial collateral ligament. It is appropriate when
possible to fit the tunnels to the anatomic notch. Routine notchplasty should be
discouraged, since with notchplasty the exposed cancellous surfaces often form
fibrocartilage and eventual reformation of the anatomic notch.
Graft Fixation Options

The goal of ACL reconstruction is to provide an ACL substitute that will reproduce as
nearly as possible the functional properties of the native ACL. For the graft to perform
this function it must be placed in a near isometric or â€œphysiometricâ€• position,
and it must have the strength and stiffness to mimic ACL function. During the first 6 to
12 weeks, before biologic healing of the grafts to the bone tunnels, the behavior of
the graft depends not only on the material properties of the graft being used but also
on the method of graft fixation used. The graft/fixation construct must be strong
enough to avoid failure with tensile loading, and it must be stiff enough to restore the
load displacement response of a normal ACL and secure enough to resist slippage with
cyclic loading.
The important parameter to consider in fixation strength is yield load. This is the point
at which plastic deformation begins and functional failure occurs (17). Most fixation
devices used for the femoral fixation of hamstring tendon grafts have yield loads of
between 350 to 800 newtons (N). The newer-generation fixation devices may increase
this strength to 1,200 N. Techniques of femoral fixation that involve rigid cross-pin
fixation provide the greatest initial strength.
Tibial fixation of hamstring tendon grafts is a more problematic consideration than

femoral fixation. Initial fixation yield strength ranges from approximately 350 N for an
interference fit screw to 1,160 N for tandem screws and washers. Graft slippage is an
additional consideration, particularly with tibial fixation methods.
The initial stiffness of an ACL substitute is highly dependent on the fixation method
used. Howell has pointed out that the stiffness of fixation methods is from four to 40
times less than that of grafts used. Increasing the stiffness of an ACL replacement
would best be achieved, therefore, by selecting fixation methods with higher stiffness
and not by shortening or increasing the cross-sectional area of the grafts used (33).
The stiffness of a doubled semitendinosus and gracilis tendon complex alone is
approximately 1,000 N/mm. The stiffness of various fixation devices of hamstring
tendons varies from approximately 25 to 230 N/mm. A normal ACL has a stiffness of
approximately 200 N/mm.
Slippage of grafts from their point of fixation during cyclic loading is a major early
problem that can compromise results (31). The slippage around interference fit screws
is considerably greater than that which occurs with a cross-pin technique. The slippage
around an interference fit screw during cyclic loading is also quite dependent on bone
quality, which can be extremely variable in a patient population. When using screw
and washer fixation of soft-tissue grafts, slippage can be considerably reduced by
utilizing an additional suture technique to secure the graft beneath the soft-tissue
washer. Supplemental suture fixation for grafts fixed with interference fit screws is
highly recommended.
Hamstring Graft Fixation

It is important in fixing hamstring tendon grafts to fix every fiber of the graft. This
cannot be done with devices such as interference fit screws alone. It is also important
to be able to tension all limbs of the graft to a desired level. To fix every fiber of the
graft on the femur it is important to cross-pin the looped end of the graft. This can be
done by a rigid cross-pin fixation or by a device such as an EndoButton. The second-
generation EndoButton with attached heavy-duty continuous loop provides the ability
to loop each fiber of the graft and gives strength and stiffness adequate to withstand
early rehabilitation techniques.
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The preferred method for tibial fixation of hamstring tendon grafts is to use a suture
technique through each end of each graft. The sutures are then tied around a post,
before direct fixation of the grafts, by a soft-tissue washer. To better stabilize the
graft within the tibial tunnel and closer to the joint line and to increase construct
stiffness, an accessory bioabsorbable screw is used in the tibial tunnel anterior to the
grafts. The interference fit screw is placed after tensioning and fixation have been
accomplished extraosseously by sutures and washer.
Technique: ACL Reconstruction with Hamstring Tendon

Autografts
Setup
The setup for this procedure includes a tourniquet placed as proximal as possible on
the thigh. The thigh holder should be low profile, particularly anteriorly, so as not to
interfere with the passage of a guide pin through the anterior thigh (Fig. 16-3). It is
our current practice to exsanguinate the limb and inflate the tourniquet at the
beginning of the case; however, diagnostic arthroscopy and any meniscal work as well
as hamstring tendon harvest could be accomplished before inflation of the tourniquet,
if desired. The operating table should have a break at the level of the knee joint so
that the knee can be easily flexed to 90 degrees.
At the beginning of the case, diagnostic arthroscopy can be performed if necessary for
precise diagnosis. Any meniscal work or joint debridement can be done at that time. It
is important to make anteromedial and anterolateral portals as close as possible to the
patellar tendon to allow easy and direct access to the intercondylar notch (Fig. 16-4).
If the diagnosis of ACL insufficiency is obvious with preoperative planning, the graft
preparation can be completed prior to starting the arthroscopic portion of the case.
Graft Harvest
A skin incision is next made vertically overlying the insertion of the pes anserinus (see
Fig. 16-4). This same incision is used as a start point to create a tibial tunnel. The
incision starts approximately 3 cm distal to the anteromedial portal and slightly medial
to it. The incision needs to extend only approximately 2 to 3 cm. Once the
semitendinosus and gracilis tendons have been identified, they are isolated and
released together from their tibial insertion (Fig. 16-5A). The grafts are then flipped
over; and from the under surface, the interface between the two tendons can be
identified, allowing separation. Each tendon is then separately clamped with a Kocher
clamp. The semitendinosus tendon is usually harvested first. Extraneous bands along
the inferior surface are identified and released. It is helpful to cross-clamp the tendon
and pull quite strongly on it to break up some adhesions and to deliver any extraneous
bands into the wound for sectioning under direct vision. It is usually possible to pass a
fingertip up the tendon to the level of the muscle belly to ensure that all bands have
been sectioned. It is also helpful to feel a â€œbounceâ€• from the muscle belly when
pulling on the tendon. The medial calf can also be observed while pulling on the
tendon to be sure the medial gastrocnemius is not being â€œdimpled.â€•
An arthroscopic grabber is then passed through a closed-end tendon stripper to deliver

the graft through the tendon stripper. The graft is then cross-clamped and held under
tension while the tendon stripper is passed in line with the direction of the tendon to
the muscle belly for harvesting (see Fig. 16-5B). The gracilis tendon is then similarly
harvested.
FIGURE 16-3
Proper setup requires a proximal tourniquet, a low-bulk thigh holder, and a table
that bends at the knee to allow flexion to 90 degrees.
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FIGURE 16-4
The incisions necessary to perform this procedure include anterolateral and

anteromedial arthroscopy portals adjacent to the patellar tendon. A tibial
incision begins approximately 3 cm distal to the anteromedial portal and extends
2 or 3 cm. This incision allows for harvesting of the semitendinosus and gracilis
tendons and for creation of the tibial tunnel.
FIGURE 16-5
The semitendinosus and gracilis tendon harvest begins by first isolating the two
combined tendons (A). The tendons are then released from their tibial
attachments and inverted to facilitate separation. Harvesting is performed with a
closed-end tendon stripper (B).
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Graft Preparation
Once the tendons have been harvested, they are shortened to approximately 22 to 24
cm in length, depending on the size of the patient's bones. It is almost always possible
to obtain this length of tendon. Muscle tissue is then scraped from the graft with
either a curette or a scalpel, and each end of each graft is then tagged with a Bunnell
or whipstitch of No. 2 nonabsorbable suture (Fig. 16-6A). The looped grafts are then
passed through a sizing tube to determine the size of tunnels to be created (see Fig.
16-6B). The prepared grafts are then soaked in an antibiotic solution until used.
Notch Preparation
The intercondylar notch is prepared by first removing a significant portion of the
anterior fat pad to allow proper visualization. This allows for the arthroscope to be
â€œbacked outâ€• enough to get a perspective on the notch and ACL. The stump of
the ACL is generally removed; however, if either the anteromedial or posterolateral
band of the ACL is intact, that intact portion can be maintained and a reconstruction
performed of the damaged band. The notch debridement is usually performed using a
basket forceps, a curette, and a motorized shaver. The intercondylar notch
debridement needs to extend to the extreme posterior aspect of the notch so that the
â€œover-the-topâ€• area can be clearly defined with a nerve hook. It is usually not
necessary or advisable to perform a bony notchplasty. This is especially true in the
case of acute disruption of the ACL, where osteophytic spurring has not occurred.
When a notchplasty is created it is not uncommon for the cancellous surfaces that are
created to rescar and reform the notch to the size that was present before
notchplasty. This often can cause problems postoperatively with graft impingement
that was not present at the completion of surgery. It is therefore best, when possible,
to adjust tunnel position to fit the anatomic intercondylar notch.
Creation of Tibial Tunnel

A tibial guide is next used to pass a guide pin from the anteromedial tibia to a point
corresponding to a central tibial attachment site (Fig. 16-7A). The intra-articular site
is just medial to the medial tibial spine and approximately 7 mm anterior to the
anterior edge of the PCL. Once the initial guide pin has been placed, it is essential to
check the position and modify it if indicated (see Fig. 16-7B). When the knee is
extended with the guide pin in place, the guide pin should move directly toward the
apex of the intercondylar notch, and at full extension it should be approximately 4 to
5 mm short of touching the superior notch (see Fig. 16-7C). If the position is not
correct, it should be modified with the use of a guide that will allow placement of a
parallel pin at the desired location (see Fig. 16-7D).
FIGURE 16-6
The hamstring tendons are cut to approximately 22 to 24 cm in length, and

muscle tissue is removed from each graft. Each end of each graft is then tagged
with a Bunnell or whipstitch of No. 2 nonabsorbable suture (A). The combined
doublelooped tendons are then passed through a sizing tube (B) so that tunnels
can be created to fit the grafts as tightly as possible.
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FIGURE 16-7
A central tibial tunnel is created by first placing a guide pin with a drill guide (A).
The position of the guide pin should be adjacent to the PCL (B), and as the knee
is extended the pin should move directly toward the apex of the notch. At
terminal extension, 4 or 5 mm of clearance should exist between the pin and the
roof of the notch (C). If the position is not ideal, a parallel drill guide can be used
to fine-tune the position (D). When the optimal position is achieved, the guide
pin is overdrilled with the appropriately sized cannulated drill (E).
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The start point of the tibial tunnel is critical since the direction of the tunnel will limit
access to the femur for preparation of the femoral socket (7). The start point must be
medial enough to provide access to the correct femoral position. This usually involves
moving the start point into the anterior fibers of the superficial medial collateral
ligament (see Fig. 16-2B). The guide should be adjusted to provide a tunnel angle of
approximately 45 degrees when viewed laterally, and it should be angled
approximately 25 to 30 degrees in the frontal plane. Once the position of the tibial
guide pin has been optimized, it is overdrilled with a cannulated drill of 7 mm. The
initial tibial tunnel is always created with a 7-mm drill and is enlarged to the final size
by the acorn drill used to create the femoral socket. We have found that this step
reduces the over enlargement of the tibial tunnel, which can potentially occur when
drilling the femoral tunnel through it.
Preparation of Femoral Socket

A socket is next created in the femur to accept the looped end of the hamstring
tendon grafts. This socket needs to be placed at approximately the 10:30 or 1:30
position and to the extreme back of the intercondylar notch. It can be created through
a properly positioned tibial tunnel. It is essential that the direction of the tibial tunnel
allow access to the extreme back of the intercondylar notch and to the 10:30 or 1:30
position. It is generally possible to do this if the tibial tunnel start point is correctly
placed.
An â€œover-the-topâ€• referencing drill guide is used to place the guide pin for the
femoral socket (Fig. 16-8A). It is best to use a femoral offset of no more than 5 mm to
ensure that the graft is to the extreme back of the intercondylar notch. It may be
preferable to use a 3-mm offset. This would often cause a â€œblowoutâ€• of the
posterior wall of the femoral socket, which is acceptable.
The guide pin is placed through the tibial tunnel and into the femur at the desired
location. This must be done with the knee flexed to approximately 90 degrees to
ensure that the guide pin can exit the exposed anterior thigh distal to the tourniquet
and drapes. The guide pin is initially passed until it engages the anterolateral femoral
cortex.
An acorn drill of the diameter predetermined to be required to accept the tendon

graft is then used to create a femoral socket from inside out to a depth of 35 mm (see
Fig. 16-8B). The 35-mm socket allows pulling the graft 25 mm into the socket and also
allows room to pull the graft initially an extra 10 mm into the socket during the
â€œflippingâ€• of the EndoButton. During the creation of the femoral socket, the
tibial tunnel is enlarged by the acorn drill to the required diameter. A 4.5-mm passing
channel is next created over the guide pin from the base of the socket to the
anterolateral femoral cortex (see Fig. 16-8C). The length of the entire tunnel is next
measured (see Fig. 16-8D). The final position of the femoral tunnel is shown in Figure
16-8E.
A continuous-loop EndoButton is next selected that will allow the looped end of the
graft to penetrate the femoral socket approximately 25 mm (Fig. 16-9). The loop
length of Endobutton is determined by subtracting 25 mm from the measured total
length of the femoral socket and passing channel. Most commonly, the continuous-loop
EndoButton selected ranges from 25 to 45 mm.
The semitendinosus and gracilis tendons are next looped through the continuous loop
of the EndoButton. Passing sutures are next attached to the EndoButton. Two sizes of
passing sutures are used, No. 5 and No. 2. This allows for pulling of the EndoButton
with the No. 5 sutures and flipping the EndoButton with the No. 2 suture (Fig. 16-10).
The knee is again flexed to the angle that was used for drilling the femoral tunnel. The
guide pin is passed through the tibial tunnel across the joint and into the femoral
tunnel and passing channel and exits through the quadriceps on the anterolateral
thigh. The passing sutures are passed through the eye of the passing pin with the
EndoButton oriented so that the No. 5 sutures enter the tunnel first. The passing
sutures are then pulled through the tibial tunnel, across the joint, and into the
femoral tunnel and out to the anterolateral thigh. The EndoButton is next pulled by
the No. 5 sutures through the tunnels to the anterolateral femoral cortex (Fig. 16-
11A,B). It is pulled until the graft â€œbottoms outâ€• into the femoral socket. The
flipping sutures are then used to â€œsetâ€• the EndoButton.
The free ends of the tendons are then pulled to secure the Endobutton on the
anterolateral femoral cortex. The â€œpassingâ€• and â€œflippingâ€• sutures are then
removed by releasing one end of each and pulling them through the thigh. If the graft
length has been properly selected there should be approximately 1 cm of each end of
each graft exiting the tibial tunnel.
Tibial Fixation
The grafts are next tensioned and fixed to the tibia by placing a low-profile screw and
washer at the distal ends of the grafts near the exit point of the tibial tunnel. Each of
the two grafts is separately tied as tightly as possible around the shaft of the tibial
screw (Fig. 16-12A). The grafts are then tensioned separately while the knee is cycled
approximately 10 times to ensure
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that there is no slack within the tunnels. The grafts, sutures, and knots are then
directly fixed with a soft-tissue washer (see Fig. 16-12B). Some fine tuning of graft
tension can be done at this point since the soft tissue washer directly fixes the graft,
sutures, and knots. This is usually done with the knee at approximately 20 degrees of
flexion.
FIGURE 16-8
An â€œover-the-topâ€• referencing drill guide with 3 to 5 mm offset (A) is used
to place the guide pin at the 10:30 or 1:30 o'clock position and to the extreme
back of the intercondylar notch. An acorn drill (B) is then used to create a socket
35 mm in depth at the desired location. A 4.5-mm drill (C) is then used to create
a passing channel through the anterolateral femoral cortex. The total depth of
the femoral tunnel and socket are then measured (D). The final position of the
femoral tunnel is at the 10:30 or 1:30 o'clock position and to the extreme back of
the notch as noted (E).
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FIGURE 16-9
The continuous-loop EndoButton provides increased strength and stiffness of the

femoral fixation when compared with the conventional EndoButton, which uses
tape and knots.
FIGURE 16-10
The completed construct (with conventional EndoButton and tape). Passing

sutures are of No. 5 for pulling and No. 2 for flipping the EndoButton.
FIGURE 16-11
The EndoButton is pulled into the femoral tunnel by the No. 5 pulling sutures,
which have been passed to the anterior thigh (A). The closed loop then delivers
the looped ends of the tendons into the femoral socket (B).
FIGURE 16-12
Tibial fixation is accomplished by placing the screw at the distal ends of the
grafts (A). The graft sutures are then tied to the post and the grafts, sutures, and
knots are directly fixed by the soft-tissue washer after final tensioning (B). To
augment tibial fixation and to increase stiffness and reduce micromotion in the
tibial tunnel, a bioabsorbable screw is next passed up the tibial tunnel anterior to
the grafts, and advanced to just short of entry into the joint.
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FIGURE 16-13
The final position of the graft (A). It is adjacent to the PCL, has excellent
clearance with the lateral wall of the intercondylar notch, and just grazes the
superior notch at terminal extension (B).
To better stabilize the grafts within the tibial tunnel, and to increase the stiffness of
the tibial fixation, a bioabsorbable interference fit screw is next selected and used in
the tibial tunnel. A screw sized 1 mm greater than the tibial tunnel drilled is passed up
the tibial tunnel anterior to the grafts and advanced to just short of entry into the
joint. The final position of the grafts is demonstrated in Figure 16-13A. The graft
avoids the lateral wall of the intercondylar notch and barely grazes the superior notch
at full terminal extension (see Fig. 16-13B).
Closure and Bracing

The tourniquet is next deflated and hemostasis is secured. The joint and wound are
both copiously irrigated. Closure is accomplished with subcutaneous and subcuticular
stitches followed by Steri-strips. Sterile dressings are applied to the wound followed by
a compressive stocking and a cold therapy device. The patient is then placed into a
hinged knee brace that is initially locked at 10 degrees of flexion. The patient will
remain in the locked knee brace for the first 6 days following surgery. No drains are
used. Prophylactic antibiotics are routinely prescribed, giving one dose when the
patient enters the operating room and one before the patient leaves the recovery
suite. In most instances further antibiotics are not administered. The patient is taken
from the operating room to recovery and in most instances is discharged to home
within 2 to 3 hours of the procedure's completion.
Pearls and Pitfalls
Setup
Proximal low-profile thigh holder
Table break at knee to allow flexion
Graft Harvest
Isolate semitendinosus and gracilis as a pair.
Invert tendons to isolate and separate.
Cross clamp and pull each tendon to expose extraneous bands.
Section all extraneous bands before advancing stripper.
Tibial Tunnel
Start medial enough to allow access to desired femoral site.
Pass provisional guide pin and observe position as knee is extended.
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Modify pin position if needed with parallel drill guide.
Drill initial tunnel 7 mm diameter. Enlarge if needed when creating femoral

socket.
Femoral Tunnel
Place guide pin at 10:30 or 1:30 position and near extreme back of notch.
Drill femoral socket to 35 mm.
Femoral Fixation
Feel EndoButton â€œbounceâ€• on femur to ensure position.
Tibial Fixation
Tie sutures for each graft separately around post.
Cycle knee before â€œpullingâ€• final tension.
Secure graft, sutures, and knots with soft tissue washer.
Place bioscrew in tunnel anterior to graft. Use size 1 mm greater than tibial
tunnel diameter.
The patients are kept in a locked knee brace for the first 6 days following surgery.
They can use crutches with weight bearing as tolerated and discontinue the use of
crutches when comfortable. In general, crutches are used for the first 10 to 14 days.
At the end of 1 week, the knee brace is opened to allow motion from 10 degrees to
unlimited flexion. Generally, at that time patients have full extension of their knee
and can easily flex to 90 degrees. The 10-degree extension block is used when weight
bearing to prevent hyperextension. The knee brace can be removed when non-weight
bearing.
The patients are generally seen in physical therapy two or three times a week for the
first 12 weeks. When other facilities are available for the patient's rehabilitation and
when no problems are encountered, formal physical therapy can be considerably
reduced. After the first week, patients can begin low-resistance stationary bicycling
out of the brace. The patients also can perform quadriceps sets, straight leg raises,
early hamstring resistance exercises, and closed-chain exercises with an elastic cord.
Bracing is discontinued at 6 weeks after surgery, when patients generally have
excellent muscle control about the knee. Other functional activities such as stair-
climbing machines and elliptical trainers are started at 6 weeks following surgery.
When the patient has reached 12 weeks postoperatively, he or she is cleared for all
activities except for running on hard surfaces, terminal knee extensions with
resistance, and sports that involve jumping and pivoting. At 6 months postoperatively,
the restrictions are removed for running and terminal knee extensions with weights. A
return to full, unrestricted sport is generally delayed until approximately 8 months
following surgery, and then begun only if the patient has regained approximately 90%
of hamstring and quadriceps strength. We continue to encourage the use of a
functional knee brace for participation in jumping and pivoting sports until 18 months
following surgery. The complete postoperative protocol is detailed in Tables 16-1, 16-
2, 16-3 and 16-4.
TABLE 16-1. Phase I-A: Physical Therapy Following

Arthroscopic Anterior Cruciate Ligament Surgery (0
to 6 Weeks)
Therapy Goals
Range of motionâ€”limiting brace locked Demonstrated independent

first 6 days, then 10 degrees to unlimited home exercise program
flexion
Passive range of motion
Weight bearing as tolerated with crutches between 0 and 5 degrees of
and brace; discontinue crutches as tolerated extension and between 115
and 125 degrees of flexion
Physical therapy will work for full passive
range of motion and patellar mobility Normal gait at 6 weeks
without a brace on level
Daily stretching out of brace for full passive
ground
extension
Stationary bike at 80 rpm
Electrical muscle stimulator used as needed
for 20 minutes
Daily low-resistance stationary biking when
range of motion allows (out of brace);
resistance as tolerated
Exercises performed in brace include

quadriceps sets, straight leg raises,
hamstrings, abductors, adductors, and
gluteals; add weights as tolerated, with the
exception of no weights on straight leg raises
Stretching of hamstrings and heel cords
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TABLE 16-2. Phase I-B: Physical Therapy Following

to 12 Weeks)
Therapy Goals
Off crutches At 8 weeks, bike with

resistance for 20
Brace worn when away from home
minutes
Range of motion in brace 10 degrees to unlimited
At 12 weeks, 0-pound
flexion
straight leg raise
Full active extension out of brace, no resistance without a quadriceps
lag
Full active flexion with resistance
Able to walk 15 to 20
Stationary biking with increasing resistance as minutes with a limp
tolerated
tolerated
Stair-climbing
Swimming kicking from the hip only, no wall push- machine, 20 minutes
offs
Normal gait without a
Physical therapy continued at least on a limited brace
basis
Upstairs step-over-
Gym weights equipment or ankle weights used for step two flights
strengthening exercises. Precaution: straight leg
raise must be limited to 5 lbs and resisted knee Sit-to-stand transfer
extension range from 90 to 45 degrees only normal
Stair-climbing machine and closed kinetic chain Full passive range of

exercises motion
Sport cord
TABLE 16-3. Phase II: Physical Therapy Following

to 24 Weeks)
Therapy Goals
No brace or crutches At 16 weeks, 5-lb straight leg

raise without a quadriceps lag
Full passive and active range of motion
At 16 weeks, stairs step-over-
More aggressive strengthening via
step for two flights ascending
cycling, stairs, closed kinetic chain
and descending
activities as tolerated
Unlimited walking without a
Patient monitors pain and swelling and
limp
reports this to physical therapist
At 24 weeks, or 6 months, 10-lb
Progressive resistive exercises
Progressive resistive exercises
straight leg raise without a
continued, straight leg raise weights
quadriceps lag
increased to 10 lbs
Stair climbing for exercise
No resisted knee extension through full
range of motion until 6 months At 24 weeks, balance on one leg
postoperatively for 2 minutes
Begin proprioception-balance training
COMPLICATIONS
Serious complications specifically related to the harvest of hamstring tendon
autografts are uncommon. Complications, however, can be associated with the
technical harvest of the hamstring tendons and also at the donor site (16). Also, as
with any ACL substitute, if tunnel placement and graft fixation are not adequate, early
failure of grafts due to overzealous rehabilitation can be encountered.
During the actual harvest of hamstring tendons, it is imperative to release all

attachments to the medial head of the gastrocnemius to ensure clear passage of the
tendon stripper to the muscle belly. If this precaution is not taken, it is possible to
obtain grafts of inadequate length to serve as an adequate ACL substitute. It is also
not uncommon to create sensory abnormalities in the distribution of the saphenous
nerve or its branches. This is usually a temporary neuroproxia, but it may take several
months for areas of decreased sensation to resolve, and it is advisable to inform the
patients of this possibility before surgery. On rare occasions patients will develop
adhesions of the posterior thigh to the healing muscle bellies. This can result in a
dimpling of the skin with knee flexion. This potential complication has been markedly
reduced with the use of tendon strippers to harvest the hamstring tendons when
compared with open harvest techniques.
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TABLE 16-4. Phase III: Physical Therapy Following
Months to 1 Year)
Therapy Goals
Aggressive strengthening exercises Able to run without a limp

and return-activity exercises
Quads and hamstrings at 90% of
(straight-line running) are initiated
uninvolved leg on a Cybex test with
Aggressive contact, pivoting, and quad-to-hamstring ratio at a
quick acceleration-deceleration minimum of 3 to 2
movements avoided until 9 months
Full muscle mass for brace fitting
following surgery
Return to unrestricted activity
If deemed appropriate, patient is
including sports
measured for a derotation brace
before starting jumping or pivoting Derotation bracing as indicated
RESULTS
The use of hamstring tendon autografts with the described technique has been used
successfully to consistently restore normal anterior laxity in ACL-insufficient knees in
both acute and chronic situations. There are several studies in the literature that
compare hamstring tendon autografts to patellar tendon autografts and demonstrate
that they are of equivalent efficacy (4,6,7,13,22,24). The use of hamstring tendon
autografts has been effective in avoiding complications, particularly motion problems
or patellofemoral pain complaints. Figure 16-14 shows the appearance of a double-
looped semitendinosus and gracilis tendon 1 year after implantation. The depicted
graft is well synovialized, and both the tibial and femoral attachments have a
â€œsplayedâ€• appearance similar to a normal ACL. The described procedure is also
cosmetically acceptable, particularly since it avoids an incision over the patellar
tendon where the skin needs to adapt to considerable motion. The incision location
below the joint line more medially on the tibia is a region of very little skin motion;
thus, the incision generally does not tend to spread. Figure 16-15 is an example of an
incision approximately 1 year following surgery in a young woman.
The importance of adhering to technical detail when performing this or any other
procedure for ACL reconstruction cannot be overemphasized. Attention to graft
preparation, tunnel placement, avoidance of notch impingement, fixation, and
rehabilitation are much more important factors in the success of ACL reconstruction
than is the origin of the autogenous graft. Although the described technique is
applicable in almost all situations of ACL insufficiency, it is particularly applicable to
those patients in whom a patellar tendon ACL reconstruction has failed, in those with
pre-existing patellofemoral disease, in skeletally immature patients, or in any other
situation in which avoidance of the extensor mechanism is desirable. The procedure is
also appropriate when cosmesis is of more than casual concern.
FIGURE 16-14
Semitendinosus and gracilis grafts 1 year after implantation. The grafts are well
synovialized. The attachment site on the tibia has a normal â€œsplayedâ€•
appearance.
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FIGURE 16-15
The skin incision 1 year after surgery. The skin incision is in an area of minimal
skin motion, and spreading of the incision is unusual. A good cosmetic result is
usually obtained.
REFERENCES
1. Aglietti P, Buzzi R, Zaccherotti G, et al. Patellar tendon versus doubled
semitendinosus and gracilis tendons for anterior cruciate ligament reconstruction.
Am J Sports Med. 1994;22:211-218.
2. Barrett GR, Papendick L, Miller C. EndoButton button endoscopic fixation

technique in anterior cruciate ligament reconstruction. Arthroscopy. 1995;11:340-
343.
3. Cho KO. Reconstruction of the anterior cruciate ligament by semitendinosus

tenodesis. J Bone Joint Surg. 1975;57A:608-612.
4. Feller JA, Webster KE. A randomized comparison of patellar tendon and

hamstring tendon anterior cruciate ligament reconstruction. Am J Sports Med.
2003;31:564-573.
5. Frank CB, Jackson DW. The science of reconstruction of the anterior cruciate
ligament (current concepts review). J Bone Joint Surg 1997;79A:1556-1576.
6. Freedman KB, D'Amato MJ, Nedeff DD, et al. Arthroscopic anterior cruciate
ligament reconstruction: a meta-analysis comparing patellar tendon and hamstring
tendon autografts. Am J Sports Med. 2003;31:2-11.
7. Gobbi A, Sanjeev M, Zanazzo M, et al. Patellar tendon versus quadrupled bone-

semitendinosus anterior cruciate ligament reconstruction: a prospective clinical
investigation in athletes. Arthroscopy. 2003;19:592-601.
8. Gomes JLE, Marczyk LRS. Anterior cruciate ligament reconstruction with a loop
or double thickness of semitendinosus tendon. Am J Sports Med. 1984;12:199-203.
9. Hamner DL, Brown CH, Steiner ME, et al. Hamstring tendon grafts for
reconstruction of the anterior cruciate ligament: biomechanical evaluation of the
use of multiple strands and tensioning techniques. J Bone Joint Surg. 1999;4:549-
557.
10. Howell SM, Berns GS, Farley TE. Unimpinged and impinged anterior cruciate
ligament grafts: MR signal intensity measurements. Radiology. 1991;179:639-643.
11. Howell SM, Taylor MA. Failure of reconstruction of the anterior cruciate
ligament due to impingement by the intercondylar roof. J Bone Joint Surg Am.
1993;75:1044-1055.
12. Howell SM, Wallace MP, Hull ML. Evaluation of the single-incision arthroscopic
technique for anterior cruciate ligament replacement (a study of tibial tunnel
placement, intraoperative graft tension, and stability). Am J Sports Med.
1999;27:284-293.
13. Jansson KA, Linko E, Sandelin J, et al. A prospective randomized study of

patellar versus hamstring tendon autografts for anterior cruciate ligament
reconstruction. Am J Sport Med. 2003;31:12-18.
14. Lane JG, McFadden P, Bowden L, et al. The ligamentization process: a four-
year case study following ACL reconstruction with a semitendinosus graft. J
Arthros Rec Surg. 1993;9:149-153.
15. Larson RV. The use of hamstring tendons in anterior cruciate ligament surgery.
In: Current techniques in arthroscopy, 2nd ed. New York: Thieme 1998.
16. Larson RV, Ericksen D. Complications in the use of hamstring tendons for
anterior cruciate ligament reconstruction. Sports Med Arthroscopy Rev. 1997;5:83-
90.
17. Magen HE, Howell SM, Hull ML. Structural properties of six tibial fixation
methods for anterior cruciate ligament soft tissue grafts. Am J Sports Med.
1999;27:35-43.
18. Marder RA, Raskind JR, Carroll M. Prospective evaluation of arthroscopically

assisted anterior cruciate ligament reconstruction: patellar tendon versus
semitendinosus and gracilis tendons. Am J Sports Med. 1991;19:478-484.
19. Mott HW. Semitendinosus anatomic reconstruction for cruciate ligament

insufficiency. Clin Orthop. 1983;172:90-92.
20. Noyes FR, Butler DL, Grood ES, et al. Biomechanical analysis of human
ligament grafts used in knee-ligament repairs and reconstructions. J Bone Joint
Surg. 1984;66A:344-352.
21. O'Neill DB. Arthroscopically assisted reconstruction of the anterior cruciate

ligament: a prospective randomized analysis of three techniques. J Bone Joint
Surg Am. 1996;78:803-813.
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22. Otero AL, Hutcheson L. A comparison of the doubled semitendinosus/gracilis

and central third of the patellar tendon autografts in arhtroscopic anterior
cruciate ligament reconstruction. Arthroscopy. 1993;9:143-148.
23. Pagnani MJ, Warner JJP, O'Brien SJ, et al. Anatomic considerations in
harvesting the semitendinosus and gracilis tendons and a technique for harvest.
Am J Sports Med. 1993;21:565-571.
24. Pinczewski LA, Deeham DJ, Salmon LJ, et al. A five-year comparison of
patellar tendon versus four-strand hamstring tendon autograft for arthroscopic
reconstruction of the anterior cruciate ligament. Am J Sport Med. 2002;30:523-
536.
25. Rodeo SA, Arnoczky SP, Torzilli PA, et al. Tendon-healing in a bone tunnel: a
biomechanical and histological study in the dog. J Bone Joint Surg. 1993;75A:1795-
1803.
26. Rosenberg TD, Deffner KT. ACL reconstruction: semitendinosus tendon is the
graft of choice. Orthopedics. 1997;20:396-398.
27. Rowden NJ, Sher D, Rogers GJ, et al. Anterior cruciate ligament graft fixation:
initial comparison of patellar tendon and semitendinosus autografts in young fresh
cadavers. Am J Sports Med. 1997;25:472-478.
28. Shino K, Nakagawa S, Inoue M, et al. Deterioration of patellofemoral articular

surfaces after anterior cruciate ligament reconstruction. Am J Sports Med.
1993;21:206-211.
29. Sidles JA, Clark JM, Garbini JL. A geometric theory of equilibrium mechanics of
fibers in ligaments and tendons. J Biomech. 1991;29:943-949.
30. Sidles JA, Larson RV, Garbini JL, et al. Ligament length relationships in the
moving knee. J Orthop Res. 1988;6:593-610.
31. Simonian PT, Williams RJ, Deng XH, et al. Hamstring and patellar tendon graft
response to cyclical loading. Am J Knee Surg. 1998;11:101-105.
32. Steiner ME, Hecker AT, Brown CH Jr, et al. Anterior cruciate ligament graft
fixation: comparison of hamstring and patellar tendon grafts. Am J Sports Med.
1994;22:240-246.
33. To JT, Howell SM, Hull ML. Contributions of fixation methods to the stiffness of
anterior cruciate ligament replacements at implantation. Arthroscopy.
1999;15:379-387.
> Table of Contents > Part Three - Ligament Injuries and Instability > 17 - Allograft
Preparation for ACL Reconstruction
17
Allograft Preparation for ACL Reconstruction
William H. Warden III
Allograft is an attractive option for anterior cruciate ligament (ACL) reconstruction, as
there is no donor site morbidity and therefore less discomfort in the early
postoperative period. In my experience, patients with allograft ACL reconstructions
are clearly more comfortable immediately after surgery than patients who have
received autograft. The most striking example I can recall is a patient who walked into
the office, did a deep knee bend, and jumped into the air 1 week after a bilateral ACL
reconstruction with no formal physical therapy.
One of the few strict contraindications to allograft reconstruction is a patient who is

unwilling to accept the risk of disease transmission. Although there are few strict
indications or contraindications, there are a number of advantages and disadvantages
to consider.
Advantages
No donor site morbidity, therefore useful for:
Revision
Multiple ligament injuries

Early return to light work
Decreased anesthesia, operating room, tourniquet time
Cosmesis
Disadvantages
Risk of disease transmission
Cost
Patellar tendon, $1,800
Achilles tendon, $1,200
Soft tissue, $800
Offset by decreased operating room time
Potential for delayed incorporation
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Although not entirely scientific, my personal bias is to avoid allograft in children and
women of childbearing age, and lean toward allograft in patients who are age 50 or
older.
An early step involves a discussion with the patient of the risk of viral transmission.
Use tissue from a well-established tissue bank certified by the American Association of
Tissue Banks (AATB) from donors under age 40. Ideally, grafts are obtained with sterile
harvest, antibiotic soaks, low-dose radiation, and freezing. Higher dose irradiation in a
radioprotectant solution is an emerging technique.
There are a number of allograft options including bone-patellar tendon-bone (BTB),

Achilles tendon, quadriceps tendon, hamstring, and tibialis anterior. The choice is
mainly surgeon preference. BTB is an excellent choice for a surgeon who prefers bone
fixation, but this allograft can potentially carry more antigens. Preoperative planning
is critical for BTB, to avoid graft-tunnel mismatch. This is not an issue with a large
patient; for a small patient, request collagen lengths of 45 mm or less. If you cannot
obtain a short graft, consider a soft tissue or single bone plug graft.
Always have a back-up graft. Occasionally, an allograft will appear discolored or

macerated. A degenerative Achilles tendon may not be evident until the graft is
sectioned. In case of the dreaded drop, one can potentially scrub an autograft, but it's
difficult to justify scrubbing an allograft that can simply be replaced by a backup.
SURGERY
Patient Positioning
Position the graft preparation table so that it does not interfere with entry into the
room, draping, or setup of arthroscopy equipment. Ideally, you should have a good
view of the arthroscopy monitor.
Technique
If it is certain that a graft will be required, graft preparation can begin before the
patient is in the room. Thaw time ranges from 5 to 30 minutes including a soak in
saline with â€œdouble antibioticâ€• (bacitracin and polymyxin B) solution. Grafts
prepared with radioprotectant typically require three separate soaks over a total of 30
minutes. Inspect the graft for discoloration or signs of structural compromise. Trim
excess fatty or loose connective tissue from the graft.
Achilles Tendon
Achilles tendon grafts have two unique features to address during graft preparation:
the collagen insertion into the bone plug is angular and the tendinous portion is fan
shaped. Although Achilles tendon grafts come with ample bone blocks, the nature of
the tendinous insertion is such that the bone plug length is always short. Therefore,
bone should not be removed from the distal end of the bone block unless necessary
during final preparation (Fig. 17-1). A prominent pump bump at the posterior aspect of
the plug may need to be trimmed along with a small amount of tendinous tissue (Fig.
17-2). The bone plug should be fashioned to fit easily through the sizing tube. We
typically use a 10-mm bone plug. Next, shorten the graft to a length of approximately
120 mm and trim excess collagen from the sides (Fig. 17-3A). A No. 2 nylon passing
suture is placed through the bone plug. Next, suture the distal portion of the graft by
placing a No. 2 Ethibond suture 40 to 50 mm distal to the bone block. Distal
interference screws seem to get better purchase on the graft if they are placed
adjacent to a sutured graft; suturing can begin more distally if interference fixation is
not used. The first suture placed should be used to tubularize the graft (see Fig. 17-
3B). A second arm of the suture is run down opposite this and then a second suture is
placed rotated 90 degrees to the initial suture. With each strand of the suture place
one or two locked passes and then run distally.
Soft Tissue
Although soft tissue allows a tighter fit in the sizing tubes compared with bone plugs,
avoid the tendency to make a collagen graft that is too large. A 10-mm bundle of
collagen may not fit well in the knee of a petite gymnast.
Most femoral-sided soft tissue fixation devices require a folded graft. Fold the graft
over a passing suture (No. 2 nylon) or fixation device and shorten if longer than 120
mm, optimizing thicker collagen sections. This should be ample length for even the
largest patient and allow extra graft if backup fixation with a post and soft tissue
washer is required on the tibial side. For a tight soft tissue graft, trim the corners of
the folded section slightly to allow easy entrance into bone tunnels (Fig. 17-4A).
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FIGURE 17-1
The uncut Achilles bone block is 30 mm in length (A). However, after 10-mm wide
coronal cuts are completed (B), it becomes clear that there are only 20 mm of
bone distal to the tendinous insertion (C). Attempts to include bone proximal to
the tendinous insertion will leave a graft that is too thick or compromise the
tendinous insertion.
FIGURE 17-2
Some grafts may have a prominent â€œpump bumpâ€• that must be trimmed.
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FIGURE 17-3
After the graft is shortened to 120 mm and the distal sides are trimmed down (A),
distal sutures are passed tubularizing the Achilles graft (B). Final graft (C).
Although some prefer suturing each distal strand independently to optimize graft
tension, I find it is more time consuming and makes a bulky graft. However, some
fixation devices such as the Intrafix require independent suturing, whereas others such
as the EndoButton are easier to use with independent suturing.
Holding distal ends in a clamp, suture the proximal portion of the graft together using
a 0-vicryl whip stitch. Start 5 to 10 mm distal to the fold (closer than this tends to
bunch up the leading edge of the graft) to a distance of approximately 25 mm from
the leading edge of your graft (this may vary depending on your fixation technique).
Be sure to mark the graft at the appropriate distance from the proximal end (usually
25 mm) so that passage to the appropriate depth can be confirmed arthroscopically.
Unless your fixation device requires independent graft ends, suture the distal ends
together using two No. 2 Ethibond sutures and a tapered needle. Start 40 to 50 mm
distal to the mark in the proximal end (more distal if interference screws are not
used), throw one or two locking passes attempting to incorporate all strands, and then
run distally with a whipstitch, including at least two strands with every bight. The
other limb of the suture is run down the graft on the opposite side. The
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second suture is then placed in a similar fashion rotated 90 degrees from the initial
suture. Be sure to confirm that your graft fits through the appropriate sizing tube after
suturing. If there is a portion of the graft that is slightly snug, trim the appropriate
section or keep the snug area in the sizing tube until passage (see Fig. 17-4B,C).
Patellar Tendon
The main concern with patellar tendon allograft is a graft that is too long for the
patient, even with an appropriate length tibial tunnel (see Preoperative Planning). Be
sure to center the patellar bone plug at the tip of the inferior pole of the patella to
minimize graft length (Fig. 17-5). A 10 mm wide section is cut from the patellar block
and then a similar plug is cut directly distal to this in the tibial plug. The 10 mm wide
strip of collagen between them is isolated from surrounding tissue. Note that larger
bone blocks may be required for revision cases. Graft preparation proceeds as with a
typical patellar tendon graft. The blocks are trimmed with a rongeur to fit the
appropriate sizing tube (9 to 11 mm) and to a length of 22 to 25 mm (Fig. 17-6). Some
find special compaction pliers helpful for contouring bone plugs. Undersize the bone
plugs slightly and taper the leading edge (Fig. 17-7) so that they pass easily through
the appropriate sizing tube, as bone plugs tend to jam in bone tunnels. Passing sutures
are inserted through drill holes, a 30-in No. 2 nylon in the lead plug, and three No. 2
Ethibonds in the distal plug at 45-degree angles to each other to minimize the risk of
suture laceration during interference screw placement.
The prepared graft is irrigated with antibiotic saline, kept under 10 to 20 lb of tension,
and covered with gauze until required (Fig. 17-8).
Pearls and Pitfalls
Undersized bone plugs slightly and taper the leading end for easy passage (see
Fig. 17-7).
Soft tissue grafts can be slightly snug in the sizing tube, but avoid too much
collagen for the patient.
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Trim and tubularize wide soft tissue grafts (see Fig. 17-3B).
Trim the entering corners of a tight folded soft tissue graft (see Fig. 17-4A).
Keep a tight section of a soft tissue graft (usually the leading loop) in a sizing
tube until required (see Fig. 17-4C).
Avoid BTB graft-tunnel mismatch (see previous discussion) with preoperative

graft length selection and appropriate tibial tunnel length.
Don't miss the â€œnoseâ€• of the patella (see Fig. 17-5).
Trim the â€œpump bumpâ€• from the calcaneal bone plug (see Fig. 17-2).
Always have a back-up graft.

FIGURE 17-4
For a tight soft tissue graft, trim the corners of the leading fold slightly to allow
easy entrance into bone tunnels (A). A very large graft such as a tibialis anterior
may require trimming along the entire length (B). If a portion of the final graft is
still slightly too tight, keep the sizing tube over that area until passage (C). Final
graft (D).
FIGURE 17-5
Center the patellar bone plug at the tip of the inferior pole of the patella to
minimize graft length, especially when using a hemipatella graft.
Although there are concerns regarding delayed incorporation of allograft compared
with autograft we use the same accelerated early rehabilitation regardless of graft
choice. Consider more vigilance regarding late effusions when allograft is used.
COMPLICATIONS
Although rare, infection can be devastating. The AATB now requires nucleic acid
testing of grafts for HIV-1 and HCV. Furthermore, emerging techniques for soft tissue
graft sterilization show promise. Nevertheless, reports of compromised
acquisition/preparation of allograft requiring recall
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continue. An important consideration is follow-up with your tissue bank in cases of
suspected infection. Delayed incorporation leading to early failure is a theoretical
complication that is difficult to assess clinically. As yet unrecognized immune
responses are another potential concern.
FIGURE 17-6
With a rongeur, trim the corners and fashion the plug to fit easily in a 12-mm
sizing tube. Excess soft tissue and bone is trimmed from the cortical sides of the
bone plugs (A). Use progressively smaller sizing tubes to locate raised areas to
trim (B). Compression with pliers may help final contouring.
FIGURE 17-7
Taper the leading edge of the bone plug for easy passage.
FIGURE 17-8
Final patellar tendon graft. The femoral plug is marked to facilitate arthroscopic
evaluation of graft passage and orientation.
RESULTS
Please see Chapters 13 and 18 for discussions of ligament reconstruction results.
RECOMMENDED READING
Jackson DW, Corsetti J, Simon TM. Biologic incorporation of allograft anterior cruciate
ligament replacements. Clin Orthop. 1996;324:126-133.
Jackson DW, Grood ES, Goldstein JD, et al. A comparison of patellar tendon autograft
and allograft used for anterior cruciate ligament reconstruction in the goat model. Am
J Sports Med. 1993;21:176-185.
Joyce MJ. Safety and FDA regulations for musculoskeletal allografts: perspective of an
orthopaedic surgeon. Clin Orthop Relat Res. 2005;435:22-30.
Vangsness CT, Jr Garcia IA, Mills CR, et al. Allograft transplantation in the knee: tissue
regulation, procurement, processing, and sterilization. Am J Sports Med.
2003;May(31):474-481.
> Table of Contents > Part Three - Ligament Injuries and Instability > 18 - Arthroscopically
Assisted Posterior Cruciate Ligament Reconstruction
18
Arthroscopically Assisted Posterior Cruciate
Gregory C. Fanelli
Craig J. Edson
Kristin N. Reinheimer
The incidence of posterior cruciate ligament (PCL) injuries is reported to be from 1%

to 40% of acute knee injuries. This range depends on the patient population reported,
and is approximately 3% in the general population and 38% in reports from regional
trauma centers (1,6,13). Our practice at a regional trauma center has a 38.3%
incidence of PCL tears in acute knee injuries, and 56.5% of these PCL injuries occur in
multiple trauma patients. Of these, 45.9% are combined anterior cruciate ligament
(ACL)/PCL tears, whereas 41.2% are PCL/posterolateral corner tears. Only 3% of acute
PCL injuries seen in our practice are isolated.
This chapter illustrates the senior author's surgical techniques for the arthroscopic
single-bundle-single-femoral tunnel, and double-bundle-double-femoral tunnel,
transtibial PCL reconstruction surgical procedure. We will also present the Fanelli
Sports Injury Clinic 2-to 10-year results of PCL reconstruction using these surgical
techniques. The information presented in this chapter has also been presented
elsewhere, and the reader is referred to these sources for additional information
regarding this topic (2,3,4,5,7,8,9,10,11,12,13,14,15,16,17,18,19,20,22,23).
The single-bundle-single-femoral tunnel transtibial tunnel PCL reconstruction is an
anatomic reconstruction of the anterolateral bundle of the PCL. The anterolateral
bundle tightens in flexion, and this reconstruction reproduces that biomechanical
function. While the single-bundle-single-femoral tunnel transtibial tunnel PCL
reconstruction does not reproduce the broad anatomic insertion site of the normal
PCL, there are certain factors that lead to success with this surgical technique:
Identification and treatment all pathology (especially posterolateral and

posteromedial instability)
Accurate tunnel placement
Anatomic graft insertion sites
Strong graft material
Minimal graft bending
Final tensioning at 70 to 90 degrees of knee flexion
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Graft tensioning
Arthrotek mechanical tensioning device
Primary and back-up fixation
Appropriate rehabilitation program
Indications
Our indications for surgical treatment of acute PCL injuries include insertion site
avulsions, tibial step-off decreased 6 tolO mm or greater, and PCL tears combined with
other structural injuries. Our indications for surgical treatment of chronic PCL injuries
are when an isolated PCL tear becomes symptomatic demonstrated by progressive
functional instability.
Contraindications
There are no significant contraindications.
History and Physical Examination
The typical history of a patient with a PCL injury includes a direct blow to the
proximal tibia with the knee in 90 degrees of flexion. Hyperflexion, hyperextension,
and a direct blow to the proximal medial or lateral tibia in varying degrees of knee
flexion, as well as a forced varus or valgus force, will induce PCL-based multiple
ligament knee injuries.
Physical examination of the injured knee compared with the noninjured knee reveals
decreased tibial step-offs, and a positive posterior drawer test. Since isolated PCL
tears are rare, collateral ligament injury is common (posterolateral and posteromedial
corner injuries), and posterolateral and posteromedial drawer tests, dial tests,
external rotation recurvatum tests, varus and valgus laxity, and even anterior laxity
may be present.
Isolated Posterior Cruciate Ligament Injury Diagnostic

Features
Abnormal posterior laxity less than 5 mm
Abnormal posterior laxity decreases with tibial internal rotation
No abnormal varus
Abnormal external rotation of the tibia on the femur less than 5 degrees
compared with the uninvolved side tested with the knee at 30 and 90 degrees
knee flexion.
Posterior Cruciate Ligament/PLC Injury Diagnostic Features
Abnormal posterior laxity greater than 5 to 10 mm
Abnormal varus rotation at 30 degrees knee flexion is variable and depends on

the posterolateral instability grade.
Abnormal external rotation thigh foot angle test greater than 10 degrees
compared with the normal lower extremity tested at 30 and 90 degrees of knee
flexion. (If you can see the difference, then PLI exists.)
Posterolateral drawer-positive
Combined Anterior Cruciate Ligament/Posterior Cruciate

Ligament Injuries Diagnostic Features
Grossly abnormal anterior-posterior tibial-femoral laxity at both 25 and 90

degrees of knee flexion
Positive Lachman and pseudo-Lachman tests
Positive pivot-shifting phenomenon
Negative tibial step-off (posterior sag)
Increased varus-valgus laxity in full extension
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Imaging Studies
Plain radiographs used to evaluate PCL injuries include the following views: anterior-
posterior weight bearing both knees, 30-degree flexion lateral, intercondylar notch,
30-degree axial of the patellas, and stress views at 90 degrees of knee flexion of both
knees. Magnetic resonance imaging (MRI) is helpful in acute cases; however, we have
found MRI to be less beneficial in chronic PCL injuries. Bone scan is used in chronic
cases of PCL instability presenting with pain to define early degenerative joint
disease.
SURGERY
Patient Positioning and Initial Setup
The patient is positioned on the operating table in the supine position, and the
surgical and nonsurgical knees are examined under general or regional anesthesia. A
tourniquet is applied to the operative extremity, and the surgical leg is prepped and
draped in a sterile fashion. Allograft tissue is prepared before beginning the surgical
procedure, and autograft tissue is harvested prior to beginning the arthroscopic
portion of the procedure. The arthroscopic instruments are inserted with the inflow
through the superior lateral patellar portal, the arthroscope in the inferior lateral
patellar portal, and the instruments in the inferior medial patellar portal. The portals
are interchanged as necessary. The joint is thoroughly evaluated arthroscopically, and
the PCL is evaluated using the three-zone arthroscopic technique (17). The PCL tear is
identified, and the residual stump of the PCL is debrided with hand tools and the
synovial shaver.
Initial Incision
An extra capsular posteromedial safety incision approximately 1.5 to 2.0 cm long is
created (Fig. 18-1). The crural fascia is incised longitudinally, taking precautions to
protect the neurovascular structures. The interval is developed between the medial
head of the gastrocnemius muscle and the posterior capsule of the knee joint, which is
anterior. The surgeon's gloved finger is positioned so that the neurovascular structures
are posterior to the finger, and the posterior aspect of the joint capsule is anterior to
the surgeon's finger. This technique enables the surgeon to monitor surgical
instruments, such as the over-the-top PCL instruments and the PCL/ACL drill guide, as
they are positioned in the posterior aspect of the knee. The surgeon's finger in the
posteromedial safety incision also confirms accurate placement of the guidewire prior
to tibial tunnel drilling in the medial-lateral and proximal-distal directions (Fig. 18-2).
This is the same anatomic surgical interval that is used in the tibial inlay posterior
approach.
FIGURE 18-1
Posteromedial extra-articular extracapsular safety incision. (Redrawn from

Arthrotek, Inc., Warsaw, Indiana, with permission.)
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FIGURE 18-2
The surgeon is able to palpate the posterior aspect of the tibia through the
extracapsular extra-articular posteromedial safety incision. This enables the
surgeon to accurately position guidewires, create the tibial tunnel, and protect
the neurovascular structures. (Redrawn from Arthrotek, Inc., Warsaw, Indiana,
with permission.)
Elevating the Posterior Capsule

The curved over-the-top PCL instruments are used to carefully lyse adhesions in the
posterior aspect of the knee, and to elevate the posterior knee joint capsule away
from the tibial ridge on the posterior aspect of the tibia. This capsular elevation
enhances correct drill guide and tibial tunnel placement (Fig. 18-3).
Drill Guide Positioning

The arm of the Arthrotek Fanelli PCL-ACL Drill Guide is inserted into the knee through
the inferior medial patellar portal and positioned in the PCL fossa on the posterior
tibia (Fig. 18-4). The bullet portion of the drill guide contacts the anterior medial
aspect of the proximal tibia approximately 1 cm below the tibial tubercle, at a point
midway between the tibial crest anteriorly and the posterior medial border of the
tibia. This drill guide positioning creates a tibial tunnel that is relatively vertically
oriented and has its posterior exit point in the inferior and lateral aspect of the PCL
tibial anatomic insertion site. This positioning creates an angle of graft orientation
such that the graft will turn two very smooth 45-degree angles on the posterior aspect
of the tibia, eliminating the â€œkiller turnâ€• of 90-degree graft angle bending (Fig.
18-5).
The tip of the guide in the posterior aspect of the tibia is confirmed with the surgeon's
finger through the extra capsular posteromedial safety incision (see Fig. 18-2).
Intraoperative anteroposterior and lateral radiographs may also be used, as well as
arthroscopic visualization to confirm drill guide and guide pin placement. A blunt
spade-tipped guidewire is drilled from anterior to posterior and can be visualized with
the arthroscope, in addition to being palpated with the finger in the posteromedial
safety incision. We consider the finger in the posteromedial safety incision the most
important step for accuracy and safety.
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FIGURE 18-3
Posterior capsular elevation using the Arthrotek PCL instruments. (Redrawn from
Arthrotek, Inc., Warsaw, Indiana, with permission.)
FIGURE 18-4
Arthrotek Fanelli PCL-ACL drill guide positioned to place guidewire in preparation

for creation of the transtibial PCL tibial tunnel. (Redrawn from Arthrotek, Inc.,
Warsaw, Indiana, with permission.)
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FIGURE 18-5
Drawing demonstrating the desired turning angles the PCL graft will make after
the creation of the tibial tunnel. (Redrawn from Arthrotek, Inc., Warsaw, Indiana,
with permission.)
Tibial Tunnel Drilling

The appropriately sized standard cannulated reamer is used to create the tibial
tunnel. The closed curved PCL curette may be positioned to cup the tip of the
guidewire (Fig. 18-6). The arthroscope, when positioned in the posteromedial portal,
visualizes the guidewire being captured by the curette and protecting the
neurovascular structures in addition to the surgeon's finger in the posteromedial safety
incision. The surgeon's finger in the posteromedial safety incision is monitoring the
position of the guidewire. The standard cannulated drill is advanced to the posterior
cortex of the tibia. The
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drill chuck is then disengaged from the drill, and completion of the tibial tunnel
reaming is performed by hand. This gives an additional margin of safety for completion
of the tibial tunnel. The tunnel edges are chamfered and rasped with the PCL/ACL
system rasp (Fig. 18-7).
FIGURE 18-6
The Arthrotek PCL closed curette may be used to cap the guidewire during tibial
tunnel drilling. (Redrawn from Arthrotek, Inc., Warsaw, Indiana, with
permission.)
Drilling the Femoral Tunnel Outside In: Single- and
Double-Bundle Posterior Cruciate Ligament
Reconstruction
The Arthrotek Fanelli PCL/ACL drill guide is positioned to create the femoral tunnel.
The arm of the guide is introduced through the inferomedial patellar portal and is
positioned such that the guidewire will exit through the center of the stump of the
anterior lateral bundle of the PCL (Fig. 18-8). The blunt spade-tipped guidewire is
drilled through the guide, and just as it begins to emerge through the center of the
stump of the PCL anterior lateral bundle, the drill guide is disengaged. The accuracy
of the placement of the wire is confirmed arthroscopically with probing and
visualization. Care must be taken to ensure the patellofemoral joint has not been
violated by arthroscopically examining the patellofemoral joint prior to drilling, and
that there is adequate distance between the femoral tunnel and the medial femoral
condyle articular surface. The appropriately sized standard cannulated reamer is used
to create the femoral tunnel. A curette is used to cap the tip of the guidewire so there
is no inadvertent advancement of the guidewire, which may damage the ACL or
articular surface. As the reamer is about to penetrate interiorly, the reamer is
disengaged from the drill and the final reaming is completed by hand (Fig. 18-9). This
adds an additional margin of safety. The reaming debris is evacuated with a synovial
shaver to minimize fat pad inflammatory response with subsequent risk of
arthrofibrosis. The tunnel edges are chamfered and rasped.
When the double-bundle PCL reconstruction is performed, the Arthrotek Fanelli

PCL/ACL drill guide is positioned to create the second femoral tunnel. The arm of the
guide is introduced through the inferior medial patellar portal and is positioned such
that the guidewire will exit through the center of the stump of the posterior medial
bundle of the PCL (Fig. 18-10). The blunt spade-tipped guidewire is drilled through the
guide, and just as it begins to emerge through the center of the stump of the PCL
posterior medial bundle, the drill guide is disengaged. The accuracy of the placement
of the wire is confirmed arthroscopically with probing and visualization. Care must be
taken to ensure that there will be an adequate bone bridge (approximately 5 mm)
between the two femoral tunnels prior to drilling. This is accomplished using the
calibrated probe and direct arthroscopic visualization. The appropriately sized
standard cannulated reamer is used to create the posterior medial bundle femoral
tunnel. A curette is used to cap the tip of the guidewire so there is no inadvertent
advancement of the guidewire, which may damage the anterior cruciate ligament or
articular surface. As the reamer is about to penetrate interiorly, the reamer is
disengaged from the drill and the final reaming is completed by hand (Fig. 18-11). This
adds an additional margin of safety. The reaming
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debris is evacuated with a synovial shaver to minimize fat pad inflammatory response
with subsequent risk of arthrofibrosis. The tunnel edges are chamfered and rasped.
FIGURE 18-7
The tunnel edges are chamfered after drilling to smooth any rough edges.
(Redrawn from Arthrotek, Inc., Warsaw, Indiana, with permission.)
FIGURE 18-8
The Arthrotek Fanelli PCL-ACL drill guide is positioned to drill the guidewire from
outside in. The guidewire begins at a point halfway between the medial femoral
epicondyle and the medial femoral condyle trochlea articular margin,
approximately 2 to 3 cm proximal to the medial femoral condyle distal articular
margin, and exits through the center of the stump of the anterolateral bundle of
the PCL stump. (Redrawn from Arthrotek, Inc., Warsaw, Indiana, with
permission.)
FIGURE 18-9
Completion of anterolateral bundle femoral tunnel reaming by hand for an

additional margin of safety. (Redrawn from Arthrotek, Inc., Warsaw, Indiana,
with permission.)
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FIGURE 18-10
The Arthrotek Fanelli PCL-ACL drill guide is positioned to drill the guidewire from
outside in for creation of the posteromedial bundle PCL femoral tunnel. (Redrawn
from Arthrotek, Inc., Warsaw, Indiana, with permission.)
FIGURE 18-11
Completion of femoral tunnel reaming by hand for an additional margin of safety.

(Redrawn from Arthrotek, Inc., Warsaw, Indiana, with permission.)
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Drilling the Femoral Tunnel Inside Out: Single- and

Reconstruction
The PCL single-bundle or double-bundle femoral tunnels can be made from inside out
using the Arthrotek Fanelli Double-Bundle Aimers. Inserting the appropriately sized
Arthrotek Fanelli double-bundle aimer through a low anterior lateral patellar
arthroscopic portal creates the PCL anterior lateral bundle femoral tunnel. The
double-bundle aimer is positioned directly on the footprint of the femoral anterior
lateral bundle PCL insertion site (Fig. 18-12). The appropriately sized guidewire is
drilled through the aimer, through the bone, and out a small skin incision. Care is
taken to ensure there is no compromise of the articular surface. The double-bundle
aimer is removed, and an acorn reamer is used to endoscopically drill from inside out
the anterior lateral PCL femoral tunnel (Fig. 18-13). The tunnel edges are chamfered
and rasped. The reaming debris is evacuated with a synovial shaver to minimize fat
pad inflammatory response with subsequent risk of arthrofibrosis. When the
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surgeon chooses to perform a double-bundle double femoral tunnel PCL
reconstruction, the same process is repeated for the posterior medial bundle of the
PCL (Figs. 18-14 and 18-15). Care must be taken to ensure that there will be an
adequate bone bridge (approximately 5 mm) between the two femoral tunnels prior to
drilling. This is accomplished using the calibrated probe and direct arthroscopic
visualization (Fig. 18-16).
FIGURE 18-12
Arthrotek Fanelli double-bundle aimer positioned to drill a guidewire for creation

of the PCL anterolateral bundle tunnel. (Redrawn from Arthrotek, Inc., Warsaw,
Indiana, with permission.)
FIGURE 18-13
Endoscopic acorn reamer is used to create the PCL anterolateral bundle femoral
tunnel through the low anterolateral patellar portal. (Redrawn from Arthrotek,
Inc., Warsaw, Indiana, with permission.)
FIGURE 18-14
Arthrotek Fanelli double-bundle aimer positioned to drill a guidewire for creation

of the PCL posteromedial bundle femoral tunnel through the low anterolateral
patellar portal. (Redrawn from Arthrotek, Inc., Warsaw, Indiana, with
permission.)
Tunnel Preparation and Graft Passage

The Arthrotek Magellan suture-passing device is introduced through the tibial tunnel
and into the knee joint, and is retrieved through the femoral tunnel with an
arthroscopic grasping tool (Fig. 18-17). The traction sutures of the graft material are
attached to the loop of the Arthrotek Magellan suture-passing device, and the PCL
graft material is pulled into position.
Graft Tensioning and Fixation

Fixation of the PCL substitute is accomplished with primary and backup fixation on
both the femoral and tibial sides. Our most commonly used graft source for PCL
reconstruction is the Achilles tendon allograft alone for single-bundle reconstructions,
and Achilles tendon and tibialis anterior allografts for double-bundle reconstructions,
although other allografts and autografts may be used as preferred by an individual
surgeon. Femoral fixation is accomplished with cortical suspensory backup fixation
using polyethylene ligament fixation buttons, and aperture opening fixation using the
Arthrotek Bio-Core bioabsorbable interference screws. The Arthrotek Graft Tensioning
Boot is applied to the traction sutures of the graft material on its distal end, and
tensioned to restore the anatomic tibial step-off. The knee is cycled through several
sets of 25 full flexion-extension cycles for graft pretensioning and settling
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(Fig. 18-18). The PCL reconstruction graft is tensioned in approximately 70 degrees of
knee flexion. Graft fixation is achieved with primary aperture opening fixation using
the Arthrotek Bio-Core bioabsorbable interference screw, and back-up fixation with a
ligament fixation button, or screw and post, or screw and spiked ligament washer
assembly (Fig. 18-19).
FIGURE 18-15
Endoscopic acorn reamer is used to create the PCL posteromedial bundle through
the low anterolateral patellar portal. (Redrawn from Arthrotek, Inc., Warsaw,
Indiana, with permission.)
FIGURE 18-16
A: Tunnel placement for double-bundle PCL reconstruction. B: Double-bundle PCL

reconstruction using Achilles tendon allograft for the anterolateral bundle, and
tibialis anterior allograft for the posteromedial bundle.
FIGURE 18-17
Arthrotek Magellan suture-passing device. (Redrawn from Arthrotek, Inc.,

Warsaw, Indiana, with permission.)
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Pearls and Pitfalls

The posteromedial safety incision protects the neurovascular structures, confirms
accurate tibial tunnel placement, and allows the surgical procedure to be done at an
accelerated pace (see Fig. 18-1). It is important to be aware of the two tibial and
femoral tunnel directions, and to have an adequate bone bridge between the PCL and
ACL tibial and femoral tunnels. This will reduce the possibility of fracture. We have
found it useful to use primary and back-up fixation. Primary fixation is with Arthrotek
BioCore interference screws, and back-up fixation is performed with a screw and
spiked ligament washer, and Arthrotek ligament fixation buttons. Secure fixation is
critical to the success of this surgical procedure (Fig. 18-19). Restoration of the normal
tibial step-off at 70 degrees of flexion has provided the most reproducible method of
establishing the neutral point of the tibia-femoral relationship in our experience. Full
range of motion is confirmed on the operating table to ensure the knee is not
â€œcapturedâ€• by the reconstruction. When multiple ligament surgeries are
performed at the same operative session, the PCL reconstruction is performed first,
followed by the ACL reconstruction, followed by the collateral ligament surgery.
FIGURE 18-18
Arthrotek knee ligament graft-tensioning boot. This mechanical tensioning device

uses a ratcheted torque wrench device to assist the surgeon during graft
tensioning. (Redrawn from Arthrotek, Inc., Warsaw, Indiana, with permission.)
FIGURE 18-19
Final graft fixation using primary and back-up fixation. (Redrawn from Arthrotek,
Inc., Warsaw, Indiana, with permission.)
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The knee is immobilized in a long leg brace in full extension for 6 weeks, non-weight
bearing using crutches. Progressive range of motion occurs during weeks 4 through 6.
The brace is unlocked between weeks 3 and 6, and progressive weight bearing at 25%
body weight per week is allowed during postoperative weeks 7 through 10. The
crutches are discontinued at the end of postoperative week 10. Progressive strength
training and range-of-motion exercises are performed. Return to sports and heavy
labor occurs after the sixth to ninth postoperative month, when sufficient strength,
range of motion, and proprioceptive skills have returned.
COMPLICATIONS
Posterior cruciate ligament reconstruction is a technically demanding surgery.
Complications encountered with this surgical procedure include failure to recognize
associated ligament injuries, neurovascular complications, persistent posterior sag,
osteonecrosis, knee motion loss, anterior knee pain, and fractures (5,18,19). A
comprehensive preoperative evaluation, including an accurate diagnosis, a well-
planned and carefully executed surgical procedure, and a supervised postoperative
rehabilitation program will help to reduce the incidence of these complications.
RESULTS
We have previously published the results of our arthroscopically assisted combined
ACL/PCL and PCL/posterolateral complex reconstructions using the reconstructive
technique described in this chapter (7,10,11,14,15,16). Our most recently published 2-
to 10-year results of combined ACL-PCL reconstructions without the Arthrotek Graft
Tensioning Boot are presented here (10).
This study presented the 2- to 10-year (24 to 120 month) results of 35 arthroscopically
assisted combined ACL/PCL reconstructions evaluated pre- and postoperatively using
Lysholm, Tegner, and Hospital for Special Surgery knee ligament rating scales, KT 1000
arthrometer testing, stress radiography, and physical examination.
This study population included 26 males, 9 females, 19 acute, and 16 chronic knee
injuries. Ligament injuries included 19 ACL/PCL/posterolateral instabilities, 9
ACL/PCL/MCL instabilities, 6 ACL/PCL/posterolateral/MCL instabilities, and 1 ACL/PCL
instability. All knees had grade III preoperative ACL/PCL laxity, and were assessed pre-
and postoperatively with arthrometer testing, three different knee ligament rating
scales, stress radiography, and physical examination. Arthroscopically assisted
combined ACL/PCL reconstructions were performed using the single incision
endoscopic ACL technique, and the single femoral tunnel-single-bundle transtibial
tunnel PCL technique. Posterior cruciate ligaments were reconstructed with allograft
Achilles tendon (26 knees), autograft BTB (7 knees), and autograft
semitendinosus/gracilis (2 knees). Anterior Cruciate ligaments were reconstructed
with autograft BTB (16 knees), allograft BTB (12 knees), Achilles tendon allograft (6
knees), and autograft semitendinosus/gracilis (1 knee). MCL injuries were treated with
bracing or open reconstruction. Posterolateral instability was treated with biceps
femoris tendon transfer, with or without primary repair, and posterolateral capsular
shift procedures as indicated. No Arthrotek graft tensioning boot was used in this
series of patients.
Postoperative physical examination results revealed normal posterior drawer/tibial

step-off in 16/35 (46%) of knees. Normal Lachman and pivot shift tests were found in
33/35 (94%) of knees. Posterolateral stability was restored to normal in 6/25 (24%) of
knees, and tighter than the normal knee in 19/25 (76%) of knees evaluated with the
external rotation thigh foot angle test. Thirty-degree varus stress testing was normal
in 22/25 (88%) of knees, and grade 1 laxity in 3/25 (12%) of knees. Thirty-degree valgus
stress testing was normal in 7/7 (100%) of surgically treated MCL tears, and normal in
7/8 (87.5%) of brace treated knees. Postoperative KT 1000 arthrometer testing mean
side-to-side difference measurements were 2.7 mm (PCL screen), 2.6 mm (corrected
posterior), and 1.0 mm (corrected anterior) measurements, a statistically significant
improvement from preoperative status (p = 0.001). Postoperative stress radiographic
side-to-side difference measurements measured at 90 degrees of knee flexion, and 32
lbs of posteriorly directed proximal force were 0 to 3 mm in 11/21 (52.3%), 4 to 5 mm
in 5/21 (23.8%), and 6 to 10 mm in 4/21 (19%) of knees. Postoperative Lysholm,
Tegner, and Hospital for Special Surgery knee ligament rating scale mean values were
91.2, 5.3, and 86.8, respectively, demonstrating a statistically significant improvement
from preoperative status (p = 0.001). No Arthrotek graft-tensioning boot was used in
this series of patients.
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The conclusions drawn from the study were that combined ACL/PCL instabilities could
be successfully treated with arthroscopic reconstruction and the appropriate collateral
ligament surgery. Statistically significant improvement was noted from the
preoperative condition at 2- to 10-year follow-up using objective parameters of knee
ligament rating scales, arthrometer testing, stress radiography, and physical
examination. Postoperatively, these knees are not normal, but they are functionally
stable. Continuing technical improvements will most likely improve future results.
A more recent study presents the 2-year follow-up results of 15 arthroscopic assisted
ACL/PCL reconstructions using the Arthrotek graft-tensioning boot (14). This study
group consists of 11 chronic and four acute injuries. These injury patterns included six
ACL/PCL/PLC injuries, four ACL/PCL/MCL injuries, and five ACL/PCL/PLC/MCL
injuries. The Arthrotek tensioning boot was used during the procedures as in the
surgical technique described previously. All knees had grade III preoperative ACL/PCL
laxity, and were assessed pre- and postoperatively using Lysholm, Tegner, and Hospital
for Special Surgery knee ligament rating scales, KT 1000 arthrometer testing, stress
radiography, and physical examination.
Arthroscopically assisted combined ACL/PCL reconstructions were performed using the

single-incision endoscopic ACL technique, and the single femoral tunnel-single-bundle
transtibial tunnel PCL technique. Posterior cruciate ligaments were reconstructed with
allograft Achilles tendon in all 15 knees. Anterior cruciate ligaments were
reconstructed with Achilles tendon allograft in all 15 knees. MCL injuries were treated
surgically using primary repair, posteromedial capsular shift, and allograft
augmentation as indicated. Posterolateral instability was treated with allograft
semitendinosus free graft, with or without primary repair, and posterolateral capsular
shift procedures as indicated. The Arthrotek graft-tensioning boot was used in this
series of patients.
Post reconstruction physical examination results revealed normal posterior

drawer/tibial step-off in 13/15 (86.6%) of knees. Normal Lachman test in 13/15 (86.6%)
knees, and normal pivot shift tests in 14/15 (93.3%) knees. Posterolateral stability was
restored to normal in all knees with posterolateral instability when evaluated with the
external rotation thigh foot angle test (9 knees equal to the normal knee, and 2 knees
tighter than the normal knee). Thirty-degree varus stress testing was restored to
normal in all 11 knees with posterolateral lateral instability. Thirty- and zero-degree
valgus stress testing was restored to normal in all nine knees with medial side laxity.
Postoperative KT-1000 arthrometer testing mean side-to-side difference
measurements were 1.6 mm (range, 3 to 7 mm) for the PCL screen, 1.6 mm (range, 4.5
to 9 mm) for the corrected posterior, and 0.5 mm (range, 2.5 to 6 mm) for the
corrected anterior measurements, a significant improvement from preoperative
status. Postoperative stress radiographic side-to-side difference measurements
measured at 90 degrees of knee flexion and 32 lbs of posteriorly directed proximal
force using the Telos stress radiography device were 0 to 3 mm in 10/15 knees (66.7%),
4 mm in 4/15 knees (26.7%), and 7 mm in 1/15 knees (6.67%). Postoperative Lysholm,
Tegner, and Hospital for Special Surgery knee ligament rating scale mean values were
86.7 (range, 69 to 95), 4.5 (range, 2 to 7), and 85.3 (range, 65 to 93), respectively,
demonstrating a significant improvement from preoperative status.
The study group demonstrates the efficacy and success of using a mechanical graft-
tensioning device (Arthrotek graft-tensioning boot) in single-bundle-single femoral
tunnel arthroscopic PCL reconstruction.
Another group of PCL-based ligament reconstructions of interest are our 2- to 10-year

results of combined PCL-posterolateral reconstruction (11). This study presented the
2- to 10-year (24 to 120 month) results of 41 chronic arthroscopically assisted
combined PCL/posterolateral reconstructions evaluated pre- and postoperatively using
Lysholm, Tegner, and Hospital for Special Surgery knee ligament rating scales, KT 1000
arthrometer testing, stress radiography, and physical examination.
This study population included 31 males, 10 females, 24 left, and 17 right chronic
PCL/posterolateral knee injuries with functional instability. The knees were assessed
pre- and postoperatively with arthrometer testing, three different knee ligament
rating scales, stress radiography, and physical examination. Posterior cruciate
ligament reconstructions were performed using the arthroscopically assisted single
femoral tunnel-single-bundle transtibial tunnel PCL reconstruction technique using
fresh frozen Achilles tendon allografts in all 41 cases. In all 41 cases, posterolateral
instability reconstruction was performed with combined biceps femoris tendon
tenodesis, and posterolateral capsular shift procedures. The paired t-test and power
analysis were the statistical tests used. Ninety-five percent confidence intervals were
used throughout the analysis.
Postoperative physical examination revealed normal posterior drawer/tibial step-off in

29/41 (70%) of knees for the overall group, and 11/12 (91.7%) normal posterior drawer
and tibial step-off in the knees tensioned with the Arthrotek tensioning boot.
Posterolateral stability was restored to
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normal in 11/41 (27%) of knees, and tighter than the normal knee was achieved in
29/41(71%) of knees evaluated with the external rotation thigh foot angle test. Thirty-
degree varus stress testing was normal in 40/41 (97%) of knees, and grade 1 laxity was
found in 1/41 (3%) of knees. Postoperative KT 1000 arthrometer testing mean side-to-
side difference measurements were 1.80 mm (PCL screen), 2.11 mm (corrected
posterior), and 0.63 mm (corrected anterior). This is a statistically significant
improvement from preoperative status for the PCL screen and the corrected posterior
measurements (p = 0.001). The postoperative stress radiographic mean side-to-side
difference measurement with 90 degrees of knee flexion and 32 lbs of posterior
directed force applied to the proximal tibia using the Telos device was 2.26 mm. This
is a statistically significant improvement from preoperative measurements (p = 0.001).
Postoperative Lysholm, Tegner, and Hospital for Special Surgery knee ligament rating
scale mean values were 91.7, 4.92, and 88.7, respectively, demonstrating a
statistically significant improvement from preoperative status (p = 0.001).
Conclusions drawn from this study were that chronic combined PCL/posterolateral
instabilities could be successfully treated with arthroscopic PCL reconstruction, using
fresh frozen Achilles tendon allograft combined with posterolateral corner
reconstruction, using biceps tendon transfer combined with posterolateral capsular
shift procedure. Statistically significant improvement is noted (p = 0.001) from the
preoperative condition at 2- to 10-year follow-up using objective parameters of knee
ligament rating scales, arthrometer testing, stress radiography, and physical
examination. Use of the Arthrotek Graft-Tensioning Boot is advised since postoperative
physical examination revealed normal posterior drawer/tibial step-off in 29/41 (70%)
of knees for the overall group, and 11/12 (91.7%) normal posterior drawer and tibial
step-off in the knees tensioned with the Arthrotek tensioning boot.
FUTURE DIRECTIONS
We have chosen to perform the double-bundle-double-femoral tunnel PCL
reconstruction surgical technique since there are convincing basic science data
supporting the efficacy of this procedure (21). This double-bundle-double femoral
tunnel technique more closely approximates the anatomic insertion site of the native
PCL, and should theoretically provide improved results. Our early clinical results are
encouraging; however, there are no long-term clinical results available as of this
writing.
Another area of interest is the incorporation of autologous platelet-rich fibrin matrix

into the grafts used in the cruciate and collateral ligament reconstructive procedures
(Fig. 18-20). There are several studies indicating favorable effects on the ligament
graft tissue and the clinical results (24,25,26). We have demonstrated favorable initial
clinical results with respect to graft incorporation, wound healing, and early stability;
however, there is no long-term follow-up as of this writing.
FIGURE 18-20
A platelet-rich fibrin matrix clot created by the Musculoskeletal Transplant
Foundation Cascade System is incorporated into the graft during the
tubularization process to potentially enhance graft incorporation in the tunnels.
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CONCLUSIONS
The arthroscopically assisted single-bundle-transtibial PCL reconstruction technique is
a reproducible surgical procedure. There are documented results demonstrating
statistically significant improvements from preoperative to postoperative status
evaluated by physical examination, knee ligament rating scales, arthrometer
measurements, and stress radiography. Factors contributing to the success of this
surgical technique include identification and treatment of all pathology (especially
posterolateral and posteromedial instability), accurate tunnel placement, placement
of strong graft material at anatomic graft insertion sites, minimizing graft bending,
performing final graft tensioning at 70 to 90 degrees of knee flexion using the
Arthrotek graft-tensioning boot, using primary and back-up fixation, and the
appropriate postoperative rehabilitation program. Due to a more anatomic
reconstruction, double-bundle reconstruction may provide better results. This will
need to be demonstrated in long-term clinical studies.
REFERENCES
1. Daniel DM, Akeson W, O'Conner J, eds. Knee ligamentsâ€”structure, function,
injury, and repair. New York: Raven Press; 1990.
2. Fanelli GC. Arthroscopic evaluation of the PCL. In: Fanelli GC, ed. Posterior
cruciate ligament injuries. A guide to practical management. New York: Springer-
Verlag; 2001.
3. Fanelli GC. Arthroscopic PCL reconstruction: transtibial technique. In: Fanelli

GC, ed. Posterior cruciate ligament injuries. A guide to practical management.
New York: Springer-Verlag; 2001.
4. Fanelli GC. Arthrotek PCL reconstruction surgical technique guide. Fanelli PCL-
ACL drill guide system. Arthrotek, Inc. Warsaw, Indiana; 1998.
5. Fanelli GC. Complications in PCL surgery. In: Fanelli GC, ed. Posterior cruciate
ligament injuries. A guide to practical management. New York: Springer-Verlag;
2001.
6. Fanelli GC. PCL injuries in trauma patients. Arthroscopy 1993;9(3):291-294.
7. Fanelli GC. Point counter point. Arthroscopic posterior cruciate ligament

reconstruction: single bundle/single femoral tunnel. Arthroscopy. 2000;16(7):725-
731.
8. Fanelli GC. Rationale and surgical technique for PCL and multiple knee ligament
reconstruction. Surgical technique guide. Arthrotek, Inc.; 2006.
9. Fanelli GC. Surgical treatment of ACL-PCL-medial side-lateral side injuries of

the knee. Operative techniques in sports medicine. 2003;11(4):263-274.
10. Fanelli GC, Edson CJ. Arthroscopically assisted combined ACL/PCL

reconstruction. 2-10 year follow-up. Arthroscopy. 2002;18(7):703-714.
11. Fanelli GC, Edson CJ. Combined posterior cruciate ligament-posterolateral

reconstruction with Achilles tendon allograft and biceps femoris tendon tenodesis:
2-10 year follow-up. Arthroscopy. 2004;20(4):339-345.
12. Fanelli GC, Edson CJ. Management of posterior cruciate ligament and
posterolateral instability of the knee. In: Chow J, ed. Advanced Arthroplasty. New
York: Springer-Verlag; 2001.
13. Fanelli GC, Edson CJ. PCL injuries in trauma patients. Part II. Arthroscopy.
1995;11:526-529.
14. Fanelli GC, Edson CJ, Orcutt DR, et al. Treatment of combined ACL-PCL medial
lateral side injuries of the knee. Journal of Knee Surgery. 2005;28 (3):240-248.
15. Fanelli GC, Giannotti BF, Edson CJ. Arthroscopically assisted combined anterior
and posterior cruciate ligament reconstruction. Arthroscopy. 1996;12(1):5-14.
16. Fanelli GC, Giannotti B, Edson CJ. Arthroscopically assisted PCL/posterior

lateral complex reconstruction. Arthroscopy. 1996;12(5).
17. Fanelli GC, Giannotti B, Edson CJ. Current concepts review. The posterior
cruciate ligament: arthroscopic evaluation and treatment. Arthroscopy.
1994;10(6):673-688.
18. Fanelli GC, Monahan TJ. Complications and pitfalls in posterior cruciate
ligament reconstruction. In: Malek MM, ed. Knee Surgery: complications, pitfalls,
and salvage. New York: Springer-Verlag; 2001.
19. Fanelli GC, Monahan TJ. Complications of posterior cruciate ligament

reconstruction. Sports Medicine and Arthroscopy Review. 1999;7(4):296-302.
20. Fanelli GC, Orcutt DR, Edson CJ. Current concepts: the multiple ligament
injured knee. Arthroscopy. 2005;21(4): 471-486.
21. Harner CD, Janaushek MA, Kanamori A, et al. Biomechanical analysis of a

double-bundle posterior cruciate ligament reconstruction. Am J Sports Med.
2000;28:144-151.
22. Malek MM, Fanelli GC. Technique of arthroscopically PCL reconstruction.

