Direct Myofacial Release
Direct Myofacial Release
Direct Myofacial Release
). Sharon
Weislefish has developed Integrative Manual
Therapy
, Hellerwork, Postural
Integration, Feldenkrais, Sensory Integration,
Neurodevelopmental Treatment and so on) while
others who use this book will have a broad sense of
integration in their work without naming that as
its deliberate endeavor. Still others will be involved
in rehabilitative settings where the workplace
defines the scope of practice and the anatomy that
can be worked on. Here there will perhaps be less
scope for overt agendas of integration. Others will
do largely corrective therapy via joint manipulation
Chapter 1 INTRODUCTION 5
and mobilization, trigger point therapy and other
procedures to influence local problems and pain.
This book is intended for all these groups. It is my
hope that the attitude to MFR described in this
book will enable the work to be presented always
in an orderly, organizing manner.
I tend to shift my foci on a seasonal basis, with
the emphasis often determined by the latest train-
ing Ive taken. I suspect we all tend to concentrate
on new information and techniques until they are
integrated and we make them our own. This sense
of mastery generally involves practice, modifica-
tion, rejection of some information and ongoing
experimentation. The approach to working with
soft tissue detailed in this book is not intended to
imply a doctrinaire, this is the only way to do it
philosophy. I think there are other profoundly use-
ful approaches to body therapy. Its just that until
now no-one has truly organized direct technique
myofascial release into a systematic workbook
approach in the way visceral, cranial and other
approaches have been.
PEDIATRIC APPLICATIONS
This book contains photos and illustrations of both
adults and children being treated. The work on
children is intended for pediatric therapists or expe-
rienced practitioners of other related therapies that
have a pediatric application. I discourage parents
with cerebral palsied children from attempting
these releases on them. While these home help
efforts are always well intended, this work requires
a foundation of training in movement develop-
ment, anatomy and physiology to be done safely.
Furthermore, more is not better. It might seem a
logical extension of the rationale behind the tech-
niques to apply them very frequently for long-term
disability. However, MFR for children with dis-
abilities needs to be placed in a broad context of
other therapies. It augments rather than replaces
these approaches. A trained pediatric therapist will
be able to determine how best to integrate these
releases into existing protocols.
LAST THOUGHTS
All the talk of fascia, thixotropy, the autonomic
nervous system and intrafascial mechanoreceptors
that follows this introduction might cloud the fact
that we work on people, not tissue. I hope not, as I
believe any technical review should illuminate, and
not blind, our view of the complex processes and
concerns of the people we touch. Studying the rel-
ative view helps illuminate the big picture.
References
1. Myers T 2000 Anatomy trains. Churchill
Livingstone, Edinburgh, UK
2. Schultz L, Feitis R 1996 The endless web. North
Atlantic, Berkeley, CA
6 Section 1 THE BASICS
PRACTICE SEEKS THEORY
Myofascial release is a practice in search of a theory.
Almost. By reviewing the histologic, mechanical,
physiologic and neurologic aspects of connective
tissue in general, and fascia in particular, a balance
can be found between the enthusiastic clinical anec-
dotes that exist about the efficacy of myofascial
release and a rational basis for understanding how
the technique works.
Connective tissue is the most pervasive substance
in the human body. For example, fascia, a specialized
type of connective tissue, surrounds, invests and pro-
tects all the visceral and somatic structures of the
body. The fascial sheaths entwined in and around
muscle account for most of the immediate lengthen-
ing of muscle after stretching. Even with joint mobi-
lization, the primary structures being affected are the
joint capsules and the periarticular connective tissue
in other words, the soft connective tissues.
CONNECTIVE TISSUE
Connective tissue is generally divided into five
principal groups:
ordinary
blood
cartilage
adipose
bone.
Manual therapists are primarily interested in ordi-
nary connective tissue which includes the sub-
groups of superficial and deep fascial sheaths, nerve
and muscle sheaths, the supporting framework of
internal organs, aponeuroses, ligaments, joint cap-
sules, periosteum and tendons.
Cells
All connective tissues consist of cells and extracellular
matrix (ECM). The cells, primarily fibroblasts, are
responsible for producing the ECM. Macrophages
and histiocytes are also found in connective tissue
and are involved in phagocytosing waste and foreign
matter. Also present are mast cells, responsible for
producing histamine and heparin, and plasma cells,
which are mostly found in pathologic situations.
With the exception of fibroblasts, all these cells are
involved in the reticuloendothelial system.
Imagine a body with all the cells removed. What
would remain? An amorphous pile of what?
Perhaps there would be not much of anything, since
we are biological beings, a sophisticated collection
of cells. There would simply be nothing at all.
Yet cells are in fact a small part of connective tis-
sues overall contribution to human structure. It is
the extracellular material that gives the various con-
nective tissues their characteristic shape, tensile
strength and texture. Without its cells, connective
tissue would still provide the body with a degree of
shape, a range of tissue textures and, perhaps, even
a semblance of our cells-included appearance.
Connective tissue is mostly about its non-cellular
characteristics. Cars the living things of the high-
way drive over bridges and through tunnels, the
non-living structures of the highway itself. Take
away the cars and the highway remains.
To understand connective tissue fascia is, in part,
to appreciate the ECM, a substance that is, by
definition, non-cellular in nature and yet, curiously,
7
Chapter 2
DEVELOPING A
HYPOTHETICAL MODEL
central to the function of all cells throughout
the body.
Extracellular matrix
The ECM of connective tissue consists of fibers and
ground substance. There are three types of fibers:
collagen, elastin and reticulin. Collagen is the most
tensile of the three and is found in fascia, bones, ten-
dons and ligaments. Elastin is less tensile but, as its
name suggests, has more elastic qualities. It is found
primarily in the lining of the arteries. Reticulin, the
least tensile and most elastic of the three, is found in
the supporting structures surrounding the glands
and lymph nodes. The ground substance is a viscous,
gel-like substance in which the cells and fibers lie.
The ground substance acts as a mechanical barrier
to foreign matter and is a medium for the diffusion
of nutrients and waste products. Of particular inter-
est is the fact that it maintains the critical distance
between adjacent collagen fibers. This distance
allows for some of the extensibility of fascia by
reducing the number of microadhesions that can
occur between collagen fibers.
The primary components of the ground sub-
stance are glycosaminoglycans sometimes called
mucopolysaccharides and water. There are two
types of glycosaminoglycans: sulfated and non-
sulfated. The non-sulfated group acts to bind water
while the sulfated types give cohesiveness to the
fascia. This capacity for binding water is an impor-
tant aspect of the physiology of fascial disorganiza-
tion, dehydration and restriction, all of which are
discussed later.
Connective tissue types
The strength of connective tissue is determined
by the arrangement of the fibers and the viscosity of
the ECM. Most histologic sources classify ordinary
connective tissue into dense and loose types. These
categories are both then divided again, into regular
and irregular types.
Dense regular connective tissue, which includes
tendons and ligaments, is characterized by a dense
parallel arrangement of collagen fibers. The paral-
lel arrangement of the fibers and a high proportion
of fibers to ground substance means the tissue is
not particularly extensible. Rather, it is extremely
compact so the vascular supply is limited, which
accounts for the increased healing time after trauma.
Dense irregular connective tissue includes
aponeuroses, joint capsules, periosteum, dermis of
the skin and fascial sheaths under high loads of
mechanical stress. The fibers are arranged in a
multidirectional manner which enables resistance
in three dimensions to various forces and stress.
The relative amount of ground substance is higher.
Loose irregular connective tissue includes super-
ficial and deep fasciae, nerve and muscle sheaths
and the endomysium which holds the individual
muscle fibers together. It is characterized by a
sparse, multidirectional framework of collagen and
elastin with more ground substance per unit area
and higher vascularity than the other types (Fig. 2.1).
Thixotropy
Many writers have proposed that connective tissue
fascia can be transformed from a gel (thickened)
state to a sol (liquid) state by the application of
energy.
1,2
This energy isnt mystical or amorphous.
In the context of a touch-based therapy such as direct
technique MFR, it is accurately defined as the shear-
ing force that is generated within the soft tissue by
the application of pressure (force) with direction
via hands, knuckles and elbows.
If youve ever stirred a can of paint then youve
seen and felt how the consistency of the paint is
changed as the shearing force the paint stick
moves through the liquid. Paint that was thick and
gluey becomes more fluid and uniform in texture.
The thick gel state gives way to a more liquid sol
state. Furthermore, the paint stays in a transformed
state for some time after the shearing force is
removed. When we apply direct myofascial tech-
niques to restricted tissue we could be seen as the
paint stick, stirring (slowly) the fascial can of paint.
This transformative process, of gel to sol, is referred
to as thixotropy.
Effects of immobility
With immobility, changes in the ground substance
occur, including losses of glycosaminoglycans and
water.
3
Since the non-sulfated glycosaminoglycans
bind water, the loss of water is easy to explain.
Lubrication between the collagen fibers is
8 Section 1 THE BASICS
maintained by the ground substance. When this crit-
ical interfiber distance is not maintained, due to low-
ered amounts of water and glycosaminoglycans,
adjacent collagen fibers move closer together and
microadhesions start to occur. As movement helps
orient newly synthesized collagen, new collagen in
immobilized (stiff) fascia will be laid down in a
haphazard manner. Additional binding occurs as
the new and randomly arranged collagen forms
microadhesions to existing fibers.
The early bonding of these crosslinks consists
primarily of weak hydrogen bonds. The hydrogen
bonds are eventually replaced by much stronger
covalent bonds which require more energy to be
broken.
This stiffness, with its attendant physiologic
changes, can be seen as the can of paint starting to
thicken, from a sol to a gel.
Furthermore, these mechanical and viscous
changes at a micro level are responsible for distor-
tions in the quality of movement of joints at a macro
level.
A restricted joint often exhibits a diminished
range of motion as well as a significant reduction
in the quality of graded movement. Even a joint
moving within acceptable ranges of motion may
exhibit a premature increase in binding when
approaching that end range. Active testing across
multiple planes of motion (real-world movement)
will reveal some zones that are grabbing, stiff and
boggy as the fascial strains appear to prevent uti-
lization of the full movement potential. Passive
range of motion reveals early binding approaching
end range and irregular soft tissue tensions. The
constant stirring of the fascia that occurs as a joint
moves freely within the parameters of its anatomic
design is further diminished or lost.
Stiffness, it seems fair to say, leads in time to
more stiffness.
Scar tissue
The histology and biomechanics of scar tissue differ
from those of non-traumatized connective tissue.
As they are frequently encountered, scars deserve
additional examination.
Scar formation consists of four major phases.
1. The inflammatory phase begins immediately after
the insult to the tissue, followed a short time later
by clotting. Soon after that there is an influx of
macrophages and histiocytes. They are involved
in debriding the area, which promotes a clean
Chapter 2 DEVELOPING A HYPOTHETICAL MODEL 9
Adductor magnus
Gracilis
Semimembranosus
Adductor longus
Sartorius
Vastus medialis
Fascia lata
Patella
Vastus intermedialis
Skin
Semitendinosus
Biceps (long head)
Sciatic nerve
Biceps (short head)
Vastus lateralis
Quadriceps tendon
Femur
Iliotibial tract
Figure 2.1 Layers of myofasciae.
environment for healing. This inflammatory stage
lasts from 24 to 48 hours. Immobility is impor-
tant at this stage to prevent further damage.
