Fracture
Fracture
Fracture
Complete: Fracture line involves entire cross-section of the bone, and bone fragments are usually displaced.
Closed: The fracture does not extend through the skin.
Open: Bone fragments extend through the muscle and skin, which is potentially infected.
Pathological: Fracture occurs in diseased bone (such as cancer, osteoporosis), with no or only minimal
trauma.
Nursing Priorities
1. Prevent further bone/tissue injury.
2. Alleviate pain.
3. Prevent complications.
4. Provide information about condition/prognosis and treatment needs.
Risk for Peripheral Neurovascular Dysfunction Fracture Nursing Care Plan (NCP)
Risk for Impaired Gas Exchange Fracture Nursing Care Plan (NCP)
Impaired Physical Mobility Fracture Nursing Care Plan (NCP)
Impaired Skin Integrity Fracture Nursing Care Plan (NCP)
Discharge Goals
1. Fracture stabilized.
2. Pain controlled.
3. Complications prevented/minimized.
4. Condition, prognosis, and therapeutic regimen understood.
5. Plan in place to meet needs after discharge.
Additional Diagnoses
1. Trauma, risk forloss of skeletal integrity, weakness, balancing difficulties, reduced muscle coordination,
lack of safety precautions, history of previous trauma.
2. Mobility, impaired physicalneuromuscular skeletal impairment; pain/discomfort, restrictive therapies
(limb immobilization); psychological immobility.
3. Self-Care deficitmusculoskeletal impairment, decreased strength/endurance, pain.
4. Infection, risk forinadequate primary defenses: broken skin, traumatized tissues; environmental exposure;
invasive procedures, skeletal traction.
A bone fracture (sometimes abbreviated FRX or Fx, Fx, or #) is a medical condition in which there is a break
in the continuity of the bone. A bone fracture can be the result of high force impact or stress, or trivial injury as
a result of certain medical conditions that weaken the bones, such as osteoporosis, bone cancer, or osteogenesis
imperfecta, where the fracture is then properly termed a pathological fracture. Nursing goal for a patient with
fracture is to relieve pain, education about upcoming surgery, promote comfort and promote healing.
Types of Fractures:
Pathophysiology
The natural process of healing a fracture starts when the injured bone and surrounding tissues
bleed, forming a fracture Hematoma. The blood coagulates to form a blood clot situated between
the broken fragments. Within a few days blood vessels grow into the jelly-like matrix of the
blood clot. The new blood vessels bring phagocytes to the area, which gradually remove the nonviable material. The blood vessels also bring fibroblasts in the walls of the vessels and these
multiply and produce collagen fibers. In this way the blood clot is replaced by a matrix of
collagen. Collagens rubbery consistency allows bone fragments to move only a small amount
unless severe or persistent force is applied.
At this stage, some of the fibroblasts begin to lay down bone matrix (calcium hydroxyapatite) in the form of
insoluble crystals. This mineralization of the collagen matrix stiffens it and transforms it into bone. In fact,
bone is a mineralized collagen matrix; if the mineral is dissolved out of bone, it becomes rubbery. Healing
bone callus is on average sufficiently mineralized to show up on X-ray within 6 weeks in adults and less in
children. This initial woven bone does not have the strong mechanical properties of mature bone. By a
process of remodeling, the woven bone is replaced by mature lamellar bone. The whole process can take up
to 18 months, but in adults the strength of the healing bone is usually 80% of normal by 3 months after the
injury.
Several factors can help or hinder the bone healing process. For example, any form of nicotine hinders the
process of bone healing, and adequate nutrition (including calcium intake) will help the bone healing process.
Weight-bearing stress on bone, after the bone has healed sufficiently to bear the weight, also builds bone
strength. The bone shards can also embed in the muscle causing great pain. Although there are theoretical
concerns about NSAIDs slowing the rate of healing, there is not enough evidence to warrant withholding the
use of this type analgesic in simple fractures
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Pathophysiology
Acute Pain
Deficient Knowledge
Self-Care Deficit
Conspitation
Activity Intolerance
Impaired Physical Mobility
Situational Low Self-Esteem
Readiness for Enhanced Therapeutic Regimen
Risk for Infection
Psychological immobility
Possibly evidenced by
Traction/immobility device
Stress, anxiety
Possibly evidenced by
Reports of pain
Administer medications as
indicated: narcotic and
nonnarcotic analgesics,
e.g., morphine, meperidine
(Demerol), hydrocodone
(Vicodin); injectable and oral
nonsteroidal anti-inflammatory
drugs (NSAIDs), e.g., ketorolac
(Toradol), ibuprofen (Motrin);
and/or muscle relaxants, e.g.,
Maintains strength/mobility of
unaffected muscles and facilitates
resolution of inflammation in
injured tissues.
Improves general circulation;
reduces areas of local pressure
and muscle fatigue.
Refocuses attention, promotes
sense of control, and may
enhance coping abilities in the
management of the stress of
traumatic injury and pain, which
is likely to persist for an extended
period.
Prevents boredom, reduces
muscle tension, and can increase
muscle strength; may enhance
coping abilities.
May signal developing
complications; e.g., infection,
tissue ischemia, compartmental
syndrome.
Reduces edema/hematoma
formation, decreases pain
sensation. Note: Length of
application depends on degree of
patient comfort and as long as the
skin is carefully protected.
Given to reduce pain and/or
muscle spasms. Studies of
ketorolac (Toradol) have proved it
to be effective in alleviating bone
pain, with longer action and fewer
side effects than narcotic agents.
cyclobenzaprine (Flexeril),
carisoprodol
(Soma), diazepam (Valium).
Administer analgesics around the
clock for 35 days.
Maintain/monitor IV patientcontrolled analgesia (PCA) using
peripheral, epidural, or intrathecal
routes of administration. Maintain
safe and effective
infusions/equipment.