Orthopaedics. 1993;16(9):961-966.
23. Miller MD, Cooper DE, Fanelli GC, et al. Posterior cruciate ligament: current
concepts. In: Beaty JH, ed. American Academy of Orthopaedic Surgeons
Instructional Course Lectures. 2002;51:347-351. Available from the American
Academy of Orthopaedic Surgeons, Rosemont, IL.
24. Sanchez M, Azofra J, Aizpurua B, et al. Application of growth factor rich

autologous plasma in arthroscopic surgery. Cuadernos de Arthroscopia.
2003;10:12-19.
25. Weiler A, Forster C, Hunt P, et al. The influence of locally applied platelet-
derived growth factor BB on free tendon graft remodeling after anterior cruciate
ligament reconstruction. Am J Sports Med. 2004;4:881-891.
26. Yasuda K, Tomita F, Yamazaki S, et al. The effect of growth factors on

biomechanical properties of the bone patellar tendon bone graft after anterior
> Table of Contents > Part Three - Ligament Injuries and Instability > 19 - Double-Bundle
Posterior Cruciate Ligament Reconstruction
19
Reconstruction
Posterior cruciate ligament (PCL) injuries are rare and infrequently occur in isolation.
Although we have an increased knowledge of the anatomy and mechanism of injury,
PCL reconstruction has not obtained the success of anterior cruciate ligament (ACL)
reconstruction. In addition, frustrating to the surgeon is the lack of consensus
regarding the optimal management of PCL injuries. Isolated PCL injuries (grades I and
II) can be treated nonoperatively with protected weight-bearing and quadriceps
muscle rehabilitation. However, some studies have demonstrated degenerative
changes and poor objective outcomes associated with conservative treatment of PCL
injuries.
The absolute indications for reconstruction of an isolated PCL tear would be for
persistent instability (grade III) or a boney avulsion. However, some authors dispute
the existence of an isolated PCL injury. Regardless, most PCL reconstructions occur in
conjunction with a knee dislocation. Advances in the understanding of the anatomy
and biomechanics of the PCL have led to an increased interest in arthroscopic
reconstruction of the PCL. Anatomic studies have delineated separate characteristics
of the anterolateral (AL) and posteromedial (PM) bundles within the PCL. The larger AL
bundle has increased tension in flexion, whereas the PM bundle becomes more taut in
extension. In addition, the smaller meniscofemoral ligaments also contribute to the
overall strength of the PCL. The timing of PCL reconstruction is controversial, but an
acute reconstruction is generally accepted for boney avulsion injuries or while
addressing combined ligamentous injuries-especially a posterolateral corner injury.
The double-bundle PCL reconstruction technique is most commonly performed in the
chronic setting, when very little native ligament remains available for augmentation.
Contraindications to an acute PCL reconstruction would be in the setting of a

traumatic knee arthrotomy or with a stiff knee. The double-bundle technique is not
recommended if any bundle remains intact. In this scenario, we prefer a single-bundle
augmentation, which is the most likely clinical presentation. We always attempt to
preserve any remnant tissue whenever possible unless located directly at the insertion
site of the target bundle. The grafts are passed around the meniscofemoral ligaments,
which does increase the complexity of the reconstruction.
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A thorough history and physical examination are obtained preoperatively in a clinic
setting. It is important to determine the chronicity of the injury. Acutely, there is
usually a history of a direct blow to the anterior lower leg or a hyperextension injury
with concomitant mild swelling. A PCL injury may also occur in the setting of a knee
dislocation with involvement of the posterolateral structures (approximately 60%).
Chronically, instability may be the only complaint.
On physical examination, it is important to assess the neurovascular status of the

injured limb. This is especially important if there is a history of a knee dislocation,
which has a higher risk of such an injury. Inspect and palpate for an effusion followed
by assessing the range of motion. With the knee in a flexed position, palpate for the
natural tibial step-off and evaluate the anterior border of tibial plateau in relation to
the femoral condyles. In addition, a Godfrey test can be performed whereby the lower
leg is elevated with the knee flexed approximately 90 degrees to assess for the
presence of a â€œsag.â€• A posterior drawer with the knee in 90 degrees of flexion is
also performed to evaluate the amount of posterior tibial translation (Fig. 19-1). It is
important to recognize the presence of a posterolateral corner knee injury, which will
accentuate the posterior drawer. The gait pattern is inspected for any varus thrust. If
a posterolateral corner injury is suspected, the physical examination should include a
reverse pivot shift test, a dial test, a posterolateral drawer, and varus stress testing at
both 30 and 90 degrees.
Radiographs of the knee should be performed to inspect for any fracture in the acute
setting or for any medial or patellofemoral compartmental arthrosis in the chronic
setting. Long-leg cassette films should be obtained if any coronal instability is
suspected. Posterior tibial subluxation can possibly be seen on the lateral radiograph.
Otherwise, a comparative stress lateral radiograph can also be obtained. A bone scan
may be useful in chronic PCL insufficiency to identify degenerative changes. A
magnetic resonance image (MRI) is essential not only to confirm a PCL injury, but more
importantly to assess for any concomitant posterolateral corner injury or meniscal
pathology that will affect the operative plan.
SURGERY
Patient Positioning
The patient is identified in the preoperative holding area and the operative site is
signed. Sciatic and femoral nerve blocks are performed by the anesthesiology staff.
The patient is taken to the operating room, where spinal or general anesthesia is
induced. The patient is positioned supine on the operating room table. A padded bump
is taped to the operating room table at the foot with the knee flexed to 90 degrees to
hold the leg flexed during the case. A side post is placed on the operative side just
distal to the greater trochanter to support the proximal leg with the knee in flexion.
Padded cushions
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are placed under the nonoperative leg. We do not routinely use a tourniquet so that
the vascular status of the extremity can be checked intraoperatively (Fig. 19-2).
FIGURE 19-1
The posterior drawer test is demonstrated. With a posteriorly directed force, the
anterior tibia can be subluxed beyond the medial femoral condyles.
Examination under Anesthesia

An examination under anesthesia (EUA) is then performed. The nonoperative knee is
examined followed by the operative knee. The alignment and range of motion are
assessed with specific attention to terminal extension and flexion. A thorough
ligamentous examination as previously described is then performed to confirm our
preoperative diagnosis and assess for any other pathology. Lower grades of posterior
drawer and reverse pivot shift tests may represent an intact AL or PM bundle of the
PCL. Fluoroscopy is used after the EUA. Lateral views are obtained with the tibia
displaced posteriorly and then reduced with an anterior force. The sloped posterior
tibial fossa is identified for later tibial tunnel placement.
Surface Anatomy and Skin Incisions

The knee is flexed to 90 degrees and the vertical arthroscopy portals are delineated.
The anterolateral portal is placed just lateral to the lateral border of the patellar
tendon and adjacent to the inferior pole of the patella. The anteromedial portal is
positioned approximately 1 cm medial to the medial border of the superior aspect of
the patellar tendon (Fig. 19-3). An accessory posteromedial portal is created just
proximal to the joint line and posterior to the medial collateral ligament under direct
arthroscopic guidance. If meniscal repair or other surgical procedures are expected,
these incisions are marked as well. The proposed incisions are then cleansed with
sterile Betadine paint and injected with a local anesthetic and epinephrine. The leg is
cleansed with an alcohol solution and Betadine, with care taken to preserve the
incision marks. The operative leg is draped using a sterile technique. A hole is cut in
the stockinette for access to the dorsalis pedis pulse throughout the case. After
draping, the arthroscopy equipment is prepared and the fluoroscopy machine is later
draped (Fig. 19-4).
FIGURE 19-2
The positioning and preparation are demonstrated. The knee rests comfortably at
90 degrees of flexion without the use of assistants.
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FIGURE 19-3
The landmarks are drawn on the skin. In this figure, the portal sites are marked as
well as the medial tibial incision for later tibial tunnel placement.
Graft Preparation
In general, two long (approximately 24 cm in length) tibialis anterior allografts are
preferred over a split allograft. The soft tissue allografts are thawed in sterile saline
solution. A whipstitch is placed in each of the free ends of the graft. A closed
Endoloop of 30 to 40 mm in length is used without the EndoButton (Smith & Nephew,
Inc., Andover, MA) (Fig. 19-5). With the standard EndoButton technique, a significant
portion of the tunnel will contain only the Endoloop suture, with some graft left
outside the tibial tunnel. The EndoButton is carefully removed using a wire cutter to
avoid damage to the loop. This is done to minimize the amount of Endoloop and
maximize the amount of graft within the femoral tunnel.
The allograft is passed through and doubled over the Endoloop, halving its length to 12
cm. A No. 5 braided suture is placed through the Endoloop and the graft is tensioned.
The graft is marked 25 mm from the Endoloop end for later referencing when passing
the graft into the femoral tunnel and 50 mm from this mark to represent the intra-
articular portion of the graft. The width of the graft is measured on the tibial and
femoral ends. The desired AL allograft width is 9 to 10 mm versus 7 mm for the PM
allograft. The graft is kept in a damp sponge until needed for graft passage.
FIGURE 19-4
Fluoroscopy is used for proper placement of the tibial tunnel. The knee can be
easily visualized in this setting when placed at 90 degrees.
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FIGURE 19-5
A tibialis anterior allograft is prepared with an Endoloop attached. Two marks are
placed, one at 25 mm of graft and the other at 75 mm. Between the designated
marks lies the intra-articular portion of the allograft. Both grafts are marked in
similar fashion.
Arthroscopy
A thorough diagnostic arthroscopy is performed to assess the cruciate ligaments and
evaluate for other cartilage or meniscal derangements. The notch is examined for any
remaining intact PCL fibers of the AL or PM bundles. If specific fibers from either
bundle or the meniscofemoral ligaments are left, care should be taken to preserve
these fibers during the case for augmentation. Frequently, the AL bundle is ruptured
and the PM bundle remains intact.
Overlying synovium and ruptured PCL fibers are debrided. An arthroscopic

electrocautery device and shaver are used to define the superior interval between the
ACL and PCL. A 70-degree arthroscope is routinely used when viewing through the
notch. The arthroscope is placed between the PCL remnants and the medial femoral
condyle (Gillquist view) to assess the posterior horn of the medial meniscus. This view
is also used for spinal needle localization of the posteromedial portal. The Gillquist
view and the interval between the intact ACL and torn PCL remnants (transseptal
view) are routinely used throughout the case to view the tibial footprint. The 30-
degree arthroscope is used when viewing via the posteromedial portal. A switching
stick may be placed into the posteromedial portal to facilitate exchange of the
arthroscope.
The 70-degree arthroscope is placed into the anterolateral portal, and a commercially
available PCL curette is introduced through the anteromedial portal. The PCL curette
(and later the PCL drill guide) is placed in proper relationship to the preserved PCL
bundle footprints. Once the curette is in place, a lateral fluoroscopic image may be
obtained to confirm its position. The 30-degree arthroscope is introduced through the
posteromedial portal. The soft tissue on the posterior aspect of the tibia is carefully
elevated centrally and slightly lateral. A transseptal portal is created for access to the
tibial PCL insertion. A shaver may be placed from the anterolateral portal to debride
some of the surrounding synovium with care taken to preserve the PCL origin. The
camera should be switched to the 70-degree arthroscope in the anterolateral portal
and the shaver placed in the posteromedial portal for further debridement. Adequate
preparation of the posterior tibia can be technically demanding and tedious, but it is
essential for proper exposure and safety in drilling the tunnel.
Tunnel Location and Preparation

Tibial Tunnel Preparation
To begin tunnel location and preparation (Figs. 19-6 and 19-7), a commercially
available PCL tibial drill guide is placed through the anteromedial portal. The guide is
set to 50 degrees, and its position is checked with a lateral fluoroscopic view and via
the posteromedial arthroscopic view (Fig. 19-8). The superior tibial insertion site of
the PCL begins in a fossa located between the two palpable prominences of the medial
and lateral plateaus of the tibia. The recommended landmark for placement of this
guide is approximately 1.5 cm distal to the articular edge of the posterior plateau,
which corresponds to the junction of the middle and distal 1/3 of the posterior tibial
slope (Fig. 19-9).
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FIGURE 19-6
Schematic representation of the femoral and tibial insertion sites of the two
bundles of the PCL. AL, anterolateral; PM, posteromedial.
FIGURE 19-7
Schematic representation of the two bundles after graft passage.
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FIGURE 19-8
With the arthroscope placed in the posteromedial portal as shown, the tibial
footprint can be visualized and the tibial guide correctly placed.
The guide is aligned on the anteromedial aspect of the tibia and an incision is made.
Dissection is performed through periosteum down to bone. In contrast, a small incision
on the anterolateral surface of the tibia can be used for placement of the tibial AL
tunnel to provide tunnel separation from the PM tibial tunnel. Otherwise, if both
tunnels are going to be made on the anteromedial aspect of the tibia, care is taken to
avoid tunnel convergence and ensure that adequate separation with a bony bridge
exists between the two tibial tunnels. The PCL guide is reset in the lateral aspect of
the PCL tibial footprint and its position is again confirmed with fluoroscopy and
arthroscopy. The guide is then clamped down onto the anterior tibial surface. A
guidewire is drilled up to but not through the posterior cortex. Another fluoroscopic
image is obtained to confirm the path of the guidewire, and the drill guide is removed
(Fig. 19-10). The PCL curette is introduced through the anteromedial portal, and the
30-degree arthroscope is placed in the posteromedial portal. The curette is used to
protect the posterior knee structures as the guidewire is carefully advanced through
the posterior cortex
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under arthroscopic visualization. A parallel pin guide can be used to make small pin
placement corrections if necessary. The same steps and precautions are repeated for
placement of the PM tibial guidewire. However, it is placed more medial and slightly
proximal to the AL tibial guidewire (Fig. 19-11). Once again, it is important to ensure
that adequate separation between the two guide pins exists to accommodate both
tunnels with a bony bridge separation. Once the guidewire positions are satisfactory, a
cannulated compaction reamer is used to drill the AL tibial tunnel first. Drill
advancement is performed under fluoroscopic guidance, but the posterior tibial cortex
is cautiously perforated by hand reaming under arthroscopic visualization. The tunnel
is irrigated and increasing serial dilators are used under arthroscopic visualization up
to the graft size (Fig. 19-12). The steps are repeated for drilling the PM tibial tunnel
with a 7-mm cannulated compaction reamer.
FIGURE 19-9
View from the posteromedial portal demonstrating the guide being placed for the
anterolateral tibial tunnel.
FIGURE 19-10
Fluoroscopic image demonstrating tibial guide pin placement for the

anterolateral tibial tunnel.
Femoral Tunnel Preparation

The shaver is used to debride any soft tissue off of the lateral wall of the medial
femoral condyle, with care taken to preserve any remnant PCL bundle tissue. An
angled awl is used to create a starting hole at the appropriate position for the bundle
being reconstructed. For the AL bundle, the starting hole is placed at the 1:00 or 11:00
position for right and left knees, respectively. This will place the superior edge of the
tunnel at the 12:00 position. The anteroposterior distance from the articular cartilage
of the medial femoral condyle depends on the size
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of the graft, but the tunnel edge should be located at the junction with the articular
cartilage. A 3/32 guidewire is placed via the anterolateral portal and impacted into
the starting hole.
FIGURE 19-11
View from the posteromedial portal demonstrating the placement of both the AL
and PM guide pins within the tibial footprint. In this figure, the AL guide pin is on
the right side and distal.
The appropriately sized cannulated acorn reamer is passed over the guidewire. The
reamer should be passed carefully given the close proximity of the patellar articular
surface. The tunnel is drilled to a depth of approximately 30 mm, with care taken to
avoid penetration of the outer cortex of the medial femoral condyle. Increasing serial
dilators are passed to match the size of the graft. A smaller EndoButton drill is used to
perforate the outer cortex of the medial femoral condyle.
This inside-out femoral tunnel preparation technique is then repeated for the PM
tunnel. The angled awl is used again to create the starting hole at the 3:00 or 9:00
position for right and left knees, respectively. The PM tunnel is placed parallel or
slightly posterior to the AL tunnel. The 3/32 guide pin is then placed via the
anterolateral portal and impacted into the starting hole. A 7-mm acorn reamer is
passed over the guidewire and drilled to a depth of approximately 30 mm. The medial
femoral condylar cortex is perforated with the EndoButton drill (Fig. 19-13).
The anterolateral portal may require a larger incision to ensure passage of the graft.
Arthroscopic graspers are placed via the anterolateral portal through the notch under
direct visualization with the 30-degree arthroscope in the anteromedial portal. The
30-degree arthroscope is then placed in the posteromedial portal. The AL graft is
passed first. A long 18-gauge bent wire loop is passed retrograde through the AL tibial
tunnel with the loop bent upward. The previously placed arthroscopic graspers are
used to retrieve the bent wire loop. The wire loop is advanced out the anterolateral
portal. Leading sutures from the free ends (tibial side) of the graft are placed through
the wire loop and the wire is pulled back through the tibial tunnel with the sutures in
an anterograde fashion. The free ends of the graft can be visualized passing through
the notch and into the tibial tunnel, with the femoral-sided leading suture remaining
in the anterolateral portal. A small scooped malleable is introduced through the
anterolateral portal and placed just posterior to the AL tunnel to retract the fat pad
and provide an unobstructed path for the beath pin. The beath pin is inserted through
the anterolateral portal and through the AL femoral tunnel and exits the skin on the
medial thigh. Care is taken to avoid penetrating soft tissue with the beath pin while
passing through the anterolateral portal because this will cause the graft to be
entangled in the soft tissue. The lead suture limbs from the Endoloop side are
threaded through the eye of the Beath pin, and the pin with the suture limbs is pulled
proximally. Traction on the suture limbs pulls the graft into the femoral tunnel to the
marked line, which is confirmed arthroscopically (Fig. 19-14). Tag the suture ends of
the graft together. This process is then repeated for the PM graft. It can be
cumbersome passing the second graft. Always keep traction on the AL graft suture
ends when passing the PM graft to ensure that the AL graft does not inadvertently get
pulled into the joint (Fig. 19-15).
FIGURE 19-12
A dilator can be seen penetrating the AL tibial tunnel. The PM tibial tunnel is
drilled and a curette is placed over the tip of the pin when reaming to protect
the neurovascular structures.
FIGURE 19-13
The AL and PM femoral tunnels have been drilled on the face of the medial
femoral condyle.
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FIGURE 19-14
The AL graft has been passed, and the PM tunnel can be visualized inferior to the
graft on the medial femoral condyle.
Next, an incision is made parallel to Langer's lines over the anteromedial aspect of the
distal medial femoral condyle, at the estimated exit of the graft suture ends from the
cortex. The vastus medialis obliquus fascia and muscle are identified and either
elevated and retracted en masse or split in line with their fibers depending where the
sutures are located. The muscle and periosteum are elevated off of the anteromedial
distal femur and the drill holes are exposed.
Graft Fixation
The closed Endoloop is placed in position along the medial femur with a tonsil to
estimate its most proximal extent. Because this is frequently positioned on a sloped
surface and bicortical purchase may not be possible, a 3.2-mm drill bit is used to drill
a unicortical hole at the most proximal extent of the Endoloop. After measuring the
length and using a 6.5-mm tap, a commercially available 6.5mm cancellous screw and
washer are placed through the Endoloop into the femur as the graft is pulled tight
distally. The fixation is palpated to ensure the Endoloop limbs are tight distal to the
screw and washer. This process is repeated for the second graft. Ensure enough
separation exists between the two screws and washers to prevent overlap.
FIGURE 19-15
A: After femoral and tibial fixation, the final placement is visualized from the
lateral portal in a left knee. The anterolateral graft almost completely obstructs
the view of the PM graft from this view. B: Final view of a double-bundle PCL
reconstruction in a right knee.
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An anterior tibial force is applied to reduce the tibia before and during final tibial
fixation. In similar fashion to the femoral side, two bicortical 4.5-mm cortical screws
and washers are placed from anteromedial to posterolateral within the proximal tibia.
The amount of knee flexion at final fixation depends on which PCL bundle is being
secured. The AL graft is secured first at 90 degrees flexion followed by the PM bundle
at approximately 15 degrees of flexion. Before the screw advances to the second
cortex, the suture limbs from the tibial side of the graft are tied with tension over the
post, and then the screw is tightened. The arthroscope is inserted to confirm adequate
position, tension, and fixation of the grafts.
Closure
Both incisions are irrigated, and the fascia in the anterolateral femoral incision is
closed with 0 vicryl suture. The subcutaneous layer is approximated with interrupted,
inverted 3-0 vicryl suture and the skin is closed with a running 4-0 absorbable suture.
The portals are closed with 3-0 nylon suture. The dorsalis pedis and posterior tibialis
pulses are assessed by palpation and a Doppler ultrasound if necessary. The incisions
are covered with adaptic gauze and sterile gauze then wrapped in cast padding and
bias wrap. Assess knee motion to ensure that it is not captured. The anterior tibial
step-off should be re-established. A hinged knee brace is applied and locked in
extension. Maintain an anterior drawer at all times when handling the leg. The patient
is awakened and taken to the recovery room, where the patient's pain and
neurovascular status are re-evaluated.
Pearls and Pitfalls
It is important to assess for concomitant posterolateral corner (PLC) injury on

the EUA and following PCL reconstruction. Deficiency of these structures may
lead to graft failure.
Exposure of the posterior tibia is essential for the success of this technique.
This can be tedious but the surgeon should be careful and patient. Both
aggressive debridement and inadequate exposure can lead to neurovascular
injury.
A parallel pin guide can be used to make small corrections for placement of the
tunnels.
Perforate the posterior tibial cortex with the guide pins or reamers in a
controlled fashion under direct arthroscopic visualization by hand to avoid
neurovascular injury.
If resistance is met when attempting to place the acorn reamer over the guide
pin in the femur, it is most likely the patella. A smaller sized acorn reamer can
be used in this situation to make a starting hole. Then larger sized reamers can
be placed freehanded to dilate the tunnel to the appropriate size.
An arthroscopic switching rod can be placed via the posteromedial portal

between the graft and the posterior tibial cortex to help facilitate graft
passage by decreasing the friction.
Fluid extravasation during arthroscopy is also a concern. Lower extremity

compartments should be monitored throughout the procedure.
Patients are kept overnight for pain management and to monitor their neurovascular
status. Patients are given instructions for exercises (quadriceps sets, straight-leg
raises, and calf pumps) and crutch use before discharge the next day. Patients are
instructed to maintain a touchdown weight-bearing status for 1 week. Partial weight
bearing (30%) is initiated after the first postoperative visit. All dressing changes are
performed with an anterior tibial force applied.
The knee is locked in extension for the first week. Symmetric full hyperextension is
achieved and passive prone knee flexion, quadriceps sets, and patellar mobilization
exercises are performed with the assistance of a physical therapist for the first month.
Mini-squats are performed from 0 to 60 degrees after the first week, and increased to
90 degrees for weeks 3 to 4. The brace is unlocked at 4 weeks postoperatively and
usually discontinued at 8 weeks. Closed-chain terminal knee extension exercises may
begin at 8 weeks. Once full, pain-free range of motion is achieved, strengthening is
addressed.
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COMPLICATIONS
Complications are rare, but can happen. Failure to carefully position the extremity
with adequate padding can lead to neurapraxia. Loss of motion (usually decreased
flexion) can result from errors in graft positioning and excessive tensioning during
graft fixation or from inadequate range-of-motion exercises during postoperative
rehabilitation. Residual laxity can also occur. Injury to the popliteal vessels is an
infrequent but very serious complication. With the transtibial technique, great care
must be taken to ensure overpenetration of the posterior tibial cortex does not occur.
The surgeon should regularly palpate the vessels about the ankle for any diminished
pulses. In addition, the thigh and calf should be routinely palpated to ensure no
compartmental syndrome develops from fluid extravasation into the soft tissues.
Avascular necrosis of the medial femoral condyle can be avoided by placing the
femoral tunnel entry-exit points proximal enough to allow preservation of the
subchondral bone.
RESULTS
Recent biomechanical in vitro cadaveric studies have documented the advantage of a
double bundle PCL construct. Race and Amis demonstrated that anatomic double
bundle PCL reconstruction with a bone-patellar tendon-bone graft more closely
approximated normal knee kinematics in a cadaver model compared with single bundle
reconstruction (2). In addition, Harner et al. published a paper supporting double
bundle PCL reconstruction with a transtibial Achilles allograft (1). Furthermore, a
biomechanical cadaveric study noted no difference regarding a transtibial versus a
tibial inlay technique for PCL reconstruction.
Clinical double-bundle PCL reconstruction studies are in evolution. Wang et al. is the
only group to publish a randomized prospective study (3). Thirty-five patients
underwent either single or double-bundle transtibial PCL reconstruction with an
Achilles allograft. No statistically significant clinical or radiographic differences were
demonstrated.
RECOMMENDED READING
Ahn JH, Chung YS, Oh I. Arthroscopic posterior cruciate ligament reconstruction using
the posterior transseptal portal. Arthroscopy. 2003;19(1):101-107.
Fox RJ, Harner CD, Sakane M, et al. Determination of the in situ forces in the human
posterior cruciate ligament using robotic technologyâ€”a cadaveric study. Am J Sports
Med. 1998;26(3):395-401.
Giffin JR, Haemmerle MJ, Vogrin TM, et al. Single- versus double-bundle PCL
reconstruction: a biomechanical analysis. J Knee Surg. 2002;15(2):114-120.
Harner CD, Janaushek MA, Kanamori A, et al. Biomechanical analysis of a double-
bundle posterior cruciate ligament reconstruction. Am J Sports Med. 2000;28(2):144-
151.
Margheritini F, Mauro CS, Rihn JA, et al. Biomechanical comparison of tibial inlay
versus transtibial techniques for posterior cruciate ligament reconstruction: analysis of
knee kinematics and graft in situ forces. Am J Sports Med. 2004;32(3):587-593.
Margheritini F, Rihn JA, Mauro CS, et al. Biomechanics of initial tibial fixation in
posterior cruciate ligament reconstruction. Arthroscopy. 2005;21(10):1164-1171.
Noyes FR, Barber-Westin S. Posterior cruciate ligament replacement with a two-strand

quadriceps tendon-patellar bone autograft and a tibial inlay technique. J Bone Joint
Surg Am. 2005;87(6):1241-1252.
Sekiya JK, Haemmerle MJ, Stabile KJ, et al. Biomechanical analysis of a combined
double-bundle posterior cruciate ligament and posterolateral corner knee
reconstruction. Am J Sports Med. 2005;33(3):360-369.
Sekiya JK, West RV, Ong BC, et al. Clinical outcomes after isolated arthroscopic single-
bundle posterior cruciate ligament reconstruction. Arthroscopy. 2005;21(9):1042-1050.
Shelbourne KD, Davis TJ, Patel DV. The natural history of acute, isolated,
nonoperatively treated posterior cruciate ligament injuries. A prospective study. Am J
Sports Med. 1999;27:276-283.
Shelbourne KD, Gray T. Natural history of acute posterior cruciate ligament tears. J
Knee Surg. 2002;15(2):103-107.
Sheps DM, Otto D, Fernhout M. The anatomic characteristics of the tibial insertion of
the posterior cruciate ligament. Arthroscopy. 2005;21(7):820-825.
Torg JS, Barton TM, Pavlov H, et al. Natural history of the posterior cruciate ligament-
deficient knee. Clin Orthop Relat Res. 1989;(246):208-216.
Yoon KH, Bae DK, Song SJ, et al. Arthroscopic double-bundle augmentation of
posterior cruciate ligament using split Achilles allograft. Arthroscopy.
2005;21(12):1436-1442.
REFERENCES
1. Harner CD, Vogrin TM, Hoher J, et al. Biomechanical analysis of a double-bundle
posterior cruciate ligament reconstruction. Deficiency of the posterolateral
structures as a cause of graft failure. Am J Sports Med. 2000;28(1):32-39.
2. Race A, Amis AA. PCL reconstruction-in vitro biomechanical comparison of

â€œisometricâ€• versus single- and double-bundle â€œanatomicâ€• grafts. J Bone
Joint Surg Br. 1998;80(1):173-179.
3. Wang JC, Weng LH, Hsu CC, et al. Arthroscopic single- versus double-bundle
posterior cruciate ligament reconstructions using hamstring autograft. Injury.
2004;35(12):1293-1299.
> Table of Contents > Part Three - Ligament Injuries and Instability > 20 - Inlay Posterior
Cruciate Ligament Reconstruction
20
Inlay Posterior Cruciate Ligament
Reconstruction
Michael A. Rauh
John A. Bergfeld
William M. Wind
Richard D. Parker
Treatment for posterior cruciate ligament (PCL) injuries has improved recently due in
part to greater understanding of the biomechanics and pathophysiology. However,
current understanding of this condition continues to lag behind that of the anterior
cruciate ligament (ACL) due in part to the relative infrequency with which this injury
is encountered. A ruptured PCL, either isolated or combined, is rare compared with
other ligamentous injuries of the knee, and accounts for only 5% to 20% of ligamentous
disruptions in the knee.
In addition, focused examination on the PCL with the â€œdialâ€• test, posterior
drawer test, and the quadriceps active test along with the development of improved
imaging techniques has greatly improved the physician's ability to provide a reliable
diagnosis differentiating between an isolated PCL injury and combined injury to the
PCL and other capsuloligamentous structures. Knowledge of the correct diagnosis and
observation of natural history of these injuries has allowed for the development of
improved treatment recommendations.
Most would agree that isolated injuries to the PCL are best treated nonoperatively
with adequate results. However, the natural history of isolated injury to the PCL is still
debated in the sports community. Definite episodes of instability are uncommon with
chronic PCL deficiency as opposed to that feature which is commonly seen after ACL
disruption. Our experience has shown a small percentage of patients with PCL
deficiency is reported to experience chronic pain and may exhibit degenerative
changes in the patellofemoral and medial compartments with time. Furthermore,
operative treatment of isolated PCL injuries has had inconsistent results.
Nonetheless, there is a subset of patients for which reconstruction of the PCL is

indicated: chronic, isolated grade 3 abnormal laxity of the PCL that has failed
nonoperative treatment, as well as acute or chronic PCL injury with combined
ligamentous injury. Identifying this subset of patients is the key to a successful
outcome from reconstruction of the PCL.
In the past, there have been multiple surgical procedures designed to reconstruct the
PCL, yet the literature reveals no long-term studies indicating normal stability
following a reconstruction. Early experience with PCL surgery using a transtibial tunnel
in a modified open procedure demonstrated that ligament reconstruction would result
in an initially stable knee; however, there would be considerable laxity by 1-year
follow-up.
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Surgical options include the transtibial and posterior tibial inlay reconstructions. The
transtibial method involves arthroscopic visualization of the tibial insertion of the PCL
with graft passage beyond the acute or commonly referred to â€œkiller turnâ€• at the
posterior aspect of the tibial plateau into the medial femoral condyle. With a single-
bundle reconstruction, the anterolateral bundle is usually reconstructed since it is
larger and stronger than the posteromedial bundle.The posterior inlay technique of
PCL replacement has gained recent popularity, and it is currently the technique used
for PCL reconstructions at our institutions. We feel that this technique is superior to
the other methods for several reasons. First, it is believed to more closely duplicates
the normal PCL anatomy. Second, it avoids the so-called â€œkiller curveâ€• found in
the arthroscopic-assisted posterior tibial tunnel method. Biomechanical studies have
demonstrated that the inlay reconstruction as well as the tibial tunnel reconstruction
both provide immediate stability (11). However, these initial studies also
demonstrated an area of thinning with the arthroscopic technique as the graft passed
around the tibial â€œkiller curve.â€• Subsequent studies demonstrated failure at this
location prior to completion of cyclic loading to 2,000 cycles (11,12). Third, the
operative technique is considerably easier and safer to perform although it is not an
all arthroscopic procedure.
Indications
Optimal management of PCL injuries continues to be an area of debate among
orthopaedic surgeons and health care professionals. Current treatment options can be
clearly categorized as operative and nonoperative. Nonsurgical treatment is advocated
for patients with acute isolated PCL injury, chronic isolated PCL injury in which the
patient has not participated in a formal physical therapy regimen, and those injuries
found in a patient who is unwilling or unable to participate in the postoperative
rehabilitation program (Fig. 20-1).
Operative indications for PCL reconstruction include patients found to have

â€œisolatedâ€• tears associated with functional disability, as well as patients with
other ligamentous injuries combined with complete PCL injury (Fig. 20-2).
Some authors extend their indications to include what are termed â€œcompleteâ€•
PCL tears. Clancy and Shelbourne has classified a PCL tear as â€œcompleteâ€• if the
anterior tibial plateau recedes beyond the distal aspect of the femoral condyles on
posterior drawer testing at 90 degrees of flexion. Bergfeld et al noted that if abnormal
laxity with the posterior drawer test does not decrease with internal rotation of the
tibia, the PCL injury is less likely to do well without surgery (2,3,4).
Two conditions which few would argue indicate for surgical intervention are:
Acute and chronic PCL injuries in combination with other capsuloligamentous

injuries
Chronic grade III isolated PCL injuries that are symptomatic.

FIGURE 20-1
Treatment algorithm for acute PCL injuries.
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FIGURE 20-2
Treatment algorithm for chronic PCL injuries.
Ultimately, the decision for surgical reconstruction of the PCL is based on the activity
level of the patient and whether the injury is isolated or in combination with other
ligament injuries. If the posterior drawer test does not decrease with tibial internal
rotation, suspicion of associated significant collateral ligament injury must be raised.
Clearly, a full discussion of the injury, natural history, as well as the risks and benefits
of operative and nonoperative treatment must occur with the patient before any
treatment decision.
Contraindications
An acute, isolated grade I or II PCL tear
Posterior drawer test with less than 10 mm abnormal posterior laxity at 90

degrees of flexion
Posterior drawer excursion decreasing with internal rotation of the tibia with
less than approximately 5 mm of abnormal posterior laxity
Less than 5 degrees of abnormal rotatory laxity
No significant varus or valgus laxity
Isolated rupture of the PCL in a patient who has not undergone a formal
physical therapy program focusing on quadriceps strengthening
Knee dislocation with arterial or venous injury resulting in limb compromise
Flexion contracture greater than 10 degrees or extension contracture greater

than 30 degrees
Regional pain syndrome
History
Complete medical history and physical examination are necessary. Orthopaedic
examination begins with a history of previous knee injury. Often, an individual has
sustained injuries to their knees which may go unnoticed or unremembered unless the
patient is specifically asked. It is important to know if the patient had issues with
patellar subluxation, meniscal symptoms, or had a prior ligamentous injury. This is
important as it can point the physician toward associated injuries and conditions that
may alter the treatment plan. Unfortunately, patients with PCL ruptures will
infrequently report feeling or hearing a â€œpopâ€• as is the case with an ACL rupture.
Motor vehicle accidents commonly induce PCL injuries. The knee is commonly flexed
when the individual is seated in a car, and the knee is subjected to a posterior-
directed force on the tibia. Similarly, the PCL can be ruptured during athletic
participation through a fall on the flexed knee, with the foot plantar flexed (Fig. 20-
3).
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FIGURE 20-3
Mechanism of injury for an isolated PCL injury.
The physical examination is crucial to identifying patients requiring reconstruction of
the PCL. Patients with an acute isolated injury to the PCL usually present with mild to
moderate effusion, and often have vague anterior or posterior soreness particularly
with flexion. Patients with acute combined injuries may have a more tense effusion.
Knee alignment should be noted. In cases of chronic PCL deficiency, attention to gait
and lower extremity alignment is performed. Individuals demonstrating a varus
alignment and varus thrust with ambulation suggest a combined PCL, posterolateral
corner injury.
A complete neurovascular examination of the lower extremity is performed on all

patients with suspected ligamentous injuries. Particular attention should be given to
the peripheral pulses and the function of the peroneal nerve. Individuals with intact
but diminished pulses or ankle-brachial indices less than 0.8 compared with the
opposite extremity should undergo observation or further investigation with an
angiogram based on vascular surgical consultation.
The integrity of the PCL is most accurately determined through the posterior drawer
test, which is performed with the knee in 70 to 90 degrees of flexion and the tibia in
neutral, external, and internal rotation. Before performing this test, the normal step-
off of the anterior tibia in relation to the femur should be assessed by comparing the
contralateral leg. Posterior subluxation of the tibia secondary to PCL insufficiency can
give a false impression on the posterior drawer test. If this is the case, an anterior
drawer should be performed first to reduce the tibia to its normal position followed by
the posterior drawer. Key information is obtained by performing the posterior drawer
test at 90 degrees in neutral, internal, and external rotation. An isolated injury to the
PCL will show a substantial decrease in the posterior drawer with internal rotation (4).
Ritchie et al found the drawer to decrease from 10 mm in neutral rotation to 4 mm
with internal rotation in an isolated PCL injury. This decrease is not seen with most
combined injuries. The posterior drawer test performed with external rotation, if
positive, is suggestive of a combined PCL/posterolateral corner injury.
Knees with isolated Posterolateral Corner (PCL) injuries should also not exhibit
increased varus or valgus laxity at 0 or 30 degrees of flexion. If present, this also
suggests a combined posteromedial or posterolateral injury. Combination PCL and
Posterolateral Corner (PLC) injuries are best determined by the tibial external rotation
(dial) test with the knee in both 30 and 90 degrees of flexion.
Although we rely heavily on the posterior drawer test, other important tests include
the quadriceps active test, posterior sag test, reverse pivot shift, external rotation
recurvatum, and external tibial rotation at 0 and 30 degrees. Again, the key is to
differentiate isolated from combined PCL injuries.
Radiographic Examination
Plain radiographs are the next step in evaluating PCL and knee injuries in general. We
routinely include bilateral standing anteroposterior, 45-degree flexion weight bearing,
Merchant patellar views, as well as a lateral view of the affected extremity. These
views allow the orthopaedic physician to document pre-existing arthritis, fractures,
joint spaces, knee alignment, as well as evaluate the slope of the proximal tibia.
Stress radiographs can be used, with only the risk of radiation and minimal cost, to
determine the sagittal translation between the injured and noninjured, presumably
intact, knee.
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FIGURE 20-4
MRI demonstrating a disrupted PCL (dark arrow) with posterior sag (white arrow).
Magnetic resonance imaging is felt to be between 96% and 100% sensitive, accurate,
and specific in determining PCL ruptures. Magnetic resonance imaging also provides
additional necessary information on the status of the remainder of the knee structures
such as the ACL, articular cartilage, and menisci (Fig. 20-4).
Graft Options
Graft options for PCL reconstruction include autografts such as bone-patellar-bone,
hamstring, and quadriceps. Limitations of these grafts include but are not limited to
donor site morbidity and increased operative duration. Allograft options such as
Achilles tendon, patella, posterior tibialis, and anterior tibialis tendon are often used
for the multiple ligament reconstructions; however, these are associated with a real
risk of viral transmission and do rely on creeping substitution for graft incorporation.
Despite these limitations, our present graft of choice is Achilles tendon allograft
obtained from a reputable bone bank.
SURGERY
Special Items to Have
Two operating room tables
Thirty- and seventy-degree arthroscopes with a full arthroscopy setup
Sterile tourniquet
PCL femoral drill guide
Graft passer (DePuy Graft Passer, Johnson & Johnson, New Brunswick, NJ)
Osteotomes
Standard 6.5-mm stainless steel cancellous screws with washer
PCL (vaginal) retractorsâ€”blunt tipped with differential angles of retraction
Patient Positioning
The patient is identified in the holding area and the operative extremity is
marked.
Preoperative prophylactic antibiotics (usually a first-generation cephalosporin)

are administered within 30 minutes of incision.
After induction of general anesthesia, an examination is performed of both

knees, again looking for associated capsuloligamentous injuries. Specifically,
the operating surgeon is responsible for determining the presence or absence
of injury to the posterolateral and posteromedial structures in the operative
extremity.
A â€œtime outâ€• is performed and again the operative extremity is identified

by the operating surgeon, the head circulating nurse, and the anesthesia team.
The surgery begins with the patient supine on the operating table.
Before the knee is prepped and draped, an intra-articular injection of 0.25%

bupivicaine with 1:400,000 epinephrine and 2 mg of morphine sulfate is given
into the operative knee.
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The opposite lower extremity is placed in a padded gynecologic stirrup that is
widely abducted away from the operative extremity. A Mayo stand cover does
well to provide a first sterile cover over the nonoperative leg.
The operative leg is positioned hanging free to allow for full knee flexion
during surgery. A lateral post is used to assist in arthroscopy treatment of
associated intra-articular pathology.
Technique
Arthroscopic Evaluation and Debridement
The procedure begins with a diagnostic arthroscopy without elevating the tourniquet.
Articular cartilage and meniscal pathology are addressed at this time. The PCL injury is
documented as well as the status of the ACL and meniscofemoral ligaments. The ACL
will usually appear lax secondary to the posterior tibial subluxation. An anterior
drawer will restore its normal appearance. The PCL remnant on the medial wall of the
intercondylar notch is debrided arthroscopically. Care is taken to preserve the intact
meniscofemoral ligament. Exposure of the notch allows for identification of the
femoral anatomic attachment site of the PCL complexâ€”composed of the PCL, and
the anterior and posterior meniscofemoral ligaments of Humphrey and Wrisberg,
respectively (FAAS). The FAAS is located anterior and distal in the notch,
approximately 8 to 10 mm from the articular margin at the 11 o'clock position in a left
knee and the 1 o'clock position in a right knee. The femoral footprint is identified, and
remnants are left to provide an anatomic landmark for placement of the femoral
tunnel guidewire. A curette is used to score the desired femoral tunnel site. The FAAS
is only marked at this stage as it will be rechecked later through a small medial
arthrotomy at the time of guide pin placement. Again, careful attention is paid to
preservation of the meniscofemoral ligaments. In particular, the anterior
meniscofemoral ligament of Humphrey remains intact and should remain so after
debridement and tunnel drilling. Note that if concomitant ACL surgery is planned, the
ACL femoral and tibial anatomic attachment sites are identified and drilled at this
time.
Graft Preparation
If using an Achilles tendon allograft, prepare the calcaneal bone plug for anticipated
implantation in the posterior tibial trough. Optimally, it should be 10 mm wide, 20 mm
long, and approximately 5 mm thick. This is the bone that will ultimately fit into the
trough to be created posteriorly. Using Krackow-type suture fixation, the tendinous
end of the Achilles graft is made tubular to allow for later passage through the femoral
tunnel and ultimate fixation. If using a bone-patellar tendon-bone autograft, the bone
plug from the patella is usually 10 mm wide and 30 mm long, whereas the bone plug
from the tibia should be 10 mm wide, 20 mm long, and approximately 5 mm thick. The
patellar bone plug should pass easily through a 9-mm sizer, and is fashioned to fit in
the femoral tunnel. The tibial bone plug should be fashioned to fit in a trough made at
the tibial attachment site of the PCL. The trough will correspond to the size of the
tibial bone plug. Once this is completed, use a 2-mm drill to make two holes in the
patellar bone plug, 5 mm and 15 mm from the distal tip, respectively. A No. 5
nonabsorbable suture is passed through each hole for later graft passage and
tensioning. The tibial plug should be fashioned into a rectangle 10 mm wide and 20
mm long.For either graft choice, a drill hole should be made with a 3.2-mm drill bit
through the center of this bone plug. The hole should be angled slightly distal to make
up for the slope of the posterior tibia, which will cause the screw to angle toward the
joint otherwise. The hole should be drilled from the cancellous side to avoid injury to
the soft tissue portion of the graft. The hole should be overdrilled with a 4.5-mm drill
and tapped accordingly. Next, a 6.5-mm partially threaded screw, 35 mm in length,
and washer should be inserted until it protrudes approximately 5 mm past the
cancellous surface. Alternatively, two small-fragment cancellous screws may be used
to fix the posterior bone plug. A cadaveric study in press performed by the lead author
showed no difference in pull-out strength between the two techniques (13). The graft
is now ready for implantation (Fig. 20-5).
Femoral Tunnel Creation

While the graft is being prepared by an assistant on the back table, the femoral tunnel
is made through a medial arthrotomy. If autograft is being used, it can be done
through the same incision as for graft harvest. A small medial arthrotomy is made
adjacent to the patellar tendon, extending to the vastus medialis at the level of the
adductor tubercle. The arthrotomy extends posteriorly along the inferior aspect of the
muscle, which is elevated anteriorly, revealing the medial femoral condyle (1). The
intercondylar notch is visualized by retracting the patella laterally. The previously
marked FAAS is identified and checked for accurate placement. Through the
arthrotomy the PCL femoral guide tip is
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placed at the FAAS, approximately 8 mm proximal to the articular margin. This should
leave the edge of the tunnel 2 to 3 mm from the articular edge after drilling. The
tunnel should be angled proximally and posteriorly to decrease the obliquity of the
femoral tunnel, which decreases the stress on the graft as it enters the tunnel. Using
this aiming guide, the femoral guide pin is then drilled from the outside to the inside.
As demonstrated by Handy et al, the PCL tracks at approximately 85 degrees. An
inside-out technique makes reproduction of this angle difficult. The anterior/distal
tunnel guide pin, reproducing the anterolateral bundle, should enter the intracondylar
notch at the 11 or 1 o'clock position as noted previously and optimally should be
approximately 8 to 10 mm proximal to the articular surface of the medial femoral
condyle. The wire should be directed lateral, posterior, and proximal, duplicating the
direction of the native PCL ligament (3). Ideally, graft preparation is occurring
simultaneously as this step. The diameter of the femoral tunnel is determined by
adding 1 mm to the size of the femoral/tendinous portion of the Achilles allograft.
Usual tunnel size is 10 mm. Tunnel sizers are used to obtain this measurement. The
edges are chamfered and smoothed with a rasp. A suture passing device is then placed
within the tunnel (DePuy Graft Passer, Johnson & Johnson, New Brunswick, NJ). Next,
an 18-gauge wire loop or commercially available graft passer is placed through the
femoral tunnel and directed posteriorly into the notch. This is placed posterior within
the notch so as to facilitate later graft passage through the posterior approach. The
wound is loosely approximated, and the tourniquet deflated if employed. A sterile
circumferential dressing is placed, and the entire leg is covered in a sterile bag (Mayo
stand cover). This is overwrapped with a long ACE wrap.
FIGURE 20-5
Prepared Achilles tendon allograft.
First Patient Turn, Burk's Approach to the Posterior Tibia

After having placed the operative extremity in a sterile circumferential dressing, a
second operating room table is positioned beside the patient. Under the direction of
the surgeon and anesthetist, the patient is turned to the prone position. We have
found the extra 10 to 15 minutes needed to perform this is well worth the effort. The
previous operating table should be cleaned and prepared to place the patient back in
the supine position after posterior dissection and graft placement. Ensure that all bony
prominences have been padded adequately. Once the patient is in the prone position,
the sterile dressing is removed. If there is any question of a break in sterile technique,
then the entire leg is reprepped and draped. A modified Burk approach to the
posterior tibia is performed (Fig. 20-6). The landmarks for the incision are the medial
border of the medial head of the gastrocnemius, the lateral border of the
semitendinosis, the popliteal crease, and the midline of the distal thigh (5). This
portion of the procedure is performed without elevating the tourniquet to ensure
adequate hemostasis is achieved. The skin is incised and the fascia exposed. The
medial sural cutaneous nerve is vulnerable near the midline at this level and should be
avoided if possible. The fascia is incised and the interval between the medial head of
the gastrocnemius and semitendinosis is developed (Fig. 20-7). The fascia over the
proximal popliteus muscle near the distal portion of the insertion of the PCL is incised
to allow for distal retraction of the popliteus muscle if necessary. A broad blunt
retractor is placed in this interval, and the medial head of the gastrocnemius is
retracted laterally protecting the neurovascular bundle. A portion of the medial
gastrocnemius origin can be released if extra exposure is needed, but we have usually
found this unnecessary. The inferior medial geniculate artery and vein may be ligated
as necessary. Next, the capsule is incised longitudinally and reflected medially and
laterally. This exposes the tibial insertion of the PCL. The remaining scar tissue and
PCL remnants are excised with care to avoid the meniscofemoral ligaments. To
facilitate exposure, the knee may be flexed 15 to 20 degrees and the tibia externally
rotated.
Creation of the Postesior Tibial Trough, Femoral Graft

Passage, and Graft Fixation
Once the insertion site is marked, a cortical window is made with an osteotome. The
window is approximately 11 Ã— 20 mm and corresponds to the bone plug previously
prepared (Fig. 20-8). The graft is now brought into the operative field. The femoral
plug is passed through the intercondylar notch and into the femoral tunnel using the
previously placed graft passer. Next, the tibial bone plug is fitted into the prepared
trough on the back of the tibia. The 6.5 screw is tightened securing the bone plug to
the posterior tibia. It is not necessary to predrill the cancellous bone. Tension is then
placed on the femoral end of the graft to ensure adequate fixation is present.
Intraoperative radiographs can be used to check screw placement. Once adequate
tibial fixation is achieved, the capsule is closed with No. 1 nonabsorbable suture, as
well as the fascia. The subcutaneous tissue is closed with a 2-0 absorbable suture, and
the skin is closed with a subcuticular closure. The leg is again covered with a sterile
Mayo stand cover and the patient is placed supine on the original operating table.
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FIGURE 20-6
Modified Burk and Shaffer posterior exposure with the surgical incision (solid line)
centered proximally on the medial head of the gastrocnemius. The incision can be
extended (dashed line) proximally for further exposure
FIGURE 20-7
Drawing (A) and clinical photograph (B) of the interval between the medial
gastrocnemius (dark arrow) and semimembranosus (light arrow).
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FIGURE 20-8
Bony trough created in the posterior tibia.
Second Patient Turn and Femoral Graft Fixation

The sterile dressing is removed. If there is any concern of breakdown of sterile
technique, the leg is reprepped and draped as described previously. With attention
and care, this step is not frequently necessary. With the knee flexed to 80 degrees in
neutral rotation, an anterior drawer is applied to the proximal tibia. The graft in the
femoral tunnel is tensioned in this position by pulling on the sutures in the femoral
bone plug. If using a bone-patellar tendon-bone graft, it is important to determine the
amount of bone within the femoral tunnel. If 20 to 25 mm of bone is in the femoral
tunnel, the graft is fixed with a 9 X 20 mm interference screw placed proximally to
secure the bone plug distally in the tunnel (closest to the articular surface). If less
than 20 mm of bone plug is within the femoral tunnel or an Achilles allograft is being
used, a trough is made in the medial femoral condyle and fixation is achieved with
either two spiked staples or by tying the sutures to a 6.5-mm post in the medial
femoral condyle. Alternatively, one may choose to use an interference screw within
the femoral tunnel with back-up staple fixation. After fixation, the knee is flexed and
extended to ensure an adequate range of motion. A gentle posterior drawer at 90
degrees can be performed, and the anterior tibial step-off should be restored. Once
the surgical team is satisfied with stability, the wounds are irrigated and a layered
closure is performed.
Pearls and Pitfalls
When using a bone patellar tendon bone graft, careful attention should be paid
to the tibial fixation site because the graft is a fixed length. Once the graft is
fixed on the posterior tibia, it is tensioned on the femoral side, and the
position of the bone plug in the femoral tunnel is assessed. If 25 mm or more of
the bone plug is within the femoral tunnel at this point, the tibial attachment
site is accepted. On the other hand, if less than 25 mm of bone is within the
femoral tunnel, the tibial attachment site can be adjusted distally. The tibial
side of the graft can be distalized 10 mm without affecting the mechanical
properties of the reconstruction. If it is felt that more than 10 mm of
distalization is needed, do not move the tibial attachment site, and fix the
femoral end in a trough as described previously.
It may be necessary to divide a portion of the upper (proximal) aspect of the

popliteus muscle to gain good exposure of the posterior tibia.
As mentioned previously, when placing the 6.5-mm screw in the tibial bone
plug, it is helpful to angle distally slightly. Otherwise, when the graft is placed
on the posterior slope of the tibia, the screw will angle toward the articular
surface.
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It is helpful, when identifying the FAAS of the PCL on the femur, to use an
angled curet to mark the site. To locate a spot 8 to 10 mm from the articular
margin, the tip of the curet is placed at the articular margin. Next, your thumb
can be placed on the curet 8 to 10 mm from the edge of the skin where the
curet enters the medial portal. Advance the curet until your thumb touches
the skin. This will place the curet tip 8 to 10 mm off the articular margin, and
this spot can be marked.
If an ACL reconstruction is being performed in conjunction with a PCL

reconstruction, care must be taken not to damage the tibial tunnel for the
ACL. This can occur in the inlay technique if the 6.5 screw securing the bone
plug to the tibia is directed into the ACL tibial tunnel. This can be avoided by
directing the screw slightly lateral. With the tibial tunnel technique the
starting point for the ACL and PCL tibial tunnels are close. Care should be
taken that these do not intersect. To avoid this, a drill can be placed and left
in one tunnel while the other is being drilled.
As previously mentioned, excess penetration with a guide pin while drilling a

tibial tunnel can cause injury to the posterior neurovascular structures. This
can be avoided by locking the pin into the drill 1 cm shorter than the distance
required to reach the guide tip. It can be advanced slowly the rest of the way
by hand. Also, a curet can be placed over the tip of the pin through the
posteromedial portal while reaming to avoid advancement of the pin.
Following wound closure, a cryotherapy pad and compressive stocking are applied. The
knee is immobilized in full extension. The postoperative regimen involves keeping the
leg immobilized for 4 weeks. During this period of immobilization, we use a cast or a
range of motion brace locked in extension and are careful not to allow posterior sag. If
using a cast, it is changed at week 1, week 2, and week 4. At 4 weeks postoperatively,
the patient begins gentle range of motion. Once the patient gains 90 degrees of
motion, the knee immobilizer is discarded. Following this, a posterior stabilizing knee
brace is applied. The patient wears the brace for all activities for the first 2 months.
The patient then is started on isotonic quadriceps, extension exercises, and closed
chain exercises to strengthen the hamstring muscles. We do not allow any isotonic
hamstring muscle exercises. Once the patient is walking well, we progress to a
â€œslideboardâ€• activity, which usually occurs at approximately 3 months. Agility
drills and sports-specific activities follow, with an anticipated return to play at
approximately 9 to 12 months following surgery.
COMPLICATIONS
As with any surgical procedure, there is a constant risk of infection. Other than
prophylactic antibiotics, attention to sterile technique, and appropriate soft tissue
handling, not much more can be done to lessen this risk. The process of repositioning
the patient for this surgery twice definitely increases the opportunity for
contamination; however, the use of a sterile tourniquet and strict attention to detail
by the surgeon and staff should limit patient exposure.
In addition, exposures that are unfamiliar to an operating surgeon can be filled with
danger. Preoperative cadaveric dissection and anatomy review is essential to the
posterior approach described herein. The surgeon needs to be aware of anomalous
branches of the genicular arteries as well. Complications can also be related to the
creation of tunnels and placement of hardware. The operating surgeon must be fully
aware of the position of bony tunnels and the implications on the survival of the graft
and the ability to perform other procedures.
RESULTS
Given the relative infrequency with which the PCL is reconstructed in the sports-
injured patient, it is difficult to obtain the numbers necessary to create a randomized
prospective study that definitively determines the optimal mode of reconstruction.
The inlay technique has been compared with the allarthroscopic technique and some
have felt that there is no true difference in outcomes. However, our decision to use
the inlay technique has been based on basic science research that has shown that
avoidance of the â€œkiller turnâ€• can lead to decreased laxity at follow-up
(1,2,3,4,8,9,10,11). Current outcomes following PCL reconstruction rely on
retrospective studies and control groups. Two good
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studies with at least 2-year follow-up have shown satisfactory outcomes with
significantly less posterior laxity at final followup (6,7). Certainly, the biomechanical
studies create speculation regarding the long-term viability of the implanted graft,
which, if failed, could lead to long-term instability and arthrosis.
CONCLUSION
Reconstructions of the PCL are uncommon compared to other knee procedures. This
procedure is generally reserved for patients with acute or chronic combined
PCL/capsuloligamentous injuries, as well as isolated PCL injuries that have failed
nonoperative treatment. Isolated PCL injuries are not routinely operated on
otherwise. Key components of the history, physical examination, and diagnostic
studies are used to identify candidates for PCL reconstruction. There is a great deal of
technical difficulty with this procedure and a relative lack of knowledge about the
natural history and reconstructive biomechanics of PCL injuries compared with ACL
injuries. In light of this, reconstruction of the PCL is currently an evolving process.
RECOMMENDED READING
Cosgarea AJ, Jay PR. Posterior cruciate ligament injuries: evaluation and
management. JAAOS. 2001;9:297-307.
Oliviero J, Miller MD. Posterior cruciate ligament reconstruction: tibial inlay

technique. Tech in Knee Surg. 2003;2(1): 63-72.
Wind WM, Bergfeld JA, Parker RD. Evaluation and treatment of posterior cruciate
ligament injuries: revisited. Am J Sports Med. 2004;32(7):1765-1775.
REFERENCES
1. Bergfeld JA, Graham SM. Tibial inlay procedure for PCL reconstruction: one-
tunnel and two-tunnel. Op Tech Sports Med. 2001;9(2):69-75.
2. Bergfeld JA, McAllister DR, Parker RD, et al. A biomechanical comparison of

posterior cruciate ligament reconstruction techniques. Am J Sports Med.
2001;29(2):129-136.
3 Bergfeld JA, Graham SM, Parker RD, et al. A biomechanical comparison of

posterior cruciate ligament reconstructions using single- and double-bundle tibial
inlay techniques. Am J Sports Med. 2005 Jul;33(7):976-981.
4. Bergfeld JA, McAllister DR, Parker RD, et al. The effects of tibial rotation of
posterior translation in knees in which the posterior cruciate ligament has been
cut. J Bone Joint Surg Am. 2001;83:1339-1343.
5. Burks RT, Schaffer JJ. A simplified approach to the tibial attachment of the
posterior cruciate ligament. Clin Orthop Relat Res. 1990;254:216-219.
6. Cooper DE, Stewart D. Posterior cruciate ligament reconstruction using single-

bundle patella tendon graft with tibial inlay fixation: 2- to 10-year follow-up. Am J
Sports Med. 2004 Mar;32(2):346-360.
7. Jung YB, Tae SK, Jung HJ, et al. Replacement of the torn posterior cruciate
ligament with a mid-third patellar tendon graft with use of a modified tibial inlay
method. J Bone Joint Surg Am. 2004 Sep;86-A(9):1878-1883.
8. Markolf K, Davies M, Zoric B, et al. Effects of bone block position and

orientation within the tibial tunnel for posterior cruciate ligament graft
reconstructions: a cyclic loading study of bone-patellar tendon-bone allografts. Am
J Sports Med. 2003;31(5):673-679.
9. Markolf KL, O'Neill G, Jackson SR, et al. Reconstruction of knees with combined
cruciate deficiencies: a biomechanical study. J Bone Joint Surg Am. 2003;85-
A(9):1768-1774.
10. Markolf KL, Slauterbeck JR, Armstrong KL, et al. A biomechanical study of the
replacement of the posterior cruciate ligament with a graft. J Bone Joint Surg Am.
1997;79(3):375-387.
11. Markolf KL, Zemanovic JR, McAllister DR. Cyclic loading of posterior cruciate
ligament replacements fixed with tibial tunnel and tibial inlay methods. J Bone
Joint Surg Am. 2002;84-A(4):518-524.
12. McAllister DR, Markolf KL, Oakes DA, et al. A biomechanical comparison of
tibial inlay and tibial tunnel posterior cruciate ligament reconstruction
techniques: graft pretension and knee laxity. Am J Sports Med. 2002;30(3):312-7.
13. Rauh MA, Munjal S, Carney K, et al. Fixation strength of tibial inlay graft with
one versus two screws. A cadaveric study. Orthopedics. In Press.
> Table of Contents > Part Three - Ligament Injuries and Instability > 21 - Posterolateral
Corner and Fibular Collateral Ligament Reconstruction
21
Posterolateral Corner and Fibular Collateral
E. Lyle Cain Jr.
William G. Clancy Jr.
Posterolateral rotatory instability (PLRI), unlike anterior cruciate ligament (ACL)

deficiency, is a relatively infrequently encountered instability pattern that has
historically been underdiagnosed and poorly treated. Because this instability pattern is
uncommon, it is frequently not diagnosed at the time of initial injury, often presenting
as chronic disability. Posterolateral corner injury may result in the worst functional
instability pattern of any type of knee ligament injury. These injuries produce
excessive tibial external rotation, posterior tibial translation, and varus laxity.
Excessive external rotation of the tibia allows for an unwinding moment on the
cruciate ligaments, which enhances joint laxity and adversely affects the cruciate
ligament reconstruction or the native ligament mechanics. External tibial rotation
brings the ACL tibial insertion laterally toward its lateral femoral condyle origin,
producing laxity in the ACL. Similarly, external tibial rotation brings the posterior
cruciate ligament (PCL) tibial insertion medially toward the medial femoral condyle
origin, producing laxity of the PCL.
Posterolateral rotatory instability of the knee, defined has excessive external tibial
rotation producing functional instability, can be one of the most disabling of all knee
instability patterns. The posterolateral corner consists anatomically of one dynamic
stabilizer and several static stabilizers that contribute to a functional stability of the
knee. The popliteus muscle tendon complex is the primary dynamic stabilizer, and the
popliteal fibular ligament has received added attention recently in the orthopaedic
literature as a static stabilizer. The other static structures important for stability
include the fibular collateral ligament (FCL) complex as well as the arcuate ligament,
posterolateral capsule, and PCL. The importance of PLRI is that if not diagnosed and
treated properly, it may lead to failure of other ligament reconstructions, particularly
ACL or PCL reconstruction procedures. Harner and colleagues have found that
sectioning of the posterolateral structures of the knee resulted in a 150% increase in
PCL forces in cadaveric knees. Posterolateral rotatory instability can be classified into
one of five types according to the etiology and associated instability patterns with or
without varus laxity or thrust.
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The first type of PLRI is traumatic. This often occurs secondary to a significant contact
injury, most commonly a varus blow from the medial surface of the knee causing a
lateral or posterolateral corner tear. The lateral stabilizing instructions of the knee
are frequently torn concomitantly, including the fibular collateral ligament as well as
other ligamentous structures including the ACL, PCL, or both. With traumatic PLRI,
treatment of the other ligament injuries such as cruciate ligament injury will correct
anterior and posterior translation of the knee; however, rotational instability may
persist. Failure to address PLRI may ultimately lead to failure of ACL or PCL
reconstruction. The second type PLRI is known as symptomatic physiologic instability.
This type of PLRI is most commonly caused by relatively minor injury occurring in
persons already having symmetric excessive external rotation in both knees. However,
it may occur without history of even minor trauma, similar to the development of
multidirectional instability of the shoulder. We believe that this type of PLRI may be
due to electromechanical dissociation of the popliteus muscle, similar to that
described by Rowe in the multidirectionally unstable shoulder. In this typem of PLRI,
the cruciate ligaments are normal as is the fibular collateral ligament. Lateral (varus)
laxity is similar to the ininvolved leg. This rotational instability produces clinical
symptoms and history of giving way and instability, particularly with rotational
maneuvers, because of excessive lateral tibial rotation with pivoting maneuvers. The
diagnosis of symptomatic physiologic instability is often overlooked because of intact
cruciate ligaments and a relatively minor trauma necessary to produce this clinical
instability. We have observed physiologic asymptomatic excessive external tibial
rotation in approximately 10% to 15% of normal knees. Cooper and others have
reported excessive physiologic external tibial rotation in 35% of patients. It is
important to note that this type of PLRI is typically bilateral and usually does not occur
with any significant varus laxity.
The third type of PLRI is combined traumatic cruciate injury with physiologic
posterolateral laxity. This occurs with isolated tearing of either the ACL or PCL with
concomitant low-grade injury to the posterolateral corner in an individual already
having excessive physiologic external rotation. We assume this injury to be the case
whenever there is a 3+ posterior drawer present along with excessive tibial external
rotation equal to the contralateral side with a PCL injury. Clinically, there is often no
increased varus laxity nor is there any injury visible to the popliteus tendon or fibular
collateral ligament seen on magnetic resonance imaging (MRI). With concomitant
tearing of the ACL, the ACL deficiency and posterolateral rotatory instability often
negate each other. Reconstruction of the ACL without attention to the posterolateral
corner eliminates the anterior posterior instability but effectively increases the
dysfunction of posterolateral excessive rotation of PLRI. Sometimes this occurs to such
extent that the individual complains of symptoms with instability worse than was felt
before ACL surgery. It is very difficult to determine whether the physiologic
posterolateral laxity is pathologic and whether it should be addressed during ACL
reconstruction. After the ACL graft has been fixed during reconstruction, the presence
of a mild increase in Lachman or pivot glide strongly suggests that the PLRI is
pathologic and should be corrected surgically.
In cases of isolated PCL injury with physiologic external rotation deformity, isolated
reconstruction of the PCL without reconstruction of the posterolateral corner results in
persistence of external rotation excess and posterior tibial sag. Increased external
rotation causes the tibial insertion of the PCL to rotate medially and anteriorly,
shortening the distance of insertion of the PCL on the medial femoral condyle, and
producing functional laxity of the PCL although the PCL reconstruction may be intact.
Therefore, symptoms may persist after PCL reconstruction if PLRI is not addressed.
The fourth type of PLRI includes posterolateral instability with varus alignment and
varus thrust. This clinical situation occurs in the patient who has an injury to the
posterolateral structures with or without fibular collateral ligament injury in a varus
knee. When this patient is weightbearing, they have a significant varus thrust which
leads to worsening of the PLRI and varus instability. With this situation, medial
compartment degenerative changes and joint space narrowing are often present as
well as the native varus alignment. We often perform valgus osteotomy in these
patients to correct both the varus position as well as a varus thrust prior to, or in
combination with, posterolateral reconstruction.
The fifth type of PLRI is posterolateral instability with medial and lateral laxity. This is
a rare entity but is seen when the femur has a medial then lateral shift on the tibia
during the stance phase of gait. This could be significant for posterolateral laxity with
varus thrust; however, it is a straight medial-lateral instability pattern not associated
posterolateral corner injury. Most likely, this represents musculoelectrical
disassociation similar to pathology in some patients with multidirectional instability of
the shoulder and will not benefit from present surgical techniques to reconstruct the
posterolateral corner.
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Our current indications for posterolateral cruciate reconstruction or fibular collateral
ligament reconstruction include a symptomatic knee with any of the PLRI patterns
classified previously. The most common patient in our practice presents with either
ACL or PCL injury with combined traumatic posterolateral corner injury as well. Any
findings on MRI or examination of ecchymosis or swelling along the posterolateral
structures of the knee including popliteus, fibular collateral ligament, or
posterolateral capsule should be assumed to have posterolateral rotatory instability
until proven otherwise by appropriate examination. In the acute setting, we generally
repair all the injured structures including the fibular collateral ligament, biceps
femorus muscle tendon complex, iliotibial band, popliteus tendon complex, as well as
the posterolateral capsule. In many acute cases, the popliteus muscle tendon complex
has been damaged to the extent that is not reasonable to repair this structure. Injuries
to the popliteus often occur in the muscle tendon junction and are not amenable to
acute repair. In these acute settings with posterolateral corner instability and poor
tissue quality or muscle tendon junction popliteus tear, we generally perform an acute
posterolateral cruciate reconstruction using allograft Achilles tendon. In the chronic
situation, we perform posterolateral cruciate reconstruction with or without fibular
collateral ligament reconstruction using Achilles tendon allograft.
Several authors have described techniques for either reattaching, repairing, or

reconstructing the posterolateral and fibular collateral structures. Hughston's
posterolateral reconstruction, described as anterior and superior advancement of the
lateral gastrocnemius tendon, superior posterolateral capsule, fibular collateral
ligament, and popliteus tendon, is possible only with intact native structures and has
not been met with much success in our experience. Jakob and associates have
recommended recession of the popliteus tendon insertion on the femur, but this often
fails to improve static or functional stability because of damage to the structures
distal to that insertion site. Until 1996, biceps femoris tendon tenodesis and rerouting
at the lateral femoral condyle was the senior author's (WCG) preference for correction
of mild to moderate PLRI without the presence of varus thrust. Although this
procedure led to predictable and satisfactory results in the mild and moderate cases,
it required an intact biceps muscle tendon complex and often required hardware
removal at a second surgical procedure. We have performed the posterolateral
cruciate reconstruction in severe cases since 1979, and have employed it exclusively in
mild, moderate, or severe PLRI since 1996.
Our current preference for acute augmentation or chronic injuries to the

posterolateral corner is the posterolateral cruciate reconstruction (PLCR) with one-
half Achilles tendon allograft as well as fibular collateral ligament reconstruction with
the opposite half of the Achilles tendon allograft. Posterolateral cruciate
reconstruction differs from the Hughston or Jakob reconstructions in that PLCR does
not rely on healthy native tissues. Immediate knee motion is possible, and hardware
removal is usually unnecessary. However, posterolateral cruciate reconstruction alone
does not correct for straight lateral (varus) laxity. Therefore, if varus laxity is
additionally present with PLRI, the fibular collateral ligament is reconstructed as
well.Valgus tibial osteotomy may be considered in conjunction with PLCR or other
cruciate ligament procedures in some cases. These patients typically have varus laxity
with medial joint space narrowing and a varus thrust in the stance phase of gait.
Failure to correct the varus alignment may cause stretching and eventual failure of the
posterolateral structures, as suggested by Noyes. At the time of surgery, the
osteotomy should be performed first followed by either concomitant or delayed
posterolateral cruciate reconstruction and fibular collateral ligament reconstruction.
A proper preoperative evaluation is essential to correctly evaluate the instability
pattern for posterolateral rotatory instability.
History
The typical history of patients with acute PLRI is significant for a discrete injury with a
blow to the medial side of the knee resulting in varus force, often combined with
hyperextension (Fig. 21-1). The mechanism of injury should alert the examiner to look
for disruption of the posterolateral structures, the lateral structures, especially the
fibular collateral ligament, and associated ACL or PCL injury.
In the chronically disabled individual, the history of significant remote trauma is

frequently elucidated but the mechanism is often unclear. More importantly, the
individual expresses a very frequent sense of instability with minimal activities,
causing giving way that may be associated with
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ACL instability. In addition, each episode of giving way is usually accompanied by pain
along the posterolateral corner of the knee.
FIGURE 21-1
Typical mechanism for acute posterolateral corner and ACL injury by a blow to
the anteromedial tibia causing varus and hyperextension.
In the acutely injured knee, findings often include marked rotational and varus
instability at 30 degrees of flexion (with fibular collateral and posterolateral injury). If
significant varus instability is present at full extension (0 degrees) or 90 degrees
flexion, suspicion for disruption of one of the cruciate ligaments must be evaluated.
The amount of excessive external rotation can be measured with the pateint supine
with hips flexed 90 degrees and the knee flexed 30 degrees. The lower leg is externally
rotated and heel and foot position is noted for both legs to determine the amount of
relative side to side exernal rotation. The knee is then flexed to 90 degrees and similar
external rotation is performed. Some believe that the amount of external rotation is
best assessed in the prone position with foot external rotation at 30 and 90 degrees
knee flexion. We believe that a more accurate method to measure excessive external
rotation is done by placing the patient supine with the hips flexed 90 degrees and
knees 30 degrees. The lateral femoral epicondyle and anterior and posterior borders of
the fibular head are marked and both legs are externally rotated. The test is repeated
at 90 degrees knee flexion. Excessive external rotation is present if the anterior
border of the fibular head passes posterior to the mark on the lateral femoral
epicondyle. Warren and Noyes have clearly demonstrated that excessive external
rotation noted to increase between 30 and 90 degrees knee flexion indicates probable
PCL injury.
Ecchymosis and soft tissue swelling may be present along the lateral and posterolateral
side of the knee, and the fibular collateral ligament frequently is not palpable as a
tight cord-like structure as is found in the normal knee. If the cruciate ligaments have
also been damaged, a positive result on Lachman, anterior drawer, posterior drawer,
pivot shift, or reverse pivot shift test may be present. As a rule, complete isolated
tear of the PCL will not have more than a 2+ posterior drawer (i.e., the tibial plateau
will be flush with the femoral condyles with the knee flexed at 90 degrees of flexion).
Any further posterior sag of the tibia on the femur (3 + posterior drawer) is almost
always indicative of concomitant posterolateral rotatory instability with posterolateral
corner injury. In the awake patient, when the acute knee is examined, further
evaluation is always difficult. One should, however, try to evaluate the amount of
external rotation of the tibia at 30 and 90 degrees of knee flexion as well as attempt
to perform a reverse pivot shift maneuver to test the posterolateral corner and PCL.
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In the individual with chronic instability, determination of external rotation compared
with the opposite knee at 30 and 90 degrees is essential. This maneuver should
reproduce a sense of giving way as well as pain along the lateral side of the knee with
PLRI. Varus alignment of the knee may or may not be present, and ambulation may
bring out a significant varus thrust. Finally, while sitting with the knee flexed at 90
degrees and the affected foot on the floor, PLRI patients can often reproduce the
sensation of dynamic instability by voluntarily firing the biceps femoris muscle,
thereby producing marked external rotation and subluxation of lateral tibial plateau
on the lateral femoral condyle. This may be considered a pathognomonic sign of
posterolateral rotatory instability but must be distinguished from the performance of
an anterior drawer in the case of isolated ACL or posterior drawer in the case of PCL
deficiency.
Additional Testing
Routine radiographs including both supine and standing anteroposterior views of both
knees should be performed to assess lower extremity alignment. In the acute setting, a
fracture of the posterolateral tibial plateau should raise the suspicion of PLRI. In the
knee with chronic PLRI, significant degeneration of the medial compartment and varus
alignment may dictate the need for concomitant osteotomy. Magnetic resonance
imaging is routinely ordered to confirm the status of the cruciates, the menisci, and
the articular cartilage structures as well as assess any fibular collateral or
posterolateral capsular injury.
SURGERY
Preoperatively, the patient is instructed on proper crutch ambulation. Information is
also provided about the expected hospital course, which is typically a 24-hour
observation stay after surgery. The patient is instructed on immediate postoperative
rehabiltation activities including quadricep exercises and early range of motion.
General anesthesia is preferred in this procedure due to the potential length and
complexity of multiligament reconstruction. Once adequate anesthesia has been
established, a thorough examination under anesthesia is performed on the affected
knee with comparison to the unaffected knee. Examination includes inspection, range
of motion, and stability testing to confirm the ligament instability as suspected
preoperatively.
Technique
With the patient in the supine position, a tourniquet is placed on the upper thigh, and
the lower extremity is prepped and draped free. The tourniquet is not generally
inflated for the procedure, but is applied for the occasional case requiring assistance
for hemostasis. Operative knee arthroscopy is performed initially with any intra-
articular pathology addressed including chondral lesions, meniscal tears, or cruciate
ligament injury. The tunnels are generally drilled for the ACL or PCL reconstruction
before the posterolateral corner reconstruction is performed. In the acute setting, the
lateral structures may be exposed initially to allow full egress of arthroscopic pump
fluid from the lateral side of the knee and prevent overdistension of the soft tissues
along the lateral knee and proximal anterior tibial muscular compartments. Once the
intra-articular work is completed, the posterolateral cruciate and femoral collateral
ligament reconstructions are performed.
A lateral curvilinear incision is made with the incision passing midway between Gerdy's
tubercle and the fibular head on the lateral surface of the knee (Fig. 21-2). The
subcutaneous tissues are pulled posteriorly using a dry lap sponge to expose the
iliotibial (IT) band and the biceps femoris tendon. The peroneal nerve should be
identified just posterior to the biceps tendon (Fig. 21-3). The surgeon should be aware
of its position at all times during the reconstruction. The IT band is then incised in line
with its fibers beginning at the point where it crosses the lateral femoral epicondyle
and proceeding distally. This exposes the insertion point on the lateral femoral
condyle of both the fibular collateral ligament as well as the popliteus tendon. The
peroneal nerve is explored and freed as it dives beneath the fascia in the anterolateral
compartment of the lower leg. In approximately 20% of cases, there is a very thick
fascial band just deep to the anterolateral muscle compartment that may lead to
common peroneal nerve palsy caused by postoperative swelling. The nerve is freed by
incising this fascia for a distance of 2 to 3 cm into the anterolateral compartment to
ensure the fascia will not entrap the nerve after completion of the procedure. A blunt
retractor is then used to retract the biceps inferiorly and the lateral head of the
gastrocnemius posteriorly to expose the popliteus muscle belly and posterolateral
capsule (Fig. 21-4).
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FIGURE 21-2
Photograph of the typical skin and bony landmarks with the intended incision
from the lateral femoral epicondyle to a point between the fibular head and
Gerdy's tubercle.
FIGURE 21-3
The initial layer of dissection for the PLCR including the peroneal nerve below the
biceps femoris tendon, the iliotibial band, and lateral gastrocnemius tendon.
FIGURE 21-4
A blunt retractor retracts the biceps inferiorly and the lateral head of the
gastrocnemius posteriorly to expose the popliteus muscle belly and posterolateral
capsule.
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FIGURE 21-5
Tibial tunnel placement. A guidewire is placed through the tibia from just below
Gerdy's tubercle to the musculotendinous junction of the popliteus muscle. A drill
guide may be used, but we typically use direct visualization with a freehand
technique for wire and tunnel preparation.
A portion of the anterior compartment musculature is dissected using sharp dissection

off Gerdy's tubercle in the anterior proximal tibia. A guidewire is placed freehand from
just below Gerdy's tubercle toward the posterolateral tibia with an attempt to exit the
wire at the muscle tendon junction of the popliteus muscle belly (Figs. 21-5, 21-6 and
21-7). This exit point should be 1 to 2 cm distal and 1 to 2 cm medial to the tibial
articular surface and the proximal tibiofibular joint. Fluoroscopy may by used as a
guide for placement of the guidewire; however, we have not generally felt that this is
necessary. A tunnel is then drilled with a 10-mm cannulated reamer over the
previously placed guidewire under careful visualization to prevent the guide pin from
proceeding further posteriorly. A No. 5 doubled suture is passed from anterior to
posterior through the tunnel using a Hewson suture passing device for later graft
passage.
The femoral insertion of the popliteus tendon on the lateral aspect of the lateral
femoral condyle is palpated. In most patients, the popliteus fovea is a palpable small
groove running obliquely along the articular surface just posterior to the midline of
the femoral condyle articular surface and is easily felt percutaneously. An incision
previously made in the IT band at the lateral femoral epicondyle allows exposure of
the popliteus tendon insertion in the area just distal to the peak of the lateral femoral
epicondyle. A K-wire is placed at the anterior/superior insertion of the popliteus
tendon for proximal placement of the bone block portion of the allograft. Work done
in our laboratory previously has shown that a physiometric insertion site is located at
the most anterior superior aspect of the popliteus tendon insertion into the fovea of
the lateral femoral condyle just distal to the lateral epicondyle. For confirmation of
the physiometric point, a suture can be fixed at this point and then passed under the
fibular collateral ligament deep to the biceps tendon and IT band through the tibial
tunnel, and the knee can be ranged to check for isometry. After many years of
measuring this physiometric
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point, we no longer perform this step. A 10-mm wide X 20-mm depth tunnel is created
using a 10-mm cannulated reamer (Fig. 21-8). Care is taken to ream perpendicular to
the lateral aspect of the femur to avoid entering the articular surface or damaging
previously placed tunnels for ACL reconstruction. An Achilles tendon allograft is
prepared on the back table in the operating room with the graft split in half, one half
to be used for the posterolateral cruciate and the other half be used for fibular
collateral reconstruction, if necessary (Fig. 21-9). For the posterolateral cruciate, the
bone is fashioned to pass easily through a 10-mm cannula with a 20-mm bone block. A
No. 5 running nonabsorbable suture is then placed through the tendon free edge. The
graft is tubularized to aid in the graft passage and fixation. If a double staple
technique is to be used for tibial fixation, sutures are placed in the free end of the
tendon and the remaining tendon is resected at the conclusion of the procedure. If the
graft is to be tied over a post, measurements must be made during the surgical
procedure to determine appropriate length of the Achilles tendon allograft that is
needed for graft fixation. The graft is cut at that appropriate length and is tubularized
and fixed with a No. 5 nonabsorbable suture for later graft fixation to a post and
washer. In general, a graft length of approximately 120 mm is appropriate for post
fixation, whereas 150 mm of graft is required to use Dale Daniel's double staple
technique for tibial fixation.
FIGURE 21-6
A: A 10-mm reamer is passed over the guidewire to exit at the popliteus muscle
tendon junction. B: Close-up view of the reamer head exiting at the popliteus
muscle tendon junction.
FIGURE 21-7
Diagrammatic representation of the proximal tibia, showing proper orientation of

the tibial tunnel.
FIGURE 21-8
Femoral tunnel placement. A 10-mm wide Ã— 20-mm deep tunnel is created using
a 10-mm cannulated reamer at the anterior superior edge of the popliteus fovea.
FIGURE 21-9
Achilles allograft preparation with graft divided into two separate grafts with 9-
Ã— 20-mm bone plugs.
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FIGURE 21-10
The graft is fixed in the femoral tunnel with a 9- Ã— 20mm interference screw,
and the tendon is passed deep to the iliotibial band and fibular collateral
ligament as well as the biceps tendon following the anatomic route of the
popliteus tendon along the posterolateral tibia.
FIGURE 21-11
Graft passed through tibial tunnel from posterior to anterior and held tight while
the knee is cycled through range of motion.
The bone block is placed in the femoral tunnel and fixed with a 9 X 20 mm
interference screw. The interference screw is placed on the side of the bone block
opposite the articular surface so that the tendon itself is more anterior and distal. The
tendon is then passed deep to the iliotibial band and fibular collateral ligament as well
as the biceps tendon following the anatomic route of the popliteus tendon along the
posterolateral tibia (Fig. 21-10). The graft is passed into a plastic graft passing tube,
which may aid in graft passage through the popliteus muscle belly and through the
tibial tunnel. A No. 5 passing suture is used to deliver the graft from posterior to
anterior through the tibial tunnel. The graft is then held taunt as the knee is taken
through range of motion to assess isometric positioning and to cycle the graft (Fig. 21-
11).
Fixation then depends on other ligaments to be reconstructed. If ACL or PCL

reconstruction is performed at the same time, these grafts are fixed on the femoral
side before fixation of the posterolateral corner graft. After femoral graft fixation for
the cruciate reconstruction, the knee is held in approximately 30 degrees of knee
flexion with internal tibial rotation and valgus stress applied. The fixation may be
performed using Daniel's two staple belt buckle technique or by tying the sutures from
the tendon end of the graft over a post and washer on the proximal lateral tibia (Figs.
21-12 and 21-13). Our current preference for tibial fixation includes intraoperative
measurements using free suture as a ruler to ensure that the graft length ends within
the tibial tunnel. The graft is prepared with a No. 5 nonabsorbable suture whip stitch
and tied over a 30-mm cancellous screw and washer. The post and washer is placed
along the anterolateral tibial crest under the anterior compartment muscles, which
helps prevent symptoms from prominent hardware. Using this fixation technique, we
have not needed to remove the tibial hardware.
The PLCR will restore rotational laxity but will not control excessive varus laxity. If
varus laxity persists due to insufficiency of the FCL, the FCL may be reconstructed
using the other half of the Achilles tendon allograft. Again, the remaining half of the
allograft is prepared and easily passed through a 10-mm cannula with a bone block
measuring 20 mm in length. A K-wire is then placed directly perpendicular into the
lateral epicondyle through the epicondylar prominence. The origin of the fibular
collateral ligament is just below the lateral femoral epicondyle and slightly posterior
and proximal to the popliteus attachment. A 10-mm reamer is then used to create a
tunnel 20 mm in depth. The bone block is affixed with a 9- X 20-mm interference
screw so that the fibers of the graft are at the posterior edge of the tunnel, which will
coincide with a normal insertion. The peroneal nerve has previously been identified
and protected. The nerve should enter the anterior tibial musculature approximately 3
to 4 cm down the fibular neck. The nerve is visualized and protected throughout
preparation of the fibular tunnel. A 7-mm tunnel is then drilled transversely from
anterior and posterior through the widest portion of the fibular head. The tunnel is
enlarged with progressively larger curets until it is large enough, typically 8 to 9 mm,
to pass the soft tissue portion of
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the Achilles tendon allograft. A suture passer is then used to pass the allograft from
posterior to anterior, and the free end of the graft is then brought proximally onto
itself. The graft is then tightened in slight valgus stress with 90 degrees of knee
flexion. The graft is fixed to a suture anchor and then sewn to itself, or it can be fixed
by using a double staple technique on the femoral surface. The anterior and posterior
limbs of the graft are then sutured to themselves using nonabsorbable suture to
produce a fibular collateral ligament reconstruction that proceeds directly from the
lateral femoral epicondyle to the fibular head attachment (Fig. 21-14). After
completion of PLCR and FCL reconstructions, tibial insertions of the cruciate ligaments
are fixed, and the knee is cycled through range of motion and assessed for stability.
FIGURE 21-12
Schematic representation of completed posterolateral cruciate reconstruction

with double staple tibial fixation.
FIGURE 21-13
Anteroposterior (A) and lateral (B) radiographs following completion of the

posterolateral cruciate reconstruction.
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The wound is closed beginning with approximation of the IT band and biceps femoris
using absorbable suture. The peroneal nerve is inspected to ensure that it is free of
any restrictions or pressure throughout its course. Drains are placed deep within the
lateral wound and brought out through separate stab incisions. The subcutaneous
tissues are closed with 2-0 absorbable suture, and the wound is generally closed with
skin staples. The patient is placed in a hinged knee brace locked in full extension and
begins physiotherapy on postoperative day 1. If a PCL reconstruction is also
performed, we prefer the knee to be placed in a spring-loaded dynasplint to
counteract the posterior tibial forces that may tension the PCL graft.
Rationale for Using the PLCR Technique

This surgical technique creates a popliteal ligament to replace the popliteus muscle
tendon complex with a much greater lever arm than the popliteofibular ligament. In
addition, we believe that fibularbased techniques using a graft to create only a FCL
will not adequately control PLRI over the long haul, resulting in late rotatory
instability. Remember, many patients with symptomatic PLRI have no varus laxity or
instability.
Pearls and Pitfalls

Proper tunnel placement is crucial as with other ligament reconstructions (ACL or
PCL). The anterior-superior portion of the popliteus tendon attachment is the best
physiometric position for the PLCR femoral tunnel, whereas the most prominent
portion of the lateral femoral epicondyle is the best placement of the tunnel for the
FCL femoral graft so the graft will lie at the inferior portion of the tunnel closest to
the insertion point of the native FCL. Graft passage through the popliteus muscle
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belly is typically the toughest portion of the procedure, and may be helped by use of a
plastic graft passage device (DePuy Mitek, Raynham, MA) to allow the Achilles tendon
to slide through the muscle belly. Peroneal nerve exploration and particularly careful
release of the fascial band of tissue at the entrance of the anterior compartment
musculature will prevent the likelihood of postoperative peroneal neuropraxia.
FIGURE 21-14
Schematic of fibular collateral ligament reconstruction with Achilles allograft.