2. Granulation, the second stage, involves an
increased vascularity of the tissue. Debris is trans-
ported away from the area, while nutrients are
transported to it. The length of this granulation
phase varies depending on the type of connective
tissue involved and the extent of the insult.
3. The third phase of scar formation is the fibro-
blastic phase. There is a proliferation of fibro-
blasts and an increase in their activity. The rate
of collagen and ground substance formation
increases. Collagen is laid down haphazardly
during this phase which lasts from 3 to 8 weeks.
4. In the maturation phase collagen production is still
accelerated. However, there is an overall shrink-
ing, solidifying and consolidation of the collagen.
In this phase collagen is strong enough to endure
some therapeutic stress without incurring further
damage. This is a phase when the deformative
properties of connective tissue fascia can be best
exploited by applying direct technique myofascial
release to help orient the newly created fibers.
Left unchecked and untreated, the localized
haystacking of collagen and the contraction of the
tissue will combine to permanently reduce local
extensibility. Prolonged periods of immobility,
which often occur in an orthopedic situation, exa-
cerbate this condition. Ground substance is lost,
with associated increases in intermolecular adhesions.
Macroadhesions form between the scar tissue
and the surrounding healthy tissue. This limits the
extensibility of large sections of tissue which in turn
initiates compensatory patterns of hypo- and hyper-
mobile tissue throughout the entire structure. These
can lead to areas of stiffness and pain developing in
areas quite distant from the initial scar formation.
Scar tissue is also associated with an undesirable
increase in the afferent signals to the central nerv-
ous system. This neurologic aspect of scar tissue is
discussed later.
ALTERNATIVE THEORIES FOR MYOFASCIAL
RELEASE
The viscoelastic explanation for the palpable changes
associated with fascial release enjoys widespread
support.
1,2,46
It has become, in a sense, a classic
theory, adopted by many schools of manual ther-
apy. According to this theory, fascia responds to
the mechanical interventions of therapy in three
related ways.
1. The ground substance changes its volume and
consistency.
2. The crosslinkages between the fibers are broken.
3. The interfiber distance is increased so that
fiber affinity is reduced, resulting in increased
extensibility in the tissue.
Others dispute the capacity of fascia to undergo
such rapid change through mechanical deforma-
tion alone.
7,8
The arguments advanced against the
thixotropy, gelsol model include the absence
of sufficient force delivered over a long enough
period of time to produce that type of change. One
study showed that moderate elongation of the
iliotibial band must be sustained for 1 hour or more
for the deformation to be permanent.
7
More force-
ful methods delivered over a significantly shorter
period of time would result in significant tearing
and inflammation. In addition, even these defor-
mations appear to require a force far greater than
even the largest manual therapist could deliver.
7
The proponents of these arguments against the
thixotropy explanation consider other factors more
important to explain the rapid changes that can
be felt under the hands of a therapist during the
delivery of myofascial release. Rarely do these con-
tacts approach even 2 minutes, let alone 1 hour.
What accounts for these quick responses in myofas-
cial extensibility and pliancy? Explanations that
go beyond the thixotropy model are based on an
exploration of what I will call neurofascial physiol-
ogy. These important theories are reviewed in
conjunction with the following sections on the
autonomic nervous system, neuromotor system
and the intrafascial mechanoreceptors.
Autonomic nervous system
Some of the mechanical and viscid effects of
myofascial release have already been highlighted.
In addition, somatovisceral and somatoparasympa-
thetic reflexes are activated by direct technique
myofascial release. These responses in the auto-
nomic nervous system (ANS) are at the heart of the
10 Section 1 THE BASICS
changes that occur in response to direct technique
MFR. Research supports this neurofascial dynamic
as being an important aspect of the types of release
observed during MFR.
9
Practitioners of other man-
ual therapies also see change in the ANS as being a
significant component of their method.
1,7,10,11
The ANS has two divisions, the sympathetic
(SNS) and the parasympathetic (PNS). Parasympa-
thetic outflow is largely through the vagus nerve. The
PNS regulates the functions required for long-term
survival and is in charge of rest, rebuilding and
rehabilitation. Increasingly, it is seen as having a
direct effect on muscle tone as well as the more
vegetative functions it has traditionally been
associated with.
The SNS, at its most extreme, is responsible for
the famous fight or flight reflex. It takes care of
crises, be they real threats to physical well-being
(the local bus headed for you at the crosswalk) or
imagined (the overwhelming physiologic responses
associated with stage fright or the total collapse in
value of a highly leveraged stock portfolio). It also
plays a role in the regulation of muscle tone. As can
easily be imagined with the fightflight system,
activation of the SNS leads to increases in muscle
tone which would obviously facilitate the ability to
fight and flee. Equally as obvious is the undesirable
state of sustained high SNS tone. A range of serious
disorders is associated with such an arousal state.
The two branches usually work as antagonists.
Gellhorn proposed a law of reciprocal inhibition
that describes a dynamic whereby the excitation of
one branch leads to the inhibition of the other.
12
He also proposed that long-term tuning of the ANS
is possible. In this situation one of the branches
dominates the other in such a way that the recipro-
cal inhibition becomes fixed and unchanging.
13
The balance between these two components of
the ANS is central to the self-regulating processes
of the body known as homeostasis. One definition of
stasis proposes it is a state of inactivity caused by
opposing equal forces while another is that it is a
stagnation in the flow of any of the fluids in a body.
In the end, neither definition conveys the need for
a flexible and adaptive nervous system that can
constantly fine-tune the well-being of the organ-
ism. A prolonged state of imbalance or stasis, asso-
ciated with Gellhorns tuning, is detrimental to
health across a broad spectrum of core bodily
processes. The term homeokinesis is probably
more suited to describing a healthy relationship
between the two branches of the ANS.
14
Ideally
there is a play between the two throughout the
day, a healthy range of flexible sinusoidal move-
ment from one branch into another (Fig. 2.2).
Various authors have attributed the success of
manual therapies to the restoration of ANS modu-
lations back into the healthy range. Upledger sees
the restoration of autonomic flexibility as one of
the primary benefits of craniosacral therapy.
10
Dr Ida Rolf held a similar view about the benefits
of Rolfing
.
5
When describing the formation of the
cranial rhythmic impulse (CRI), McPartland &
Mein proposed that:
if our hypothesis and findings from
entrainment studies are true, then the common
denominator and underlying mechanism gen-
erating CRI is the balance between the sympa-
thetic and parasympathetic nervous systems. If
there is autonomic nervous system balance then
Chapter 2 DEVELOPING A HYPOTHETICAL MODEL 11
Figure 2.2 Autonomic nervous system showing healthy
range and out-of-range problems. Gellhorns tuning takes
place when the ANS stays fixed in one of the overload zones.
Generally, with MFR pain and stiffness this is in the extreme
of the SNS range (after Bradley,
16
with permission).
the bodys rhythms harmonize into a strong,
coordinated, sinusoidally fluctuating entrain-
ment frequency, palpated by the practitioner as
a strong healthy CRI. To wit, health as assessed
by CRI becomes dependent on sympathovagal
balance.
15
The importance of the ANS for health should not
be underestimated. Consider that all cells have
sympathetic innervation. The impact of a chroni-
cally aroused SNS, coupled with the outflow of the
associated stress hormones, will therefore be pro-
found. It can stall healing, generate hypertension,
contribute to the formation of facilitated segments,
impair metabolism via endocrine imbalance and
ultimately all homeostatic (kinetic) mechanisms.
Chronic hyperventilation, with an associated loss
of oxygen to the brain, resulting in multiple dimin-
ished functions, is another serious consequence.
16
Considering the frequency of asthma, hyper-
tension, glaucoma, ulcer disease, and abnormal-
ities of sweating, temperature, cardiac rhythm,
respiration, sexual, bowel and bladder function,
it is amazing that the autonomic nervous system
gets essentially no direct treatment.
11
This observation accurately describes the scope of
the health problems associated with the ANS.
However, the assertion that there is little direct
treatment of the ANS is not accurate. Direct tech-
nique MFR provides that treatment.
Studies have found that certain forms of tactile
stimulation produce predictable changes in the
ANS. Deep slow pressure into the skeletal muscle of
cats produces a decrease in blood pressure (under
control of the ANS).
17
More germane to a prac-
titioner of direct technique MFR are studies into
the relationship of tactile stimulation and ANS
responses in humans. Using a vagal tone monitor
to assess the activity of the vagus nerve, researchers
have demonstrated that the PNS was stimulated by
direct technique MFR to the sacrum and low
back.
18,19
These studies indicate that soft tissue
pelvic manipulation is useful for certain types of
low back dysfunction, as well as musculoskeletal
disorders associated with autonomic stress and
imbalance.
From my own clinical practice, I observe that
deep calming of the type associated with increased
PNS activity often occurs during a treatment
using direct technique MFR. The signs of this
include:
borborygmus (bowel sounds gurgling, ping-
ing and the like)
hypnogogia (the dreamy fluid state between
waking and sleeping)
muscle twitching
deep abdominal jerks and twitches
fasciculation (skin ripples)
increased salivation (sometimes drooling)
full body lowering of muscle tone
reduced respiratory rate
full sleep, although this is nowhere near as
common as hypnogogia
lowered heart rate.
Just as important, of course, are the subjective expe-
riences of the client. These include feeling at peace,
languid, centered, calm with less rigid thinking and
sometimes dream images. This psychosomatic state
and its associated dream images are generally pleas-
ant. The sense of the passing of time also alters,
usually toward a more timeless, fluid state. These
changes are all a function of increased PNS activity.
However, this is not always the case. The oppo-
site effect is sometimes witnessed. Sweating, rapid
pulse, increased breathing rate, dry mouth and full
body increases in muscle tone can occur. The sub-
jective reports are of irritability, agitation, anger,
fear and disordered thinking which are a function
of activation of the SNS. These unpleasant sensa-
tions are not long lasting. Rather, they appear to
be part of an internal psychosomatic integration
cycle that is necessary for the restoration of ANS
flexibility and homeokinesis.
I definitely favor an increase in the PNS! While
spikes in the SNS can occur in manual therapy set-
tings, they should not be provoked through aggres-
sive, invasive approaches. When spikes occur, stop
working and allow for the self-regulatory processes
of the client to calm them. This is often as simple
as waiting for a minute or two at which point the
person will report that the effect has passed. Occa-
sionally, more time is needed. I want to emphasize
that the goal is to avoid these spikes in SNS activa-
tion. Working with direct technique MFR in the
mindful, sensitive manner described in this work-
book should result in consistent reductions in the
chronically aroused SNS the state associated with
musculoskeletal and myofascial pain.
12 Section 1 THE BASICS
Clearly, the ANS controls, from the top down, a
wide range of somatic functions. Appropriate and
continuous sensory stimulation from the bottom up,
to the brain, is essential for normal brain function.
By providing these sensory inputs, direct technique
MFR favorably influences the brain and therefore
the CNS controls on the ANS. Consider that dam-
age to the sensory cortex is generally more problem-
atic than damage to the motor cortex. Disordered
or absent sensory input is catastrophic for the func-
tion of the brain itself, as well as the efferent neuro-
motor system, whereas damage to the motor cortex
is often less disintegrative as the underlying sensory
afferents are intact. In time, the brain is able to use
new regions to formulate motor efferents.
The work of Cottingham and others into the
effects of soft tissue manipulation on the ANS is
the best explication of these dynamics to date.