Note the suture connecting the anterior and posterior limbs.
Initially, the patient is allowed to bear full weight in a brace locked in full extension
with crutch ambulation. The patient performs range-of-motion exercises from 0 to 90
degrees of flexion three times a day for the first 2 weeks postoperatively, and usually
goes to supervised physical therapy 2 to 3 days a week during this period. At 4 weeks,
we allow full range of motion as tolerated and expect full range of motion about 6
weeks postoperatively. Active hamstring contractions are discouraged for the first 6
weeks to prevent excessive tibial translation or rotation and to prevent tension on the
posterior cruciate or posterolateral corner grafts. The hinged knee brace is allowed to
be unlocked during weightbearing at the 6-week mark based on ability of the patient
to control his or her body weight with quadriceps contraction. The patient generally
wears the hinged knee brace for 8 to 10 weeks postoperatively to protect the lateral
and posterolateral structures. Quadriceps exercises are begun as soon as possible
postoperatively. Running and plyometrics are generally started 16 weeks after surgery,
with return to play anticipated approximately 6 to 8 months postoperatively. We do
not routinely use functional postoperative braces for posterolateral corner
reconstructions, although with certain contact sports, we use braces to protect the
collateral ligaments.
COMPLICATIONS
Common Peroneal Nerve Palsy
Common peroneal nerve palsy is the most worrisome potential complication of
posterolateral corner acute repair or reconstruction because of the proximity of the
nerve during surgical exposure. In many cases of posterolateral and fibular collateral
ligament injury, the peroneal nerve has been injured or has some damage at the time
of initial traumatic event. During the surgical procedure, we generally explore the
nerve proximally and distally to make certain that it is released from any restrictions
within the anterior compartment of the tibia or along the posterior biceps tendon
proximal to the area of injury. Since we began releasing the fibro-osseous tunnel at
the anterior compartment of the tibia, we have not had any actual cases of peroneal
nerve injury as an intraoperative complication.
If peroneal nerve palsy occurs postoperatively, this most likely is secondary to

postoperative hematoma or surgical traction injury. In the case of large
hematomeformation, re-exploration and evacuation of hematoma should be
reconsidered if no improvement is seen by simple release of the bandages and
dressings.
From 1977 to 1979, we were unaware of the potential presence of a fibrotic band
distal to the entrance of the peroneal nerve into the anterolateral musculature. We
have not had any cases of neuropraxia since we began exploring the anterior
compartment entrance and releasing this band when present. In the cases of peroneal
nerve palsy during the first 2 years of our case series, peroneal nerve weakness did not
usually begin until postoperative day 3 and was completely resolved by 6 to 9 months.
Sensation usually returns first, followed by anterior compartment muscle control. In
our experience, the long toe extensor tendon function takes 2 to 3 months to recover,
and the extensor halluces longus recovers at 6 to 9 months as the last muscle to
improve. As a general rule, if no return of function has occurred by 6 weeks post
injury, electrodiagnostic studies are done to determine the injury to the nerve as well
as its regeneration potential. In addition, an ankle-foot orthosis (AFO) may be required
for the involved foot for ambulation until there is adequate return of active
dorsiflexion of the ankle.
Reconstruction Failure
In our previous technique for posterolateral corner reconstruction using biceps
tenodesis procedure, biceps tendon rupture and failure were possible due to excess
tension and tendon fixation using a spiked washer on the lateral femoral condyle (Fig.
21-15). With the current technique of posterolateral cruciate reconstruction, failure,
if it occurs, is generally due to allograft reinjection or failure to
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incorporate. In the case of allograft rejection, the operative leg may remain swollen
and painful and erythematous with no other clinical or laboratory evidence of
infection. In the potential case of allograft rejection, the graft may have to be
removed surgically followed by delayed alternative surgical procedures to restore
mechanical stability. This has not occurred in our series.
FIGURE 21-15
Intraoperative photograph of biceps tenodesis fixed to the lateral epicondyle with

a spiked soft tissue washer.
Fixation Failure
With regard to graft fixation using a post and washer technique, appropriate length
must be determined and the allograft must be prepared appropriately to allow fixation
of sutures over a post and washer construct. If the double staple technique is used,
adequate graft length must be present to allow a distal staple to be applied prior to
the graft being folded onto itself and the proximal stable applied. We have had a case
of graft failure due most likely to improper, insufficient tibial fixation of the graft
which allowed slippage of the graft and loss of tension along the posterolateral corner.
In cases where the graft is not long enough, we have placed several No. 5 Krackow-
type nonabsorbable sutures tied to post and screw fixation with an additional back-up
staple fixation or interference screw fixation. We have not used interference screw
fixation as our primary mode of fixation on the tibial surface for the posterolateral
cruciate reconstruction or fibular collateral ligament reconstruction.
Stiffness
Loss of motion is a rare complication with our current rehabilitation protocol, which
allows 0 to 90 degrees of flexion immediately after PLCR. During the biceps tendon
tenodesis procedure, stiffness was an issue because most patients were kept with
limited range of motion for up to 6 weeks post surgery. Range of motion is encouraged
with the PLCR rehabilitation protocol compared to the immobilization required for
many of the other methods of posterolateral corner reconstruction. Although the
multiple ligament injured knee can be a rehabilitation challenge, the occurrence of
arthrofibrosis and need for manipulation or further surgery is rare with current
techniques.
Infection
As with most open knee procedures, the risk of infection should be less than 1%.
Prophylactic antibiotics are used as well as postoperative drainage to avoid hematoma
formation. Copious irrigation and hemostasis are employed prior to wound closure.
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CONCLUSIONS
Posterolateral corner instability is an often unrecognized, but extremely disabling
injury that is usually misdiagnosed in the acute setting. Early repair of injured
structures, including the FCL, biceps femoris, posterolateral capsule, popliteus,
arcuate complex, and popliteofibular ligament generally results in return of knee
stability. However, in cases of severe acute injury, or chronic posterolateral and/or
varus instability, posterolateral cruciate ligament reconstruction and fibular collateral
reconstruction with Achilles tendon allograft have proven to allow return of normal
knee function and stability. Appropriate recognition and diagnosis are the keys to
successful treatment.
RECOMMENDED READING
Clancy WG. Repair and reconstruction of the posterior cruciate ligament. In: Chapman
MW, ed. Operative Orthopaedics. 2nd ed, vol 3. Philadelphia: JB Lippincott;1993:2093.
Clancy WG, Shelbourne KD, Zoellner GB, et al. Treatment of knee joint instability
secondary to rupture of the posterior cruciate ligament: report of a new procedure. J
Bone Joint Surg. 1983;65A:310-322.
Cooper DE. Tests for posterolateral instability of the knee in normal subjects. J Bone
Joint Surg. 1991;73A:30-36.
Daniel DM, Stone ML, Barnett P, et al. Use of the quadriceps active test to diagnose
posterior cruciate ligament disruption and measure posterior laxity of the knee. J
Girgis FG, Marshall JL, Al Monajem ARS. The cruciate ligaments of the knee joint:
anatomical, functional, and experimental analysis. Clin Orthop. 1975;106:216-231.
Gollehon DL, Torzilli PA, Warren RF. The role of the posterolateral and cruciate
ligaments in the stability of the human knee: a biomechanical study. J Bone Joint
Surg. 1987;69A:233-242.
Grood ES, Stowers SF, Noyes FR. Limits of movement in the human knee: the effects of
sectioning the PCL and posterolateral structures. J Bone Joint Surg. 1988;70A:88-97.
Harner CD, Janaushek MA, Kanamori A, et al. Biomechanical analysis of a double-

bundle posterior cruciate ligament reconstruction. Am J Sports Med. 2000;28:144-151.
Hughston JC, Andrews JR, Cross MJ. Classification of knee ligament instabilities, II: the
lateral compartment. J Bone Joint Surg. 1976;58A:173.
Hughston JC, Jacobsen KE. Chronic posterolateral rotatory instability of the knee. J
Bone Joint Surg. 1985;67A:351.
Jakob RP, Hassler H, Staeubli HU. Observations on rotatory instability of the lateral
compartment of the knee. Acta Orthop Scand. 1981;191(suppl):1-32.
Noyes FR, Stowers SF, Grood ES. Posterior subluxations of the medial and lateral
tibiofemoral compartments: an in vitro ligament sectioning study in cadaveric knees.
Am J Sports Med. 1993; 21:407-414.
Veltri DM, Deng XH, Torzilli PA, et al. The role of the cruciate and posterolateral
ligaments in stability of the knee: a biomechanical study. Am J Sports Med.
1995;23:436-443.
> Table of Contents > Part Three - Ligament Injuries and Instability > 22 - Medial Collateral
22
Medial Collateral Ligament Reconstruction
Of the four major ligaments in the knee, the medial collateral ligament (MCL) has the
greatest chance to heal after injury. This is in large part due to its anatomic and
biologic properties. Because the MCL heals, there are few cases in which surgical
intervention is required. In fact, it has been shown that surgery may even be
detrimental in certain situations, since the risk of postoperative stiffness is significant.
Clinical classification of MCL injuries can be organized by timing, severity, and

associated injuries. All these factors must be taken into account when formulating a
treatment plan. Timing, although somewhat artificial, can be divided into acute (< 3
weeks), subacute, and chronic (>4 to 6 weeks). This is based on the natural progression
of the healing process, with the majority of MCL injuries healing to some extent within
3 to 4 weeks. Severity of the MCL injury is assessed by valgus stress testing at 0 and 30
degrees of knee flexion on physical examination. It is classified as per the guidelines
established by the American Medical Association and discussed further later.
Finally, it is important to determine what other injuries are associated with an MCL
tear. There are essentially three main scenarios: (a) no associated injuries (i.e.,
isolated); (b) associated with either anterior cruciate ligament (ACL) or posterior
cruciate ligament (PCL) disruption alone; and (c) multiliagementous injury. The
anatomic location of the injury (i.e., femoral insertion, tibial insertion, or
midsubstance tear) may be helpful in predicting the potential for healing. Femoral
avulsions seem to heal better than tibial avulsions, although the risk of loss of motion
is greater. The overall alignment of the limb also is an important factor, as an
underlying genu varum alignment will protect the ligament more than genu valgum. Of
course, patient activity level and expectations may also play a role in treatment.
Acute Cases
Isolated MCL Injuries
Isolated grade I and II MCL injuries are treated nonoperatively (Fig. 22-1). A guided
program of early, protected (i.e., braced) motion, weight-bearing, and controlled
exercises is instituted. The ACL acts as a secondary restraint to resist valgus load, and
it is important to confirm its integrity before embarking on a course of nonoperative
treatment.
Grade III, or â€œcomplete,â€• tears may also be treated nonoperatively, but only
after careful exclusion of any associated injuries that may necessitate surgical
intervention. These would include cruciate ligament injury, meniscal tear requiring
repair, and entrapment of the MCL (Figs. 22-2 and 22-3).
With ACL Disruption

The indications for ACL reconstruction remain the same despite the presence of an
MCL injury. In cases of grade I or II MCL tears, if the ACL is to be reconstructed, it is
delayed 4 to 6 weeks, giving the MCL a chance to heal and allowing the knee to
recover from the initial
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trauma. Grade III MCL tears with ACL disruption pose more of a controversial scenario.
There have been studies supporting acute surgery on both the MCL and the ACL as well
as those supporting delayed reconstruction of the ACL along with nonoperative
management of the MCL injury.
FIGURE 22-1
An algorithm for evaluating and treating injuries to the medial side of the knee.
It has been our experience that most grade III MCL injuries heal if protected. The
major risk with early surgery is the development of arthrofibrosis. However, in a
certain subset of patients with this
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combination of injury (grade III MCL/ACL), the MCL does not heal and there is residual
valgus and rotatory laxity that is very difficult to correct later. We try to detect this
group initially by physical examination and magnetic resonance imaging (MRI). If the
patient has gross opening in full extension, he or she is considered a candidate for MCL
repair. The MRI can be helpful in this setting, as it will reveal if the injury is an
avulsion off of the tibia or the femur. We believe that the tibial avulsion is more
problematic, as the synovial fluid has access to the ligament/bone interface, which
may prevent healing. In this scenario, we do a surgical repair of the MCL through a
limited approach to the tibial insertion combined with an ACL reconstruction. We
prefer allograft tissue for the ACL to minimize iatrogenic trauma. Furthermore, if
significant laxity is noted with an anterior drawer (>10 mm), we elect to repair the
posterior oblique ligament (POL) at the site of injury to decrease anteromedial
rotatory instability that may complicate the ACL reconstruction in this scenario. We
also repair meniscal pathology at the same time. When we do elect to repair an
MCL/ACL injury, we delay the surgery at least 10 to 14 days and brace the knee in the
interim. This allows for the acute effects of the trauma to subside.
FIGURE 22-2
The normal anatomy of the MCL complex.
FIGURE 22-3
Entrapment of the superficial portion of the MCL.

With PCL Disruption
Although a rare injury, this pattern can result in a very unstable knee. Because both
the PCL and the MCL attach to the medial femoral condyle, complete disruption of
both results in gross laxity of the medial side of the knee. In the setting of grade III
injury to both of these structures, we recommend surgical treatment.
With ACL and PCL Disruption

In the setting of an ACL/PCL/MCL injury, we almost always repair or reconstruct all
three injured ligaments. Delaying the intervention more than 3 weeks in this case
often results in significant residual MCL laxity that is very difficult to correct. The only
exception is if the MCL injury is only partial (i.e., grades I or II), in which case we
postpone the surgery until final healing of the MCL occurs.
Chronic Cases
Chronic instability results following a grade III MCL injury that does not heal. There is
usually more than single-plane (i.e., valgus) instability present with rotational and
translational (i.e., cruciate ligaments) components. The overall alignment of the limb
plays a critical part in the treatment. Knees with underlying genu valgum are more
likely to have residual instability. In this setting both soft-tissue reconstruction and
bony alignment may have to be addressed. The usual indication for reconstruction in
chronic cases is persistent instability that affects activities of daily living.
History
The MCL can be injured with valgus contact to the knee or through a twisting,
noncontact mechanism. In the acute setting, depending on the severity and associated
injuries, the patient will present with varying degrees of swelling, pain, and loss of
motion. In combined ligament injuries, the patient is very uncomfortable and has
significant loss of motion. The patient often presents with a large effusion and more
pain than would be expected with an isolated ACL tear. If a combined ligament injury
is suspected, the neurovasculature must be assessed, as a knee dislocation may have
occurred. In the chronic setting, patients will present with instability, which usually
affects activities of daily living and results in an inability to participate in athletic
activities.
Examination
The goal of the examiner is to determine the degree of injury to the MCL as well as
the presence of any associated injuries. In this regard, the examination of the MCL
must be done in the setting of a comprehensive knee examination that includes
assessment of the neurovascular status of the extremity. In the acute setting, the
patient will have obvious discomfort, making the examination limited. The key to an
informative examination is getting the patient to relax as much as possible. A pillow
under the affected knee and gentle examination go a long way toward not only
allowing the patient to guard less but also gaining his or her confidence.
The knee may or may not be grossly swollen, and the absence of a large effusion
should not lead the examiner to a false sense of security. Tenderness to palpation over
the MCL should be elicited.
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Midsubstance tears may be associated with joint-line tenderness, creating potential
difficulty in distinguishing meniscal from collateral injury. Concomitant injury to both
structures, however, is possible.
Valgus stress testing of the affected knee in comparison to the contralateral knee is
the cornerstone of the diagnosis. This is done at both 30 degrees of flexion and full
extension, if possible. Flexing the knee to 30 degrees isolates the MCL. Putting the leg
over the side of the examining table helps stabilize the femur, and placing a finger on
the medial joint line helps in assessing the amount of opening. The amount of medial
compartment opening is graded according to the American Medical Association (AMA)
guidelines. A grade I injury allows 0 to 5 mm of opening with a firm end point; a grade
II injury allows 5 to 10 mm with a firm end point; a grade III injury, or a
â€œcompleteâ€• tear of the superficial and the deep portions of the ligament, allows
greater than 10 mm of opening and the end point is usually soft. These numbers are all
with reference to the uninvolved side. The following figures demonstrate sequentially
the Lachman (Fig. 22-4), posterior drawer (Fig. 22-5), positioning the knee at 30
degrees (Fig. 22-6) and then applying a valgus load (Fig. 22-7); then positioning the
knee at 0 degrees (Fig. 22-8) and applying a valgus load (Fig. 22-9).
Any opening of the medial joint line to valgus stress with the knee at 0 degrees is a
much more ominous sign and is seen only with grade III injuries. It implies a large
magnitude of force to the knee and mandates careful scrutiny of cruciate ligaments,
the menisci, and the patella for stability. The posteromedial capsule and the vastus
medialis obliquus must also be suspected of involvement.
Another provocative test to determine the degree of injury is the external rotation
anterior drawer test. This maneuver assesses the amount of anterior translation of the
tibia under the femur at roughly 80 degrees of knee flexion and the foot in maximal
external rotation. It is not always possible to perform this test on the acutely injured
knee due to patient discomfort. It is positive if the amount of translation is
significantly increased compared with the contralateral knee. The sine qua non for this
test to be positive is disruption of the meniscotibial ligaments, allowing the meniscus
to move freely. If the meniscotibial ligaments are intact, the meniscus remains firmly
seated on the tibia, providing a buttress against the posterior femoral condyle and
thus resisting anterior translation of the tibia.
FIGURE 22-4
The Lachman test. With the knee at 30 degrees of flexion, an anterior stress is
placed on the tibia. Note the position of the examiner's right thumb on the
anteromedial joint line, allowing an accurate assessment of the degree of
anterior translation of the tibia as well as the quality of the endpoint.
FIGURE 22-5
The posterior drawer test. With the knee at 90 degrees of flexion, a posteriorly
directed force is placed on the tibia. Again, note the position of the examiner's
right index finger on the anteromedial joint line, allowing an accurate assessment
of tibial translation and the quality of the endpoint.
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FIGURE 22-6
The knee is positioned at 30 degrees of flexion.

FIGURE 22-7
A valgus stress is placed on the knee at 30 degrees of flexion. The amount of

medial joint space opening is assessed.
Imaging
A standard radiographic knee series is indicated in all cases of suspected MCL injury.
This should include a 45-degree flexion weight-bearing view and lateral and sunrise
views. If weight bearing is not possible, a simple anteroposterior view should be
substituted. Avulsions or osteochondral fragments seen on radiograph could
significantly affect the treatment plan. Any patient with increased laxity to valgus
stress and open physes should have stress views to localize the source of the laxity. In
chronic cases with instability, long-standing cassette radiographs of both lower
extremities should be obtained to assess alignment.
Magnetic resonance imaging offers an abundance of information that may alter the
treatment plan. The presence of meniscal tears, the location of the MCL tear, and any
concomitant injuries, ligamentous or otherwise, can be discovered. This study is
particularly useful when the physical examination is compromised but can also reveal
pivotal information, such as the presence of an entrapped MCL lesion (Figs. 22-10 and
22-11), even in apparently straightforward cases.
FIGURE 22-8 The knee is positioned at full extension.

FIGURE 22-9 A valgus stress is placed on the knee at full extension. The amount
of medial joint space is assessed.
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FIGURE 22-10
Magnetic resonance image demonstrating entrapment of the superficial MCL in

the joint with displacement of the meniscus out of the joint. Although a rare
finding, this mandates operative intervention to reduce and fix the MCL.
FIGURE 22-11 A magnified view of the area of interest in Figure 22-10.
In cases of disruption of the ACL, PCL, and MCL, careful consideration must be given to
evaluating the vasculature by angiogram, as the incidence of vascular injury in the
setting of knee dislocation is dangerously high. It is routine at our institution to obtain
an angiogram in this setting.
SURGERY
Positioning and Draping
Position the patient supine on the operating room table (Fig. 22-12). We do not
routinely use a tourniquet for this procedure, even in combined surgery. Examination
under anesthesia is performed at this time. The anticipated surgical incisions are
drawn on the skin along with appropriate anatomic landmarks (Fig. 22-13). The
surgical sites are prepared with alcohol and betadine and then injected with lidocaine
with epinephrine (1:100,000). The joint is injected as well. Allowing the epinephrine
to sit for the few minutes while the remainder of the setup, preparation, and drape
are completed achieves an excellent hemostatic effect. With this routine, the need for
inflating a tourniquet is obviated. A 5-lb sand bag is taped to the table so that the foot
can rest on it with the knee in 90 degrees of flexion. A side post is affixed to the table
adjacent to the tourniquet to prevent the limb from abducting at the hip while the
knee is flexed. The limb is prepared with Betadine and draped in standard fashion.
After sterile draping has been completed, a bolster of soft goods is placed between
the thigh and the side post so that the limb can be maintained at 90 degrees of flexion
and vertically oriented throughout the case without assistance. Preoperative
antibiotics are routinely administered.
In multiligament cases, the dorsalis pedis and posterior tibialis pulses are palpated and
marked on the skin. If any difficulty is encountered with palpation, a sterile Doppler
device is brought to the field and pulses are established before any surgical
intervention. They are checked intermittently throughout the case. This setup allows
for unencumbered physical examination of the limb at any time.
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FIGURE 22-12
The limb is draped free for easy intraoperative access and examination. Range of
motion is unencumbered. Note the window in the stockinette over the dorsalis
pedis pulse for intermittent monitoring in multiligament cases.
FIGURE 22-13
Anatomic landmarks of the knee for a medial approach.
Operative Technique
Acute Repairs
Acute repairs (<3 weeks) are performed in conjunction with cruciate ligament surgery.
It is important to restore the central pivot (ACL and PCL) to optimize repair of the
MCL. To restore normal knee stability, one must address all primary and secondary
restraints that are injured. We begin by reconstructing the cruciates (ACL, PCL, or
both). Our preferred graft for the ACL and PCL is tibialis anterior allograft. In most
cases, the cruciate surgery is done arthroscopically. However, especially in the acute
setting, fluid extravasation can be a problem. If there is any palpable increase in calf
pressure during the case, then arthroscopy is abandoned and the case is performed in
an open fashion. In performing these cases it is critical that the operating surgeon be
skilled with both open and arthroscopic techniques.
Based on the examination under anesthesia, MRI, and arthroscopy, selective

approaches with varying incisions are made. In most cases we use a medial hockey
stick incision. In certain cases we can identify on the MRI either a femoral- or tibial-
sided avulsion, and limited incisions directly over the injured area can be made (Figs.
22-10, 22-11, and 22-14, 22-15 and 22-16).
In all cases of medial-sided repair, knowledge of the anatomy of the medial side of the
knee is requisite to a successful operation. We endorse a layered approach to the
medial side of the knee (Fig. 22-17). We use a variety of fixation devices to repair the
MCL, but our most common material is sutures and/or suture attached to bone
anchors. Our primary goal is to achieve an anatomic repair and to â€œseal offâ€• the
MCL from synovial fluid from the joint. The leakage of synovial fluid onto the injured
MCL will cause delay and often complete cessation of the healing response.
The landmarks for the medial hockey stick incision include the adductor tubercle, the
medial epicondyle, the medial joint line, and the patella. Often the knee is too
swollen to palpate some of these superficial landmarks, and one must rely on an
estimated path of the MCL from femur to tibia. This incision balances well and
preserves the skin bridge between incisions (Fig. 22-18). We do not advocate a midline
incision over the patella. Make the incision through the skin and subcutaneous fat to
the level of the fascia of layer 1 (sartorial fascia). Gently sweep the fat layer off the
fascia with a
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lap sponge to create skin flaps (Fig. 22-19). The infrapatellar branch of the saphenous
nerve is always identified and preserved (Fig. 22-20). It emerges from under the
anterior border of the sartorius and travels on the fascia. It can usually be found about
1 cm above the joint line. Tag the nerve with a vessel loop. The saphenous nerve and
vein are rarely in the field and are easily avoided.
FIGURE 22-14
Intraoperative view of the entrapped MCL under the medial meniscus.
The fascia of layer 1 is incised in line with the skin incision. A convenient landmark
proximally is between the adductor tubercle and the medial epicondyle. This incision
extends from the epicondyle proximally to the gracilis distally. The sartorius muscle is
retracted posteriorly along with the hamstring tendons, whereas the vastus medialis
and the medial patellar retinaculum are taken medially. The pes bursa is teased away
and this exposes layer 2, comprised of the superficial MCL, the POL, and the joint
capsule anterior to the superficial MCL. Immediately deep to the superficial MCL is the
deep MCL, which is considered part of layer 3 (see Fig. 22-17). Further access to the
joint is established by performing an arthrotomy in the interval between the posterior
border of the superficial MCL and the anterior border of the POL. Exercise caution
when near the joint line to avoid transecting the meniscus. With the medial side of the
knee exposed, the surgeon should carefully inspect all structures including the medial
meniscus, the deep MCL, the superficial MCL, the POL, as well as the arms of the
semimembranosus.
FIGURE 22-15
Limited approach to the tibial insertion of the MCL.
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FIGURE 22-16
The avulsed tibial insertion of the superficial MCL. This was repaired with suture
anchors anatomically.
FIGURE 22-17
The anatomy of the medial side of the knee, organized by layers as described by
Warren et al.
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FIGURE 22-18
The skin incision.

FIGURE 22-19
Sharp dissection is continued to layer 1. Fairly generous skin flaps are raised.
The joint may be inspected arthroscopically either before or after the approach to the
medial side. Performing the exposure before the arthroscopy allows for easier
identification of tissue planes, as there is no distortion of the tissue by extravasated
fluid. However, the inconvenience of arthroscopy after opening the medial aspect of
the joint must be weighed. The sequence of repair should follow an orderly
progression from deep to superficial. First, repair meniscal tears that are accessible.
This includes peripheral tears that must be repaired back to the overlying ligament or
capsule using nonabsorbable suture. The capsule or ligament to which the meniscus is
being sewn must, of course, be reduced in an anatomic position as the sutures pass
through the tissue to ensure proper placement. Position the knee in extension for this
part of the case to avoid overconstraining the knee and restricting motion. Tag these
sutures and tie them only after the medial side has been reconstructed.
Next, the MCL itself is repaired. At the time of the MCL repair, we place â€œsoft
goodsâ€• under the knee so that it remains reduced and is in 30 to 40 degrees of
flexion. We take great care while repairing the MCL to not overconstrain the knee and
cause a flexion contracture. If the MCL has been avulsed from its femoral insertion,
repair both the superficial and the deep portion back to the epicondyle. A 0 or No. 1
nonabsorbable suture is used and the anatomy restored, sewing to the periosteum if
available or using bone anchors (Fig. 22-21). If it has been torn midsubstance, then it
is repaired anatomically. If the tibial insertion of the deep MCL is avulsed, this should
be sutured back to the periosteum of the tibia under the superficial portion of the
ligament. Fixing the deep MCL creates
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a stable post on the medial side of the knee from which to continue the medial-sided
repair. In the event that the tibial insertion of the superficial MCL has been avulsed as
well, this should be repaired either with suture to the periosteum at its insertion or
with suture anchors similarly placed. If the capsule has been avulsed, repair it with
suture anchors placed just below and parallel to the joint line. Next, the POL is
assessed.
FIGURE 22-20
The infrapatellar branch of the saphenous nerve is identified and preserved.

FIGURE 22-21
Suture anchors at the medial epicondyle.
If a POL repair is planned in isolation (as in anteromedial rotary instability [AMRI]), a

limited incision can be employed (Fig. 22-22). The medial joint line is marked and an
incision is carried down along the posterior one third of the medial femoral condyle.
This incision is approximately 4 cm in length, with two thirds of the incision being
placed below the joint line. A similar approach as used with the MCL repair is used
through layer 1. Layer 2 is then identified and the junction of the superficial MCL and
POL is located. This can be facilitated with an intraoperative valgus stress to the knee
with palpation of the medial joint line. With this maneuver, the MCL becomes taught,
and the POL remains lax allowing its identification. The junction between MCL and
POL is then divided and the underlying capsule is exposed (Fig. 22-23). The capsule is
incised superior to the medial meniscus, and the meniscus is tagged with
nonabsorbable suture for later repair into the MCL (Fig. 22-24). The POL and MCL are
separated off of the capsule with the use of an elevator. We use three to four braided,
No. 2 nonabsorbable sutures through the POL in a horizontal mattress fashion and
advance the POL to the MCL to recreate the posteromedial tension (Fig. 22-25). The
repair is zone specific to the POL, advancing only the portion of the ligament that has
been traumatized. The suture depth in the superficial MCL can be adjusted depending
on the severity and location of the injury
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to vary the degree of imbrication that is achieved (Fig. 22-26). The sutures are then
tied down over the MCL in slight flexion (15 degrees), and the repair is completed
after repair of the sartorius fascia (layer 1) with absorbable suture (Fig. 22-27).
FIGURE 22-22
A limited medial incision can be employed when only repairing or imbricating the
POL.
FIGURE 22-23
The demarcation between the POL and MCL is identified and split for the
imbrication.
FIGURE 22-24
A: The capsule is incised superior to medial meniscus and the joint is exposed
(MFC, medial femoral condyle; MM, medial meniscus). B: The medial meniscus is
tagged (black suture) and this suture is incorporated into the final repair.
At this point, the knee is carefully taken through a full range of motion to ensure that
the repair has not â€œcapturedâ€• the knee. The tension can be adjusted if a full
range of motion is not achieved. The surgeon must manually maintain anteroposterior
reduction during this maneuver because the cruciate(s) has not been attached on the
tibial side (in the setting of combined injury). Next, the knee is taken to 90 degrees
extension and the PCL is tensioned and secured. Finally, the knee is brought out to full
extension and the ACL graft is tied over a post. Closure is performed with the knee
flexed to 90 degrees, and a postoperative drop-lock brace is applied with the knee in
full extension.
Clironic Cases
In chronic cases, if surgery is indicated, every effort is made to repair the medial side
primarily, and this is often possible. It is important to remember that the tissues in
cases of chronic instability are not easily identified and are often severely attenuated.
The surgeon must have several different techniques in his repertoire for managing
chronic MCL laxity. The goal is to
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achieve an anatomic repair or reconstruction. This may necessitate the use of both
primary repair and reconstructive techniques. Preoperatively, the MRI is helpful in
identifying the location of the injury to the MCL. This can usually be seen on the
coronal images and correlated with the arthroscopic view of the compartment under a
valgus stress. Depending on where the pathology is (i.e., femoral, midsubstance, or
tibial) the focus of the repair should be to this area.
FIGURE 22-25 A: The mattress sutures have been placed in the POL and are ready
for advancement through the MCL. The depth is varied depending on the degree
of imbrication desired. The joint line is indicated by the blue marker (cadaver
knee) B: The sutures have been passed and the repair is shown (live surgery).
FIGURE 22-26
The sutures are passed and the depth is determined by the degree of instability
present on examination (cadaver knee).
On occasion the medial structures are attenuated to the point where repair is not
possible, although in most cases we have been able to use existing tissue to repair the
MCL. The underlying principle is to build off of the superficial and deep MCL and
advance the POL on this complex if laxity persists. The choice of graft can be either
allograft or autograft. We have used a number of different grafts, including Achilles
tendon and tibialis anterior allograft, as well as semitendinosus autograft (Fig. 22-28).
The principle of reconstruction remains the same regardless of graft choice.
The semitendinosus is easily harvested through the medial incision. Isolate it just
inferior to gracilis and release it from its tibial insertion, preserving as much length as
possible. Place a whipstitch of 0 suture in the free end. Carefully dissect the tendon
free of all investing soft tissue with a combination of blunt and scissor dissection. In
chronic cases, the tendon may be relatively adherent to surrounding scar, and
patience is required, as careful preparation is crucial to successful harvesting. A
closed-ended tendon stripper is used to harvest the tendon. Pay close attention to the
direction of the tendon, cognizant that it inserts on the ischial tuberosity, and
recreate that trajectory with the stripper. Once harvested, take the graft to the back
table, divest it of all soft tissue, preserve all usable
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length, and place a whipstitch in the other end of the tendon. Fold the graft onto
itself, doubling it over a piece of No. 5 suture, and then sew it into one bundle with 0
absorbable suture. It is obviously expedient if the graft preparation can be undertaken
by a second assistant, so as not to interrupt the flow of the reconstruction.
FIGURE 22-27 A: The sutures are tied over the MCL and the repair is completed
(cadaver knee). B: The final repair after closure of the sartorial fascia (live
surgery).
FIGURE 22-28
A prepared hamstring autograft.
Using the exposure as described previously, the medial epicondyle and the site of the
tibial insertion of the superficial MCL should be clearly identified. The graft should
recreate the path of the superficial MCL. The graft should also recreate the isometry
of the native MCL. Toward this end, drill a hole at the tibial insertion site and place a
screw with a soft-tissue washer through the graft into the pilot hole and tighten it.
Next, palpate the medial epicondyle and hold the femoral side of the graft at the
anticipated insertion. Take the knee through flexion and extension to assess whether
the graft is placed isometrically. Adjust the femoral insertion point until the isometric
point is found. Then drill a pilot hole at this site. Position the knee in full extension
and apply gentle tension on the graft in an anatomically correct position. Thread a
screw with a soft-tissue washer through the graft and secure it in the pilot hole. Trim
the excess graft appropriately. Having established a stable post, the remainder of the
medial-sided repair proceeds in an orderly sequence as described previously. It is our
experience that the POL must be advanced more often in chronic cases than in acute
cases. We assess it carefully and advance it onto the reconstructed MCL as necessary.
Both the superior and inferior ends of the POL should be anatomically restored as
shown in Figure 22-29. Take great care not to overtighten the POL, as this will result in
a flexion contracture. Pass these sutures in a horizontal mattress-type stitch through
the superficial MCL and into the underlying periosteum to increase the security of the
repair. This portion of the procedure should be done with the knee at 20 to 30 degrees
of flexion. The cruciate ligaments are tensioned and fixed in the same fashion as in
the acute setting.
FIGURE 22-29
The POL is advanced according to the zone of injury. With femoral lesions,
advancement is carried out along 1,2, and possibly 3 depending on the injury
severity. With tibial lesions, 3,4, and 5 are more routinely advanced.
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Pearls and Pitfalls
Always be sure to maintain the knee at approximately 15 degress of flexion

(near full extension) while the POL is advanced so as to not capture the knee
and compromise postoperative motion.
Identifying the POL from the MCL can be facilitated by a valgus stress at 30
degrees of knee flexion. With this maneuver, the POL remains lax while the
MCL is placed under tension.
In chronic cases, there is typically a substantial amount of AMRI contributing to

the instability pattern, which can usually be addressed with POL advancement
as described.
Use the preoperative MRI to define where the MCL injury is and focus POL
advancement at this point. For example, if it is on the femoral side, you would
focus the advancement in this area.
For reconstruction (using a graft), placing a K-wire in the medial epicondyle

and looping a suture can help to locate the isometric point of the ligament
prior to pilot hole placement.
Cruciate ligament reconstruction takes precedence over the MCL with respect to the
postoperative management. In general, the rehabilitation follows the regimen that is
used for isolated ACL or PCL reconstructions, with careful attention to any decrease in
range of motion. Achieving full extension is emphasized. If medial repair or
reconstruction is the only procedure, the knee is braced in extension for 2 to 4 weeks
to allow for healing. A slight varus bend can be applied to the brace to protect the
repair. During this time, continuous passive motion is used two or three times a day.
The brace is then unlocked, and range-of-movement exercises begun. Strengthening
begins at 6 weeks. Bicycling and low-impact closed-chain exercises are an integral part
of the strengthening phase. The brace is typically removed at 6 to 8 weeks. Small
flexion contractures are tolerated through 8 to 12 weeks of rehabilitation, as it is
thought to be protective of the repair and allows for maturation of scar tissue. A small
flexion contracture is also postulated to allow the dynamic stabilization of the
semimembranosus to strengthen the repair. If full extension is achieved in the
operating room, it is possible to regain it through aggressive therapy at the
appropriate time. Weight bearing is initially touch down and progresses to full
gradually after 2 weeks. Crutches are discontinued only when ambulation is without a
limp.
Complications
Complications regarding surgery of the medial collateral ligament itself are few.
Certain risks are associated with anesthesia, surgery in general, and the procedures
that may be performed concomitant with MCL repair. Specific to the medial-side
reconstruction itself, the main complication is loss of motion. This is a problem that
may occur with any knee surgery and, specifically, any knee ligament surgery.
However, it has been recognized that MCL surgery is a particular risk factor for loss of
motion after ligament reconstruction. Of course, technical aspects of the surgery
come into play, as the repair of the medial side must not be so aggressive as to restrict
motion. Tensioning the repaired or advanced tissue with the knee in full extension
helps to prevent this complication. The timing of knee ligament reconstruction was
also found to be important, as surgery less than 1 month after the injury was a risk
factor, unless the knee was grossly unstable. In general, decreased postoperative
range of motion can be avoided by operating on knees that have full range of motion
preoperatively. In addition, an intraoperative confirmation of full knee excursion
should be performed after the reconstruction is complete but before closure.
RESULTS
Not surprisingly, the less severe the injury, the better the prognosis. In cases of
isolated MCL injury, rehabilitation is fairly aggressive, tempered by the severity of the
injury to the knee. Return to sports is allowed when pain, tenderness, and instability
are gone, range of motion is full, and strength and agility are nearly normal. This
typically takes from 2 to 6 weeks for partial tears.
The prognosis for cases of acute, isolated grade III MCL tears is a bit less optimistic.
The rehabilitative protocol is similar in nature to that for partial tears, but the
progression is typically dramatically slower. The initial phase of pain and inflammation
control may last up to 6 weeks in and
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of itself. The maintenance of range of motion is more difficult than it is for partial
tears. Tears from the femoral insertion have been shown to heal quicker and more
reliably than tears of the meniscotibial ligament with their associated capsular tears.
The key to successful nonoperative management of isolated MCL injuries is ensuring
that it is truly an isolated injury. Even so, return to sports may take more than 12
weeks.
For combined injuries, the prognosis is worse. MCL tears in the setting of a knee
dislocation are potentially sport-ending injuries. Medial collateral ligament tears
associated with only ACL tears are surmountable obstacles, but the risk of persistent
residual laxity is not insignificant. The criteria for return to sport are the same as
those described previously, but the time frame is longer. Return of full strength and
proprioception may take up to a year.
RECOMMENDED READING
American Medical Association, Committee on the Medical Aspects of Sports. Standard
nomenclature of athletic injuries. Chicago: The Association; 1966.
Baker CL, Liu SH. Collateral injuries of the knee: operative and nonoperative
approaches. In: Fu FH, Harner CD, Vince KG, eds. Knee surgery. Baltimore: Williams &
Wilkins; 1994:794-795.
Clayton ML, Weir GJ, Jr. Experimental investigation of ligamentous healing. Am J Surg.
1959;98:373-378.
Fetto JF, Marshall JL. Medial collateral ligament injuries of the knee: a rationale for
treatment. Clin Orthop. 1978;132:206-218.
Frank C, Woo SL-Y, Amiel D, et al. Medial collateral ligament healing: a

multidisciplinary assessment in rabbits. Am J Sports Med. 1983;11:379-389.
Harner CD, Irrgang JJ, Paul J, et al. Loss of motion after anterior cruciate ligament
reconstruction. Am J Sports Med. 1992;20:499-506.
Hart DP, Dahners LE. Healing of the medial collateral ligament in rats: the effects of
repair, motion, and secondary stabilizing ligaments. J Bone Joint Surg Am.
1987;69A:1194-1199.
Hughston JC, Andrews JR, Cross M J, et al. Classification of knee ligament instabilities.
I. The medial compartment and cruciate ligaments. J Bone Joint Surg Am.
1976;58A:159-172.
Hughston JC, Barrett GR. Acute anteromedial rotatory instability: long-term results of
surgical repair. J Bone Joint Surg Am. 1983;65A:145-153.
Hunter SC, Marascalco R, Hughston JC. Disruption of the vastus medialis obliquus with
medial knee ligament injuries. Am J Sports Med. 1983;11:427-431.
Indelicato PA. Nonoperative management of complete tears of the medial collateral

ligament. Orthop Rev. 1989;18:947-952.
Kannus P. Long-term results of conservatively treated medial collateral ligament

injuries of the knee joint. Clin Orthop. 1988;226:103-112.
Mok DW, Good C. Nonoperative management of acute grade III medial collateral
ligament of the knee: a prospective study. Injury. 1989;20:277-280.
Muller W. The knee: form, function, and ligament reconstruction. Berlin: Springer-
Verlag; 1982.
Sandberg R, Balkfors B, Nilsson B, et al. Operative versus nonoperative treatment of

recent injuries to the ligaments of the knee: a prospective randomized study. J Bone
Joint Surg Am. 1987;69A:1120-1126.
Shelbourne KD, Patel DV. Management of combined injuries of the anterior cruciate
and medial collateral ligaments. Instr Course Lect. 1996;45:275-280.
Shelbourne KD, Porter DA. Anterior cruciate ligament-medial collateral ligament

injury: nonoperative management of medial collateral ligament tears with anterior
cruciate ligament reconstructionâ€”a preliminary report. Am J Sports Med.
1992;20:283-286.
Thornton GM, Johnson JC, Maser RV, et al. Strength of medial structures of the knee
joint are decreased by isolated injury to the medial collateral ligament and
subsequent joint immobilization. J Orthop Res. 2005;23:1191-1198.
Tipton CM, James SL, Mergner KW, et al. Influence of exercise on strength of medial
collateral ligaments of dogs. Am J Physiol. 1970;218:894-902.
Warren LF, Marshall JL. The supporting structures and layers on the medial side of the
knee. J Bone Joint Surg Am. 1979;61A:56-62.
Wilson TC, Satterfield WH, Johnson DL. Medial collateral ligament tibial injuries:
indication for acute repair. Orthopedics. 2004;27:389-393.
Woo SL-Y, Inoue M, McGurk-Burleson E, et al. Treatment of the medial collateral

ligament injury. II. Structure and function of canine knees in response to different
treatment regimens. Am J Sports Med. 1987;15:22-29.
Woo SL-Y, Young EP, Ohland KJ, et al. The effects of transection of the anterior
cruciate ligament on healing of the medial collateral ligament: a biomechanical study
of the knee in dogs. J Bone Joint Surg Am. 1990;72A:382-392.
> Table of Contents > Part Three - Ligament Injuries and Instability > 23 - Surgical Technique
for Knee Dislocations
23
Surgical Technique for Knee Dislocations
Bryan T. Kelly
Struan H. Coleman
Russell F. Warren
Knee dislocations occur whenever the articular surfaces of the distal femur and
proximal tibia displace completely from one another (Fig. 23-1). Dislocations are most
usefully described according to the associated degree of local soft-tissue injury (e.g.,
closed or open grade I, II, III), and the primary direction of displacement. The
direction of a particular knee dislocation is designated according to the direction of
displacement of the tibia with respect to the femur (6,7). Anterior, posterior, medial,
lateral, and rotatory dislocations have been described. Straight anterior or posterior
dislocations are the most commonly reported. Other forms of dislocation (medial,
lateral, or rotary) are far less common and are often combined with anterior or
posterior dislocation (6,7,9,13,14,15).
The majority of reported dislocations of the knee occur in association with motor-
vehicle accidents. The high velocity and ultra-high-force nature of this type of trauma
often leads to associated head and thoraco/abdominal injuries of higher priority and,
not uncommonly, to rapid mortality. Of primary importance in the multiple trauma
victim is the immediate treatment of life-threatening conditions. Once these issues
have been properly addressed, attention should be given to a dislocated knee.
We believe it is appropriate to attempt immediate closed reduction and
immobilization of all knee dislocations if possible. The massive displacement of the
bony architecture that occurs during dislocation causes extensive disruption of the
joint capsule and ligaments. Usually, at least three major ligaments and the posterior
joint capsule are damaged. Therefore, the degree of instability is usually so severe
that gentle manipulation will easily achieve reduction. Once reduced, a thorough
evaluation of the neurologic and vascular status of the injured extremity is critical.
Adequate determination of pedal pulses, in addition to ankle-brachial indexes
bilaterally, should be performed routinely in this setting. Failure of early diagnosis of
vascular injuries has resulted in extremely high amputation rates, up to 90% in some
series (2,4).
In some medial dislocations, it may be impossible to reduce the knee by closed

manipulation, since the medial femoral condyle may have â€œbutton-holedâ€•
through the medial joint capsule and retinaculum. These cases require open
reduction. Furthermore, the joint is often rendered so unstable in medial and lateral
dislocations that closed reduction, once achieved, is very difficult to maintain. Under
these circumstances, after a careful vascular and neurologic examination, the knee
should be immediately immobilized in a brace, cast, or splint and a radiographic
evaluation made of the reduction, with adjustment if required. In some cases
placement of an external fixator is required to maintain the reduction. Immobilization
in a significant degree of flexion (45 degrees or more) may be needed to achieve and
maintain an adequate reduction. In contrast, reduction is often relatively easy to
maintain after manual reduction of straight anterior or posterior dislocations in which
the medial and lateral soft tissues have simply been â€œpeeled offâ€• anteriorly or
posteriorly in a longitudinally intact sleeve of tissue. When these tissues are reduced
to their original positions, they tend to provide enough stability to maintain adequate
reduction with relatively minimal external support.
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FIGURE 23-1
Lateral radiograph of an acute anterior knee dislocation.
NONOPERATIVE TREATMENT
In closed dislocations, without intra-articular fracture, and where a good reduction
can be easily maintained in a cast or brace, nonoperative treatment with early range
of motion (ROM) therapy may achieve an acceptable functional outcome. However, if
this course is chosen, frequent radiographic assessment of the adequacy of the
reduction must be performed during the first several weeks. This is because posterior
subluxation will often lead to a poor outcome, and later attempts at surgical
correction of this problem will have a low success rate once the soft tissues have
â€œmatured,â€• creating a fixed, posteriorly subluxed position. A hinged external
fixator may be used to prevent posterior subluxation but allow for progressive ROM in
cases where the reduction is difficult to maintain.
Delayed Arthroplasty or Arthrodesis

In older patients with massively comminuted intra-articular fractures, the most
prudent course of action may be to simply immobilize the knee in a position of
acceptable alignment for a period of time, during the initial phase of bone and soft
tissue recovery and healing, before performing a primary knee arthroplasty or
arthrodesis as the definitive surgical treatment. Immobilization may take the form of a
cast, brace, or external fixator device.
It is of the utmost importance to assess the condition of the local skin and soft tissues
(Fig. 23-2), neurologic status, and, particularly, the vascular status of the involved
extremity, both initially and at frequent intervals after initial evaluation. Accurate
monitoring of the pedal pulses is critical. Determination of the status of the knee
ligaments, although of interest, is an issue of lower priority early after a knee
dislocation. The evaluation and early management of a dislocation should be done in a
manner that does not jeopardize or hinder the evaluation of the vascular status of the
limb.
After the initial reduction, the factors that we consider most important in making
surgical decisions about dislocations of the knee include:
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FIGURE 23-2
Typical skin trauma after closed knee dislocation.
The adequacy of the reduction. If closed reduction is impossible because of

interposed soft tissue, open reduction should be performed at the earliest
opportunity (within several hours after dislocation). If an adequate closed (or
open) reduction can be achieved but cannot be maintained, the surgical repair
of damaged ligaments should be performed as soon as the patient's general
condition and the condition of the local soft tissues permit (from several hours
or days to 2 weeks after dislocation). If the interval between injury and
definitive fixation is anticipated to be a prolonged period, the use of an
external fixator may be employed.
The condition of the soft tissues. In open injuries, surgical irrigation and
debridement should be performed at the earliest opportunity (within several
hours after injury). In most of these cases, and in closed dislocation with
severe blunt injury to the local skin and soft tissues, attention to surgical
stabilization of the knee should be delayed until the condition of these tissues
improves to a level appropriate to the performance of extensive open-knee
ligament surgery (within several days to 2 weeks after injury).
The status of arterial blood flow to the distal extremity. The impairment of
popliteal arterial blood flow is the most common and severe complication of
knee dislocations, with a reported incidence ranging from 5% with low energy
trauma to 40% with high energy, anterior or posterior dislocations (2,5,15). Our
plans for surgery are guided by the following criteria. The absence of pulses at
the ankle and foot is an absolute indication for immediate surgical exploration
and repair of the popliteal artery. Arterial blood flow must be re-established
within 6 hours after the initial injury; otherwise, severe ischemic effects
leading to amputation are inevitable in a great majority of cases. The absence
or diminution of pulses obviates the need for preoperative angiography in favor
of immediate exploration and repair of the popliteal artery, with
intraoperative angiography, in this setting, reserved for identifying the site of
injury. Patients with normal pulses may be monitored by careful vascular
examination. In a study by Treiman et al (16), of 115 unilateral knee
dislocations, the incidence of popliteal artery injury was 23%. However, there
was a 79% incidence of popliteal injury associated with abnormal pedal pulses.
In the remaining 86 patients with normal pulses, the arteriogram was normal in
77, showed spasm in 5 patients, and revealed an intimal flap in 4 patients.
None of the patients with normal pedal pulses required arterial repair. Hollis et
al (4) also concluded that routine arteriography is unnecessary in patients with
a normal physical examination after reduction of the knee dislocation. The role
of magnetic resonance angiography in making the diagnosis of a vascular injury
in this setting remains to be determined, but appears to be particularly useful
for evaluating those patients with extensive soft tissue injury and normal
pulses when surgery is being planned (10,11).
In approaching soft-tissue reconstruction in a dislocated knee, the surgeon should

consider and prepare options for repairing and grafting any or all of the main
stabilizers of the knee, including the anterior cruciate ligament (ACL), posterior
cruciate ligament (PCL), medial collateral ligament
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(MCL), lateral collateral ligament (LCL), popliteus tendon and popliteofibular
ligament, patellar tendon, patella, and quadriceps tendon. These may include
autografts from the injured or contralateral knee, allografts and, very occasionally,
ligament augmentation devices (LADs). The surgeon should assess the availability of
these resources and obtain appropriate informed consent for their use before taking
the patient to the operating room.
If vascular reconstruction has been performed, the use of a tourniquet may be

contraindicated. Intraoperative fluoroscopy is often helpful in the placement of bone
tunnels, which may be required for reconstruction of the PCL. Intraoperative plain
radiographs should also be available to assess the correction of posterior subluxation
before the conclusion of surgery.
Inspection of the menisci and articular surfaces, and preparation of the intercondylar
notch for ligament reconstruction, is most easily and thoroughly accomplished
arthroscopically. However, capsular disruption may preclude routine arthroscopy
because of excessive extravasation of fluid into the soft tissues. Under these
circumstances, we usually favor a brief arthroscopic inspection, with low-pressure flow
and systemic wrapping of the calf, before proceeding to an open approach. We have
also found â€œdryâ€• or â€œsemi-wetâ€• arthroscopy, with intermittent fluid flushes,
to be a viable option in inspection, debridement, and reconstruction in the
intercondylar notch. The surgeon should be prepared, however, to perform the entire
procedure in an open fashion, if necessary, and if arthroscopy is used, an awareness of
fluid extravasation is paramount. Significant fluid extravasation may make open
procedures (e.g., MCL and LCL reconstruction) more difficult, and can lead to a lower-
leg compartment syndrome.
SURGERY
Technique
The patient is placed supine on an operating table that is adjustable to allow variable
amounts of flexion at the hip and knee. We position the patient longitudinally on the
table in such a way that the patient's knee-flexion crease is just distal to the distal
flexion joint of the table, so that the foot of the table may be dropped to allow up to
90 degrees of knee flexion during the surgical procedure. If available, an operating
table that has these features and is also designed for use with an image intensifier is
optimal. Our preferred method of anesthesia is continuous lumbar epidural blockade.
In our experience, this has provided excellent, adjustable, and long-lasting muscle
relaxation and pain relief.
The first event after reaching an appropriate level of anesthesia should be a thorough
examination of the knee. This should be done before prepping and draping the patient
to allow easy manipulation of the knee and comparison with the opposite knee. A
thorough assessment should be made of the status of the ACL, PCL, MCL, LCL, and
posterolateral corner (Figs. 23-3, 23-4, 23-5 and 23-6).
FIGURE 23-3
Assessment of lateral collateral ligament injury.

FIGURE 23-4
Severe posterior and lateral injury.
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FIGURE 23-5
Assessment of external rotatory stability of the knee.

FIGURE 23-6
Assessment of posterior stability of the knee.
If possible, a tourniquet is then placed around the upper thigh at as high a level as
possible, over a layer of protective cast-padding material. We then seal off the
tourniquet area with adhesive plastic drapes to avoid the skin irritation occasionally
seen when Betadine (povidone/iodine) solution seeps under the tourniquet and
remains there during periods of tourniquet inflation. The entire extremity is then
prepped by first scrubbing with Betadine scrub solution and then painting with
concentrated-Betadine swabs. We seal off the lower leg and foot with an adhesive
plastic dressing. Further standard draping is done with sterile paper drapes. If the use
of contralateral autografts is anticipated, the opposite leg is also prepped and draped
in the same manner.
A separate sterile table is set up away from the primary surgical field, at which an
assistant is provided with the appropriate instruments for preparing graft material. If
allografts are to be used, an assistant may begin graft preparation simultaneously with
the start of the surgical procedure on the knee.
If arthroscopy is to be performed as part of the procedure, it is often done first. We

occasionally harvest an autograft from the middle third of the patellar tendon, before
the arthroscopic examination, to allow the assistant to prepare the graft while the
rest of the procedure continues. We avoid using the tourniquet during arthroscopy by
adding 1 mg of epinephrine to each 3,000-mL bag of arthroscopic irrigation solution,
and have been quite satisfied with the level of hemostasis and clarity of visualization
this affords. Arthroscopy begins with a routine visual inspection of the entire knee in a
predetermined sequence as follows: suprapatellar pouch, patella, lateral gutter,
popliteus recess, femoral trochlea, medial gutter, medial compartment, intercondylar
notch and ligaments, and lateral compartment. Once the visual inspection is
complete, we proceed to the required repairs (Fig. 23-7).
FIGURE 23-7
Arthroscopic view of midsubstance rupture of the PCL.
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At this point, we may discontinue arthroscopy to harvest patellar tendon,
semitendinosus, and gracilis grafts, as necessary. After the graft harvest is completed,
we resume arthroscopy to perform any required meniscal work, as well as a
debridement and preparation of the intercondylar notch. If fluid extravasation
precludes further arthroscopy, we proceed to an open approach.
All remnants of ruptured ligaments, if unusable in the reconstruction, are removed.

Enough bone is also removed from the roof and sides of the intercondylar notch to
allow unencumbered movement of the knee so as to prevent impingement of the
intended grafts.
At this stage, we prepare the tibial attachment site of the PCL arthroscopically using a
posteromedial portal for visualization while debridement is performed through an
anterior portal and the intercondylar notch (Fig. 23-8).
As previously mentioned, extensive capsular injury may compromise or even

contraindicate arthroscopy in a fluid medium. If fluid loss is marked, the repair
medially may be performed before addressing the ACL or PCL to allow better
arthroscopic evaluation and ligament reconstruction. For open reconstruction of the
knee ligaments, we have been satisfied with the utility of a long, oblique skin incision
that courses over the front of the knee in a proximal-lateral to distal-medial direction
(Figs. 23-9 and 23-10). With appropriate extension of the incision and the elevation of
skin flaps, this type of incision allows access to all aspects of the knee.
Our approach to multiple ligament reconstruction in the acute setting is guided by the
following principles:
The sequence of reconstruction to which we adhere is PCL, ACL, MCL,

posterolateral corner, LCL, and others (e.g., extensor mechanism, iliotibial
band).
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Ligaments that have been avulsed from a bony attachment with little or no
apparent intrasubstance injury are treated with primary repair and with
augmentation if necessary (Figs. 23-11 and 23-12).
Allografts for PCL and ACL will greatly facilitate the reconstruction process. We
prefer an Achilles allograft for the PCL reconstruction and a bone-tendon-bone
(BTB) patella tendon allograft for ACL reconstruction.
Intrasubstance ruptures of the PCL and ACL are reconstructed with a BTB
patella tendon, semitendinosus, or gracilis autograft. However, these
autografts may be needed for the medial or lateral side reconstructions.
For combined ACL and PCL reconstructions, we make all of the required bone
tunnels before passing any grafts, to avoid injury to the grafts.
Intrasubstance ruptures of other ligaments are treated with primary repair and
augmentation.
If ipsilateral autografts are the only available biologic grafts, and both the ACL
and PCL are ruptured, we give priority to a BTB autograft from the middle
third of the patella in the PCL reconstruction. In these cases, we reconstruct
the ACL with a semitendinosus-gracilis autograft.
Injuries to the MCL are treated with primary repair, advancement of posterior
tissues (if appropriate), and/or augmentation with a hamstring tendon
(Bosworth technique) or LAD.
Injuries to the popliteus tendon and popliteofibular ligament are usually

treated with primary repair with augmentation, using a strip of iliotibial band.
If the muscle-tendon junction has been disrupted, we perform a tenodesis of
the tendon to the posterolateral tibia or posterior fibular head after ensuring
the competence of the tendon's femoral attachment.
Injuries to the LCL are treated with primary repair and/or augmentation with a
strip of biceps tendon graft. Reconstruction with an achilles tendon allograft
has been used more recently.
FIGURE 23-8
Preparation of the posterior tibial ledge may be accomplished arthroscopically by

viewing through a posteromedial portal.
FIGURE 23-9
A long oblique skin incision for open multiple ligament reconstruction.

FIGURE 23-10
Initial exposure after long oblique skin incision.
Reconstruction of the Posterior and Anteriar Cruciate

Ligaments
We recommend using image intensification for making the tibial bone tunnel used for
reconstruction of the PCL, especially if the procedure is to be done arthroscopically. In
our experience, even a small portable system, used in conjunction with any standard
drill guide, aids accurate and safe placement of a guidewire into the appropriate site
on the posterior tibia. The anterior entry site for the tibial tunnel for the PCL is well
distal to the usual ACL tunnel site. Alternatively, the tibial tunnel can be started
lateral to the tibial tubercle to improve the tibial angle and thus decrease the angle
posteriorly. The exit site of the tunnel, at the back of the tibia, is critical. Ideally, the
guidewire should exit in the middle of the posterior facet of the natural PCL origin.
This ensures that the reconstructed ligament will
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be anchored far enough posteriorly to function properly. A common error is to accept
guidewire placement that is too close to the level of the joint line. The more
proximally the wire is placed, the closer the top of the eventual tunnel will be to the
articular cartilage of the joint. As the top of the tunnel is elevated superiorly, it also
advances anteriorly owing to the geometry of the ledge, which has a gentle anterior
slope. Furthermore, a tunnel exiting too close to the joint line has an area of weakness
(a thin bone bridge) near its exit site, which will often break off during ligament
tensioning, causing further anterior movement of the graft-attachment site on the
tibia. A properly placed guidewire, when seen radiographically on a lateral view,
parallels the angle made by the joint of the proximal tibia with the fibula and runs just
superior and parallel to the posterior curve of the proximal tibial cortex, just
underneath the posterior ledge (Fig. 23-13). Tips for this procedure are outlined in
Table 23-1.
FIGURE 23-11
Open view of knee showing femoral avulsion of the ACL controlled with sutures
before primary repair.
FIGURE 23-12
The semitendinosus tendon is being harvested for use as an augment for the ACL
repair.
If the surgeon wants to create the tibial tunnel for the PCL reconstruction
arthroscopically, we find that a posteromedial arthroscopic portal is helpful. With a
posteromedial portal the surgeon can view with a 30- or 70-degree arthroscope either
from the posteromedial position or through the notch, and obtain an excellent view of
the posterior tibial ledge. Preparation of the area can then be easily accomplished by
working through either an anterior portal or the posteromedial portal. We locate the
position for the posteromedial portal by pushing the arthroscope to the posteromedial
corner of the medial compartment, behind the MCL if it is present, or simply to the
junction of the A and B zones of the medial meniscus. If the scope is pushed up against
the capsule at this point, a correctly positioned portal can be placed by cutting in the
area of skin transilluminated by the scope. Table 23-2 lists a few extremely helpful
guides to PCL graft preparation (Fig. 23-14).
FIGURE 23-13
Fluoroscopic view of proper interference-screw placement for fixation of the PCL

graft in the tibial tunnel. Note that the orientation of the screw is nearly parallel
to the posterior tibial cortex and at the same level.
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TABLE 23-1. Pearls of Tibial Tunnel Creation
1. Set the initial length of the guidewire, from tip to drill chuck, such
that when the guidewire has been advanced to the point at which the
drill chuck hits the drill guide, the guidewire has just reached the
inner table for the posterior tibial cortex.
2. Advance the guidewire under radiographic control from the beginning

of the procedure. Stop advancement of the wire under power when
the posterior cortex is reached.
3. Penetrate the posterior cortex slowly, viewing it directly from the

posterior medial portal and radiographically.
4. Grasp the guidewire with an arthroscopic grasper or a clamp to ensure

that it is not inadvertently driven further posteriorly during reaming
over the wire.
5. Set the length of the reamer, from tip to drill chuck, to prevent
inadvertent overpenetration of the reamer past the posterior tibial
cortex. Drill over the guidewire while viewing the advancing reamer
with frequent fluoroscopic radiographic spot pictures.
6. Chamfer the edges of the tunnel with a rasp.
TABLE 23-2. Pearls of Posterior Cruciate Ligament

Graft Preparation
1. In using a bone-tendon-bone graft, we have found that the lead bone
plug on the graft should not exceed 20 mm in length or it will have a
great degree of difficulty turning the corner at the tibial exit site to
head up into the femoral tunnel.
2. We have also found it helpful to allow the trailing bone plug to be 30

to 40 mm long or longer. This makes fixation in the tibial tunnel, with
an interference screw placed from anteriorly, much easier.
3. If an autograft from the middle third of the patellar tendon is being

used, we fashion the short bone plug from the patella and the long one
from the tibial tubercle, since the tibial tubercle can easily take such
a long plug.
4. In passing the graft from the tibial tunnel to the femoral tunnel, it is
helpful to manipulate the graft with an instrument that has a wide
rolled edge that acts as a sort of â€œpulleyâ€• from anteriorly, so that
as traction is applied to the draw-sutures, the pulley causes the part
of the graft that has not emerged from the back of the tunnel to be
drawn posteriorly. As this happens, the part of the graft that has
already emerged from the tunnel curls around the instrument,
following the actual traction vector, which is directed anteriorly
toward the femoral bone tunnel (see Fig. 23-14).
5. Alternatively, an Achilles tendon allograft may be used for

reconstruction of the PCL. In this case, we pass the free end first and
impact the trailing bone plug into the tibial tunnel. The graft is fixed
on the femoral side with an AO screw and washer.
The site for the femoral bone tunnel in PCL reconstruction is chosen at a point
anterior and superior on the medial wall of the intercondylar notch. We have been
satisfied with the results obtained by selecting a point for guidewire placement that
corresponds to the anterolateral component of the PCL insertion (Fig. 23-15). When
the notch is viewed with the knee flexed at 70 to 90 degrees, and is at the axilla
(junction) of the medial wall and the roof of the notch (the notch usually lies
approximately 1 cm proximal to the edge of the articular surface), the point of
guidewire placement is optimal. The guidewire is placed from the anteromedial cortex
of the distal femur into the chosen position in the intercondylar notch, using any
standard drill guide, and the tunnel is made by reaming over the guidewire with the
appropriate-sized reamer.
Whenever possible, we now prefer a double-bundle PCL reconstruction which recent

reports have demonstrated to be biomechanically superior to a single bundle. The two
femoral tunnels attempt to recreate the positions of the posteromedial bundle, which
is tight in extension, and the anterolateral bundle, which tightens in flexion. An
Achilles tendon graft is split to create the two components for the femoral attachment
sites.
The following is a guide for the double-bundle PCL reconstruction technique:
The Achilles allograft bone plug is cut to 12 mm thick. The tendinous portion is
then split into equal 8-mm halves (or into a 6-mm limb and a 10-mm limb to
recreate the anterolateral and posteromedial bundles, respectively). The graft
ends are tubularized and sized to fit into the 8-mm tunnel (Fig. 23-16).
The femoral footprint of the native PCL is identified arthroscopically, and a

resection of the PCL stump is performed. A notchplasty is also performed if
needed. A posteromedial arthroscopy portal is often used for the notchplasty.
A longitudinal incision is then made on the femur midway between the medial
femoral epicondyle and the anteromedial articular margin. Two guide pins are
then drilled from this region into the notch in an outside-in technique (Fig. 23-
17). The two guide pins are placed a minimum of 12 mm apart such that the
two 8-mm tunnels will be separated by at least a 4-mm bone bridge (Fig. 23-
18). The anterolateral pin is placed first. The femoral drill guide targeting tip
is positioned at the planned center of the tunnel in the notch (the â€œwallâ€•-
â€œroofâ€• junction). The anterolateral guide pin is then placed. A pin guide
is then placed over the first pin on the femoral cortex such that the drill hole
for the second pin is 12 mm away. The second pin is then directed proximal
and posterior to the first pin. Following pin placement, the tunnels are reamed
to 8 mm (or to 6 mm and 10 mm). The tunnels are then smoothed with a shaver
to aid the passage of the graft (Fig. 23-19).
Mersilene tape is passed through the two tunnels in an outside-in technique

and placed on the tibial plateau to approximate the required length of the
graft. Bunnell stitches are then placed on the tendinous ends of the graft with
No. 5 Ethibond (Ethicon, Inc., Somerville, NJ).
Immediately after the PCL tunnels are finished, we attend to the bonework
required for ACL reconstruction. If the ACL is avulsed, we perform primary
repair and augmentation with a hamstring tendon. If the ACL has suffered
intraligamentous rupture, and the middle third of the patellar tendon has been
used for the PCL, we reconstruct the ACL with a semitendinosus-gracilis graft
or allograft.
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FIGURE 23-14
Lateral diagrams of proper tibial and femoral tunnel placement for PCL
reconstruction.
FIGURE 23-15
Anteroposterior diagram of proper tibial and femoral tunnel placement for PCL
reconstruction.
FIGURE 23-16
Split Achilles allograft used for a double-bundle PCL reconstruction before

insertion.
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FIGURE 23-17
A diagram of guide pin placement for a double femoral tunnel PCL reconstruction.
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FIGURE 23-18
A diagram of two femoral tunnels.
FIGURE 23-19
An arthroscopic image of a double femoral tunnel before passage of the graft.
The tibial tunnel for the ACL is made in typical fashion, using a standard drill guide
and reamer system. A femoral tunnel for the ACL is also made in standard endoscopic
fashion on the wall of the lateral femoral condyle.
When all required tunnels have been constructed, we prepare to pass the PCL and ACL
grafts. The PCL graft is positioned first, followed by the ACL graft. Likewise, when
both grafts are in place, the PCL graft is tensioned and fixed in place first, followed by
tensioning and fixation of the ACL graft.
To facilitate passage of the PCL graft, we use a 22-gauge wire with a loop to lead the
graft-draw sutures into the tunnels. We start by inserting the wire in an anterior-to-
posterior direction through the tibial bone tunnel for the PCL. The leading end is
grasped and brought into the area of the intercondylar notch, where a second grasper,
reaching into the notch from the femoral tunnel for the PCL, can draw the wire up
through the femoral tunnel. It is important during these maneuvers to control the
trailing end of each wire with a clamp. Next, a second wire is looped through the
looped end of the first wire and the first wire is used to draw the second wire back
down through the knee. The second wire is then retracted to pull the PCL draw
sutures, and the graft itself, into the tibial tunnel and intercondylar notch and out the
femoral tunnel (Figs. 23-20 and 23-21).
To draw the ACL graft into position, we use a beath pin that is driven up through both
the tibial and femoral tunnels and finally through the soft tissues of the distal, lateral
thigh. The sutures that have been passed through the femoral bone plug of the graft
are threaded through the end of the beath needle, and then the needle and sutures
are advanced through the knee under direct visualization. A controlled tension is
maintained on both sets of sutures as the femoral bone plug is guided into the femoral
tunnel to the desired depth.
After both the PCL and ACL grafts have been positioned, the PCL graft is tensioned and
fixed in place with interference screws. The knee must be properly reduced during
fixation of the PCL graft to recreate the normal step-off. Proper tensioning of the PCL
is best achieved with the knee at approximately 70 degrees of flexion. If the fixation
of either interference screw is deemed tenuous, we provide additional fixation by
fastening the draw sutures with either a ligament button or staples. If a double bundle
technique is used for the PCL reconstruction, the anterolateral component is
tensioned and fixed at 90 degrees of flexion, and the posteromedial component is
tightened and fixed with the knee in extension.
FIGURE 23-20
PCL graft being drawn into the tibial tunnel.

FIGURE 23-21
The ACL is controlled with several sutures in preparation for primary repair; the
PCL graft is drawn forward in the notch in preparation for pulling it up into the
femoral tunnel.
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After the PCL graft has been fixed in place, the ACL graft is tensioned and fixed with
interference screws with the knee in full extension and properly reduced. Again, if
either screw appears tenuous, the fixation is reinforced by tying the sutures over an
EndoButton (Smith & Nephew, Andover, MA) on either the femur or the tibia.
Reconstruction of the Medial Collateral Ligament

Reconstruction of the medial or lateral collateral ligaments requires a functional
â€œcentral pivotâ€• for accurate determination of isometricity. For this reason, we
recommend performing the PCL reconstruction first, followed by the ACL
reconstruction, and then the MCL and/or LCL-posterolateral reconstruction. Other
repairs, such as to the iliotibial band or extensor mechanism, are left for last.
The first thing to do in approaching the MCL is to assess the type and amount of
damage to the entire medial side. In cases in which the tibial attachment of the MCL
has been relatively cleanly avulsed, a simple reattachment with a staple or a bone
screw and ligament washer may be all that is necessary to effect a satisfactory repair.
More often, the MCL injury is on the femoral side, leaving a frayed end that is much
more difficult to control and repair. If the posterior MCL and capsular tissues appear
to be intact and of good quality in such cases, we have often been satisfied with the
results of suturing the remnants of the MCL back to bone with suture anchors, and
anteriorly advancing or â€œreefingâ€• the posterior tissues to establish medial
stability. In knee dislocations, however, these tissues are often completely destroyed,
and we find it necessary to augment the medial side with an ipsilateral semitendinosis
tendon graft, as described by Bosworth, or use Achilles tendon allograft. The
hamstring tendon graft is left attached at its tibial insertion and dissected proximally.
The tendon is advanced to the center of rotation of the MCL on the femur, where it is
fixed in place with an AO screw and washer or with a staple. If the tibial attachment
site of the original MCL is still present, the graft should first be secured to the tibia
directly over this site at a point that is approximately at the same level as the tibial
insertion of the semitendinosus tendon. If the tibial attachment of the original MCL is
no longer apparent, a site approximately 3 cm distal to the tibial joint line, at the
level of the insertion of the semitendinosus tendon, will be effective. We prefer to use
a cortical-bone screw with a ligament washer for fixation.
The next task is to determine the proper isometric femoral attachment site for the
graft. We do this by first selecting a likely site on the medial cortex of the medial
femoral condyle, approximately 25 mm proximal to the medial joint line. A guidewire
is inserted into the femur at this site. Next, the graft is drawn taut over this wire at
approximately 30 degrees of knee flexion. The knee is then taken through a range of
motion from 0 to 100 degrees while the graft excursion over the wire is observed. For
this observation, it is helpful to mark the graft where it lies directly over the wire
when the knee is at 30 degrees of flexion. The proposed attachment site is then
adjusted, according to whether the graft lengthens significantly in flexion or
extension. If the graft lengthens in flexion, the attachment site is too far anterior. If
the graft lengthens in extension, the site is too far posterior (Fig. 23-22). The
guidewire is adjusted accordingly until minimal excursion (1 to 2 mm) is obtained. The
graft should be attached to this site with a bone screw and ligament washer while the
medial side of the knee is held closed at 30 degrees of knee flexion. The remaining
remnants of MCL are then reefed over the â€œnewâ€• anterior band of MCL thus
formed, and the posterior medial corner is repaired while keeping the tibia internally
rotated and applying a varus stress.
Lateral Collateral Ligament and Pasterolateral Corner

In approaching the posterolateral corner of the knee, our attention is first directed to
the peroneal nerve. We routinely identify and expose the nerve to protect it from
inadvertent injury during ligament reconstruction (Fig. 23-23). During exposure the
nerve is inspected for signs of injury. If a nerve palsy was noted preoperatively, or an
intraneural hematoma is seen, the epineurium is opened to allow blood to evacuate. It
is particularly important to avoid traction on the peroneal nerve when dissecting the
peroneal nerve from the neck of the fibula.
In cases of knee dislocation, the LCL, popliteus tendon, and popliteofibular ligament
are often avulsed from their bony attachments, and may be reconnected directly to
bone (Fig. 23-24). Recently, our basic science research has enabled us to appreciate a
previously poorly recognized attachment of the popliteofibular ligament to the fibular
head. Our anatomic dissections and biomechanical testing have shown us that the
popliteofibular ligament is an important restraint to external rotation and posterior
translation of the tibia (i.e., the â€œstaticâ€• function of the popliteus tendon).
On the lateral side we restore the function of the popliteus tendon first. We perform a
primary reattachment of the tendon to bone if the tendon is avulsed from its femoral
attachment. More commonly
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we find that the popliteofibular ligament and/or the popliteus muscle-tendon junction
are severely damaged or destroyed, and we therefore tenodese the tendon to the
posterolateral corner of the tibia or, preferably, the posterior part of the fibular head
at the usual insertion of the popliteofibular ligament (Fig. 23-25). This attachment site
is on the gently sloping surface of the posterior fibular head, approximately 8 to 10
mm posterior and 3 to 5 mm medial to the insertion of the LCL and biceps tendon. The
attachment can be reconstructed by detaching part or all of the popliteus tendon from
its muscular junction, controlling it with sutures, and fixing it to bone on the posterior
fibula. The tension of the reconstruction should be set with the knee flexed at
approximately 70 degrees and the lateral tibia drawn forward to its neutral position. It
should be noted that after tightening the reconstruction in this position, it will
become slightly slack when the knee is extended, owing to the cam effect of the
femoral attachment site, since the latter is anterior to the center of rotation on the
femur. The reconstruction will, however, continue to become taut and resist
significant external rotation at
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all angles of knee flexion. If there is attenuation of the popliteus tendon, it may be
repaired and augmented with a thin strip of iliotibial band left attached to Gerdy's
tubercle. This is placed through a drill hole made from the anterior to the posterior of
the tibia and then routed along the popliteus tendon (Fig. 23-26).
FIGURE 23-22
Reattachments of the MCL to the femur must be adjusted to obtain proper

isometricity and function.
FIGURE 23-23
The peroneal nerve is exposed during the posterolateral repair.

FIGURE 23-24
Open view of lateral side of knee showing where the popliteus and LCL have been
avulsed from the bone.
FIGURE 23-25
Popliteus tendon injury at the popliteofibular ligament or popliteus muscle-

tendon junction may be treated with tenodesis of the popliteus tendon to the
posterior lateral corner of the tibia.
If the LCL is avulsed from its femoral insertion, it is reattached to the bone. If the
proper site for reattachment is not obvious, it is determined in a manner that is
completely analogous to the way in which the femoral attachment site is selected for
reconstruction of the MCL. To do this, we use a guide pin at a level approximately 25
mm proximal to the joint line, with the knee at 30 degrees of flexion, to judge
ligament excursion, and adjust the selected site accordingly. Next, we drill a shallow
bone tunnel (about 5 to 10 mm deep) at the intended site of attachment with a one-
quarter-inch (6 to 7 mm) bit, and then drive a Steinmann pin with holes in its trailing
end (beath pin) through the bone tunnel, across the distal femur, and out the medial
femoral cortex. We use the holes in the trailing end of the beath pin to draw the
ligament sutures across the femur and advance the ligament into the bone tunnel. The
sutures are then securely tied down over a ligament button.
In cases in which the LCL is avulsed from its fibular attachment, it may likewise be
recessed into a shallow bone tunnel in the fibular head by using draw sutures taken
through two drill holes brought
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out distally on the fibula. Fixation is achieved by tying the sutures down over a bone
bridge or ligament button (Fig. 23-27).
FIGURE 23-26
Technique for popliteus tendon augmentation using an iliotibial band graft.
If direct repair of the LCL is impossible, the primary graft source for reconstructing the
ligament is the biceps tendon, since the fibular insertion of this tendon actually
envelops the fibular insertion of the LCL. To use the biceps tendon for LCL
reconstruction, we first proceed by leaving the fibular insertion of the tendon
undisturbed. We then elevate from the underlying muscle a segment of biceps tendon
just proximal to the fibular insertion and approximately 20 mm wide and 8 to 10 cm
long. Next, we â€œtubularizeâ€• this sheet of aponeurotic material by rolling one half
to the midline and then rolling the other half around the first half. We secure the
resulting tube with several simple sutures spaced a few millimeters apart along the
length of the graft. The end of the graft is controlled by placing several loop sutures.
Finally, the graft is attached to the femur (Fig. 23-28). It is our standard practice to
perform anterior and lateral compartment releases at the completion of the
reconstruction to decrease the chances of compartment pressure elevation.
Other graft materials that may be used for LCL reconstruction include hamstring
tendon, tubularized iliotibial band, and a patellar allograft or the Achilles tendon. All
of these graft sources are easily adapted to the techniques described previously, with
the appropriate shaping of bone plugs when
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used and the judicious use of draw sutures. When bone plugs are used as part of the
graft construct, interference screws may be used to augment graft fixation.
FIGURE 23-27
Repair of a fibular avulsion of the LCL.

FIGURE 23-28
Technique for LCL reconstruction using a biceps tendon graft.
Surgical dressings are loosely applied to avoid neurovascular compromise, and the leg
may be braced and slightly elevated. Surgical drains should be left in place for as long
as significant drainage (>50 mL/4 hr) persists. The wound should be observed and the
dressing changed at least once daily. We maintain intravenous antibiotics for 48 hours
postoperatively.
Management of the hospitalized patient after surgical reconstruction of a dislocated
knee focuses on the neurovascular status of the extremity and the condition of the
wounds and local soft tissues. Frequent (every 4 hr) assessment of the pedal pulses,
microcirculatory appearance of the foot, and distal sensorimotor function is top
priority during the first several postoperative days. If difficulties arise, neurosurgical
and vascular surgical consultations are promptly obtained. Repeat angiography and
exploration of the popliteal artery should be performed rapidly if questionable
symptoms arise.
After surgical reconstruction of a knee dislocation, we place the knee in a long-leg

hinged brace before leaving the operating room, and we instruct that it is to be worn
at all times. The brace is kept locked in full extension except during ROM exercises.
If possible, we begin continuous passive motion (CPM) immediately after surgical

reconstruction. The arc of motion is started at 0 to 30 or 40 degrees and increased
slowly until a 0- to 70- or 80-degree arc is established and comfortably maintained.
Adjunctive cold therapy applied directly to the knee during passive motion has been
effective in decreasing pain and edema, and allowing more rapid mobilization of the
joint, especially in the early postoperative period.
In the absence of any wound or neurovascular complaints or severe associated

traumatic injuries, most patients are ready to leave the hospital by 5 to 7 days after
surgery. Ambulation is allowed on crutches, with no weight bearing on the affected
side until 3 weeks after surgery. At 3 weeks, progression to partial weight bearing is
allowed with the brace locked in full extension. At 6 weeks, the brace is unlocked to
allow a 0- to 30-degree arc of motion and supervised gait training while the
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crutches are gradually discarded. Over 1 to 2 weeks, a long brace ceases to protect
the posterolateral corner reconstruction.
After 6 weeks, aggressive therapy is begun to restore full ROM of the knee. Active
isolated quadriceps and hamstring exercises (open kinetic-chain exercises) are avoided
since they may create joint-shear forces and displacement. Closed kinetic-chain
exercises are started, including leg presses, half squats, and stair exercises. Stationary
bicycling is also an important part of the therapy program. Over the course of several
months, exercises involving increasing resistance and various proprioceptive and agility
drills are added to the therapeutic regimen.
Bracing and early motion are the hallmarks of our postoperative rehabilitation
protocol. In this regard, we treat patients who have had a multiple ligament
reconstruction more slowly and progressively than those who have had a routine ACL
reconstruction, especially if there is lateral or posterolateral injury.
COMPLICATIONS
By far, the most common complication of dislocation of the knee is variable loss of
knee motion. It is our impression that early surgical intervention and subsequent early
motion therapy will lead to improved motion and stability that are reliably superior to
what can be achieved with a program of cast immobilization as the definitive
treatment for knee dislocations. The exception to this would apply in a straight
anteroposterior dislocation in which there is sufficient stability to allow a nonsurgical
approach and early motion. In older patients, a â€œwait and seeâ€• approach may
also be advisable if there is sufficient stability after the knee is reduced.
Continuous passive motion is instituted early after surgery with the goal of achieving a
0- to 90degree arc of knee flexion by 6 weeks postoperatively. If this goal is not
achieved, we perform manipulation under anesthesia (MUA) to gently lyse adhesions.
In general, our guidelines for MUA are to manipulate the knee at about 8 to 10 weeks
postoperatively if the ROM is less than 70 to 80 degrees, and to manipulate at about 12
weeks if the ROM is less than 110 degrees. After MUA, we reinstitute CPM and resume
the rest of the rehabilitation process.
A reasonable goal for ROM of the knee is an arc of 120 degrees. It is important to
identify patients who might not be progressing toward this goal and to manipulate
them at an early stage after surgery, since tissue maturation makes manipulation
impossible and/or ineffective beyond 4 to 5 months postoperatively.
RESULTS
Knee dislocations often result in severe soft tissue injuries that make return of normal
knee function challenging. Although surgical techniques have improved, a complete
return to normal function remains difficult. In three recent studies (3,8,17) patients
reported their postoperative function as nearly normal 39% of the time, abnormal in
40% of cases, and severely abnormal in 21% of the cases. In general, ROM and
functional outcomes scores are better in patients treated with ligamentous
reconstructions as outlined previously (1,12). However, residual impairment of
function is the rule rather than the exception. The most common complications after
surgical repair include postoperative stiffness, failure of some portion of the
ligamentous reconstruction, and posttraumatic osteoarthritis (12).
CONCLUSION
Knee dislocations are challenging injuries that result in severe soft tissue injury around
the knee joint. Surgical intervention appears to provide the most predictable return to
improved function; however, despite newer surgical techniques, return to normal
function is difficult. Close attention to complete presurgical evaluation is important to
rule out associated neurovascular injury. Detailed surgical planning is critical for
optimal outcomes. With appropriate preoperative planning and meticulous surgical
technique return to near normal function can be anticipated.
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REFERENCES
1. Dedmond BT, Almekinders LC. Operative versus nonoperative treatment of knee
dislocations: a meta-analysis. Am J Knee Surg. 2001;14(1):33-38.
2. Green NE, Allen BL. Vascular injuries associated with dislocation of the knee. J
Bone Joint Surg Am. 1977;59(2):236-239.
3. Harner CD, Waltrip RL, Bennett CH, et al. Surgical management of knee
dislocations. J Bone Joint Surg Am. 2004;86-A(2):262-273.
4. Hollis JD, Daley BJ. 10-year review of knee dislocations: is arteriography always
necessary? J Trauma. 2005;59(3):672-676.
5. Hoover NW. Injuries of the popliteal artery associated with fractures and
dislocations. Surg Clin North Am. 1961;41:1099-1112.
6. Johner R, Ballmer PM, Rogge D, et al. Dislocation of the knee. In: Jacob RP,
Stubli HU, eds. The knee and the cruciate ligaments. Anatomy biomechanics.
Clinical aspects reconstruction complications rehabilitation. Berlin: Springer
Verlag; 1990.
7. Kennedy JC. Complete dislocation of the knee joint. J Bone Joint Surg Am.
1963;45:889-904.
8. Liow RY, McNicholas MJ, Keating JF, et al. Ligament repair and reconstruction
in traumatic dislocation of the knee. J Bone Joint Surg Br. 2003;85(6):845-851.
9. Meyers MH, Moore TM, Harvey JP, Jr. Traumatic dislocation of the knee joint. J
Bone Joint Surg Am. 1975;57(3):430-433.
10. Potter HG. Imaging of the multiple-ligament-injured knee. Clin Sports Med.
2000;19(3):425-441.
11. Potter HG, Weinstein M, Allen AA, et al. Magnetic resonance imaging of the
multiple-ligament injured knee. J Orthop Trauma. 2002;16(5):330-339.
12. Robertson A, Nutton RW, Keating JF. Dislocation of the knee. J Bone Joint Surg
Br. 2006;88(6):706-11.
13. Shields L, Mital M, Cave EF. Complete dislocation of the knee: experience at
the Massachusetts General Hospital. J Trauma. 1969;9(3):192-215.
14. Sisto DJ, Warren RF. Complete knee dislocation. A follow-up study of operative
treatment. Clin Orthop Relat Res. Sep 1985(198):94-101.
15. Thomsen PB, Rud B, Jensen UH. Stability and motion after traumatic
dislocation of the knee. Acta Orthop Scand. 1984;55(3):278-283.
16. Treiman GS, Yellin AE, Weaver FA, et al. Examination of the patient with a
knee dislocation. The case for selective arteriography. Arch Surg.
1992;127(9):1053-1062.
17. Wascher DC, Dvirnak PC, DeCoster TA. Knee dislocation: initial assessment and
implications for treatment. J Orthop Trauma. 1997;11(7):525-529.
> Table of Contents > Part Three - Ligament Injuries and Instability > 24 - High Tibial
Osteotomy in Knees with Associated Chronic Ligament Deficiencies
24
High Tibial Osteotomy in Knees with Associated
Chronic Ligament Deficiencies
Frank R. Noyes
Sue D. Barber-Westin
Proximal tibial osteotomy has gained wide acceptance as a treatment option for patients with
medial tibiofemoral osteoarthrosis and varus deformity of the lower extremity. Generally, high
tibial osteotomy (HTO) provides beneficial results when performed early in the course of the
arthrosis process in younger individuals (15 ,62 ). There are questions, however, about the level
of physical activity that should be pursued after an HTO, as progression of the underlying knee
arthrosis can be expected. The presence of varus angulation alone is not sufficient to justify a
HTO. Rather, the symptoms, medial tibiofemoral arthrosis, and functional limitations are the
primary indicators for the procedure. An added complexity in the varus angulated knee with
medial compartment arthrosis is the presence of anterior cruciate ligament (ACL) deficiency.
Patients who have these combined abnormalities often experience pain, swelling, giving-way,
and functional limitations that sometimes result in a truly disabling condition. There may also be
an associated deficiency of the lateral and posterolateral ligamentous structures that adds to
the varus angulation and clinical symptoms.
The terms primary, double , and triple varus knee simplify the clinician's task in logically
assessing the abnormal alignment and ligamentous deficiencies in a knee with varus alignment
(Fig. 24-1 ). In short, primary varus refers to the overall tibiofemoral varus osseous alignment,
including increased varus alignment due to loss of the medial meniscus and medial tibiofemoral
articular cartilage.
As the tibiofemoral weight-bearing line (WBL) at the knee joint shifts onto the medial
tibiofemoral compartment, a tendency is created for greater loads to be placed on the medial
compartment, and less loads placed on the lateral compartment. The medial shift of the axis
may result in excessive medial compressive loads, which may promote arthrosis in that
compartment (5 ,68 ). Eventually, excessive tensile forces develop in the lateral ligaments,
iliotibial tract, and other lateral soft tissues; and separation
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of the lateral tibiofemoral compartment will occur during standing and walking activities (lateral
condylar lift-off) (30 ,68 ). This condition is termed the double-varus knee because the lower
limb varus alignment results from two factors: the tibiofemoral osseous and geometric alignment
and separation of the lateral tibiofemoral compartment due to lateral ligamentous damage.
FIGURE 24-1
Schematic illustration of primary, double, and triple varus knee angulation (see text). WBL,
weight-bearing line. (Reprinted with permission from Noyes FR, Simon R. The role of high tibial
osteotomy in the anterior cruciate ligament-deficient knee with varus alignment. In: DeLee JC,
Drez D, eds. Orthopaedic Sports Medicine, Principles and Practice , Philadelphia: WB Saunders,
1993.)
There is a hierarchy of soft tissues on the lateral side that resists lateral tibiofemoral joint
separation (9 ,36 ). The biceps femoris, quadriceps, and gastrocnemius muscles act in a dynamic
manner to resist adduction moments on the knee during gait, maintaining lateral tibiofemoral
contact (36 ). If the muscle forces do not provide a functional restraint due to excessive lateral
tensile forces, separation of the lateral tibiofemoral joint occurs. While the fibular collateral
ligament (FCL) will normally allow a few millimeters of lateral joint opening due to physiologic
slackness, studies have found that the lateral ligamentous structures may stretch out in chronic
varus angulated knees (5 ,22 ).
With injury to the posterolateral structures (FCL, popliteus muscle tendon-ligament unit,
posterolateral capsule), a varus recurvatum position of the limb may occur (16 ). The triple
varus knee refers specifically to varus alignment due to three causes: (a) tibiofemoral osseous
alignment, (b) increased lateral tibiofemoral compartment separation due to deficiency of the
FCL and popliteal muscle tendon-ligament complex, and (c) added varus recurvatum in extension
due to the abnormal increase in external tibial rotation and knee hyperextension with
involvement of the entire posterolateral ligament complex. An abnormal increase in
hyperextension may also indicate damage to other ligamentous structures (ACL or posterior
cruciate ligament [PCL], posterior capsule) (10 ). However, in some knees with physiologic
slackness of the cruciate ligaments, there may be a significant varus recurvatum due to damage
to the posterolateral ligamentous structures alone. The
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selection of an appropriate treatment must take into account the associated ligamentous
injuries in addition to the varus malalignment.
Subjective symptoms of pain, swelling, and recurrent loss of stability related to activity are
wellknown effects of chronic ACL deficiency (56 ). What is unique to the varus-angulated ACL-
deficient knee is that there may be two or three different knee subluxations that produce the
symptoms of instability. Symptoms of instability may be due to subluxation of the anterior tibia,
separation of the lateral tibiofemoral joint on walking (varus thrust), posterior subluxation of
the lateral tibial plateau (with knee flexion and external tibial rotation), excessive
hyperextension or varus recurvatum, or a combination of these subluxations. It is incumbent on
the clinician to take a careful history to determine which of the symptomatic subluxations is
present. A varus thrust on walking and a varus tibiofemoral osseous alignment are indications for
a valgus-producing osteotomy. An ACL reconstruction in such knees would not address the
instability and would be expected to fail due to the abnormal lateral joint opening. A varus
recurvatum or â€œbackkneeâ€• instability indicates a triple varus knee requiring reconstruction
of the posterolateral ligamentous structures in addition to the ACL. The complaint of medial
joint line pain may or may not correlate with the degree of medial compartment arthrosis (13
,19 ). In the early stages, the patient usually complains of medial pain with sports activities but
rarely with daily activities. When pain does occur with daily activity, the probability exists for
extensive articular cartilage damage and exposed subchondral bone. Loss of the medial meniscus
is the major risk factor for the progression of arthrosis of the medial compartment.
The timing of the HTO and ligament reconstructive procedures in knees with deficient knee
ligaments is based on several factors (Table 24-1 ) (49 ). In double-varus knees, the gap test is
used at arthroscopy to determine whether it is safe to proceed with ACL reconstruction
concomitantly with the osteotomy, or whether it should be staged to allow adaptive shortening
of posterolateral tissues. In knees that do not demonstrate abnormal lateral joint opening or
external tibial rotation, the HTO and ACL reconstruction may be performed at the same setting.
The tibial fixation of the ACL graft is performed by placing interference screws proximal to the
osteotomy site and by adding sutures that are tied to a suture post for additional fixation. An
ACL reconstruction should not be performed if there is excessive abnormal lateral tibiofemoral
compartment opening because this would place the graft under undue forces. In triple-varus
knees, we stage the ligament reconstructive procedures after the osteotomy. Performing all
procedures (HTO, ACL reconstruction, and posterolateral ligament reconstruction)
simultaneously results in a lengthy operation with increased risk of complications, prolonged
rehabilitation, and knee motion problems (4 ,26 ,74 ). We perform the HTO and then, after
adequate healing of the osteotomy, an arthroscopically assisted ACL reconstruction and open
posterolateral ligament reconstruction. The ACL and posterolateral ligament reconstructive
procedures must be performed together to allow both structures to function together to resist
abnormal lateral tibiofemoral joint opening and varus recurvatum.
When a primary ACL reconstruction is performed with the HTO, our first graft choice remains a
bone-patellar tendon-bone. In knees undergoing ACL revision reconstruction and osteotomy, we
prefer to use a contralateral bone-patellar tendon-bone graft (44 ), followed by an ipsilateral
quadriceps tendon-bone graft (43 ) (Fig. 24-2 ), or a four-strand semitendinosus-gracilis tendon
autograft. We avoid allografts for ACL reconstructions in revision knees whenever possible due to
the higher failure rates of these grafts compared to autografts (40 ,41 ,50 ). If an allograft is
required and the knee demonstrates marked anterior displacement indicating involvement of the
secondary ligament restraints, consideration may be given to also perform an iliotibial band
extra-articular Losee-type procedure (41 ).
The recommendation for HTO is derived from careful consideration of subjective symptoms,
findings on physical examination, radiographic findings of alignment and arthrosis, and gait
analysis data when available. In our clinical setting the predominant indication for HTO is to
obtain limb alignment in younger patients experiencing medial tibiofemoral joint pain and who
wish to continue an active lifestyle. Unfortunately, a previous medial meniscectomy in a varus
aligned knee is a major factor in the progression of the deterioration. Every attempt should be
made to preserve and repair both simple and complex meniscal tears. We have described
elsewhere techniques that allow for repair of meniscus tears that extend into the avascular
region (34 ,67 ). After HTO, there is the necessity for modification of athletic activities and
common sense guidelines as to what activities will be pursued. The goal of HTO is to allow a
more active lifestyle, with recreational pursuits that impose a lower demand on the knee,
avoiding strenuous and high-impact sports activities.
The second major indication for HTO is in a varus-aligned knee with combined cruciate injury
and posterolateral ligament injury. It is necessary to achieve a normal axial alignment prior to
the ligament
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reconstructive procedures. If there is no medial arthrosis, it is not necessary to produce a valgus
overcorrection. Rather, a normal 50% weight-bearing line is achieved by corrective osteotomy.
Anterior cruciate ligament
Autograft options: Central third bone-patellar tendon-bone
At or preferably after HTO
Any patient who had instability before HTO should not risk a further trial of function and
possible reinjury. Considerwhen secondary ligament restraints are lost (pivot shift 3 +
impingement, >10 mm increased anterior displacement involved knee) and associated medial or
lateral ligament deficiency is present. Consider when meniscus repair is performed. Athletically
active patient desiring best knee possible for return to sports activities.
Quadriceps tendon-patellar bone
Semitendino susgracilis
Lateral collateral ligament (posterolateral complex intact, no increased external rotation or

varus recurvatum)
Usually not required

Expect adaptive shortening LCL in the majority of patients after valgus-producing osteotomy. At
HTO, avoid disrupting proximal tibiofibular joint, which would allow proximal migration and
laxity of posterolateral ligament structures.
Posterolateral complex, including lateral collateral ligament
Posterolateral complex proximal advancement LCL, posterolateral complex reconstruction with

autograft or allograft (see text)*
Staged procedure:
HTO first
ACL,
lateral/posterolateral reconstruction
Patients usually have increased lateral joint opening of 8 mm at the intercondylar notch (12 mm
or more at periphery), increased external rotation of 10 to 15 degrees, and require
posterolateral reconstruction at time of ACL reconstruction. Combined posterolateral
reconstruction and ACL reconstruction always performed together to limit hyperextension and
varus rotations.
*A bicepstendon transposition is contraindicated in these knees. The biceps muscle function is
required to maintain compressive forces across the lateral tibiofemoral compartment and to
allow active external tibial rotation. Both of these functions may be lost if the biceps tendon is
transposed and fixated to the lateral femoral condyle.
Reprinted with permission from Noyes FR, Barber-Westin SD, Hewett TE. High tibial osteotomy
and ligament reconstruction for varus angulated anterior cruciate ligament-deficient knees. Am
J Sports Med. 2000;28:282-296.
Ligament Surgical Timing Related to Indications/Comments

Deficiency Procedure HTO
TABLE 24-1. Indications for and Timing of Ligament

Reconstructive Procedures
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FIGURE 24-2
Algorithm for the treatment of ACL-deficient knees with varus or valgus malalignment requiring
surgery.
Relative contraindications for HTO include:
Medial tibiofemoral compartment bone exposure over an area >15 Ã— 15 mm on both

surfaces
Prior lateral meniscectomy, or lateral tibiofemoral cartilage damage
Extension deficit >10 degrees
Significant lateral tibial subluxation
Moderate to marked symptomatic patellofemoral arthrosis

Depression and concavity of the medial tibiofemoral joint, due to advanced arthrosis
which prevents loading the lateral compartment (teeter-totter knee)
Poor motivation or unrealistic expectations of HTO by the patient. In this situation, the
patient may do better with a joint replacement when the arthrosis advances, rather than
with an HTO to buy time until a future total knee arthroplasty.
The methodology for our preoperative evaluation and surgical planning is shown in Figure 24-3 .
The comprehensive physical examination includes particular attention to:
Patellofemoral abnormalities, including extensor mechanism malalignment and arthrosis
Detection of tibiofemoral crepitus on varus and valgus loading as an indicator of early

articular cartilage damage prior to radiographic changes
Palpation to detect inflammation, or neuroma of the soft tissues
Detection of gait hyperextension abnormalities that require gait retraining

preoperatively (46 ,57 )
Examination for abnormal motion limits and joint subluxations of the affected knee
compared to the opposite knee
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FIGURE 24-3
Algorithm for preoperative planning in knees with angular malalignment and associated ligament
injury. Tibial width 62% for valgus overcorrection if medial arthrosis, and 50% for neutral
alignment. WBL, weight-bearing line. (Reprinted with permission from Noyes FR, Goebel SX,
West J. Opening wedge tibial osteotomy. The 3-triangle method to correct axial alignment and
tibial slope. Am J Sports Med. 2005;33:378-387.)
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The clinician must determine three factors (54 ):
The abnormal motion limits (e.g., increases in tibial rotation and knee extension)
The subluxations that occur in the medial and lateral tibiofemoral joint in the physical
examination, and the knee positions in which they occur
The anatomic ligament deficiencies that explain the abnormal motion limits and
subluxations
The clinical test for FCL insufficiency is based on the varus stress test performed at 0 and 30
degrees of knee flexion. The examiner must estimate the amount (in millimeters) of joint
opening from the initial closed contact position of each tibiofemoral compartment to the
maximal opened position. The tibiofemoral rotation test, which we previously described (Fig. 24-
4A,B ), is performed at 30 and 90 degrees of knee flexion to diagnose posterior tibial
subluxations in a qualitative manner after ligament injury (52 ). It is important at arthroscopy to
perform a lateral and medial joint opening gap test to measure the amount of joint opening with
a calibrated nerve hook (Fig. 24-5A,B ). Knees that have insufficient posterolateral structures
will demonstrate 8 mm of joint opening at the intercondylar notch, and 12 mm or more of joint
opening at the periphery of the lateral tibiofemoral compartment.
Radiographic assessment of lower-limb alignment is based on an examination of radiographs of

the full length of the extremity with the patient in the standing position. Separation of the
lateral tibiofemoral compartment may occur, preventing a correct assessment of true
tibiofemoral osseous alignment. We recommend double-stance, full-length anteroposterior
radiographs showing both lower extremities from the femoral heads to the ankle joints (6 ). The
knee is flexed 3 to 5 degrees to avoid a hyperextension position. If separation of the lateral
tibiofemoral joint is observed, it is necessary to make appropriate calculations to subtract the
lateral compartment opening so that the true tibiofemoral osseous alignment is determined (6 ).
We also look for the â€œteeter-totterâ€• effect in which simultaneous contact of the medial
and lateral tibiofemoral compartments is not possible due to advanced changes of tibial
obliquity and bone loss in the medial tibiofemoral joint. This finding indicates that overall limb
alignment will probably remain in a varus position after HTO, since lateral tibiofemoral contact
cannot be re-established. The required radiographic measurements include the tibiofemoral
mechanical axis and tibiofemoral WBL (Fig. 24-6 ). It is possible for a patient to have a varus
thrust on walking due to complete disruption of the posterolateral ligaments and still have a
valgus tibiofemoral osseous alignment.
The results of our study (6 ) indicate that determination of the WBL at the knee joint represents
the most precise method of preoperative planning. A WBL crossing the knee lateral to the 75%
coordinate has the potential for a â€œlift-offâ€• of the medial femoral condyle (23 ).
Unicondylar weight-bearing resulting from distraction of the medial compartment is undesirable
and could result in rapid lateral compartment deterioration, gradual medial collateral ligament
(MCL) failure, and a possible progressive valgus deformity (21 ). The weight-bearing line-tibial
intersection depends on two separate variables, the final mechanical axis angle and the femoral
and tibial lengths of the patient.
We use two methods to determine the correction wedge on preoperative radiographs. First, the
centers of the femoral head and tibiotalar joint are marked on the full-length radiograph (Fig.
24-7 ). The selected WBL coordinate of the tibial plateau is identified and marked. This is
usually placed at 62% of the tibial width, which allows the WBL to pass through the lateral
tibiofemoral compartment, providing a 3-degree angular overcorrection. The 62% WBL
coordinate is used if there is medial tibiofemoral articular cartilage damage and the desire
exists to transfer loads to the lateral compartment. A 50% WBL coordinate is chosen if there is
no medial tibiofemoral joint damage and the surgeon wishes to correct to a normal axial
alignment. One line is drawn from the center of the femoral head to the selected percent WBL
intersection at the tibia, and a second line is drawn from the center of the tibiotalar joint to
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the same tibial coordinate position. The angle formed by the two lines intersecting at the tibia
represents the angular correction required to realign the WBL through this coordinate.
FIGURE 24-4
The tibiofemoral rotation test, shown at 90 degrees of knee flexion, during (A) neutral rotation
and (B) external tibial rotation. The position of the medial and lateral tibial plateaus is palpated
and compared with the normal knee to assess whether a subluxation (anterior or posterior) of
the medial or lateral tibial plateau is present. The test is performed at 30 and 90 degrees of
knee flexion. The position of the medial and lateral tibial plateau is assessed at the starting
position (neutral tibial rotation) and at the final test position (external or internal tibial
rotation). The axis of tibial rotation is also observed in the involved knee and compared with the
normal knee to detect a shift to the medial or lateral tibiofemoral compartment during tibial
rotation. (Modified with permission from Noyes FR, Simon R. The role of high tibial osteotomy in
the anterior cruciate ligament-deficient knee with varus alignment. In: DeLee JC, Drez D, eds.
Orthopaedic sports medicine, principles and practice. Philadelphia: WB Saunders; 1993.)
FIGURE 24-5
Arthroscopic gap test for determining the amount of lateral joint opening. A calibrated nerve
hook is used to measure the millimeters of joint space. (Reprinted with permission from Noyes
FR, Barber-Westin SD, Hewett TE. High tibial osteotomy and ligament reconstruction for varus
angulated anterior cruciate ligament deficient knees. Am J Sports Med. 2000;28:282-296.)
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FIGURE 24-6
The weight-bearing line positions are shown for different degrees of the mechanical axis. In this
example, a correction beyond 186 degrees results in a weight-bearing line lateral to the desired
target area. TW, tibial width. (Reprinted with permission from Dugdale TW, Noyes FR, Styer D.
Preoperative planning for high tibial osteotomy: effect of lateral tibiofemoral separation and
tibiofemoral length. Clin Orthop. 1991;271:105-121.)
FIGURE 24-7
Graphic depiction of the method used to calculate the correction angle of an HTO using a full-
length, non-weight-bearing anteroposterior roentgenograph of the lower extremity. (Reprinted
with permission from Dugdale TW, Noyes FR, Styer D. Preoperative planning for high tibial
osteotomy: effect of lateral tibiofemoral separation and tibiofemoral length. Clin Orthop.
1991;271:105-121.)
The second method of determining the correction wedge involves cutting the full-standing
radiograph horizontally through the line of the superior osteotomy cut (Fig. 24-8 ). A vertical cut
of the lower tibial segment is then made to converge with the first cut at the level of the medial
cortex. The distal portion of the radiograph is aligned until the center of the femoral head, the
selected weight-bearing line coordinate point on the tibial plateau, and the center of the
tibiotalar joint are all colinear. With the radiograph taped in this position, the angle of the
wedge formed by the overlap of the two radiographic segments is measured and compared with
the value obtained using the first method. The mechanical axis is measured to determine the
angular correction. If there is a discrepancy between the two correction wedge angles, the
procedures should be repeated.
Lateral radiographs are examined for abnormal tibial slope, such as an excessive posterior
sloping of the tibial surface greater than 8 degrees. Abnormal posterior sloping of the tibia in
the sagittal plane is usually seen after tibial fractures or growth abnormalities, and may increase
forces on an ACL reconstruction, resulting in failure. It is important not to induce during the
tibial osteotomy an abnormal tibial slope.
A high adduction moment may be anticipated in varus angulated knees; however, it is also
known that the moments and loads on the knee joint cannot be reliably predicted from the
static alignment of the lower limb measured on radiographs. We reported on the results of gait
analyses in 32 patients
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with ACL deficiency and varus angulation (57 ). The majority of patients (20 of 32) had an
abnormally high adduction moment. The adduction moment showed a statistically significant (p
<.05) correlation to predicted high medial tibiofemoral compartment loads and high lateral
ligament tensile forces (p <.01). The data indicated the likelihood, in knees with high lateral
ligament tensile forces, for separation of the lateral tibiofemoral joint to occur with
â€œcondylar lift-offâ€• during periods of the stance phase. We also found that approximately
one third of the patients had normal or low adduction moments and corresponding normal to low
medial tibiofemoral compartment loads. These patients had gait characteristics or adaptations
that tended to lower medial tibiofemoral loads despite the varus angulation of the knee joint.
Gait analysis allowed identification of patients with very high medial joint loads suggesting a
poorer prognosis for progression of the arthrosis. Alternatively, patients with a low adduction
moment have a potentially better overall prognosis, and an HTO would result in a substantial
lowering of these loads.
FIGURE 24-8
Graphic depiction of alternate method used to calculate the correction angle of a HTO using a
full-length, non-weight-bearing radiograph of the lower extremity. (Reprinted with permission
from Dugdale TW, Noyes FR, Styer D. Preoperative planning for high tibial osteotomy: effect of
lateral tibiofemoral separation and tibiofemoral length. Clin Orthop. 1991;271:105-121.)
Opening Wedge HTO: Methodology for Correction of Axial

Alignment and Tibial Slope
On cross-section, the proximal anteromedial tibial cortex has an oblique or triangular shape,
whereas the lateral tibial cortex is nearly perpendicular to the posterior margin of the tibia.
Because of this relationship, a medial opening wedge osteotomy that has an anterior tibial
tubercle gap (width) equal to the gap (width) at the posteromedial margin would increase tibial
slope, decrease knee extension, and potentially increase ACL tensile loads (13 ). A lateral closing
wedge osteotomy that has an equal anterior-to-posterior gap along the lateral tibial cortex
would have a small effect on tibial slope. Although authors have recognized that a change in
tibial slope may occur when performing a medial opening wedge osteotomy, operative
techniques or calculations have not been precisely defined to address this problem (8 ,14 ,24 ).
We conducted a study to mathematically calculate through three-dimensional analysis of the

proximal tibia (using fine cut axial CT imaging) how the angle of the opening wedge along the
anteromedial tibial cortex influences the tibial slope (sagittal plane) and valgus correction
(coronal plane) when performing a medial opening wedge osteotomy (53 ). The obliquity of the
anteromedial cortex of the proximal tibia relative to the posterior tibial cortex (Fig. 24-9 ) was
measured on 35 magnetic resonance imaging (MRI) films in healthy young individuals (mean age,
32.7). Serial CT images of a cadaveric tibia were made in 1.25 mm slices and digitized using a
computer-aided design
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package to create a solid model of the proximal tibia. A medial opening wedge osteotomy was
created along the anterior-posterior axis to include a hinge axis on the lateral margin of the
tibia. The open wedge osteotomy was just proximal to the tibial tubercle, ending 20 mm below
the cortical margin of the lateral tibial plateau.
FIGURE 24-9
An axial cut of the proximal tibia at the diaphyseal-metaphyseal junction. The angle formed by
the intersection of the medial-lateral axis along the posterior tibia and the axis along the
anteromedial tibial cortex is called the anteromedial cortex oblique angle. This angle measures
approximately 45 degrees to the coronal plane. (Reprinted with permission from Noyes FR,
Goebel SX, West J. Opening wedge tibial osteotomy. The 3-triangle method to correct axial
alignment and tibial slope. Am J Sports Med. 2005;33:378-387.)
The distal portion of the tibia was rotated on the virtual model about the hinge axis (Fig. 24-10 )
through the lateral point of the osteotomy maintaining the anatomic tibial slope (sagittal plane).
Measurements of the wedge angle and gap angle along the anteromedial tibial cortex were made
from the resulting computerized model. Standard algebraic calculations were made using the
law of triangles to determine the effect of different degrees of opening wedge osteotomy on
coronal (valgus) and sagittal (tibial slope) alignment.
The MRI measurement of the anteromedial tibial cortex oblique angle at the site of the opening
wedge osteotomy was 4 Â± 6 degrees (range, 34 to 56 degrees) in the 35 patients.
The opening wedge angle, along the anteromedial tibial cortex to maintain the tibial slope, was
found to be dependent on the angle of coronal valgus correction (HTO coronal angle) and the
angle of obliquity of the anteromedial tibial cortex. In Figure 24-11 , the results are shown for
the calculation of the opening wedge angle (along the anteromedial tibial cortex) for five
different osteotomy corrections in the coronal valgus plane (2.5 to 12.5 degrees). As an
example, a 10-degree coronal valgus correction (assuming a 45-degree obliquity of the
anteromedial tibial cortex with respect to the hinge axis) would result in a 7-degree opening
wedge angle along the anteromedial tibial cortex. A larger wedge angle would decrease the
tibial slope, and a smaller wedge angle would increase the tibial slope.
The gap angle is perpendicular to the anteromedial oblique surface of the tibia with a vertex on
the hinge axis posterior to the tibia. This is shown in Figure 24-12 , where the gap angle in
degrees is shown for five different osteotomy corrections as a function of the obliquity of the
anteromedial tibial cortex. As an example, a 10-degree coronal valgus correction (assuming a
normal 45-degree obliquity of the anteromedial tibial cortex) would result in a 7-degree gap
angle at the osteotomy site.
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FIGURE 24-10
The distal portion of the tibia was rotated about an AP axis, which ran through the hinge point
of the osteotomy to maintain the posterior tibial slope. A solid model of a high tibial osteotomy
plate was placed at a series of positions around the medial aspect of the tibia, just anterior to
the medial collateral ligament. (Reprinted with permission from Noyes FR, Goebel SX, West J.
Opening wedge tibial osteotomy. The 3-triangle method to correct axial alignment and tibial
slope. Am J Sports Med. 2005;33:378-387.)
FIGURE 24-11
The anteromedial cortex opening wedge angle depends on the oblique angle of the tibial cortex
with respect to the hinge axis. Each line represents the desired calculated degrees of correction
for the opening wedge osteotomy in the true coronal (90-degree) plane. (Reprinted with
permission from Noyes FR, Goebel SX, West J. Opening wedge tibial osteotomy. The 3-triangle
method to correct axial alignment and tibial slope. Am J Sports Med. 2005;33:378-387.)
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FIGURE 24-12
The magnitude of the gap angle changes with the obliquity of the anteromedial tibial cortex
angle. Each line represents the calculated degrees of correction for the opening wedge
osteotomy in the coronal plane. (Reprinted with permission from Noyes FR, Goebel SX, West J.
Opening wedge tibial osteotomy. The 3-triangle method to correct axial alignment and tibial
An error in the anteromedial tibial cortex opening wedge angle would result in an error in the
tibial slope (Table 24-2 ). For example, an error of 5 mm in the Y2 gap (Fig. 24-13 ) at the tibial
tubercle, assuming the length of the anteromedial cortex (L) is 40 mm, would result in an
unexpected change in the tibial slope of 10 degrees. As a general rule in this example, every
millimeter of alteration in the gap height induces a change of 2 degrees in the tibial slope.
The opening wedge angle can be set at surgery by measuring and altering the vertical gap at two
points along the osteotomy site, Y1 and Y2 (see Fig. 24-13 ). This has importance in determining
that the correct wedge angle is obtained prior to internal fixation at the osteotomy site. The
site at which the vertical gap measurement is taken depends on the coronal distance from the
hinge axis, obliquity of the anteromedial tibial cortex, and the distance along the osteotomy site
on the anteromedial surface (Tables 24-3 and 24-4 ). In Table 24-3 , the millimeters of opening
at the osteotomy site are based
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on the width of the tibia and the angle of correction. In Table 24-4 , an average 45-degree
oblique angle of the anteromedial cortex and a tibial width of 60 mm are assumed. This allows
the surgeon to calculate at the time of surgery making simple measurements, the desired gap
height at two points along the osteotomy to maintain the tibial slope. For example, the
measurement of the posterior tibial width is made at the osteotomy site (X1 ). The opening
height at the most posteromedial point (Y1 ) and the distance between the two measurement
points (Y1 to Y2 ) is used in Table 24-4 to determine the second opening height at a defined
distance (L) along the osteotomy site to maintain tibial slope.
4.0
3.2
2.7
2.3
2.0
1.8
1.6
1.5
8.1
6.5
5.4
4.6
4.0
3.6
3.2
2.9
12.0
9.6
8.1
6.9
6.1
5.4
4.9
4.4
15.8
12.8
10.7
9.2
8.1
7.2
6.5
5.9
5
19.5
15.8
13.3
11.4
10.0
8.9
8.1
7.3
23.0
18.8
15.8
13.6
12.0
10.7
9.6
8.8
26.3
21.6
18.3
15.8
13.9
12.4
11.2
10.2
29.5
24.4
20.7
17.9
15.8
14.4
12.8
11.6
32.5
27.0
23.0
20.0
17.7
15.8
14.3
13.0
10
35.3
29.5
25.2
22.0
19.5
17.4
15.8
14.4
* Results are given in degrees.
Reprinted with permission from Noyes FR, Goebel SX, West J. Opening wedge tibial osteotomy.
The 3-triangle method to correct axial alignment and tibial slope. Am J Sports Med.
2005;33:378-387.
Length of Anteromedial Cortex, mm
Error at Y2 (mm) 20 25 30 35 40 45 50 55
TABLE 24-2. Errors in the Tibial Slope Caused by Errors in

Vertical Gap Height (Y2 )*
FIGURE 24-13
The opening wedge angle along the anteromedial tibial cortex can be calculated using the three
linear measurements along the osteotomy opening wedge. Y2 , posterior gap; Y1 , gap anterior
to Y2 ; L, length between Y1 and Y2 . (Reprinted with permission from Noyes FR, Goebel SX, West
J. Opening wedge tibial osteotomy. The 3-triangle method to correct axial alignment and tibial
If a buttress wedge plate is used, the appropriate size plate may be selected for the opening
wedge depending on the site where the plate is placed. An example is given if the surgeon
selects to place the buttress plate just anterior to the superficial MCL on the anteromedial tibial
cortex approximately 20 mm anterior to the most posteromedial point of the osteotomy. In
Table 24-4 , with a 10-mm
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posteromedial opening (Y1 ), the correct width of the buttress plate would be 7.6 mm (tibial
width 60 mm). For a 12-mm posteromedial opening, the correct width of the buttress plate
would be 9.2 mm. A wider buttress plate gap would result in excessive valgus alignment and
altered tibial slope. The overall method to follow to correct lower limb alignment is summarized
in Table 24-5 .
50
4.37
5.25
6.15
7.00
8.00
8.80
9.70
10.85
11.55
55
4.81
5.78
6.77
7.70
8.80
9.68
10.67
11.94
12.71
60
5.25
6.30
7.38
8.40
9.60
10.56
11.64
13.02
13.86
65
5.69
6.83
8.00
9.10
10.40
11.44
12.61
14.11
15.02
70
6.12
7.35
8.61
9.80
11.20
12.32
13.58
15.19
16.17
75
6.56
7.88
9.23
10.50
12.00
13.20
14.55
16.28
17.33
80
7.00
8.40
9.84
11.20
12.80
14.08
15.52
17.36
18.48
85
7.44
8.93
10.46
11.90
13.60
14.96
16.49
18.45
19.64
90
7.87
9.45
11.07
12.60
14.40
15.84
17.46
19.53
20.79
95
8.31
9.98
11.69
13.30
15.20
16.72
18.43
20.62
21.95
100
8.75
10.50
12.30
14.00
16.00
17.60
19.40
21.70
23.10
* TW, coronal tibial width at osteotomy site.
2005;33:378-387.
Degrees of Angular Correction
TW* 5 6 7 8 9 10 11 12 13
TABLE 24-3. Millimeters of Opening at the Osteotomy Site

Based on the Width of the Tibia and the Angle of Correction
8.0
8.0
8.0
8.0
8.0
20
5.7
5.9
6.1
6.3
6.4
25
5.2
5.4
5.6
5.8
6.0
30
4.6
4.9
5.2
5.4
5.6
35
4.0
4.4
4.7
5.0
5.2
40
3.5
3.9
4.2
4.5
4.8
45
2.9
3.4
3.8
4.1
4.4
10
0
10.0
10.0
10.0
10.0
10.0
20
7.2
7.4
7.6
7.8
8.0
25
6.5
6.8
7.1
7.3
7.5
30
5.8
6.1
6.5
6.7
7.0
35
5.1
5.5
5.9
6.2
6.5
40
4.3
4.9
5.3
5.6
6.0
45
3.6
4.2
4.7
5.1
5.5
12
12.0
12.0
12.0
12.0
12.0
20
8.6
8.9
9.2
9.4
9.6
25
7.8
8.1
8.5
8.7
9.0
30
6.9
7.4
7.8
8.1
8.4
35
6.1
6.6
7.1
7.4
7.8
40
5.2
5.8
6.3
6.8
7.2
45
4.4
5.1
5.6
6.1
6.5
*By measuring the width of the tibia, the opening wedge height at the most medial point (Y1 ),
and the distance between vertical measurement points (L), the vertical height at the second
measurement point (Y2 ) can be found on the table. Calculations based on 45-degree angle of
the anteromedial tibial cortex at osteotomy site.
2005;33:378-387.
Tibial Width at Osteotomy (X1 ), mm
Opening at Osteotomy (Y1 ), mm L, mm 50 55 60 65 70
TABLE 24-4. Opening Wedge Height Measurements*

The change in the oblique angle of the anteromedial cortex, within the standard deviations
reported, would have only a small and negligible effect on the width measurements of the
opening wedge (see Table 24-4 ). For example, assuming a 10-mm width to achieve the desired
valgus correction, the computed width for the buttress plate (20 mm anteriorly) and width at
the tibial tubercle (45 mm anteriorly) would be 7.6 mm and 4.7 mm, respectively. For a 40-
degree oblique angle, the width would be 7.4 mm and 4.3 mm, respectively. For a 50-degree
oblique angle, the width would be 7.9 mm and 5.2 mm, respectively, at the two sites.
In general, the rules to maintain tibial slope are:
The most anterior gap of the osteotomy wedge at the tibial tubercle should be one half
of the posteromedial gap to maintain the tibial slope.
Every millimeter of gap error at the tibial tubercle results in approximately 2 degrees of
change in the tibial slope.
Preoperative radiographic measurement of the tibial slope is recommended.
The buttress plate height at Y2 (placed anterior to the MCL; see Fig. 24-13 ) will be 2 to 3
mm less than the posteromedial height at Y1 (gap) to maintain tibial slope during the
coronal valgus correction.
Since small millimeters of change at the osteotomy site affect slope and coronal
angulations, radiographic confirmation of final alignment at surgery and postoperatively
is required.
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1.
Determine the coronal valgus angular correction in degrees based on preoperative full-standing
radiographs for the desired placement of the weight-bearing line at the tibial plateau and
measure the tibial slope based on a lateral radiograph.
2.
Determine the millimeters of opening at the posteromedial osteotomy site at surgery (Y1 gap)
using the width of the tibia (X1 distance) for the coronal valgus correction based on the law of
triangles (first triangle; Table 24-3 ).
3.
Determine the proper gap width of the osteotomy opening wedge along the anteromedial cortex
to maintain tibial slope and the proper width of the tibial buttress plate based on its location
along the anteromedial cortex (Table 24-4 ). If the tibial slope is to be increased or decreased,
the effect on the degrees of tibial slope is shown in Table 24-2 .
4.
Confirm final angular correction based on intraoperative radiographs and later, postoperative
full-standing and lateral radiographs.
2005;33:378-387.
TABLE 24-5. Proposed Method to Follow for Opening Wedge
Osteotomy to Correct Coronal and Sagittal Malalignment
SURGICAL TECHNIQUE
Selection of Opening Wedge or Closing Wedge Osteotomy
Currently, the two most frequently used techniques for correcting varus deformity are the
opening and closing tibial wedge osteotomies. We recommend opening wedge osteotomy under
the following circumstances:
When a small angular correction is indicated, the lateral dissection and fibular osteotomy
are avoided
When a large angular correction greater than 12 to 15 degrees is required to avoid

excessive tibial shortening
When distal advancement or reconstruction of the MCL is also required
When an extensive FCL and posterolateral reconstruction is required, avoiding proximal

fibular osteotomy since ligament grafts are anchored to the proximal fibula
When patella alta or leg length shortening is positively affected by the millimeters of
opening wedge osteotomy
There are disadvantages of the opening wedge osteotomy in that appropriate autograft or
allograft bone is required at the opening wedge site to restore the anteromedial and
posteromedial cortex to add fixation strength and promote osseous union. We prefer autogenous
bone grafting of the open defect, which aids in achieving stability at the osteotomy site. There
are no published clinical trials we are aware of on the outcome of opening wedge osteotomy
where bone allografts were used to fill the osteotomy site. Numerous authors have reported on
the outcome of opening wedge osteotomy where autogenous bone was used to fill the gap site
(31 ,63 ,65 ,79 ). We believe the potential problem of early varus collapse of an opening wedge
osteotomy due to delayed union may be lessened by autogenous iliac crest bone grafting. In
addition, a T- or L-shaped tibial buttress plate with locking screws may be required to achieve
more rigid fixation (71 ).
There is a greater chance of increasing the posterior tibial slope if the opening wedge buttress
plate is placed in a more anterior position. However, with attention to this problem at surgery,
the normal tibial slope in the sagittal plane can be preserved. Another disadvantage of an
opening wedge osteotomy is that transection of the superficial MCL's distal attachment is usually
required. In an opening wedge osteotomy of 5 to 7.5 mm, the MCL fibers may be incompletely
transected at several different levels (pie-crust approach) to maintain the distal attachment,
effectively lengthening the ligament.
The closing wedge osteotomy has the advantage of faster healing and earlier resumption of
weight bearing, since contact of two large cancellous surfaces of the proximal tibia is achieved.
The initial internal fixation of the osteotomy is more secure than an opening wedge procedure,
and there is less chance for a change in osteotomy position and loss of correction. The closing
wedge osteotomy does involve more dissection and a meticulous approach and osteotomy of the
proximal fibular neck region avoiding the peroneal nerve. It is also somewhat more tedious to
resect more bone and alter the lower limb correction at surgery, should it be necessary.
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In the preoperative planning, the surgeon should measure the height of the patella on lateral
radiographs to determine if an abnormal patella infera or alta position exists which may
contraindicate an opening or closing wedge osteotomy, respectively, as these procedures would
further decrease or elevate the patella position. The use of an external fixator has been
advocated with tibial osteotomy (80 ); it has the advantage of allowing minor adjustments to be
made in lower limb alignment, which is postoperatively helpful in difficult or biplanar
corrections. A disadvantage of an external fixator is the risk of pin tract infections and the 10-
to 12-week period of time that the fixator is used.
Authors' Preferred Technique for Closing Wedge Tibial

Osteotomy
In preparing for the HTO, the entire lower extremity is prepped and draped free. Arthroscopy is
performed at the time of the HTO to evaluate articular surfaces of the medial and lateral
tibiofemoral compartments and patellofemoral joint. Anterior cruciate ligament-deficient knees
frequently have associated meniscus tears that require repair (67 ) or partial removal.
Appropriate debridement of tissues, inflamed synovium, and notch osteophytes limiting knee
extension is performed. A sterile tourniquet is inflated before the skin incision. An oblique
incision is made from the head of the fibula to the anterior crest of the tibia, 2 cm distal to the
tibial tubercle (Fig. 24-14A ). The subcutaneous tissues are incised down to the fascia of the
anterolateral tibial musculature. A fascial incision is made from the lateral aspect of the tibial
tubercle sloping up proximally to the distal aspect of Gerdy's tubercle, and is then extended
posteriorly and laterally to the anterior bare area of the fibula (Fig. 24-14B ). The anterior bare
area of the fibula is an important and safe anatomic landmark. The FCL and peroneal nerve are
safely avoided when one identifies this bare area to initiate the limited subperiosteal exposure
of the fibular neck for the fibular osteotomy.
Subperiosteal dissection on the tibia is initiated just distal to Gerdy's tubercle, using a scalpel
followed by a Cobb elevator. The dissection is continued just lateral to the patellar tendon, and
should
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be done cleanly, with a relatively bloodless field and without damage to muscle tissue. The
retinaculum adjacent to the patellar tendon is incised and the retropatellar space entered. The
patellar tendon is retracted anteriorly. The dissection is continued posteriorly on the tibia. It is
important to remain in a subperiosteal plane because of the proximity of neurovascular
structures. Posteriorly, the dissection is completed across the width of the tibia in one location,
and is then carefully extended proximally and distally in this safe subperiosteal plane.
FIGURE 24-14
A: Oblique skin incision. B: Dome-shaped fascial incision.

FIGURE 24-14
(Continued) C: The proximal fibula is subperiosteally dissected and the peroneal nerve is
identified and protected in preparation for the proximal fibular osteotomy. D: The proximal
guide pin is placed parallel to the joint and 2.5 cm from it. The width of the proximal tibial is
determined by measuring the length of this pin. The preoperative planning notes are consulted
to determine the starting point for the distal guide pin with reference to the proximal pin,
which has been computed for the desired angle of correction. The distance between the two
guide pins is essentially the width of the base of the osteotomy wedge. E: The proximal tibial
osteotomy is made by using the two guide pins as an external jig system to guide the osteotome.
F: The outer half of the tibial width of the wedge of bone is removed as a single piece.
There are three basic options for the fibula when performing a HTO: proximal slide, proximal
fibular osteotomy, and distal fibular osteotomy. A proximal slide (disruption of tibiofibular joint)
is
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strongly contraindicated in ACL-deficient knees as this shortens the lateral and posterolateral
ligaments and may lead to a posterolateral instability in an already unstable knee. Our
procedure of choice is a proximal fibular osteotomy through the fibular neck region (Fig. 24-
14C,D ). Meticulous surgical technique, and protection and palpation of the peroneal nerve, are
essential. To protect the peroneal nerve, the lateral and posterior periosteal sleeve is carefully
preserved and not retracted under tension. If there is any question as to potential peroneal
nerve damage, exposure of the nerve is indicated with direct visualization and protection. The
fibular bone wedge that is removed is 2 to 3 mm less than the computed tibial wedge to allow
compaction of the fibula osteotomy. Excellent bony apposition is achieved when the osteotomy
is closed, and it is not necessary to add internal fixation.
An alternative choice for a fibular osteotomy is at the junction of the middle and distal thirds of
the fibula through a 3-cm posterolateral incision. The peroneal tendons are retracted anteriorly
and two bunion retractors can be used for exposure after subperiosteal exposure. A portion of
the medial cortex of the proximal fibula is preserved to maintain bone contact and promote
union. A bone graft from the tibial site is added.
The proximal tibial closing wedge osteotomy may be performed using a commercially available
calibrated osteotomy guide system (3 ) (NexGen Osteotomy System, Zimmer, Warsaw, IN). As an
alternative, bone cuts can be determined using a freehand method (Fig. 24-14E,F ). A smooth
guide pin is placed transversely just through but not beyond the medial cortex, 25 mm distal to
the joint line with the use of an image intensifier to ensure proper placement. It is critical to
leave at least 25 mm of proximal tibia to avoid a tibial plateau fracture. The transverse length
of the guide pin determines proximal tibia width. The method described by Slocum (70 )
involving a series of congruent right triangles is used to determine the entry point of the second
guidewire, again confirming proper positioning of the guidewires with fluoroscopy. These guide
pins determine the osteotomy triangular bone wedge that is removed to achieve the desired
correction.
We prefer to make our initial osteotomy cuts using a micro-oscillating saw to only cut the outer
cortex and then complete the osteotomy with thin osteotomes. An oscillating saw in the
cancellous bone may wander and potentially change the correction angle. A malleable retractor
is placed in the subperiosteal tissues posteriorly to protect the neurovascular structures, and the
knee is flexed 15 degrees. The lateral one half of the wedge is removed as a single piece. This
wedge of bone may occasionally need to be replaced if an inadvertent overcorrection is
obtained at surgery. The remaining wedge of bone is removed under direct visualization. The
surgeon is seated, using a headlamp to view the depth of the osteotomy. The osteotomy plane is
maintained in a perpendicular manner, noting that at the midpoint of the tibia, the tibial width
is one half of that at the lateral cortex.
The medial 7 to 10 mm of the wedge adjacent to the medial cortex is not disturbed. The medial
cortex is preserved to provide stability and prevent tibial medial or lateral translation or varus
recurrence. The posterior tibial cortex of the wedge is removed under direct visualization, with
a retractor placed subperiostealy at all times to protect the neurovascular structures. The
patellar tendon is carefully protected.
Two to three perforations of the medial cortex with a guidewire are often required before the
osteotomy gap can be closed with a gentle valgus force which is applied over a few minutes to
gradually deform the remaining medial cortex. Apposition of the bony surfaces of the tibia and
fibula should be visualized and inspected.
It is important as already stressed that HTO does not increase or decrease the normal posterior
tibial slope. An increase in the posterior tibial slope would result in a loss of knee extension and
place higher forces on an ACL reconstruction. A decrease or reverse in the tibial slope (anterior
tibia slopes distally) would produce knee hyperextension and place high forces on a PCL
reconstruction. The sagittal plane of the osteotomy should be perpendicular to the long axis of
the tibia with an equal width of cortex removed both anteriorly and posteriorly to preserve the
normal posterior tibial slope. In rare cases, usually after a tibial shaft fracture that healed in an
abnormal anterior or posterior position, a biplanar osteotomy for correction of an abnormal
tibial slope may be required.
Using fluoroscopy, an alignment guide rod (rigid 3 to 4 mm rod, 1 m in length) is positioned over
the center of the femoral head and the center of the tibiotalar joint to determine the newly
corrected WBL intersection at the tibial plateau. A large single staple may be placed across the
lateral tibial osteotomy site for provisional fixation. During this procedure the lower limb is
axially loaded at the foot to maintain closure of both the medial and lateral tibiofemoral
compartments. The knee is held at 5 degrees of flexion to avoid hyperextension. The alignment
guide rod represents a new WBL which should agree with preoperative calculations. If necessary,
further bone may be removed from
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the osteotomy to adjust the WBL as required. Internal fixation of the osteotomy is achieved
using an L-shaped plate. Two 6.5-mm cancellous screws are placed in the proximal tibia, and
two to three cortical screws are placed distal to the osteotomy (Fig. 24-15A,B ). Rarely, a 6.5-
mm cancellous screw is placed in a lag fashion across the osteotomy site into the medial aspect
of the proximal tibial bone for additional fixation. The final WBL is determined after fixation.
The tourniquet is released and hemostasis obtained. The fascia of the anterior compartment
musculature is loosely reattached to the anterolateral aspect of the tibial border. A Hemovac
drain is used for 24 hours. A soft Jones-type dressing is applied. The patient is admitted
overnight for ice, elevation, pain control, and monitoring of the neurovascular status.
Authors' Preferred Technique for Opening Wedge Tibial

Osteotomy
The same preoperative calculations are made as described for the closing wedge technique,
except the correction is achieved by performing an oblique opening wedge just proximal to the
tibial tubercle. The lower limb is positioned and arthroscopy performed as already described.
The ipsilateral anterior iliac crest is also prepped.
A 4-cm incision is made over the anterior iliac crest and deepened to the periosteum (Fig. 24-
16A, B ), which is sharply incised and reflected medially only to the pelvic brim. Laterally,
meticulous subperiosteal dissection is carried along the outer table of the pelvis. The graft size
is defined on the bone using electrocautery. In most patients, the graft dimension is 40 mm in
length, 10 mm in width, and 30 mm in depth. However, in smaller patients, the graft may be
smaller in width, approximately 8 mm in depth. Patients undergoing large osteotomies may
require a longer graft in length of approximately
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45 mm. The inner iliac cortex is not dissected, the muscle attachments are not disturbed, and a
spacer of the outer table defect is not required. Additional cancellous bone is removed from the
inner pelvic cortex. The graft is later fashioned into three triangles. One triangle is placed
posterior to the plate to close the gap at the posterior tibial cortex; one triangle is placed in the
midportion of the osteotomy deep to the plate; and the smaller triangle is placed in the gap in
the anterior tibial cortex. A drain is not required at the iliac crest harvest site.
FIGURE 24-15
Anteroposterior (A) and lateral (B) radiographs show the postoperative appearance after
proximal tibial osteotomy, fibular osteotomy, and internal fixation. (Reprinted with permission
from Noyes FR, Barber-Westin SD, Hewett TE. High tibial osteotomy and ligament reconstruction
for varus angulated anterior cruciate ligament-deficient knees. Am J Sports Med. 2000;28:282-
296.)
FIGURE 24-16
A: A 4-cm incision over the anterior iliac crest is made to harvest the iliac crest bone graft. The
graft is comprised of the anterior crest and outer iliac cortex; the inner table is not removed. B:
The usual iliac crest bone graft dimensions are 40 mm in length, 10 to 12 mm in width, and 30
mm in depth. (Reprinted with permission from Noyes FR, Mayfield W, Barber-Westin SD, et. al.
Opening wedge high tibial osteotomy. An operative technique and rehabilitation program to
decrease complications and promote early union and function. Am J Sports Med. 2006;34:1262-
1273.)
A 5-cm vertical skin incision is made medially midway between the tibial tubercle and
posteromedial tibial cortex, starting 1 cm inferior to the joint line (Fig. 24-17 ). The sartorius
fascia is incised in line with its fibers, proximal to the gracilis tendon. The sartorius and pes
anserinus are retracted posteriorly, exposing the superficial medial collateral ligament (SMCL)
and posterior border of the tibia. The SMCL is transected at its most distal tibial attachment.
Anteriorly, the retropatellar bursa is entered by incising the medial patellar retinaculum,
allowing the patellar tendon to be lifted to expose the anterior tibia. A small portion of the
patellar tendon fibers attaching medially may be incised at the tibial tubercle to achieve an
oblique osteotomy line.
A sharp periosteal incision is made at the posteromedial tibial border, just posterior to the
SMCL, to allow meticulous posterior tibial subperiosteal dissection by a Cobb elevator. Care is
taken to protect the inferior medial geniculate artery which lies just beneath the distal fibers of
the SMCL. A malleable retractor is placed in the subperiosteal posterior tibial space. Only
sufficient posterior tibial subperiosteal dissection is used to protect the neurovascular
structures, and wide dissection is not necessary (Fig. 24-18 ).
Management of Superficial Medial Collateral Ligament.

There are three surgical approaches for management of the SMCL. In a small opening wedge
osteotomy of 5 to 7.5 mm, a â€œpiecrustingâ€• procedure using multiple transverse incisions at
different places may effectively lengthen the SMCL. In larger opening wedge osteotomies, it is
necessary to transect the distal attachment, usually 6 cm from the joint, and perform a distal
elevation of the attachment in a subperiosteal plane. This allows for the SMCL to be reattached
distally after the opening wedge osteotomy with the posteromedial portion of the SMCL bridging
the osteotomy site. Transecting the SMCL at the most distal attachment site preserves its
length, allows excellent exposure and bone grafting, and allows the tibial fixation plate to be
placed in a correct midline position. The tibial slope is not altered, and function of the SMCL is
retained.
A third approach is used when distal advancement of the SMCL is required as a reconstructive
procedure due to SMCL insufficiency and abnormal medial joint opening. The SMCL is dissected
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to the medial joint which is entered anterior and posterior to the SMCL fibers. The posterior
incision is at the junction of the SMCL and posteromedial capsule (short oblique fibers). The
anterior incision preserves the attachment of the medial patellofemoral ligament. The medial
meniscus position is carefully examined when a distal advancement of the SMCL is performed. It
may be necessary to incise the medial meniscus capsular attachments and then resuture the
attachment to preserve the correct anatomic location of the meniscus when the SMCL is
advanced. The SMCL should appear relatively normal without scar replacement, as advancement
of only scar tissue would be expected to fail and not provide medial stability. In select cases, a
semitendinosus tendon augmentation of the SMCL may be necessary. The tendon is detached
proximally, passed through a small drill hole at the femoral attachment just anterior and
posterior to the SMCL femoral attachment, and then both anterior and posterior tendon arms are
sutured to the SMCL after the osteotomy is completed. The distal attachment of the
semitendinosus is not disturbed. In some instances, both the gracilis and semitendinosus tendons
may be used. The need to surgically reconstruct the SMCL is infrequent; however, the details as
presented should allow for restoration of function when required. At the conclusion of the SMCL
reconstruction, plication of the posteromedial capsule anteriorly to the SMCL is performed to
remove any abnormal redundancy with the knee at full extension. Overtightening of the SMCL
and posteromedial capsules is avoided; the repair allows for full knee extension, and a normal
range of flexion.
FIGURE 24-17
Skin incision over opening wedge osteotomy midway between the tibial tubercle and posterior
tibia along the anterior border of the superficial medial collateral ligament. The gracilis and
semitendinosus tendon insertions are marked.
FIGURE 24-18
Subperiosteal dissection of the superficial medial collateral ligament which is later transected at
its distal attachment in cases where a large opening wedge osteotomy is necessary.
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Placement of Tibial Guide Pins.

The patellar tendon and retropatellar bursa are exposed medially. The superior attachment of
the tendon is recessed 5 mm using a scalpel to provide adequate exposure for the osteotomy.
Retractors are placed anteriorly and posteriorly to complete the exposure. A Keith needle is
placed in the anterior medial joint just above the tibia, and the distance is marked on the
desired point of the osteotomy along the anteromedial cortex. A second Keith needle is placed
at the posteromedial tibial joint space, and the same millimeters are marked to provide a
measurement of the tibial slope (Fig. 24-19 ).
A commercial guide system (Arthrex Opening Wedge Osteotomy System, Arthrex Inc., Naples, FL)
is used to facilitate guidewire placement (Fig. 24-20A-D ). A 2-mm guide pin is placed at the
posteromedial cortex at the marked line and advanced across the tibia at an oblique angle. This
pin is usually placed at approximately 15 degrees of obliquity to the tibial shaft and is verified
by intraoperative fluoroscopy. To prevent fracture of the lateral tibial plateau, it is important to
retain as much lateral tibial width as possible. The error is to have too much obliquity to the
guide pin compromising postoperative tibial internal fixation. A second pin is placed anterior
and parallel to the first pin. At this point, it is imperative to ensure that the medial osteotomy
line (from anterior to posterior) is perpendicular to the joint line. A measurement of the
perpendicular cut is accomplished to confirm the distance of each guide pin from the articular
surface of the tibia. The distances should be equal in order to maintain the original posterior
tibial slope. The length of the posterior pin is measured and used following the law of triangles
(53 ) to determine the millimeters of osteotomy opening to obtain the desired angular
correction.
The osteotomy is performed using an oscillating saw for the outer medial and anterior cortices,
followed by a 3/4-inch osteotome, placed over the guide pin and verified by fluoroscopy (Fig.
24-21A,B ). A 1/2-inch osteotome is used for the posterior cortex, with the edge palpated
posterior to the tibia as the osteotome is advanced. The osteotomy is carried to within 10 mm of
the lateral cortex. Calibrated opening wedges are then gently inserted into the osteotomy site
to achieve the desired
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angular correction, hinging on the intact posterolateral cortex. This step requires several
minutes to prevent fracture of the lateral tibial pillar. Intraoperative fluoroscopy verifies the
correct hip-knee-ankle weight-bearing line at the tibia. In addition, the surgeon should
determine that there is closure of the medial and lateral tibiofemoral compartments using axial
weight bearing against the foot with the knee at 5 to 10 degrees of flexion. The alignment is
verified and adjusted if required to achieve the desired angular correction. The anterior gap of
the osteotomy site should be one half of the posterior gap, following rules previously described
to maintain the tibial slope (53 ). The width of the tibial buttress plate along the anteromedial
cortex is measured and is always less than the millimeters at the posterior medial gap due to the
angular inclination of the anteromedial tibial cortex. In select cases, the slope may be
purposefully increased in PCL-deficient knees or decreased in ACL-deficient knees.
FIGURE 24-19
The intraoperative photograph shows the placement of two Keith needles (arrows ) at the joint
line, which assists the surgeon in obtaining an osteotomy that is perpendicular to the normal
tibial slope. In the photograph, the Army-Navy retractor is beneath the patellar tendon. The
osteotomy site starts at the anteromedial cortex, 35 mm from the joint line in an oblique
manner just proximal to the tibiofemoral joint. This maintains sufficient width of the proximal
tibia and limits the risk of a tibial plateau fracture. (Reprinted with permission from Noyes FR,
Mayfield W, Barber-Westin SD, et al. Opening wedge high tibial osteotomy. An operative
technique and rehabilitation program to decrease complications and promote early union and
function. Am J Sports Med. 2006;34:1262-1273.)
FIGURE 24-20
Correct placement for guide pin and subsequent osteotomy with a thin osteotome. The lateral
cortex is preserved.
The three cancellous bone graft triangular segments are impacted tightly into the osteotomy site
to obliterate the space and provide added stability, particularly in the sagittal plane (Fig. 24-
22A,B ). An appropriately sized plate with trapezoidal block is selected and secured with 6.5-mm
cancellous screws proximally and 4.0-mm cortical screws distally (Fig. 24-23A-C ). We avoid the
use of plates with a square buttress block, since this geometry increases the posterior tibial
slope. For osteotomies 15 mm or greater, a low-profile T- or L-shaped locking plate is used.
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FIGURE 24-21
The use of a commercial (Arthrex Opening Wedge Osteotomy System, Arthrex Inc., Naples, FL)
osteotomy wedge to gradually open the osteotomy site.
FIGURE 24-22
A: The opening wedge triangular osteotomy opening is shown, along with the harvested and
prepared (see inset) iliac crest bone grafts. The bone segments are oversized by 1 to 2 mm to
achieve impaction into the osteotomy site. One graft is placed along the posterior tibial cortex
at the osteotomy site (A) , one is wedged beneath the buttress plate (B) , one graft is placed
anteriorly at the tibial tubercle (which is one-half the width of the posterior segment to
maintain tibial slope) (C) , and the remaining bone graft is packed deep into the osteotomy site
to promote healing. B: The final appearance at the osteotomy site. The iliac crest bone grafts
have been placed to restore the entire medial cortex from posterior to anterior to provide a
buttress at the osteotomy site. The senior author now prefers a locking path design for more
secure fixation. (Reprinted with permission from Noyes FR, Mayfield W, Barber-Westin SD, et al.
Opening wedge high tibial osteotomy: an operative technique and rehabilitation program to
decrease complications and promote early union and function. Am J Sports Med. 2006;34:1262-
1273.)
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FIGURE 24-23
Demonstration of a 15-mm opening wedge osteotomy, with incorporation of a large tibial plate.
A: Standing bilateral preoperative radiograph shows the desired opening wedge (gap) to be
obtained at the proximal tibia. B: An AO tibial side plate is shown, with cortical iliac crest
autograft implanted. C: Immediate postoperative anteroposterior radiograph. D: Tibial locking
screw plate designs are available for superior fixation as shown. (Reprinted with permission from
Noyes FR, Mayfield W, Barber-Westin SD, Albright JC, Heckmann TP. Opening wedge high tibial
osteotomy. An operative technique and rehabilitation program to decrease complications and
promote early union and function. Am J Sports Med. 2006;34:1262-1273.)
The SMCL fibers are sutured distally and secured to either the plate screws or to suture anchors
to maintain tension. The pes anserine tendons and sartorius fascia are reapproximated.
Pearls and Pitfalls

To avoid complications and inadequate correction following opening wedge osteotomy, the
following technical pearls are suggested:
Avoid too proximal placement of guide pin and osteotomy, as this can lead to lateral
tibial plateau fracture.
Verify the osteotomy starting point at the medial cortex and use anterior and posterior
Keith needles to verify tibial slope.
Perform subperiosteal dissection to protect the superficial medial collateral ligament.
Ensure a secure fixation of the medial collateral ligament repair to avoid valgus
instability.
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Use iliac crest bone graft, especially for larger opening wedges.
Use newer screw locking plate designs for secure fixation.
Measurement pitfalls: anterior tibial gap should be one half of the posteromedial gap.
Downsize plate from the posteromedial coronal gap measurement.
Begin immediate knee motion and patellar mobilization to avoid arthrofibrosis.
Avoid too oblique of an osteotomy by maintaining a 15-degree pin angulation to the tibial
axis.
Careful preoperative planning is required to avoid inadequate correction. During surgery,

confirmation of adequate correction should be accomplished under fluoroscopy. The knee will be
placed in an overall excessive valgus position if excessive tibiofemoral joint opening is not
evaluated on preoperative standing radiographs. Even though an ideal position is verified at
surgery, there may be a change when postoperative full-standing radiographs are taken under
the patients' partial or full weight postoperatively. These radiographs should be obtained by 6 to
8 weeks postoperatively (or sooner if there is a concern) to document the surgical correction
under weight-bearing conditions. If necessary, it is relatively easy to correct the coronal and
sagittal angles at the osteotomy by reoperation early, before bone healing.
The incidence of delayed or nonunion after osteotomy may be reduced by attention to technical
details. We do not perform the osteotomy distal to the tibial tubercle. The cancellous surfaces
should be cut in a manner that will maximize the amount of surface area that will be in
opposition. Adequate internal fixation is believed important with toe-touch weight-bearing in
the initial 4-week postoperative period to protect the osteotomy site. Any loss in the overall
alignment is an indication for a reoperation. From an empirical standpoint, we insist that no
nicotine be consumed for at least 16 weeks before surgery with total abstinence until healing of
the osteotomy is evident.
Most if not all of the complications reported in the literature regarding iliac crest bone graft
harvest may be avoided by the surgical technique described in this report. For example, we limit
the dissection to include only 10 mm of the superior iliac crest. A meticulous subperiosteal
exposure of the outer iliac crest is performed without violating the muscle plane. The inner iliac
cortex is never dissected, and the muscle attachments are kept intact. We acknowledge that the
iliac crest harvest procedure can be painful with trunk flexion activities for up to 4 weeks
postoperatively, and patients are advised accordingly. The advantage of the autogenous bone
graft is prompt healing, less time on crutches, less overall disuse, and lowered incidence of
delayed or nonunion (55 ).
Peroneal nerve palsy most commonly results from pressure from a tight bandage or lying with
the lower limb in external rotation postoperatively. The nerve may also be directly injured
during surgery. Osteotomy of the fibula is performed at different sites by different authors
depending on the operative technique. There is a risk of injuring the peroneal nerve if the
osteotomy is performed in the proximal third of the fibula (77 ). Fibular osteotomy in the middle
portion of the fibula may injure the peroneal nerve innervation to the extensor hallicus longus.
A patella infera condition may develop postoperatively due to quadriceps weakness and
concomitant contracture of peripatellar and fat pad tissues. If not recognized and treated
promptly, developmental patella infera can rapidly progress to a permanent patella infera and
disabling patellofemoral arthrosis (59 ,60 ). A diagnosis is established by measuring a decrease in
the vertical height ratio of the patella, either from preoperative measurements or from the
opposite knee, of 11% to 15%, depending on the method used (60 ).
We use immediate knee motion and patellar mobilization exercises, and a rehabilitation
protocol of straight-leg raises, multiangle isometrics, and electrical muscle stimulation to
decrease the incidence of quadriceps weakness and knee motion limitation following HTO. In
addition, a phased rehabilitation treatment program for limitations of motion is implemented
early postoperatively when restriction of either extension or flexion is noted (51 ). We had no
cases of patella infera in our prospective HTO studies following this program. Any suspect case
where there is a postoperative limitation of knee motion and patellar mobility requires lateral
radiographs to determine the patellar vertical height (ratio).
Fracture of the tibial plateau can be avoided by meticulously following the previously described
operative techniques. The proximal osteotomy should remain parallel to the joint surface and
maintain a minimum of 25 mm of tibial width proximally. The medial cortex may be penetrated
by a few drill holes, avoiding forceful closure of the wedge. If plateau fracture does occur during
a closing wedge osteotomy, anatomic reduction and internal fixation of the plateau fragments
may be performed and the osteotomy completed. In an opening wedge osteotomy, the
osteotomy is not completed, but delayed until adequate healing of the tibial plateau fracture is
demonstrated.
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Recognition of the anatomic relationship of the vessels to the surgical dissection is imperative.
The anterior tibial artery is at risk because it pierces the proximal interosseous membrane.
Injury to this artery may result in an anterior compartment syndrome, which must be recognized
and dealt with promptly. The popliteal artery is at risk during posterior dissection and osteotomy
of the posterior tibial cortex. By flexing the knee and gently retracting the popliteal structures,
the risk of injury to these structures will be significantly reduced.
POSTOPERATIVE REHABILITATION
The protocol for rehabilitation following HTO is shown in Table 24-6 . The program includes
immediate range of motion (0 to 90 degrees), quadriceps muscle isometric exercises, straight-
leg raises, patellar mobilization, and electrical muscle stimulation (49 ). For 1 week after
surgery, ice or a commercial cooling system, mild compression, and maximal elevation are used
to prevent edema and swelling. Patients are ambulatory for short periods of time, but are
instructed to elevate their limb, remain home, and not resume usual activities. Prophylaxis for
deep venous thrombosis includes intermittent compression foot boots in both extremities,
immediate knee motion exercises, antiembolism stockings, ankle pumps performed hourly, and
aspirin (600 mg daily for 10 days). Doppler ultrasound is routinely performed at 5 days
postoperative and at any time the patient demonstrates abnormal calf tenderness, a positive
Homan's sign, or increased edema.
A long-leg postoperative brace is worn for the first 8 weeks postoperatively. Initially, patients
are only allowed toe-touch weight bearing. At 4 weeks, 25% weight bearing is allowed and
gradually increased to full by week 8 if radiographs demonstrate adequate healing and
maintenance of the osteotomy position. If allograft bone is used in an opening wedge osteotomy,
a delay in full weight bearing is advocated until radiographic evidence of healing is
demonstrated. This may take 12 to 16 weeks for osteotomies greater than 10 mm. The protocol
emphasizes strengthening of the quadriceps, hamstring, hip, and gastrocsoleus musculature.
Closed-chain exercises and stationary bicycling are begun at week 4 postoperatively, and weight
machine exercises are begun at week 7 to 8 postoperatively. Excessive use of bicycling and
weight machines is not allowed in patients with articular cartilage damage. By week 9 to 12
postoperatively, other aerobic conditioning exercises are begun as appropriate including
swimming, ski machines, and walking.
Patients who express the desire to return to sports activities are advised of the risk of further
cartilage deterioration if damage was present at the index procedure. Muscle strength tests
should demonstrate quadriceps and hamstring strength values of at least 70% that of the
contralateral limb. We advise that there should be no pain or swelling with activities. The
majority of patients who undergo HTO usually return to only light recreational activities.
COMPLICATIONS
Inadequate or Loss of Correction of Lower Limb Alignment
Inadequate or overcorrection of lower limb alignment has been reported by many authors (13
,18 ,19 ,61 ). Hernigou et al reported persistent varus occurred in 10 of 93 knees immediately
following closing-wedge osteotomy (13 ). At 10 to 13 years postoperatively, 71 of 76 knees
showed some degree of recurrent varus deformity. Magyar et al reported a loss of correction
after closing-wedge osteotomy (28 ). Although normal alignment was demonstrated immediately
postoperatively, 9 of 16 knees in this series had collapsed into valgus by 1 year postoperatively.
Loss of the axial alignment obtained at surgery may be attributed to several factors, including
lack of internal fixation, inadequate internal fixation, or collapse of the distal fragments settling
into the cancellous bone of the plateau. Bauer et al reported that 15 of 65 knees (23%)
experienced shifting of the distal fragments medially after HTO; no internal fixation was used (2
). Hernigou et al reported that 21 of 93 patients (23%) lost correction within the first
postoperative year; displacement of the osteotomy prior to healing occurred in 11 knees, and
the osteotomy failed to correct the varus deformity in 10 knees (13 ). Stuart et al evaluated
standing tibiofemoral angles, which decreased from a mean of 9.3 degrees of varus to 7.8
degrees of valgus at final follow-up (73 ). Using the Kaplan-Meier survival analysis method, the
authors predicted that varus alignment was likely to occur in 18% of knees; lateral compartment
arthrosis was likely to progress in 60%; and medial and lateral compartment arthrosis was likely
to progress in 83% by 9 years after surgery. Recent survival rates
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reported by authors following closing and opening wedge osteotomy are shown in Tables 24-7
and 24-8 .
Brace: Long-leg postoperative
Range of motion minimum goals:
0-110 degrees
0-135 degrees
Weight bearing:
None to toe touch
1/4 body weight
1/2 to 3/4 body weight
Full
Patella mobilization
Modalities:
Electrical muscle stimulation
X
X
Pain/edema management (cryotherapy)
Stretching:
Hamstring, gastrocsoleus, iliotibial band, quadriceps
Strengthening:
Quad isometrics, straight-leg raises, active knee extension
Closed-chain: gait retraining, toe raises, wall sits, minisquats
Knee flexion hamstring curls (90 degrees)
Knee extension quads (90-30 degrees)

X
Hip abduction-adduction, multi-hip
Leg press (70-10 degrees)
Balance/proprioceptive training:
Weight-shifting, minitrampoline, BAPS, KAT, plyometrics

X
Conditioning:
UBE
Bike (stationary)
Aquatic program
X
X
Swimming (kicking)
Walking
Stair climbing machine

X
Ski machine
Running: straight
Cutting: lateral carioca, figure 8
Full sports
X
BAPS, Biomechanical Ankle Platform System (BAPS, Camp, Jackson, MI); KAT, Kinesthetic
Awareness Trainer (Breg, Inc., Vista, CA); UBE, upper body ergometer.
Reprinted with permission from Noyes FR, Mayfield W, Barber-Westin SD, et al. Opening wedge
high tibial osteotomy. An operative technique and rehabilitation program to decrease
complications and promote early union and function. Am J Sports Med. 2006;34:1262-1273.
Postop Weeks Postop Months
1-2 3-4 5-6 7-8 9-12 4 5 6
TABLE 24-6. Cincinnati Sportsmedicine and Orthopaedic

Center Rehabilitation Protocol Summary for High Tibial
Osteotomy
Delayed Union and Nonunion

Jackson and Waugh reported 19 of 226 (8%) patients had a delayed union following closing wedge
HTO; 14 were immobilized for more than 12 weeks before union was achieved, and the
remaining five required bone grafting (20 ). Naudie et al reported 15 of 106 (14%) patients had a
delayed union or nonunion after HTO (38 ).
Warden et al reported low incidence rates of delayed union and nonunion (6.6% and 1.8%,
respectively) in 188 opening wedge osteotomies where an iliac crest bone autograft was used in
the
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majority of knees (79 ). There was a trend toward an increased incidence of delayed union or
nonunion when a coral wedge was used either alone or in conjunction with the autogenous graft
(15%, 5 of 33 knees) compared to when an iliac crest graft was used alone (6%, 8 of 128 knees).
No problems with union were reported by Marti et al (32 ), Pace and Hofmann (63 ), or Patond
and Lokhande (65 ) following opening wedge osteotomy and iliac crest bone autograft
procedures.
Koshino 2004
N = 75 59 y (46-73 y)
1. TKA or unicompartmental arthroplasty
97.8%
96.2%
93.2%
None
2. Moderate pain at final f.u. >15 y p.o.
Aglietti 2003
N = 91 58 y (36-69 y)
1. TKA
96%
78%
57%
Alignment at healing, muscle strength, male gender
2. HSS score <70 points
Sprenger 2003
N = 76 69 y (47-81 y)
1. TKA
86%
74%
56%
Alignment at 1 y p.o.
2. HSS score <70 points
3. Patient dissatisfaction
Billings 2000
N = 64 49 y (23-69 y)
1. TKA
85%
53%
NA
None
Naudie 1999
N = 106 55 y (16-76 y)
1. TKA
73%
51%
39%
Body weight, delayed or nonunion, age, preop flex
Coventry 1993
N = 87 63 y (41-79 y)
1. TKA
87%
66%
NA
Body weight, alignment at 1 yp.o.
2. Moderate or severe pain in patients who declined TKA
Stuart 1990*
N = 75 58 y (33-75 y)
1. Recurrence varus deformity >5 degrees from femorotibial angle achieved at HTO
96%
82%
NA
None
62%
17%
NA
2. Medial compartment radiographic arthritic progression
81%
40%
NA
3. Lateral compartment radiograpic arthritic progression
HTO, high tibial osteotomy; TKA, total knee arthroplasty; y, years; f.u., follow-up; HSS, Hospital
for Special Surgery; p.o., postoperative.
* Follow-up for survival rates calculated at 9 years postoperative.
Survival Rates
Study Number End 5 Years 10 Years 15 Years Factors

of Points Postoperative Postoperative Postoperative Correlated
Patients, for with
Age at Survival Survival
HTO Analysis Rate
TABLE 24-7. Survival Rates Following Closing Wedge High

Tibial Osteotomy
Sterett 2004
N = 38 51 y (34-79 y)
1. TKA
84%
NA
NA
None
2. Revision HTO
Weale 2001
N = 76 54 y (36-70 y)
1. TK A
88.8%
63%
NA
None
2. Waiting for TKA
3. Postoperative sepsis precluded revision
Hernigou 2001
N = 215 61 y (48-72 y)
1. TKA
94%
85%
68%
None
HTO, high tibial osteotomy; TKA, total knee arthroplasty; y, years.
Survival Rates
Study Number End 5 Years 10 Years 15 Years Factors

of Points Postoperative Postoperative Postoperative Correlated
Patients, for with
Age at Survival Survival
HTO Analysis Rate
TABLE 24-8. Survival Rates Following Opening Wedge High
Tibial Osteotomy
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The effectiveness of allograft or other bone substitute materials for opening wedge osteotomy
has not been adequately determined, even though the introduction of commercially available
triangularshaped cortical-cancellous allografts (along with rigid plate designs) has led to their
increased usage. The clinical presumption that large osteotomy gaps would proceed to union
with the use of allograft or other bone substitute materials without loss of correction cannot be
currently answered. A porous hydroxylapatite wedge was used in conjunction with a fibular
autograft in 21 knees in one series, with no problems reported with union (24 ). The patient
population in that study averaged 66.6 years of age, and the osteotomies were small in
magnitude (maximum, 10 mm). A high 55% rate of complications was reported by one
investigation in which tricalcium phosphate was used to fill the wedge defect in 20 knees during
opening wedge osteotomy (66 ). Nonunions occurred in 35%; loss of correction in 15%; infections
in 10%; and material failure in 30%. Hernigou and Ma described the implementation of a block of
acrylic bone cement which was placed into the posteromedial portion of the osteotomy, in
combination with an anteromedial plate, in 203 knees (12 ). Only two patients had a delayed
union with loss of correction, and one patient had nonunion of the osteotomy. To our
knowledge, no report published to date has provided results using allograft bone to fill the
defect during opening wedge osteotomy.
Staubli et al reported on 92 opening wedge osteotomy cases in which no bone or bone

substitutes was used to fill the gap; a medial tibial plate with locking screws provided fixation
and support to the osteotomy site (71 ). There was a small (2%) incidence of delayed union and a
2% incidence of loss of correction; however, 40% required removal of the implant. Many
investigators have demonstrated that external fixators can accomplish adequate union and
correction, such as the series reported by Sterett et al., in which only 1 of 33 patients had a loss
of correction early postoperatively (72 ). However, pin tract infections were reported in 45% by
these authors, which were similar to the pin tract infection rate reported by Weale et al of 38%
(28 of 73 knees) (80 ) and Magyar et al of 51% (157 of 308 cases) (29 ).
Peroneal Nerve Injury or Palsy

In 1974, Jackson and Waugh reported 27 of 226 (12%) cases of partial or complete injury to the
peroneal nerve during osteotomy (20 ). There was a higher incidence of injury during curved
osteotomies located below the tibial tuberosity, and the authors concluded that osteotomy in
this location was too dangerous. A 7% incidence of peroneal nerve injury was reported by
Sundaram et al (75 ), and a 6% incidence was described by Harris and Kostuik (11 ). The rate of
injury is extremely high (25%) after dome osteotomy below the tibial tubercle (20 ). In three
reports by Noyes et al (42 ,49 ,55 ) of 137 osteotomies (including both opening and closing
wedge), no peronal nerve injuries were reported.
Arthrofibrosis and Patella Infera

Arthrofibrosis and developmental patella infera after knee surgery require prompt recognition
and treatment (59 ,60 ,69 ). Many early HTO studies cited unacceptably high rates of
arthrofibrosis and patella infera following postoperative immobilization. Windsor et al reported
that 80% of 45 knees had patella infera following closing wedge HTO and postulated that
postoperative plaster immobilization allowed the quadriceps muscle to relax and the patellar
ligament to shorten (82 ). Westrich and colleagues reported that 16 of 34 knees (47%) treated
with cast immobilization developed patella infera, compared with only 3 of 35 (8%) that received
immediate postoperative motion (81 ).
A decrease in the patellar height Blackburn-Peel ratio may be expected following opening wedge
osteotomy. Wright et al reported that all 28 patients in their series of medial opening wedge
osteotomies had a decrease in patellar height; however, no significant change in patellar
ligament length was detected using the Insall-Salvati ratio (17 ,83 ). The authors reasoned that
because the osteotomy increased the distance between the tibial tubercle and tibial articular
surface, the patella migrated distally. Other authors have reported similar findings in decreased
patellar height following
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opening wedge osteotomy (12 ,37 ,39 ,76 ). Patella infera has been noted to correlate with the
magnitude of angular correction after HTO (76 ,83 ).
Tibial Plateau Fracture

The incidence of tibial plateau fracture following osteotomy has been reported from 1% to 20%
(13 ,33 ). Matthews et al reported that 8 of 40 patients (20%) sustained a tibial plateau fracture
intraoperatively, which became the obvious cause for recurrence of the varus deformity in five
of these patients (33 ). Bauer et al (2 ) reported that six fractures occurred in 61 patients (10%),
and a similar incidence was published by Hernigou et al (13 ) (10 of 93 knees, 11%).
Tibial plateau fracture appears to be a rare problem following opening wedge osteotomy,
although Amendola et al reported 7 of 37 patients (19%) in their initial series of opening wedge
osteotomies had intra-articular fractures that extended into the lateral compartment (1 ). The
fractures were believed to be caused by a combination of a vertical osteotomy site (closer to the
lateral tibial plateau joint line than the lateral cortex) and use of thick osteotomes. After
adjusting the obliquity of the osteotomy and switching to thin, flexible osteotomes, the authors
did not experience any further cases of fracture.
Vascular Injury
Vascular complications are exceedingly rare following osteotomy (2 ,7 ,13 ,32 ). Flierl et al
reported the incidence of neurologic complications after two opening wedge osteotomy
techniques, one which used a conventional oscillating saw and another that created multiple
drill holes and osteoclasis (7 ). In the conventional sawing method group, acute transient
peroneal nerve palsy developed in 15.7% of 89 patients, with persistent deficits found in 12.4% 6
months postoperatively. In the osteoclasis group, 14% had acute transient events, and 4.7%
reported permanent weakness. Other than the report by Flierl et al, nerve injury after opening
wedge HTO appears to be extremely rare (13 ,32 ).
Deep-Vein Thrombosis
The incidence of deep-vein thrombosis (DVT) has not been adequately studied following HTO.
Turner et al reported a 41% rate of DVT using venography after HTO in 84 patients; only 15% of
these had been diagnosed clinically (78 ). Only calf clots were diagnosed clinically; three
proximal and 12 mixed clots were diagnosed with a venogram. Leclerc et al performed a
randomized, prospective trial comparing low-molecular-weight heparin (LMWH) to placebo after
129 osteotomies (27 ). The incidence of DVT was 17% in the LMWH group compared to 58% in the
placebo group. While 19% of the placebo group had femoral vein clots, none were detected in
the treatment group.
RESULTS
We completed three prospective studies assessing treatment results for closing wedge (42 ,49 )
and opening wedge (55 ) osteotomies. The first study reported the results of 41 patients (100%
follow-up) who were followed for an average of 58 months after closing wedge osteotomy
(range, 23 to 86 months) (42 ). These patients also had ACL-deficiency, of which 30 were treated
with a reconstructive procedure. Significant improvements were found at follow-up for pain,
swelling, and giving-way (p <.001) and 24 patients (59%) had returned to mostly light sports with
no symptoms. Thirty-six patients (88%) stated they would undergo the HTO again and 32 (78%)
felt that their knee condition was improved compared to its preoperative status. The
radiographic evaluation showed that 37 (90%) patients were surgically corrected with a WBL
between 50% and 80%. Patients who had advanced arthrosis with bone exposure in the medial
tibiofemoral compartment avoided ACL reconstruction by returning only to activities of daily
living.
The second study determined the results of patients with double or triple varus knees who had
varus malalignment treated by closing wedge osteotomy, ACL-deficiency treated by bone-
patellar tendon-bone graft reconstruction, and lateral ligament deficiency treated in certain
cases with a posterolateral ligament reconstruction (49 ). Forty-one patients remained in follow-
up a mean of 4.5 years (range, 2 to 12 years) after the HTO. At follow-up, significant
improvements were found for pain, swelling, and giving-way (p <0.001) (Fig. 24-24A-C ). Twenty-
nine patients (71%) had improvements in their pain score, and 27 patients (66%) had returned to
mostly light athletics without symptoms. The preoperative mean adduction moment of the study
group was 35% higher (p <
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0.001) than the control group value; 10 of the 17 patients (59%) had values that were greater
than 1 standard deviation above control values. Postoperatively, the adduction moment
decreased to significantly lower than control values. At surgery, all knees were corrected to a
WBL of 62%. At the most recent follow-up, 33 knees (80%) remained in an acceptable position.
We noted, as reported by other authors (13 ,18 ,19 ,61 ), a gradual return to a varus alignment
in certain knees due to continued wear and collapse of the medial tibiofemoral compartment
despite achieving an initial valgus correction.
FIGURE 24-24
Statistically significant improvements were found for pain (A) , swelling (B) , and giving-way (C)
from preoperative to follow-up (p < 0.01). ADL, activities of daily living. (Reprinted with
permission from Noyes FR, Barber-Westin SD, Hewett TE. High tibial osteotomy and ligament
reconstruction for varus angulated anterior cruciate ligament-deficient knees. Am J Sports Med.
2000;28:282-296.)
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Our third study followed 55 consecutive patients who underwent opening wedge osteotomy to
determine the rate of complications including nonunion, delayed union, alteration in tibial
slope, patellar infera, and arthrofibrosis (55 ). The patients were observed a mean of 20 months
(range, 6 to 60 months) postoperatively. All underwent the technique described in this report,
followed by the rehabilitation program detailed in Table 24-6 . The osteotomy opening size
ranged from 5 to 17.5 mm; 35 knees (64%) had openings of 10 mm or less, and 20 knees (36%)
had openings greater than 11 mm. The osteotomy united in all patients. Three knees had a delay
in union, which resolved by 6 to 8 months postoperatively. The iliac crest bone graft healed
without complications. There were no infections, loss of knee motion, nerve or arterial injuries,
alterations in tibial slope, or cases of patellar infera. Full weight bearing was achieved a mean
of 8 weeks (range, 4 to 11 weeks) postoperatively. A loss of fixation occurred in one patient who
admitted to resuming full weight bearing immediately after surgery, and the osteotomy was
successfully revised.
Reconstruction of the Posterolateral Knee Structures

Indications for reconstruction of the posterolateral structures include abnormal lateral joint
opening, increased external tibial rotation, and a varus recurvatum position with hyperextension
of the knee. The primary functioning posterolateral structures are the FCL and popliteus muscle-
tendon-ligament unit (PMTL), including the popliteofibular ligament (PFL) and posterolateral
capsule. These structures function together to resist lateral joint opening, posterior subluxation
of the lateral tibial plateau with tibial rotation, knee hyperextension, and varus recurvatum (10
,58 ,64 ). We previously described in detail the surgical options for correcting deficiency or
ruptures to the posterolateral structures (45 ,46 ,47 ). These options are based on the quality
and integrity of these tissues determined at the time of surgery (Fig. 24-25A,B ).
In cases of acute ligamentous disruptions, primary repair of the FCL is only indicated for bony
avulsions that are amendable to internal fixation. Otherwise, graft reconstruction of the FCL is
recommended, especially if an immediate knee motion program is to be used postoperatively, as
will be detailed following. We prefer to perform acute posterolateral reconstruction procedures
within the first 10 days of injury. Since posterolateral ruptures are usually accompanied by
injuries to one or both cruciates and may represent a knee dislocation, we observe these
patients for 1 week to evaluate the neurovascular structures and skin condition. This short delay
also allows appropriate planning of the surgical procedure and most importantly, the institution
of rehabilitation to initiate supervised range of motion and muscle exercises before surgery.
Our preferred reconstruction for either acute posterolateral injuries not amendable to primary
repair, or chronic ruptures with abnormal lateral joint opening, external tibial rotation, and
varus recurvatum is an anatomic FCL reconstruction with a bone-patellar tendon-bone graft. The
posterolateral reconstruction is performed using a straight lateral incision, approximately 12 to
15 cm in length centered over the lateral joint line (Fig. 24-26A-C ). The incision is extended
distally to allow exposure of the fibular head and proximally to allow exposure of the femoral
attachment of the FCL. The skin flaps are mobilized beneath the subcutaneous tissue and fascia
to protect the vascular and neural supply. An incision is made along the posterior border of the
iliotibial band and continued proximally, overlying the vastus lateralis. The attachment of the
iliotibial band to the lateral intra-muscular septum is preserved. The posterolateral structures
are identified both proximally at their femoral attachment (25 ) and distally. The interval
anterior to the lateral gastrocnemius tendon and muscle and posterior to the posterolateral
capsule is entered. The approach is similar to that described for meniscus repairs (34 ,35 ). This
allows exposure of the PMTL junction, posterolateral capsule, and PFL. The peroneal nerve is
protected throughout the procedure and usually is not dissected from its anatomic position.
The lateral joint capsule is incised vertically 10 mm anterior to the popliteal tendon's femoral
attachment site. An incision is made in the posterolateral capsule posterior to the FCL femoral
attachment,
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avoiding the popliteus tendon, to expose the lateral meniscus attachments and popliteal tendon
recess.
FIGURE 24-25
Algorithm for the treatment of acute (A) and chronic (B) posterolateral injuries. (Reprinted with
permission from Noyes FR, Barber-Westin SD, Albright JC: An analysis of the causes of failure in
57 consecutive posterolateral operative procedures. Am J Sports Med. 2006;34:1419-1430.)
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FIGURE 24-26
A: Straight lateral incision over the epicondyle, extended distally for an HTO. B: Exposure of the
posterolateral structures through an iliotibial band-splitting approach. C: The posterolateral
structures are identified and their quality and size determined.
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FIGURE 24-26
(Continued) D: Incision just proximal and superior to the attachment of the LCL to the
epicondyle. E: Elevation of a 6- to 7-mm wedge of bone. F: Alice clamps placed on the wafer of
bone. G: Release of the posterolateral capsule.
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FIGURE 24-26
(Continued) H,I: Reattachment of the bone with a four-pronged staple.
A 9 to 10 mm bone-patellar tendon-bone autograft or allograft may be used to replace the FCL.