18,19
Given the relative ease of measuring the activity of
the vagus nerve via a vagal tone monitor, as shown
by Cottingham, there could be more research
of this kind. Perhaps in the near future more stud-
ies will explore these fascinating and important
relationships.
Neuromotor controls and the central
nervous system
Myofascial release also elicits obvious and predictable
responses in neuromotor control. Co-contraction,
which results in loss of strength, poor joint stabil-
ity and fatigue through excessive demands by the
muscles for energy, is reduced. There is a subsequent
increase in muscle recruitment efficiency. This
change can be measured by palpation, functional
strength testing and range-of-motion tests. The
clients subjective report post release will often focus
on freedom of movement, decreased stiffness, light-
ness and better coordination.
An increase in the quality of movement at the
joint nearest the site of the myofascial release will be
consistently observed. Quality refers to the establish-
ment of a balanced relationship between the agonists
and antagonists. The joint is both free moving and
stable at all points of its range of motion. Even brief
myofascial release can bring about such changes.
Clearly, the effects of the MFR work extend into
the nervous system. How does the neuromotor sys-
tem get involved with manipulation of the bodys
connective tissue structures? The most obvious
explanation for the observed changes in tonus can be
found in the process of reciprocal inhibition. This is
a muscle tone process, not the reciprocal inhibition
of Gellhorn and his descriptions of the ANS. When
a muscle on one side of a joint contracts, the muscles
on the opposite side should be inhibited to allow for
passive lengthening. Without this dynamic, move-
ment would be impossible as muscles on all sides
of a joint might fire at the same time. As shortened
muscles are lengthened through MFR, the antago-
nist muscles are released from a long, weakened and
inefficient position. This enables the antagonist to
resume a tonus that more adequately stabilizes the
joint. The process is accelerated via active client
movements which activate the antagonist while the
therapist applies MFR to the agonist.
A more comprehensive understanding of MFR
and the nervous system can be formed by a review
of the relationships between structure and func-
tion. The ability to maintain posture, or the gravity
response, and all movement requires an integrated
interplay between a number of systems: the struc-
tural or connective tissue elements, the coordina-
tion or muscular-motor system and the perceptual
or sensory system.
20
For example, the joint receptors (sensory) pro-
vide information to the nervous system that a joint
is stable or not. A balanced, even pressure at the
joint sends the signal that the joint is working well.
To maintain this type of balanced relationship at
the articular surfaces requires a sophisticated level
of coordinated muscular work. Smooth concentric
contraction of the agonists must match well-graded
eccentric contraction of the antagonists. The
receptors in ligaments, fascia, tendons and viscera
are also involved with sensory feedback to the
central nervous system, which in turn develops
appropriate, or inappropriate as the case may be,
coordination via the motor system.
The efficient, well-ordered firing of muscles is
dependent on appropriate sensory information that
is processed to generate a normal efferent signal. A
feedback loop is developed here. Any disruptions
to the sensory signals, from the joints, ligaments,
tendons or muscle spindles, can alter the tone of
muscles as well as their firing order. Disruptions to
these signals occur for a variety of reasons trauma,
asymmetrical postural demands on the joints,
excessive physical demands, fluid pressure increase,
Chapter 2 DEVELOPING A HYPOTHETICAL MODEL 13
visceral strain, psychomotor posturing, and so on.
The list is long but the effect is the same: the bom-
bardment of the CNS with multiple sensory signals
from distressed viscera, muscles and joints leads to
changes in the neuromotor system. These exces-
sive signals are distributed throughout the CNS
and are not confined to one spinal segment cor-
tex, brainstem and nearby spinal segments will also
be affected. The underlying and largely uncon-
scious muscle tone that is essential for maintaining
the anti-gravity response posture is governed
by the gamma motor system. This sustained back-
ground tone is referred to as the gamma bias.
When the CNS is bombarded with continuous and
excessive sensory input, the gamma bias, or sustained
efferent outflow, often becomes what is called the
gamma gain. Muscle spindles, each contained
in their own connective tissue sheaths, become sen-
sitized so that the reflex contraction of a stretched
muscle increases. A muscle in this condition resists
lengthening. If the gamma gain is sufficiently high
the sensitized spindle may force a contraction even
when the muscle is shorter than its resting length.
Of course, such a condition is extremely dysfunc-
tional with severe negative effects on joint range
and stability. This in turn generates more noxious
sensory stimuli into the nervous system.
Left unchecked, these sensorimotor disturbances
become long-term changes in the structural, con-
nective tissue system. Fascia shortens and thickens
in an attempt to provide support where the imbal-
anced relationship between hypertonic and hypo-
tonic muscles cannot. Further disruptions to the
normal physiology of fasciae occur as the full range
of movement at the joints and in the muscles is lost,
preventing the orientation of newly created collagen
fibers.
21
Without appropriate orienting forces, the
fibers tend to cluster and thicken. The longer these
processes of tightening, compression and misalign-
ment go uninterrupted, the more noxious afferent
stimuli there are. These stimuli are not coming solely
from the proprioceptors in the muscles, the spindles,
but from many types of sensory endings that exist
in the connective tissue network. These are elabo-
rated on in the section on intrafascial mechano-
receptors below. Whatever its origins, the cycle is
self-perpetuating: constriction, left uninterrupted,
leads to more and more serious constriction.
Direct technique MFR as described in this book
works into the myofasciae and other connective
tissue types. The thixotropy theory proposes that
the action of this mechanical pressure is sufficient to
elongate and decompress restricted fascia. Taken
alone, this may not be a viable explanation. However,
that same deep, slow and directed pressure is also
14 Section 1 THE BASICS
Tonus
regulation
of specific
muscle
fibers
M
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)
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t
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(
p
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s
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)
Therapists
hand
Cortex
Midbrain
Brainstem and cerebellum
Spinal cord reflexes
Sensory input Motor output
Figure 2.3 The practitioners touch alters the signals being
sent to the CNS. Such disruption to the habitual patterns of
the nervous system is a central part of direct technique MFR
(after Schleip,
7
with permission of the author).
affecting a range of sensory fibers via stimulation
of the joint, ligament, muscle and tendon receptors.
In Figure 2.3, we see the therapists hand involved
in altering the dynamics of the biofeedback loops
outlined above. The noxious stimuli that result in the
self-perpetuating pain, compression and dysfunction
cycle are replaced with new afferent signals. These,
in turn, alter the efferent signals.
For example, changes in tone create a change in
joint position, which in turn helps normalize tone.
Over the long term these initial and rapid changes
in tone, coordination and perception lead to a
more appropriate arrangement of collagen fibers
within fascia. Contrast this with the more con-
ventional view that this restructuring of the con-
nective tissue happens immediately, as proposed
in the thixotropy model.
Intrafascial mechanoreceptors
In the opening section of this chapter, we saw that
fascia was part of the large ECM of the body.
A theory of fascial release, generally identified
as thixotropy, was developed. The release is seen as
involving a mechanical stretching of the fibers as
well as an associated change in the hydration of the
ground substance.
Next I introduced alternative theories of fascia
release that included somato-autonomic reflexes.
Reflexes require an initiating sensation and that
means sensory fibers. As this section will show, they
are in fact enormously important to understanding
what happens under our hands when we touch
someone.
Sensory fibers within the fascia itself have a
highly developed capacity to communicate with
both the ANS and the CNS.
22
For myofascial ther-
apists seeking to flesh out a theory of why MFR
works, these sensory fibers Ruffini, Pacinian and
interstitial are the El Dorado of neurophysiology.
Within dense regular connective tissue there
are two types of mechanoreceptors: the Pacinian/
Paciniform corpuscles and the Ruffini bodies.
23
Thus, they occur within myofascia, tendons,
aponeuroses and ligaments, the very soft tissues we
focus on in direct technique MFR. These are in
addition to the sensory fibers that lie within the
muscle spindles and some of the Golgi tendon
organs (GTOs). The role of the GTOs in inhibiting
tone is well documented although it is now thought
that they discharge only when muscles actively
contract. They respond to changes in force, not
length. Direct technique MFR applied to a muscle
that is actively contracting against resistance, usually
eccentrically, increases the discharge from the GTOs
and elicits inhibition of any further tensioning in
that muscle.
The Pacinian corpuscles are stimulated by high-
velocity, low-amplitude (HVLA) thrust manipula-
tions, as well as vibratory techniques while the
Ruffini bodies respond to slow and deep melting
techniques.
7
Furthermore, the stimulation of the
Ruffini bodies is linked to a reduction in the activ-
ity of the SNS. This certainly helps in understand-
ing the effects of soft tissue manipulation on the
ANS that were described earlier where both local
and systemic changes in that system occur.
Gamma neurons can be inhibited by supraspinal
structures. The medial reticular formation plays a
role in this inhibition. As we saw in Figure 2.3, the
various sensory fibers found in fascia make connec-
tions to the brainstem and above. They are not
directly involved in the local myotactic reflex arc
taking place at the spinal segment. It is likely that
these various sensory fibers are involved in carrying
information to the CNS that reestablishes inhibition
of gamma gain from the top down. This would
account for the fact that many rapid changes in tone
take place when applying direct technique MFR to
tissue that is completely devoid of muscle fibers.
For example, a deep slow MFR technique applied to
the calcaneus will elicit a predictable change in range
of motion at the ankle, with obvious reductions in
gastrocnemius tone with an increase in its resting
length. Additionally, coordination and stability will
improve even though no deliberate attempt is made
to balance the action of agonists and antagonists. The
spindles have not been directly treated. Although
the processes are not clear, I propose that these
changes are a function of sensory inputs ascending
to supraspinal levels and influencing the formation
of inhibition.
The third group of nerves is the interstitial mus-
cle receptors. Researchers have identified their
involvement in the fine tuning of the blood flow.
This also points to a direct connection to the ANS.
Sakadas study of the periosteum of the mandible
shows slow and rapid responding receptors. The
slow receptors sense pressure and pain, as well as
Chapter 2 DEVELOPING A HYPOTHETICAL MODEL 15
low-frequency vibration. The rapid-response recep-
tors are involved in sensing pressure and pain but also
much higher frequency vibration, up to 500Hz.
22
The interstitial fibers have been shown to have
control over plasma extravasation.
24
This refers to
the extrusion of plasma from blood vessels into the
interstitial fluid matrix. Now we see a nervous sys-
tem component to the gelsol model described
earlier. When certain forms of stimulation are pres-
ent, of the kind provided by an MFR practitioner,
the interstitial fibers signal the blood vessels to
increase the renewal speed of the ground substance.
Hydration may occur but it is initiated through
sensory fibers rather than mechanical force alone.
An increase in the quantity of ground substance
helps maintain the interfiber distance and lubri-
cates the space between the fibers. This is fascial
cohesiveness the affinity of fibers that drives
them to bind with their neighbors is balanced via
an appropriate volume of ground substance. As the
neuromotor system is released from dysfunction via
MFR, fascia is stressed via appropriate and orderly
movement. Collagen will be laid down according
to the general direction of the stress. Movement
forces fibers into extensibility and this prevents
the clumping of collagen fibers. A combination
of increased levels of ground substance with more
orderly arrangements of fibers means fewer crosslinks
and increased extensibility.
Once the intrafascial mechanoreceptors are
included in the discussion, a bigger, fuller picture
emerges, of therapist-induced forces acting to trig-
ger complex neurologic reflexes that quickly alter
the tonus of both the ANS and the CNS.