The collagenous portion of the graft is usually 50 mm in length. The approximate length required
for the FCL bone-patellar tendon-bone graft is determined before surgery by measuring the
distance from the anatomic femoral insertion site to the anatomic fibular insertion site on a
lateral radiograph (using appropriate magnification markers). The length is also verified
intraoperatively. In some patients, the autogenous patellar tendon is insufficient in size and,
therefore, an allograft of the desired size is required for the procedure.
A tunnel at the proximal fibular attachment site is made over a guide pin that matches the same
depth and diameter of the bone portion of the graft. Only the anterior and proximal portion of
the fibula is exposed. The bone portion of the graft is gently tapped into the fibular tunnel and
attached with two 3.5-mm cortical screws. A guide pin is placed 5 mm proximal and eccentric to
the FCL femoral attachment to allow the collagenous portion of the graft to be anatomically
placed at the femoral attachment site. The femoral tunnel is drilled with a beath pin, and a
suture is attached to the bone to tension the graft. The FCL graft is tensioned at 30 degrees of
knee flexion with the lateral joint closed by placing approximately 9 newtons on the graft to
avoid overconstraining the lateral tibiofemoral compartment. An interference screw is placed at
the femoral anatomic insertion site of the FCL (Fig. 24-27A,B ).
In some patients, the autogenous patellar tendon is insufficient in size and therefore, an
allograft of the desired size is required for the FCL anatomic replacement procedure. We ensure
that sufficient allograft tissue is available during surgery if required. There are two methods
available for femoral fixation of the graft. While we normally use tunnel fixation with an
interference screw as described in this study, an inlay technique may be used with a four-prong
staple for grafts in which secure tunnel fixation may not be accomplished. The FCL graft
reconstruction forms the cornerstone and, along with repair or substitution of the posterolateral
structures, provides sufficient tensile strength for an immediate protected knee motion
program.
The use of a bone-patellar tendon-bone graft provides the advantages of bony incorporation at
the femoral and fibular insertion sites, avoiding the potential prolonged incorporation that may
occur
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with soft tissue grafts. Even so, asymmetrical loading at the bone-patellar tendon-bone interface
could be expected to occur with knee flexion-extension cycles. We believe that this graft
remodels at the insertion sites in a manner similar to that seen in bone-patellar tendon-bone
ACL reconstruction due to these sagittal plane motions. This is our current graft of choice for
FCL replacement.
In our experience, there are two procedures for restoration of the PMTL. The first, indicated in
chronic cases in which the distal attachments of the PMTL are elongated but intact, is a
proximal advancement of the popliteus tendon at its femoral attachment. The popliteus tendon
is transected at its femoral attachment site and recessed into a 7-mm tunnel using a beath
needle with one suture at the tendon end. Internal fixation is done with a reabsorbable
interference screw. In severe cases of acute ruptures of the posterolateral tissues or in chronic
cases in which the distal attachments of the PMTL are disrupted or deficient with scar tissue, it
is necessary to perform a graft substitution of the PMTL. We prefer to use an Achilles tendon-
bone allograft and place the bone portion of the graft at the anatomic femoral insertion site. An
alternative graft to consider is a bone-patellar tendon-bone allograft or a semitendinosus and
gracilis two-strand autograft. The graft is brought out through an 8-mm tunnel placed at the
most lateral tibia margin, 15 mm distal to the joint line, adjacent to the popliteus muscle.
Fixation is accomplished with a soft-tissue interference screw and suture post at the tibia (Fig.
24-28A-E ). This represents a reconstruction of the static portion of the popliteal-tibial
attachments. The graft is passed through an appropriately sized drill hole at the femoral
anatomic attachment of the popliteus tendon (25 ) and fixated by a soft tissue interference
screw and suture post if required. The graft is tensioned at 30 degrees of knee flexion in neutral
tibial rotation under approximately 9 newtons of force to avoid overconstraining the normal
amount of external tibial rotation.
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We do not recommend drilling a second tunnel for the PMTL reconstruction at the fibula, as this
may compromise the fixation strength of the FCL graft at the fibular attachment site. Two
nonabsorbable sutures placed between the PMTL graft and FCL graft secure the static arm of the
popliteus tendon to the fibula (PFL). We prefer to place the popliteus tendon graft on the
posterolateral aspect of the tibia at the popliteus muscle attachment.
FIGURE 24-27
Internal fixation of the LCL bone-patellar tendon-bone graft is accomplished on the femur by an
interference screw and on the fibula by two small cortical screws (A) . Ideally, the graft is
placed at the femoral site in a tunnel which is angulated 30 degrees to decrease stress
concentration effects. Alternatively, the fibula fixation may be accomplished with an
interference screw (with or without additional suture fixation) (B) in certain cases such as
revision knees where osteopenia may weaken graft fixation strength. (Redrawn with permission
from Noyes FR and Barber-Westin SD: Treatment of complex injuries involving the posterior
cruciate and posterolateral ligaments of the knee. Am J Knee Surg. 1996;9:200-214.)
FIGURE 24-28
Popliteus tendon-ligament reconstruction with two-strand semitendinosus autograft and lateral

collateral ligament reconstruction with bone-patellar tendon-bone autograft. A: A soft tissue
interference screw is used for tibial fixation of the popliteus semitendinosus autograft. B:
Passage of popliteus semitendinosus graft beneath lateral collateral ligament bone-patellar
tendon-bone autograft. The posterior exit tunnel is at the posterolateral corner of the tibia. C:
Final fixation of the popliteus and lateral collateral ligament graft reconstructions. D: Suture of
popliteus two-strand semitendinosus graft to posterior margin of lateral collateral ligament
autograft at the fibular attachment site to restore the popliteal fibular ligament. E: Suture
plication of posterolateral capsule to posterior margin of lateral collateral ligament bone-
patellar tendon-bone autograft.
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In our experience, most knees require a combined reconstruction of the posterolateral
structures and cruciate ligaments (Fig. 24-29A,B ). The order of the reconstruction of multiple
ligament procedures is as follows. First, the cruciate ligaments are reconstructed; however, the
tibial or femoral attachment site for the ACL or PCL, respectively, is not fixated. Second, the
posterolateral dissection is performed and the graft tunnels are drilled. Third, the knee is
reduced in the anteroposterior plane and the cruciate grafts secured (48 ). Finally, the FCL and
PMTL grafts are passed and the final fixation of the posterolateral structures is performed.
A second operative option we described for chronic posterolateral instability is a nonanatomic

femoral-fibular graft reconstruction. This procedure is indicated when the FCL is intact but
deficient and the PMTL does not require graft substitution. In addition, the procedure is
advantageous when operative time needs to be considered in dislocated knees and a relatively
easy stabilizing procedure can be performed (45 ). An Achilles tendon allograft is passed from
anterior to posterior through the anatomic FCL fibular attachment site and 3 to 4 mm anterior
and posterior to the FCL femoral attachment site. The circular graft provides the first arm of the
reconstruction and replaces important tensile bearing tissues laterally. The plication or
advancement of the posterolateral structures produces a dense collagenous plate of tissues
extending from the FCL and posterolateral corner of the knee joint. We described the results of
this operation in a consecutive group of patients (45 ). Twenty of 21 patients (95%) were
followed a mean of 42 months postoperatively (range, 24 to 73 months). The success rate of the
operative procedure was 76% as determined by stress radiographs and knee stability
examinations. The postoperative program of immediate knee motion restored 0 to 135 degrees
of motion in all knees and was not deleterious to the reconstructions. Significant improvements
were noted for functional limitations in sports activities (p <0.05), symptoms (p <0.01), and in
the overall rating score (p <0.0001).
A third operative approach may be considered in knees in which chronic insufficiency of the
posterolateral structures results from either a minor injury (without a traumatic disruption) or
from varus osseous malalignment and a varus recurvatum thrust on walking (46 ). The
posterolateral insufficiency is due to interstitial tearing, as a definitive FCL of normal width and
integrity (although lax) can be identified and the PMTL appears functionally intact even though
elongated. A graft reconstruction is not indicated in these knees. Instead, the posterolateral
tissues are proximally advanced in a more simplified manner that avoids the added operative
complexity and morbidity that may occur with major graft reconstructive procedures. Note that
the FCL must be at least 5 to 7 mm in width, and the posterolateral structures must be at least 3
to 4 mm thick, to be functional when the residual slackness is removed by the advancement
procedure. This procedure will not be effective if the posterolateral structures consist of scar
tissue or if the distal attachment site has been disrupted. One advantage of the proximal
advancement technique is that the ligamentous tissues are tightened in a more simplified
manner without the added operative complexity and morbidity that occurs with major graft
reconstructive procedures. We described the outcome of this procedure in a consecutive series
of knees (46 ). Twenty-one of 23 patients (91%) were followed a mean of 42 months (range, 23 to
94 months) postoperatively. The posterolateral advancement was found to be fully functional in
64%, partially functional in 27%, and nonfunctional in 9%. Overall, 71% of the patients showed
improvement in the pain score (p = 0.02), and all but one showed improvement in the overall
rating score (p = 0.0001).
Rehabilitation
The postoperative rehabilitation protocol following posterolateral reconstruction allows
immediate knee motion the first postoperative day, but includes protection against excessive
joint loads to prevent graft stretching and failure. Patients are warned to avoid hyperextension
and activities that would incur varus loading, external tibial rotation, or lateral joint opening.
Patients are placed initially in a bivalved cylinder cast for 4 weeks, which is removed for careful
active-assisted range of
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motion exercises in a range of 0 to 90 degrees 4 times a day. The cast is used in this time period
instead of a soft-hinged postoperative brace, as we think a brace will not provide sufficient
protection against excessive lateral joint opening that could occur with ambulation. At 4 weeks
postoperatively, a lower extremity, hinged, double-upright brace is applied locked at 10 degrees
of extension. The brace is removed 4 times daily for range of motion exercises. At 6 weeks, the
brace is unlocked, motion from 0 to 110 degrees is encouraged, and partial weight bearing from
25% to 50% of the patient' s body weight is allowed. At 7 to 8 weeks, a custom medial unloading
brace is applied as weight bearing progresses to full and flexion is advanced to 120 degrees. The
brace is also used as the patient returns to activity to provide some protection against knee
hyperextension and excessive varus loads.
FIGURE 24-29
Postoperative anteroposterior (A) and lateral (B) radiographs of a 22-year-old male who
underwent a PCL tibial inlay two-strand reconstruction, an ACL reconstruction, and a
posterolateral reconstruction. The LCL bone-patellar tendon-bone graft fixation is shown. The
method of fixation at the femur was a bone inlay at the anatomical LCL fixation site and at the
fibula by two small fragment screws. An advancement of the popliteus tendon at the femur and
repair of the PFL were also performed. (Reprinted with permission from Noyes FR, Barber-Westin
SD. Posterolateral knee reconstruction with an anatomical bone-patellar tendon-bone
reconstruction of the fibular collateral ligament. Am J Sports Med. 2007;35:259-273.)
In select athletes, a running program is begun at approximately 9 months postoperatively, and

plyometric and sports-specific training programs are initiated at 12 months postoperatively.
However, the majority of patients who require multiple ligament reconstructive procedures do
not want to return to high-impact sports and therefore, this advanced conditioning and training
is usually not required. We advise patients who have articular cartilage damage to return to low-
impact activities only to protect the knee joint.
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REFERENCES
1. Amendola A, Fowler PJ, Litchfield R, et al. Opening wedge high tibial osteotomy using a
novel technique: early results and complications. J Knee Surg. 2004;17:164-169.
2. Bauer GC, Insall J, Koshino T. Tibial osteotomy in gonarthrosis (osteoarthritis of the

knee). J Bone Joint Surg. 1969;51A:1545-1563.
3. Billings A, Scott DF, Camargo MP, et al. High tibial osteotomy with a calibrated osteotomy
guide, rigid internal fixation, and early motion. Long-term follow-up. J Bone Joint Surg Am.
2000;82:70-79.
4. Boss A, Stutz G, Oursin C, et al. Anterior cruciate ligament reconstruction combined with
valgus tibial osteotomy (combined procedure). Knee Surg Sports Traumatol Arthrosc.
1995;3:187-191.
5. Chao EYS. Biomechanics of high tibial osteotomy. In: Evarts CM, ed. AAOS Symposium on
Reconstructive Surgery of the Knee. St. Louis: Mosby; 1978;143-160.
6. Dugdale TW, Noyes FR, Styer D. Preoperative planning for high tibial osteotomy: the
effect of lateral tibiofemoral separation and tibiofemoral length. Clin Orthop Relat Res.
1992;274:248-264.
7. Flierl S, Sabo D, Hornig K, et al. Open wedge high tibial osteotomy using fractioned drill
osteotomy: a surgical modification that lowers the complication rate. Knee Surg Sports
Traumatol Arthrosc. 1996;4:149-153.
8. Fowler PJ, Tam JL, Brown GA. Medial opening wedge high tibial osteotomy: how I do it.
Oper Tech Sports Med. 2000;8:32-38.
9. Grood ES, Noyes FR, Butler DL, et al. Ligamentous and capsular restraints preventing
straight medial and lateral laxity in intact human cadaver knees. J Bone Joint Surg Am.
1981;63:1257-1269
10. Grood ES, Stowers SF, Noyes FR. Limits of movement in the human knee. Effect of
sectioning the posterior cruciate ligament and posterolateral structures. J Bone Joint Surg
Am. 1988;70:88-97.
11. Harris WR, Kostuik JP. High tibial osteotomy for osteoarthritis of the knee. J Bone Joint
Surg. 1970;52A:330-336.
12. Hernigou P, Ma W. Open wedge tibial osteotomy with acrylic bone cement as bone
substitute. Knee. 2001;8:103-110.
13. Hernigou P, Medevielle D, Debeyre J, et al. Proximal tibial osteotomy for osteoarthritis
with varus deformity. A ten- to thirteen-year follow-up study. J Bone Joint Surg.
1987;69A:332-354.
14. Hernigou P, Ovadia H, Goutallier D. Mathematical modelling of open-wedge tibial

osteotomy and correction tables. Rev Chir Orthop Reparatrice Appar Mot. 1992;78:258-263.
15. Holden DL, James SL, Larson RL, et al. Proximal tibial osteotomy in patients who are
fifty years old or less. A long-term follow-up study. J Bone Joint Surg. 1988;70A:977-982.
16. Hughston JC, Jacobson KE. Chronic posterolateral rotatory instability of the knee. J Bone
Joint Surg Am. 1985;67:351-359.
17. Insall J, Salvati E. Patella position in the normal knee joint. Radiology. 1971;101:101-
104.
18. Insall JN, Joseph DM, Msika C. High tibial osteotomy for varus gonarthrosis. A long-term
follow-up study. J Bone Joint Surg Am. 1984;66:1040-1048.
19. Ivarsson I, Myrnerts R, Gillquist J. High tibial osteotomy for medial osteoarthritis of the
knee. A 5- to 7- and 11- to 13-year follow-up. J Bone Joint Surg. 1990;72B:238-244.
20. Jackson JP, Waugh W. The technique and complications of upper tibial osteotomy. A
review of 226 operations. J Bone Joint Surg. 1974;56B:236-245.
21. Johnson F, Leitl S, Waugh W. The distribution of load across the knee. A comparison of
static and dynamic measurements. J Bone Joint Surg. 1980;62B:346-349.
22. Kettelkamp DB. Tibial osteotomy. In: Evarts CM, ed. Surgery of the musculoskeletal
system. New York: Churchill Livingstone; 1990;3551-3567.
23. Kettelkamp DB, Jacobs AW. Tibiofemoral contact areaâ€”determination and

implications. J Bone Joint Surg Am. 1972;54:349-356.
24. Koshino T, Murase T, Saito T. Medial opening-wedge high tibial osteotomy with use of
porous hydroxyapatite to treat medial compartment osteoarthritis of the knee. J Bone Joint
Surg Am. 2003;85-A:78-85.
25. LaPrade RF, Ly TV, Wentorf FA, et al. The posterolateral attachments of the knee: a
qualitative and quantitative morphologic analysis of the fibular collateral ligament,
popliteus tendon, popliteofibular ligament, and lateral gastrocnemius tendon. Am J Sports
Med. 2003;31:854-860.
26. Lattermann C, Jakob RP. High tibial osteotomy alone or combined with ligament
reconstruction in anterior cruciate ligament-deficient knees. Knee Surg Sports Traumatol
Arthrosc. 1996;4:32-38.
27. Leclerc JR, Geerts WH, Desjardins L, et al. Prevention of deep vein thrombosis after
major knee surgeryâ€”a randomized, double-blind trial comparing a low molecular weight
heparin fragment (enoxaparin) to placebo. Thromb Haemost. 1992;67:417-423.
28. Magyar G, Toksvig-Larsen S, Lindstrand A. Changes in osseous correction after proximal

tibial osteotomy: radiostereometry of closed- and open-wedge osteotomy in 33 patients.
Acta Orthop Scand. 1999;70:473-477.
29. Magyar G, Toksvig-Larsen S, Lindstrand A. Hemicallotasis open-wedge osteotomy for

osteoarthritis of the knee. Complications in 308 operations. J Bone Joint Surg Br.
1999;81:449-451.
30. Markolf KL, Bargar WL, Shoemaker SC, et al. The role of joint load in knee stability. J
31. Marti CB, Gautier E, Wachtl SW, et al. Accuracy of frontal and sagittal plane correction
in open-wedge high tibial osteotomy. Arthroscopy. 2004;20:366-372.
32. Marti RK, Verhagen RA, Kerkhoffs GM, et al. Proximal tibial varus osteotomy. Indications,
technique, and five to twenty-one-year results. J Bone Joint Surg Am. 2001;83-A:164-170.
33. Matthews LS, Goldstein SA, Malvita TA, et al. Proximal tibial osteotomy. Factors that
influence the duration of satisfactory function. Clin Orthop Relat Res. 1988;229:193-200.
34. McLaughlin JR, Noyes FR. Arthroscopic meniscus repair: recommended surgical
techniques for complex meniscal tears. Techniques in Orthopaedics. 1993;8:129-136.
35. Medvecky MJ, Noyes FR. Surgical approaches to the posteromedial and posterolateral
aspects of the knee. J Am Acad Orthop Surg. 2005;13:121-128.
36. Muller W. Kinematics of the cruciate ligaments. In: J.A. Feagin, ed. The cruciate
ligaments. Diagnosis and treatment of ligamentous injuries about the knee. New York:
Churchill Livingstone; 1988:217-233.
37. Nakamura E, Mizuta H, Kudo S, et al. Open-wedge osteotomy of the proximal tibia with
hemicallotasis. J Bone Joint Surg Br. 2001;83:1111-1115.
P.358
38. Naudie D, Bourne RB, Rorabeck CH, et al. Survivorship of the high tibial valgus
osteotomy. A 10- to 22-year followup study. Clin Orthop Relat Res. 1999;367:18-27.
39. Naudie DD, Amendola A, Fowler PJ. Opening wedge high tibial osteotomy for
symptomatic hyperextension-varus thrust. Am J Sports Med. 2004;32:60-70.
40. Noyes FR, Barber SD. The effect of a ligament-augmentation device on allograft
reconstructions for chronic ruptures of the anterior cruciate ligament. J Bone Joint Surg Am.
1992;74:960-973.
41. Noyes FR, Barber SD. The effect of an extra-articular procedure on allograft
reconstructions for chronic ruptures of the anterior cruciate ligament. J Bone Joint Surg Am.
1991;73:882-892.
42. Noyes FR, Barber SD, Simon R. High tibial osteotomy and ligament reconstruction in
varus angulated, anterior cruciate ligament-deficient knees. A two- to seven-year follow-up
study. Am J Sports Med. 1993;21:2-12.
43. Noyes FR, Barber-Westin SD. Anterior cruciate ligament revision reconstruction. Results
using a quadriceps tendon-patellar bone autograft. Am J Sports Med. 2006;34:553-564.
44. Noyes FR, Barber-Westin SD. Revision anterior cruciate surgery with use of bone-patellar
tendon-bone autogenous grafts. J Bone Joint Surg Am. 2001;83-A:1131-1143.
45. Noyes FR, Barber-Westin SD. Surgical reconstruction of severe chronic posterolateral
complex injuries of the knee using allograft tissues. Am J Sports Med. 1995;23:2-12
46. Noyes FR, Barber-Westin SD. Surgical restoration to treat chronic deficiency of the
posterolateral complex and cruciate ligaments of the knee joint. Am J Sports Med.
1996;24:415-426.
47. Noyes FR, Barber-Westin SD. Treatment of complex injuries involving the posterior
cruciate and posterolateral ligaments of the knee. Am J Knee Surg. 1996;9:200-214.
48. Noyes FR, Barber-Westin SD, Grood ES. Newer concepts in the treatment of posterior
cruciate ligament ruptures. In: Insall JN, Scott WN, eds. Surgery of the knee. Philadelphia:
WB Saunders; 2001:841-877.
49. Noyes FR, Barber-Westin SD, Hewett TE. High tibial osteotomy and ligament
reconstruction for varus angulated anterior cruciate ligament-deficient knees. Am J Sports
Med. 2000;28:282-296.
50. Noyes FR, Barber-Westin SD, Roberts CS. Use of allografts after failed treatment of
rupture of the anterior cruciate ligament. J Bone Joint Surg Am. 1994;76:1019-1031
51. Noyes FR, Berrios-Torres S, Barber-Westin SD, et al. Prevention of permanent

arthrofibrosis after anterior cruciate ligament reconstruction alone or combined with
associated procedures: a prospective study in 443 knees. Knee Surg Sports Traumatol
Arthrosc. 2000;8:196-206.
52. Noyes FR, Cummings JF, Grood ES, et al. The diagnosis of knee motion limits,
subluxations, and ligament injury. Am J Sports Med. 1991;19:163-171.
53. Noyes FR, Goebel SX, West J. Opening wedge tibial osteotomy: the 3-triangle method to
correct axial alignment and tibial slope. Am J Sports Med. 2005;33:378-387.
54. Noyes FR, Grood ES, Torzilli PA. Current concepts review. The definitions of terms for
motion and position of the knee and injuries of the ligaments. J Bone Joint Surg Am.
1989;71:465-472.
55. Noyes FR, Mayfield W, Barber-Westin SD, et al. Opening wedge high tibial osteotomy: an
operative technique and rehabilitation program to decrease complications and promote
early union and function. Am J Sports Med. 2006; 34:1262-1273.
56. Noyes FR, Mooar PA, Matthews DS, et al. The symptomatic anterior cruciate-deficient
knee. Part I: the long-term functional disability in athletically active individuals. J Bone
Joint Surg Am. 1983;65:154-162
57. Noyes FR, Schipplein OD, Andriacchi TP, et al. The anterior cruciate ligament-deficient
knee with varus alignment. An analysis of gait adaptations and dynamic joint loadings. Am J
Sports Med. 1992;20:707-716
58. Noyes FR, Stowers SF, Grood ES, et al. Posterior subluxations of the medial and lateral
tibiofemoral compartments. An in vitro ligament sectioning study in cadaveric knees. Am J
Sports Med. 1993;21:407-414
59. Noyes FR, Wojtys EM. The early recognition, diagnosis and treatment of the patella
infera syndrome. In: Tullos HS, ed. Instructional Course Lectures. Rosemont, IL: AAOS;
1991:233-247.
60. Noyes FR, Wojtys EM, Marshall MT. The early diagnosis and treatment of developmental
patella infera syndrome. Clin Orthop. 1991;265:241-252.
61. Odenbring S, Egund N, Knutson K, et al. Revision after osteotomy for gonarthrosis. A 10-
19 year follow-up of 314 cases. Acta Orthop Scand. 1990;61:128-130.
62. Odenbring S, Tjornstrand B, Egund N, et al. Function after tibial osteotomy for medial
gonarthrosis below aged 50 years. Acta Orthop Scand. 1989;60:527-531.
63. Pace TB, Hofmann AA, Kane KR. Medial-opening high-tibial osteotomy combined with
Magnuson intraarticular debridement for traumatic gonarthrosis. J Orthop Tech. 1994;2:21-
28.
64. Pasque C, Noyes FR, Gibbons M, et al. The role of the popliteofibular ligament and the
tendon of popliteus in providing stability in the human knee. J Bone Joint Surg Br.
2003;85:292-298.
65. Patond KR, Lokhande AV. Medial open wedge high tibial osteotomy in medial
compartment osteoarthrosis of the knee. Nat Med J India. 1993;6:105-108.
66. Patt TW, Kleinhout MY, Albers RG, et al. Paper #142. Early complications after high tibial
osteotomyâ€”A comparison of two techniques (open vs. closed wedge). Arthroscopy.
2003;19:74.
67. Rubman MH, Noyes FR, Barber-Westin SD. Arthroscopic repair of meniscal tears that
extend into the avascular zone. A review of 198 single and complex tears. Am J Sports Med.
1998;26:87-95.
68. Schipplein OD, Andriacchi TP. Interaction between active and passive knee stabilizers
during level walking. J Orthop Res. 1991;9:113-119.
69. Scuderi GR, Windsor RE, Insall JN. Observation on patellar height after proximal tibial
osteotomy. J Bone Joint Surg. 1989;71A:245-248.
70. Slocum DB, Larson RL, James SL, et al. High tibial osteotomy. Clin Orthop Relat Res.
1974;104:239-243.
71. Staubli AE, De Simoni C, Babst R, et al. TomoFix: a new LCP-concept for open wedge
osteotomy of the medial proximal tibiaâ€”early results in 92 cases. Injury. 2003;34(suppl
2):B55-62.
72. Sterett WI, Steadman JR. Chondral resurfacing and high tibial osteotomy in the varus
knee. Am J Sports Med. 2004;32:1243-1249.
73. Stuart MJ, Grace JN, Ilstrup DM, et al. Late recurrence of varus deformity after proximal
tibial osteotomy. Clin Orthop Relat Res. 1990;260:61-65.
74. Stutz G, Boss A, Gachter A. Comparison of augmented and non-augmented anterior

cruciate ligament reconstruction combined with high tibial osteotomy. Knee Surgery Sports
Traumatology Arthroscopy. 1996;4:143-148.
P.359
75. Sundaram NA, Hallett JP, Sullivan MF. Dome osteotomy of the tibia for osteoarthritis of
the knee. J Bone Joint Surg. 1986;68B:782-786.
76. Tigani D, Ferrari D, Trentani P, et al. Patellar height after high tibial osteotomy. Int
Orthop. 2001;24:331-334.
77. Tjornstrand BE, Egund N, Hagstedt BV. High tibial osteotomy: a seven-year clinical and
radiographic follow-up. Clin Orthop Relat Res. 1981;160:124-135.
78. Turner RS, Griffiths H, Heatley FW. The incidence of deep-vein thrombosis after upper
tibial osteotomy. A venographic study. J Bone Joint Surg Br. 1993;75:942-944.
79. Warden SJ, Morris HG, Crossley KM, et al. Delayed- and nonunion following opening
wedge high tibial osteotomy: surgeons' results from 182 completed cases. Knee Surg Sports
Traumatol Arthrosc. 2005;13:34-37.
80. Weale AE, Lee AS, MacEachern AG. High tibial osteotomy using a dynamic axial external
fixator. Clin Orthop Relat Res. 2001;382:154-167.
81. Westrich GH, Peters LE, Haas SB, et al. Patella height after high tibial osteotomy with
internal fixation and early motion. Clin Orthop Relat Res. 1998;354:169-174.
82. Windsor RE, Insall JN, Vince KG. Technical considerations of total knee arthroplasty after
proximal tibial osteotomy. J Bone Joint Surg. 1988;70A:547-555.
83. Wright JM, Heavrin B, Begg M, et al. Observations on patellar height following opening
wedge proximal tibial osteotomy. Am J Knee Surg. 2001;14:163-173.
> Table of Contents > Part Three - Ligament Injuries and Instability > 25 - Guidance System
for ACL Reconstruction
25
Guidance System for ACL Reconstruction
Jason L. Koh
Anterior cruciate ligament (ACL) reconstruction is a commonly performed operation,

with over 175,000 procedures performed in the United States with a cost of more than
$1 billion. Precise graft placement has a significant effect on clinical outcomes, with
poor tunnel location related to pain, loss of motion, impingement, graft failure, and
arthritis. According to the American Orthopedic Society for Sports Medicine (AOSSM),
there is a relatively high revision rate (10% to 20%), and these failures are usually
associated with technical factors, specifically tunnel placement. Several studies have
documented the difficulty in manually placing ACL tunnels, even in the hands of
experienced surgeons.
Current computer-aided navigations systems primarily use optical tracking of markers

attached to the femur, tibia, and instruments. Almost all systems will allow the use of
any graft choice or type of fixation, and standard manual instruments can be used,
and position verified with navigation. Navigation systems provide additional
information about the location of tunnels, isometry, and impingement data. They also
can document movement in multiple planes, such as rotational stability as well as pure
anterior-posterior stability.
Computer-aided navigation systems improve the accuracy of ACL tunnel placement and
can reliably document translational and rotational outcomes. Reasons for navigation
include:
Manual tunnel placement has been demonstrated to have errors.
Navigation improves accuracy of ACL tunnel placement.
Real time information about impingement is provided.
Real time information about isometry is provided.
Real time information about location from the over-the-top position is

provided.
Postoperative stability is improved with navigation.
Navigation allows documentation of rotation and translation.
Indications
Indications for computer-aided navigation are knee ligament reconstructions where
accuracy and precision are desired. My preference is to use navigation in almost every
case. Particular situations in which navigation is useful include:
Revision ACL reconstruction, where the anatomy may be altered and navigation
may provide additional information about alternative tunnel locations
Single-bundle (anteromedial or posterolateral bundle) reconstruction of partial

ACL tears
Double-bundle ACL reconstruction
Combined osteotomy/ligament reconstruction procedures
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Contraindications
Relative contraindications for computer-aided navigation are situations where there is
an inability to palpate and register intra-articular landmarks accurately, or when
kinematic data are unable to be obtained. The result would be potential errors in
parts of the navigation; however, some of the data will remain useful. Caution should
be used when there is significant bony loss or inability to obtain consistent flexion-
extension data. When used in cases of multiple ligament injury, it is critical to keep
the tibia reduced on the femur during acquisition of data. If this cannot be
accomplished in the grossly unstable knee, the projection of the intercondylar notch
on the tibia (impingement screen) will be incorrect, and the isometry data will also be
incorrect.
Essentially no preoperative preparation is needed for the Orthopilot system. All
measurements can be obtained intraoperatively. Optional preoperative radiographic
data can be input into the machine, but I typically do not find this necessary.
SURGERY
In many ways the technique is identical to the standard ACL procedure, except that
prior to the drilling of tunnels, points are registered in the computer and kinematic
data are obtained. It is important to remember that navigation serves as a precise
measuring tool; it does not dictate the course of surgery or tunnel position. Typically,
to improve workflow, all intra-articular work such as stump removal and notch
preparation is performed prior to marker placement.
Marker Placement
Reflective balls are attached to the tibial, femoral, and instrument trackers, and the
trackers are then placed in a secure location on the back table. The surgeon's
preferred graft type is harvested and prepared as usual. Data regarding the type of
graft and size of graft are input into the computer. The computer is carefully
positioned opposite the operative leg toward the foot of the bed with the camera
facing the operative site (Fig. 25-1). Two stab incisions are made in the distal tibia,
and two unicortical K-wires are placed into the tibia distally to avoid the tibial tunnel
site (Fig. 25-2). Arthroscopy fluid can be used to irrigate and cool the K-wires as they
are drilled in to the tibia. A pin clamp is attached to the K-wires and the tibial tracker
is then attached to the clamp. Two K-wires are then drilled into the medial
epicondyle, and the femoral tracker is attached to a clamp attached to the pins. I
prefer to place the femoral tracker in an area with relatively less soft tissue motion to
avoid stress on the pins.
Registration
Extra-articular landmarks including the tibial tubercle, anterior tibial spine, and
medial and lateral tibial plateau are registered in the computer by using a pointer
with attached markers. The tip of the pointer is placed on the structure and stabilized
by the finger of the surgeon, and the right foot pedal is used to register the data. This
is followed by acquisition of kinematic data. The tibia is reduced onto the femur and
the knee is evaluated in full extension, at 90 degrees of flexion, and through an arc of
motion. This is followed by acquisition of intra-articular landmarks. The anterior
portion of the posterior cruciate ligament (PCL) is lightly touched with the tip of the
probe and registered, followed by the medial tibial eminence and anterior horn of the
lateral meniscus. It is important to make sure that the tibia is not anteriorly
subluxated when the PCL is palpated, since this will falsely place the PCL more
posterior than its normal anatomic position with respect to the ACL footprint. The
anterior edge of the intercondylar notch is then sequentially palpated and registered.
The instrument tracker is then attached to the hook probe, and the over-the-top
position is palpated at 12 o'clock (Fig. 25-3) and 1:30/10:30 with the tip of the
instrument at the junction of the bone and soft tissue fringe. This step is particularly
useful in avoiding palpation of â€œresident's ridgeâ€• since instantaneous feedback on
the length of Blumensaat's line is provided. (Standard length is approximately 30 to 32
mm; values of 25 to 26 mm suggest that there is too anterior palpation of the over-
the-top position). The ACL origin points are then acquired. Preoperative kinematic
data are then acquired by placing the reduced knee at the desired angle (typically 30
degrees) and applying an anterior and posterior
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force. The rotational laxity of the tibia with respect to the femur is also registered by
internally and externally rotating the tibia.
FIGURE 25-1
Operating room setup.
FIGURE 25-2
Tracker placement and palpation. Note that tibial tracker pins are placed
relatively distal to the origin of the tibial tunnel and that the femoral pins are
placed near the epicondyle.
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FIGURE 25-3
Palpation of the over the top position (Blumensaat's line). Average length is 30 to
32 mm; shorter distances suggest incorrect palpation.
Tibial Tunnel Mapping

The tibial drill guide is then placed intra-articularly, and an approximate position for
the tibial tunnel is chosen. The instrument tracker is attached to the drill guide, and
the relationship of the proposed tunnel to intra- and extra-articular landmarks is seen
on the navigation screen (Fig. 25-4). Information provided includes the location of the
intercondylar notch with the knee in full extension projected onto the tibial plateau;
the distance from the PCL, and the location of the medial tibial eminence and the
anterior horn of the lateral meniscus. The amount and location of any impingement
are also shown. The percentage of the medial-lateral distance is calculated. The
coronal and sagittal angle of the tibial tunnel with respect to the tibia is also shown,
which aids in aiming the tunnel to the appropriate position on the femur. Taking the
provided information into account, the surgeon chooses a tunnel location, and a
guidewire is drilled into the elbow of the guide. The tunnel location is then registered
using the foot pedal. The tibial tracker can be removed from the tibial pin clamp at
this point prior to drilling the tunnel, and reattached at the end of the case for
evaluation of knee stability.
Femoral Tunnel Mapping

Typically, a standard manual offset guide is used to identify an approximate position
for the femoral tunnel, and a dimple is made by placing a guide pin through the guide.
The offset guide can be placed transtibially or through a low anterior portal;
alternatively, an outside-in technique can be used and a pin placed. The instrument
tracker is then attached to the femoral probe, and the approximate starting point is
palpated. The femoral navigation screen (Fig. 25-5) provides real-time information on
the distance to the over-the-top position, graft isometry, location on the clock face,
and the location and amount of any potential impingement. Using this information the
surgeon can decide on and register an appropriate starting point. The guidewire is
then drilled into this point and the reamer is used to create the femoral tunnel.
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FIGURE 25-4
Tibial navigation screen demonstrating the projected notch, indicating areas of

impingement, distance from the PCL, anterior horn of the lateral meniscus, and
medial tibial eminence. The coronal and sagittal angle of the tunnel are also
shown.
FIGURE 25-5
Femoral navigation screen demonstrating the areas of potential impingement,

isometry, distance from the over-the-top position, and location on the clock face.
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Final Evaluation
After tunnel creation, the graft is passed into the knee, tensioned and cycled, and
secured with the surgeon's preferred fixation devices. The tibial tracker is reattached
to the tibial pin clamp, and knee is placed in the appropriate degree of flexion to
retest anterior-posterior and rotational stability. Similar forces are applied to the
reconstructed knee to evaluate laxity, and the pre- and postoperative anteroposterior
(AP) translation in millimeters and the degrees of rotation are displayed (Fig. 25-6).
These measurements are then recorded, and then the markers and pins can be
removed. A summary data sheet can be printed out. Typically, the AP stability
improves from a mean of 15 to 5 mm, and the rotational arc improves from 28 to 18
degrees. Steristrips are placed over the pin sites.
Pearls and Pitfalls
The markers should be positioned to avoid interfering with tunnel drilling and
placement, and the pins should be securely fastened to bone. Slightly
divergent pins improve the stability of the clamps. The markers can be
attached to a larger screw if sufficient bony stability cannot be achieved.
Placing the femoral pins in an area with relatively less soft tissue motion over
bone will decrease the chance of pin loosening. If the pins become loose, they
can be reinserted, but reregistration should be performed.
The reflective markers need to be kept dry during the case. Placing gauze or a
sponge on the shaft of instruments exiting the joint can help reduce spray of
fluid onto the trackers. If the markers get wet, a dry sponge and a quarter turn
of the reflective ball can improve the ability of the camera to track the
marker.
Intra-articular palpation must be done accurately. During palpation of the PCL,

it is important to keep the tibia appropriately reduced, avoiding anterior
translation of the tibia. The tip of the probe should be barely touching the
anterior fibers of the PCL. The notch palpation can proceed in only one
direction. If there are obvious discrepancies between the navigation screen
and the arthroscopic appearance of the tibia or femur, it is recommended to
reregister data points. In addition, anterior translation of the tibia on the
femur during the kinematic registration will result in abnormal impingement
and isometry data.
At any point during the registration and measurement process, previous

screens can be accessed and points can be removed using the left foot pedal.
Pressing both the pedals will allow screenshots or the use of an intra-articular
measuring tool.
FIGURE 25-6
Postoperative knee stability screen showing both pre- and postoperative anterior
translation and rotation values.
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The surgeon's standard postoperative protocol can be used.
COMPLICATIONS
The goal of navigation is to reduce the rate of improperly placed tunnels.
Complications associated with computer-assisted navigation can be associated with
equipment or software. The information displayed on computer navigation screens is
only as accurate as the registration points input by the user. The surgeon should
always correlate the information on the screen with the anatomy visualized
arthroscopically. In its current format, the Orthopilot system is quite robust and rarely
has a significant error or freezes. If at any point there is a discrepancy between the
navigation screens and actual anatomy, re-registration should be performed. If there
are persistent abnormalities, the surgeon always has the option to perform the
standard manual instrumentation technique.
The only other complication noted has been some minimal discomfort associated with
the unicortical tibial pin sites. One patient developed superficial erythema and some
drainage associated with one site that resolved over a period of 2 weeks. Given the
size of the K-wires used to attach the trackers, it is unlikely that fracture will occur
through the pin sites.
RESULTS
Results using a variety of computer-aided navigation systems have consistently
demonstrated improved tunnel position and clinical measurements. My experience
(4,5) has demonstrated improved tunnel placement, with the tibial tunnel located
slightly more anterior in the anatomic footprint and a substantial improvement in both
anterior-posterior and rotational stability when compared to injured knees. Eichhorn
has demonstrated improved tunnel position, with precision to within 1.6 mm on the
tibia and 2 mm on the femur; and a more anatomic tibial and femoral tunnel
placement at 1:30/10:30 clock face position. Picard and the Pittsburgh group (6)
demonstrated on foam knees that the manual instrumentation had errors on the tibia
of 4.9 mm and femur of 4.2 mm that decreased to 3.4 mm and 2.7 mm. Plaweski (7)
showed radiographic impingement decreased from 75% to 0% with navigation, and that
96.7% have less than 2 mm translation postoperatively, compared to 83% with manual
tools.
RECOMMENDED READING
Koh JL. Computer-assisted navigation and anterior cruciate ligament reconstruction:
accuracy and outcomes. Orthopaedics. 2005; 28(10)(suppl):S1283-1287.
Koh JL, Koo SS, Leonard J, Kodali P. Anterior cruciate ligament (ACL) tunnel
placement: a radiographic comparison between navigated versus manual ACL
reconstruction. Orthopaedics. 2006;29(10)(suppl):S122-124.
Picard F, DiGioia AM, Moody J, et al. Accuracy in tunnel placement for ACL
reconstruction. Comp Aid Surg. 2001;6(5):279-289.
Plaweski S, Cazal J, Rosell P, et al. Anterior cruciate ligament reconstruction using

navigation. Am J Sports Med. 2006;34(4):542-552.
REFERENCES
1. Eichhorn H-J. Image-free navigation in ACL replacement with the OrthoPilot
System. In: Stiehl JB, Konermann WH, Haaker RG, eds. Navigation and robotics in
total joint and spine surgery. Berlin: Springer-Verlag; 387-396.
2. Eichhorn J. Three years of experience with computer-assisted navigation in

anterior cruciate ligament replacement.
http://www.aclstudygroup.com/Powerpoint-pdf02/Eichhorn.pdf.
3. Eichhorn J. Three years of experience with computer navigation-assisted

positioning of drilling tunnels in anterior cruciate ligament replacement (SS-67).
Arthroscopy. 2004;20(5)(suppl):31-32.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 26 - Arthroscopic
Chondroplasty and/or Debridement
26
Arthroscopic Chondroplasty and/or
Debridement
Douglas W. Jackson
Chondroplasty is defined as the â€œsurgical shaping of cartilage.â€• This shaping
includes contouring and removing devitalized or detached cartilage fragments. The
debridement of loose or fragmented articular cartilage may relieve mechanical
symptoms associated with the diseased articular surfaces.
Chondroplasty of the knee is usually always part of an overall debridement procedure,

and at the same time is an associated joint lavage and irrigation. This washout
removes small particulate debris and bioactive molecules that may include
inflammatory mediators and enzymes. Although the natural
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history of the arthritic process in the knee joint is probably unaffected by arthroscopic
chondroplasty, debridement, and lavage, in selected patients the procedure may
reduce or eliminate certain mechanical symptoms.
A definition for arthroscopic debridement of the knee when used in the treatment of
degenerative articular cartilage disease (degenerative arthritis) has not been defined
in the literature. Therapeutic arthroscopic techniques for knee joint osteoarthritis
include debridement and/or chondroplasty, partial meniscectomy, osteophyte
excision, joint lavage, and loose-body removal. At the time of the arthroscopic
assessment, in addition to a chondroplasty and debridement of the articular surfaces,
the surgeon may also address exposed subchondral bone.
Several of these associated or concomitantly performed procedures are described in

other chapters in this book, including the microfracture technique to stimulate
localized repair over the exposed boney surface (Chapter 27), chondrocyte
transplantation (Chapter 29), osteochondral autografts (Chapter 28), osteochondral
allografts (Chapter 33) and associated osteotomy (Chapters 24, 30, and 31).
The indications for considering arthroscopic chondroplasty and debridement are

primarily for mechanical symptoms in the knee that are felt to be secondary to
articular cartilage fragmentation and loosening, loose bodies, meniscal fragments, and
tears and impingements. These symptoms may include localized joint pain with
specific rotation and/or flexion, catching or locking, and recurrent effusions
associated with the shedding of articulate cartilage debris and fragments. These
symptoms should be interfering with daily activities and adversely affecting the
patient's quality of life. The symptoms have not been responsive or relieved by
nonsurgical treatments, including activity modification, anti-inflammatory medication,
and/or glucosamine sulfate, viscosupplementation, corticosteroid injection (with or
without aspiration), physical therapy, stress off-loading with bracing or orthotics, and
activity restriction.
The long-term management of degenerative articular disease involves patient

education and weight loss (if appropriate). The photos (images) of patient's articular
surfaces taken at the time of chondroplasty (debridement) can be used to reinforce
the need to lose weight, build and maintain lower extremity strength, and proactively
select nonaggravating activities and exercises.
While arthroscopic chondroplasty and debridement can provide palliative treatment to

reduce symptoms from knee osteoarthritis in carefully selected individuals, it is often
difficult to predict which patients will definitely benefit. The patient and surgeon
should have realistic expectations regarding its possible outcome. The duration of
symptomatic relief following chondroplasty is variable and is influenced by the natural
history and predisposing factors of the individual's underlying articular cartilage
disease. This is further impacted by the patient's future use of the knee and
subsequent aggravating injuries. Symptomatic improvement is less likely in the
presence of more advanced articular cartilage disease (articulating exposed bone
particularly on opposing sides of the joint), altered subchondral bone, extremity
malalignment, and obesity. Those patients with more advanced articular cartilage
degeneration can on occasion experience worsening of their preoperative symptoms
following this procedure.
While imaging techniques for articular cartilage lesions are improving, arthroscopy
remains the best method for assessing the grade, size, and location of degenerative
articular cartilage changes in the knee joint. Most patients presenting for an
orthopaedic surgeon's evaluation with symptomatic articular cartilage disease have
mild to moderate osteoarthritis of the knee and are between 40 and 70 years of age.
They usually are coming because of the sudden onset or significant change in their
mechanical symptoms and/or effusion. Their assessment begins with a history and
physical examination. The duration and severity of symptoms should be established.
The onset of the pain related to symptomatic degenerative articular cartilage disease
may be insidious; it may develop following an aggravating activity or be associated
with a specific traumatic event. The patients who have a more sudden onset tend to
respond better to arthroscopic chondroplasty and debridement. In addition to their
pain, common symptoms include grinding, clicking, and catching. The mechanical
symptoms may be related to loose fragments of cartilage and/or their interaction with
degenerative meniscal tissue. Patients may describe a feeling of tightness and/or a
sense of swelling related to intra-articular effusions after aggravating activities. The
effusion often contributes to their difficulty when squatting or kneeling.
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The patient's discomfort may be exacerbated by twisting motions of the knee, when
squatting and turning over in bed at night. The pain is often increased with prolonged
standing, walking, or running. The symptoms are usually relieved by rest and
restrictions in motion of the knee. The severity of the associated pain may escalate
with certain activities and motion and interfere with activities of daily living; night
pain can interrupt sleep.
Physical examination includes documentation of the standing lower-extremity

alignment in both coronal and sagittal planes. An associated genu varum malalignment
is a more common deformity in symptomatic osteoarthritic knees than those with
abnormal genu valgus. The presence of a fixed flexion deformity is not unusual in
those patients with more advanced joint involvement.
An effusion may be palpable, and the affected joint may feel warmer in the presence
of an underlying inflammatory process. The ranges of active and passive motion of the
knee without effusion are usually within normal limits in the early stages of articular
cartilage disease. Localized joint-line pain may be present on palpation, and the
McMurray test may elicit some discomfort in the involved compartment. Associated
degenerative meniscal tears and articular cartilage disease are commonly associated
with older patients. The knee is usually stable to ligament evaluation in most patients,
but excluding underlying ligamentous instability is an important part of the
examination and future management.
Aspiration of the joint, with appropriate synovial fluid analysis, should be considered
when an effusion is a persistent finding. The shedding of particulate debris from
degenerative articular surface(s), and its subsequent digestion and removal, may
evoke an inflammatory response within the joint. The synovial aspiration may show
particulate debris on gross examination if the needle bore for aspiration is of adequate
size. (I prefer an 18-gauge needle for aspirations of symptomatic synovial effusions).
Crystal analysis (gout and pseudogout), cell count, and other synovial fluid studies
should be performed when an aspirate is sent for laboratory evaluation.
In assessing the patient's complaints, it is helpful to establish clinically whether the

medial, lateral, or anterior compartment(s) of the knee are involved. Is the problem
related to predominantly one compartment, or is a tricompartment problem
contributing to the patient's disability? Weightbearing radiographs are useful adjuncts
to supplement the clinical findings. They document leg alignment and the extent of
articular cartilage interval narrowing, subchondral bone changes, and osteophyte
formation.
Radiographic imaging of knees with chondral disease should include weight-bearing

views. The joint space is best evaluated with a posteroanterior (PA) view, taken of the
weight-bearing knees in 20 to 30 degrees of flexion. These PA flexion views may give
narrowing changes on weight-bearing views that are not apparent on the same knee
fully extended. They document the most frequent region of earlier joint space
narrowing (i.e., the middle one third of the femoral condyle) (Fig. 26-1). Radiologic
features such as sharpening of the tibial spines, squaring of the joint margins,
subchondral sclerosis, and cyst formation may be associated with more advanced
degeneration. Tibiofemoral alignment can be assessed on the weight-bearing views of
the knee, but a full-lengthextremity film may be required to assess the mechanical
axis (if thought to be needed). The greater the varus or valgus deformity and the
closer to bone on bone articulation, the less likely the arthroscopic chondroplasty will
provide symptomatic relief. The reader is referred to the accompanying chapters on
osteotomy, which discuss treatment options for these patients.
Magnetic resonance imaging (MRI) is a useful noninvasive means that may help
demonstrate symptomatic articular cartilage lesions. There is often a high correlation
with radiographs, but MRI may be particularly helpful when standard radiographs
appear normal. A wide variety of MRI pulse sequences have been investigated with
regard to their utility in assessment of cartilage image. Among the most useful are fat
suppressed proton density (FSPD) and T2-weighted sequences (FST2). A number of
gradient recalled sequences continue to be used although no clear consensus on the
best pulse sequence has emerged. An example of a MRI grading system is shown in
Table 26-1. Current techniques are more accurate in detecting partial or full-thickness
cartilage loss (grades III or IV). Experience suggests that MRI and arthroscopic findings
may not always correlate. A clinically practical and reproducible system of staging
cartilage loss using MRI on a wide scale has yet to emerge.
Nevertheless, MRI is excellent for demonstrating the integrity of the meniscus in the
degenerative compartment being evaluated. Magnetic resonance imaging is also
capable of providing information on the condition of the underlying subchondral bone
and adjacent marrow space (Fig. 26-2).
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FIGURE 26-1
Radiographs of a single knee. Weight-bearing posteroanterior view with knee

flexion (A) illustrates complete loss of joint space in the lateral compartment,
whereas the weightbearing view with the knee in extension (B) suggests the
presence of a reasonable joint space.
A radionuclide bone scan may provide additional information in those patients whose
pain is out of proportion to the objective findings. As an indicator of increased blood
flow, radiotracer uptake may be localized to a single joint surface or compartment. A
more generalized uptake may indicate more advanced changes, or even at times an
alternate pathology. Recent advances in nuclear medicine techniques include the use
of tomographic techniques (SPECT imaging) in conjunction with performing the bone
scan. This technique offers increased sensitivity for lesion depiction compared with
conventional planar nuclear imaging methods.
The MRI and/or bone scan can be very helpful in the diagnosis of avascular necrosis
and its variants of localized bone involvement. Its presence should be explained
preoperatively to the patient. Some of these patients may eventually need partial or
total knee replacement regardless of what is done. The natural history as best it is
known should be explained to the patient, giving the patient the option of additional
watchful waiting with possible limited weight bearing.
TABLE 26-1. Grading of Cartilage Lesions
Grade MRI Arthroscopy
I Signal heterogeneity Softening
II Fissuring Fraying or fissuring
III Partial thickness loss Partial thickness loss
IV Complete loss of cartilage Full thickness loss (exposed bone)
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FIGURE 26-2
MRI images demonstrating chondropathy. Articular cartilage thinning over the

middle third of the medial femoral condyle is shown on a coronal image (A). The
sagittal image (B) shows changes in the subchondral bone with articular cartilage
breakdown.
Eighty-five percent of early symptomatic chondral lesions occur in the medial

tibiofemoral compartment. Chondral lesions are most commonly encountered during
arthroscopy on the femoral side of the joint, and less commonly cartilage changes are
noted on the opposing tibial surface. When there is disruption of the chondral surface
(grade IV) encountered during arthroscopy, an associated degenerate meniscal tear in
the involved compartment is present at least 70% of the time.
Whenever arthroscopy of the knee is performed, particularly in middle-aged and older

patients, the surgeon should be prepared to perform some type of arthroscopic
chondroplasty and/or debridement. This includes the debridement and/or removal of
loose, undermined, and partially detached articular cartilage, lavage out articular
cartilage debris, and excise abnormally mobile and/or degenerative meniscus
fragments leaving smoothly contoured and stable rims. The lavage provided at the
time of arthroscopy is a method of irrigating and removing the smaller particulate
intra-articular cartilaginous debris and degradative enzymes present in the joint.
The informed and written consent obtained before surgery should explain the
possibility of this additional procedure(s), risks, rehabilitation, and prognosis. We try
to give a clear understanding to each patient of the possibility of the debridement of a
potentially symptomatic articular cartilage. This includes an explanation of
degenerative cartilage disease and the objectives of a chondroplasty in trying to
reduce certain specific symptoms. Patients are also informed that there is a small
chance that their knee may be made more symptomatic following the procedure. In
our patients we think this risk is less than 5%. The poor results that are seen following
arthroscopic debridement are more often in those knees with advanced degenerative
disease. This is common when there are articulating bone-on-bone surfaces, significant
malalignment, obesity, and in those patients with secondary gain factors (legal or
industrial injury) related to their symptoms.
Since there is a possibility of performing the microfracture technique in association

with the chondroplasty, this should be discussed, particularly in relation to the
postoperative course. See Chapter 27 on the indications and details of microfracture.
Surgeons should be prepared to deal with fragmented and loose articular cartilage as
well as exposed bone.
SURGERY
On the day of surgery, the â€œcorrectâ€• limb is confirmed and marked. The patient's
medical history should be reviewed for allergies to drugs that may be used during and
after surgery. We seldom inflate a tourniquet during this procedure; but if is to be
used, special attention should be made regarding patients with underlying vascular
disease, vascular grafts, or previous deep vein thrombosis.
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The patient is placed on the operating table in a supine position, and the correct limb
is once again confirmed. General, regional, or local anesthesia can be used, depending
on patient and surgeon preference. If local anesthesia is used, we prefer an
anesthesiologist present to provide sedation if necessary and additional pain relief and
muscle relaxation if needed. If the patient prefers to observe the procedure on the
television monitor, sedation is minimized or eliminated.
An examination of the knee is routinely performed at the beginning of the case before
anesthesia is complete to document the preoperative passive range of motion and
ligamentous stability in the affected and contralateral knee. A tourniquet is applied
proximally on the thigh but is only inflated during the case if intra-articular bleeding
slows the progress of the surgical procedure. If this circumstance occurs during the
case, we inflate the tourniquet to a pressure of 300 mm Hg after elevating the leg for
60 seconds. We prepare and drape the leg in the usual orthopaedic manner using
iodine solution and impervious arthroscopy drapes. The foot and entire lower
extremity are placed in a sterile impervious stocking. An adhesive elastic wrap is used
to contour the stocking to the leg and seal the opening below the knee to prevent fluid
accumulation within the sterile stocking.
The operating room and equipment are arranged to facilitate the surgical procedure.
We prefer to stand beside the affected limb and view the TV monitor placed on the
opposite side of the operating table. This allows a straight, unobstructed view of the
arthroscopic image for the surgeon. The surgical assistant, if present, stands beside
the patient's waist to assist with limb positioning. The presence of skilled and
experienced operating room personnel is invaluable.
We prefer to use gravity-flow irrigation to maintain knee joint distension and fluid
pressure throughout the procedure, and we do not use an automated (pump) inflow
system. The circulating nurse has the role of obtaining additional instrumentation as
needed, overseeing the fluid delivery and suction system, and facilitating the
documentation procedure. The relevant arthroscopic findings are documented with
photographic prints and hand drawn on a specific form at the conclusion of the case.
Both of these are included as a part of the operative record in our office charts. The
grading and description of articular cartilage lesions are helpful in the future
management of the patient's symptoms. The improved technology in the integrated
operating suites, digital imaging and computerized documentation, can be used to
enhance patient education, medical and operative records, and report generation and
billing (Fig. 26-3).
FIGURE 26-3
The new integrated operating rooms enable enhanced digital documentation for
the medical records, operative report, patient education, coding, billing,
transmission to an off-site office, and for use in education forums.
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Technique
The same systematic approach is used for all our knee arthroscopies. After the lower
extremity has been prepared and draped in the usual orthopaedic manner, the desired
portals are infiltrated with a solution containing equal amounts of Xylocaine (1%) and
Marcaine (0.25%), with epinephrine (1:200,000) to minimize portal site bleeding and
reduce postoperative pain.
An anterolateral portal is created for the placement of the arthroscope. With the knee
flexed in a 90-degree position, the portal site is identified as a â€œsoft spotâ€•
located at the lateral border of the patella tendon, one fingerbreadth above the
lateral tibial plateau. The skin is incised with a No. 11 scalpel blade. My preference is
to make a puncture that is oriented vertically and directed centrally toward the
intercondylar notch as it penetrates the joint capsule. A blunt trochar is then used to
pass the arthroscopic cannula into the joint. Having penetrated the joint capsule, the
trochar is carefully inserted into the suprapatellar pouch, while extending the knee.
Gently inserting a blunt trochar with the knee in extension minimizes the risk of
damaging the patellar, trochlear, and condylar articular surfaces. The camera and
light source are attached to the arthroscope, and the camera image is â€œwhite
balancedâ€• before placing it into the sheath.
The 30-degree (4-mm) arthroscope is inserted through the high-flow cannula placed in
the anterolateral portal. The inflow tubing is then attached to the cannula, and the
joint is distended with saline. The caliber of the cannula is large enough to allow
sufficient irrigation during the procedure. Intra-articular placement of the scope is
confirmed visually on screen. An outflow tubing is attached to the cannula's second tap
to allow suction drainage as required. Initial joint lavage may be required to disperse
intra-articular bleeding so that a clear image is visible on the TV monitor. The amount
of intra-articular cartilaginous debris present during initial washout is noted (Fig. 26-
4). A more extensive lavage can be achieved by introducing a large bore in flow
cannula or suction via an anteromedial or superior portal. We do not hesitate to create
additional portals, including posterior medially, if necessary to facilitate adequate
visualization, irrigation, and removal of loose bodies and/or fragments.
Inspection
Once the arthroscope position and image are satisfactory, a diagnostic arthroscopy is
performed. This involves a complete inspection of the knee joint, made in a sequential
and systematic fashion. Each compartment is evaluated, and the status of the articular
cartilage on both sides of the joint compartment; the integrity of the meniscus,
anterior cruciate ligament (ACL), and posterior cruciate ligament (PCL); the condition
of the synovium; and the presence of osteophytes are documented.
We begin with inspection in the suprapatellar pouch, noting any synovial changes,
plicae, intra-articular debris, or loose bodies. The articular surfaces of the
patellofemoral joint are inspected with the knee in full extension. Each facet of the
patella is viewed, and the contour of the femoral sulcus
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is followed downward to the intercondylar notch. Special attention is directed to the
trochlear groove and the tracking of the patella during 40 degrees of knee flexion and
extension. The presence of enlarged or impinging synovial plica is evaluated before
passing the arthroscope into the medial gutter, and the presence of osteophytes and
articular debris is documented (Fig. 26-5).
FIGURE 26-4
Initial drainage of knee joint effusion containing particulate debris.
FIGURE 26-5
Particulate debris located in medial gutter.
The arthroscope is then passed into the medial compartment to assess the articular
surfaces of the medial femoral condyle and tibial plateau. Inspection of the medial
meniscus is also undertaken. To visualize the entire articulating surface of the femoral
condyle, the knee is observed as it is moved from extension into flexion. Maximum
knee flexion affords the best visualization of the posterior third of the condylar
articular surface. A gentle valgus force, generated by controlled manual pressure, is
usually required to view the posterior horn of the medial meniscus adequately. This
opening of the medial joint space can be facilitated by a leg holder, lateral post,
and/or knowledgeable assistant.
Inspection of the intercondylar notch is carried out with the knee in a flexed position.
The ACL and PCL are observed, and the presence of notch osteophytes is noted. Once
the structures of the intercondylar notch have been visualized, the scope is carefully
passed into the posteromedial compartment of the knee to ensure there are no loose
bodies. The integrity of the posterior attachment of the medial meniscus and status of
the synovium are also assessed.
The arthroscope is then directed across the notch to inspect the lateral compartment
and gutter. To improve visualization of the lateral compartment, the knee is moved
into the figure-4 position. With the knee at 90 degrees or less, the ankle can be held
or rested on the contralateral tibia. Under its own weight, the leg falls outward,
externally rotating and abducting the femur, creating a varus force that opens up the
lateral joint. In this position, the articular surfaces and lateral meniscus are inspected
by rotating the arthroscope to take advantage of the 30-degree lens. Finally, the
lateral joint margin and its gutter are inspected for osteophytes, debris, and loose
bodies.
Intra-articular Probing and Palpation

The purpose of initial visual inspection is to identify areas of pathology within the
joint. However, a diagnostic arthroscopy is not complete unless the intra-articular
structures are palpated and probed. We prefer to use a blunt nerve hook as the
probing instrument to assess articular cartilage defects and the mobility of meniscal
fragments or tears.
The probe is usually inserted through the second portal, most frequently created
anteromedially. With the knee in 90 degrees of flexion, the â€œsoft spotâ€• is
palpated adjacent to the medial edge of the ligamentum patella. This location is
confirmed by transillumination with the arthroscope tip positioned at the intra-
articular site of the portal. Precise placement of the portal is enhanced by first using
an 18-gauge spinal needle to determine the position and trajectory of subsequent
instrumentation and its ability to access the target tissue (Fig. 26-6). Surgical
instruments should be as parallel to the tibial plateau as possible. To be certain that
the meniscus is not damaged when creating the portal, the lens of the arthroscope is
rotated to view the position of the penetrating knife blade. A No. 11 blade is used to
create a vertically oriented portal. By creating these surgical portals under direct
arthroscopic visualization, injury to the menisci can be avoided, and the most
advantageous angles of approach with the instrumentation can be achieved.
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FIGURE 26-6
An 18-gauge spinal needle is used for selective portal placement. Operative

portals are best created under direct arthroscopic vision.
With the nerve hook placed through the anteromedial portal, palpation of the articular
surfaces and the menisci is performed. It is important to characterize and describe the
chondral lesions. The extent of the segments of loose or undermined cartilage may
only become apparent when probed (Fig. 26-7). This process is helpful to grade
chondral pathology more accurately. Each compartment of the knee is palpated. At
times, it may be advantageous to switch portals for the arthroscope and nerve hook to
improve visualization and instrumentation.
Documentation
Depending on the surgeon's preference and available equipment, documentation can
be in the form of written text, diagrams, arthroscopic photography, videotape, or
digital image recording. Articular cartilage lesions are recorded in terms of
grade/stage, size, and location. The size of the lesion is measured for length and
width. Using the 5-mm tip of the nerve hook probe may be helpful as a sizing tool (Fig.
26-8). We grade chondral lesions according to the Outerbridge classification I to IV
(Figs. 26-9, 26-10, 26-11 and 26-12), although numerous other grading systems exist.
We prefer to record the data using a diagram and photography. To ensure
documentation is complete, all intra-articular structures are recorded in a systematic
fashion on our postoperative data sheet.
FIGURE 26-7
A blunt hook is used to palpate undermined cartilage.
FIGURE 26-8
Probe may be used to size chondral lesions.
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FIGURE 26-9
Outerbridge grade I lesion. Arthroscopic views (A,B) and diagrammatic

representation (C) with probe demonstrating articular cartilage softening.
FIGURE 26-10
Outerbridge grade II lesion. Arthroscopic view (A) and schematic diagram (B)
demonstrating cartilage fibrillation.
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FIGURE 26-11
Outerbridge grade III lesion. Arthroscopic view (A) and diagram (B) showing
partial-thickness cartilage loss.
FIGURE 26-12
Outerbridge grade IV lesion. Arthroscopic view (A) and diagram (B) showing full-
thickness loss of articular cartilage and exposed underlying bone.
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Chondroplasty and Debridement

Using a combination of manual and powered arthroscopic tools, the surgical
techniques that may be applicable to osteoarthrosis include the following:
Lavage of intra-articular debris

Loose body removal
Partial meniscectomy
Debridement of loose and fragmented articular cartilage
Shaving and contouring of detaching cartilage surfaces
Occasional selective excision of osteophyte(s)
Limited synovectomy
Microfracture of selected grade IV exposed bone (see Chapter 27)
Following the diagnostic palpation with the nerve hook, if indicated the debridement
of chondral lesions is undertaken. The goal is to excise loose articular cartilage and
remove that represent undermined lesions. Larger fragments may be detached using a
hand-held punch, cutting forceps, or curette. Smaller partial-thickness lesions of the
condyle are smoothed with a power shaver. A nonaggressive tip has less risk of causing
unwanted damage to healthy cartilage. The tip is glided across the contoured surface
of the condyle, with the cutting surface at right angles to the articular surface (Fig.
26-13). It should be kept under direct visualization. With suction applied to the power
tool, the debrided cartilage will be sucked into the resector tip. Care is always taken
to avoid damage to healthy, normal-appearing articular cartilage.
There is usually a gradual transition from normal to abnormal articular cartilage in

degenerative knee joints. If, however, there is an abrupt transition between the
diseased and normal areas, the ridge separating the two zones is tapered with the
power shaver or curette. The treated area is reprobed to ensure there are no
remaining undermined flaps of loose articular cartilage.
In regions where the underlying subchondral bone is exposed, the procedure is the
same, and then the surgeon decides if in addition to debridement, marrow-stimulation
techniques may be employed. The exposed bone of the grade IV lesion may be
penetrated using microfracture. This technique penetrates the subchondral bone,
inducing clot formation and the potential migration of marrow cells. If a healing
response is initiated, the defect is repaired with fibrocartilage scar tissue, which has
inferior structural and functional properties to hyaline cartilage. The reader is
referred to Chapter 27, which covers the microfracture technique in detail.
Joint margin osteophytes can occasionally be the source of localized pain or popping
as soft tissue rolls over them. Impinging osteophytes within the notch or on the tibial
shelf can occasionally contribute to restricting knee extension. Removal of
symptomatic or impinging osteophytes can be done arthroscopically and may include
using a small intra-articular osteotome, curette, or a power tool fitted with an
arthroscopic burr or other aggressive resector tip.
Radiofrequency (RF) energy has also been used to ablate diseased articular cartilage,
in place of the usual mechanical shaving methods for debridement. A variety of
monopolar and bipolar probes are available to deliver the thermal energy, and lower
RF settings appear to inflict less thermal damage. We do not use it on articular
cartilage. Studies on the effect of RF have shown the potential for deleterious
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alterations in adjacent chondrocytes, cartilaginous matrix, and even the subchondral
bone. Caution is advised until definitive guidelines emerge for the application of RF
electrodes in thermal-assisted chondroplasty. Currently, we prefer mechanical
techniques for articular cartilage debridement.
FIGURE 26-13
Arthroscopic power-shaver with nonaggressive resector tip glides over condylar

surface, excising loose fibrillated cartilage drawn into its jaws with suction.
FIGURE 26-14
Arthroscopic view of a basket forceps making the initial cut across the anterior
base of a flap tear of the meniscus.
FIGURE 26-15
Powered meniscal resector contours stable meniscal rim.
Dealing with symptomatic degenerative meniscus pathology is an important part of the

arthroscopic treatment of knee osteoarthritis. The meniscal tears found in association
with chondral disease often have associated degenerative flap tears of the posterior
and/or middle one third of the meniscus. Debridement of mobile meniscus fragments
or flap-based resection is performed using basket forceps (Fig. 26-14) and/or power
instruments with suction (Fig. 26-15). The remaining unstable rim is then trimmed and
balanced with a basket forceps or power shaver (Fig. 26-16). A probe can be used at
this point to test for stability of the remaining meniscal rim and to confirm there are
no undersurface flaps or residual irregularities of the meniscus. While attending to
meniscal pathology, care must be taken not to inflict damage to adjacent articular
surfaces. Often degenerative meniscal and cartilage lesions are present in the same
compartments.
The techniques of arthroscopic debridement may be used in all three compartments of
the knee. During patellofemoral chondroplasty, instrumentation through the standard
portals may not be satisfactory to gain access to the involved chondral surface. To
facilitate surgical access in these cases, it may be necessary to create additional
portals into the suprapatellar pouch (Fig. 26-17). Superior
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portals can be placed medially or laterally, and the trajectory for instrumentation
through a portal can be determined using an 18-gauge needle technique to confirm
access to the desired surface. This additional portal facilitates the application of the
techniques of arthroscopic chondroplasty to the femoral trochlea and undersurface of
the patella (Figs. 26-18 and 26-19).
FIGURE 26-16
Completed partial meniscectomy and chondroplasty.

FIGURE 26-17
Suprapatellar portal for better access to involved articular cartilage in the

patellofemoral joint.
FIGURE 26-18
Arthroscopic view of frayed patellar articular cartilage.

FIGURE 26-19
Patella chondroplasty.
A final inspection of the knee joint is made to seek residual loose fragments of
menisci, intra-articular debris, and loose pieces of articular cartilage. Particular
attention is paid to joint recesses where debris may collect, namely, the suprapatellar
pouch, medial and lateral gutters, the popliteus tendon sheath, and the posteromedial
and posterolateral compartments. Larger loose bodies may require extraction using a
grasping forceps, and the exit portal may require enlargement, depending on the
fragment's size. If indicated, synovial biopsy or partial synovectomy may be
performed.
Lavage
Joint lavage is a part of arthroscopic chondroplasty and debridement. The same
irrigation fluid that distends the joint flushes out the smaller debris generated during
the debridement process. Frequently, 3 to 4 L of irrigation fluid are used during a
routine arthroscopic chondroplasty and debridement. In addition to the clearance of
particulate intra-articular debris, irrigation can wash out inflammatory mediators and
enzymes.
When arthroscopic instrumentation has been completed, the fluid inflow is turned off.
The remaining intra-articular fluid is flushed out with any residual debris (Fig. 26-20).
If desired, the fluid inflow is turned back on and the cannula is inserted into another
compartment to repeat the final irrigation process. Before removing the cannula at
the end of the procedure, we prefer that all the fluid is drained from the joint. To aid
postoperative analgesia, we inject the knee with a 25-mL solution containing a long-
acting local anesthetic (bupivacaine, 0.25%) and morphine (Duramorph, 5 mg). We
close each portal with an interrupted suture. Band-Aids and gauze dressing are placed
over the wounds. A long leg support hose secures the dressing in place and applies
pressure to the knee (Fig. 26-21).
Pearls and Pitfalls
My experience has been that up to 5% of patients who undergo chondral

debridement describe increased knee symptoms following the procedure. It is
important to discuss the possibility of increased symptoms postoperatively with
the patient before surgery to avoid any patient surprise. This is particularly
true in patients with malalignment who have areas of bone on bone
articulation. Often these patients should have had a knee replacement;
instead, the surgeon performed the debridement in hopes of prolonging the use
of the knee before the need for replacement.
One area to be cautious of in extensive debriding is the femoral sulcus that has
a large area of loosening and degenerating articular cartilage. These lesions
are often an incidental finding or have been mildly symptomatic. These
patients may have a sudden increase in symptoms when their degenerating
articular cartilage in the femoral sulcus is aggressively debrided. On occasion,
following
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extensive debridement of the femoral sulcus articular cartilage, the patella
can start mechanically giving a catching or giving-way sensation to some of
these patients.
Be alert for those patients who have knee symptoms related to the early
phases of osteonecrosis or avascular changes in the subchondral bone. These
can usually be identified by a preoperative MRI. I see patients every year who
have an MRI after an arthroscopic debridement and feel their MRI changes of
osteonecrosis and increasing symptoms were caused by their arthroscopic
debridement.
FIGURE 26-20
Lavage: pressurized distension fluid flushes out the debris through the cannula
into a sterile basin.
FIGURE 26-21
Long-leg support hose to maintain pressure and hold dressing in place.
Patients undergoing arthroscopic chondroplasty and debridement are treated on an
outpatient basis. Elevation and cooling of the knee are provided in the recovery room,
and the patient is instructed in the use of crutches with partial weight bearing on the
affected limb. Nonnarcotic oral analgesia is all that is usually required following this
procedure. At discharge, the patient is given an instruction sheet explaining the
postoperative protocol, with specific mention of symptoms suggestive of postoperative
complications.
Patients are advised to elevate and intermittently ice the knee during the first 48
hours postoperatively. They may change the Band-Aid dressings on the day after
surgery, and showering of the extremity is permitted after 48 hours. At 1 week most
patients are able to ambulate without crutches, and weight bearing is allowed as
tolerated. We suggest crutches and partial weight bearing for those who have
persistent effusions. The first postoperative office visit is scheduled 3 to 5 days
following surgery. A wound check is performed and sutures are removed. We explain
the surgical findings and treatment, and discuss the prognosis.
Rehabilitation
Our patients begin exercises at home without supervision following surgery. Printed
instructions are provided outlining the techniques of isometric and limited-arc
quadriceps- and hamstring-strengthening exercises. Range of motion is gradually
increased, aiming to achieve full active extension and flexion within 1 to 2 weeks. At
this stage, the patient is allowed to weight bear as tolerated on the operated limb.
Should an effusion persist and prevent active range-of-motion exercises, aspiration of
the joint may be performed. The patient is instructed to return to crutch walking for
any distance outdoors, until the joint effusion abates.
Once the acute reaction to surgery subsides, patients are encouraged to begin
bicycling, swimming, or water aerobics. For most patients, a home program of
unsupervised rehabilitation is sufficient for recovery, provided that the surgeon
monitors the patient's progress. Those patients lacking either the motivation or the
facility to rehabilitate without supervision are referred to a rehabilitation therapist for
a supervised program. Rehabilitation for this group of patients differs from the
aggressive
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approach offered to the young athlete. It involves progressive resistance exercises and
may need to be restricted to limited-arc exercises, rather than using full-arc
strengthening programs. The importance of maintaining body weight within reason,
general conditioning, and strength of the leg musculature on a regular basis has been
stressed preoperatively and is stressed again before the patient is discharged from
care. Patients are advised to avoid jogging as their form of endurance exercising and
preferably substituting activities that reduce axial and impact loading of the knee
joint.
COMPLICATIONS
The morbidity of arthroscopic surgery is low. Complications such as hemarthrosis (2%),
deep venous thrombosis (0.5%), and septic arthritis (0.1%) require early detection and
intervention. Cutaneous nerve injury can occur during portal skin incision, resulting in
a small area of postoperative paresthesia. Sinus formation at the portal site is
uncommon, and significant vascular injury during arthroscopic chondroplasty is
unusual. Patients need to have realistic expectations and know that 30% to 40% of
patients may have no improvement in their symptoms.
RESULTS
The literature is lacking prospective and/or randomized studies that apply defined
techniques of arthroscopic chondroplasty and debridement with long-term follow-up.
There are many retrospective articles that report patient benefits. When performed
for the indications outlined, I tell our patients that arthroscopic debridement and
lavage may provide symptomatic improvement 60% to 70% of the time. The
postoperative result may take as long as 3 months to be apparent, and the relief of
pain and stiffness can last 5 years or more. Approximately 30% to 40% of my patients
have no change in preoperative symptoms and may require further surgery or choose
to live with their disability until it becomes worse. A small percentage of patients (5%)
report they are more symptomatic following a chondroplasty and debridement.
Several factors determine the prognosis after arthroscopic chondroplasty. Those

patients who benefit the most present with mechanical symptoms of shorter duration
and do not have pronounced angular deformity of their lower extremity, with only
mild to moderate radiographic evidence of osteoarthritis.
Arthroscopic lavage and debridement can be a successful temporizing treatment for

the symptomatic osteoarthritic knee. The natural history of the arthritic process is
probably not altered, and it is important to counsel patients that the goal of surgery is
to reduce symptoms in the knee joint during non-aggravating activities.
RECOMMENDED READING
Browne JE, Branch TP. Surgical alternatives for the treatment of articular cartilage
lesions. J Am Acad Othrop Surg. 2000;8:180-189.
Ewing JW. Arthroscopic treatment of degenerative meniscal lesions and early

degenerative arthritis of the knee. In: Ewing JW, ed. Articular cartilage and knee joint
function: basic science and the arthroscope. New York: Raven Press; 1990:137-145.
Hanssen AD, Stuart MJ, Scott RD, et al. Surgical options for the middle-aged patient
with osteoarthritis of the knee joint. J Bone Joint Surg. 2000;82-A:1768-1781.
Harwin SF. Arthroscopic debridement for osteoarthritis of the knee: predictors of

patient satisfaction. Arthroscopy. 1999;15:142-146.
Jackson DW, Simon TM, Aberman HA. Symptomatic articular cartilage degeneration:
the impact in the new millennium. Clin Orthop Relat Res. 2001;391S:S14-S25.
Jackson RW. Arthroscopic surgery and a new classification system. Am J Knee Surg.
1998;11:51-54.
Jackson RW, Dieterichs C. The results of arthroscopic lavage and debridement of

osteoarthritic knees based on the severity of degeneration: a 4- to 6-year follow-up.
Arthroscopy. 2003;19:13-20.
Lu Y, Edwards RB, Cole BJ, et al. Thermal chondroplasty with radiofrequency energy:
an in vitro comparison of bipolar and monopolar radiofrequency devices. Am J Sports
Med. 2001;29:42-49.
McCauley TR, Disler DG. Magnetic resonance imaging of articular cartilage of the knee.
J Am Acad Orthop Surg. 2001;9:2-8.
Mosely JB, O'Malley K, Petersen NJ, et al. A controlled trial arthroscopic surgery for
osteoarthritis of the knee. N Engl J Med. 2002;347:81-88.
Puddu G, Cipolla M, Cerulio G, et al. Arthroscopic treatment of flexed arthritic knee in

active middle-aged patients. Knee Surg Sports Traumatol Arthrosc. 1994;2:73-75.
Simon TS, Jackson DW. Articular cartilage: injury pathways and treatment options.
Sports Med Arthrosc Rev. June 2006.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 27 - Microfracture
Technique: Treatment of Full-Thickness Chondral Lesions
27
Microfracture Technique: Treatment of Full-
Thickness Chondral Lesions
J. Richard Steadman
William G. Rodkey
The â€œmicrofractureâ€• technique was developed to enhance chondral resurfacing

by providing a suitable environment for new tissue formation and to take advantage of
the body's own healing potential. Specially designed awls are used to make multiple
perforations, or â€œmicrofractures,â€• into the subchondral bone plate. Microfracture
is our preferred method to treat chondral defects. The senior author (JRS) first
described this technique more than 20 years ago. He has treated more than 3,000
patients with the microfracture procedure.
Indications
Microfracture was designed initially for patients with posttraumatic articular cartilage
lesions of the knee that had progressed to full-thickness chondral defects. The
microfracture technique still is most commonly indicated for full-thickness loss of
articular cartilage in either a weight-bearing area between the femur and tibia or in
an area of contact between the patella and trochlear groove. Unstable cartilage that
overlies the subchondral bone is also an indication for microfracture. If a partial
thickness lesion is probed and the cartilage simply scrapes off down to bone, we
consider this to be a full-thickness lesion. Degenerative joint disease in a knee that
has proper axial alignment is another common indication for microfracture. These
lesions all involve loss of articular cartilage at the bone-cartilage interface.
Patients with acute chondral injuries are treated as soon as practical after the
diagnosis is made, especially if the knee is being treated concurrently for meniscus or
anterior cruciate ligament (ACL) pathology. Patients with chronic or degenerative
chondral lesions often are treated nonoperatively (conservatively) for at least 12
weeks after a suspected chondral lesion is diagnosed clinically. This treatment regimen
includes activity modification, physical therapy, nonsteroidal anti-inflammatory drugs,
viscosupplement injections, and perhaps dietary supplements that may have cartilage-
stimulating properties. If nonoperative treatment is not successful, then surgical
treatment is considered.
No limitations are placed on how large an acute lesion can be and still be considered
suitable for microfracture. We have observed that even very large acute lesions
respond well to microfracture. We
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have noted empirically that traumatic lesions, acute or chronic, less than 400 mm2
tend to respond better to microfracture than those lesions greater than 400 mm2, but
we have not observed this difference to be statistically significant. Treatment of
chronic degenerative lesions is not specifically limited by size, but more emphasis is
placed on proper axial alignment and the presence of global degenerative changes
throughout the knee.
General considerations for use of the microfracture procedure include patient age,
acceptable biomechanical alignment of the knee, the patient's activity level, and the
patient's expectations. If all of these criteria define a patient who could benefit from
chondral resurfacing, then microfracture is considered.
Contraindications
Specific contraindications for microfracture include axial malalignment (as described
following), patients unwilling or unable to follow the required strict and rigorous
rehabilitation protocol, partial thickness defects, and inability to use the opposite leg
for weight bearing during the minimal or non-weight-bearing time. Other specific
contraindications include any systemic immune-mediated disease, disease-induced
arthritis, or cartilage disease. A relative contraindication is for patients older than 65
years because the authors have observed that some patients older than 65 years
experience difficulty with crutch walking and the required rigorous rehabilitation.
Other contraindications to microfracture include global degenerative osteoarthrosis or
if the cartilage surrounding the lesion is too thin to establish a perpendicular rim to
hold the marrow clot. In these advanced degenerative cases, axial malalignment is
often a confounding factor (Table 27-1).
Patients who present with knee pain undergo a thorough physical and orthopaedic
examination. The cartilage lesions can be on the joint surfaces of the femur, tibia,
and/or patella. At times the physical diagnosis can be difficult and elusive, especially
if only an isolated chondral defect is present. Identification of point tenderness over a
femoral condyle or tibial plateau is a useful finding, but in itself is not diagnostic. If
compression of the patella elicits pain, this finding might be indicative of a patellar or
trochlear lesion.
For diagnostic imaging, we use long-standing radiographs as described following to

observe for angular deformity and for joint space narrowing that is often indicative of
loss of articular cartilage. We also obtain standard anteroposterior (AP) and lateral
radiographs of both knees as well weightbearing views with the knees flexed to 30 to
45 degrees. Patellar views are also useful to evaluate the patellofemoral joint.
Magnetic resonance imaging (MRI) that employs newer diagnostic sequences specific
for articular cartilage has become a mainstay of our diagnostic workup of patients with
suspected chondral lesions.
Two methods for radiographic measurement of the biomechanical alignment of the

weight-bearing axis of the knee are used in our facility: (a) the angle made between
the femur and tibia on AP views obtained with the patient standing, and (b) the
weight-bearing mechanical axis drawn from the center of the femoral head to the
center of the tibiotarsal joint on long standing (approx. 51 in/130 cm) radiographs. If
the angle drawn between the tibia and femur is greater than 5 degrees of varus or
valgus compared to the normal knee, this amount of axial malalignment would be a
relative contraindication for microfracture. Preferably the mechanical axis weight-
bearing line should be in the central quarter of the tibial plateau of either the medial
or lateral compartment. If the mechanical axis weight-bearing line falls outside the
central-most quarter of the plateaus, medial or lateral, this weight-bearing shift also
would be a relative contraindication if left uncorrected (Fig. 27-1). In such cases, a
realignment procedure should be included as a part of the overall treatment regimen.
TABLE 27-1. Indications and Contraindications for

Microfracture
Indications Contraindications
Full-thickness defect (grade IV), acute or Partial thickness defects

chronic
Unstable full-thickness lesion Uncorrected axial

malalignment
Degenerative joint disease lesion Unable to commit to

(requires proper knee alignment) rehabilitation protocol
Patient capable of rehabilitation protocol Global degenerative

osteoarthrosis
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FIGURE 27-1
If the mechanical axis weight-bearing line falls outside the central-most quarter
of the plateaus, medial or lateral, this weight-bearing shift also would be a
relative contraindication if left uncorrected.
TECHNIQUE
The patient is placed supine, and routine skin preparation and draping are completed.
Standard arthroscopic portals can be used for microfracture; however, we find that
making the portals slightly higher and slightly further off the midline may provide
additional visualization (Fig. 27-2). We make three portals: a superior and medially
placed inflow portal and medial and lateral parapatellar portals (Fig. 27-3). Accessory
portals may occasionally be made for lesions in difficult-to-reach locations. Typically
we do not use a tourniquet during the microfracture procedure; instead, we adjust the
arthroscopic fluid pump pressure to control bleeding. An initial thorough diagnostic
examination of the knee should be done before beginning the actual microfracture.
We carefully inspect all geographic areas of the knee including the suprapatellar
pouch, the medial and lateral gutters, the patellofemoral joint, the intercondylar
notch and its contents, and the medial and lateral compartments including the
posterior horns of both menisci. We do all other intra-articular procedures before
completing microfracture, with the exception of ACL reconstruction. This sequence
helps prevent loss of visualization when the fat droplets and blood enter the knee from
the marrow cavity via microfracture holes. This technique also decreases the amount
of time that the microfractured bone is exposed to the elevated intra-articular
pressures and fluid flow that possibly could decrease the formation of the surgically
induced marrow clot (â€œsuper clotâ€•) that is critical to the success of the
microfracture procedure. We pay particular attention to soft tissues such as plicae and
the lateral retinaculum that potentially could produce increased compression between
cartilage surfaces.
FIGURE 27-2
Standard arthroscopic portals can be used for microfracture; however, we find

that making the portals slightly higher and slightly further off the midline may
provide additional visualization.
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FIGURE 27-3
We make three portals: a superior and medially placed inflow portal and medial
and lateral parapatellar portals.
After carefully assessing the full-thickness articular cartilage lesion, we debride the
exposed bone of all remaining unstable cartilage using a hand-held curved curette and
a full radius resector. It is critical to remove all loose or marginally attached cartilage
from the surrounding rim of the lesion (Fig. 27-4). Establishment of a stable full-
thickness border of cartilage surrounding a central lesion is optimal for microfracture
to provide some degree of protection to the regenerating tissue that is forming in the
treated lesion. The calcified cartilage layer that remains as a cap to many lesions must
be removed, preferably by using a curette (see Fig. 27-4). Thorough and complete
removal of the calcified cartilage layer is extremely important based on animal studies
we have completed. Care should be taken to maintain the integrity of the subchondral
plate by not curetting too deeply; otherwise, the joint shape and geometry might be
negatively altered. This prepared lesion, with a stable perpendicular edge of healthy
well-attached viable cartilage surrounding the defect, provides a pool that helps hold
the â€œsuper clot.â€• After preparation of the lesion, we use an arthroscopic awl to
make multiple holes, or â€œmicrofractures,â€• in the exposed subchondral bone
plate. We use an awl with an angle that permits the tip to be approximately
perpendicular to the bone as it is advanced, typically 30 or 45 degrees (Fig. 27-5).
There also is a 90-degree awl that should be used only on the patella or
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other soft bone. The 90-degree awl should be advanced only manually, not with a
mallet. The holes are made as close together as possible, but not so close that one
breaks into another, thus damaging the subchondral plate between them and
potentially altering the joint shape. This technique usually results in microfracture
holes that are approximately 3 to 4 mm apart. When fat droplets can be seen coming
from the marrow cavity, the appropriate depth (approx. 2 to 4 mm) has been reached
(Fig. 27-6). The arthroscopic awls likely produce no thermal necrosis of the bone
compared with handdriven or motorized drills. We make microfracture holes around
the periphery of the defect first, immediately adjacent to the healthy stable cartilage
rim (see Figs. 27-5 and 27-6). Then, we complete the process by making the
microfracture holes toward the center of the defect (Fig. 27-7). We assess the treated
lesion at the conclusion of the microfracture to ensure a sufficient number of holes
have been made before reducing the arthroscopic irrigation fluid flow (Fig. 27-8).
FIGURE 27-4
It is critical to remove all loose or marginally attached cartilage from the

surrounding rim of the lesion. The calcified cartilage layer that remains as a cap
to many lesions must be removed, preferably by using a curette.
FIGURE 27-5
We use an awl with an angle that permits the tip to be approximately

perpendicular to the bone as it is advanced, typically 30 or 45 degrees.
FIGURE 27-6
When fat droplets can be seen coming from the marrow cavity, the appropriate
depth (approx. 2 to 4 mm) has been reached. We make microfracture holes
around the periphery of the defect first, immediately adjacent to the healthy
stable cartilage rim.
After the arthroscopic irrigation fluid pump pressure is reduced, under direct
visualization we are able to observe the flow of marrow fat droplets and blood from
the microfracture holes into the prepared lesion (Fig. 27-9). The quantity of marrow
contents flowing into the joint is judged to be adequate when we observe marrow
elements flowing from all microfracture holes (Fig. 27-10). We
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then remove all instruments from the knee and evacuate the joint of fluid. Intra-
articular drains should not be used because the goal is for the super clot rich in
marrow elements to form and to stabilize while covering the lesion.
FIGURE 27-7
We complete the process by making the microfracture holes toward the center of
the defect.
FIGURE 27-8
We assess the treated lesion at the conclusion of the microfracture to ensure a

sufficient number of holes have been made before reducing the arthroscopic
irrigation fluid flow.
FIGURE 27-9
After the arthroscopic irrigation fluid pump pressure is reduced, under direct
visualization we are able to observe the flow of marrow fat droplets and blood
from the microfracture holes into the prepared lesion.
FIGURE 27-10
The quantity of marrow contents flowing into the joint is judged to be adequate
when we observe marrow elements flowing from all microfracture holes.
Chronic degenerative chondral lesions commonly have extensive eburnated bone and
bony sclerosis with thickening of the subchondral plate, thus making it difficult to do
an adequate microfracture procedure. In these instances and when the axial alignment
and other indications for microfracture are met, we first make a few microfracture
holes with the awls in various locations of the lesion to assess the thickness of the
sclerotic or eburnated bone. We often use a motorized burr to remove the sclerotic
bone until punctate bleeding is evident. After the bleeding appears uniformly over the
surface of the lesion, a microfracture procedure can be performed as previously
described. We have observed noticeably improved results for these patients with
chronic chondral lesions since we began using this technique. However, if the
surrounding cartilage is too thin to establish a perpendicular rim to hold and protect
the super clot, then likely we would not do a microfracture procedure in patients with
such advanced degenerative lesions.
The microfracture awl produces a rough surface in the subchondral bone to which the
marrow clot can adhere more easily, yet the integrity of the subchondral plate is
maintained for joint surface shape and geometry (see Fig. 27-8). The microfracture
procedure virtually eliminates thermal necrosis and provides a roughened surface for
blood clot adherence. The different angles of arthroscopic awls available provide
easier access to difficult-to-reach areas of the knee. The awls not only provide
perpendicular holes but also improved control of depth penetration compared to
drilling. We believe that the key to the entire procedure is to establish the surgically
induced marrow clot to provide the optimal environment for the body's own
pluripotential marrow cells (mesenchymal stem cells or progenitor cells) to
differentiate into stable tissue within the lesion.
Pearls and Pitfalls

Complete a thorough arthroscopic diagnostic examination, inspecting all
geographic areas of the knee. Perform all other intra-articular procedures
before completing microfracture, with the exception of ACL reconstruction.
Assess the chondral lesion. Remove all loose or marginally attached cartilage
down to exposed bone.
Thoroughly and completely remove the calcified cartilage layer with a hand
held curette, but do not penetrate the subchondral bone.
Use a microfracture awl to make microfracture holes in the subchondral bone,

first working all the way around the periphery and then into the center of the
lesion.
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Microfracture holes should be about 3 to 4 mm apart so that one does not
break into another, and they should penetrate approximately 2 to 4 mm deep
to access the marrow elements.
Reduce the irrigation pump pressure (or tourniquet) and observe for marrow
element flow from all holes.
Remove all instruments and evacuate the joint. Do not use a drain in the joint.
We have designed the postoperative management program to promote the ideal
physical environment in which the newly recruited mesenchymal stem cells from the
marrow can differentiate into the appropriate articular cartilage-like cell lines. These
differentiation and maturation processes must occur slowly but consistently. Our
animal studies have confirmed that both cellular and molecular changes are an
essential part of the development of a durable repair tissue. Our experience and
clinical research data indicate that improvement can be expected to occur slowly but
steadily for at least 2 years. During this protracted period, the repair tissue matures,
pain and swelling resolve, and the patient regains confidence and comfort in the knee
during increased levels of activity.
The rehabilitation program after microfracture for treatment of chondral defects in

the knee is crucial to optimize the results of the surgery. The surgically induced
marrow clot provides the basis for the most ideal chemical environment to
complement the physical environment. This newly proliferated repair cartilage then
fills the original defect. The postoperative rehabilitation program after microfracture
necessitates consideration of several factors. The specific protocol recommended
depends on both the anatomic location and the size of the defect. These factors are
critical to determine the ideal postoperative plan. For example, if other intra-articular
procedures are done concurrently with microfracture, such as ACL reconstruction, we
will alter the rehabilitation program as necessary. All of the possible variations of the
rehabilitation program are not within the scope of this chapter, but here we describe
two different protocols.
Rehabilitation Protocol for Patients with Lesions on

the Femoral Condyle or Tibial Plateau
After microfracture of lesions on the weight-bearing surfaces of the femoral condyles
or tibial plateaus, we initiate immediate motion with a continuous passive motion
(CPM) machine in the recovery room. The initial range of motion (ROM) typically is 30
to 70 degrees, and then it is increased as tolerated by 10 to 20 degrees, until full
passive ROM is achieved. The rate of the machine is usually 1 cycle per minute
initially, but the rate can be varied based on patient preference and comfort. Many
patients tolerate use of the CPM machine at night. For those who do not, we have
observed that intermittent use during the day is as beneficial. Regardless of when the
CPM machine is used, the goal is to have the patient in the CPM machine for 6 to 8
hours every 24 hours. If the patient is unable to use the CPM machine, then
instructions are given for passive flexion and extension of the knee with 500
repetitions three times per day. We encourage patients to gain full passive ROM of the
injured knee as soon as possible after surgery. We also prescribe cold therapy for all
patients postoperatively. Our experience and observations indicate that the cold helps
control pain and inflammation, and most patients state that the cold provides overall
postoperative pain relief. Cold therapy is typically used for 1 to 7 days
postoperatively. Crutch-assisted touchdown weight-bearing ambulation (10% of body
weight initially) is prescribed for 6 to 8 weeks, depending on the size of the lesion. For
most patients, 6 to 8 weeks is adequate time to limit weight bearing. However, for
patients with small lesions (<1 cm diameter), weight bearing may be initiated earlier.
Patients with lesions on the femoral condyles or tibial plateaus rarely use a brace
during the initial postoperative period. However, we may prescribe an unloading type
brace when the patient becomes more active and the postoperative swelling has
resolved.
The patient begins therapy immediately after surgery with an emphasis on patellar
mobility and range of motion. Patients are instructed to perform medial to lateral and
superior to inferior movement of the patella as well as medial to lateral movement of
the quadriceps and patellar tendons. This mobilization is essential to prevent patellar
tendon adhesions and associated increases in patellofemoral joint reaction forces.
Range-of-motion exercises (without range-of-motion limitations), quadriceps sets,
straight-leg raises, hamstring stretching, and ankle pumps are also initiated the day of
surgery.
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Stationary biking without resistance and a deep water exercise program are initiated
at 1 to 2 weeks postoperatively. The deep water exercises include use of a flotation
vest for deep water running. It is imperative that the foot of the injured leg does not
touch the bottom of the pool during this exercise to avoid placing excessive loads on
the microfracture site.
After 8 weeks of touchdown weight bearing, the patient is progressed to weight

bearing as tolerated, typically weaning off crutches within 1 week. Restoration of
normal muscular function through the use of low-impact exercises is emphasized
during weeks 9 through 16. Stationary biking with increasing resistance, treadmill
walking on a 7% incline, and elliptical training are all initiated during this phase.
Closed-chain double knee bends and elastic cord exercise are added to the regimen at
about 12 weeks. Our observations indicate that the ability to achieve predetermined
maximum levels for sets and repetitions of elastic resistance cord exercises is an
excellent indicator for progressing to weight training.
We permit free or machine weights when the patient has achieved the early goals of
the rehabilitation program, but not before 16 weeks after microfracture. We strongly
emphasize the importance of proper technique when beginning a weight program. The
patient is instructed to avoid pain during weight training and may be counseled to
avoid ranges of motion that stress the microfracture site. A progressive run-walk
program and initial agility drills start between 16 to 24 weeks postoperatively. Initial
running is completed on a forgiving surface, using a ratio of 1 minute running followed
by 4 minutes walking. Once the patient can perform 20 minutes at a 1:4 ratio, the
ratio is gradually progressed to 4 minutes running and 1 minute walking before the
patient is allowed to run continuously. Initial agility drills include single plane
activities completed at 25% speed with increases in duration made before increases in
intensity. Depending on the clinical examination, size of the patient, the sport, and
the size of the lesion, we usually recommend that patients do not return to sports that
involve pivoting, cutting, and jumping until at least 4 to 9 months after microfracture.
Rehabilitation Protocol for Patients with

Patellofemoral Lesions
Without exception, all patients treated by microfracture for patellofemoral lesions
must use a brace set at 0 to 20 degrees for the first 8 weeks postoperatively. This
brace limits compression of the regenerating surfaces of the trochlea or patella, or
both. We allow passive motion with the brace removed, but otherwise the brace must
be worn at all times. Patients with patellofemoral lesions are placed into a continuous
passive motion machine set at 0 to 50 degrees immediately postoperatively. Apart
from the ROM setting, parameters for the CPM are the same as for tibiofemoral
lesions. We also use cold therapy as described previously. With this regimen patients
typically obtain a pain-free and full passive ROM soon after surgery.
For patients with patellofemoral joint lesions, we carefully observe joint angles at the
time of arthroscopy to determine where the defect comes into contact with the
patellar facet or the trochlear groove. We make certain to avoid these areas during
strength training for approximately 4 months. This avoidance allows for training in the
0- to 20-degree range immediately postoperatively because there is minimal
compression of these chondral surfaces with such limited motion. Patients are allowed
to weight bear as tolerated in their brace 2 weeks after surgery. After 8 weeks, we
open the knee brace gradually before it is discontinued. When the brace is
discontinued, patients are allowed to advance their training progressively. Stationary
biking without resistance is allowed 2 weeks postoperatively; resistance is added at 8
weeks after microfracture. Starting 12 weeks after microfracture, the exercise
program is the same as used for femorotibial lesions.
COMPLICATIONS
Most patients progress through the postoperative period with little or no difficulty.
However, some patients present with mild transient pain, most frequently after
microfracture in the patellofemoral joint. Small changes in the articular surface of the
patellofemoral joint may be detected by a grating or â€œgrittyâ€• sensation of the
joint, especially when a patient discontinues use of the knee brace and begins normal
weight bearing through a full ROM. Patients rarely complain of pain at this time, and
this grating sensation usually resolves spontaneously in a few days or weeks. Similarly,
if a steep perpendicular rim was made in the trochlear groove, patients may notice
â€œcatchingâ€• or â€œlockingâ€• as the apex of the patella rides over this lesion
during joint motion. Some patients may even perceive these symptoms while in the
CPM machine. These symptoms usually gradually dissipate within 3 months. If this
perceived locking is painful, the patient is advised to limit weight bearing and avoid
the symptomatic joint angle for an additional period.
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Swelling and joint effusion typically resolve within 8 weeks after microfracture.
Occasionally, a recurrent effusion develops between 6 and 8 weeks after
microfracture, most commonly when a patient begins to bear weight on the injured
leg after microfracture of a defect on the femoral condyle. While this effusion may
mimic the preoperative or immediate postoperative effusion, usually it is painless. We
treat this type of painless effusion conservatively. It usually resolves within several
weeks after onset. Rarely has a second arthroscopy been required for recurring
effusions.
RESULTS
We published the first long-term outcomes paper on the microfracture technique. This
study followed 72 patients an average of 11 years following microfracture, with the
longest follow-up being 17 years. This study included knees with no joint space
narrowing, no degenerative arthritis, and no ligament or meniscus pathology which
required treatment. All patients were under 45 years of age. The microfracture
technique used on these patients did not include recent improvements to the
technique as described in the technique section of this chapter. With a 95% follow-up
rate, the results showed improvement in symptoms and function. Patient-reported
pain and swelling decreased at postoperative year 1, continued to decrease at year 2,
and the clinical improvements were maintained over the study period. The study
identified age as the only independent predictor of functional (Lysholm) improvement.
Patients over 35 years of age improved less than patients under 35; however, both
groups showed improvement.
We also have documented the outcome of microfracture in patients who played

professional football in the United States. Twenty-five active National Football League
players were treated with microfracture between 1986 and 1997. The study found that
76% of players returned to play the next football season. Following return to play,
those same players played an average of 4.6 additional seasons. All players showed
decreased symptoms and improvement in function. Of those players who did not
return to play, most had pre-existing degenerative changes of the knee.
Arthroscopic treatment of the degenerative knee is controversial. However, we

documented outcomes at 2 years in patients with degenerative chondral lesions
treated with microfracture. Our goals were to alleviate pain, maximize function, and
prevent further degenerative changes. With strict patient selection, proper surgical
technique, and compliance with a well-defined rehabilitation program, patients
showed improvement in their function and had decreased symptoms. Pain and swelling
significantly decreased with most patients reporting only mild symptoms, and patients
were highly satisfied with their results. Factors that were associated with less
functional improvement included bipolar lesions, lesions greater than 400 mm2, and
meniscus-deficient knees. Failures, as defined by revision microfracture or total knee
replacement, were documented in 6% of the patients. These results confirm excellent
short-term outcomes; however, further studies will be needed to determine how long
the results last. A contraindication to microfracture in the degenerative knee is axial
malalignment of the joint. Medial opening wedge high tibial osteotomy (HTO) has
gained popularity as a means of correcting malalignment in patients with medial
compartment arthrosis and varus malalignment who wish to stay active. We recently
reported on 39 patients who underwent an opening wedge osteotomy on the medial
side of the proximal tibia in conjunction with the microfracture procedure in their
degenerative varus knee. Patients showed improvement in function and activity level,
as well as decreased symptoms. Most patients had a greater than 20-point increase in
their Lysholm score. This study also showed that patients with no prior surgeries had
more improvement. Two patients went on to total knee replacement at 3 years and 5
years following the HTO. The study concluded that at a minimum of 2 years following
surgery, patients with varus alignment and chondral surface lesions of the knee can be
treated effectively with the high tibial osteotomy and microfracture. These patients
returned to an active lifestyle as demonstrated by their significantly higher activity
(Tegner) scores.
RECOMMENDED READING
Frisbie DD, Morisset S, Ho CP, et al. Effects of calcified cartilage on healing of
chondral defects treated with microfracture in horses. Am J Sports Med.
2006;34:1824-1831.
Frisbie DD, Oxford JT, Southwood L, et al. Early events in cartilage repair after
subchondralbone microfracture. Clin Orthop. 2003;407:215-227.
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Frisbie DD, Trotter GW, Powers BE, et al. Arthroscopic subchondral bone plate
microfracture technique augments healing of large osteochondral defects in the radial
carpal bone and medial femoral condyle of horses. J Vet Surg. 1999;28:242-255.
Hagerman GR, Atkins JA, Dillman C. Rehabilitation of chondral injuries and chronic
degenerative arthritis of the knee in the athlete. Oper Tech Sports Med. 1995;3:127-
135.
Kocher MS, Steadman JR, Briggs KK, et al. Reliability, validity, and responsiveness of
the Lysholm knee scale for various chondral disorders of the knee. J Bone Joint Surg.
2004;86A:1139-1145.
Miller BS, Steadman JR, Briggs KK, et al. Patient satisfaction and outcome after
microfracture of the degenerative knee. J Knee Surg. 2004;17:13-17.
Steadman JR, Briggs KK, Rodrigo JJ, et al. Outcomes of microfracture for traumatic
chondral defects of the knee: average 11-year follow-up. Arthroscopy. 2003;19:477-
484.
Steadman JR, Miller BS, Karas SG, et al. The microfracture technique in the treatment
of full-thickness chondral lesions of the knee in National Football League players. J
Knee Surg. 2003;16:83-86.
Steadman JR, Rodkey WG, Briggs KK, et al. Debridement and microfracture for full-
thickness articular cartilage defects. In: Scott WN, ed. Insall & Scott surgery of the
knee. Philadelphia: Elsevier; 2006:359-366.
Steadman JR, Rodkey WG, Briggs KK. Microfracture chondroplasty: indications,

techniques, and outcomes. Sports Med Arthrosc Rev. 2003;11:236-244.
Steadman JR, Rodkey WG, Briggs KK. Microfracture to treat full-thickness chondral
defects. J Knee Surg. 2002; 15:170-176.
Steadman JR, Rodkey WG. Microfracture in the pediatric and adolescent knee. In:
Micheli LJ, Kocher M, eds. The pediatric & adolescent knee. Philadelphia: Elsevier;
2006:308-311.
Steadman JR, Rodkey WG. Microfracture technique: treatment of full-thickness

chondral lesions. In: Jackson DW, ed. Reconstructive knee surgery. 2nd ed. New York:
Lippincott Williams & Wilkins; 2002:329-335.
Steadman JR, Rodkey WG, Rodrigo JJ. â€œMicrofractureâ€•: surgical technique and
rehabilitation to treat chondral defects. Clin Orthop. 2001;391S:S362-S369.
Sterett WI, Steadman JR. Chondral resurfacing and high tibial osteotomy in the varus
knee. Am J Sports Med. 2004;32:1243-1249.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 28 - Osteochondral Plug
Transplantation
28
Osteochondral Plug Transplantation
LÃ¡szlÃ³ Hangody
ZsÃ³fia Duska
ZoltÃ¡n KÃ¡rpÃ¡ti
Efficacious treatment of full-thickness cartilage defects of the weight-bearing surfaces
represents a multifaceted challenge for the orthopaedic surgeon. During the recent
decade many efforts have been made to improve the quality of different cartilage
repair techniques aiming to provide a hyaline-like surface in the resurfaced area.
Autologous osteochondral transplantation represents one solution, enabled by
promotion of a hyaline or hyaline-like repair of the affected area. Several series of dog
and horse experiments and 14 years of clinical experience proved that autologous
osteochondral mosaicplasty is a useful alternative in the treatment of full-thickness
cartilage defects. Open and arthroscopic use of the mosaicplasty technique has
already extended clinical experience, and recent data suggest both indications and
contraindications. Besides femoral and tibial condylar and patellotrochlear
applications, talar, femoral head, humeral head, and capitulum humeri implantations
were carried out. Preclinical animal studies and subsequent clinical practice have
confirmed the survival of the transplanted hyaline cartilage as well as fibrocartilage
filling of the donor sites (located on the non-weight-bearing and less-weightbearing
surfaces of the knee joint). Clinical scores, several types of different imaging
techniques, control arthroscopies, histologic examination of biopsy samples, and
cartilage stiffness measurements have been used to evaluate the clinical outcome and
quality of the transplanted cartilage, and have confirmed the clinical efficacy of the
mosaicplasty technique. This chapter discusses the indications, contraindications,
technical details, rehabilitation, and pitfalls and complications of the procedure.
If the preoperative differential diagnosis includes a small or medium-sized focal defect

of the weight-bearing surfaces, the patient should be advised of the possibility of an
autologous osteochondral mosaicplasty. In such cases, the patient should also be
prepared for an open procedure. The site or the size of the lesion can require a
miniarthrotomy or an open procedure; however, the main goal should be an
arthroscopic approach for this type of cartilage transplantation. Open procedures can
lead to an overnight stay and altered weight-bearing status for several weeks.
Transplantation of osteochondral autografts is not new, but its clinical application was
restricted because of certain technical difficulties, such as congruency and donor site
problems. Osteochondral autograft donor sites must be taken from relatively non-
weight-bearing surfaces, which limits the procurement field. On the other hand, use of
large grafts can cause certain incongruities at the recipient site, permanently altering
the biomechanics of the joint. Instead of single-block transfer, the use of small
multiple cylindrical grafts may permit more tissue to be transplanted while preserving
donor site integrity, and their use in a mosaic-like implanting fashion would allow
progressive contouring of the new surface. Autologous osteochondral mosaicplasty has
been introduced in clinical practice in 1992 in Hungary. This type of resurfacing can be
indicated for 1- to 4-cm2 focal chondral or osteochondral defects of the weight-
bearing area. The most common recipient sites are the femoral condyles, but clinical
experience with the patellotrochlear, tibial, and talar applications is fairly extensive.
As far as exceptional applications are concerned, a few implantations for certain
osteochondral defects of the humeral and femoral head as well as the capitulum
humeri were carried out.
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As with other types of resurfacing, mosaicplasty requires the elimination of the
underlying causes (if this is possible) in the same step. According to clinical
experience, anterior cruciate ligament (ACL) reconstruction, meniscus surgery, and
femorotibial realignment procedures are the most common concomitant procedures,
but patellofemoral realignment techniques, lateral release, and so on, may also be
required.
Any disadvantageous condition hindering the survival of the transplanted hyaline

cartilage on the recipient site can be a contraindication, such as the following:
Generalized arthritis, rheumatoid and/or degenerative type
Lack of appropriate donor area
Infectious or tumor defects
Age greater than 50 years
Osteochondral defects deeper than 10 mm
Localized full-thickness cartilaginous lesions are usually defined at arthroscopy, but
preoperative clinical investigation may also give some useful information. Anamnestic
data and actual clinical findings (e.g., tenderness in the medial joint space, swelling,
clicking) may support the presence of a cartilage defect, but there are no specific
signs for determining the exact type and location of existing chondral damage.
Osteochondral lesions usually show more expressed complaints. Despite that small- or
medium-sized chondral and osteochondral damages do not give a characteristic
picture, the clinical examination is necessary to eliminate the actual biomechanical
problems. Stability of the affected knee, femorotibial alignment, and patellofemoral
traction conditions should be cleared at the clinical examination.
Standard and standing radiographic examinations are basic elements of the

preoperative diagnostics. Computed tomography (CT) may inform about the
subchondral bony condition. Ultrasound investigation or special sequences of magnetic
resonance imaging (MRI) can give useful information about the location and extension
of the chondral defects, but severity cannot always be determined exactly. The last
step is usually the arthroscopic examination, which should determine the exact
location and stage of the damage, evaluate the quality of the donor area, and check
all the other intra-articular conditions.
Preoperative preparations should include antibiotic prophylaxis. General or local
anesthesia with tourniquet control is recommended.
SURGERY
The patient is positioned supine with the knee capable of 120 degrees of flexion. The
contralateral extremity is placed in a stirrup. Standard arthroscopic instrumentation
and MosaicPlasty Complete System (Smith & Nephew, Inc., Endoscopy Division,
Andover, MA) are required. Beside these reusable instruments, disposable chisels,
drillbits, and tamps are also available to provide ideal conditions for precise graft
harvest and tunnel preparation (Dispoposplasty System, Smith & Nephew, Inc.,
Endoscopy Division, Andover, MA). A fluid management system may support the
procedure.
Choosing a procedure (arthroscopic or miniarthrotomy) depends on the type, size, and

exact location of the defect determined during arthroscopy. As placing the grafts
perpendicular to the surface is paramount to the success of the operation, the first
task is to determine whether an arthroscopic or open procedure is required. Although
certain trochlear defects can be resurfaced arthroscopically, patellotrochlear and
tibial lesions always require an open procedure. Most of the femoral condylar defects
can be managed arthroscopically. As for most of these lesions, central anterior medial
and central anterior lateral portals will allow correct perpendicular access.
An open procedure may be chosen in the learning curve or when an arthroscopic

approach is not practical due to size or location of the lesion. Arthroscopic or open
mosaicplasties have the same steps and technique.
Arthroscopic Mosaicplasty
Portal Selection
As has been emphasized previously, perpendicular access to the lesion is critical to
proper insertion of the grafts. Take care in making the viewing and working portals.
Use a
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1.2-mm K-wire or 18-gauge spinal needle initially to locate the portal sites. It should
be noted that these portals tend to be more central than the standard portals due to
the inward curve of the condyles (Fig. 28-1). For osteochondritis dissecans on the
medial femoral condyle, the approach should be from the lateral side. Standard lateral
portal is sometimes too oblique. Therefore, use the central patellar tendon portal,
which gives good access to the inner positions of both the medial femoral condyle and
the lateral femoral condyle.
Defect Preparation
Use a full-radius resector or curette and a knife blade to bring the edges of the defect
back to good hyaline cartilage at a right angle. Clean the base of the lesion with an
arthroscopic burr (Abrader, Acromionizer) or half-round rasp to viable subchondral
bone (Fig. 28-2). Abrasion arthroplasty of the defect site promotes fibrocartilage
grouting from the bony base. Because tapping the cutting edge of the guide into the
bony base and removal of it can mark the defect site, use the drill guide to determine
the number and size of grafts needed (Fig. 28-3). Filling of the defect by same-sized
contacting rings allows a filling rate of about 70% to 80%, but use of additional sizes to
cover the dead spaces and cutting the grafts into each other can improve the coverage
to 90% to 100% (Fig. 28-4). Finally, measure the depth of the defect with the laser
marks of the dilator.
Graft Harvest
The medial femoral condyle periphery of the patellofemoral joint above the line of the
notch is the preferred arthroscopic harvest site. The lateral femoral condyle above the
sulcus terminalis and, in exceptional cases, the notch area can serve as additional
donor areas (Fig. 28-5). Grafts harvested from the notch area are less favorable
features, as they have concave cartilage caps and less elastic underlying bone. The
medial patellofemoral periphery has easier access than the lateral one as fluid
distension can promote lateral positioning of the patella and may provide easier
perpendicular positioning for the harvesting chisel.
FIGURE 28-1
A: Use of a spinal needle to determine the perpendicular access to the defect. B:

Arthroscopic picture of the same step.
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FIGURE 28-2
A: Abrasion arthroplasty of the bony base of the defect. B: Arthroscopic picture

of the same step.
The best view for harvesting grafts is obtained by introducing the scope through the
standard contralateral portal. Extend the knee and use the standard ipsilateral portal
to check the perpendicular access to the donor site. Extended position should provide
perpendicular access to the most superior donor hole. Step-by-step flexion allows the
harvest of additional grafts from the lower portions of the patellofemoral periphery. If
the standard portals do not allow a perpendicular approach, use a spinal needle or a K-
wire to determine the location of additional harvesting portals.
Once the necessary portal has been determined, introduce the proper-sized tube
chisel filled with the appropriate harvesting tamp. Once the site has been clearly
identified, the chisel is located perpendicular
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to the articular surface and driven by a hammer to the appropriate depth. The
minimal length of the graft should be at least two times its diameter, but, as a rule,
take 15-mm-long grafts to resurface chondral lesions and 25-mm-long plugs for
osteochondral defects. It is important to hold the chisel firmly to avoid its shifting at
the cartilage-bone interface, producing a crooked graft. By flexing the knee, lower
sites can be obtained. The lower limit is the level of the top of the intercondylar notch
(sulcus terminalis). Insert the appropriate harvesting tamp into the cross-hole in the
tubular chisel and use it as a lever. The chisel should be toggled, not rotated, causing
the graft to break free at the chisel tip (Fig. 28-6). Eject the grafts from the chisel by
sliding the appropriately sized chisel guard over the cutting end. Use the tamp to push
out the graft onto gauze in a salinewetted basin (Fig. 28-7). The donor site holes will
eventually be filled up with initial repair tissue by bleeding mediated mesenchymal
stem cell invasion in a few hours. Proper rehabilitation during the first postoperative
weeks may support a transformation of the primary repair tissue into cancellous bone
and fibrocartilage as final coverage.
FIGURE 28-3
A: Abrasion arthroplasty of the bony base of the defect. B: Arthroscopic picture

of the same step.
FIGURE 28-4
A: Eighty percent, 90%, and 100% filling of a defect. B-D: Intraoperative pictures
of the same filling rates.
FIGURE 28-5
Location of the recommended donor sites.
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FIGURE 28-6
A,B: Graft harvest by toggling the harvesting chisel.
During the learning curve, the grafts can also be obtained through a miniarthrotomy
(1.5 to 2.0 cm).
Implantation of the Grafts: â€œDrill-Dilate-Deliverâ€•

(Three-Dimensional Grafting)
DRILL
Flex the knee and establish good distension. Reintroduce the drill guide using the
dilator as an obturator. Place these tools perpendicularly to the defected surface. By
rotating the arthroscope, the drill guide and the perpendicularity of the laser mark
can be seen from different angles, ensuring proper orientation. Tap the cutting edge
of the guide into the subchondral bone. Insert the appropriate size drill bit and drill to
the desired depth (Fig. 28-8). Generally, a recipient hole a few millimeters deeper
than the length of the graft is desirable to minimize high intraosseal pressure. Reduce
the inflow to minimize leakage. Finally, remove the drill bit.
DILATE
Insert again the conical-shaped dilator into the drill guide. Tap it to the desired depth,
depending on the actual features of the recipient bone. Stiff bone needs more dilation
than normal or soft bone. Hold the drill guide firmly and remove the dilator from the
hole (Fig. 28-9).
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FIGURE 28-7
A-C: Graft removal from the harvesting chisel.
DELIVER
Adjust the delivery tamp by turning the handle to initially allow the graft to sit slightly
higher than the depth of the defect. This will minimize the likelihood of
overpenetrating the graft. Stop the inflow; otherwise, fluid flow can push the graft out
of the tube. Deliver the graft under direct visualization into the recipient hole through
the drill guide with the delivery tamp (Fig. 28-10). Insert the graft deeper by turning
the delivery tamp handle counterclockwise. The graft should be flush with the original
articular surface. Remove the drill guide to inspect the graft. If the graft is proud,
reinsert the drill guide and tap the graft down gently with the tamp of the appropriate
size. Insert the subsequent grafts in a similar fashion by placing the drill guide
immediately adjacent to the previously placed grafts. Such step-by-step graft
implantation has several advantages. Dilation of the actual recipient hole allows an
easy graft insertion (low insertion force on the hyaline cap), but dilation of the next
hole affects the surrounding bone to the previously implanted grafts, which can result
in a very safe press fit fixation (Fig. 28-11).
When all the holes are filled and the grafts are in place, put the knee through a range
of motion, creating varus or valgus stress, depending on the site of the resurfacing
(Fig. 28-12). Close the portals and introduce suction drainage into the joint through a
superior portal. Use an elastic bandage to fix the appropriate dressing.
Complications
One of the most common problems is to neglect the main requirement of the
operation. Perpendicular harvest and implantation of the grafts are crucial for
successful transplantation. Oblique harvest and insertion may result in steps on the
surface. Careful control by the arthroscope from different angles should help avoid
such problems.
Another frequent mistake is to implant a graft deeper than the desired level. First of
all, appropriate use of the delivery tamp can help to avoid deep insertion of the
grafts. If the graft has been inserted too deep, the following steps are recommended:
Insert the drill guide next to the previously implanted graft. Drill the appropriate
recipient hole. Remove the guide and use the arthroscopic
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probe to remove the previously implanted graft to the proper level (Fig. 28-13). The
recipient hole adjacent to the implanted graft should allow such manipulation. As soon
as the expected graft level has been achieved, continue the recommended sequence
for the other insertions. Dilation of the adjacent tunnel will provide perfect press fit
fixation of the previously implanted graft.
FIGURE 28-8
A-C: Drilling of the recipient tunnel. D: Arthroscopic picture of the same step.
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FIGURE 28-9
A-C: Dilating of the recipient tunnel. D: Arthroscopic picture of the same step.
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FIGURE 28-10
A,B: Insertion of the graft. C,D: Arthroscopic pictures of the same step.
Open Mosaicplasty
If an arthroscopic approach is impractical, it may be necessary to create a medial or
lateral anterior sagittal incision or an oblique incision to perform a miniarthrotomy
mosaicplasty (Fig. 28-14). Patellotrochlear and tibial implantations may require an
extended anteromedial approach. Further steps and technique of the implantation are
identical to those for the open procedure.
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FIGURE 28-11
A,B: Step-by-step implantation supports a safe press fit fixation.

FIGURE 28-12
A,B: Functional test of the resurfaced area.
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FIGURE 28-13
Elevation of a too deeply implanted graft.
Postoperatively, the drain should be removed at 24 hours. Appropriate pain and cool
therapy as well as nonsteroidal anti-inflammatory drugs can lessen patient discomfort.
Postoperative thrombosis prophylaxis is recommended.
Rehabilitation
In the rehabilitation protocol following mosaicplasty at our institutions, there is no
immobilization. The main point of the rehabilitation is to ensure the early motion of
the treated joint to promote appropriate nutrition of transplanted cartilage. Cool
therapy can be used during the first week to avoid postoperative bleeding and to
decrease pain. In the case of a concomitant procedure requiring external fixation of
the affected joint (e.g., meniscus reinsertion), limitation of range of motion for a
short period by bracing can be allowed. Table 28-1 provides the details of our
rehabilitation protocol.
FIGURE 28-14
Intraoperative picture of a miniarthrotomy mosaicplasty.
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TABLE 28-1. Mosaicplasty Rehabilitation Protocol

(Uzsoki Hospital and Sanitas Private Clinic, Budapest,
Hungary)
Ambulation
Extent, type (chondral or osteochondral), and location of the defect may

modify weight bearing.
Two-crutch ambulation, Immediately

non-weight bearing
Two-crutch ambulation, 2 to 4 weeks

partial loading (30 to 40
kg/sec)
Discontinue crutches, full 4 to 5 weeks

weight bearing
Functional Exercises
Form walking, gait 4 to 5 weeks

evaluation
Step-up 4 to 5 weeks
Step-down 5 to 6 weeks
Range of Motion
Early range of motion is encouraged.

CPM for 2 to 4 cm2 lesions Immediately (first week)
(in painless range)
Full extension, flexion as Immediately

tolerated
Stationary bicycle 3 weeks
Strength Return
Quadriceps
Open-chain exercises, leg Immediately

raises
Concentric contraction to 1 week (or earlier if tolerated)

full extension
Concentric contraction 2 weeks

against resistance
Isometric exercises at Immediately

different angles
Excentric exercises 3 to 4 weeks

against resistance
Hamstrings
Isometric exercises in Immediately
different angles
Concentric and excentric 1 to 2 weeks

strengthening
Against resistance 3 to 4 weeks
Closed-Chain Exercises
Partial loading promotes the transformation of connecting tissue (between

transplanted plugs) into fibrocartilage, so these exercises are important,
especially in the half-weight-bearing period. However, with some closed-
chain exercises (e.g., cycling) it is possible to ensure cyclic loading that
makes the fluid and nutrition transport much more efficient between
synovial fluid and hyaline cartilage.
Pushing a soft rubber ball Immediately

with foot
Closed-chain exercises 2 to 3 weeks

with half weight bearing
With full weight 5 to 6 weeks

bearing
Stationary bicycle with 2 to 4 weeks (if 90 degrees knee flexion

resistance achieved)
Stair Master 6 to 8 weeks

Proprioception Return
Balance exercises standing 5 to 6 weeks

on both feet
Standing on one foot 6 to 8 weeks

(hard ground)
Standing on one foot 8 to 10 weeks

(trampoline or
aerostep)
Return to Activity
Jogging 10 weeks
Straight-line running 3 months
Directional changes 4 to 5 months
Shear forces 5 months (approx. 4 to 5 months are needed

to form a composite hyaline-like surface on a
transplanted area that tolerates shear forces)
Sport-specific adaptations 5 months
Sport activity 5 to 6 months (depending on depth and extent

of the defect; if strength, power, endurance,
balance, and flexibility are not sufficient,
sport activity is allowed later)
Special Viewpoints
Weight Bearing at Different Defects of the Knee
Femur or tibia condyle,

chondral defect, d <15
mm
Non-weight bearing 1 week
Partial weight 1 to 3 weeks

bearing

chondral defect, d â‰¥ 15
mm
Non-weight bearing 2 weeks

bearing

osteochondral defect
Non-weight bearing 3 weeks

bearing
Patellar defect, d <15 mm
Partial weight 2 weeks

bearing
Patellar defect, d â‰¥15

mm
Partial weight 3 weeks

bearing
Quadriceps Strengthening and Patellar Mobilization-Differences at Patellar

Defects
Vastus medialis
strengthening
Isometric exercises in Immediately

extension
Patellar mobilization Immediately
Isometric exercises in 1 week

different angles
Open-chain exercises 2 weeks
Against resistance 3 to 4 weeks

Eccentric exercises 4 to 5 weeks
against resistance
Closed-chain exercises 2 to 3 weeks
The treatment of underlying causes can also modify the rehabilitation

program. The most frequent combinations at knee applications follow.
Anterior Cruciate Ligament (ACL)-Reconstruction Combined with

Mosaicplasty
2 to 4 weeks non-weight bearing (up to the mosaicplasty)
2 more weeks of partial weight bearing
0 to 90 degrees ROM for 3 weeks
Mainly closed-chain exercises for quadriceps strengthening
Hamstring strengthening in open and closed chain
Proprioceptive training
Meniscus Reinsertion Combined with Mosaicplasty
4 weeks of non-weight bearing

Retinaculum Patellae Reconstruction Combined with Mosaicplasty
2 to 4 weeks of non-weight bearing (up to the mosaicplasty)
High Tibial Osteotomy (HTO) Combined with Mosaicplasty
Weight bearing (for 4 weeks only with crutches and only in extension) is up
to the mosaicplasty, pain, and degree of the correction of the varus
(undercorrectionâ€”non-weight bearing, overcorrectionâ€”early weight
bearing).
COMPLICATIONS
Septic or thromboembolic complications may result in a negative influence on the
clinical outcome. Correct aseptic conditions, one-shot antibiotics, and thrombosis
prophylaxis can decrease the chance of these complications.
According to 14 years of follow-up, long-term donor site morbidity does not occur
frequently. Patellofemoral complaints, such as pain or swelling after strenuous
physical activity, follow the mosaicplasty procedure in fewer than 3% of cases.
However, excessive postoperative bleeding occurred in 8%. Precise postoperative
drainage, cool therapy, and elastic bandages can lessen the chance of this
complication.
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RESULTS
Between February 1992 and February 2002, 831 mosaicplasties were accomplished at
our institutions. These implantations involved the femoral condyles in 597 procedures,
the patellofemoral joint in 118, the talar dome in 76, the tibial condyles in 25, the
capitulum humeri in 6, the femoral head in 9, and later, the humeral head in 3 cases
as well. Two thirds of the patients underwent surgery because of a localized grade III
or grade IV cartilage lesion, according to the Outerbridge classification system,
whereas one-third underwent surgery because of osteochondral defects. In 85% of the
patients, concomitant surgical interventions also were done, including ACL
reconstructions, realignment osteotomies, and meniscal surgery. In most of the
patellar or trochlear graftings, patellofemoral realignment or lateral release was also
done. The results of the resurfacing procedures were evaluated at regular intervals
with use of standardized clinical scores and radiography, and selected patients were
also assessed with MRI, second-look arthroscopy, histological analysis of biopsy
materials, and cartilage stiffness measurement. Femoral, tibial, and patellar implants
were evaluated according to the modified Hospital for Special Surgery scoring system,
the modified Cincinnati knee-rating scale, the Lysholm scale, and the International
Cartilage Repair Society scoring system, and donor-site disturbances were evaluated
according to the Bandi score system. According to our investigations, good-to-
excellent results were achieved in more than 92% of the patients treated with femoral
condylar implantations, 87% of those treated with tibial resurfacing, 79% of those
treated with patellar and/or trochlear mosaicplasties, and 94% of those treated with
talar procedures. Long-term donor-site discrepancies, assessed with use of the Bandi
score, showed that patients had 3% morbidity after mosaicplasty.
Nineteen patients had a second-look arthroscopic examination performed between 2

months and 6 years postoperatively because of persistent or recurrent pain, swelling,
or postoperative intra-articular bleeding; 23 had arthroscopy performed between 1
and 9 years postoperatively because of a second trauma; and 41 had arthroscopy
performed between 2 and 4 months postoperatively to evaluate the quality of the
resurfaced area and to determine the earliest date to return to a professional sports
activity. Sixty-nine of the 83 patients who were followed arthroscopically showed
congruent gliding surfaces, histologic evidence of the survival of the transplanted
hyaline cartilage, and fibrocartilage filling of the donor sites. In a limited series
involving 23 patients, cartilage stiffness measurements were performed with use of a
computerized arthroscopic indentometric device, the Artscan 1000 (Artscan Oy,
Helsinki, Finland), at a pressure of 10 N. Nineteen of them had similar values to the
healthy hyaline cartilage.
RECOMMENDED READING
BodÃ³ G, Hangody L, MÃ³dis L, et al. Autologous osteochondral grafting (mosaic
arthroplasty) for the treatment of subchondral cystic lesions in the equine stifle and
fetlock. Vet Surg. 2004;33:588-596.
FeczkÃ³ P, Hangody L, Varga J, et al. Experimental results of donor site filling for
autologous osteochondral mosaicplasty. Arthroscopy. 2003;19(7):755-761.
Hangody L. Mosaicplasty. In: Insall J, Scott N, eds. Surgery of the knee. New York:
Churchill Livingstone; 2000:357-361.
Hangody L. The mosaicplasty technique for osteochondral lesions of the talus. Foot
Ankle Clin North Am. 2003;8:259-273.
Hangody L, FeczkÃ³ P, KemÃ©ny D, et al. Autologous osteochondral mosaicplasty for

the treatment of full thickness cartilage defects of the knee and ankle. Clin Orthop.
2001;391(suppl):328-337.
Hangody L, Kish G. Surgical treatment of osteochondritis dissecans of the talus. In:

Duparc, J, ed. European textbook on surgical techniques in orthopaedics and
traumatology. Editions Scientifiques et Medicales. New York: Elsevier; 2000.
Hangody L, Kish G, KÃ¡rpÃ¡ti Z, et al. Arthroscopic autogenous osteochondral

mosaicplasty for the treatment of femoral condylar articular defects. Knee Surg Sports
Traumatol Arthros. 1997;5: 262-267.
Hangody L, Kish G, KÃ¡rpÃ¡ti Z, et al. Osteochondral plugs: autogenous osteochondral

mosaicplasty for the treatment of focal chondral and osteochondral articular defects.
Oper Tech Orthop. 1997;7:312-322.
Hangody L, Kish G, KÃ¡rpÃ¡ti Z, et al. Treatment of osteochondritis dissecans of talus:

the use of the mosaicplasty technique. Foot Ankle Int. 1997;18:628-634.
Hangody L, RÃ¡thonyi G, Duska Zs, et al. Autologous osteochondral mosaicplasty-

surgical technique. J Bone Joint Surg. 2004;86-A(suppl I):65-72.
Kish G, MÃ³dis L, Hangody L. Osteochondral mosaicplasty for the treatment of focal

chondral and osteochondral lesions of the knee and talus in the athlete. Clin Sports
Med. 1999;18:45-61.
KordÃ¡s G, SzabÃ³ J, Hangody L. Primary stability of osteochondral grafts used in

mosaicplasty. Arthroscopy. 2006;22(4):414-422.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 29 - Chondrocyte
Transplantation
29
Chondrocyte Transplantation
Lars Peterson
Lesions of the articular cartilage in the knee joint present great diversity in size,
location, depth, and containment. These factors have to be evaluated to decide the
right treatment. Autologous chondrocyte transplantation (ACT) is a treatment option
for symptomatic full-thickness chondral or osteochondral lesions (OCD). The patients
(15 to 55 years old) often present with pain and catching or locking on activity and
pain and swelling after activity.
Chondral lesions possible for treatment with ACT should be between 1 to 2 and 16 cm2
with grade III or IV Outerbridge classification (grade 3 or 4 ICRS classification). The
opposing articular surface has to be undamaged or have only minor cartilage pathology
(grade I or II Outerbridge classification, grade 1 or 2 ICRS classification).
Autologous chondrocyte transplantation is indicated for lesions on the femoral

condyles, the femoral trochlea, and patella. Patients with tibial plateau defects
treated with ACT are showing improved results over time; therefore, this locale can be
considered for ACT. Long-term follow-up on kissing lesions (i.e., bipolar lesions in the
femurotibial and patellotrochlear joints) show at 4 to 10 years postoperatively
satisfactory results in 75% of the cases. Autologous chondrocyte transplantation can be
tried in these cases in younger patients as a salvage procedure. Multiple lesions, two
or more lesions in one joint excluding kissing lesions, have shown satisfactory results in
84% at 5 to 11 years postoperatively. Almost all of these patients needed concomitant
procedures to create optimal environment for the transplanted chondrocytes.
Stabilizing or unloading procedures will improve the short- and long-term survival of
the repair tissue in the injured surfaces. Autologous chondrocyte transplantation is not
a treatment for osteoarthritis (general joint disease), gout, and rheumatoid arthritis or
other systemic joint diseases.
Autologous chondrocyte transplantation is indicated for osteochondritis dissecans

(OCD) with unstable fragments, osteochondral flaps and empty defects, and a size
between 1 to 2 and 16 cm2. If the bony defect is between 6 and 8 mm deep, ACT alone
can be used. If the bony defect is deeper, concomitant autologous bone grafting
(sandwich technique) has to be used.
The preoperative planning includes a careful history to find out the cause of the
cartilage lesion: traumatic or microtraumatic. Clinical assessment of symptoms and
signs, radiologic imaging including standing radiographs, and arthroscopic assessment
are essential.
Examination and Evaluation

The preoperative assessment is important when deciding if ACT is an option for the
patient and should include all pathology of the knee; but an arthroscopic evaluation
must precede the final decision. Try to find out the cause of the cartilage defect,
since this affects the treatment. Crepitations during extension and flexion against
resistance may indicate cartilage pathology. Check for any ligament instability or
malalignment that might need surgical correction. It is important to have a stable
joint since instability greatly decreases the chances of a successful surgery.
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Regarding lesions to the patella, it is very important to examine for possible patellar
instability, malalignment, or maltracking, since this often is the cause of the cartilage
damage.
Radiography, Magnetic Resonance Imaging,

Arthrography
Radiologic imaging is useful to rule out patients with osteoarthritis. Standing
radiographs in 45 degrees of knee flexion and extension and hip-knee-ankle
radiographs show joint space reduction and varus or valgus malalignment that may
need surgical correction. Computerized tomography with and without quadriceps
contraction may reveal patellofemoral dysplasia or instability. Magnetic resonance
imaging (MRI) with or without contrast medium (gadolinium) may be helpful to assess
the articular cartilage and pathology of the underlying bone.
Arthroscopic Evaluation
If ACT so far is the choice of treatment, the next step is an arthroscopic examination
performed by the surgeon. The examination proceeds as follows:
Administer general or spinal anesthesia.
Test the joint stability.
Examine all the articular surfaces, the menisci, the synovial lining, and the
cruciate ligaments using an arthroscopic probe.
Remove loose bodies or debris in the joint, if present.
Evaluate the location, size, containment, and depth of the cartilage lesion
(Fig. 29-1).
Make sure the lesion is accessible through an optimal arthrotomy.
Harvest cartilage if the lesion is suitable for chondrocyte transplantation.
Slice cartilage specimens from the unloaded proximal, medial edge of the
trochlea using a ring curette or a gouge and (Fig. 29-2).
When necessary use other locations for the cartilage biopsy, such as the
proximal, lateral edge of the trochlea or the lateral intercondylar notch.
Cut down through the subchondral bone to cause a bleeding, so the donor site
area may heal with fibrocartilage.
Let one end of the specimen still be attached, then use a grasper to take out
the specimen.
Harvest cartilage from a surface of approximately 5 by 10 mm is, resulting in
the required 200 to 300 mg of cartilage.
Place the specimens in a sterile tube containing a medium of DMEM with

phenolred and gentamycin.
Treat meniscal injuries after the harvesting of cartilage.
The harvested cartilage is enzymatically digested and the chondrocytes are isolated
and cultured for 2 weeks, which results in an increase in cell number by 20 to 30 times
(Fig. 29-3). The implantation of the chondrocytes can thus at the earliest take place 2
weeks after the harvesting. If the implantation is scheduled at a later date, the
chondrocytes can be frozen after 1 week of culturing, and then thawed and cultured
for another week before the implantation.
SURGERY
Chondral Lesions
Preparation and Approach
Place the patient supine on the operating table.
Use general or spinal anesthesia and a tourniquet-controlled bloodless field.
Perform medial or lateral parapatellar miniarthrotomy to open the joint.
Adjust the arthrotomy for good access to the defect.
To expose multiple femoral or patellar lesions, the patella may have to be

dislocated.
Consider the approach for concomitant procedures, for example, high tibial
osteotomy.
Debridement of Defect
Excise all damaged and undermined cartilage.

Get vertical edges to healthy cartilage.
Cut down to the subchondral bone around the lesion using a knife with a No. 15
blade.
Keep the lesion contained by cartilage, if possible.
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It is better to leave a 3 to 4 mm rim of cartilage of questionable quality than
have a lesion bordering bone or synovium.
Remove the damaged cartilage using a ring curette or a periosteal elevator.
Be careful not to violate the subchondral bone and cause bleeding and possible
fibrous ingrowth in the defect.
The debrided defect should be as circular or oval as possible (Fig. 29-4).
Measure the defect and use sterile aluminum foil or paper to make a template
of the defect (Fig. 29-5).
Check for any bleeding of the subchondral bone; at this time the tourniquet
can be let down for easier detection.
If there is a bleeding, place an epinephrine-wet sponge in the defect to stop it.
Keep the explored cartilage moist throughout the procedure.

FIGURE 29-1
A-C: ICRS classification of the location and depth of a cartilage lesion. (With
permission from International Cartilage Repair Society, ICRS Cartilage Injury
Evaluation Page, 2000.)
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FIGURE 29-1
(Continued)
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FIGURE 29-1
(Continued)
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FIGURE 29-2
Schematic drawing of the cartilage harvest (A). Arthroscopic pictures of a full-
thickness cartilage lesion (B) and cartilage harvesting with a ring curette (C).
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FIGURE 29-3
From the harvested cartilage slices the chondrocytes are isolated and cultured for
2 weeks, before the implantation can take place.
Periosteal Flap Harvest

It is then time for the harvesting of a periosteal flap to cover the cartilage defect. The
periosteum is easily accessed at the proximal medial tibia, distal to pes anserinus and
the medial collateral ligament insertion. The medial or lateral femoral condyles
proximal to the articular cartilage can also be used as harvest sites but the periosteum
is thicker, less elastic, and covered by numerous vessels, which may cause
postoperative bleeding and arthrofibrosis if not taken care of. More adequate
periosteum is found on the distal shaft of the femur proximal to the condyles. The
steps for periosteal flap harvest are as follows:
Incise to get good exposure.
Dissect gently and remove the overlying fibrous tissue, fat, and crossing vessels
covering the periosteal flap.
Use the template to measure how a large flap you need (Fig. 29-6).
Oversize the periosteal flap with 1 to 2 mm at the periphery of the template.
Incise the periosteal flap using a knife with a No. 15 blade.
Carefully dissect it with a periosteal elevator with small movements and lift
with a nontoothed forceps.
Go close down to the bone so the cambium layer of the periosteum is

preserved and take care not to penetrate the flap. A healthy cambium layer
interacts with the chondrocytes and is thus important.
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Mark the outside of the periosteal flap with a sterile pen for orientation at
fixation.
Be sure to keep the flap moist at all times.

FIGURE 29-4
Drawings and photographs showing a femoral condyle defect before (A,B) and
after (C,D) radical excision and debridement of all damaged and undermined
cartilage.
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FIGURE 29-5
The debrided defect is measured (A) and a template of aluminum foil is made (B).
Periosteal Flap Suturing
Return to the debrided defect and inspect once again for bleeding.
Cover the defect with the periosteal flap with the cambium layer facing the
defect bed.
Anchor the periosteal flap with one suture in each corner using a cutting
needle with 6.0 resorbable sutures, soaked in glycerin or mineral oil (Fig. 29-
7A,B).
Place the knots over the periosteal flap.
Tension the flap like a skin over a drum with interrupted sutures in a Z pattern.
The distance between the sutures should be 3 to 5 mm (Fig. 29-7C,D).

Leave an opening in the most superior portion for the injection of the
chondrocytes.
Use fibrin glue to seal the distances between the sutures.
A good periosteal fit and fixation is crucial for the surgical outcome. If the
lesion is not contained by cartilage the periosteal flap has to be sutured to the
synovium. If there is no synovial lining or osteophytes interfere, suture to small
holes drilled through the bone or use resorbable minitacks.
Testing Waterlightness
With a saline-filled tuberculin syringe and a plastic 18-gauge 2-inch angiocath, test the
watertightness of the periosteum-covered defect.
FIGURE 29-6
The template is used to excise a periosteal flap of the right size and form from
the proximal medial tibia.
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FIGURE 29-7
The flap is first anchored with one suture in one corner (A,B). The flap is then
sutured to the cartilage rim with interrupted sutures (C,D).
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FIGURE 29-8
Test the watertight integrity by injecting saline under the periosteal flap.
Gently insert the angiocath through the superior opening and slowly inject the
saline (Fig. 29-8).
Any leakage may easily be detected at the periosteum-cartilage border around

the defect.
If needed, add another suture and fibrin glue so watertight integrity is

obtained.
Check for leakage again.
If satisfied, aspirate the saline but make sure that the periosteal flap does not
stick to the defect bed.
Implantation of Cells
The defect is now ready for the chondrocyte implantation.
Aspirate the chondrocytes in a tuberculum syringe using an 18-gauge or larger

needle.
Replace the needle with a plastic 18-gauge 2-inch angiocath.
Start filling the inferior portion of the defect by placing the angiocath deep
into the defect and gently inject the chondrocytes.
Withdraw the angiocath as you inject the chondrocytes; the cells should be
evenly distributed throughout the defect (Fig. 29-9).
Close the superior opening with sutures and fibrin glue.
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If the defect is very large you may have to use an upper and a lower opening
for proper filling of the defect. Inject through the lower opening, then close it
before you start filling through the upper opening.
FIGURE 29-9
Patella defect showing the syringe placed deep into the defect and under
withdrawal the chondrocytes are injected.
When the opening is sealed, the defect is contained and watertight, but the periosteal
flap still allows diffusion of fluid and nutrition to the transplanted chondrocytes. Intra-
articular drains should not be used as they could harm the periosteum and suck out the
transplanted chondrocytes. If a drain must be used make sure it is without suction.
Close the wound in layers and bandage the leg from the foot and up over the knee.
Postoperative Management
The patient usually stays in the hospital for 1 to 3 days depending on the severity of
the case. Prophylactic antibiotics are used for 48 hours postoperatively. Continuous
passive motion (CPM) is started 6 to 8 hours after the surgery. The chondrocytes can
then be expected to have attached to the defect bed and the rim of healthy cartilage.
Continuous passive motion is continued intermittently until the patient is released
from the hospital. The day after surgery range of motion (ROM) and isometric
quadriceps training are started. The patient is mobilized with crutches. Depending on
the size and location of the defect, weight bearing to 20 kg or to the limit of pain is
allowed for the first 6 weeks. Over the next 6 weeks, a gradual increase in weight
bearing is allowed until full weight bearing is reached. After release from the hospital,
the patient returns after 3 and 6 weeks and after 3 months for check-ups. Follow-ups
are then as needed.
Concomitant Surgery
It is important to create an optimal environment for the repair tissue. This means
correcting any background factors such as instability or malalignment, and if
necessary, unloading the repair tissue. These procedures may be done separately, for
example an anterior cruciate ligament (ACL) reconstruction or a tibial osteotomy, or
at the same time as the biopsy is taken. Often it is done at the same time as the
implantation. Pathologic mechanics in the joint reduce the chances of a successful
repair. Patellar lesions are often related to an unstable patella, and the patella must
thus be stabilized for a good healing. Stabilizing procedures may include
anteromedialization of the tibial tuberosity, lateral release, proximal medial soft
tissue shortening, and trochlear groove plasty (if it is dysplastic). In trochlear and
patellar lesions, unloading with ventralization of the tibial tuberosity should be
considered. A torn ACL is reconstructed after the cartilage lesion is debrided and
covered with periosteum, but before the chondrocytes are injected. To unload the
transplanted area when a varus or valgus malalignment is present, a high tibial or
femoral osteotomy is performed. When these corrective surgeries are performed, a
brace limiting the range of motion to 0 to 60 degrees is used postoperatively for 3
weeks and for the following 3 weeks with a ROM of 0 to 90 degrees.
Osteochondral Lesions (Osteochondritis Dissecans)

When treating osteochondritis dissecans with ACT, attention must be paid to the depth
of the defect. If the bony defect is shallower than 6 to 8 mm, the lesion is treated the
same way as a chondral lesion. Gently debride the sclerotic bottom of the defect, but
be careful not to cause bleeding; and the cartilage is debrided to vertical edges to
healthy cartilage. Cover the defect with a periosteal flap, seal, and check for leakage.
Then implant the chondrocytes and close the last opening.
If the bony defect is deeper than 6 to 8 mm only ACT is not enough, concomitant
autologous bone grafting is needed (Fig. 29-10A,B).
Abrade away the sclerotic bottom of the defect to spongious bone and
undercut the subchondral bone.
Drill holes in the spongious bone with a 2-mm burr.
Debride the cartilage to healthy cartilage with vertical edges (Fig. 29-10C,D).
If the bony defect is small, use cancellous bone from the tibial or femoral
condyle; but if the defect is larger, bone has to be harvested from the iliac
crest.
Start packing the defect from the bottom and contour the cancellous bone
over the defect just below the subchondral bone level (Fig. 29-10E,F).
Harvest a periosteal flap and cover the contoured bone grafted defect at the
level of the subchondral bone with the cambium layer facing the joint.
Anchor it with horizontal or mattress sutures into the cartilage or subchondral

bone and use fibrin glue under the flap for fixation to the grafted bone.
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After depositing the glue richly between the bone graft and periosteal flap,
compress the area with a dry sponge for 2 to 3 minutes to seal off the bone
marrow cavity. This will avoid bleeding into the cartilage defect (Fig. 29-10G).
Suture another periosteal flap to the cartilage edges, with the cambium layer
facing the defect (Fig. 29-10H).
Use fibrin glue to seal off the intervals between the sutures.
Test the watertightness with a gentle saline injection.

If there is no leakage, aspirate the saline and inject the chondrocytes (Fig. 29-
10I).
Close the opening and seal with fibrin glue.

FIGURE 29-10
MRI (A) and photo (B) of an osteochondritis dissecans with a deep bony defect on
the medial femoral condyle. The defect is abraded and multiple holes are drilled.
The subchondral bone is undercut (C,D).
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FIGURE 29-10
(Continued) Cancellous bone (E) is used to fill the bony defect and should contour
the subchondral bone (F). A periosteal flap with the cambium layer facing the
joint is glued on top of the bone graft and sutured to the surrounding cartilage
(G). Another periosteal flap is sutured to the cartilage rim with the cambium
layer facing the defect and the chondrocytes are injected underneath (H,I).
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FIGURE 29-10
(Continued) MRI (J) 18 months after concomitant bone and chondrocyte

transplantation, and arthroscopic picture of the transplanted area 2 years after
the transplantation (K).
Pearls and Pitfalls

Pearls
For a good result, only defects that fit the indications should be transplanted. Kissing
lesions should not be transplanted other than as a salvage procedure. It is important
that the surgeon performs the initial arthroscopy himself or herself, to be able to
adequately decide if the patient is a candidate for ACT or not. At the time of
implantation, excise the cartilage defect radically. All undermined, fissured, and
damaged cartilage should gently be debrided to get a good fit of the periosteal flap.
Accurate sizing, harvesting, and suturing of the flap are crucial for a good and durable
outcome. There is definitely a learning curve for this procedure. Begin with at least
ten simple cases to become comfortable with the technique before treating multiple
lesions or cases combined with corrective surgery, such as ACL reconstruction.
Pitfalls
PERIOSTEUM
It is important that the flap used to cover the defect is of healthy periosteum.
Atrophic periosteum may be found in inactive, obese, and older patients; it is also
more common in females and smokers. A very thin and fragile periosteum must not be
used; try to find periosteum of good quality at the femoral condyles. Incise the flap
and carefully dissect thicker vessels, fat, and fibrous tissue off the flap. A clean and
thin flap leaves more room for cell expansion and matrix filling
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of hyaline cartilage and causes less periosteal complications. Compress the fat away
with a wet sponge but be careful not to harm the periosteum. After the flap is
removed, use electrocoagulation to seal the vessels and thus avoid unwanted bleeding
at the harvest site. Bleeding after harvest from the femoral condyles may cause
problems with pain and arthrofibrosis with difficulties in regaining range of motion. If
you find rifts or vessels penetrating the flap, close these with 6.0 resorbable sutures
and fibrin glue after the flap has been sutured to the defect. In large defects you may
have to use two flaps. Suture the two flaps separately. Then adapt the two flap
borders with a few interrupted sutures and secure it with a running suture across.
DEBRIDEMENT
When debriding the defect, be careful and meticulous so all pathologic cartilage is
removed without causing bleeding. If bleeding occurs, place an epinephrine sponge in
the defect during periosteal harvesting. If this is not enough, stop the bleeding by
dripping a drop of fibrin glue and then press your fingertip over for 30 seconds, or
carefully use punctual electrocoagulation (risk for bone necrosis).
If the cartilage defect has been previously treated with drilling, microfracture,
abrasion, arthroplasty, or other procedures harming the subchondral bone, internal
osteophytes can be present. If they interfere with the periosteal anchoring, they must
be handled. Gently tap them down to the level of the surrounding subchondral bone
(Fig. 29-11). You may gently debride a large and high osteophyte with a curette. Any
resultant bleeding must be treated accordingly. Treatment penetrating the
subchondral bone may also result in fibrous plugs; debride them down to the level of
the surrounding subchondral bone.
What I Have Learned from Experience

Radical excision is important. Incise in normal cartilage in the periphery, including
pathologic cartilage. In the beginning I was not radical enough in the excision
especially in patellar lesions. It is sometimes difficult to separate macroscopically
normal from abnormal cartilage. Add a margin of 1 to 2 mm to include apoptotic
chondrocytes in the border.
Suture with interrupted sutures, not running sutures, and grasp 4 to 6 mm into the
cartilage to avoid cutting through with the sutures. If you loose one interrupted
suture, it is not so bad but if you loose a
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running suture, it may cause delamination. Tie the suture with four knots, placed on
the vertical edge of the cartilage over the periosteal flap. Three knots only may have a
tendency to untie the suture.
FIGURE 29-11
An intralesional osteophyte (A) that has been tapped down to the level of the
subchondral bone (B).
Pay great attention to background factors such as ligament instability, varus-valgus

deformities, patellar instability, and maltracking. The results are improved by
correction of these factors. On large patellar or trochlear lesions, elevate the tibial
tuberosity.
The meniscus has an important role and should be preserved and sutured when
possible. Resection should be optimal but kept as conservative as possible. Total
meniscectomy in the injured compartment may need meniscus replacement. Initial
results when combining ACT with meniscus transplantation are promising.
An overly aggressive rehabilitation program with dynamic strength training in open

kinetic chains, and too early weight bearing may cause pain, swelling, and periosteal
complications. However, in contained isolated lesions, the increased weight bearing
can start earlier-at about 3 weeks if no pain is experienced. If the patient experiences
a clicking, snapping sensation, sometimes catching or locking, usually between 3 to 4
or 8 to 9 months, analyze the activity that elicits the symptoms and change the
program accordingly. In half the cases the symptoms will disappear. If symptoms
persist, arthroscopically examine and debride either hypertrophic periosteal tissue or
intra-articular adhesions. Slow progress with ROM may occur postoperatively.
Supervise the training and intensify the ROM exercises; frequent check-ups of progress
are important. Usually the motion will increase, but slowly. Do not interfere during
the first 4 months; any manipulation could jeopardize the repair or cause graft
delamination. If it is necessary, use arthroscopy to visualize and remove adhesions and
then gently mobilize the knee. Leave a drainage and start continuous passive motion
(CPM) immediately.
The rehabilitation following autologous chondrocyte transplantation is a slow process
with gradual progression that extends over a long period of time. The new cartilage
matures over time and must have the right amount of stimulation to form a durable
tissue, able to withstand the forces in the knee. There is a large degree of individual
variation in the rehabilitation process, and the program should be formed according to
the patient's status and needs. Factors such as the size and location of the defect and
possible concomitant corrective surgery are important to have in mind when forming
the rehabilitation program. It is important that there is regular contact between the
patient and physical therapist. An aggressive rehabilitation with too early or heavy
dynamic strength training could jeopardize the transplanted area. Keys to a successful
rehabilitation are mobility exercises, isometric strength training, functional exercises,
and patience.
COMPLICATIONS
More then 1,300 transplantations have been made in Sweden since 1987, and there
have been no major general complications such as intra-articular infections. Local
complications from the periosteal flap or the graft of transplanted chondrocytes are
not uncommon, but most of them can be corrected with only minor surgery. The
periosteal flap covering the defect can become fibrillated and cause crepitations and
swelling (Fig. 29-12A,B). As a response to the friction of joint movement, it may also
become hypertrophic and cover the normal cartilage surrounding the defect in the
direction of motion or become thicker (Fig. 29-12C). When symptomatic, this causes
crepitations and clicking, and is sometimes associated with pain and swelling. All these
periosteal complications should be evaluated arthroscopically. Examine the graft
incorporation to the surrounding cartilage and the subchondral bone. If a fibrillated or
hypertrophic periosteal flap causes the symptoms, shave or excise the flap down to
the level of the surrounding cartilage.
A delaminated periosteal flap could cause early problems with catching or locking. The
delamination could be either marginal (less than 10 mm), partial (less than 50% of the
flap is delaminated), or total (more than 50% of the flap is detached) (Fig. 29-12D, E-
F). A marginal or partial periosteal flap delamination is treated with a gentle
debridement of the loose part. If the delamination is total and the periosteal flap
appears as a loose body, it should be extracted; but if it is still attached to the
underlying repair tissue, remove the whole flap. The remaining repair tissue is capable
of filling the
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defect, but be careful with excessive loading of the repair tissue and encourage
bicycling the first 6 to 8 weeks.
FIGURE 29-12
Periosteal complications: fibrillation (A,B), periosteum overlaps surrounding
cartilage (C,D), marginal (E), partial (F), and total (G) periosteum delamination.
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FIGURE 29-12
(Continued)
The graft of transplanted chondrocytes can also show incomplete incorporation to the
surrounding cartilage and the underlying subchondral bone. A marginal delamination
with less than 10 mm of the graft loosened (Fig. 29-13A) is excised and the
subchondral bone of the failed area is microfractured. A partial delamination means
that less than 50% of the graft is delaminated (Fig. 29-13B). This is treated with a
gentle debridement, and cartilage for a retransplantation is harvested.
A total graft failure in which the whole graft is loose or only a rim is still attached is
seldom seen (Fig. 29-13C). It could be caused by a distorsion of the knee or too
aggressive rehabilitation and early return to sports. In this case, the patient
experiences pain, swelling, and locking. Remove the graft and harvest chondrocytes
for a new ACT.
RESULTS
You can expect hyaline-like repair tissue in 80% of cases. If you have a good/excellent
result at 2-year follow-up, it will stay good/excellent at 5 to 11 years (mean 7.4). In
femoral condyle lesions and OCD, you can expect 85% to 90% good/excellent results. In
patellar lesions, there are about
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70% good/excellent results. In salvage procedures in kissing lesions in the
patellotrochlear or tibiofemoral joints, 3 out of 4 patients may improve (Fig. 29-14).
FIGURE 29-13
Drawings of marginal (A), partial (B), and total (C) graft delamination.
Arthroscopic picture showing a partial graft delamination (D).
What to Tell Patients to Expect

Patients are told that this is a repair process starting with cultured chondrocytes in
suspension injected into the defect. The cells undergo some mitosis to confluence,
adhere to the subchondral bone and cartilage edges, and start to produce matrix. It is
important that the collagen is anchored in the subchondral bone. At 3 months, the
defect is filled with soft and compressive repair tissue. From 3 to 9 to 12 months,
there is an ongoing maturation with improved organization of the cells and filling with
matrix, and increased weight bearing stimulates this process. The maturation process
continues slower after 12 months but is active at 2 to 3 years. The ultimate goal is that
the turnover of repair cartilage is equal to the normal surrounding cartilage. There is a
5% to 10% rate of complications leading to reoperation; however, most complications
are benign and can be handled with arthroscopic procedures (e.g., periosteal
complications, adhesions, and arthrofibrosis).
Crutches are used and only partial weight bearing is allowed for 10 to 12 weeks. Walk
increasing distances when full weight bearing is achieved. Bicycling is used with low
resistance for stimulation and adaptation of the repair tissue. Expect a year before
returning to impact sports activities. The patient should be followed by the surgeon
throughout the whole rehabilitation period.
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FIGURE 29-14
A 39-year-old female soccer player had total meniscectomy at age 16.

Arthroscopy showing bipolar lesions to medial femoral condyle and medial tibial
condyle (A), that were treated with ACT (B,C) concomitant with high tibial
osteotomy as a salvage procedure. Follow-up arthroscopy at 4 years shows healing
with good integration to the surrounding cartilage (D).
RECOMMENDED READING
Brittberg M, Lindahl A, Nilsson A, et al. Treatment of deep cartilage defects in the
knee with autologous chondrocyte transplantation. New Engl J Med. 1994;331:889-895.
Minas T, Peterson L. Advanced techniques in autologous chondrocyte transplantation.