7,13,18,19
In turn these changes have numerous direct and
indirect effects on the ground substance and fascial
cohesiveness in general.
AND THERES MORE
Smooth muscle cells have been found in the fascia
cruris (fascia of lower leg). Using electron photo-
microscopy, two researchers observed not only
widespread existence of the intrafascial nerve fibers
mentioned above but also, unexpectedly, smooth
muscle cells.
25
While we are already in a kind of
neurofascial El Dorado, this discovery amounts to
a jackpot in the exploration of the relationship
between fascia and the nervous system. With
smooth muscle cells being under the control of the
ANS, it seems likely that neural-regulated tension-
ing occurs within fascia. This fascial tonus is con-
trolled via the state of the ANS, separate but
related to the much stronger tonus regulation of
muscles via the neuromuscular system. What the
purpose of this pretensioning might be is not
clear. While this is debated, it seems possible to
conclude that an ANS tuned toward the SNS
branch might exert an overtensioning effect on the
fascia. MFR has been shown to reduce the severity
of this tuning and increase the activity of the vagus
nerve. It could well be that part of the release and
lengthening the client and therapist both feel dur-
ing the application of MFR is a tension release in
the intrafascial smooth muscle cells.
PIEZOELECTRIC EFFECT
A lesser developed theory for the kind of fascial
deformation we are seeking to understand is the
piezoelectric effect. Piezo (pressure) electricity
refers to the generation of an electrical charge
when a crystal is compressed. Connective tissue
may act as a liquid crystal. At least one author has
proposed that the application of the therapists
pressure increases the electrical charge within the
tissue.
26
This, in turn, stimulates the fibroblasts
to increase the secretion of collagen fibers in that
local area.
A number of problems exist with this theory.
First and foremost, the secretion of fibers could not
occur so rapidly that a therapist would sense that
production. Also, the secretion of fibers across any
timespan would not account for the rapid changes
in tissue texture that are palpated during direct
technique MFR. Nor would the increased prod-
uction of fibers necessarily be a desirable state for
the body.
However, activation of the piezoelectric effect
may provide the necessary charge for the stimula-
tion of the sensory branch of the interstitial fibers.
In turn, these have control over plasma extravasa-
tion, which is associated with ground substance
hydration/dehydration. Sakadas research shows
interstitial fibers as being sensitive to low- and high-
frequency vibrations. Perhaps it is here, at the level
of the interstitial fibers, that the piezoelectric effect
is playing a part in fascial structure and function.
16 Section 1 THE BASICS
PSYCHOSOCIAL FACTORS
Manual therapy often describes the body as if there
is, in fact, a pure body that exists separately from
social and cultural contexts. I write this book in
Australia as if there is a universal body to be dis-
cussed and engaged with. Its a complex dilemma.
Certainly, our biological body can be understood
in terms of a semi-universal anatomy and physiol-
ogy. To a great extent, a muscle spindle is a muscle
spindle in whatever body it might be found, with a
predictable relationship to the central nervous sys-
tem and muscle tone.
However, what of the way we think about and
shape our bodies in other contexts? How could any
one author write a book that speaks to the multi-
tude of social settings that shape the self-image of
their inhabitants? A woman in Somalia? A teenage
boy in central China? An elderly Afghan refugee
incarcerated indefinitely in an Australian detention
center? A child with cerebral palsy in South Texas?
What is the self-image body in these contexts?
What is the meaning of touch therapies in the social
context in which they are delivered? As numerous
social constructivists have proposed when warning
about the blinders put on when adopting a purely
biological determinism, The impact of any biologi-
cal feature depends in every instance on how that
biological feature interacts with the environment.
27
This is the realm of the psychosomatic body and,
to coin a phrase, the sociosomatic body as well.
Numerous authors in the social sciences have of
course articulated, from a variety of perspectives,
the view that self-image forms in the context of
cultural, social and psychological environments.
That self-image can, to a great extent, shape many of
the processes at work in the biological landscape.
Changes made through direct technique MFR could
be looked at, although they are not in this book, sim-
ply in terms of the anatomic, physiologic and kinesi-
ologic effects described so far. Desirable changes in
those three alone would certainly be enough to make
direct technique MFR a powerful therapy.
Still, when manual therapy techniques are
described and the rationale for using them is elab-
orated, it can sound like were talking about simply
tuning a soft machine. A form of biological reduc-
tionism starts to creep in: all we are is an assemblage
of fluid-filled bags moved from one place to another
by contractile fibers that are instructed from the
nervous system. Of course, this is a convenient way
to analyze and understand certain parts of the
whole person. With hard science on their side,
these descriptions then sound reassuringly final and
conclusive. But what happens in social and cultural
contexts when a person moves from a compressed,
fatigued and painful state into one that is more at
ease and expressive? An example of this non-linear
systems thinking is shown in Figure 2.4 where a
state of thoracic flexion and tightness is seen as
existing in relationship with a number of other
aspects of a person.
Are there changes that include, but go beyond,
the biological? The observation has been made by
many somatic therapists that release from chronic
tightness can influence both the psychosomatic and
sociosomatic body. The non-linear approach can be
developed even further than in Figure 2.4, to include
a wide range of relationships that occur between bio-
logical, psychologic and sociologic factors (Fig. 2.5).
And the opposite is also true. These same psycho-
social factors may contribute to the development
of disruptions to the normal function of the neuro-
myofascial net. The shift in thinking is from cause
and effect to non-linear interdependencies. Using
this dynamic model makes the discussion on what
happens in MFR a much more complex one.
The famous Whitehall studies show that the
most significant factors for general health are not
fitness, diet, genetics or whatever they are social
rank and socio-economic status.
28
In these studies
a steep inverse relationship was found between
social class and morbidity from a wide range of
diseases. A similar relationship was seen between
Chapter 2 DEVELOPING A HYPOTHETICAL MODEL 17
Thorax flexion
Vitality
Breathing
Headache
Indigestion
Neck tension
Figure 2.4 Viewing the effects of direct technique MFR
from a systems perspective, rather than a linear cause and
effect model (from Schleip,
7
with permission of the author).
self-observed social status and sick days taken. This
finding alone does not prove any direct causal rela-
tionship between psychosocial factors and myofas-
cial, or neurofascial, restrictions. Nor is it intended
to do so. It becomes of even more interest, though,
when other related findings are included. For exam-
ple, Sapolskys famous study of baboons showed
that a lack of social control leads to high stress as
measured by cortisol and adrenaline (epinephrine)
levels. Furthermore, his ongoing studies at Stanford
University, building on the work of Syles, show that
sustained stress can damage the hippocampus, a
region of the brain central to learning and memory.
His work has identified glucocorticoids, a class of
steroids secreted from the adrenal gland during
stress, as critical to such neurotoxicity.
29
As for the psychosocial influences of consumerism
and affluence in many Western countries, the
same author writes, We live well enough to have
the luxury to get ourselves sick with purely social,
psychological stress.
30
In other words, we do not
need an external pathogen or a collision with a brick
wall to introduce serious disturbance into our being.
Our self-image, formed in relation to our perceived
social standing, can be enough to steer us toward
neurophysiological imbalance.
If lack of social control leads to high levels of
stress, then understanding the dynamics of power
relations within cultural and social contexts might
be just as important in responding to chronic
myofascial restriction as a good set of manual ther-
apy techniques. Developing social supports and
networks that enable a greater level of personal sat-
isfaction might be a central part of successful out-
comes in somatic therapy. Mentoring programs
and, for those with more income, personal coaches
could be important pieces of the puzzle for people
moving away from disabling patterns of constric-
tion! Fostering relationships that nurture is impor-
tant, as is identifying ones that do not. Education
can lead to feelings of control and participation.
Social control leads to a change in physiology. The
term preventive medicine takes on a new and
challenging meaning here!
I started this section by pointing to the difficulty
of talking about the body in a universal biological
sense. I proposed that self-image arises in social and
cultural contexts. My intention was to suggest a
fuller, and hopefully more productive, questioning
of what might be affected when deep release and
repositioning in the support, transport and coordi-
nation systems of a person take place. It also
enables a more realistic assessment of the limits of
our work. These limitations are not always structural
but can have their genesis in functions that are
intricately tied to social forces.
18 Section 1 THE BASICS
Thorax flexion
Vitality
Breathing
Headache
Indigestion
Courage/
vulnerability
Relationship
with partner
Sexual
attractiveness
Physical size
Feeling good
about yourself
Gait/movement
quality
Neck tension
Financial
situation
Figure 2.5 An expanded version of these complex interdependencies that includes psychosocial and psychological
factors (from Schleip,
7
with permission of the author).
Still, despite this journey into areas outside the
biological, I must acknowledge that the breadth of
the discussion has been limited. In particular, it
focuses on these issues in a Western consumer-
capitalist context where personal, individual sub-
jectivities are highly valued, especially within certain
socio-economic groups.
31
Certainly, not all cultures,
or classes within Western society, place the same
emphasis on the formation of these highly individ-
ual subjectivities.
A GRAND UNIFYING THEORY?
In examining the causes for disruption to the nor-
mal function of the myofascial complex, Chaitow
has used the delightfully simple phrase something
happens.
32
He then lists the various possible com-
ponents leading to this disruption (Box 2.1). It is
Chapter 2 DEVELOPING A HYPOTHETICAL MODEL 19
Box 2.1 Causes of soft tissue dysfunction.
The something happens proposal (from
Chaitow,
32
with permission).
Congenital factors (short/long leg, small
hemipelvis, short upper extremity, fascial, cranial
and other distortions)
Overuse, misuse and abuse (and disuse)
factors (such as injury or inappropriate patterns
of use involved in work, sport or regular
activities)
Postural stresses
Reflexive factors (trigger points, facilitated spinal
regions)
Chronic negative emotional states (anxiety,
repressed anger, etc.)
Nutritional deficits
Toxic accumulations
Infection
Endocrine (hormonal) imbalances
Box 2.2 The everything happens response to direct technique MFR. The net is cast wide to
include the co-emergent relationships that exist between the biological and the social.
Golgi tendon organs stimulated tone inhibited.
Ruffini endings are stretched, resulting in
inhibition of overall sympathetic nervous system
activity.
Reductions in SNS activity affect smooth muscle
fibers found in fascia fascial and whole organism
decompression. Increased parasympathetic activity
enhances whole organism well-being.
Interstitial receptors are stimulated especially via
work on the periosteum, resulting in increased
proprioceptive acuity as well as ground substance
renewal.
Muscle spindles are slowly stretched resulting in a
lowering of muscle tone.
Supraspinal inhibition of gamma gain leads
to resetting of gamma bias. Gravity response
improves.
Self-defeating cocontraction patterns of movement
(agonist and antagonist firing at the same time) are
reduced, resulting in heightened energy for creative
and expressive movement.
Mechanical restrictions to breathing are released
leading to overall improvement in physiology and
balance in CO
2
/O
2
ratios in the blood. Further
changes in the ANS take place.
Lymph and all fluid return is improved. Fascial
cohesiveness begins to improve.
Postures of defeat are reduced, allowing for
explorations of a new self-image that is oriented
in real time and space (here and now, take up
your space) rather than via inner narrative
which is oriented to the there and then
(if only).
Whole organism decompression leads to new
expanded relationship with the environment and
more satisfying interactions with it. Self-confidence
and esteem are boosted.
It takes two to know one. The communication
taking place in the therapeutic relationship is a
springboard to a new formulation of self (for client
and therapist). Movement and touch behaviors
modeled by the therapist generate new potentials
for the client.