Clin Sports Med. 1999;18(1):13-44.
Peterson L. Cartilage cell transplantation. In: Malek MM, ed: Knee surgery. New York:
Springer; 2001:440-449.
Peterson L. International experience with autologous chondrocyte transplantation. In:

Insall JN, Scott WN, eds. Surgery of the knee. 3rd ed. New York: Churchill Livingstone;
2001:341-356.
Peterson L, Brittberg M, Kiviranta I, et al. Autologous chondrocyte transplantation:

biomechanics and long-term durability. Am J Sports Med. 2002;30(1):2-12.
Peterson L, Minas T, Brittberg M, et al. Treatment of osteochondritis dissecans of the

knee with autologous chondrocyte transplantation: results at two to ten years. J Bone
Joint Surg Am. 2003;85-A(suppl 2):17-25.
Peterson L, Minas T, Brittberg M, et al. Two- to 9-year outcome after autologous

chondrocyte transplantation of the knee. Clin Orthop Relat Res. 2000;374:212-234.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 30 - Opening Wedge
Osteotomyâ€”Proximal Tibia and Distal Femur
30
Opening Wedge Osteotomyâ€”Proximal Tibia
and Distal Femur
Giancarlo Puddu
Vittorio Franco
Massimo Cipolla
Guglielmo Cerullo
Enrico Gianni
Osteoarthrosis of the knee has many causes. Degenerative changes of the articular
cartilage can occur through tension, compression, or shear. These changes are very
much related to the forces exerted on the bearing surfaces. Genetic factors are known
to play a part. Specific trauma and trauma from the overload caused by obesity or
occupational factors are etiologically important. In essence, the biophysical cause of
osteoarthrosis is an overload or a concentration of forces beyond the ability of the
cartilage and subchondral bone to cope.
In any discussion of osteoarthrosis of the knee and its treatment, the operation of
knee replacement (arthroplasty) versus knee realignment (osteotomy) is always
pertinent. Historically, osteotomy preceded replacement by about 10 years. As
replacement became a reality in the 1970s, the indications for each operation became
clearer. While the annual number of osteotomies has remained stable, the
proportional rate of total arthroplasties has increased dramatically since the early
1960s.
It follows that alignment of the lower extremity plays an important role in

osteoarthrosis of the knee. Malalignment into a varus position overloads the medial
condyles of the femur and tibia; valgus malalignment, similarly, overloads the lateral
condyles. The rationale behind the osteotomy is to correct the angular deformity at
the knee and therefore to decrease the excessive weight-bearing load across the
affected compartment, which is the most involved by the degenerative process. Since
alternative treatments for severe osteoarthrosis of the knee in the 1960s were limited,
tibial osteotomy at the beginning was used for osteoarthrosis, rheumatoid arthritis,
and secondary arthrosis, regardless of the etiopathogenesis and the magnitude of the
angular deformity. After several years' experience, Coventry (1), Insall (3), and others
narrowed the indications for tibial osteotomy. By the late 1970s, high tibial osteotomy
was felt to be for the younger, more active patient in whom total knee replacement
was thought to be unwise.
Now we think that the patients selected for proximal tibial/distal femur osteotomy
should have mostly unicompartmental osteoarthrosis with axial malalignment.
However, fracture and other trauma, congenital and acquired deformities, and
idiopathic osteonecrosis are also reasons for osteotomy.
A patient is typically a candidate for high tibial osteotomy when the orthopaedic
surgeon can clinically detect a varus standing alignment associated with a medial
compartment arthritis in a stable knee; a medial compartment cartilage damage
associated with anterior cruciate ligament (ACL), posterior cruciate ligament (PCL),
posterolateral corner, or combined ligament deficiencies; a painful knee after
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a medial meniscectomy; articular cartilage defects requiring a repair technique; an
osteochondritis dissecans lesion; or a patient who requires a meniscal allograft.
A typical candidate for distal femur osteotomy is young or middle-aged, athletic

patient who has developed a disabling painful knee due to an early arthritis of the
lateral compartment after lateral meniscectomy. Also candidate to the antivalgus
procedure is the patient with chondral damages of the lateral femoral condyle
associated with an ACL or PCL acute tear or chronic laxity; the patient with congenital
valgus deformity often associated with overweight and lateral compartment arthritis;
and the patient with a cyst of the lateral meniscus or with a discoid meniscus followed
by a subtotal meniscectomy.
There is no definite patientage below which one should have an osteotomy, or above
which one should have an arthroplasty. The age of 65 years is cited most often, but
activity level, lifestyle, and general health must be considered. As long-term studies of
arthroplasty demonstrate, age considerations may change. But the fact remains that
osteotomy patients are generally younger than those who undergo knee arthroplasty.
Osteotomy is best done for unicompartmental osteoarthrosis in knees with generally

well-maintained range of motion (ROM) (at least 90 degrees of flexion; less than 15
degrees of flexion contracture). Osteotomy should probably not be done in patients
with rheumatoid arthritis, patients with very unstable knees, or those whose knees
have greater than 20 degrees of varus deformity or 15 degrees of valgus deformity,
according to Insall (2). These knees are complicated by an associated severe
ligamentous laxity and subluxation; the latter is a relative contraindication. In fact,
even if the correction of the mechanical axis in such a great deformity can result in a
relaxed collateral ligamentous complex, often the knee finds a new functional stability
so that many authors, including ourselves, do not routinely perform retightening of the
ligamentous structures.
A patient who has varus deformity and ACL insufficiency may be treated with an ACL
reconstruction in addition to the proximal tibial osteotomy. A technically demanding
procedure, the osteotomy associated with the ligament reconstruction addresses the
underlying disorder and corrects the problems. Because this is not a routine
procedure, it will not be discussed here. However, the symptoms of pain and
instability must be separated as clearly as possible, because when pain prevails,
especially in sedentary patients, correction of alignment, with consequent relief of
medial compartment pain, can be a satisfactory treatment.
The varus arthritic symptomatic knee very often shows articular cartilage lesions,
Outerbridge (4) grades III and IV, that affect the involved medial compartment. Full-
thickness chondral defects rarely heal without a direct intervention. Together with the
modern proposals of treatment of the articular cartilage tears (autologous chondrocyte
implantation and osteochondral autografts), the methods based on bone marrow
stimulation are still in action. Historically, the first technique of marrow stimulation
proposed was the Pridie drillings, introduced by the author in 1959. Presently,
Steadman's (7) microfracture arthroplasty seems a more effective and safer option, as
it is completely arthroscopic, and has the advantage, compared with the former, of
not inducing local thermal necrosis. When performed together with the HTO,
microfractures fully complement the surgical treatment, satisfying the necessity of a
direct treatment of the intra-articular pathology, without increasing the risk of
complications or lengthening time of surgery.
Overweight is a controversial topic. Obesity has a negative effect on the outcome of

surgery in many orthopaedic operations. Most would agree that excess weight could
make a patient a better candidate for osteotomy than for arthroplasty, but it is also
true that obesity itself represents a negative factor in view of the possible general
postoperative complications. When a patient is overweight, the weight should be
brought to near normal before surgery, since osteotomy has a worse long-term
outcome when the patient is obese.
Another contraindication to osteotomy is severe bone loss (more than a few

millimeters) of the medial or lateral tibia or femur. When medial or lateral
compartment bony support is insufficient, congruent weight bearing on both tibial
plateaus following the osteotomy is impossible. In this situation tibiofemoral contact
will teeter on the relatively prominent intercondylar tibial spines.
Severe varus or valgus deformity may be associated with lateral or medial subluxation
of the tibia, respectively. Subluxation of more than 1 cm is an absolute
contraindication to osteotomy, and some authors suggest that osteotomy should not be
performed if any translation or subluxation is present.
Studies on the biomechanics of the dynamic gait accurately addressed the issue of
varus or lateral thrust of the knee during ambulation. The term adductor moment was
used to describe the amount of lateral or varus thrust of the knee observed during
gait. Patients with high adductor moment have
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worse results following osteotomy than those with a low adductor moment.
Furthermore, patients with a high adductor moment are more likely to have a
recurrent varus deformity following valgus osteotomy. When osteotomy is chosen for
those patients in spite of a high adductor moment, overcorrection of the deformity
may be helpful.
Osteotomy of the proximal tibia or distal femur is designed to relieve pain caused by
medial or lateral tibiofemoral osteoarthrosis. Slight degenerative changes of the
patellofemoral joint are not a contraindication to osteotomy. However, following
proximal tibial osteotomy with a medial opening wedge, the anterior tibial tubercle is
lowered to about one half of the angular correction; therefore, the condition of
â€œpatella bajaâ€• is a contraindication to this kind of operation.
The goal of knee osteotomy is to realign the mechanical axis of the limb, thereby
shifting weightbearing forces from a diseased compartment to a more normal
compartment.
The alignment of the limb is best measured by full-length radiographs of the lower
extremity.
The mechanical axis is determined by drawing a line from the center of the femoral
head through the center of the knee to the center of the talus. The anatomic axis is
depicted by drawing a line through the center of the shaft of the femur and through
the center of the shaft of the tibia. In the normal knee, the two lines cross each other
in the center of the joint, making an angle of 5 degrees (physiologic valgus), and the
mechanical axis passes through the center of the joint, or slightly varus (about 1
degree medially).
The deformity is measured by using these parameters as the reference points of

â€œnormality.â€• With a varus knee we want to move the mechanical axis through the
neutral alignment (approximately the center of the knee) up to a more lateral point at
about two thirds (63%) of the tibial plateau to get 5 degrees of mechanical valgus
alignment and thereby overcorrect the anatomic valgus from the â€œnormalâ€• 5
degrees to 9 or 10 degrees (Fig. 30-1). Extensive experience has shown that
overcorrection is absolutely essential if one wants to optimize a long-standing result
from valgus osteotomy. It is inconvenient to correct to normal valgus that brings the
extremity back to the position from which it originally became deformed, and that
does not properly unload the medial compartment. In the normal knee, approximately
60% of the weight-bearing forces are transmitted through the medial compartment and
approximately 40% are transmitted through the lateral compartment. Even in severe
valgus deformity (up to 30 degrees valgus), the medial plateau load never falls below
30%.
FIGURE 30-1
In the varus knee we want to correct the deformity, moving the mechanical axis
up to a lateral point of the tibial plateau (62%, about two thirds of the articular
surface) to get 10 degrees of anatomic valgus alignment (hypercorrection).
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There is a controversy about full-length radiographs made with the patient supine
compared with those made with the patient standing. Originally these radiographs
were made with the patient supine. Then, some surgeons noted that, in this position,
the amount of deformity was underestimated in certain patients who had osseous
defects or ligamentous laxity, and this was presented as a cause of undercorrection by
the osteotomy. For these reasons, most authors currently recommend that radiographs
be made with the patient standing. However, two problems remain. First, because the
patient can take the weight off the painful limb when standing and thus negate much
of the benefit of this position, it could be better to take the radiographs with the
patient standing on only the affected limb. Second, when there is severe ligamentous
laxity, to plan the operation on radiographs made with the patient standing can lead
to overcorrection, a potentially greater problem than undercorrection.
When we have a reasonable doubt from the physical examination, we calculate the
joint line convergence angle (JLCA), which is formed by a line tangent to the distal
femoral condyles and a line tangent to the tibial plateau. A single-leg weight-bearing
full radiograph and a double-leg full radiograph, always in standing position, are
obtained to measure the lower limb alignment. The difference in the JLCA between
the two radiographs represents the component of malalignment associated with
ligamentous laxity. To prevent overcorrection, the soft tissue laxity is subtracted from
the overall valgus correction.
A young patient with a symptomatic congenital varus could be an early candidate for a
valgus osteotomy, but the correction must not be an overcorrection, because in such
patients it is sufficient to restore the physiologic alignment of the knee.
With the valgus knee, we want to reposition the mechanical axis to neutral alignment
at approximately 0 degrees in the center of the joint. This means 5 degrees of
anatomic and physiologic valgus (Fig. 30-2). The biomechanics of varus and valgus
deformities of the knee differ. In fact, the intrinsic valgus angle between femur and
tibia determines an asymmetric overload of the medial compartment, at about 60% of
the whole, already in the normal knee. Thus, the overbalance of the knee toward a
varus alignment will result in a functional disaster because of the additional overload
of the medial compartment and the consequent, dramatic acceleration of the
degenerative changes of the more normal side.
FIGURE 30-2
In the valgus knee we want to correct the deformity, moving the mechanical axis
up to approximately the center of the tibial plateau to get 5 degrees of
physiologic valgus alignment (normocorrection).
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FIGURE 30-3
The Rosenberg view. Comparative posteroanterior weight-bearing knee

radiograph at 45 degrees of flexion facilitates the diagnosis if the standard
standing AP view is not sensitive enough.
The osteotomy we propose here is based on the opening wedge technique. Special
plates with a spacer â€œtoothâ€• were designed for this aim. We need to know the
size of the base of the wedge, calculated in millimeters, to choose the plate and fix
the osteotomy to the planned angle. Different widths of the tibia, at the level of the
osteotomy cut, correspond to different wedge sizes, for the same degree of angular
correction.
To complete evaluation of the knee, we look at the different radiographic views,

including the standard lateral and the axial views of the patellofemoral joint.
The Rosenberg view, comparative posteroanterior weight-bearing radiograph at 45

degrees of knee flexion, facilitates the diagnosis in case the standard anteroposterior
(AP) view is not sensitive enough (Fig. 30-3). The Rosenberg examination has a strong
predictive value when the deformity is associated with a cruciate insufficiency, and
therefore the chondral wear prevails in the posterior part of the tibial plateaus.
Most authors don't recommend computed tomography (CT) scans or magnetic

resonance imaging (MRI) in studying a candidate for knee osteotomy; but we think that
the stress reaction of the subchondral bone, detectable by MRI (Fig. 30-4), could be
the only sign of a degenerative process at its earlier stage.
FIGURE 30-4
MRI can be positive for a stress reaction of the subchondral bone as the only
diagnostic sign of an early degenerative process.
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SURGERY
Dedicated Surgical Instrumentation
The object of valgus osteotomy is to obtain a new mechanical axis overcorrected up to
5 degrees of valgus. Our purpose with varus osteotomy is to reposition the lower limb
to align to the physiologic 0 degrees of the neutral mechanical axis.
We present here our technique to perform the opening wedge osteotomy. To achieve
more reproducible results with the fewest technical difficulties, the senior author (GP)
developed a complete but simple and easy system of dedicated instruments and
plates.
We get the valgus correction of the knee by means of proximal tibia osteotomy and
the varus correction by means of distal femoral osteotomy. In varus deformity the
tibiofemoral joint line is usually parallel to the floor. Proximal tibia osteotomy has
been demonstrated to transfer load effectively from the medial to the lateral
compartment. In valgus deformity, the joint line has a valgus tilt with a correspondent
obliquity from superolateral to inferomedial direction. Although tibial varus osteotomy
may realign a valgus limb, it cannot correct the joint-line tilt because the procedure is
performed distally to the joint. The mechanical consequence of this, in patients with
severe valgus deformities (more than 10 to 12 degrees according to various authors), is
the effect of transferring the load transmission medially not more than the lateral
portion of the tibial spine. The resultant increased valgus tilt of the joint line leads to
greater shear forces and lateral subluxation during gait. However, distal femoral varus
osteotomy may realign a valgus limb and correct valgus tilt of the joint line when used
to treat lateral tibiofemoral osteoarthrosis with valgus deformity.
The plates specially designed for this osteotomy are butterfly shaped, with four holes
for the tibia, and T shaped, with seven holes for the femur (Fig. 30-5) (Arthrex,
Naples, FL). Their peculiarity is a spacer, a tooth as it were, available in many
different sizes from 5 to 17.5 mm thick (up to 20 mm for the femur), with the tooth
increasing one size for each additional millimiter. Tibial plates with trapezoidal
spacers are also available to permit the correction of the coronal and, eventually, the
sagittal deformity, the so-called tibial posterior slope, in one operation. The tooth
enters the osteotomic line, holding the position and preventing a later collapse of the
bone with the recurrence of the deformity. The thickness of the spacer must coincide
with the desired angle of correction, calculated in the preoperative planning. The two
upper holes of the tibial plate and the three of the horizontal lower arms of the
femoral T plate allow the introduction of the AO 6.5-mm cancellous screws, and the
lower holes of the tibial plate and the holes in the vertical arm of the femoral plate
are cut for the AO 4.5-mm cortical screws.
The new plates are now made in a titanium alloy (see Figs. 30-14 and 30-22). The most
important improvement from the former ones is the new special holes of the plate.
Through them the screws can be freely oriented in every preferred direction and then
even locked in the plate, like an internal mini fixator (thanks to a special device of the
hole itself, created by the manufacturer). Dedicated cortical and cancellous screws for
the new plates, also of titanium alloy, are now available.
FIGURE 30-5
The plates are specially designed for opening wedge osteotomies in different
shapes and sizes.
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The crucial point of the operation is the opening of the metaphysis, where the
osteotomy has cut the bone, at the desired angle of correction and holding the
position to allow the introduction of the plate tooth. Recently we introduced a new
innovative tool, the â€œopener jackâ€• (Fig. 30-6), which greatly facilitates this step.
Introduced into the osteotomic line, the jack gently retracts the bone to create the
space for the plate and the grafts. It consists of two osteotomes coupling with a screw
long enough to move away the blades and open the osteotomy. Then, a very simple
â€œopener wedgeâ€• enters into the already prepared osteotomic site. It looks like a
fork with two wedge-shaped tines, graduated to hold the opening at the correct rate,
and a removable handle to allow the positioning of the plate.
The other two dedicated tools are the special Homan retractor for the vastus lateralis
(Fig. 30-7), to be used in the femoral osteotomy, and a long rod guide with an ankle
support to check intraoperatively the mechanical femorotibial alignment (Fig. 30-8).
Surgical Technique of Tibial Osteotomy

Step 1: Patient Position
We prefer a normal operating table with the patient in a supine position and the C-
arm of an image intensifier set up opposite the surgeon. The patient is draped as usual
in knee surgery; we also prepare the iliac wing and cover the foot using a very fine
stockinette and a transparent adhesive drape to minimize the bulging at the ankle so
that it will be possible to better realize the femorotibial alignment after the
correction. The tourniquet may be inflated.
Step 2: Arthroscopy
Arthroscopy of the knee is carried out before the osteotomy to assess the relative
integrity of the controlateral tibiofemoral compartment and that of the patellofemoral
joint and to treat any intra-articular pathology.
FIGURE 30-6
The â€œosteotomy jackâ€• consists of two osteotomes coupling with a screw long
enough to move away the blades and open the osteotomy.
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FIGURE 30-7
The special Homan is designed for the lateral femoral approach to retract the
vastus lateralis.
After the careful arthroscopic examination has been completed, we perform all the
other opportune procedures before microfracture arthroplasty. This caution is to
prevent loss of visualization that could happen when droplets and blood enter the
knee from the microfracture holes, after the penetration of the subchondral bone.
We dedicate particular attention to the treatment of meniscal tears and synovial

pathology and, of course, removal of any eventual, loose body.
Then we examine the full thickness defects of the cartilage, candidate to the
microfracture, to assess that the exposed bone is intact. It is critical to remove all
loose or marginally attached flaps of cartilage in and surrounding the affected area to
leave a lesion with a stable rim. Also very important is to remove the calcified
cartilage layer that is often present but preserve an intact subchondral plate without
violating it with a courette or motorized burr. The aim of this accurate preparation is
the creation of a firm â€œcratereâ€• with stable perpendicular edge of healthy
cartilage that will trap in the clot formed by the bone marrow coming from the
microfractures. The special surgical instrument to use is the dedicated awl, suggested
by Steadman, with an angled tip, typically 30 or 45 degrees, that allows it to be
perpendicular to the bone as it is advanced in the tibial, or femoral, tear. Chondral
lesions up to 3 or 4 cm2 are treated in this manner, and the holes must be
approximately 3 to 4 mm apart, or about 4 or 5 holes per cm2. The major advantage of
this technique, compared to the older drilling procedure, is that the awl produces no
thermal necrosis of the bone at all.
When the holes in a sufficient number have been made, marrow fat droplets and blood
are released from the microfracture and form a pluripotential clot which fills in the
chondral defect. The defect on the joint surface had been carefully prepared in
advance to get the cratere with the firm perpendicular edge of healthy cartilage that
becomes the ideal site for the clot and provides an optimal environment for the
pluripotential marrow cells to differentiate into stable repairing tissue and cover the
whole lesion. Moreover the awl produces, because of the microfractures, a roughened
surface of the subchondral bone to which the clot can adhere more easily.
FIGURE 30-8
The long rod guide with an ankle support is dedicated to intraoperatively check
the tibiofemoral alignment after the osteotomic correction.
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When the cartilage that surrounds the critical area of the lesion is too thin, it may be
impossible to create a stable rim with a well-defined perpendicular edge and,
consequently, the technique is contraindicated.
Chronic degenerative chondral lesions commonly have extensive sclerotic bone with
thickening of the subchondral plate. In these cases, it is very important to remove the
sclerotic bone, using a motorized burr or an aggressive courette, until a punctuate
bleeding of the surface appears; but the integrity of the subchondral plate should be
always respected to preserve the joint surface's original shape and congruence.
The marrow clot, which Steadman named â€œsuperclot,â€• promotes the healing
process of the joint surface toward a neoformed tissue very close in substance and
function to the normal articular cartilage.
Step 3: Incision and Exposure

We expose the anteromedial aspect of the tibia through a vertical skin incision
centered between the medial border of the anterior tibial tubercle and the anterior
edge of the medial collateral ligament and extending 6 to 8 cm distally to the joint
line. Sharp dissection is carried to the sartorius fascia, and pes anserinus tendons are
identified and detached from the bone. The pes anserinus is now retracted and the
anterior half of the underlying superficial collateral ligament is cut horizontally (Fig.
30-9). There is no risk of instability because the deepest, and much more stabilizing,
tibiomeniscal bundle of the ligament remains intact. A blunt retractor is placed
dorsally, deep to the collateral ligament, to protect the posterior vessels and to
clearly expose the posteromedial corner of the tibia. Anteriorly, a second retractor is
placed under the patellar tendon. The procedure is facilitated by flexion of the knee.
Step 4: Osteotomy
The authors' preferred method is a â€œfreeâ€• technique. With the knee in extension
and under fluoroscopic control, a guide pin (Steinmann) is drilled, by the â€œfree
hand,â€• through the proximal tibia from medial to lateral. This is oriented obliquely,
starting approximately 4 cm distally to the joint line and directed across the superior
edge of the tibial tubercle to a point 1 cm below the joint line (Fig. 30-10).
The original instruments system also provides an osteotomy guide assembly to help the
surgeon in the proper placement of the guide pin and an osteotomy cutting guide to
facilitate the use of the oscillating saw. The guide may be oriented to accommodate
variations in size and anatomy. Different choices in tilting the osteotomy cut in both
the coronal and sagittal planes are also possible.
The osteotomy is then performed, keeping the oscillating saw blade below and parallel
to the guide pin, to prevent an intra-articular fracture. The saw is used to cut the
medial cortex only. Then a sharp osteotome (Fig. 30-11) is used to finish the
osteotomy, making certain that the all the cancellous metaphysis and especially the
anterior and the posterior cortices are completely interrupted but preserving a lateral
hinge of about 0.5 cm of intact bone. Fibular osteotomy is not necessary.
FIGURE 30-9
The pes anserinus tendons are retracted to expose the superficial collateral
ligament.
FIGURE 30-10
A guide Steinmann pin is drilled through the proximal tibia from medial to lateral,
obliquely oriented and directed across the superior edge of the tibial tubercle to
a point 1 cm below the joint line.
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FIGURE 30-11
The osteotomy is performed, keeping the osteotome below and parallel to the
guide pin to prevent an intra-articular fracture.
Step 5: Wedge Opening

The osteotomic line is easily opened by the help of the jack, which gently moves the
tibial axis and realigns the knee. Then the opener wedge is introduced and slowly
advanced into the metaphysis (Fig. 30-12). The surgeon measures the dimension of
bone gap directly on the graduated tines of the wedge and chooses the plate.
Step 6: Plate Fixation

By removing the handle and, if necessary, one of the wedges, with the other one still
into the osteotomy, the plate can be positioned easily on the medial cortex of the
tibia
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with the spacer tooth introduced into the osteotomic line (Fig. 30-13). Before fixing
the plate, we check the mechanical axis under fluoroscopy by means of the special
guide rod, which is long enough to extend from the center of the femoral head through
the knee to the center of the ankle (Fig. 30-14). When the rod crosses the knee at a
lateral point about two-thirds (63%) of the tibial plateau, we know the angular
correction corresponds to the 10 degrees of anatomic valgus we had planned in
advance. But if the correction is under- or oversized, we can still change the plate,
with one having a thicker or thinner tooth as needed. Next, the plate is fixed
proximally with two 6.5-mm cancellous screws and distally with two 4.5-mm cortical
screws. In the valgus knee, the medial side is the convex side so that the plate can act
as a tension band device, obeying an important biomechanical principle for the
effectiveness of the internal fixation.
FIGURE 30-12
The opener wedge is introduced and slowly advanced until the osteotomy has
been opened to obtain the planned realignment of the knee.
FIGURE 30-13
Before fixing the plate, we check under fluoroscopy the mechanical axis by means
of the special guide rod, which is long enough to extend from the center of the
femoral head through the knee to the center of the ankle.
Step 7: Bone Grafting

With a skin incision extended from the anterosuperior iliac spine 8 to 10 cm above the
iliac crest, we take two or three corticocancellous bone grafts with the same wedge
shape of the osteotomy. The larger one measures the full correction; the others are
proportionately smaller. The grafts are press-fit and introduced to fill the defect. It is
also possible to use different grafts, such as bone from the bank; or bovine, freeze-
dried bone; or, according to some other authors, no grafts at all. The correct position
of the plate and grafts is confirmed with AP and lateral
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radiographs (Fig. 30-15). One or two drains (the second, intra-articular, if needed) are
prepared and the wound is closed in a routine fashion.
FIGURE 30-14
A new titanium plate positioned on the tibia medial cortex with the spacer tooth
into the osteotomy and fixed to the bone with two proximal 6.5-mm cancellous
screws and two distal 4.5-mm cortical screws.
FIGURE 30-15
The correct position of the plate and grafts is confirmed by the postoperative AP
and lateral radiographs.
Surgical Technique of Femoral Osteotomy

Steps 1 and 2: Patient Positionâ€”Arthroscopy
There are no differences in positioning and preparing the patient for femoral
osteotomy compared with the tibial procedure. The arthroscopy for the femoral
osteotomy has the same justification and is performed with the same intentions as in
the tibial procedure.
Step 3: Incision and Exposure

We expose the lateral aspect of the femur with a standard straight incision through
the skin and the fascia, starting two finger-breadths distally to the epicondyle and
extending the incision about 12 cm proximally (Fig. 30-16A). The dissection is carried
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down to the vastus lateralis, which is retracted from the posterolateral intermuscular
septum by the special dedicated Homan retractor placed ventrally (Fig. 30-16B).
Perforating vessels are to be expected and should be controlled with ligature or
electrocautery. We leave the joint capsule intact. The lateral cortex is now exposed.
The procedure is facilitated by flexion of the knee.
FIGURE 30-16
A: The lateral aspect of the femur is exposed with a standard straight skin
incision. B: The dissection is carried down through the fascia to the lateral
cortex, retracting the vastus lateralis with the special Homan.
FIGURE 30-17
The Steinmann guide pin is drilled in a slightly oblique direction from lateral to
medial, safely off from the troclear groove.
Step 4: Osteotonry
Again the authors' preferred method consists of drilling the guide pin into the femur
with the â€œfree hand.â€• The osteotomy guide assembly and the cutting guide may
be helpful in properly positioning the guide pin when the oscillating saw must be used.
The Steinmann guide pin should be positioned in a slightly oblique direction (about 20
degrees) from a proximal point on the lateral cortex, three finger-breadths above the
epicondyle, safely off from the trochlear groove, to a distal point on the medial cortex
(Fig. 30-17). A second Homan is placed dorsally to avoid soft-tissue damage, and the
osteotomy is started with the power saw just to cut the cortical bone. It is very
important to do the osteotomy with the blade, the saw, and then the osteotome
parallel and proximal to the guide pin to help prevent intra-articular fracture (Fig. 30-
18).
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After the first few centimeters with the saw, a sharp, flexible, thin osteotome is
introduced and driven in all directions into the femur. This will preserve a hinge of
intact bone by separating the cancellous bone and the anterior and posterior cortices,
ending the osteotomy 0.5 cm before the medial cortex.
FIGURE 30-18
The osteotome must be kept parallel and proximal to the guide pin to help
prevent intra-articular fracture.
FIGURE 30-19
The wedge opener is inserted into the osteotomy and advanced until the opening
corresponds to the planned correction.
Step 5: Wedge Opening

After the distraction by the jack, the opener is introduced and slowly advanced until
the osteotomy has been opened to obtain the planned realignment of the knee (Fig.
30-19); the handle is removed. The surgeon measures the dimension of bone gap
directly on the graduated wedges of the opener and chooses the plate.
Step 6: Plate Fixation
By removing the handle of the opener, the plate can be easily positioned on the
lateral cortex of the femur with the spacer tooth introduced into the osteotomic line
(Fig. 30-20). If the plate does not fit the femur cortex properly, we must precontour it
by modeling with the bending pliers. Before fixing the plate, we make an
intraoperative control of the mechanical axis by means of the special guide rod, long
enough to extend from the center of the femoral head to the center of the ankle,
which we check under fluoroscopy at the passage on the knee joint, approximately the
center of the tibial spine for neutral mechanical axis (Fig. 30-21). When the correction
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is under- or oversized, we choose a different plate with a thicker or thinner tooth as
needed. We then fix the plate with four cortical screws proximal to the osteotomy and
two (rarely three) cancellous screws distally (Fig. 30-22). We recommend a lateral
plate instead of a medial one for an important biomechanical reason. When a normal
knee with a valgus femorotibial angle is loaded in single-leg stance, the lateral femur
is the tension side secondary to the extrinsic varus component of the body weight. In
severe genu valgum, the mechanical axis moves laterally and therefore the convex
medial side is subjected to tensile forces. After osteotomy, the mechanical axis is
again moved medially, which returns the tension side of the knee to the lateral side.
To act as a tension band, the plate must be applied to the lateral femur. Application
of the plate to the medial femur after the osteotomy, as in the closing wedge
osteotomy with the AO 90-degree angled blade plate, violates this principle and will
probably lead to a high rate of failure.
FIGURE 30-20
The new femoral titanium plate with the locking holes.

FIGURE 30-21
In distal femur varus osteotomy we check under fluoroscopy the mechanical

alignment and move the axis to the center of the tibial plateau in a neutral
position.
Step 7: Bone Grafting

We always fill the osteotomic defect with autologous bone harvested from the
omolateral iliac wing or grafts from the bank. According to other authors, bone
grafting is recommended in all opening wedge osteotomies greater than 7.5 mm to
prevent delayed union or nonunion and/or fixation failure. In osteotomies 7.5 mm or
smaller, the decision to bone-graft should be individualized. The correct position of
the plate and grafts is confirmed with AP and lateral radiographs (Fig. 30-23). One or
two drains (one intra-articular) are prepared and the wound is closed in a routine
fashion.
TECHNICAL PITFALLS AND COMPLICATIONS

The risk of intra-articular fracture always exists. This is more often due to a mistake in
positioning the guide pin too close to the joint, leaving a very poor metaphysary bone
stock between the osteotomy and the articular surface; or it may be due to imperfect
finishing of the osteotomy without
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complete interruption of the anterior cortex. More often, however, an intact posterior
cortex produces an articular fracture to stress the knee, in valgus or varus, and open
the osteotomy. Most often it is possible, although not easy, to fix the fracture with the
proximal screws in tibia, or the distal ones in femur, introduced as usual through the
plate.
FIGURE 30-22
The plate is secured to the femur cortex with two (rarely three) distal cancellous
screws and all four cortical screws.
FIGURE 30-23
The correct position of the plate and grafts is confirmed by the postoperative
When the surgeon does not respect the hinge of intact bone, the osteotomy will
dislocate. Checking the fixation and the alignment of the bone under fluoroscopy, the
osteotomy angle looks subluxated with the tibia, or the femur, diaphysis, slipped
laterally or medially, respectively. The way to prevent this technical problem begins
with the proper choice of the site of the osteotomy cut that should be proximal
enough, in the tibia, or distal in the femur, to avoid the maximum step-off of the bone
profile and address a more stable fixation. Mainly, however, an intact bone hinge is
essential for stability; when the hinge is correctly preserved, it prevents any
dislocation. But if the undesired subluxation has occurred, then a possible solution is a
staple fixation by a contralateral incision.
Failure of the hardware, especially the plates, should be a rare event. However, it can
happen that a screw breaks when the patient returns to weight bearing too early
postoperatively against the rehabilitation protocol. But sometimes, a screw breaks due
to a technical pitfall. An imperfect congruence of the tooth plate into the osteotomic
space overloads the screws with a lever arm that the screws cannot resist. An intact
hinge maintains a sort of intrinsic elasticity and, as a spring, closes the osteotomy on
the tooth making the system plate-bone congruent and tight; conversely, a fissured
hinge misses to exert this elastic compression on the opposite side of the bone. The
plate could be loose into the bone and the spacer tooth not in contact with both
cortices. The screws have to support part of the effort to prevent the osteotomy
collapse until the bone heals, but they break because of a fatigue fracture of the
metal before the complete recovery.
The lateral positioning of the â€œTâ€• plate is critical. The osteotomy has to be
perfectly oriented in the sagittal plane, perpendicular to the longitudinal axis of the
femur to have the long arm of the plate completely lying on the bone, just in the
center of the diaphysis. In fact the spacer tooth forms a right angle with the plate that
prevents the correct positioning of the long arm on the bone when the osteotomy is
oblique with the femur. If the vertical arm is not parallel to the diaphysis, the last
upper holes of the plate fall out from the bone, anteriorly or posteriorly to the cortex,
making it very difficult to fix the all screws properly (Fig. 30-24).
Injuries to the vessels are infrequent. Accidental tears to the anterior tibial artery are
reported in the literature, but only when an extensive lateral approach to the tibia is
taken, or to the posterior vessels, which could be safely protected by correct use of a
posterior Homan retractor and keeping the knee flexed during surgery.
Thrombophlebitis and infections are generic complications in common with all the
other surgical procedures involving the inferior limb.
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FIGURE 30-24
Badly oriented osteotomy, oblique to the longitudinal axis, leads to a malposition

of the plate.
Delayed union may occur, but most osteotomies will go on to union with time and
partially assisted early weight bearing. Nonunion is also a possibility. In our series (55
tibial and 21 femoral osteotomies with a minimum follow-up of 4 years) we had no
nonunions. This probably is the result of the systematic use of bone grafts to fill the
osteotomy.
Peroneal palsy was always a potential complication of tibial valgus closing wedge
osteotomy. We have never seen this complication in opening wedge technique.
However, in severe valgus deformities, when distal varus osteotomy is performed, a
transitory peroneal apraxia can occur from overstretching the nerve.
It may be incorrect to include loss of the desired correction as a true complication. In

opening wedge, the bone collapse of the grafts might determine a decrease of the
angular correction, but the new plates have been demonstrated to be effective in
preventing it. Of course continuing degenerative changes and high adduction moment
contribute to a gradual loss of correction as time goes by.
After the operation the knee is immobilized with a range-of-motion brace in full
extension or at slight flexion of about 10 degrees that allows a full ROM when
unlocked. Passive flexion and extension in a continuous passive motion device are
started the day after surgery. The drains are removed 48 hours later. Patients are
allowed to walk with no weight bearing on the operated limb from the second
postoperative day, and they are dismissed from the hospital in 4 to 5 days. When
postoperative knee pain and effusion have been minimized, restoring normal lower
limb ROM and musculotendinous extensibility (with consideration for bi-articular
muscles) is foundational to implementing an exercise program that integrates the
trunk, hip, and ankle muscles into dynamic knee stabilization challenges while
addressing isolated quadriceps femoris deficiencies. Physical therapy intervention with
the knee osteotomy patient requires continual attention to the balance of protection
and function. Although progressive weight bearing and ROM exercises are vital to
recovery, early excessive joint loading and terminal knee flexion-extension with
external loads can compromise the integrity of the surgical realignment. Usually within
the first 4 weeks, the patients are able to completely flex the knee. After 4 weeks (6
weeks in femoral osteotomy), functional weight bearing
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is allowed. Full weight bearing is normally possible after 6 to 7 weeks (8 or 9 weeks in
femoral osteotomy) when the radiographs show satisfactory healing of the bone. Then
greater emphasis needs to be placed on restoring proprioceptive-kinesthetic normalcy
at the involved lower limb. While the rehabilitation program progress to address both
anaerobic and aerobic physiologic energy systems, increasing fatigue resistance as
evidenced by prolonged maintenance of appropriate body control during functional
exercises without apparent discomfort or movement-avoidance patterns assures the
therapist that neuromuscular control for dynamic knee stabilization is improving. All
progressions may need to be delayed for older patients, particularly if they have not
recently participated in a physical exercises program, and rehabilitation should
emphasize active ROM to facilitate articular cartilage nourishment and preservation.
RECOMMENDED READING
Puddu G. Osteotomies about the athletic knee. In: Drez D Jr, DeLee JC, eds. Operative
techniques in sports medicine. Vol. 8, No. 1. Orlando: WB Saunders; 2000.
Puddu G, Fowler PJ, Amendola A. Opening wedge osteotomy system by Arthrex.

Surgical technique. Naples, FL: Arthrex Inc.; 1998.
REFERENCES
1. Coventry MB. Osteotomy about the knee for degenerative and rheumatoid
arthritis. J Bone Joint Surg. 1973;55:23-48.
2. Insall JN, Joseph DM, Msika C. High tibial osteotomy for varus gonarthrosis. J
Bone Joint Surg. 1984;66:1040-1048.
3. Insall JN, Shoji H, Mayer V. High tibial osteotomy: a five-year evaluation. J Bone
Joint Surg. 1974;56:1397-1405.
4. Outerbridge RE. The etiology of chondromalacia patellae. J Bone Joint Surg

(Br). 1961;43:752-757.
5. Steadman JR, Rodkey WG, Singleton SB, et al. Microfracture techniquefor full-
thickness chondral defects: technique and clinical results. Op Tech Sports Med.
1997;7:300-304.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 31 - Computer-Assisted
Opening Wedge High Tibial Osteotomy
31
Computer-Assisted Opening Wedge High Tibial
Osteotomy
Douglas W. Jackson
Blaine Warkentine
After using computer assistance in various knee surgery applications, we found this
technology particularly helpful in obtaining the desired alignment in an opening-wedge
proximal tibial osteotomy. Prior to its use, intraoperative fluoroscopy, radiographs,
and/or visual assessments were the tools to assist in trying to obtain the desired
intraoperative alignment. Computer assistance offers real time alignment information
related to rotation of the limb as well as the exact sagittal and coronal planes for the
osteotomy. This information is obtained with less radiation exposure (to patient, staff,
and surgeon) and has enabled us to use smaller incisions as well as decrease our
overall operative time. This application of computer-assisted surgery is based on and
draws heavily from the contributions and techniques described by Dr. Guancarlo Puddu
(see Chapter 30).
Indications
Our current indications for computer-assisted opening wedge proximal osteotomy
reflect our practice's patient profile. This procedure is considered in our physically
active patients and in combination with certain other knee procedures:
The patient has pain and limitations primarily related to the knee's medial
compartment.
The patient is â€œphysiologicallyâ€• younger and wants to continue high-

demand activities after surgery.
The varus alignment is less then 12 degrees. In our experience caring for an
active patient population, those requiring greater than 12 degrees of
correction usually have more than unicompartment disease. In addition, those
with larger corrections require significant bone grafting at the opening
osteotomy site and run a higher risk of nonunion. In our experience, patients
with high degrees of malalignment represent more individualized approaches;
and while they may benefit from computer-assisted surgery applications, those
techniques used are beyond the scope of this chapter.
The opening wedge osteotomy is in combination with other surgical procedures

in the knee where there is the desire to unload the diseased medial
compartment of the knee. It is used in attempt to provide a more favorable
environment for a desired biologic response or protected healing for another
procedure. These include:
Microfracture
Chondrocyte transplantation, osteochondral autografts and allografts
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Meniscal allograft transplant
Helping to reduce loads associated with varus and posterolateral

ligament insufficiency and/or reconstructions
Anterior cruciate ligament (ACL) reconstruction associated with

symptomatic arthritic medial compartment
Contraindications
Inflammatory arthritis
Tricompartmental disease
A flexion arc of less the 110 degrees (this and those contraindications below
are in our patients whose objective is to remain physically active)
Greater than a 5-degree flexion contracture
Greater than 12 degrees of varus
Tibiofemoral subluxation on standing radiograph
Previous meniscectomy in the contralateral compartment
The history and physical examination establish that the physiologically younger, active
patient being evaluated has symptoms primarily related to the knee's medial
compartment. The knee range of motion (ROM) is documented; weight-bearing
radiographs allow joint space determinations. If an osteotomy is being considered in
those patients with varus alignment that is felt to contribute to their current
complaints or is over weighting their medial compartment, we obtain an overall leg
alignment on long-standing weight-bearing radiograph. In the group of patients who
are also being evaluated for other surgical procedure(s) of the knee, it is part of the
initial evaluation. These include any biologic restoration of their cartilage surfaces or
a meniscus replacement in the overloaded compartment. It is unlikely that a biologic
replacement will survive any better than the native tissues if the abnormal forces
continue to be present. In addition, we consider it in varus alignment associated with
posterolateral and varus instability (see Chapter 24).
SURGERY
Patient Positioning
The patient is positioned on the operating room table in the supine position with a
tourniquet (which we may or may not use for portions or all of the procedure) applied.
The entire lower extremity is prepared and draped separately from the groin distally
in the usual orthopaedic manner. We use an adhesive wrap on the leg to decrease the
bulky surgical stocking, allowing better visualization of the leg alignment.
Technique
An arthroscopic examination is performed and the treatable intra-articular cartilage
and meniscal pathology addressed before the osteotomy. It varies among patients, but
it is common to remove, debride, and contour a portion of a degenerative medial
meniscus and loose or fragmented articular cartilage in the medial compartment. In
addition, loose bodies and intra-articular debris and localized chondroplasties may be
performed (see Chapter 26). Exposed bone that meets the indications in Chapter 27
may be addressed with microfracture or chondrocyte transplantation (see Chapter 29).
Autograft (see Chapter 28) and allograft (see Chapter 33) osteochondral
transplantation, meniscal allograft (see Chapters 10 and 11) or ACL reconstruction (see
Chapter 12) may be performed in conjunction with the tibial osteotomy (see Chapter
24).
There are several companies that offer computer chosen software to assist in proximal
tibial osteotomy, and use different methods for acquiring the position and alignment
of the limb. Ideally, newer versions will allow less invasive data acquisition
methodology. We currently use a system that requires femur and tibia reference
arrays. The distal femur array is placed along the lateral intermuscular septum. For
the tibia they are placed percutaneously along the lateral aspect of proximal one-third
of the tibia (Fig. 31-1). In addition, the system we currently use has a navigated drill
guide (Fig. 31-2), a round tip surface pointer, and a sharp percutaneous pointer.
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FIGURE 31-1
The femoral and tibial reference arrays are in place. A percutaneous pointer is
being used to select the level of the osteotomy on the lateral side of the tibia.
FIGURE 31-2
Using computer-assisted navigation, the reference guide is used to select the

desired slope of the osteotomy and the exact level on the tibia. Once the position
is chosen and confirmed, guides pins are inserted and used with the guide in
making the osteotomy cut.
The operating room is arranged to facilitate the workflow and enhance the visibility of
the reference rays by the camera. We place the computer and the associated infrared
camera at the foot of the bed approximately 2 to 3 meters from the extremity (Fig.
31-3).
We prefer using a fluoroscopic mini C-arm because of the reduced radiation; it can be
used without a radiology technician and we do not wear lead aprons during the
procedure.
Registering Anatomic Landmarks
Eight anatomic landmarks are registered. The sequence of the registration points is
shown in Figure 31-4.
The kinematic hip center is defined by the software as the femur is moved in a circular
motion (Fig. 31-4A). This allows an accurate assessment of the center of femoral head,
which is very helpful in obtaining the desired correction and confirming it.
FIGURE 31-3
When performing the osteotomy on a left lower extremity, we place and drape
the mini-image intensifier so that it can be moved into position for confirmation
and site-selection registration. The infrared camera should have an unobstructed
line of sight of the reference arrays.
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FIGURE 31-4
The sequence of registering the eight anatomic landmarks. A: The computer

calculates three sets of center points within the femoral head. These points are
compared, and the worst center point is discarded. Distance between the
remaining two center points is calculated and read as the error of registration. In
this example the accuracy of the center of the femoral head is 2.31 mm. We
prefer the accuracy for the femoral points to be 3 mm or less. If it is not, we
perform the pivoting registration again until we have registered 3 mm or less.
This corresponds with a deviation of less then 0.7 degrees varus/valgus on a 33-
cm femur. B: The medial and lateral malleoli points are registered using the blunt
pointer. C: The tibial joint line is located at the midcoronal point of both the
medial and lateral tibial plateau and registered using the blunt pointer. D: The
medial and lateral femoral epicondyles are located and registered. E: The
pointer's tip is placed at the tibial tubercle and rotated to represent the rotation
of the lower limb. This establishes the axis that is important in calculating the
osteotomy correction.
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During the registration of the data to calculate the kinetic hip center, it is important
that the pelvis is not moving. If we feel this is a problem in our accuracy, we use an
assistant to stabilize the pelvis and re-enter the data. We start with small circles at
approximately 90 degrees of hip flexion and gradually continue to do the circular
motion until we reach full extension at the hip
Both medial and lateral malleoli points are registered using the blunt pointer. The
software bisects these two points, which defines the distal point of the mechanical
axis of the affected limb (Fig. 31-4B)
The tibial joint line is located with two points, at the midcoronal point of both the
medial and lateral tibial plateau (Fig. 31-4C). The computer then bisects the medial
and lateral registered points to give the center of the tibial mechanical axis.
The medial and lateral femoral epicondyles are located and registered (Fig. 31-4D).
The software bisects these points to develop the distal mechanical axis of the femur.
The rotation for the tibia is registered. The pointer tip is placed at the tibial tubercle
and rotated to represent the rotation of the lower limb. We use the second metatarsal
as the landmark and the apex to the spine of the anterior tibia as an additional check
(Fig. 31-4E).
This rotation must be known for the computer to calculate the midsagittal axis on
which the osteotomy is calculated. The osteotomy is made in an oblique fashion, and
this axis is the pivoting axis on which the software calculates the correction.
Initial Angle Definition

The leg is placed in the fully extended position, and the computer stores the
preoperative leg alignment. Figure 31-5A demonstrates the several choices the surgeon
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has in placing the point at which the mechanical axis will bisect the tibia. Noyes has
recommended surgical corrections with the mechanical axis bisecting the tibia at 62%
of the width, and Fujisawa has recommended a point at 69% of the width. Figure 31-5B
shows a preoperative example of a patient with 8.0 degrees of genu varus and a
mechanical axis (Misculicz line) that bisects the tibial plateau at 11 % of its total
medial-lateral width. For example, achieving a correction to 2.6 degrees of genu
valgus in this patient would occur by placing the mechanical axis bisecting the plateau
at 62% of its width (Fig. 31-5C). We prefer using corrections between 58% and 62%
when combining the osteotomy with other surgical procedures. Based on end points
collected with the pointers, the software calculates the length of the osteotomy that
will leave a 5-mm lateral cortical hinge (Fig. 31-6). After defining the exact point on
the medial tibial cortex for the desired beginning point for the osteotomy, it is used as
the mid point of our surgical incision and exposure to the medial tibia.
FIGURE 31-5
A: The software allows you to select your precise preference for the amount of
correction desired. The surgeon can decide where the mechanical axis (Misculicz
line) bisects the tibial plateau as it crosses the chosen point in the medial-lateral
width of the tibia. B: Demonstrates where the mechanical axis bisects the tibial
plateau in a patient with 8 degrees of genu varum. C: Demonstrates the degree of
correction (2.6 degrees of valgus) if the surgeon chooses the mechanical axis to
bisect the tibia at 62% of it width.
The software sets the cut plane of the osteotomy perpendicular to the mechanical axis
according to the sagittal plane. If desired by the surgeon, additional slope can be fine-
tuned into the planned osteotomy.
The computer will give the necessary data to correct the coronal plane so that the
mechanical axis bisects the tibia at 62% of tibial width (which was defined by the two
landmarks acquired at the joint line). This roughly represents between 2 to 4 degrees
of valgus, depending on the length of the extremity, and can be fine-tuned to any
position desired.
Tibia cutting plane navigation is defined from the start point and end point of the
osteotomy. The software allows the use of a pre-calibrated K-wire drill guide to
navigate two K-wires according to the trajectory given by these previously defined
points (Fig. 31-7A,B).
Through the small surgical incision we use the navigated drill guide and two K-wires.
We penetrate the far cortex with these wires to maintain stability of the K-wires and
to stress relax the far cortex allowing plastic deformation of the far cortex instead of
fracture. The unslotted low profile cutting block is slipped over the K-wires, and the
osteotomy is cut to the desired length. The software has calculated this from the
distance between start and end points minus the desired far cortical hinge. Our
preference is to intermittently check the progression of the saw blade with the mini C-
arm to confirm and assure ourselves of the exact location of the saw blade.
We then apply a valgus force slowly to the extremity until the desired correction is
achieved. (Fig. 31-8).
We pack bone graft into the wedge and select an appropriately sized medial
distraction plate. The real time alignment can be checked at anytime, and once it is
chosen, the appropriate plate is selected. The final alignment is confirmed on the
computer screen, and the correction is depicted prior to closure.
Using the mini C-arm we confirm the placement of the fixation and the length of the
screws (Fig. 31-9). The ligament status, final ROM, and coronal and sagittal alignment
are documented. The surgical wound is closed; a long leg support hose and a knee
immobilizer are applied.
FIGURE 31-6
Using fluoroscopy (mini C-arm), the sharp percutaneous pointer defines and
registers where we want the osteotomy (the oblique bone cut) to start (point a)
and end (point b) in relation to the near and far cortex.
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FIGURE 31-7
A-D: The precalibrated K-wire drill guide is used to navigate two K-wires
according to the trajectory given by previously defined points. These pins are
placed to achieve the wedge size and placement in both the coronal and sagittal
plane. This aids the surgeon to prevent adversely affecting the slope of the tibia
during the osteotomy. Through the small surgical incision we use the navigated
drill guide and the two K-wires. The unslotted low-profile cutting block is slipped
over the K-wires, and the osteotomy is cut to the desired length.
Pearls and Pitfalls
The reference arrays fixed to the femur and tibia need to be rigid. The fixation
pins need to be checked for any extraneous movement and may need to be
advanced for better fixation. Care must be taken with the exact position of the
tips of the fixation pins to prevent possible nerve and vascular injury.
Errors in navigation may occur from poor fixation and if the reference arrays
position are altered during the case.
The camera should be positioned for easy and accurate visualization of the
arrays. This point will eliminate a significant amount of frustration during the
case if visualization is difficult or if the camera needs constant positioning
adjustments.
The registration of the required landmarks for high tibial osteotomy takes a
few minutes but is critical. Each entry point has a purposeful use within the
software's calculations for both the precise osteotomy bone cuts, the degree of
correction, and the AP rotation of the tibia. When these are registered
incorrectly, the information will be significantly altered.
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There are other options in the software for distal femoral osteotomy as well as
closing wedge tibial osteotomy. This chapter has focused on the application of
computer-assisted orthopedic surgery with the opening wedge proximal tibial
osteotomy.
Additionally, you can transfer the femoral array to the proximal wedge after
the osteotomy has been accomplished. In this way you will now be tracking the
true opening of the wedge as opposed to some instability within the knee. Our
opinion is that if we axial load the knee in extension, this issue is negligible. If
you decide to do this, then you must try your best to be as close to full
extension as possible when assessing alignment.
FIGURE 31-8
A: A wedge can be placed and used to open the osteotomy and check the
correction. The computer screen will give the degree of progressive correction,
and the point the mechanical axis is bisecting the tibia (B,C).
FIGURE 31-9
The fluoroscopic images are obtained to confirm the plate and screw placement.
If these are as desired, the arrays are removed and the wounds closed.
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We currently prefer the patients be discharged the day following their osteotomy with
touch weight bearing. It is possible some of these procedures can be done on an
outpatient basis. We delay active ROM for 1 to 2 weeks until the patient returns to the
office for their first postoperative evaluation. We take radiographs at their first
postoperative office visit (Fig. 31-9), and again at 6 weeks postoperatively.
Radiographs are then taken at 3 and 6 months to confirm bony union if necessary.
COMPLICATIONS AND PATIENT INFORMATION

The patient should understand there is a period of limited weight bearing
which depends on the degree of correction and the rate of time for the
individual osteotomy to heal.
There may be incomplete resolution of the patient's knee arthritic symptoms.
There is a likely chance that the patient will undergo a later conversion to
total knee arthroplasty (hopefully not needing that option for at least a 7- to
10-year period).
Potential complications that may occur include delayed unions, nonunions, loss
of correction, return of symptoms, peroneal nerve palsy, vascular injury,
compartment syndrome, intra-articular fracture, patella baja, infection, and
thromboembolism.
RESULTS
We have been using this technique since 2002 and have a limited number of patients.
Our desired alignment corrections have been the best we have obtained to date. If one
looks at the existing literature following proximal tibial osteotomies, it appears the
results are better when the authors adhered to specific indications and obtain the
desired surgical correction. Several long-term studies (primarily closing-wedge
proximal tibial osteotomies) indicate that accurate correction has been the leading
predictor for success. The reported initial high success rates of 70% to 90% seem to
deteriorate with time. Some studies suggest that more than half of high tibial
osteotomies have remained effective at 7 to 10 years. Undercorrection tends to cause
failure from continued symptoms. Overcorrection produces a poor cosmetic result and
can make later total knee replacement more difficult to perform.
There is very little literature on active patients undergoing osteotomy with

concomitant surgeries. The many variables make it difficult to specifically address and
randomize for comparisons. As our techniques improve and particularly for those
active physiologically younger patients wishing to defer replacement, we find the
opening wedge proximal tibial osteotomy with tibial varus is a strong consideration.
The surgical incisions we use are 4.0 to 6.0 cm in length.
Our operative time (including the limited intra-articular arthroscopic

procedures and for the osteotomy) averages 68 minutes.
Total fluoroscopy time averages 12 seconds using a mini C-arm.
Follow-up radiographic assessment in the office setting confirms corrections

within 2.0 degrees of the planned correction.
RECOMMENDED READING
Coventry MB, Ilstrup DM, Wallrichs SL. Proximal tibial osteotomy: a critical long-term
study of eighty-seven cases. J Bone Joint Surg Am. 1993;75:196-201.
Insall JN, Joseph DM, Msika C. High tibial osteotomy for varus gonarthrosis: a long-term
follow-up study. J Bone Joint Surg Am. 1984; 66:1040-1048.
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Kessler OC, Jacob HA, Romero J. Avoidance of medial cortical fracture in high tibial
osteotomy: improved technique. Clin Orthop. 2002;395:180-185.
Marti RK, Verhagen RA, Kerkhoffs GM, et al. Proximal tibial varus osteotomy:
indications, technique, and five- to twenty-one-year results. J Bone Joint Surg Am.
2001;83:164-170.
Sprenger TR, Doerzbacher JF. Tibial osteotomy for the treatment of varus
gonarthrosis: survival and failure analysis to twenty-two years. J Bone Joint Surg Am.
2003;85:469-47 Therapeutic study, level III-2 (retrospective cohort study).
> Table of Contents > Part Four - Articular Cartilage and Synovium > 32 - Allograft
Transplantation for Articular Defects of the Knee
32
Allograft Transplantation for Articular Defects
of the Knee
Petros J. Boscainos
Catherine F. Kellett
Allan E. Gross
The use of fresh osteochondral allografts for osteochondral defects of the knee is
based on a scientific rationale and on long-term clinical experience.
Fresh avascular osteochondral allografts, if harvested within 24 hours of death and

preserved at 4Â° C, show 100% viability of cartilage at 4 days (7,12,33). Although
freezing decreases the immunogenicity of the bone to some degree, it also results in
decreased chondrocyte viability (13,38). Even cryopreservation and controlled rates of
freezing and thawing will not achieve an acceptable degree of cartilage viability (13).
The matrix that surrounds the chondrocytes isolates them from the host's immune cells
and prevents host sensitization (23). The avascular bone remains structurally intact
and mechanically strong until it is replaced by host bone by creeping substitution (30).
Although the osteocytes will not survive unless the graft is vascularized, the use of
immunosuppressive drugs to counter the increase in immunogenicity, that this would
produce, cannot be justified in this setting. In a series of failed fresh osteoarticular
allografts between 12 and 84 months after transplantation, none has demonstrated
histologic evidence of transplant rejection (21). Long-term chondrocyte viability of
fresh osteochondral allografts has been confirmed in a number of studies, even at 17
years after transplantation (9,11,26,30).
Based on that scientific rationale, cadaveric allografts with viable cartilage and
avascular bone, which provides an intact structure until host bone replaces it by
creeping substitution, provide a reconstructive solution for young, high-demand
patients where implants or an arthrodesis is not desirable (Fig. 32-1) (17,36).
Osteochondral allografts provide flexibility in terms of the size of defect that can be
reconstructed. Both femoral and tibial defects can be addressed and where needed,
the allograft meniscus can also be transplanted (17).
Appropriate patient selection is paramount to satisfactory outcome. A number of

patient characteristics have been identified that are predictive of success.
Diagnosis
McDermott et al (25) reviewed the first 100 patients who received fresh, small-
fragment osteochondral allografts for articular defects in and around the knee.
Initially, these grafts were performed for unicompartmental
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osteoarthritis, spontaneous osteonecrosis of the knee, steroid-induced avascular
necrosis of the femoral condyles, osteochondritis dissecans, and most commonly,
traumatic defects. Grafts performed for trauma had the best results (10). Those done
for primary osteoarthritis had poor results. Meyers and coworkers also had poor results
with fresh grafts placed into osteoarthritic knees (27). Garret (15) produced excellent
midterm results in treating patients with osteochondritis dissecans.
FIGURE 32-1
A: Intraoperative pictures of an osteochondral posttraumatic defect of a lateral

femoral condyle of 5 cm diameter and 1.5 cm in depth. B: Intraoperative picture
of the same knee 2 years after grafting. The graft appears healthy.
It may be concluded that posttraumatic defects and osteochondritis dissecans of the

knee are the best indications for fresh grafting (6,8,9,15,17,25,27,30,41).
It has been our experience that the best results with fresh osteochondral allografts are
in patients with unipolar posttraumatic defects and osteochondritis dissecans of the
knee. At our institution osteochondral allografts are no longer used for treating
osteoarthritis, spontaneous osteonecrosis, or steroid induced osteonecrosis, or in
patients with inflammatory arthropathy. If the posttraumatic defect has been present
for long enough to cause severe degenerative changes in the opposing articular
surface, the graft is contraindicated. Also the patient must be compliant and capable
of rehabilitation.
Age
Beaver et al (3), using Kaplan-Meier survivorship analysis, demonstrated that patients
younger than 60 years of age with posttraumatic defects had better results with
osteochondral allografts than patients older than 60 years. Fortunately, the great
majority of posttrauma patients are in their second and third decades.
Site
The use of allografts for bipolar lesions (femur and tibia) has not been reported to be
as successful as unipolar transplants (3,8,17,25,30,40). Patients therefore should be
referred for surgery before secondary changes occur on the nontraumatized side.
Size
Recent advances in other techniques for cartilage repair and resurfacing, such as
microfracture technique, autologous chondrocyte transplantation, osteochondral
autografts, and periosteal grafts (6,
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24,28,29,31,37), have reduced the role of allograft transplantation to defects larger
than 3 cm in diameter and 1 cm deep (20).
Deformity
Most osteoarticular defects have a concomitant malalignment of the limb. In order to
unload the compartment that has received the transplant, this malalignment should be
addressed with a realignment osteotomy of the tibia or femur, as appropriate (18,19).
For this purpose, a lateral-closing wedge high tibial osteotomy is performed for valgus
realignment and a medial-closing wedge, distal femoral osteotomy is used to realign
the limb into varus (20) (Fig. 32-2). Lately, the senior author has been using a medial-
opening wedge high tibial osteotomy for valgus realignment and fixation with a Puddu
plate (32) (Fig. 32-3). If indicated, the osteotomy should be performed at the same
time as the osteochondral graft transplantation. Delayed osteotomy could be reserved
as a salvage procedure for a deteriorating graft when the mechanical axis passes
through the grafted compartment in very young patients (20).
The preoperative assessment includes a clinical and radiographic evaluation of the
injured knee. The presence of previous scars or hardware that needs to be taken out
might determine the configuration of the skin incision.
FIGURE 32-2
Lateral tibial plateau allograft and distal femoral varus osteotomy. A: Old
fracture of lateral tibial plateau with secondary valgus deformity. B: The plateau
is resected to healthy cancellous bone, and the lateral plateau osteochondral
allograft is inserted. C: The lateral tibial plateau allograft is fixed with two 4.0-
mm cancellous screws. A distal femoral varus osteotomy is performed to
decompress the lateral compartment after the allograft is fixed.
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FIGURE 32-3
A: Osteochondral defect of the medial femoral condyle. B: Press-fit osteochondral

allograft implantation in the defect. C: Medial-opening wedge high tibial
osteotomy and fixation with a Puddu plate.
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Routine views of the knee and a 3-foot standing radiograph will provide the data
necessary to estimate the location, size, and structure of the defect and required
graft. A careful assessment for the presence of degenerative changes and of the need
to correct alignment must be done. The biomechanical axis is used to evaluate
deformity and plan correction. Computed tomography (CT) scans and magnetic
resonance imaging (MRI) are not required routinely but, if available, can help define
the defect.
In some patients, previous arthroscopic data might help evaluate the size of the
defect, secondary degenerative changes, and status of the opposing surface.
Graft Procurement and Handling

The local organ procurement agency (in our case The Trillium Gift of Life
Networkâ€”Ontario's Organ and Tissue Donation Agency) identifies potential donors.
The donor must meet the criteria outlined by the American Association of Tissue Banks
(14). The donors must be less than 30 years old to provide healthy, viable cartilage and
strong bone. Graft procurement is carried out within 24 hours of death, under strict
aseptic conditions with the specimen consisting of the entire joint with the capsule
intact. Before harvesting, standardized radiographs of the knee are obtained, and the
recipient is matched to the donor with regard to size. Appropriate cultures (aerobic,
anaerobic, fungal, and tuberculosis) and blood for serology are taken, but no tissue
typing or HLA matching is performed. The graft is stored in a sealed container in
sterile Ringer's lactate solution at 4Â°C with cefazolin (1 g) and bacitracin (50,000 U)
per liter of solution.
The recipient patient is notified as soon as a donor has been located and is admitted
to the hospital as prearranged. Implantation is performed within 48 hours of the
harvest (graft procurement), but we are willing to extend it to 96 hours based on
previous research (12,22,33,34).
SURGERY
Two surgical teams work simultaneously. One team performs the arthrotomy, while
the other prepares the graft. The graft is prepared on a separate table with a separate
set of instruments. At the time of soft-tissue removal, care must be taken to preserve
the donor menisci and the articular cartilage. The osteochondral fragment is gradually
shaped to fit the defect. It is kept in Ringer's lactate and antibiotics until
implantation.
The patient is lying supine on a radiolucent table. One gram of cefazolin is given
intravenously to the patient on induction of anaesthesia. The leg is prepared and
draped to allow an extensile anterior approach, the old scars are marked, and the
tourniquet is inflated. It is important after draping to have access to the anterior
superior iliac spine by palpation and the ankle to evaluate alignment.
The Surgical Approach

A straight midline incision is performed if possible. This allows access to the defect
site as well as permits a proximal tibia or distal femoral osteotomy, if one is indicated.
Furthermore, the same midline incision can be used for possible future procedures,
thus preserving the skin flap blood supply. If the knee has incisions other than midline,
an alternative approach may be needed to avoid potential skin necrosis. The incision is
approximately 25 to 35 cm long and is centered over the patella; proximally, it
overlies the quadriceps tendon and distally, it overlies the tibial tubercle. Minor skin
flaps are raised to facilitate later closure. A medial or lateral parapatellar arthrotomy
is made, depending on the site of the defect. Where varus deformity exists, we prefer
to perform a medial opening-wedge tibial osteotomy, but in the rare case where a
lateral closing wedge is necessary, the skin incision is carried further distal to allow
reflection of the muscles of the anterior tibial compartment. There is usually no
difficulty doing a standard high tibial osteotomy through this approach. In the case of
a distal varus femoral osteotomy, the medial aspect of the distal femur is approached
through a midline skin incision and a subvastus approach is carried out.
Preparation of the Recipient Bed and Grafting

Once exposure is obtained, the reconstruction is straight forward and quite
conservative. The articular defect is â€œsquared offâ€• with the goal of removing as
little bone as possible. The resection is down to bleeding cancellous bone. Precise
measurements are done and the graft is machined to fit the recipient bed accurately.
The graft is orthotopic and the graft's articular surface has to lie flush
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with that of the host joint. Femoral allografts are usually inserted with a press-fit
technique (Fig. 32-4), whereas tibial allograft stability has to be tested prior to
fixation (Fig. 32-5). This is done by moving the knee slowly into full extension and
flexion before internal fixation. If the graft fits well and is relatively stable in flexion
and extension, it is fixed with two or three 4.0-mm partially threaded cancellous
screws. Enough bone must be retained on the graft to accommodate screw fixation (at
least 1 cm).
FIGURE 32-4
A: Anteroposterior knee radiograph of a medial femoral condyle allograft plug and

an opening wedge high tibial osteotomy fixed with a Puddu plate. The distal
femoral screws are from previous surgery. The opening wedge space has been
filled with a mixture of cortical allograft and autologous bone. B: Lateral
postoperative radiograph of the same knee.
Realignment
According to the preoperative planning a realignment procedure should be carried out.
High Tibial Osteotomy (Medial-Opening Wedge)
If varus alignment exists, a lateralclosing wedge high tibial osteotomy is performed in
concert with a medial femoral condyle graft for valgus realignment (19). Lately the
senior author is using a medial-opening wedge high tibial osteotomy for valgus
realignment and fixation with the Puddu plate (32) (see Fig. 32-4). The characteristic
of this plate is that is has a spacer that fits into the osteotomy site. Calculating the
degrees of correction will establish the width of the spacer to be used. It is important
to aim for overcorrection from the physiologic valgus so that after the correction the
mechanical axis of the lower limb passes 3 to 4 mm into the lateral tibiofemoral
compartment.
A straight midline incision is performed from the upper pole of the patella to 4 to 6 cm
distal to the tibial tubercle. Sharp dissection is done to the sartorius fascia, and the
pes anserinus is retracted.
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A retractor is placed under the medial collateral ligament to expose the posteromedial
aspect of the proximal tibia. This is further facilitated by knee flexion. Another
retractor is placed under the patellar tendon.
FIGURE 32-5
A: Anteroposterior knee radiograph of a lateral tibial plateau allograft and a

distal femoral varus osteotomy showing screw fixation of the lateral tibial
allograft. B: Lateral postoperative radiograph of the same knee.
A 3.0-mm osteotomy guide pin is inserted into the tibia from medial to lateral within 1
cm of the lateral cortex. The use of fluoroscopy is advised at this point. The pin should
be 1 to 2 cm from the joint line. An osteotomy guide assembly is mounted onto the
guidewire. A parallel guide sleeve assembly is inserted into the osteotomy guide in
such a way so that it reproduces the existing anteroposterior slope of the tibia. Two
distal pins are passed through the parallel guide sleeves (above the tibial tubercle)
within 1 cm of the lateral tibia cortex. The sleeves and the osteotomy guide assembly
are removed, and the cutting guide is placed over the pins. An oscillating saw is used
to perform the osteotomy within 1 cm of the lateral cortex. Osteotome blades are
used to complete the lateral aspect of the osteotomy but we aim to keep the lateral
cortex intact. The osteotomy is confirmed by fluoroscopy. The osteotomy wedge is
inserted with a mallet to the predetermined correction. Autologous bone graft from
the iliac crest is packed into the osteotomy space. The plate is inserted between the
wedge tines. The plate usually sits just anterior to the medial collateral ligament. Two
6.5-mm screws cancellous screws are inserted proximally. The tines are removed and
two 4.5-mm cortical screws are inserted distally. More bone graft is packed in the
osteotomy space if necessary. Anteroposterior and lateral radiographs are taken to
confirm staple position and adequate correction.
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Distal Fentoral Varus Osteotomy

The distal femoral varus osteotomy that is performed concurrently with a lateral tibial
plateau graft is a closing wedge osteotomy (18) (see Fig. 32-5). The medial aspect of
the distal femur is approached through a midline skin incision and a subvastus
approach is carried out. The fascia over the vastus medialis is incised. The muscle is
dissected from the medial intermuscular septum and reflected anteriorly and laterally,
exposing the medial aspect of the distal femoral shaft and the medial femoral condyle.
A medial arthrotomy is made to enable visual control of the guidewires during the
procedures.
The knee is flexed to 90 degrees' and a guidewire is inserted across the joint space
from medial to lateral through the medial arthrotomy resting on the most distal aspect
of the condyles. A second guidewire is inserted into the medial femoral condyle,
parallel to the first guidewire and about 1 cm proximal to the femoral articulating
surface. A third guidewire may be inserted through the patellofemoral compartment
to mark the anterior part of the femoral articular surface. An anteroposterior
radiograph or a view with an image intensifier is then made to confirm that the second
guidewire is parallel to the articular surface. This is very important as the second wire
guides the blade plate into the femoral condyles. The amount of correction achieved
after the osteotomy depends on the angle of the blade plate with the condition that
the blade is parallel to the femoral articular surface.
If the guidewire is in the correct position, three 4.5-mm drill holes are made on the
cortex of the medial femoral condyle, along the line where the chisel will be inserted,
to prevent comminution. The chisel is then inserted in the anterior half of the medial
femoral condyle (to ensure that the plate is going to sit on the femoral shaft) at a
point 2.0 to 2.5 cm from the distal femoral articular surface and to a depth of 50 to 70
mm, depending on the size of the distal part of the femur. The chisel is kept parallel
to the second guidewire, which is parallel to the articulating surfaces. It is also aimed
10 to 15 degrees posteriorly to avoid cutting out at the anterior aspect of the lateral
femoral condyle. The plate holder is used to guide the chisel and, as a guide, to obtain
correct apposition of the plate to the long axis of the femur. After insertion of the
chisel, anteroposterior and lateral radiographs are made if there is any doubt as to the
position of the chisel. Visualization through the medial (or lateral) arthrotomy can
confirm that the chisel has not penetrated the intercondylar notch or the anterior part
of the femoral articular surface.
The femoral osteotomy is done from the medial side, just proximal to the adductor
tubercle and the anterior part of the femoral articular surface, which may be easily
palpated through the medial (or the lateral) arthrotomy. Using methylene blue, a line
is drawn on the medial part of the femoral cortex parallel to the long axis of the
femur, to extend proximally and distally to the line of the osteotomy. This line is used
to ensure the correct rotational alignment of the femur after completion of the
osteotomy.
The cut in the medial part of the femoral cortex is made with an oscillating saw. The
lateral part of the cortex is perforated at several points with a drill bit and, if needed,
a small osteotome. This allows easier correction of the deformity while it prevents
lateral translocation of the proximal fragment. A medially based wedge of bone is then
removed from the proximal femoral fragment. The base of the wedge should be 5 to
10 mm wide, or just wide enough to allow a 90-degree angle between the chisel and
the medial part of the femoral cortex after the wedge is removed. With our
technique, only a small wedge of bone is removed initially; the proximal cortical
fragment can be easily impacted into the cancellous bone of the distal fragment
because the diameter of the bone proximal to the osteotomy differs from that distal to
the ostetomy. This allows an increase in the angle of the osteotomy if desired; it also
provides more stability and promotes more rapid healing of the osteotomy.
The 90-degree offset dynamic compression blade plate is then inserted. Three sizes of
the offset (10, 15, and 20 mm) are available. Therefore, selection of the plate
depends on the length of blade and the degree of offset that is required, both of
which can be measured from the radiographs, using the available templates for these
plates. If, after inserting the blade, the plate cannot be brought into contact with the
medial part of the femoral cortex, more bone is removed from the base of the wedge
until contact is made. The bone that is removed from the wedge is cut into small
pieces and used for grafting along the medial part of the femoral cortex. The screws
are inserted according to the principles of the dynamic compression plate. The medial
part of the femoral cortex and the distal femoral articular surface are then at 90
degrees to each other. This results in a tibiofemoral angle of approximately 0 degrees,
which is the desired position. The vastus medialis is fastened back to the medial
septum with interrupted sutures (Figs. 32-6 and 32-7).
Before closure, the knee is checked again for full range of motion, stability, and
alignment.
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FIGURE 32-6
Technique of distal femoral varus osteotomy. A: Valgus knee. B: A guidewire is
inserted parallel to the transcondylar axis of the distal femur 1 cm from the joint
line and confirmed on a radiograph. C: Seating chisel for the blade plate is
inserted 2.5 to 3 cm from the joint line, parallel to the guidewire. D: Osteotomy
is performed parallel to the chisel at least 1.5 cm proximal and a 5-mm based
wedge is removed. E: Osteotomy is held with a 90-degree offset blade plate.
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FIGURE 32-7
A: Anteroposterior radiographs of the right knee in a 31-year-old woman 2 years

after open reduction and internal fixation of the lateral tibial plateau fracture.
The lateral tibial plateau has collapsed, causing a secondary valgus deformity of
the knee. B: Postoperative standing radiograph in the same patient at 8 years. A
fresh lateral tibial plateau allograft has been fixed with three screws. A distal
varus femoral osteotomy was done in conjunction with the allograft. The
alignment of the knee is well corrected. The allograft is united and no evidence
of resorption or fragmentation, and the joint space is maintained.
Closure
The tourniquet is deflated and careful hemostasis is done. The surgical site is well
irrigated. The soft tissues are closed in layers with interrupted absorbable sutures over
a vacuum drain. A Jones-type bandage is applied supplemented by an above knee full
leg cast.
Pearls and Pitfalls

Tibial Graft Fixation
When the graft is fixed to the recipient site with screws, we drill the first hole with a
2.5-mm drill bit and leave it in place while drilling the second hole. After inserting the
screw into the second hole, the first drill bit is taken out and replaced by a screw. This
sequence provides good stabilization of the small fragment while drilling the holes for
the screws. Make sure that the screws do not interfere with other hardware, do not
protrude into the soft tissues, and are not close to the articular surface.
Realignment Procedure
The graft itself should not be used to correct alignment. This is achieved by an
osteotomy either before or at the time of allograft implantation. This decision
depends on whether the graft involves the same side of the joint as the osteotomy.
When a realignment procedure is done at the same time of grafting, it should be
carried out after graft fixation. If the realignment procedure involves the same side of
the joint as the graft, is should be carried out several months before transplantation
to allow sufficient time for revascularization of host bone.
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The drain is taken out after 24 hours, and the patient receives intravenous cefazolin
for 5 days. The limb is placed in an above knee cylindrical fiberglass cast after 2 or 3
days. At 2 weeks the cast is removed, the patient is fitted with a hinged knee brace,
and physiotherapy is begun. Physiotherapy consists of active and active-assisted range
of motion exercises, isometric strengthening exercises, and non-weight-bearing
ambulation with crutches for 3 months. No resisted exercises are performed until
there is radiographic evidence that the osteotomy is healed.
RESULTS
The use of fresh osteochondral allograft for posttraumatic joint defects is based on
long-term clinical and experimental evidence of maintenance of viability and function
of chondrocytes as well as replacement of the grafted bone by host bone (17). Recent
advances in other techniques for cartilage repair and resurfacing, such as
microfracture technique, autologous chondrocyte transplantation, osteochondral
autografts, and periosteal grafts (5,24,28,29,31,37), have reduced the indication of
allograft tissue transplantation to defects larger than 3 cm in diameter and 1 cm in
depth.
There are some disadvantages with the use of osteochondral allograft tissue. A well-
organized transplant program is necessary. The surgery cannot be performed on an
elective basis. Disease transmission in fresh allograft remains a concern. Risks are
described to prospective recipients as equal to those for homologous blood transfusion
(HIV 1:493,000 [39], HCV 1:103,000, HBV 1:63,000 [35]), although there are published
estimates of lower risk (4). If the antigen test for viruses becomes precise enough to
eliminate the window period for detection to less than 24 hours, then allograft tissue
will become the tissue of choice for all the previously mentioned techniques because
there is no sacrifice of host tissue and it is less expensive.
There are several advantages in using allograft tissue to repair osteochondral defects.
There is no donor site morbidity. The exact size and shape of the osteochondral defect
may also be duplicated using allograft tissue, and the use of multiple grafts is
obviated.
Encouraging mid- to long-term results have been published related to the use of fresh
osteochondral allograft for posttraumatic defect of the knee joint. In a recent report,
survival of fresh femoral allografts has been reported to be 74% at 15 years with 61% of
the patients achieving excellent or good functional outcomes. These results include
development of degenerative joint disease as a cause of failure (20). Agnidis et al
compared the Short Form-36 scores of 47 patients with transplants to large articular
defects with normative data from an age-matched group. At an average of 12 years
follow-up, the patients with transplants had favorable results in every category, and
93% considered their operation a success (1).
In an earlier study performed at the authors' institution (16), 126 knees of 123 patients
with osteochondral defects secondary to trauma (111 cases) or osteochondritis
dissecans (15 cases) were reviewed. The average age was 35 years (range, 15 to 64);
there were 81 males and 42 females. The defects were located in the tibial plateau
(55 lateral, six medial, and two combined medial and lateral), femoral condyle (27
medial and 23 lateral), bipolar tibial and femoral (seven lateral and one medial
compartment) and patellofemoral (one in patellar groove of the femur and one in the
patella). The grafts, which were between 8 and 40 mm thick, were fixed to good
bleeding cancellous bone after resecting the defect. In 47 cases the meniscus was
included in the transplant. Sixty-eight knees underwent osteotomy to correct
alignment (37 distal femoral, 31 upper tibial). Patients were assessed clinically pre-
and postoperatively, using a rating score based on subjective and objective criteria.
Radiographic assessment included alignment, graft union, fracture and resorption,
joint-space narrowing, and osteoarthritis.
The average follow-up was 7.5 years (range, 1 to 22). Failure was defined as a
decrease in knee score or the need for further surgery. Kaplan-Meier survivorship
analysis demonstrated 95% successful results at 5 years (95% confidence limits: 87 to
98), 71% at 10 years (95% confidence limits: 56 to 83), and 66% at 20 years (95%
confidence limit: 50 to 81) (Fig. 32-8). Among 18 failures, one patient had an
arthrodesis, eight had total knee replacement, one had removal of the graft, and eight
experienced failure because of a decrease in score but still retain these grafts. The
success rate was 85%.
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FIGURE 32-8
Kaplan Meier survivorship curve with 95% confidence limits (Greenwood's

method).
COMPLICATIONS
In the previously described prospective cohort, five complications were noted and
included three stiff knees, one wound hematoma, and one rupture of the patellar
tendon. Long-range analysis reveals a statistically significant relationship of failure
with bipolar grafts (p <0.05) and patients receiving worker's compensation (p = 0.0396)
but no significant relationship with other factors such as osteotomy, meniscus
transplant, sex, and medial and lateral side of the knee.
In addition, analysis of variance in successful cases did not show any statistically
significant effect of patient age or sex, postoperative complications, or preoperative
scores. Radiographic assessment of 18 patients experiencing failure showed four
collapsed grafts, seven patients with loss of joint space, and 10 with significant
osteoarthritis.
Except for two instances of questionable union, all grafts solidly united to the host
bone 6 to 12 months after surgery. Among clinically successful patients, we noted five
instances of mild graft collapse (less than 3 mm), 11 of decreased joint space, and 18
of osteoarthritic changes.
Although most patients experiencing failure showed radiographic changes of collapse

(four), joint-space narrowing (seven), and osteoarthritis (13), similar changes were
found in some patients in whom the procedure was successful.
In a functional outcome study, the SF-36 scores of 47 patients with successful

transplants were compared to normative data from an age-matched group (1). The
greatest discrepancies in the SF-36 scores were in the categories of physical
functioning and â€œrole physical.â€• Despite this, patients returned to a normal
lifestyle, including leisure sports but not competitive sports.
A recent study was performed on 60 patients who were a minimum of 5 years

(average, 10 years) from the time of transplantation of fresh osteochondral allografts
of the femoral condyles (2). Excellent long-term survival has been demonstrated, with
85% (confidence limits, 41% to 100%) of cases surviving without further surgery at 10
years, and a projected survivorship of 74% at 15 years. Of the 12 patients with more
than 15 years of follow-up, only four have failed. Of equal importance is the mean 10-
year Hospital for Special Surgery (HSS) score of 83, with 84% of patients reporting good
to excellent overall outcomes. The mean score for pain was 28 (mild pain requiring
occasional analgesics), and most patients were able to walk more than 1 mile with few
problems.
Overall, the high success rate in mid- to long-term follow-up of this procedure, which
not only does not compromise salvage surgery but facilitates it by restoring bone
stock, makes it an appropriate procedure for unipolar osteochondral defects of the
knee secondary to trauma, or for osteochondritis dissecans in properly selected
patients.
REFERENCES
1. Agnidis Z, Stimec J, Krajbich J, et al. Health-related quality-of-life following
fresh osteochondral allograft of the knee: a minimum five-year follow-up. J Bone
Joint Surg. 1999;81B(suppl 1):106.
2. Aubin PP, Cheah HK, Davis AM, et al. Long-term follow-up of fresh femoral
osteochondral allografts for posttraumatic knee defects. Clin Orthop Relat Res.
2001;391(suppl):S318-S327.
3. Beaver RJ, Mahomed M, Backstein D, et al. Fresh osteochondral allografts for

posttraumatic defects in the knee. A survivorship analysis. J Bone Joint Surg.
1992;74B:105-110.
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4. Buck BE, Malinin TI, Brown MD. Bone transplantation and human
immunodeficiency virus. An estimate of risk of acquired immunodeficiency
syndrome (AIDS). Clin Orthop Rel Res. 1989;240:12-136.
5. Buckwalter JA, Mankin HJ. Articular cartilage repair and transplantation.

Arthritis Rheum. 1998;41:1331-1342.
6. Bugbee WD, Converty FR. Osteochondral allograft transplantation. Clin Sports

Med. 1999;18:67-75.
7. Campbell CJ, Ishida H, Takahashi H, Kelly F. The transplantation of articular

cartilage. An experimental study in dogs. J Bone Joint Surg. 1963;45A:1579-1592.
8. Chu Cr, Convery FR, Akeson WH, et al. Articular cartilage transplantation.
Clinical results in the knee. Clin Orthop Rel Res. 1999;360:159-168.
9. Convery FR, Akeson WH, Amiel D, et al. Long-term survival of chondrocytes in

an osteochondral articular cartilage allograft. A case report. J Bone Joint Surg.
1996;78A:1082-1088.
10. Convery FR, Meyers MH, Akeson WH. Fresh osteochondral allografting of the
femoral condyle. Clin Orthop Relat Res. 1991;273:139-145.
11. Czitrom AA, Keating S, Gross AE. The viability of articular cartilage in fresh
osteochondral allografts after clinical transplantation. J Bone Joint Surg.
1990;72A:574-581.
12. DePalma AF, Tsaltas TT, Mauler GG. Viability of osteochondral grafts as
determined by uptake of S35. J Bone Joint Surg. 1963;45A:565-578.
13. Enneking WF, Mindell ER. Observations on massive retrieved human allografts.
J Bone Joint Surg. 1991;73A:1123-1142.
14. Fawcett KJ, Barr HR, eds. Tissue banking. Arlington, VA: American Association
of Blood Banks; 1987:97-107.
15. Garrett JC. Fresh osteochondral allografts for treatment of articular defects in
osteochondritis dissecans of the lateral femoral condyle in adults. Clin Orthop
Relat Res. 1994;303:33-37.
16. Ghazavi MT, Pritzker KP, Davis AM, et al. Fresh osteochondral allografts for
post-traumatic osteochondral defects of the knee. J Bone Joint Surg.
1997;79B:1008-1013.
17. Gross AE. Use of fresh osteochondral allografts to replace traumatic joint
defects. In: Czitrom AA, Gross AE, eds. Allografts in orthopaedic practice.
Baltimore: Williams & Wilkins;1992: 67-82.
18. Gross AE, Hutchison CR. Realignment osteotomy of the knee. Part 1: Distal
femoral varus osteotomy for osteoarthritis of the valgus knee. Oper Tech Sports
Med. 2000;8:122-126.
19. Gross AE, Hutchison CR. Realignment osteotomy of the knee. Part 2: Proximal
valgus tibial osteotomy for osteoarthritis of the varus knee. Oper Tech Sports Med.
2000;8:127-130.
20. Gross AE, Shasha N, Aubin P. Long-term follow-up of the use of fresh
osteochondral allografts for posttraumatic knee defects. Clin Orthop Relat Res.
2005;435:79-87.
21. Kandel RA, Gross AE, Ganel A, et al. Histopathology of failed osteoarticular
shell allografts. Clin Orthop Relat Res. 1985;197:103-110.
22. Kwan MK, Wayne JS, Woo SL, et al. Histological and biomechanical assessment
of articular cartilage from stored osteochondral shell allografts J Orthop Res.
1989;7:637-644.
23. Langer F, Czitrom A, Pritzker KP, Gross AE. The immunogenicity of fresh and
frozen allogeneic bone. J Bone Joint Surg. 1975;57A:216-20.
24. Mandelbaum BR, Browne JE, Fu F, et al. Articular cartilage lesions of the knee.
Am J Sports Med. 1998;26:853-861.
25. McDermott AG, Langer F Pritzker KP, et al. Fresh small-fragment osteochondral
allografts: Long-term follow-up study on first 100 cases. Clin Orthop Rel Res.
1985;197:96-102.
26. McGoveran BM, Pritzker KPH, Shasha N, et al. Long-term chondrocyte viability
in a fresh osteochondral allograft. J Knee Surg. 2002;15:97-100.
27. Meyers MH, Akeson W, Convery FR. Resurfacing of the knee with fresh
osteochondral allograft. J Bone Joint Surg. 1989;71A:704-713.
28. Moran ME, Kim HK, Salter RB. Biological resurfacing of full-thickness defects in
patellar articular cartilage of the rabbit. Investigation of autogenous periosteal
grafts subjected to continuous passive motion. J Bone Joint Surg. 1992;74B:659-
667.
29. Newman AP. Articular cartilage repair. Am J Sports Med. 1998;26:309-324.
30. Oakeshott RD, Farine I. Pritzker KP, et al. A clinical and histologic analysis of
failed fresh osteochondral allografts. Clin Orthop Rel Res. 1988;233:283-294.
31. O'Driscoll SW. The healing and regeneration of articular cartilage. J Bone Joint
Surg. 1998;80A:1795-1812.
32. Puddu G, Franco V, Cipolla M, et al. Opening wedge osteotomy. Proximal tibia
and distal femur. In Jackson DW, ed. Master techniques in orthopaedic surgery:
reconstructive knee surgery. 2nd ed. Philadelphia: Lippincott Williams & Wilkins;
2000:375-390.
33. Rodrigo JJ, Thompson E, Travis C. Deep-freezing versus 4 degrees preservation

of avascular osteocartilaginous shell allografts in rats. Clin Orthop Relat Res.
1987;218:268-275.
34. Schachar N, McAllister D, Stevenson M, et al. Metabolic and biochemical status

of articular cartilage following cryopreservation and transplantation: a rabbit
model. J Orthop Res. 1992;10:603-609.
35. Schreiber GB, Busch MP, Kleinman SH, et al. The risk of transfusion-
transmitted viral infections. The Retrovirus Epidemiology Donor Study. N Engl J
Med. 1996;334:1685-1690.
36. Shasha N, Aubin PP, Cheah HK, et al. Long-term clinical experience with fresh
osteochondral allografts for articular knee defects in high-demand patients. Cell
Tissue Bank. 2002;3:175-182.
37. Steadman JR, Rodkey WG, Briggs KK. Microfracture to treat full-thickness
chondral defects: surgical technique, rehabilitation, and outcomes. J Knee Surg.
2002;15:170-176.
38. Stevenson S, Dannucci GA, Sharkey NA, et al. The fate of articular cartilage
after transplantation of fresh and cryopreserved tissue-antigen-matched and
mismatched osteochondral allografts in dogs. J Bone Joint Surg. 1989;71A:1297-
1307.
39. Tomford WW. Transmission of disease through transplantation of

musculoskeletal allografts. J Bone Joint Surg. 1995;77A:1742-1754.
40. Zukor DJ, Oakeshott RD, Gross AE. Osteochondral allograft reconstrucion of
musculoskeletal allografts. Part 2: Experience with successful and failed
osteochondral allografts. Am J Knee Surg. 1980;2:182-191.
41. Zukor DJ, Paitich B, Oakeshott RD, et al. Reconstruction of posttraumatic

articular surface defects using fresh small-fragment osteochondral allografts. In:
Aebi M, Regazzoni P, eds. Bone transplantation. New York: Springer-Verlag, 1989.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 33 - Allograft
Osteochondral Plugs
33
Allograft Osteochondral Plugs
William Bugbee
F resh osteochondral allografts are becoming increasingly popular in articular

cartilage reconstruction in the knee. The ability to restore diseased or damaged
cartilage with mature hyaline cartilage tissue is attractive, and the surgical
technique for implanting plugs or dowel-type grafts in simple femoral condyle
lesions is fairly straightforward. A large body of basic science and clinical outcome
data support the use of fresh osteochondral allografts in clinical practice (3 ).
Fresh osteochondral allografts possess the ability to restore a wide spectrum of
articular and osteoarticular pathology, as a result, the clinical indications cover a
broad range of pathology. As is true for other restorative procedures, in addition to
evaluating the particular articular lesion, the careful assessment of the entire
joint, as well as the individual, is important. Many proposed treatment algorithms
suggest the use of allografts for large lesions (>2 or 3 cm) or for salvage in difficult
reconstructive situations where bone loss is also present. In our experience,
allografts can be considered as a primary treatment option for osteochondral
lesions greater than 2 cm (approximately) in diameter, as is typically seen in
osteochondritis dissecans (OCD) and osteonecrosis. Fresh allografts are also useful
as a salvage procedure when other cartilage-restorative procedures, such as
microfracture, osteoarticular transfer system (OATS), and autologous chondrocyte
implantation, have been unsuccessful.
Additionally, allografts often are used for salvage reconstruction of post traumatic
defects of the tibial plateau or the femoral condyle. Other indications for
allografting in the knee include treatment of patellofemoral chondrosis or arthrosis
and in very select cases of unicompartmental tibiofemoral arthrosis (Table 33-1 ).
Relative contraindications to the allografting procedure include uncorrected joint

instability or uncorrected malalignment of the limb. An allograft may be considered
in combination or as part of a staged procedure in these settings. Allografting
should not be considered an alternative to prosthetic arthroplasty in an individual
with symptoms and acceptable age and activity level for prosthetic replacement. In
the younger individual, bipolar and multicompartmental allografting have been
modestly successful. The presence of inflammatory disease or crystal-induced
arthropathy is considered a relative contraindication, as well as is any unexplained
synovitis. The use of fresh osteochondral allografts in individuals with altered bone
metabolism, such as is seen in chronic steroid use, smoking, or even nonsteroidal
anti-inflammatory agents, has not been studied extensively. Unlike other cartilage
procedures, relative size of the lesion (large or small) is not considered a
contraindication to allografting.
Common to all fresh allografting procedures is matching the donor with recipient.
This is done on the basis of size. In the knee, an anteroposterior radiograph with a
magnification marker is used, and a measurement
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of the medial-lateral dimension of the tibia, just below the joint surface, is made
(Fig. 33-1 ). This corrected measurement is used, and the tissue bank makes a
direct measurement on the donor tibial plateau. Alternatively, a measurement of
the affected condyle can be performed. A match is considered acceptable at Â± 2
mm; however, it should be noted that there is a significant variability in
anatomythat is not reflected in size measurements. In particular, in treating OCD,
the pathologic condyle typically is larger, wider, and flatter; therefore, a larger
donor generally should be used. When using allograft plugs or dowels, one is
generally safe with a larger donor. Small donor condyles lead to problems with
matching radius of curvature, particularly for grafts over 20 mm in diameter. Most
femoral condyle lesions can be treated using dowel-type grafts. Commercially
available instruments (Arthrex, Naples, FL) simplify the preparation, harvesting,
and insertion of these grafts, which may be up to 35 mm in size (Fig. 33-2 ).
Perhaps the most important step in preoperative planning is understanding that one
is using human tissue and performing a transplantation. Respect for the donation
process, understanding the recovery, processing, and safety issues are minimum
requirements necessary for the surgeon wishing to use fresh allograft tissue.
1. Chondral lesions: traumatic or degenerative
2. Osteochondritis dissecans
3. Post fracture reconstruction
4. Osteonecrosis
5. Salvage of previous cartilage procedure (microfracture, OATS, autologous

chondrocyte implantation)
TABLE 33-1. Major Indications for Fresh

Osteochondral Allografting of the Femoral Condyle
SURGERY
For most femoral condyle lesions, allografting can be performed through a
miniarthrotomy. In most situations, a diagnostic arthroscopy has been performed
recently and is not a necessary component of the allografting procedure. However,
if there are any unanswered questions regarding meniscal status, or the status of
the other compartments, a diagnostic arthroscopy can be performed prior to the
allografting procedure. Examination under anesthesia is done in the standard
fashion, as is the diagnostic arthroscopy when the surgeon feels this is indicated.
Only rarely do we perform arthroscopy, as adequate data have typically been
collected prior to beginning the process of graft acquisition.
FIGURE 33-1
Radiographic technique for graft sizing. In this example the corrected tibial width is
81 mm (9.41 Ã— 10/11.52).
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FIGURE 33-2
Commonly used instruments for allograft plug technique.
Patient Positioning
The patient is positioned supine, with a tourniquet on the thigh. A leg holder is
valuable in this procedure to position the leg in between 70 and 100 degrees of
flexion, and to access the lesion (Fig. 33-3 ). At least one surgical assistant is
necessary to provide adequate retraction and leg position for working through the
mobile window of the small arthrotomy.
Technique
The fresh graft is inspected to confirm the adequacy of the size match and quality
of the tissue prior to opening the knee joint. It is important to keep the graft moist
during the procedure; generally, it is left in the packaging media when not being
instrumented.
A midline incision is made with the knee in flexion from the center of the patella to
the tip of the tibial tubercle. This incision is preferred in anticipation of further
surgery in the patient's lifetime. This incision is elevated subcutaneously, either
medially or laterally to the patellar tendon, depending on the location of the lesion
(either medial or lateral). A retinacular incision is then made from the superior
aspect of the patella inferiorly. Great care is taken to enter the joint and to incise
the fat pad without disrupting the anterior horn of the meniscus. In some cases in
which the lesion is posterior or very large, the meniscus must be detached and
reflected; generally, this can be done safely, leaving a small cuff of tissue adjacent
to the anterior attachment of the meniscus for later repair. This is most common
for large OCD lesions of the lateral femoral condyle.
FIGURE 33-3
Position of the leg for access to a typical medial femoral condyle lesion.
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FIGURE 33-4
Miniarthrotomy with Z retractor and bent Hohman in notch, exposing the OCD
lesion of the medial femoral condyle.
FIGURE 33-5
Guidewire in place and sizing dowel measuring lesion diameter.
Once the joint capsule and synovium have been incised and the joint has been
entered, retractors are placed medially and laterally. Care is taken in the
positioning of the retractor within the notch to protect the cruciate ligaments and
the articular cartilage. This notch retractor is essential to adequate mobilization of
the patella. The knee is then flexed and/or extended until the proper degree of
flexion is noted that presents the lesion into the arthrotomy site (Fig. 33-4 ).
Excessive degrees of flexion limit the ability to mobilize the patella. If access is
difficult, extending the arthrotomy proximal is warranted. The lesion then is
inspected and palpated with a probe to determine the extent, margins, and
maximum size. After a size determination is made, a guidewire is driven into the
center of the lesion, perpendicular to the curvature of the articular surface. It is
critical to place the guidewire perpendicular to the joint surface. The size of the
proposed graft is determined using sizing dowels (Fig. 33-5 ), the remaining
articular cartilage is scored, and a coring drill is used to remove the remaining
articular cartilage and 3 to 4 mm of subchondral bone (Figs. 33-6 and 33-7 ). In
deeper lesions, the pathologic bone is removed until there is healthy, bleeding
bone. In cases of very deep lesions, the depth of this coring should not exceed 10
mm, and bone grafting should be performed to fill any deeper or more extensive
osseous defects. Our experience suggests that the minimal amount of allograft bone
should be transplanted, and our grafts are rarely more than 5 to 8 mm in thickness.
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The guide pin then is removed, depth measurements are made in the four
quadrants of the prepared recipient site, and a simple map is created (Fig. 33-8
).The corresponding anatomic location of the recipient site then is identified on the
graft. The graft is placed into a graft holder (or alternately, held with bone-holding
forceps). A saw guide then is placed in the appropriate position, again
perpendicular to the articular surface; and an appropriate sized tube saw is used to
core out the graft (Figs. 33-9 and 33-10 ). Prior to removing the graft from the
condyle, an identifying mark is made to ensure proper orientation. Once the graft
is removed, depth measurements, which were taken from the recipient, are
transferred to the graft; this graft then is cut with an oscillating saw, and then
trimmed with a rasp to the appropriate thickness in all four quadrants (Figs. 33-11
and 33-12 ). Often, this must be done multiple times to ensure precise thickness
and to match the prepared defect in the patient.
FIGURE 33-6
The cutting reamer placed over the guidewire with soft tissues well protected.
FIGURE 33-7
Recipient socket after preparation. Note the minimal depth.

FIGURE 33-8
Depth map of recipient site. The position of the free edge is marked.
FIGURE 33-9
Harvesting of the plug from the allograft condyle. The graft and guide are held as
one unit.
The graft now is irrigated copiously with a high-pressure lavage to remove all
marrow elements, and the recipient site is dilated to ease the insertion of the graft
and to prevent excessive impact loading of the articular surface when the graft is
inserted. At this point, any remaining osseous defects are grafted.
FIGURE 33-10
Allograft plug is marked and ready for removal from condyle.

FIGURE 33-11
Depth measurements from the recipient socket are transferred to the plug.
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FIGURE 33-12
Allograft plug is trimmed carefully to the appropriate thickness.

FIGURE 33-13
After lavage the graft is ready for implantation. Correct rotation is determined.
The graft is then inserted by hand in the appropriate rotation and is gently tamped
in place until it is flush (Figs. 33-13 and 33-14 ). Recent studies have shown that
impact loading during insertion of osteochondral grafts causes chondrocyte death (1
). Thus, gentle manual pressure followed by joint range of motion may be a more
reasonable method of graft insertion. If the graft does not fit, refashioning of
either the recipient site (deepening or dilating) or the graft itself (tapering or
thinning) is performed carefully. An excessively tight fit is not necessary. We
accept mismatches of no more than 1 mm from flush with the surrounding joint
surfaces and avoid countersinking of the graft.
Once the graft is seated, a determination is made whether additional fixation is

required. If the graft is captured and not rocking with direct pressure, then no
further fixation is generally required. Absorbable polydioxanone pins or chondral
darts can be used, particularly if the graft is large or has an exposed edge within
the notch (Fig. 33-15 ). We have used small diameter absorbable pins through the
graft surface for years without any deleterious effect. Occasionally, the graft needs
to be trimmed in the notch region to prevent impingement. The knee is then
brought through a complete range of motion to confirm that the graft is stable and
there is no catching or soft-tissue obstruction
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noted. At this point, the wound is irrigated copiously, and routine closure is
performed. We always use a small drain for 24 hours to prevent painful
hemarthrosis from developing.
FIGURE 33-14
The graft is seated and rotation, step-off, and stability are checked. At this time
the joint is carried through a range of motion to completely seat the graft.
FIGURE 33-15
Three PDS pins have been placed through the graft for additional fixation.
Lesions of the trochlea are approached in a similar manner; however, these are
much more technically challenging, as the anatomy of the trochlea is much more
complex, leading to technical issues in creating symmetric matching recipient sites
and donor grafts. In this setting, extensive care must be taken to match the
anatomic location and the angle of approach, as most larger grafts will end up
being elliptical in shape due to the anatomy of the trochlear groove.
Pearls and Pitfalls
Preoperative assessment of the biological and mechanical environment of

the joint is critical. Be sure of the necessity of concomitant procedures,
such as finding and removing loose bodies, meniscectomy or repair, anterior
cruciate ligament reconstruction, and osteotomy.
Adequate confirmation of appropriateness of the lesion for fresh
osteochondral allografting is necessary. Don't be surprised by new or
enlarging lesions.
A careful, informed consent process, including risks of infection and disease

transmission, should be in place.
Confirm graft recipient match both in size and side. Do this before
induction of anesthesia.
Inspect the allograft before making an incision. If it is a small graft, don't

try to make a big plug (30 mm) from it. The curvature will not match the
recipient condyle.
All cutting instruments should contact the condyle (recipient and graft)
perpendicular to the articular surface.
Use reamers rather than drills for all cutting and irrigate copiously.
Save reamings for possible bone graft.
For in-between sizes, always start preparing for the smaller graft. One can
always enlarge the recipient site. Avoid removing too much normal
cartilage.
Two or more plug grafts can be placed overlapping for large lesions
(snowman technique).
Be prepared to use a freehand technique, if necessary, for large or

posterior lesions.
Minimize the osseous portion of the allograft at 3 to 6 mm. Use bone graft
from the recipient site reaming (or the allograft) to fill any deeper defects.
Always remove all soft tissue and perform pressurized lavage of graft prior
to insertion.
Avoid excessive impacting of the graft during insertion. Loose fitting grafts
can be pinned.
Use adjunctive fixation, including absorbable pins, chondral darts, or even

screws.
If the graft ends up too thin, remove it and add bone graft to the base of
the recipient site. We prefer the graft flush, but 1 mm proud is better than
1 mm countersunk.
Adequate and prolonged quadriceps rehabilitation is essential. Often these

patients are multiply operated and have had a long period of disability prior
to allografting.
Nonunion of the graft is very rare. Late fragmentation is a more common

event.
POSTOPERATIVE MANAGEMENT AND REHABILITATION

The rehabilitation of the knee allograft patient in the early postoperative period
includes management of pain, swelling, restoration of limb control, and range of
motion. Assuming rigid graft fixation, patients are allowed touch-down weight
bearing for a minimum of 6 to 8 weeks postoperatively, or until bony union is
determined by radiographs. For femoral condyle allografts, no bracing is needed;
but if an osteotomy is performed, a hinged range-of-motion brace is used for
protection until healing is apparent. Weight bearing is progressed slowly between
the second and fourth month, with full weight bearing using a cane or crutch.
Continuous passive motion is considered optional. The main objective is restoration
of range of motion and strengthening of the quadriceps/hamstrings using isometric
exercises and avoidance of open-chain exercises. We use the stationary bicycle at 4
to 6 weeks in addition to pool therapy if available.
Clinical follow-up includes radiographs at 4 to 6 weeks, 3 months, 6 months, and

yearly thereafter. Careful radiographic assessment of the graft-host interface is
important. Any concern of delayed healing should lead to a more cautious approach
to weight-bearing and other high-stress activities.
Patients will typically experience continued improvement over the first

postoperative year and often continue to demonstrate functional improvement
between years 1 and 2. This often depends on patient motivation, desired activity
level, and adherence to a rehabilitation program. Return to sports and recreational
activities is individualized in the period between 4 months and 1 year (Table 33-2 ).
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1. Non-weight-bearing, 6 to 12 weeks
2. Range-of-motion exercises
3. Quadriceps sets
4. Stationary bicycle at 4 weeks
5. Progressive weight bearing beginning 8 to 12 weeks
6. Sports/recreation at 6 months
TABLE 33-2. Postoperative Management of Femoral

Condyle Allografts
COMPLICATIONS
Early complications unique to the allografting procedure are few. There does not
appear to be any increased risk of surgical site infection with the use of allografts
as compared with other procedures. The most unique issue regarding possible
postoperative complications with fresh allografts relates to transmission of disease
from the graft itself. In our series of over 500 allografts, we have yet to record a
graft-associated bacterial or viral infection.
The use of a miniarthrotomy in the knee decreases the risk of postoperative

stiffness. Occasionally, one sees a persistent effusion, which is typically a sign of
over use, but which may indicate an immune-mediated synovitis. Delayed union or
nonunion of the fresh allograft is the most common early finding. This is evidenced
by persistent discomfort and/or visible graft-host interface on serial radiographic
evaluation. Delayed or nonunion is more common in larger grafts, or in the setting
of compromised bone, such as in the treatment of osteonecrosis. In this setting,
patience is essential, as complete healing or recovery may take an extended
period. Decreasing activities, the institution of weight-bearing precautions, and
possible use of external bone stimulators may be helpful in the early management
of a delayed healing. In this setting, careful evaluation of serial radiographs can
provide insight into the healing process. Magnetic resonance imaging scans are
rarely helpful, particularly before 6 months postoperatively, as they typically show
extensive signal abnormality that is difficult to interpret. It should be noted that
with adequate attention to postoperative weight-bearing restrictions and adequate
graft fixation, delayed or nonunion requiring repeat surgical intervention within the
first year is extremely uncommon. The natural history of the graft that fails to
osseointegrate is unpredictable. Clinical symptoms may be minimal, or there may
be progressive clinical deterioration and radiographic evidence of fragmentation,
fracture, or collapse. Typical symptoms of this type of graft failure include increase
of pain or sudden onset, often associated with minor trauma. Effusion, crepitus, or
focal, localized pain are commonly seen. Careful evaluation of serial radiographs
typically will demonstrate collapse, subsidence, fracture, or fragmentation. Fresh
osteochondral allografts rarely fail due to the cartilage portion of the graft; most
failures originate within the osseous portion. It is important to note that the
allografted joint may suffer from the same pathology that is present in any other
joint, such as meniscus or ligamentous injury. It should also be noted that
radiographic and magnetic resonance abnormalities are commonly noted, even in
well-functioning allografts, and great care must be taken in interpreting and
correlating the imaging studies with clinical findings.
Treatment options for failed allografts include observation, if the patient is

minimally symptomatic and the joint is thought to be at low risk for further
progression of disease. Arthroscopic evaluation and debridement also may be used.
In many cases, revision allografting is performed and generally has led to a success
rate equivalent to primary allografting. This appears to be one of the particular
advantages to fresh osteochondral allografting, in that fresh allografting does not
preclude a revision allograft as a salvage procedure for failure of the initial
allograft.
RESULTS
We have reported the results of 69 knees in 66 patients with OCD of the femoral
condyle, treated with fresh osteochondral allografts (Table 33-3 ). All allografts
were implanted within 5 days of their recovery. Patients were prospectively
evaluated using an 18-point modified D'Aubigne and Postel scale, which measures
function, range of motion, and absence of pain, allotting 1 to 6 points each for a
maximum of 18 points. Subjective assessment was performed with a patient
questionnaire. There were 49 males and 17 females, with a mean age of 28 years
(range, 15 to 54). Forty lesions
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involved the medial femoral condyle and 29 the lateral femoral condyle. An
average of 1.6 surgeries had been performed on the knee prior to the allograft
procedure. Allograft size was highly variable, with a range from 1 to 13 cm2 . The
average allograft size was 7.4 cm2 . Six knees were lost to follow-up. Mean follow-
up in the remaining 63 knees was 4.3 years (range 1 to 14). Overall, 50/69 (79%)
knees were rated good or excellent, scoring 15 or above on the 18-point scale;
11/69 (17%) were rated fair; and 2/69 (3%) were rated poor. The average clinical
score improved from 12.9 preoperatively to 16.1 postoperatively (p <0.01). Six
patients had reoperations on the allograft, one was converted to total knee
arthroplasty, and five underwent revision allografting at 1, 2, 5, 7, and 8 years
after the initial allograft. Thirty-six of 66 patients completed questionnaires: 95%
reported satisfaction with their treatment; 86% felt they were significantly
improved. Subjective knee function improved from a mean of 3.2 to 7.8 on a 10-
point scale.
Meyers
Knee
Multiple
31
3.5
77%
Chu
Knee
Multiple
55
6.2
84% G/E
Ghazavi
Knee
Trauma
126
7.5
85% SVS
Aubin
Femur
Trauma
60
10.0
85% SVS
GÃ¶rtz
Femur
Trauma
43
4.5
88% G/E
Garrett
Femur
OCD
17
2.9
94% G/E
Bugbee
Femur
OCD
69
5.2
80% G/E
Bugbee
Knee
Osteonecrosis
21
5.3
88% G/E
Park
Knee
Osteoarthrosis
37
5.3
76% G/E
Jamali
Patellofemoral
Multiple
29
4.5
52% G/E
G/E,good/excellent; OCD, osteochondritis dissecans; SVS, survivorship.
From Gortz S, Bugbee WD. Allografts in articular cartilage repair. J Bone Joint Surg.
2006;88:1374-1384. With permission.
Author Site of Diagnosis/ Number of Mean Successful

Lesion Indication Patients Follow-up Outcome
(Years)
TABLE 33-3. Selected Outcomesâ€”Osteochondral

Allografting in the Knee
Between 1997 and 2004, 43 patients with isolated cartilage lesions of the femoral
condyle were treated with fresh osteochondral allografting at our institution. The
study population included 23 males and 20 females and had a mean age of 35 years.
Twenty-nine lesions involved the medial femoral condyle, 13 the lateral femoral
condyle, and one was bilateral. All patients had undergone prior surgery. Mean
allograft area was 5.9 cm2 . Thirty-eight of 43 (88%) were considered successful
(score 15 or greater on the 18-point modified D'Aubigne and Postel scale) at mean
4.5 years of follow-up.
SUMMARY
Fresh osteochondral allograft plugs are useful for a wide variety of chondral and
osteochondral lesions of the femoral condyle of the knee. The surgical technique is
straightforward but does require special instruments and careful attention to
detail. Postoperative management is not complex and complications are
uncommon.
RECOMMENDED READING
Gortz S, Bugbee WD. Allografts in articular cartilage repair. J Bone Joint Surg.
2006;88:1374-1384.
GÃ¶rtz S, Ho A, Bugbee WD. Fresh osteochondral allograft transplantation for

cartilage lesions in the knee. American Academy of Orthopaedic Surgeons 73rd
Annual Meeting, Paper A151, 2006.
REFERENCES
1. Borazjani BH, Chen AC, et al. Effect of impact on chondrocyte viability
during insertion of human osteochondral grafts. J Bone Joint Surg Am.
2006;88:1934-1943.
2. Emmerson BC, Jamali A, Bugbee WD. Fresh osteochondral allografting in the

treatment of osteochondritis dissecans of the femoral condyle. Am J Sports
Med. 2007;35:907-914.
3. Gortz S, Bugbee WD. Fresh osteochondral allografting: graft processing and

clinical applications. J Knee Surg. 2006;19:231-240.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 34 - Osteochondritis
Dissecans of the Knee
34
Osteochondritis Dissecans of the Knee
Frederick M. Azar
S. Terry Canale
O steochondritis dissecans (OCD) of the knee is a common entity in both adults and
children. Its incidence has historically been estimated to be between 0.02% and
0.03% based on knee radiographs, and 1.2% based on knee arthroscopy (5 ,11 ,27 ).
More recent investigators have reported much higher frequencies (1 ,21 ).
Osteochondritis dissecans occurs twice as often in males as in females.
Osteochondritis dissecans is most common in the medial femoral condyle, followed
by the lateral femoral condyle; its occurrence in the patella is rare. It primarily
affects young athletic individuals and can significantly impair knee function and
activity and lead to long-term disability (10 ).
The etiology of OCD is controversial and remains unclear. Theories include

ischemia, repetitive microtrauma, familial predisposition, endocrine imbalance,
epiphyseal abnormalities, accessory centers of ossification, growth disorders,
osteochondral fracture, repetitive microtrauma with subsequent interruption of
interosseous blood supply to the subchondral area of the epiphysis, anatomic
variations in the knee, and congenitally abnormal subchondral bone.
It is important to distinguish OCD from acute osteochondral fracture (Fig. 34-1 ). In

addition, an OCD lesion that has separated from a vascular bony bed should be
distinguished from one that has separated from an avascular bony bed
(osteonecrosis). Osteochondritis dissecans is more common in adolescents and
young adults than is osteonecrosis.
Several classification schemes for OCD have been proposed. Bradley and Dandy (5 )
developed a classification for the knee and limited the term OCD to expanding
concentric lesions of the medial femoral condyle that appear during the second
decade of life and progress to concave, steep-sided defects. Cahill and Berg (6 )
proposed a classification scheme based on the appearance of the lesion on
scintigraphy. Dipaola et al (12 ) classified lesions according to their appearance on
magnetic resonance imaging (MRI) and associated specific findings with the
potential for fragment detachment. Guhl (19 ) developed an intraoperative
classification based on cartilage integrity and fragment stability noted at the time
of arthroscopy.
Although the natural history of these lesions has not been conclusively defined,
current evidence suggests that they persist and can lead to further cartilage
degradation (32 ). Primary prognostic factors, in addition to the age of the patient,
include progression, size, stability, amount of subchondral bone present, and
location of the lesion, especially as it relates to weight bearing. Lateral femoral
condylar lesions tend to be more posterior, larger, less stable, and more
fragmented than medial lesions.
Although nonoperative treatment has a limited role in patients with symptomatic

osteochondral lesions, multiple forms of nonoperative treatment have been
described, including periods of immobilization, activity modification, and non-
weight bearing. Prolonged immobilization should be avoided because joint motion
affects articular cartilage attrition. Cahill et al (7 ) noted a 50% failure rate with
conservative treatment in a juvenile population with OCD.
A number of operative procedures have been devised for treatment of OCD, ranging
from simple debridement to autograft/allograft or chondrocyte implantation (Table
34-1 ). Arthroscopy is preferable to arthrotomy; however, it may be necessary to
convert an arthroscopic procedure to an arthroscopic-assisted mini arthrotomy to
obtain adequate fixation of an unstable lesion.
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FIGURE 34-1
Unstable lesion that requires fixation.
See Table 34-1 for surgical treatment options and their indications.
There are few contraindications to surgical treatment of OCD. Contraindications to

specific surgical techniques may include the size of the lesion, with some
procedures suited for only small (<2 cm2 ) lesions; the activity demands of the
patient (high vs. low demand), and the age of the patient (osteochondral allo- and
autografts, autologous chondrocyte implantation usually not indicated in patients
with open physes). The location of the lesion also may be a contraindication to a
particular technique. Patient willingness to comply with postoperative weight-
bearing restrictions also must be considered.
History and Physical Examination
The most common complaint of patients with OCD is pain, which usually is difficult
to localize and typically is present for several months. Patients also may report
swelling or mechanical symptoms such as catching and popping if the lesion is
partially or completely separated. Approximately 80% of juvenile patients have
symptoms for an average of 14 months before initial presentation. A history of
trauma to the knee is given by 40% to 60% of patients (8 ). Examination also may
reveal effusion, joint line tenderness or tenderness over the lesion, limitation of
motion, a positive McMurray's sign, and quadriceps atrophy. The patient may walk
with an externally rotated gait to avoid contact of the medial femoral condyle with
the medial tibial spine. A thorough history and physical examination are necessary
to rule out other causes of knee pathology.
Imaging
Imaging techniques are important not only for diagnosis but for treatment decision-
making. The size, location, and stability of the lesion are critical to choosing the
appropriate technique.
Radiographs
Most OCD lesions can be easily identified on plain radiographs. Anteroposterior,
lateral, notch or tunnel, and patellofemoral views are recommended. Most lesions
occur in the lateral portion of the medial femoral condyle, and they are more
clearly seen on the notch and lateral views. Lesions of the lateral femoral condyle
tend to be more posterior than those in the medial
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condyle (Fig. 34-2 ). The size of the lesion usually can be accurately determined on
plain radiographs. Most small lesions are stable; lesions with a sclerotic margin of 3
mm or more and lesions larger than 0.8 cm2 have an increased risk of being loose.
Comparison radiographs should be obtained in juvenile and adolescent patients
because an OCD lesion may be confused with ossification centers, which may cause
transient symptoms but usually resolve spontaneously within 6 to 12 months. In
addition, OCD is bilateral in approximately 30% of patients (Fig. 34-3 ).
Arthroscopic debri dement
Shaving of loose chondral flaps
Small defects (<2 cm2 ) in areas of limited weight bearing
May provide short-term relief.
Internal fixation with K-wires, screws, biodegradable rods, or pins (arthroscopy or

mini arthrotomy)
Removal of fibrous tissue from crater, antegrade or retrograde internal fixation.

Cancellous graft from proximal tibia or femoral condyle can be added.
Unstable lesion, hinged or loose in crater.
Risk of hardware breakage or loosening, protruding hardware causing articular

wear; second procedure may be needed for hardware removal.
Perichondral/periosteal graft
Graft harvested from distant (perichondrium) or adjacent (periosteum) site,

implanted over defect to serve as joint buffer, source of mesenchymal stem cells
Small defect in non-weight-bearing area in young patient.
Long-term (5 to 10 year) results have been disappointing.
Abrasion chondroplasty/ microfracture
Arthroscopic drilling, microfracture with sharp awl, abrasion with a burr. Combined
with removal of loose bodies and debridement of loose cartilage.
Creation of bleeding surface to access pluripotential stem cells from marrow.
Younger patient Normal alignment Mechanical symptoms (catching, locking,

swelling) Lesion <2 cm2 with stable borders
Good initial treatment

Results in mainly fibrocartilage matrix formation, which has more type I collagen,
cannot withstand compressive and shear loads as well; may degenerate over time.
Osteochondral autograft (OATS, COR, mosaicplasty)
Transplantation of single or multiple plugs from areas with less weight-bearing to

fill defects in areas with more weight-bearing function.
Defect â‰¤2 cm2
Inability to treat large defects due to limited availability of donor cartilage.
Potential problems from donor site.
Osteochondral allograft
Transplantation of single or multiple plugs from fresh allograft femoral condyles
Defect >2 to 3.5 cm2 Isolated lesion Limited donor site Young patient
No morbidity from donor site, improved ability to shape and tailor graft. Risk of
disease transmission.
Autologous chondrocyte implantation (ACI)
Cells obtained from arthroscopic cartilage biopsy, released from matrix and culture
expanded, then placed in defect under periosteal patch.
Younger (15 to 55 years) patient Active patient Single or multiple defects 2 to 10

cm2
Ligamentous or meniscal lesions, joint malalignment, patellofemoral instability

must be corrected; complete meniscal deficit may be contraindication.
COR, chondral osseous replacement; OATS, osteochondral autograft transfer

systems.
Procedure Technique Indications Comments

TABLE 34-1. Surgical Treatment Options for
Osteochondritis Dissecans of the Femoral Condyle
Magnetic Resonance Imaging

Magnetic resonance imaging is recommended to determine the extent of the lesion.
Common MRI characteristics include a high-signal-intensity line or cystic area
beneath the lesion, a high-signal-intensity line through the articular cartilage, and
a focal articular cartilage defect on T2-weighted spin echo and short tau inversion
recovery (STIR) images
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(Fig. 34-4 ). Currently, a spoiled gradient (SPGR) echo sequence using fat
suppression and three-dimensional acquisition is considered the optimal technique
for evaluating articular cartilage lesions. The presence of fluid around the fragment
or focal cystic areas beneath the fragment is the best indicator of instability; the
absence of a zone of high-signal intensity at the interface of the fragment and the
bone is a reliable sign of lesion stability (28 ). Magnetic resonance imaging also is
useful for identification of associated ligamentous and meniscal injuries.
FIGURE 34-2
Recommended radiographic views for evaluation of OCD of the knee. A:

Anteroposterior. B: Lateral. C: Tunnel.
FIGURE 34-3
Bilateral OCD lesions of lateral femoral condyles.
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FIGURE 34-4
MRI evaluation of OCD. A: AP-coronal plane view. B: Lateral sagittal plane view.
Computed Tomography Scanning

Computed tomography (CT) scanning can delineate the bony contours and allow
determination of the amount of cortical bone present in the osteochondral
fragment (8 ). Healing potential is determined by the quantity of bone attached to
the major fragment, and healing can be followed with tomograms until union.
Bone Scanning
Bone scanning (technetium bone scintigraphy) may be helpful to determine the
extent of activity within the lesion and to monitor progress of healing (6 ,37 ). This
has been shown to be more reliable in patients with open physes than in skeletally
mature patients.
SURGERY
Patient Positioning
The patient is placed supine with the prepared and draped limb angled off the
lateral aspect of the table; a leg holder or lateral post can be used.
Technique
Typically, a 30-degree arthroscope is used, although a 70-degree
arthroscope can sometimes be helpful when the lesion is posterior.
A complete and systematic examination of the joint with the 30-degree

arthroscope in the anterolateral portal is the first step in any arthroscopic
procedure about the knee. The articular surfaces of the femoral condyles
should be inspected carefully. Moving the knee from 20 to 90 degrees of
flexion during viewing will help with inspection of the posterior extent of
the lesion.
The articular surfaces should appear smooth except for a slightly raised
irregularity at the borders of the lesion. A probe inserted through the
anteromedial portal is used to carefully probe this irregular line to be sure
there is no break in the continuity of the articular surface overlying the
subchondral bone (Fig. 34-5 ).
Arthroscopic Drilling of the Intact Lesion
If the lesion is intact, a 0.062-inch Kirschner wire is used to perforate it

with multiple holes.
The wire is positioned perpendicular to the articular surface, with the soft
tissues protected by a sleeve or cannula over the wire.
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Drilling of inferior lateral lesions of the medial femoral condyle usually is
done through the anterolateral portal; lateral central lesions may be better
accessed with the wire through the anterolateral portal and the arthroscope
in the anteromedial portal. Large lesions may require drilling through both
portals.
The wire should penetrate the articular surface, the subchondral lesions,
and the underlying bone to a depth of 1 to 1.5 cm to ensure vascular
access. In patients with open physes, imaging is recommended when drilling
to avoid entering the physis (Fig. 34-6 ).
FIGURE 34-5
Arthroscopic view of OCD of knee.
Arthroscopic Removal and Curettage/Microfracture

If the lesion does not have a salvageable fragment, it can be treated with curettage
or abrasion arthroplasty, with or without microfracture for small lesions in non-
weight-bearing areas where fibrocartilage growth may be satisfactory (Fig. 34-7 ).
FIGURE 34-6
Intraoperative fluoroscopy helps avoid the physis in skeletally immature patients.

FIGURE 34-7
Microfracture of OCD lesion of knee.
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Internal Fixation
Two or three 0.062-inch threaded pins can be used for antegrade or

retrograde fixation. The pins should be left long within the adjacent medial
femoral condylar soft tissues so that they can be easily removed once
healing has occurred.
Headless metal screws with variable pitch also can be used but should be
countersunk to the level of subchondral bone. Most authors recommend
removal of screws once healing has occurred (Fig. 34-8 ). Bioabsorbable
screws also can be used to avoid a second procedure for hardware removal
(Fig. 34-9 ).
For smaller, relatively stable lesions, we generally prefer to use an

absorbable fixation device. Implant removal is not necessary, and loading
stress on the fracture site is gradually increased as the implants degrade.
The general technique for insertion is the same regardless of the exact
device used (Fig. 34-10 ).
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Before reduction and fixation of partially or completely detached lesions,
the bed and fragment must have all fibrous tissue debrided with a curet or
shaver (Fig. 34-11 ). If bone grafting is indicated, cancellous bone grafts can
be packed into the base of the crater to obliterate step-off. Cancellous
graft can be obtained from the proximal tibia or a femoral condyle with a
trephine coring needle or similar device.
The graft is placed arthroscopically or through a mini arthrotomy behind the

OCD lesion and packed to make a smooth surface before fixation. This also
can be accomplished retrograde.
FIGURE 34-8
Arthroscopic internal fixation with cannulated headless screws. A,B: Radiographic

appearance. C,D: Arthroscopic views. Screw head should be buried down to
subchondral bone.
FIGURE 34-9
Bioabsorbable screw can be used for fixation.

FIGURE 34-10
Insertion of bioabsorbable pin.
Osteochondral Autograft Transplantation
The osteochondral defect is inspected arthroscopically, and the size of the

lesion is measured.
A set of sizer/tamps with varying head sizes is used to determine the exact
diameter of the defect.
A donor harvester is driven with a mallet into subchondral bone to a depth

of approximately 15 mm, and a bone core is removed by axially loading the
harvester and rotating the driver 90 degrees clockwise and then 90 degrees
counterclockwise.
The recipient harvester and protector caps are inserted into the driver and
placed over the defect at a 90-degree angle to the articular surface to
create the recipient socket.
With the knee in a constant flexion angle, a mallet is used to drive the
recipient tube harvester into subchondral bone to a depth of about 13 mm
(2 mm less than the length of the donor core), and the recipient bone core
is extracted in the same manner as the donor core; the depth of the core is
measured and recorded.
A calibrated alignment stick of the appropriate diameter is used to measure

the recipient socket depth.
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With the harvester stabilized, a mallet is used to lightly tap the end of the
collared pin and drive the bone core into the recipient socket. A stable
knee flexion angle and the position of the harvester must be maintained
during this step.
Multiple smaller autografts (4 to 8 mm) can be implanted (mosiacplasty) in

the same manner for lesions less than 2 cm in diameter.
FIGURE 34-11
Debridement of OCD lesion on lateral femoral condyle.

Osteochondral Allografting
Osteochondral allografts can be used for larger lesions (approx. 2 to 3.5 cm);
however, they have some disadvantages: risk of transmission of disease and
problems with storage and stabilization (Fig. 34-12 ). More recently, fresh articular
allografts have been used, but time-dependent loss of chondrocyte viability is a
concern. Malinin et al (31 ) reported a gradual, significant decrease in the number
of chondrocytes after 9 days; cell outgrowth occurred from specimens stored for up
to 15 days but not in samples stored for longer than 34 days. Using knee condyles
from sheep, Williams et al (43 ) found 98% chondrocyte viability at 8 days, but only
80% at 15 days. In a canine model, Glenn et al (17 ) showed that fresh
osteochondral autografts and allografts were not statistically different with respect
to bony incorporation, articular cartilage composition, or
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biomechanical properties up to 6 months after implantation. Shasha et al (41 )
reported 95% survival of fresh osteochondral allografts at 5 years, 71% at 10 years,
and 66% at 20 years after implantation.
FIGURE 34-12
Osteochondral allografting. A,B: Preparation of recipient bed. C: Removal of graft

from donor bone. D: Harvested allograft. E: Graft in place.
Autologous Chondrocyte Implantation

Cartilage is harvested arthroscopically from the margin of the knee joint, cultured
for 4 weeks, and then transplanted into the damaged area. The cells are held in
position by a membrane of periosteum that is taken from the upper tibia and
sutured into position over the cartilage defect before the cells are injected (Fig.
34-13 ). For deep osteochondral defects, staged open or arthroscopic bone grafting
has been done, followed 4 to 9 months later by autologous chondrocyte
implantation (ACI) (16 ,39 ). To avoid multiple operative procedures, Peterson (38 )
advocated an ACI â€œsandwichâ€• technique in which cancellous bone graft is used
to fill the bone defect, periosteum is sutured above the bone graft at the level of
the subchondral bone with the cambium layer facing the joint, more periosteal
membrane is sutured to the rim of the chondral defect with the cambium layer
facing the defect, fibrin glue is used to secure a water-tight seal, and chondrocytes
are injected between the membranes. Bartlett et al (2 ) described a variation of
this sandwich technique using a porcine type I/III membrane rather than
periosteum. The same authors described a matrix-induced autologous chondrocyte
implantation (MACI) technique that uses a porcine collagen bilayer seeded with
chondrocytes and secured directly to the base of a prepared chondral defect by
fibrin glue.
Pearls and Pitfalls
Flexing the knee as much as possible during arthroscopy can help expose
lesions that may not be clearly visible with the knee flexed at 90 degrees.
When drilling an intact lesion, care should be taken not to destabilize the
lesion by drilling too many holes; the holes should be approximately 3 to 4
mm apart.
When drilling an intact lesion in an adolescent with open physes, image

guidance is recommended to avoid entering the physis.
It is important to remove all fibrous tissue from the crater and the fragment
to optimize healing potential.
If fixation is done arthroscopically, the proper cannula must be used; a mini

arthrotomy should be used if adequate fixation cannot be obtained
arthroscopically.
Screw heads should be buried down to subchondral bone to avoid abrasion.
During autograft or allograft transplantation, when creating the recipient

socket, a 90-angle to the articular surface is maintained to obtain and flush
transfer, and the harvester must be rotated so that the depth markings can
be seen.
If an osteochondral autograft technique is used, the harvest tunnels should

be 1 to 2 mm apart.
A restricted motion brace is used, with the arc of motion controlled to prevent
contact of the tibial articular surface with the lesion. Use of crutches with non-
weight bearing is encouraged until early healing is seen on radiographs, usually 6 to
8 weeks. An unlocked functional brace is then recommended for an additional 2 to
4 months. Sports-specific exercises and return to unrestricted activity is allowed
between 6 and 12 months after surgery, depending on the individual patient and his
or her outcome. Postoperative management and rehabilitation may vary according
to the exact procedure done and the individual patient (20 ).
OSTEOCHONDRITIS DISSECANS OF THE PATELLA

Osteochondritis dissecans of the patella is less frequent than involvement of the
femoral condyles and typically has a worse prognosis. It usually occurs in the second
and third decades and is most often located in the lower half of the patella.
Involvement is bilateral in up to one third of patients and should be distinguished
from dorsal defects of the patella. Fixation can be technically demanding. At
present, arthroscopic excision, curettage, drilling, and debridement (Fig. 34-14 )
are recommended. Surgery is indicated for loose bodies and for lesions with
sclerotic margins. Prognosis is believed to be related to the size of the lesion, with
those in the 5- to 10-mm range reported to have good to excellent results.
However, about half of patients have significant patellofemoral pain at
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long-term follow-up. Overall, the relationship between prognosis and skeletal
maturity for patellar lesions is less clear than that for femoral lesions.
FIGURE 34-13
Autologous chondrocyte implantation. Cells held in position by periosteum sutured

over defect.
FIGURE 34-14
Arthroscopic view of OCD of the patella.
COMPLICATIONS
Complications, in addition to typical postoperative knee complications such as
infection and hemarthrosis, include iatrogenic cartilage damage, hardware
loosening, and abrasion of the articular cartilage by hardware. Aggressive drilling of
an intact lesion can cause fragmentation of the lesion. Metallic screws that are
prominent or become prominent as surrounding articular cartilage wears down can
damage adjacent articular cartilage. Absorbable fixation devices have been
reported to occasionally cause foreign-body reactions. Damage to adjacent
articular cartilage has been attributed to loosening and failure of bioabsorbable
screws that backed out, and unabsorbed screw heads have been found as intra-
aticular loose bodies (13 ,40 ). LaPrade and Botker (25 ) reported two patients who
had fibrocartilage hypertrophy at osteochondral autograft donor sites that caused
knee pain and occasional locking; arthroscopic trimming of the fibrocartilge was
required in both. Delayed union or nonunion of an internally fixed lesion may
require an additional procedure if symptomatic.
RESULTS
Reported results of operative treatment of osteochondral lesions of the knee vary
considerably among techniques and among authors. Levy et al (26 ) reported good
and excellent results at 1-year follow-up in all 15 patients treated with
debridement, while Mithoefer et al (37 ,38 ,39 ) reported 67% good and excellent
results in 48 patients 24 months after microfracture. Makino et al (32 ) found stable
fragments and intact smooth surfaces in 14 of 15 knees at second-look arthroscopy
an average of 50 months after fixation with a Herbert screw.
Several series (9 ,23 ,24 ,29 ) have reported good and excellent results in 83% to
89% of patients after osteochondral autograft procedures. Karataglis et al (24 )
reported improvement in preoperative symptoms in 32 (86.5%) of 37 knees at 3-
year follow-up, Ma et al (31 ) reported 89% good results in 18 patients at 42-month
follow-up, and Chow et al (9 ) reported 83% good or excellent results in 33 patients
at 45-month follow-up. The sizes of the lesions treated with autografts ranged from
1 to 4.1 cm.
In the largest series of mosiacplasty procedures (831 patients), Szerb et al (42 )

reported good to excellent results in 92% of femoral condylar lesions. Gudas et al
(18 ), in a prospective randomized study, compared mosaic osteochondral
autografting with microfracture in 57 young athletic individuals. Functional results
were much better in those with autografts (96% good or excellent) than
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in those with microfracture (52% good or excellent); MRI showed more good results
after autografting (94%) than after microfracture (49%); and more patients returned
to sports activities after autografting (93%) than after microfracture (52%). Bently
et al (4 ), however, compared mosaicplasty with ACI in 100 patients with an
average defect size of 4.66 cm2 and found 88% good and excellent clinical results
after ACI and 69% after mosiacplasty; arthroscopic examination found 82% good and
excellent results after ACI, compared to only 34% after mosiacplasty. In another
prospective comparative trial, Horas et al (22 ) found that results were equally
good after ACI and osteochondral cylinder transplantation, although recovery was
slower after ACI.
Allografting procedures, generally reserved for larger lesions, were reported by

Ghazavi et al (15 ) to be successful in 85% of 126 knees at 7.5 years after fresh
allografts were implanted. Gross et al (16 ) reported failure of 12 of 60 allografts at
10-year follow-up. Mega-OATS procedures have been developed for very large
lesions, but few reports of their results are available and follow-up is relatively
short. Karataglis and Learmonth (24 ) reported functional improvements at 2.5
years after distal femoral allografts in 5 patients with large defects (average of 30
X 30 mm).
Autologous chondrocyte implantation procedures have been reported to be

successful in 66% to 96% of patients. Peterson et al (38 ) reported successful clinical
results in more than 90% of 48 patients at follow-up ranging from 2 to 10 years.
Bentley et al (4 ), in a prospective randomized comparison, found excellent or good
results in 82% of 58 ACI procedures and only 34% of 42 mosaicplasty procedures at
an average follow-up of 19 months. Fu et al (14 ) compared ACI (58 patients) to
debridement (58 patients) for treatment of full-thickness chondral defects and
found that those who had ACI obtained higher levels of knee function and had
greater relief of pain and swelling at 3-year follow-up. Micheli et al (33 ) reported
marked clinical improvements after ACI in 14 patients younger than 18 years who
had OCD lesions of the knee. Bartlett et al (3 ) reported improvements in the
clinical scores of 91 patients who had either ACI or MACI grafting procedures; the
authors concluded that the clinical, arthroscopic, and histologic outcomes of the
two procedures are comparable, but that longer follow-up is needed before MACI is
widely adopted.
RECOMMENDED READING
Gross AE, Shasha N, Aubin P. Long-term followup of the use of fresh osteochondral
allografts for posttraumatic knee defects. Clin Orthop Rel Res. 2005;435:79-87.
Kocher MS, Tucker R, Ganley TJ, Flynn JM. Management of osteochondritis

dissecans of the knee: current concepts review. Am J Sports Med. 2006;34:1181-
1191.
Marcacci M, Konn E, Zaffagnini S, et al. Multiple osteochondral arthroscopic

grafting (mosiacplasty) for cartilage defects of the knee: prospective study results
at 2-year follow-up. Arthroscopy. 2005;21:462-470.
REFERENCES
1. Aroen A, Loken S, Heir S, et al. Articular cartilage lesions in 993 consecutive
knee arthroscopies. Am J Sports Med. 2004;32:211-215.
2. Bartlett W, Gooding CR, Carrington RWJ, et al. Autologous chondrocyte

implantation at the knee using a bilayer collagen membrane with bone graft: a
preliminary report. J Bone Joint Surg Br. 2005;87B:330-332.
3. Bartlett W, Skinner JA, Gooding CR, et al. Autologous chondrocyte

implantation versus matrix-induced autologous chondrocyte implantation for
osteochondral defects of the knee: a prospective, randomised study. J Bone
Joint Surg Br. 2005;87B:40-45.
4. Bentley G, Biant LC, Carrington RW, et al. A prospective, randomized

comparison of autologous chondrocyte implantation versus mosaicplasty for
osteochondral defects in the knee. J Bone Joint Surg Br. 2003;85B:223-230.
5. Bradley J, Dandy DJ. Osteochondritis dissecans and other lesions of the

femoral condyles. J Bone Joint Surg Br. 1989;71B:518-522.
6. Cahill BR, Berg BC. 99m-technetium phosphate compound joint scintigraphy

in the management of juvenile osteochondritis of the femoral condyles. Am J
Sports Med. 1983;11:329-335.
7. Cahill BR, Phillips MR, Navarro R: The results of conservative management of

juvenile osteochondritis dissecans using joint scintigraphy. A prospective study.
Am J Sports Med. 1989;17:601-605.
8. Cain EL, Clancy WG. Treatment algorithm for osteochondral injuries of the
knee. Clin Sports Med. 2001;20:321-342.
9. Chow JC, Hantes ME, Houle JB, Zalavras CG. Arthroscopic autogenous
osteochondral transplantation for treating knee cartilage defects: a 2- to 5-
year follow-up study. Arthroscopy. 2004;20:681-690.
10. Crawford DC, Safran MR. Osteochondritis dissecans of the knee. J Am Acad
Orthop Surg. 2006;14:90-100.
11. Curl WW, Krome J, Gordon ES, et al. Cartilage injuries: a review of 31,516
knee arthroscopies. Arthroscopy. 1997;13:456-460.
12. Dipaola JD, Nelson DW, Colville MR. Characterizing osteochondral lesions by
magnetic resonance imaging. Arthroscopy. 1991;7:101-104.
13. Friederichs MG, Greis PE, Burks RT. Pitfalls associated with fixation of
osteochondritis dissecans fragments using bioabsorbable screws. Arthroscopy.
2001;17:542-545.
P.497
14. Fu FJ, Zurakowski D, Browne JE, et al. Autologous chondrocyte

implantation versus debridement for treatment of full-thickness chondral
defects of the knee: an observational cohort study with 3-year follow-up. Am J
Sports Med. 2005;33:1658-1666.
15. Ghazavi MT, Prtizker KP, Davis AM, Gross AE. Fresh osteochondral allografts
for post-traumatic osteochondral defects of the knee. J Bone Joint Surg Br.
1997;79B:1008-1013.
16. Gillogly SD, Myers TH. Treatment of full-thickness chondral defects with
autologous chondrocyte implantation. Orthop Clin North Am. 2005;36:433-446.
17. Glenn RE Jr, McCarty EC, Potter HG, et al. Comparison of fresh
ostoechondral autografts and allografts: a canine model. Am J Sports Med.
2006;34:1084-1093.
18. Gudas R, Kalesinskas RJ, Kimtys V, et al. A prospective randomized clinical

study of mosaic osteochondral autologous transplantation versus microfracture
for the treatment of osteochondral defects in the knee joint in young athletes.
Arthroscopy. 2005;21:1066-1075.
19. Guhl JF. Arthroscopic treatment of osteochondritis dissecans. Clin Orthop

Rel Res. 1982;167:65-74.
20. Hambly K, Bobic V, Wondrasch B, Van Assche D, Marlovits S. Autologous

chondrocyte implantation postoperative care and rehabilitation: science and
practice. Am J Sports Med. 2006;34:1020-1038.
21. Hjelle K, Solheim E, Strand T, Muri R, Brittberg M. Articular cartilage

defects in 1,000 knee arthroscopies. Arthroscopy. 2002;18:730-734.
22. Horas U, Pelinkovic D, Herr G, Aigner T, Schnettler R. Autologous

chondrocyte implantation and osteochondral cylinder transplantation in
cartilage repair of the knee joint. A prospective, comparative trial. J Bone
Joint Surg Am. 2003;85A:185-192.
23. Karataglis D, Green MA, Learmonth DJ. Autologous osteochondral

transplantation for the treatment of chondral defects of the knee. Knee.
2006;13:32-35.
24. Karataglis D, Learmonth DJ. Management of big osteochondral defects of

the knee using osteochondral allografts with the MEGA-OATS technique. Knee.
2005;12:389-393.
25. LaPrade RJ, Botker JC. Donor-site morbidity after osteochondral autograft
transfer procedures. Arthroscopy. 2004;20:69-73.
26. Levy AS, Lohnes J, Sculley S, LeCroy M, Garret W. Chondral delamination of

the knee in soccer players. Am J Sports Med. 1996;24:634-639.
27. Linden B. Osteochondritis dissecans of the femoral condyles: a long-term

follow-up study. J Bone Joint Surg Am. 1977;59A:769-776.
28. Loredo R, Sanders TG. Imaging of osteochondral injuries. Clin Sports Med.
2001;20:249-2778.
29. Ma HL, Hung SC, Wang ST, Chang MC, Chen TH. Osteochondral autografts
transfer for post-traumatic osteochondral defect of the knee-2 to 5 years
follow-up. Injury. 2004;35:1286-1292.
30. Makino A, Muscolo DL, Puigdevall M, Costa-Paz M, Averza M. Arthroscopic

fixation of osteochondritis dissecans of the knee: clinical, magnetic resonance
imaging, and arthroscopic follow-up. Am J Sports Med. 2005;33:1499-1504.
31. Malinin T, Temple HT, Buck BE. Transplantation of osteochondral allografts

after cold storage. J Bone Joint Surg Am. 2006;88A:762-770.
32. Messner K, Maletius W. The long-term prognosis for severe damage to

weight-bearing cartilage in the knee: a 14-year clinical and radiographic
follow-up in 28 young athletes. Acta Orthop Scand. 1996;67:165-168.
33. Micheli LJ, Moseley JB, Anderson AF, et al. Articular cartilage defects of
the distal femur in children and adolescents: treatment with autologous
chondrocyte implantation. J Pediatr Orthop. 2006;26:455-460.
34. Mithoefer K, Minas T, Peterson L, Yeon H, Micheli LJ. Functional outcome of

knee articular cartilage repair in adolescent athletes. Am J Sports Med.
2005;33:1147-1153.
35. Mithoefer K, Williams RJ III, Warren RF, et al. The microfracture technique
for the treatment of articular cartilage lesions in the knee. A prospective
cohort study. J Bone Joint Surg Am. 2005;87A:1911-1920.
36. Mithoefer K, Williams RJ III, Warren RF, Wickiewicz TL, Marx RG. High-
impact athletics after knee articular cartilage repair: a prospective evaluation
of the microfracture technique. Am J Sports Med. 2006;34:1413-1418.
37. Paletta GA, Bednarz PA, Stanitski CL, et al. The prognostic value of
quantitative bone scan in knee osteochondritis dissecans: a preliminary
experience. Am J Sports Med. 1998;26:7-14.
38. Peterson L. Chondrocyte transplantation. In: Jackson DW, ed.: Master

techniques in orthopaedic surgery: reconstructive knee surgery. Philadelphia:
Lippincott Williams & Wilkins, 2003;353-373.
39. Peterson L, Minas T, Brittberg M, Lindahl A. Treatment of osteochondritis

dissecans of the knee with autologous chondrocyte transplantation:results at
two to ten years. J Bone Joint Surg Am. 2003;85A(suppl 2):17-24.
40. Scioscia TN, Giffin JR, Allen CR, Harner CD. Potential complication of
bioabsorbable screw fixation for osteochondritis dissecans of the knee.
Arthroscopy. 2001;17:E7.
41. Shasha N, Aubin PP, Cheah HK, et al. Long-term clinical experience with
fresh osteochondral allografts for articular knee defects in high demand
patients. Cell Tissue Bank. 2002;3:175-182.
42. Szerb I, Hangody L, Duska Z, Kaposi NP. Mosaicplasty: long-term follow-up.

Bull Hosp Joint Dis. 2005;63:54-62.
43. Williams RJ III, Dreese JC, Chen CT. Chondrocyte survival and material
properties of hypothermically stored cartilage: an evaluation of tissue used for
osteochondral allograft transplantation. Am J Sports Med. 2004;32:132-139.
> Table of Contents > Part Four - Articular Cartilage and Synovium > 35 - Synovectomy for
Pigmented Villonodular Synovitis
35
Synovectomy for Pigmented Villonodular
Synovitis
Edward Y. Cheng
Vineet Sharma
Pigmented villonodular synovitis (PVNS) is a unique condition affecting synovial tissues

in joints, tendon sheaths, and bursa. The disease is classified into three categories: (a)
extra-articular PVNS (also known as giant cell tumor of the tendon sheath), (b)
localized intra-articular PVNS, and (c) diffuse intra-articular PVNS. It is unknown
whether or not this disease is a true neoplasm or reactive in origin (24), but recent
evidence of monoclonality suggests a neoplastic etiology (3,18). The disease may occur
in either young adults or the elderly and commonly presents with symptoms related to
the intra-articular nature of the mass. The knee is the most common joint involved but
any synovial joint including the hip (Fig. 35-1) (33), ankle (Fig. 35-2) (13), shoulder
(Fig. 35-3) (22), or elbow (30) may be affected. Polyarticular disease in children has
also been reported (12,34). Pain and intermittent effusions, either traumatic or
nontraumatic in origin, are caused by the inflammatory synovitis and recurrent
hemarthroses that are typical. In the knee, locking may occur or the disease may be
misdiagnosed as a popliteal cyst.
The mainstay of treatment of PVNS is surgical excision by either open or arthroscopic
means. No medical therapy is known to be effective at eradicating the disease.
Untreated, some patients may develop stable disease but most patients will require
treatment for symptomatic reasons or to prevent the eventual progression to
periarticular erosive cyst formation and subsequent destruction of the cartilaginous
joint surface. In about one third of cases, an aggressive course may ensue with
multiple recurrences despite surgical resection (9). In these cases, radiation may be
considered, administered by either external beam (26) or via intra-articular radiation
synovectomy (11,16). Once joint destruction has occurred, total knee arthroplasty is
the only means of addressing the disabling symptoms; however, a synovectomy must
still be performed as recurrences after knee arthroplasty have been documented
(1,14).
Indications
Symptomatic PVNS
Asymptomatic PVNS
Locally recurrent PVNS
Contraindications (Relative)
Multiply recurrent PVNS
Medical comorbidities precluding surgery
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FIGURE 35-1
A: Coronal fat suppression MRI of PVNS of the hip. Note effusion and dark areas of
hemosiderin deposition. B: Axial T1 MRI of PVNS of the hip. Note how the extent
of disease is seen much more readily than on computed tomography scan (C).
Examination and Evaluation
Patients with PVNS demonstrate evidence of an effusion or a hemarthrosis when minor
trauma results in a hemorrhage. In the localized type, locking due to the presence of
an intra-articular mass is common, and in both the diffuse and localized from, there
may be limited joint arc of motion. A palpable mass is not usually appreciated unless
there are extra-articular disease manifestations. Most commonly, the extra-articular
disease occurs in the popliteal fossa or intercondylar notch (Fig. 35-4). In advanced
cases, findings consistent with degenerative arthrosis are evident.
Imaging Studies
Plain radiographic findings in the early stage of disease are either normal or may show
a displaced suprapatellar fat plane due to presence of an effusion. A capsular-based
noncalcified soft tissue mass may be present (7). In later stages, periarticular cysts are
present and cartilage thinning is seen (4). The findings on magnetic resonance imaging
(MRI) are more dramatic and in many cases diagnostic. A soft tissue mass with
alternating areas of bright signal and dark signal void on both T1 and T2 sequence
represents the fluid (bright on T2) and fat (bright on T1) accumulation as well as
hemosiderin deposits within the soft tissue (dark on both T1 and T2), respectively (Fig.
35-5). In the appropriate clinical setting, these findings are nearly diagnostic of PVNS,
although rheumatoid arthritis may have a similar appearance (23,31). The nodular
form may not always be localized to one location and instead may occur in multiple
compartments within the joint. It is essential to precisely
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determine the extent of the disease on MRI as this will directly affect the surgical
approach to the disease. In doubtful cases, the diagnosis can be established with a
biopsy.
FIGURE 35-2
A: Sagittal T2 MRI image of PVNS of the tibio talar and subtalar joint. B: Sagittal
T1 MRI image of PVNS of the tibio talar and subtalar joint.
FIGURE 35-3
A: Coronal STIR (Short T1 inversion recovery) MRI image of PVNS of the

glenohumeral joint. Note large effusion and synovial thickening. B: Axial T2 MRI
image of PVNS of the glenohumeral joint. Note erosion into the glenoid fossa.
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FIGURE 35-4
Sagittal T2 MRI image of PVNS in an extra-articular location extruding outward

from the intercondylar notch.
FIGURE 35-5
A: Axial T1 MRI. B: Coronal T2 MRI.
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FIGURE 35-5
(Continued) C: Axial fat suppression MRI images of PVNS.
Gross and Histologic Appearance

The intraoperative gross appearance of disease is characteristic. In the diffuse form,
multiple fronds of reddish-brown pigment-stained synovial tissue represent villous
projections floating in synovial fluid. In the nodular form, a thickened reddish-brown
and yellow stained xanthomatous soft tissue mass with cavitary areas of fluid
accumulation is seen and the mass may spread dramatically in the suprapatellar pouch
(Fig. 35-6) or intercondylar notch.
FIGURE 35-6
Clinical intraoperative photo of markedly thickened PVNS with reddish-brown

pigmentation and xanthomatous appearance in the suprapatellar pouch.
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FIGURE 35-7
Histologic photomicrograph (20x power) of PVNS.
Histologically, PVNS is indistinguishable from a giant cell tumor of tendon sheath

(nodular tenosynovitis). A proliferation of round synovial-like cells is present with
occasional multinucleated osteoclast-type giant cells, xanthomatous cells, and
inflammatory cells interspersed throughout the lesion (Fig. 35-7) (5,15).
Specific Approaches
The controversy regarding open versus arthroscopic synovectomy is difficult to resolve.
With either technique, in the diffuse form, the recurrence rate ranges from 8% to 50%
(10,25,32). The only factor which is conclusively known to prevent local recurrence is
complete and aggressive resection of the affected tissues. The benefits of arthroscopic
synovectomy are a quicker rehabilitation and avoidance of arthrotomy and
postoperative knee stiffness. The disadvantages are related to performing an
intralesional excision or debridement, potential for intra-articular spread of disease
converting a localized form to diffuse disease, seeding of portal sites (Fig. 35-8),
difficulty accessing posterior and extra-articular locations of disease, and difficulty in
removal of markedly thickened
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synovial tissue. Advantages of an open synovectomy over the arthroscopic technique
are the ability to do a marginal or en bloc excision of the mass, complete a total
synovectomy as opposed to a only a debridement, and the expeditious nature of an
arthrotomy. Excellent results using cryosurgery in addition to open synovectomy have
been reported (20). However, the open procedure can be technically challenging to
perform adequately through a popliteal exposure and may result in postoperative
adhesions if a normal arc of knee motion is not regained promptly after surgery. No
prospective trials directly comparing these techniques have been performed, and they
would be considerably difficult to power adequately due to the rarity of the disease.
FIGURE 35-8
Axial T2 MRI image of local recurrence of PNVS in prior arthroscopic portal site
adjacent to patellar tendon.
Arthroscopic Approach
Disease is limited to a single location
Accessible arthroscopically
Nodular form
Open Approach
Extra-articular disease
Posterior disease inaccessible arthroscopically
Diffuse form (8,9,19,21).
SURGERY
Positioning, Draping, and Organizing the Operating
Room
When open synovectomy is performed, the operative team must be prepared to
undertake the surgical approach mandated by the anatomic location of disease. If both
the anterior and posterior compartments of the knee are affected, an anterior and
posterior synovectomy is required. In the supine position, a roll is placed beneath the
buttock and a sandbag taped to the table at midcalf level to support the heel while
the knee is flexed. In the prone position, blanket rolls or a spinal frame are adequate
for positioning. It is helpful to have a second table to turn the patient onto after
completion of the anterior portion of the surgery.
Details of Procedure and Avoiding Pitfalls

Anterior Exposure
The anterior arthrotomy is performed first through an exposure similar to total knee
arthroplasty. Using a tourniquet facilitates visualization. Care must be taken to avoid
injury to the menisci upon exposing the knee. Areas where an incomplete resection
may occur include the medial and lateral gutters, beneath the menisci, and in
between the anterior and posterior cruciate ligaments. A bone hook placed within the
femoral notch to distract the femur assists exposing the ligaments. Flexing and
rotation the tibia assist exposing the gutters. Once the anterior synovectomy is
completed, the tourniquet is deflated, hemostasis secured, wound is closed, drapes
removed, the patient is turned prone onto another table, and the knee is reprepped
and draped (Fig. 35-9).
Posterior Exposure
The posterior exposure is usually more challenging for most surgeons as it is performed
less frequently than the anterior exposure and one must dissect out the popliteal
vessels, and tibial and peroneal nerves. With practice, however, one can become
comfortable with this dissection and maneuver around the critical structures with
ease. Under tourniquet control, a gentle â€œSâ€• shaped curvilinear incision is made
with care taken to avoid an abrupt turn in the corners resulting in a corner edge at risk
for necrosis. Ideally, a turn no greater than 45 degrees is fashioned and the flaps are
kept as thick as possible by keeping the plane of dissection deep to the popliteal fascia
before raising the flap (Fig. 35-10). This greatly facilitates the wound closure as well.
The incision can be based from either the lateral or medial border of the thigh
depending on which side has the greatest extent of disease. The semitendinosis tendon
is readily identified in the medial border of the incision (Fig. 25-10B and C). A few
gentle spreading motions with the blunt-tipped scissors allow one to identify the
popliteal vein and artery together. Similarly, the peroneal nerve is found coursing
obliquely toward the biceps tendon. This can then be dissected proximally toward the
tibial-peroneal bifurcation. Vessel loops are placed around the neurovascular
structures to aid in retraction (Fig. 35-11A). Multiple small venous structures around
the popliteal vein will need to be divided when skeletonizing the vein. It is necessary
to dissect a sufficient distance to allow sufficient mobility for retraction without
undue tension. Hemoclips aid in the speed of dissection but have the
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disadvantage of causing artifactual degradation of any subsequent MRI studies that will
likely be necessary for postoperative surveillance. The distal extent of the vascular
dissection is limited by the vessel branches to the gastrocnemius and the origin of the
anterior tibial artery.
FIGURE 35-9
A: Intra-operative picture showing the anterior exposure with extent of synovitis.

B: Same knee after complete anterior synovectomy. C: The synovial mass
removed en masse.
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FIGURE 35-10
A: Intraoperative photo of incision. Note curvilinear incision avoids corner angles

<135 degrees to prevent flap necrosis in corners. Intraoperative (B) and
diagrammatic views (C) of superficial dissection keeping dissection deep to fascial
layer, avoiding dissection through the subcutaneous tissue to keep the flap as
thick as possible, thus avoiding necrosis.
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FIGURE 35-11
Line drawing (A) and intraoperative photo (B) of popliteal exposure with
gastrocnemius muscle heads reflected from origin on posterior femur.
The key to exposing the posterior joint capsule adequately is taking down the
gastrocnemius muscle origins (Fig. 35-11A,B). Depending on the lesion location on
preoperative imaging, one or both gastrocnemius muscle heads will need to be
divided. If there is any question, it is advisable to divide both as the MRI frequently
underestimates the extent of disease in this locale. A right angle clamp is placed
beneath the musculotendinous origin just proximal to the femoral condyle. Cautery is
used to divide the tendon and separate the remaining muscle fibers from the posterior
knee capsule. Extra-articular extensions of PVNS are frequently found in this location.
Tagging sutures placed in the tendon are used to retract the muscle distally and away
from the vessels. Homan retractors placed around the femoral condyle as well as slight
knee flexion may aid in exposure. At this juncture, the posterior knee capsule should
be fully exposed with the vessels coursing in the middle. Another extra-articular site
frequently involved with PVNS is located in the intercondylar notch, posterior to the
posterior cruciate ligament (Fig. 35-12). Retracting both gastrocnemius muscle origins
to either side allows visualization of the ligament of Wrisburg and posterior cruciate
ligament just beyond this structure. A small No. 15 scalpel blade is used to perform
the capsulotomy along the periphery of the femoral condyle. This affords an intra-
articular view of any disease that may be seen as fronds within the joint or attached
to the synovial surface of the joint capsule (Fig. 35-13). The entire capsule and
adjacent synovium can be resected leaving only the posterior cruciate ligament
remaining behind the knee (Fig. 35-14). Again, the posterior menisci must be
protected. The limits of the prior anterior dissection are now encountered in the
gutters and intercondylar notch regions. These areas should be carefully inspected for
any remaining tissue resembling PVNS.
After excising all abnormal tissue, the tourniquet is deflated to ensure satisfactory
hemostasis. A drain is placed deep within the wound prior to reattaching the
gastrocnemius muscles using a
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Kessler-type tendon suture and oversewing with No. 0 nonabsorbable sutures. The
repair should be strong enough to withstand the tension applied when the knee is
straight and the foot is placed in a neutral position. Approximating the popliteal fascia
is facilitated by placing â€œfar-near-near-farâ€• type self-retention sutures to
prevent tearing of the thin fascia. A sound fascial closure is important as this is the
only layer of any significant tensile strength other than the skin, and the wound
closure must be able to tolerate immediate motion postoperatively.
FIGURE 35-12
Intraoperative photo of PVNS tumor mass (highlighted circle) in the intercondylar

notch adjacent to lateral femoral condyle.
FIGURE 35-13
Intraoperative photo of PVNS involving posterior joint capsule, reflected portion

held in forceps (highlighted circle).
Arthroscopic Synovectomy
Performing an adequate arthroscopic synovectomy is considerably more difficult than
most arthroscopic procedures. The proliferative synovitis usually present makes
visualization challenging. Using a tourniquet is essential, and a variable pressure pump
for irrigating solution with epinephrine also is helpful. To afford sufficient visualization
of the entire knee, additional portals other than the conventional 3-portal technique
are required (Fig. 35-15). Both superior medial and lateral portals are needed to
access the suprapatellar pouch, and switching among them with the arthroscope and
operative instruments shaver allows complete excision of both sides of the pouch.
While the standard anterior portals are used, using the midpatellar lateral or medial
portal may assist in visualizing the lateral and medial gutters, respectively (28,29).
The intercondylar notch can be accessed using conventional inferior medial and lateral
portals. To see the posterior regions adequately, a 70-degree arthroscope is necessary,
and a posterior medial or lateral portal allows the arthroscopist an additional port for
operative instruments in addition to the arthroscope (Fig. 35-16). Illuminating the
posterior portal sites and using a spinal needle may minimize an injury to the
infrapatellar branch of the saphenous nerve or the peroneal nerve. A motorized full-
radius shaver in oscillating mode is a necessity for performing the synovectomy. The
larger 5.5-mm shaver is used as it is more aggressive, unless the anatomic confines of
areas such as beneath the menisci or posterior recesses require using the smaller 4.5-
mm shaver. Additional techniques helpful in the posterior recesses include flexing the
knee and using the arthroscopic sheath for fluid inflow. If hemorrhage is considerable
after tourniquet deflation, a drain should be considered.
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FIGURE 35-14
Line drawing (A) and intraoperative photo (B) of posterior knee joint after
excision of the posterior joint capsule, synovium and PVNS.
FIGURE 35-15
Arthroscopic portal sites for midpatellar lateral and midpatellar medial

placement to facilitate visualization of lateral and medial gutters.
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FIGURE 35-16
Arthroscopic view of PVNS.
The postoperative management of the synovectomy patient depends on the type of
procedure performed. For open synovectomy, the goals are as follows:
Early and aggressive rehabilitation to gain range of motion
Detection of any wound healing problems
The institution of early continuous passive motion devices the first day after surgery
and an epidural catheter for some patients are quite beneficial. The drain is removed
the first or second day after surgery. A supervised course of physiotherapy after
hospital discharge is advisable until an arc from 0 to 90 degrees is attained and the
patient is capable of a home therapy program. Protected weight bearing with crutches
for 4 weeks is recommended until the gastrocnemius tendinous reattachment has
healed sufficiently. Afterward, a strengthening program with progressive resistance
exercises is commenced. Prophylaxis for deep venous thrombosis using anticoagulation
is not routinely incorporated unless there are extenuating factors such as a
hypercoagulable state or prolonged vascular retraction.
For the arthroscopic synovectomy patient, a compression dressing is left on for the
first few days, followed by range of motion exercises. A long-acting anesthetic in
instilled into the knee upon would closure. Immediate weight bearing is allowed as
tolerated.
For either surgical procedure, careful assessment of the vascular and neurologic status
of the limb after surgery is necessary. While more dissection takes place in the open
procedure, the neurovascular structures are also at risk for injury during the
arthroscopic procedure. An occult injury is more likely in a closed procedure and
recognition may be especially problematic if the patient is discharged as an outpatient
postoperatively.
Follow-up Surveillance
Pigmented villonodular synovitis has been reported to recur as late as 17 years after
prior excision (27). A baseline MRI scan after the surgical changes in the knee have
resolved, about 6 months postoperatively, is useful for comparison to any future
studies that may be necessary if there is a question of recurrence. Recurrent pain or
swelling of the knee most frequently will herald the onset of a relapse. Taking
adequate time to educate the patient both pre- and postoperatively about the natural
history of PVNS and its propensity for recurrence is the best means of monitoring for
relapse and mitigating negative feelings on behalf of the patient.
If a local recurrence is diagnosed, management usually involves repeat excision. A

careful assessment of the MRI is necessary to determine the extent of recurrence. If
repeat excision is not a possibility, then radiation synovectomy or moderate dose
external beam radiation are alternative means of treating this disease. Once joint
destruction has already occurred, either arthrodesis or arthroplasty become
considerations depending upon the age of the patient.
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COMPLICATIONS
The main complications encountered with open synovectomy are:
Wound healing issues with the posterior incision
Arthrofibrosis and limitation of motion
If there is a localized area of flap necrosis despite taking the precautions described
previously, conventional treatment with saline gauze dressing changes is usually
satisfactory. If the patient has not regained a 0 to 90-degree arc of motion by the first
month after surgery, a knee manipulation should be considered.
The main complications associated with the arthroscopic procedure are:
Occult neurovascular injury
Persistent pain and effusion
A vascular injury requires immediate attention and evaluation. Limiting activities and
immobilizing the knee can manage any persistent pain and swelling that is
bothersome.
RESULTS
In the localized form of PVNS, recurrences are rare, regardless of type of
excision (i.e., open vs. arthroscopic).
In the diffuse form of PVNS, results vary according to extent of disease and
completeness of excision.
The reported recurrence rate for diffuse PVNS in various studies is from 8% to 50%
(6,10,25). This wide range in reported results is due to many reasons. Most studies are
small retrospective series or case reports. Even among the larger reported series,
there are numerous uncontrolled factors resulting in bias (such as nonuniform methods
of treatment or usage of adjuvant radiation) and use of nonactuarial analysis to
determine local recurrence rates. In addition, although most authors have
distinguished between the localized and diffuse forms of disease, the criteria for these
two types is poorly defined and at times, quite subjective. For the localized form of
PVNS, the results of both open and arthroscopic treatment are good and the
recurrence rate is low (6,17,21,25). Most of evidence supporting this conclusion is
quite poor, consisting of mainly case reports and small retrospective studies.
For the diffuse form of PVNS, most centers have undertaken a more aggressive
treatment approach. Flandry et al (10) reported a series of 23 patients with diffuse
PVNS of the knee who underwent open total synovectomy. The recurrence rate was
8%; however, eight of the patients required a manipulation for loss of motion
postoperatively. The outcome was good or excellent in 92% of the patients. Zvijac (35)
reported on 14 patients with PVNS of knee treated with arthroscopic synovectomy. The
average follow-up was 41.9 months. In the two patients with localized disease, there
P.513
was no recurrence; but in diffuse form, there was recurrence in 2 of 12 patients. De
Ponti (6) has reported on 19 patients treated with arthroscopy with an average follow-
up of 60 months. There were 15 patients with diffuse disease, seven treated with
extended arthroscopic synovectomy and eight with partial synovectomy. The
recurrence was 50% in patients treated with partial synovectomy, and 20% in patients
with complete synovectomy at 2 years. Chin et al (2) reported on 40 cases treated
with one of the three modalities, surgery alone (5 patients), surgery combined with
intra-articular radiation synovectomy with use of dysprosium-165 (30 patients), and
surgery with external beam radiation (5 patients). The overall recurrence rate was 18%
(7 cases) at a mean follow-up of 5 years. Five recurrences were in patients who had
surgery combined with intra-articular radiation synovectomy and two in the surgery
and external beam radiation group.
TABLE 35-1. Results of Surgical Treatment for
Pigmented Villonodular Synovitis of the Knee at the
University of Minnesota
Type of Treatment Number Mean Recurrence

Disease of Follow-Up (No. of
Patients (Months) patients/%)
Nodular Arthroscopy. 2 24 None
Open 2 28 None
synovectomy
Diffuse Open anterior 21 42.8 5 (23.8%)

and posterior
Arthroscopy and 6 40.3 2 (33.3%)

open posterior
Arthroscopy 3 30 3 (100%)
Open anterior 3 16 None
Open posterior 3 30 1 (33.3%)
Observation 1 24 NA
We reviewed the results of surgery done at the University of Minnesota from 1990 to
2005 (unpublished data). A total of 41 patients were available for review.
Postoperative MRI examinations were done to detect recurrence. Different methods of
treatment were performed according to the disease extent and surgeon preferences.
Recurrence rates are cited in Table 35-1.
REFERENCES
1. Ballard WT, Clark CR, Callaghan JJ. Recurrent spontaneous hemarthrosis nine
years after a total knee arthroplasty. A presentation with pigmented villonodular
synovitis. J Bone Joint Surg Am. 1993;75(5):764-767.
2. Chin KR, Barr SJ, Winalski C, et al. Treatment of advanced primary and
recurrent diffuse pigmented villonodular synovitis of the knee. J Bone Joint Surg
Am. 2002;84-A(12):2192-2202.
3. Choong PF, Willen H, Nilbert M, et al. Pigmented villonodular synovitis.

Monoclonality and metastasis-A case for neoplastic origin? Acta Orthop Scand.
1995;66(1):64-68.
4. Cotten A, Flipo RM, Chastanet P. et al. Pigmented villonodular synovitis of the

hip: review of radiographic features in 58 patients. Skeletal Radiol. 1995;24(1):1-
6.
5. Darling JM, Goldring SR, Harada Y, et al. Multinucleated cells in pigmented

villonodular synovitis and giant cell tumor of tendon sheath express features of
osteoclasts. Am J Pathol. 1997;150(4):1383-1393.
6. De Ponti A, Sansone V, Malchere M. Result of arthroscopic treatment of

pigmented villonodular synovitis of the knee. Arthroscopy. 2003;19(6):602-607.
7. Dorwart RH, Genant HK, Johnston WH, et al. Pigmented villonodular synovitis of
synovial joints: clinical, pathologic, and radiologic features. Am J Roentgen.
1984;143(4):877-885.
8. Eisold S, Fritz T, Buhl K, et al. Pigmented villonodular synovitis. Case reports
and review of the literature. Chirurg. 1988;69(3):284-290.
9. Flandry F, Hughston JC. Pigmented villonodular synovitis. J Bone Joint Surg Am.
1987;69(6):942-949.
10. Flandry FC, Hughston JC, Jacobson KE, et al. Surgical treatment of diffuse
pigmented villonodular synovitis of the knee. Clin Orthop Relat Res. 1994;300:183-
192.
11. Franssen MJ, Boerbooms AM, Karthaus RP, et al. Treatment of pigmented
villonodular synovitis of the knee with yttrium-90 silicate: prospective evaluations
by arthroscopy, histology, and 99mTc pertechnetate uptake measurements. Ann
Rheum Dis. 1989;48(12):1007-1013.
12. Garcia Sanchez A, Utrilla Utrilla M, Casals Sanchez JL, et al. Pigmented
villonodular synovitis with polyarticular presentation. Anales de Medicina Interna.
1996;13(7):341-343.
13. Ghert MA, Scully SP, Harrelson JM. Pigmented villonodular synovitis of the foot
and ankle: a review of six cases. Foot & Ankle International. 1999;20(5):326-330.
14. Hamlin BR, Duffy GP, Trousdale RT, et al. Total knee arthroplasty in patients
who have pigmented villonodular synovitis. J Bone Joint Surg Am. 1998;80(1):76-
82.
15. Jaffe HL, Lichtenstein L, Sutro CJ. Pigmented villonodular synovitis, bursitis,
and tenosynovitis: a discussion of the synovial and bursal equivalents of the
tenosynovial lesions commonly denoted as xanthoma xanthogranuloma, giant cell
tumor, or myeloplaxoma of the tendon sheath, with some consideration for the
tendon sheath lesion itself. Arch Pathol. 1941;31:731.
16. Kat S, Kutz R, Elbracht T, et al. Radiosynovectomy in pigmented villonodular
synovitis. Nuklearmedizin. 2000;39(7):209-213.
17. Kim SJ, Shin SJ, Choi NH, et al. Arthroscopic treatment for localized
pigmented villonodular synovitis of the knee. Clin Orthop Relat Res.
2000;(379):224-230.
18. Kobayashi H, Kotoura Y, Hosono M, et al. Case report: uptake of pentavalent

technetium-99m dimercaptosuccinic acid by pigmented villonodular synovitis:
comparison with computed tomography, magnetic resonance imaging and gallium-
67 scintigraphy. Brit J Radiol. 1994;67(802):1030-1032.
19. Le Tiec T, Hulet C, Locker B, et al. Villonodular synovitis of the knee. Analysis
of a series of 17 cases and review of the literature. Revue de Chirurgie
Orthopedique et Reparatrice de l Appareil Moteur. 1998;84(7):607-616.
20. Mohler DG, Kessler BD. Open synovectomy with cryosurgical adjuvant for
treatment of diffuse pigmented villonodular synovitis of the knee.
Bulletinâ€”Hospital for Joint Diseases. 2000;59(2):99-105.
21. Moskovich R, Parisien JS. Localized pigmented villonodular synovitis of the

knee. Arthroscopic treatment. Clin Orthop RelatRes. 1991;(271):218-224.
22. Mulier T, Victor J, Van Den Bergh J, et al. Diffuse pigmented villonodular
synovitis of the shoulder. A case report & review of literature. Acta Orthopaedica
Belgica. 1992;58(1):93-96.
23. Muscolo DL, Makino A, Costa-Paz M, et al. Magnetic resonance imaging

evaluation and arthroscopic resection of localized pigmented villonodular synovitis
of the knee. Orthopedics (Thorofare, NJ). 2000;23(4):367-369.
24. Neale SD, Kristelly R, Gundle R, et al. Giant cells in pigmented villo nodular
synovitis express an osteoclast phenotype. J Clin Pathol. 1997;50(7):605-608.
25. Ogilvie-Harris DJ, McLean J, Zarnett ME. Pigmented villonodular synovitis of

the knee. The results of total arthroscopic synovectomy, partial, arthroscopic
synovectomy, and arthroscopic local excision. J Bone Joint Surg Am. 1992;74(1):
119-123.
P.514
26. O'Sullivan B, Cummings B, Catton C, et al. Outcome following radiation

treatment for high-risk pigmented villonodular synovitis. International Journal of
Radiation Oncology, Biology, Physics. 1995;32(3):777-786.
27. Panagiotopoulos E, Tyllianakis M, Lambiris E, et al. Recurrence of pigmented

villonodular synovitis of the knee 17 years after the initial treatment. A case
report. Clin Orthop Relat Res. 1993;(295):179-182.
28. Patel D. Proximal approaches to arthroscopic surgery of the knee. Am J Sports

Med. 1981 ;9(5):296-303.
29. Patel D. Superior lateral-medical approach to arthroscopic meniscectomy.

Orthop Clin North Am. 1982;13(2):299-305.
30. Pimpalnerkar A, Barton E, Sibly TF. Pigmented villonodular synovitis of the

elbow. Journal of Shoulder & Elbow Surgery. 1998;7(1):71-75.
31. Poletti SC, Gates HS III, Martinez SM, et al. The use of magnetic resonance
imaging in the diagnosis of pigmented villonodular synovitis. Orthopedics
(Thorofare, NJ). 1990;13(2):185-190.
32. Rader CP, Barthel T, Hendrich C, et al. Pigmented villonodular synovitis of the
knee joint-long-term follow-up and therapeutic concept. Zentralblatt fur
Chirurgie. 1995;120(7):564-570.
33. Sharafuddin MJ, Sundaram M, McDonald D. Progression from simple joint
effusion to extensive pigmented villonodular synovitis of the hip within 2 years:
demonstration with MR imaging [letter]. Am J Roentgen. 1995;165(3):742.
34. Vedantam R, Strecker WB, Schoenecker PL, et al. Polyarticular pigmented

villonodular synovitis in a child. Clinical Orthopaedics & Related Research. 1998;
(348):208-211.
35. Zvijac JE, Lau AC, Hechtman KS, et al. Arthroscopic treatment of pigmented
villonodular synovitis of the knee. Arthroscopy. 1999;15(6):613-617.

Master Techniques in Orthopaedic Surgery Knee11

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Editors: Jackson, Douglas W.

Title: Master Techniques in Orthopaedic Surgery: Reconstructive Knee

> Front of Book > Editors

E. Lyle Cain Jr., MD

Craig J Edson MS, PT, ATC

Mario Ferretti M.D.

Freddie Fu MD, DSc, DPS

John P. Fulkerson M.D.

LÃ¡szlÃ³ Hangody MD, PhD, DSc

Brant Richardson PAC

William G. Rodkey DVM

Wei Shen MD, PhD

Fotios Paul Tjoumakaris MD

Albert Mu-Hong Tsai MD

William H. Warden III, MD

William M. Wind Jr., MD

> Front of Book > Dedication

> Front of Book > PREFACE

Because each chapter represents a selected individual author's approach to a

Tightness of the lateral retinaculum associated with lateral patellar tilt

Patellofemoral arthritis with lateral patellar and/or trochlear chondral damage

Recurrent patellar subluxations/dislocations (typically performed in

Painful bipartite patella with associated lateral pressure syndrome

Decreased tension in the lateral retinaculum

Partial denervation of the patella

Correction of patellar tilt with decreased loading of the lateral

Conservative therapy is successful in treating most cases of anterior knee pain

Various mechanical devices may also be used, including a Polumbo-style brace, a

Hamstring tightness may be associated with increased loads on the patellofemoral

In general, both extremities should be examined for side-to-side differences, and

Radiographic evaluation consists of a standing PA view in 30 degrees of flexion, a

Calculation of the congruence angle.

A: The radiofrequency wand is inserted through the anterolateral portal adjacent

The lateral patellofemoral and patellotibial ligaments should be released.

Patellar tracking after lateral release. A: Central tracking. B: Correction of

The patella should be manually everted 90 degrees to confirm adequate lateral

Lateral release should be performed only with the appropriate surgical

Avoid injury to the muscle fibers of the vastus lateralis proximally.

Mechanical debridement of the fat pad around the anterolateral portal

Electrical stimulation of the VMO and biofeedback may be selectively employed if

Thermal injury to the skin

Medial subluxation following excessive lateral release

Transection or rupture of the vastus lateralis insertion and/or quadriceps

Reflex sympathetic dystrophy

Persistent knee pain

The incidence of hemarthrosis can be reduced by careful attention to surgical detail,

Superficial infections require special attention because of the potential for

Pre-existing complex regional pain syndrome should be recognized at the stage of

We believe that approximately 75% of our patients have achieved significant

4. Fulkerson JP. Patellofemoral pain disorders: evaluation and management. J

8. Lombardo SJ, Bradley JP. Arthroscopic diagnosis and treatment of

9. Merchant AC. Classification of patellofemoral disorders. Arthroscopy.

MPFL reconstruction is indicated in skeletally mature patients with symptomatic

Tibial tuberosity osteotomy procedures, such as the Elmslie-Trillat, have a theoretical

Exam under Anesthesia

The tourniquet is raised while the knee is in an extended position. A longitudinal

Comparison of patellar translation between symptomatic left and asymptomatic

Medial Patellofemoral Ligament Repair

Graft Harvest for Medial Patellofemoral Ligament

Muscle is removed from the gracilis tendon.