Introduction of neural plasticity (ANS and CNS)
leads to better movement that orients new collagen
fibers into more supportive and less constrictive
arrangements. Refreshing the ground substance
creates greater interfiber distance that reduces
binding between fibers, long-term changes in
fascial cohesiveness now possible.
Self-regulating function of the body enhanced,
resulting in better overall health, especially in
all systems regulated by the ANS basically
everything!
possible to use a similarly useful vernacular term
and say that the response to direct technique MFR
is that everything happens. While this is, of course,
not completely true, it does convey the breadth of
the witnessed effects. The response is not localized
to one system but percolates into many. Non-linear
systems theory, with its multitudes of interdependent,
co-emergent relationships, makes the everything
happens hypothetical model viable, even as it takes
the interested reader outside the comfort zone of the
more usual cause and effect approach (Box 2.2).
Further reading
Bradley D 1998 Hyperventilation Syndrome: Breathing
Pattern Disorders, 3rd edn. Tandem Press, Birkenhead
Dinah Bradley is a New Zealand-trained
phsyiotherapist who has worked in a wide range of
settings. However, her long-time interest has been
respiratory therapy, especially the poorly documented
syndrome of chronic hyperventilation. This book is
written for the lay person who seeks to understand
the range of problems caused by hyperventilation.
It contains excellent material on the relationship
between the ANS, CO
2
, anxiety and other unpleasant
physiologic events. It provides sufficient information
to inform both the health practitioner seeking more
understanding of this surprisingly common problem
as well as clients who want tools for self-management.
I recommend it as a primer. If the material in here is
especially relevant to your practice then I suggest
going onto the more substantial and excellent
Multidisciplinary Approaches to Breathing Pattern
Disorders by Chaitow, Bradley and Gilbert (Churchill
Livingstone, Edinburgh, 2002).
Chaitow L 1999 Cranial Manipulation Theory and
Practice: Osseous and Soft Tissue Approaches. Churchill
Livingstone, Edinburgh
The text is easy to follow and links the theory to the
practical problems of the clinician. The book
describes both soft tissue and osseous applications as
well as providing guidance on which option to select
in different clinical situations. Practical exercises are
included to help improve clinical skills. Chaitow does
a thorough literature review of recent research into
cranial motion and rhythm. In fact, this review is one
of the books most important elements. Chaitow
uses the information he gathers to challenge much of
the previously unquestioned dogma that some of the
principal teachers of cranial manipulation still adhere
to. While Chaitows role as iconoclast might offend
some, most readers will find his frank reevaluation of
the main cranial theories to be a breath of fresh air in
an area that has for too long been muddied by
jargon, dogma and isolationist politics.
Chaitow L, Bradley D, Gilbert C 2002
Multidisciplinary Approaches to Breathing Pattern
Disorders. Churchill Livingstone, Edinburgh
For many years, decades even, I observed a strong
link between breathing patterns that seemed less than
ideal and anxiety states. Or to put it another way,
high rapid breathing was associated with SNS arousal
as well as, generally, hypertonic myofascia. Bradleys
first book gave real insight into the physiologic
processes accompanying chronic hyperventilation
syndrome (CHS). Then this much more complex
volume appeared, complete with a variety of manual
therapy techniques for addressing the physical
restrictions of the rib cage that perpetuate the
syndrome. This is a great text. If 12% of the general
population suffers with CHS, as Bradley suggests,
then that alone is significant. But given the
relationship between CHS, the SNS, anxiety and
changes in myofascial tone, the percentage of people
presenting at a manual therapy clinic is probably
much higher. Its worth knowing about!
Cottingham JT 1985 Healing Through Touch: A
History and Review of the Physiological Evidence. Rolf
Institute, Bouder, CO
While somewhat dated now, Cottinghams book
offers a perspective on aspects of physiology that
have immediate relevance for bodyworkers. His
investigation into this area turned up many
important research findings that might otherwise
have gone unnoticed. His seminal research into the
autonomic nervous system makes his contribution
to the area of manual therapies a significant one.
Ive hoped for some time that Cottingham might
rework this slender text into an updated and more
comprehensive version.
Grossinger R 1995 Planet Medicine, Vols I & II.
North Atlantic Books, Berkeley, CA
The author, who holds a doctorate in anthropology,
with a specialization in medical anthropology, has
developed a two-volume work that can easily be
described as encyclopedic. For anyone with a desire
to understand the real history of medicine and be
able to place any healing practice in its historical
context, this work is required reading. Its especially
strong in the area of somatics. While I do not share
all of the authors sensibilities, I consider this to be a
well-articulated work and highly recommend it.
20 Section 1 THE BASICS
Juhan D 1987 Jobs Body: A Handbook for
Bodyworkers. Station Hill Press, New York
The style is extremely approachable yet this text
contains a great deal of complex information that is
useful to anyone interested in touch therapies. The
section on muscle tone is excellent and will satisfy
the needs of most bodyworkers. This is basically a
foundation text for all bodyworkers.
Rolf IP 1978 Rolfing: The Integration of Human
Structures. Harper and Row, New York
When it was initially published this book was the first
of its kind an attempt to develop a view of holism
that included the relationship of human structure
to the gravitational field. For Rolf this was the
culmination of decades of research, clinical practice
and philosophical thought. She drew on her
knowledge of fascia (the subject of her doctoral
dissertation) to give her work an impressive depth of
understanding. Rolf s writing is characterized by
precise use of language (influenced no doubt by her
studies in General Semantics) and passion for her
ideas about balanced posture. Many of her ideas are
now so integrated into bodywork practice and jargon
that its easy to forget how original and unique her
line of inquiry was. Few people have articulated so
fully an original formulation of what integration
means, let alone a set of protocols to consistently
achieve it. Rolf is definitely one of them.
Schleip R 2003 Explorations of the Neuro-myofascial
Net. Journal of Bodywork and Movement Therapies
7(1):1119
Schleip is a master synthesizer. Working from the
perspective of a practicing Rolfer
and Feldenkrais
practitioner, he has gathered an impressive array of
research that is related to his ongoing passion what
happens when we touch someone in therapy? Why
does it work? This article, and another from April
of the same year, are some of the best contributions
to this understanding that I have seen. Schleip blends
the findings of solid research into a wonderful
narrative about talking to schools of fish, wet tropical
neurofascial jungles and other lively analogies. While
there is academic rigor displayed throughout, the
lasting impression of these articles is that they are fun
to read.
Schultz L, Feitis R 1997 The Endless Web. North
Atlantic Books, Berkeley, CA
Schultz is a long-time Rolfer
before training as an
osteopath and now practices medicine and Rolfing
instructor, holds a
doctorate in philosophy and is a long-time practitioner
of Zen. Although it is at times repetitious, this work
nevertheless rewards the reader with important insights
into the lesser world of somatics and the larger world
of life itself. Its relevance to this chapter lies primarily
in the exploration of various ways of viewing self and
how these can give way to a greater sense of no self.
32 Section 1 THE BASICS
Chapter 5
TOOLS OF THE TRADE
A MANUAL THERAPISTS COMMUNICATION MEDIA
Straw polls conducted in my classes reveal that as many as 45% of participants
report some peripheral neuropathies involving the fingers, wrist and/or fore-
arm. Since not all participants are massage therapists around 20% of them
come from other manual therapy traditions, especially physiotherapy and
osteopathy it appears that all types of practitioners are at risk.
Massage therapists injure the carpal ligaments via grasping motions
with the hand during kneading-type procedures, especially the abductors
digiti minimi and pollicis longus as well as the opponens digiti minimi and
pollicis, all of which pull on the flexor retinaculum at the wrist. General
narrowing and compression of the thoracic outlet due to sustained thoracic
and cervical flexion while working is a related problem. Mobilizing physio-
therapists also appear to put the thoracic outlet under strain for much the
same reason. When asked about the onset of their problems, most practi-
tioners associate the peripheral neuropathies with their work. Clearly, this is
not good.
All practitioners need a set of working tools that can serve them well
across decades of manual therapy. Insight into structure and function, along
with a big bag of manual therapy techniques, is not going to be useful if a
practitioner disables themselves via patterns of overuse, strain and subsequent
fatigue.
Apart from the obvious benefits to our patients from the use of these tech-
niques, another equally important one is the benefit to the practitioner of
doing direct technique MFR . A variety of tools including the fingers, elbows,
knuckles, forearms and, less frequently, the thumbs can be employed. As the
following photos illustrate, the slowness of the work enables the therapist to
pay considerable attention to body use while treating in this manner. In fact,
its possible to explore many aspects of ones own coordination and economi-
cal movement while still paying close attention to the client. This results in
much better contact and communication.
Developing these various aspects of coordination, stability and appropriate
strength takes around 2 years although refinement may continue across a life-
time. While this conditioning takes place, the practitioner will want to shift
frequently from one tool to another.
33
THE FINGERS
Always keep the fingers slightly flexed with the wrists in a neutral position.
Keep a slight arch at the MP joints as well as at the carpal tunnel (Fig. 5.1).
34 Section 1 THE BASICS
Figure 5.1
Correct: the wrists are
in neutral and the fingers
are in a slightly flexed
position.
Figure 5.2
Correct: working with one
hand on top of the other.
Developing strength and stability in the fingers takes time and practice but
unless you are truly double jointed, this can be accomplished. Another way
to utilize the sensitivity of the fingers without overworking them is to lend sta-
bility by working with one hand over the other. This is excellent for sustained
contact with minimal effort (Fig. 5.2).
While this conditioning takes place, rest the fingers by making use of the
elbows and knuckles. Resting is indicated when the fingers shake, collapse or
cannot be prevented from hyperextending (Fig. 5.3).
Chapter 5 TOOLS OF THE TRADE 35
Pain and inflammation in the PIP or DIP joints strongly suggest that earlier
signs of strain have been ignored. Even though sensitivity in the elbow and
knuckles improves with use, experience shows that the fingers remain the most
sensitive tool of all. Its worth spending the time to condition them correctly
from the outset.
THE FIST
The fist refers to the use of the four knuckles between the metacarpals and
phalanges (MP joints). It is usually a soft fist where the fingers are left extended
and folded into the thenar and hyperthenar eminences while the thumb rests
lightly on the first finger (Fig. 5.4).
Figure 5.4
Correct: the thumb is
relaxed and the forearm is
pronated sufficiently to
adopt the shaking hands
attitude with the fist.
Figure 5.3
Incorrect: ouch! The wrists
are flexed while the fingers
are hyperextended with
excessive force at the MP
joints.
Just as with the fingers, the wrist should be at neutral while the elbow is
best kept straight, without taking it into hyperextension (Fig. 5.6).
36 Section 1 THE BASICS
Figure 5.6
Incorrect: a variation
on using the fist that is
sometimes used. The
weight is going through
the carpal bones, placing
strain on both the carpal
tunnel and the median
nerve.
Turn the thumb up into the position you take for shaking hands and you will
present your wrist and fist to your contacts in a stable and stress-free manner.
Configured in this way, the fist becomes an amazingly adaptable tool that is
quite capable of artfully following the contours of bones, shearing fascial layers
or melting through large muscles like the gluteus, all with a wonderful econ-
omy of effort (Fig. 5.5).
Bracing the elbow against your body can be helpful at times in which case it
will obviously have to be more flexed.
Figure 5.5
Correct: the arm is stable
and straight. The weight is
able to transmit directly
through to the contact
site. This is using gravity
to bring about release.
Chapter 5 TOOLS OF THE TRADE 37
The therapists skin can become sensitized and even painful during the first
few months of working with the elbow. The skin feels like its being peeled off
the bone in a most non-therapeutic manner. Once again, the best response is
to shift to other tools and give the overly stressed tissues a rest. Generally, con-
ditioning occurs rapidly and once its established, there are few complications
associated with using the elbow.
Be mindful of the attitude that you bring to working with the elbow. Elbow
work need not be synonymous with deep and painful. Use its broad surface
area to sink into tight, fibrous tissue with precision. This feels fantastic.
Try to avoid working with extreme internal rotation at the shoulder as, over
time, this will cause damage to that joint. Also avoid collapsing into the contact
and narrowing the thoracic outlet. The fist should not be clenched (Fig. 5.8).
THE KNUCKLES
The thumbs are vulnerable to injury from overuse and the knuckles PIPs
can often be used in their place. Like elbows, knuckles can sound like theyre
simply about intensity of contact. However, there can be a great deal of nuance
to their use. The list of situations where they are useful includes the plantar
THE ELBOW
These days most therapists are happy to work with their elbows as theyve dis-
covered they are excellent tools in a wide range of situations. Our collective
carpal ligaments, forearm flexors, median nerves and thoracic outlets no doubt
enjoy this development. Although the term elbow is used this is not neces-
sarily accurate as the point of contact can vary. The olecranon process itself is
only one possibility. Often, the contact is slightly distal to the olecranon and
involves contact through 34cm of the ulna (Fig. 5.7).
Figure 5.7
Correct: the contact
is close to the olecranon
but not right on it. The
fingers are not tense or
making a fist.
Avoid deviation of the fingers as well as prolonged periods of wrist exten-
sion, both of which can lead to sore joints (Fig. 5.10).
THE THUMBS
Of course, sometimes a thumb is just right. However, pay attention to the
angles. Hyperextension over a long period of time can render the thumb
38 Section 1 THE BASICS
Figure 5.9
Correct: the MP and wrist
joints are at neutral.
Figure 5.8
Incorrect: the fist
is clenched and the
shoulder is in full internal
rotation. Too much effort
is being used.
fascia, galea aponeurotica, the retinacula of the ankles and the palmar aspect of
the hand. Like the elbow, the contact is rarely right on the points of the joints
but spread onto the shafts of the phalanges (Fig. 5.9).
Chapter 5 TOOLS OF THE TRADE 39
extremely painful, if not fully inoperable. Ive known some excellent Shiatsu
therapists who did long-term damage to the thumb in this way (Fig. 5.11).
Figure 5.11
Incorrect: good palpation
tools but not useful for
MFR or any transmission
of force.
Figure 5.10
Incorrect: the fingers are
deviated and the wrist is
away from neutral.
At times, having the thumbs in this hyperextended position can be
useful when palpating. For example, the thumbs work well to assess the
relative position of the transverse processes, other bony landmarks or soft
tissue tone.
For good support, keep the thumb snug against the first finger, which in
turn is held in the soft fist position. This provides a great deal of stability with
no strain to the carpometacarpal joint and surrounding ligaments (Fig. 5.12).
EFFECTIVE FULL BODY USE
Most manual therapists are given at least basic instruction on body mechanics
in their courses. While the quality of this input varies a great deal, one recur-
ring theme is the need to use bodyweight rather than muscular effort where
possible. This is useful but insufficient. For instance, it fails to address the fact
that transmitting the force of bodyweight through the upper extremity into a
clients body requires an equivalent amount of work to be done in stabilizing
the shoulder joint and girdle. Otherwise we would lean into our work through
our hands only to have our shoulders move in the opposite direction and ren-
der the contact ineffective. Since this only happens to a small extent, its clear
that we are stabilizing that joint, whether we are aware of it or not.
There are necessary additions to be made to this advice about gravity. Try
working with the hip hinge as the primary axis of movement for lowering the
bodyweight towards the client. At the same time, maintain an awareness of the
sacrum and coccyx dropping down, away from the movement of the head,
which is reaching forward. (For more information on the value of reaching in
a specific direction, see below.) Bringing your weight forward in this way will
contribute to an overall lengthening of the spine, with an associated opening
of the chest, while working (Fig. 5.13).
Activating the hip, while allowing the coccyx to reach in the opposite
direction to the contact being made with the client, will enable the therapist to
elongate the hamstrings, lengthen the front line of the trunk and maintain
access to the diaphragm. Working in this manner means the therapist can be
exploring internal space, stability and elongation during treatments! The same
attitudes can be brought to work done from the seated position (Fig. 5.14).
This has to be better than getting locked into exaggerated thoracic kypho-
sis, with accompanying internal rotation of the humeral heads, dropped clavi-
cles, depression of the upper ribs, exaggerated cervical flexion with associated
capital extension, a posterior pelvis, short hamstrings and disconnection from
the feet and ground (Figs 5.15 & 5.16).
40 Section 1 THE BASICS
Figure 5.12
Correct: the thumb is
protected from any strain
at the MC joint.
Chapter 5 TOOLS OF THE TRADE 41
Try to get the pelvic and shoulder girdles facing in the same direction as
much as possible. Too much counterrotation between the two girdles can
cause facet joint pain and stiffness in the thoracic and lumbar spines, asymmet-
rical muscle tone and even functional scoliosis a surprisingly frequent set of
problems brought on by the work of manual therapists.
Figure 5.14
Correct: seated position
enables the same sense of
support, direction
and span.
Figure 5.13
Correct: the hips are
engaged as the major
point of flexion while the
spine is stable, elongating
and dynamic.
Working from a position of support means much more than saving our bod-
ies from excessive wear and tear. Doing direct technique MFR is about com-
munication. When there is ease and balance in the body of the therapist, this is
transmitted to the client as clear intention and a purposeful, responsive touch.
This same attitude of economy of effort frees up energy for the therapist to
feel, or in other ways sense, the variety of responses of the client to the input
42 Section 1 THE BASICS
Figure 5.16
Incorrect: loss of support
and direction leads to
shortening and collapse.
Figure 5.15
Incorrect: loss of support
and direction leads to an
overall shortening of the
therapists body. Resting
on the elbows forces the
shoulders into the ears.
Chapter 5 TOOLS OF THE TRADE 43
Try again but this time the person with the extended arm reaches with
their intention to a point on the wall behind you or even through the wall. In
other words, they have a specific sense of direction rather than simply resist-
ance. Generally, the pusher will find the arm much harder to push down while
the reacher will feel much stronger and more stable (Fig. 5.18).
What is happening here? And how does this Karate Kid energy stuff help us
in our work? Although its beyond the scope of this book to fully explore the
neurology of this cute parlor trick (some of you may have already come across
it in schools of chi development and so on), it can be said that movement made
with a sense of direction recruits the appropriate muscles but not their antago-
nists. The first movement, where effort was made to resist the downward force,
without any deliberate direction, generates a high degree of contraction in the
agonist and the antagonists. The muscles work against each other and effec-
tively weaken in a mistaken attempt to make stronger.
The Feldenkrais teacher Ruthy Alon suggests that these kinds of contrac-
tions involve what she terms parasitic muscles whose involvement we are not
they are receiving. This dynamic feedback loop is at the heart of doing good
therapy. Establishing a consistency in economical body use goes a long way
toward cultivating this potential.
An experiment
Try this simple experiment. Stand beside a friend whose shoulder should be
flexed to 90 with the elbow extended. Grasp their arm and attempt to push
it toward the ground while asking them to resist (Fig. 5.17). Both persons
should note the effort involved.
Figure 5.17
Stability attempted
without direction.
usually aware of as they are habitual and not easily brought into awareness.
1
These automatic subroutines are generally going on all the time and they inter-
fere with many aspects of balanced tonus.
2
This is a shot-gun approach to mus-
cle contractions; getting everything to fire will certainly move something but
without finesse. And with fatigue.
For therapists this has far-reaching consequences, if youll pardon the pun.
As we do the various actions to explore the positions suggested above, we can
add another dimension that will significantly diminish the effort we make
as we work. Its about direction rather than effort. If you work into the myo-
fasciae of the hip triangle but think about reaching through that anatomic region
into the table or through to the floor (or the center of the earth?), you add
direction. The muscular effort will diminish while the effectiveness of your
work will increase. While this can be hard to learn the Protestant work ethic
may have captured vast tracts of your nervous system it can happen if you
pay attention to it. Think direction rather than wrestling match.
The process of decompressing and releasing is a potential in the clients body.
The best way to activate it is to communicate with just the right amount of
effortlessness. Zero effort and you have, perhaps, energy work, Reiki and the
subtlest ends of the cranial spectrum. This might not be the agreement for ther-
apy that you have made with your client. Too much effort and you can be goug-
ing, ripping or thumping. This is probably not the agreement for therapy that
you have with your client either. (Please, really, no ripping or thumping, ever.)
If you like this fun game then it can be developed in other ways. While stand-
ing, and before making contact with the arms/fingers/elbows onto the client,
find, clearly, the awareness of your feet. This is all about sensation temperature,
pressure and texture can each be contacted. Then extend this into an imagined
sense of being supported at a point about a meter below you or the center of
44 Section 1 THE BASICS
Figure 5.18
Stability with a deliberate
sense of direction.
Chapter 5 TOOLS OF THE TRADE 45
the earth if you have a good imagination rather than at the floor and allow
your imagination to take your awareness through to that point. The first time
it may take a few seconds; subsequent visits take much less. Note the relative
sense of support when contrasting this more deliberate direction-specific con-
nection with your normal habit. One consistent feature of the sensation-rich
approach to orienting against gravity is a sharp reduction in tiring patterns of
muscular co-contraction.
For the technique driven this can appear to be a frivolous diversion from the
real tasks of manual therapy. Of course, some people are blessed with a natu-
rally flexible, robust nervous system. Many are not, though. I see many clini-
cians who are fatigued from doing soft tissue work with a lot of over-exertion.
A friend calls work like this white collar laboring. The burnout rate amongst
the manual therapy laboring class is, regrettably, very high. Perhaps theres a
place here for the old Work smarter, not harder maxim, given here with some
hints that may allow it to integrate into your working days.
So feeling tired or crunched in a session? Disconnected? Check and see if
youve lost sensory awareness of your feet/hands/back/head. Orient to sen-
sation and then add direction to feet/hands/back/head and see if this unwraps
you without any big attempt to adopt correct form (remember the wasted
effort made in the unbendable arm game).
I suspect that many of the really fine manual therapists who contact their
clients across a spectrum of levels, from the energetic to the dense, are engaged
with this type of low-effort, direction-specific contact. Milne describes his
approach to cranial work in the beautifully titled The Heart of Listening.
3
This
book is rich in the kind of evocative imagery and metaphoric language that
can shift us away from the hard yards of soft tissue slug fests in resistant tis-
sues. One can see how this style of sensation-rich contact, married with a sense
of direction, could lead us to a new appreciation of what it means to touch,
work and listen at the same time. This kind of dynamic, of saying hello through
touch and listening for the response, is central to what this book is about.
COMMENTS FOR PEDIATRIC THERAPISTS
In the pediatric settings where I work, we treat children all over the place
floors, wheelchairs, cradled in the mothers arms, mats on conference tables,
and so on. Rarely have I worked on anything resembling a true treatment
table, adjusted to the correct height for my body. Electric height-adjustable
tables seem even less common.
The most common place is a mat on the floor. Take care of your own body
when working on the floor. Ive found its easy to start feeling crunched after
spending a few hours doing MFR down there. Then the quality of touch starts
to change. Contact made with a clear sense of direction on the part of the
therapist is often replaced, at best, by the application of a correct technique.
The hand holds will be right, the anatomic structures being touched are cor-
rect but the communication changes. Mechanical pushing into the intrafascial
mechanoreceptors replaces a sustained hello. Enter the Law of Diminishing
Returns: the harder you work, the less effect it seems to have.
Learning how to maintain a stable core is important for everyone. For manual
therapists and somatic practitioners who spend a lot of time as floor workers,
it is essential. Working in the standing position makes it easier to engage and
work from the core stabilizers. This is because of the closed chain kinetic con-
nection through to the feet. Working on the floor, this chain is then opened,
which results in movement with increased amounts of co-contraction. Large
phasic muscles are used as stabilizers. This is tiring, exhausting even.
In my highly informal straw polls taken in the various classes I teach, pedi-
atric therapists report a higher incidence of SIJ and lumbar pain than other
therapists. I suspect that this is associated with the failure to successfully stabi-
lize the pelvis, sacrum and lumbar spine due to the amount of time spent in
the open chain relationship to gravity. It might also have something to do with
the fact that the majority of pediatric therapists are women. Many have had
children, another contributing factor to weakening of the deep abdominal sta-
bilizers. These suggestions for better body use may be helpful no matter what
the underlying cause of the instability may be.
Work in a dynamic two-point kneel. This closes the chain. Augment this by
dorsiflexing the back foot sufficiently to get the toes on the floor rather than
resting on its dorsal surface in a passive plantarflexed position. Reach/step
through to the toes to initiate forward movement into the hands. This acti-
vates the same vital connection between the foot and thoracolumbar fascia as
walking. This is where the support for the low back can come from.
Test the difference. Pay attention to the hips, the internal spaces of the pelvis
and the breath while doing this. Do the movement as described, with the toes
on the ground and the plantar surface of the foot on slight stretch. Reach into
the back foot while extending both arms into the floor/pillow/person, etc.
Now drop the foot into plantarflexion and make the same forward-reaching
motion through into the hands. Generally, the first approach will leave the
hips feeling loose and open, also the pelvic floor and breath. In the second
approach, the hip muscles will have to stabilize the pelvis, leading to feelings
of restriction there as well as in the breathing. This can easily be verified by
attempting to wag your tail while in each position (Fig. 5.19).
46 Section 1 THE BASICS
Figure 5.19
Body position for stability
while working on the floor.
Chapter 5 TOOLS OF THE TRADE 47
GENERAL POSTURAL STRATEGIES FOR FLOOR WORKERS
Keep the waistline at neutral as much as possible. Avoid pulling up into
hyperlordosis or crunching over into lumbar kyphosis.
Feel the spine as long without forcing it the coccyx drops down and the
top of the head lifts up. (Waistline at neutral!) This works especially well
when there is a closed chain situation in the lower extremity but can cer-
tainly be explored at all other times.
Allow the back of the head to feel wide and the suboccipital triangle
to open.
Soften the gaze frequently. The effect of this is amazing. Really. Try it.
Stay sensory; notice texture, temperature, pressure, light, shadow, color and
shape. Coming to our senses is one of the most central components of
changing posture. This is not labored, serious stuff but as simple as noticing
in an instant the position of a hand, the feeling of our skin on anothers
skin, the texture of the carpet, the mortar in the bricks. See Chapter 4 for
more on this line of thinking.
Figure 5.20
Use of a kneeling stool to
support stability while
treating on the floor.
Try working from a kneeling bench a heavy-duty meditation cushion is
used in Figure 5.20. While this is not as dynamic as the two-point position
described above, it offers advantages over sitting cross-legged, mainly that the
knees are lower than the hips, leaving the pelvis free to find an easy neutral posi-
tion. Sitting cross-legged, where the knees are invariably higher than the hips,
pushes the pelvis into posterior tilt, quite often to an extreme degree. Once
there, postural stability is almost impossible to maintain, with the spinal erec-
tors eventually exhausted by their efforts. Burning, painful hot spots develop at
various sites along the back and in the suboccipital area. These hot spots give
the nervous system plenty of noxious stimulation and diminish the chance of
discrete muscular balance of the type required for easy posture (Fig. 5.20).
References
1. Alon R 1996 Mindful spontaneity. North Atlantic Books, Berkeley, CA
2. Frank K 1995 Tonic function: a gravity response model for Rolfing structural
integration and movement integration. Self-published. Available online at:
www.somatics.de
3. Milne H 1995 The heart of listening, vols I & II. North Atlantic Books,
Berkeley, CA
48 Section 1 THE BASICS
INDEX
A
Abdominal muscles 106108
lower 8284, 89
Abductor pollicis brevis 169,
170
Adductors (thigh)
posterior release 63
sidelying release 6466
standing release 7071
Analgesics 155
Anatomy 3
Ankle retinaculum release 5152
Ankle stiffness 52, 53
Antagonists, muscle 26
Anterior compartment/interosseus
membrane release 5253
Anterior/lateral cervical region
129132
sidelying (children) 141143
Anterior trunk see Front of trunk
Anxiety states, chronic 108,
112113, 132
Arthritis, hand 175
Asthma 84, 97, 106
Autonomic nervous system (ANS)
1013, 15, 16
hand releases and 171, 174175
signs of rebalancing 12, 2627
suboccipital work and
139140
temporalis fascia work and
147148
thoracic outlet work 154
see also Parasympathetic nervous
system; Sympathetic nervous
system
Axillary zone releases 158160,
176177
B
Back 27, 92102
children, seated work
118119
deeper muscles 100102
prone work 95100
upper/mid trapezius and levator
scapula 9295
Back pain
floor workers 46, 47
low 66, 82, 86
Balance training 53, 56
Biceps brachii tendon 165166
Biceps femoris release 6263
Bilateral work 26
Bladder infections 80
Blood pressure 12
Body use, effective full
4047
Bodyweight, applying 2425,
40
Brachial fascia 161
Brachial nerve restrictions 165
Bradley, D 20
Breathing
accessory muscles 122, 132
functional releases 114118
Breathing patterns
disordered 20, 112113
expiration fixed 113
inspiration fixed 113
Breaths
monitoring 26
rectus abdominis and sheath
release 108
releasing-type 97
therapeutic 56, 113
Buddhism 2930
C
C7T1 93, 97, 136137
Calcaneus, standing release 6970
Calm abiding (shamatha) 29, 30
Calm state, deep 12, 2627,
139140
Carpal ligament injuries 33
Carpal tunnel 170171
Carpal tunnel syndrome 160, 163,
165, 171
Cells 78
Central nervous system (CNS)
1315, 16
lesions 28
Cerebral palsy (CP) 6, 28
pelvic techniques 87, 88, 89
upper extremity work 175176,
178, 179
see also Pediatric applications
Cervical region 26, 27, 129143
cranial base and suboccipital
myofasciae 138140
deep posterior myofasciae
136138
infrahyoid 132133
lateral/anterior 129132
longus colli 134136
occipital condyles 140141
pediatric supplement (sidelying)
141143
suprahyoid 133134
Chaitow, L 18, 20
Chaperones 80
Children see Pediatric applications
Chronic fatigue 26, 175
Collagen 8
crosslinks 9, 10
effects of immobility 89
182 INDEX
Collagen (contd)
effects of myofascial release 10, 16
scar formation 10
Colon 81
Communication 34, 2425, 26
Connective tissue 710
cells 7
dense irregular 8
dense regular 8
effects of immobility 89
extracellular matrix 78
loose irregular 8
scar 910
thixotropy 8, 10
types 8
Conoid ligament 163164
Constipation 58
Contemplative approach 2932
Coracoid process
pectoralis release 108, 109, 110
release 157158
Cottingham, J 13, 20
Cranial base 138140, 153
Cranial manipulation therapy 20
Cranial rhythmic impulse (CRI)
1112
Cranial sacral therapies 148
Cranium, posterior 150151
Cross-legged position, sitting 47
D
Deep posterior myofasciae, neck
136138
Deeper back muscles
lower 100101
upper 101102
Defacilitation 2930, 31
Diaphragm, respiratory 112113
Digital fibrous flexor sheaths 167
Direction of working 4345
Disintegration 5
Double crush syndrome 160, 165,
168
Dreamy, relaxed state 12, 2627,
139140
E
Effort of working 4345
Elastin 8
Elbow
flexor release, children 177178
release 165166
working with 37
Embodiment 4
Emotional responses 115, 118
Epicranial aponeurosis 148149,
153
Erectors, spinal 100, 101
Exhaustion, signs 26
Extensor retinaculum, wrist 173
External obliques 102104
Extracellular matrix (ECM) 78
F
Face see Head and face
Feathering 2526
Feedback 4, 24
Feitis, R 21
Feldenkrais, Moshe 4
Femur, derotation 71
Fibroblasts 7, 10
Fibromyalgia 26, 175
Fingers
occupational injuries 33, 35
working with 3435
Fist, working with 3536
Flexor digitorum superficialis 167,
168
Flexor pollicis brevis 169, 170
Flexor retinaculum 169, 170171
Flexors carpi radialis and ulnaris
167, 168
Floor work 4547
Foot-awakening exercise 30
Forearm
extensor muscles 172174
flexor muscles 166168
Forefoot, standing release 6869
Freeing the breathing
number 1 114115
number 2 115116
number 3 116118
Front of trunk
deep 110118
superficial 106110
Frontal sinus 150
G
Gag reflex 125
Gait problems 53, 56, 69
Galea aponeurotica (epicranial
aponeurosis) 148149, 153
Gamma bias 14, 24
Gamma gain 14, 15
Gastrocnemius release 5354
Glucocorticoids 18
Gluteus maximus release 7576
Gluteus medius 58
Glycosaminoglycans 89
Golgi tendon organs (GTOs) 15
Granulation 10
Gravity, using 2425, 40
Grossinger, R 20
Ground substance 8, 10, 16
Guarding response 2425, 82
H
Hamstrings release
lateral 6263
medial 6061
pediatric 7172
Hands
dorsum release 174175
methods of working with
3436, 3739, 40
occupational injuries 33
palmar anatomy 169
palmar aspect release 168171
Hard palate release 125126
Head and face 145152
pediatric supplement 151152
Headache 153155
cranial base and suboccipital
region 139, 153
epicranial aponeurosis 149, 153
pelvic floor 80, 154
self-help 155
temporalis fascia 148
therapeutic goals 153155
upper/mid trapezius and levator
scapula 9495
Heel cord release 7274
Hip
extensors, stiff 72
flexors, tight 88
therapists 40, 41
Hip hiking 87
Histiocytes 7, 910
Homeostasis 11
Hyperventilation
acute 118
chronic 12, 20, 112113
Index 183
Hypnogogic state see Dreamy,
relaxed state
Hypothetical model 722
I
Iliac crest release 5657
children 8687
Iliacus release 8081
Iliocostalis lumborum 103
Iliotibial band (ITB)
release 5960
vastus lateralis border 66, 67
Immobility, effects 89
Incontinence 80
Inflammation 910
Infrahyoid region 132133
Integration 46
Internal obliques 102104
Interosseus membrane/anterior
compartment release 5253
Intrafascial mechanoreceptors
1516
Intraoral treatment 121128,
154155
pediatric supplement 126128
Irritable bowel syndrome 58
J
Joint
sensory receptors 13
stiffness 9
Juhan, D 21
K
Kneeling bench 47
Kneeling two-point position 46
Knuckles, working with 3738
Kyphosis, thoracic 106
L
Lateral/anterior cervical region
129132
sidelying (children) 141143
Lateral aspect of trunk 104106
Lateral hamstrings release 6263
Lateral pterygoid release 123124
Latissimus dorsi 102104, 106, 160
Levator scapula 9295, 130, 131
Line of tension 23
Longissimus capitus 139
Longus colli release 134136
Low back pain 66, 82, 86
Lower back
deeper muscles (seated)
100101
prone position 101102
Lower extremities 5174
pediatric clients 7174
Lower leg 5155
Lumbar lordosis, increased 82
M
Macrophages 7, 910
Maitland, J 32
Mandibular release 121122
Massage therapy 4
Masseter release 145146
Mast cells 7
Mastoid process 129130
Maxillary release 122123
Maxillary sinus 150
Mechanoreceptors, intrafascial 1516
Medial hamstrings release 6061
Medial pterygoid release 124125
Median nerve 168, 171
Medical manual therapy 4
Meditation 2930
Menstruation, painful 58, 80
Method, direct myofascial release
see Technique, direct myofas-
cial release
Milne, Hugh 45, 148
Modesty 25
Mouth see Intraoral treatment
Movement, therapeutic 2324
client-generated 2324
direction and effort 4345
practitioner-induced 23
Mucopolysaccharides 8
Multifidus 57, 9899, 100, 101,
102
Muscle spindles 14, 15
Muscle tone
autonomic control 11
central nervous control 1314
effects of direct myofascial
release 15
Myofascial layers 9
N
Nasalis muscle 149150
Neck stiffness 136
Neck work see Cervical region
Nerve fibers, intrafascial 1516
Neurologic disorders 27
Neuromotor controls 1315
Neuropathies, peripheral see
Peripheral neuropathies
Non-verbal communication 26
O
Obturator internus release 7980
Obturator nerve 80
Occipital condyles, decompression
140141
Occupational injuries 33, 46, 158,
160
Omohyoid 133
Opponens pollicis 169, 170
Orthopedic disorders 27, 28
Overuse injuries, occupational 33
P
Pacinian/Paciniform corpuscles 15
Painter, Jack 4
Palatine fascia release 125126
Palmar aponeurosis 167
Parasympathetic nervous system
(PNS) 1113, 25
signs of activation 12, 2627,
140
suboccipital work and
139140
see also Vagus nerve
Pectoralis major 108110, 154
deeper portion 161163
Pectoralis minor 154, 158159
children 177
investing layer 108110
Pediatric applications 6, 28
cervical region 141143
head and face 151152
intraoral treatment 126128
lower extremities 7174
pelvis 8690
trunk 118119
upper extremities 175179
Pediatric therapists 4547
184 INDEX
Pelvic floor
headaches and 80, 154
releases 66, 7980
Pelvic roll with lumbosacral traction
8486
Pelvis 57, 7590
anterior tilt 58, 67, 81
pediatric supplement 8690
Peripheral neuropathies
occupational 33, 160
releases 160, 163, 165, 168
see also Carpal tunnel syndrome
Piezoelectric effect 16
Pilates 2728, 132
Piriformis release 7779
Plantar myofasciae release 5556
Plasma cells 7
Plasma extravasation 16
Posterior compartment release
5355
children 7274
Posterior cranium 150151
Posture
central nervous control 1314
floor workers 47
therapists 4043
Pressure, applying 24
Procerus and nasalis muscles
149150
Pronator teres 167, 168
Prone back work
lower 97100
upper 9597
Psoas release 8182
advanced 110112
children 8890
Psoas tightness, Thomas test 67,
68
Psychosocial factors 1719, 25
Q
Quadratus lumborum (QL) 57
Quadriceps/anterior aspect of thigh
6667
R
Reciprocal inhibition 11, 13
Rectus abdominis
pelvic portion 8284
and sheath 106108
Reports, client 26
Respiration see Breathing
Respiratory diaphragm 112113
Respiratory disorders 106
Restricted layer, engaging 24
Reticulin 8
Ribs 108
axillary zone 158, 159, 176,
177
first 131, 132, 154
functional release 114118
lateral aspect 105, 106
posterior 97
timing of work on 27
Rolf, Dr Ida 4, 11, 21
Rolfing 4, 21
Ruffini bodies 15
S
Sacroiliac joint (SIJ)
movement 77
pain and dysfunction 47, 66,
79, 82
Sacrotuberous ligament (STL)
release 7677
Scalene muscles 122, 130, 132
Scapula
inferior angle 160
instability 158
Scar tissue 910, 74
Schleip, R 21
Schultz, L 21
Sciatic nerve 79
Seated back work, children
118119
Seated position, therapist 40, 41,
42
Self-help techniques, headaches
155
Self-image 17
Self-mobilization 2324
Semis (medial hamstrings) release
6061
Semispinalis capitis 139
Sensory awareness, therapists
3032, 4445
Sensory fibers, intrafascial 1516
Sensory inputs, disrupted 1314
Serratus anterior 106
Shamatha (calm abiding) 29, 30
Shoulder girdle
functional release 116118
occupational problems 158
Shoulder joint, fibrous restrictions
164165, 172, 175176
Shoulder region releases 157165
Sides, trunk 102106
Sinuses, paranasal 122, 150
Sitting cross-legged position 47
Smooth muscle cells, intrafascial 16
Social class 1718
Social support/networks 18
Soleus release 55
Somatic therapy 4
Something happens proposal,
Chaitows 18
Spinal erectors 100, 101
Splenius capitis 130, 139
Splenius cervicis 139
Standing position, therapist 4042
Standing release
adductors 7071
calcaneus 6970
forefoot 6869
Sternocleidomastoid (SCM) 122,
130, 131, 132
Sternohyoid 133
Sternothyroid 133
Stiffness, joint 9
Stress
psychosocial 18, 25
therapeutic 23
Subclavius 108, 109
Suboccipital region 26, 138140
headaches 139, 153
self-help techniques 155
Subscapularis 159160
pediatric 176
Suprahyoid region 133134
Sympathetic nervous system (SNS)
1113, 15, 16
chronic anxiety 113
signs of activation 12, 27
Systems non-linear approach 17
T
Technique, direct myofascial release
2328
developing a strategy 27
dynamic approach 2728
lesions 28
movement 2324
ordered approach 2527
using gravity 2425
Index 185
Temporalis fascia 147148
pediatric supplement 151152
Temporomandibular joint (TMJ)
121, 122, 154155
Tennis balls 155
Tensor fascia lata release 5758, 67
children 8788
Theory 722
alternatives 1016
grand unifying 1920
piezoelectric effect 16
psychosocial factors 1719
viscoelastic/thixotropy model
710, 14
Thigh
adductors see Adductors
anterior aspect 6667
Thixotropy 8, 10, 14
Thomas test 67, 68
Thoracic outlet
occupational injuries 33
releases 154
Thoracic spine stiffness 97, 101
Thoracic surgery 97
Thoracolumbar fascia 100,
102104
Thumbs, working with 3839, 40
Toe walkers 74
Tools of the trade 3348
Touch 34, 25
Transverse carpal ligament 169,
170171
Transversus abdominis 108
Trapezius, upper/mid 9295, 139,
153, 154
Trapezoid ligament 163164
Triceps release 171172
pediatric 178179
Trunk 91119
back 92102
deep front 110118
lateral aspect 104106
pediatric supplement 118119
sides 102106
superficial front 106110
U
Ulna, periosteum 173174
Undressing 2425
Upper back
deeper muscles (seated) 101102
prone position 9597
trapezius and levator scapula
9295
Upper crossed syndrome 162,
164
Upper extremities 157179
pediatric supplement 175179
V
Vagus nerve 11, 12, 16, 25
see also Parasympathetic nervous
system
Vastus lateralis, iliotibial band (ITB)
border 66, 67
Vipashana 2930
Voice production 134
W
Weislefish, Sharon 4
Whiplash, cervical 80, 132, 134,
136
Z
Zygomaticus major and minor
146147
CHURCHILL LIVINGSTONE
An imprint of Elsevier Limited
2004, Elsevier Limited. All rights reserved.
The right of Michael Stanborough to be identified as author of this work has been
asserted by him in accordance with the Copyright, Designs and Patents Act 1988.
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First published 2004
ISBN 0 443 07390 2
British Library Cataloguing in Publication Data
A catalogue record for this book is available from the British Library
Library of Congress Cataloging in Publication Data
A catalog record for this book is available from the Library of Congress
Notice
Medical knowledge is constantly changing. Standard safety precautions must be followed,
but as new research and clinical experience broaden our knowledge, changes in treatment
and drug therapy may become necessary or appropriate. Readers are advised to check
the most current product information provided by the manufacturer of each drug to be
administered to verify the recommended dose, the method and duration of administration,
and contraindications. It is the responsibility of the practitioner, relying on experience and
knowledge of the patient, to determine dosages and the best treatment for each individual
patient. Neither the publishers nor the author will be liable for any loss or damage of any
nature occasioned to or suffered by any person acting or refraining from acting as a result
of reliance on the material contained in this publication.
The Publisher
Printed in China
There are books which offer just another spin on a
common subject, maybe with a different emphasis
than previous authors, yet which add nothing sub-
stantially new to the vast field of literature that is
already available in the field of manual therapies.
The book which you hold in your hands, dear
reader, clearly belongs to a different kind. It is
the first time that an internationally respected
teacher of deep tissue work offers an easy to follow
and clearly organized manual for direct myofascial
techniques.
Among the field of myofascial release, two main
streams can be described. There are the more
recent schools of indirect release, which have been
influenced by Lawrence Jones, Rolin Becker, Jean
Pierre Barral and others. Their hands tend to first
go with the direction of the somatic dysfunction,
and then they allow the system to rewind itself
from there. For example if the right shoulder of
a client is chronically pulled forward, an indirect
approach practitioner will manually support the
shoulder going exactly in that forward direction
until some release is felt towards a new and less
contracted direction. Naturally these approaches
tend to be experienced as more gentle and less
intrusive by the client. Yet they also tend to have
their limits (or need to be repeated for years) in
many cases of severe tissue shortening or adhesion.
Several excellent courses, textbooks and manuals
are easily available on these techniques.
On the other side are the direct release tech-
niques, in which the practitioner works directly
towards the preferred and more healthy direction.
To loosen a tight myofascial area, the practitioners
hands or elbow slowly sink directly into the tight-
ened myofascial tissue. Often the client is then
invited to contact the same place from the inside
(via breathing or subtle joint movements) while the
manual pressure is gradually increased (up to sev-
eral pounds of pressure) until the tissue softens.
For example in the client with the protracted
shoulder, one might work directly on the tissues of
pectoralis major or minor or on the ligaments
around the coracoid process (see pp161163) in
order to release the shoulder directly out of its pro-
tracted pattern. This approach is often referred to
as deep tissue work and tends to be seen among
practitioners as more traditional. While being criti-
cized as too violent and as old fashioned by some,
it is also experienced as magically powerful and as
deeply profound by others. Most, yet not all, prac-
titioners of this approach have been influenced
by the work of Ida Rolf (18961979), founder of
the Rolfing