Case Study of Paranoid Schizophrenia
Case Study of Paranoid Schizophrenia
Case Study of Paranoid Schizophrenia
VALENZUELA CAMPUS
College of Nursing
General Objectives:
General Objective:
This case study aims to gain a comprehensive knowledge about F20.0 Paranoid Schizophrenia
including the practical exercise about the health problem and the practica. experience working
with the patient having the disease in order to provide holistic care to patient.
Specific Objectives
Introduction:
Schizophrenia is a mental disorder characterized by the disturbances in
thoughts, sensory perception and deterioration in psychosocial functioning. It is also
characterized by a weak ego. The common defense mechanisms used by individual are
regression, projection, withdrawal and denial. There are four As to acknowledge in
having schizophrenia, first, the associative looseness, the blunted affect, ambivalence
and the autistic thinking.
Paranoid schizophrenia is the most common type of schizophrenia in most parts
of the world. The clinical picture is dominated by relatively stable, often paranoid,
delusions usually accompanied by hallucinations particular auditory variety, and
perceptual alterations. Disturbances of affect, volition and speech, and catatonic
symptoms are not prominent. Paranoid Schizophrenia is manifested primarily through
impaired thought processes, in which the central focus is on distorted perceptions or
paranoid behavior and thinking. Delusions are in most cases grandiose, persecutory or
both. (WHO 2005)
With paranoid schizophrenia, the ability to think and function in daily life is better
compare with other types of schizophrenia. It may not have as many problems with
memory, concentration or dull emotions. Still, paranoid schizophrenia is a serious,
lifelong condition that can lead to many complications, including suicidal behavior.
Those individuals who diagnosed with paranoid schizophrenia are not especially
prone to violence; often prefer to be alone. Studies show that if people have no record
of criminal violence prior to develop schizophrenia and are not substance abusers, then
they are unlikely to commit crimes after they become ill. Most violent crimes are not
committed by people with paranoid schizophrenia, and most people with schizophrenia
do not commit violent crimes. Substance abuse always increases violent behavior,
whether or not the person has schizophrenia.
If someone with paranoid schizophrenia becomes violent, their violence is most
often directed at family members and takes place at home. These individuals may
spend an extraordinary amount of time thinking about ways to protect themselves from
their persecutors.
In the US paranoid schizophrenia reports issued by Centers for Disease Control
and Prevention (CDC) for 2000 revealed 121,000 diagnoses of paranoid schizophrenia
in non-Federal, short-stay hospitals (73,000 men and 47,000 women). Most individuals
(62,000) were between the ages of 15 and 44; none were under age 15; 37,000 were
between 45 and 64; and 21,000 were 65 or older. According to geographic distribution,
the highest prevalence is in the South and Northeast regions of the US with the lowest
prevalence in the West and Midwest are almost equal. (Medical Disability Advisor,
2010)
The onset of the disorder is usually later than catatonic or disorganized
schizophrenia. Men have earlier onset, and more frequent than women. Women have a
bimodal onset with peaks in their 20s and early 40s. One study demonstrated within
subtype age of institutionalization gender differences only for paranoid schizophrenia
(Salokangas et al., 2003).
The present etiology of the paranoid schizophrenia are the following, genetics it
is known because people believed that mental disorder can be inherit. Other causes are
decreased dopamine, stress, alcohol abuse and substance abuse.
Prognosis of the disease is good when there is no familial history of the disease,
the patient has good social and professional adjustment prior to onset of symptoms, if
the disease come suddenly and the disorder is treated early, quickly, consistently. And
onset symptoms occur at later years of life and there is an absence of symptoms
between psychotic episodes.
Paranoid schizophrenia is usually treated with a combination of therapies,
tailored to the individual's symptoms and needs. Anti-psychotic medications can reduce
hallucinations and disordered thinking, but do not affect the social withdrawal that is
common among those with paranoid schizophrenia. Failure to take medication even
during remission periods can result in a relapse. Psychotherapy is used to address the
emotional and social issues that result from paranoid schizophrenia. Group therapy can
be especially helpful, because it creates opportunities for socialization for individuals
with paranoid schizophrenia.
Nursing History
Patients Profile:
Patients Name:
PATIENT EG
Ward Rm:
Pavillion 3 Female
Age:
51 years old
Sex:
Female
Civil Status:
Single
Birth Place:
Tacloban
Nationality:
Filipino
Religion:
Catholic
Admission Date:
December 3, 2013
Physicians Diagnosis:
Chief Complaint:
According to the informant ( brother ): nanghahabol ng kitchen knife at biglang
nagagalit.
According to the patient: wala po akong sakit
Family History:
Patient denied history of psychiatric illlness.
Brain
The brain is a spongy organ made up of nerve and supportive tissues. It is
located in the head and is protected by a bony covering called the skull. The base, or
lower part, of the brain is connected to the spinal cord. Together, the brain and spinal
cord are known as the central nervous system (CNS). The spinal cord contains nerves
that send information to and from the brain. The CNS works with the peripheral nervous
system (PNS). The PNS is made up of nerves that branch out from the spinal cord to
relay messages from the brain to different parts of the body. Together, the CNS and
PNS allow a person to walk, talk, throw a ball and so on.
breathing
body temperature
blood pressure
heart rate
Cranial nerves emerge from the brainstem. These nerves control facial sensation, eye
movement, hearing, swallowing, taste and speech.
Other important parts of the brain
NEUROTRANSMITTERS
NEUROTRANSMITTERS are the brain chemicals that communicate information
throughout our brain and body. They relay signals between nerve cells, called
neurons. The brain uses neurotransmitters to tell your heart to beat, your lungs to
breathe, and your stomach to digest. They can also affect mood, sleep, concentration,
weight, and can cause adverse symptoms when they are out of balance.
Neurotransmitter levels can be depleted many ways.
Inhibitory Neurotransmitters
SEROTONIN is an inhibitory neurotransmitter which means that it does not
stimulate the brain. Adequate amounts of serotonin are necessary for a stable mood
and to balance any excessive excitatory (stimulating) neurotransmitter firing in the
brain. If you use stimulant medications or caffeine in your daily regimen it can cause
a depletion of serotonin over time. Serotonin also regulates many other processes such
as carbohydrate cravings, sleep cycle, pain control and appropriate digestion. Low
serotonin levels are also associated with decreased immune system function.
GABA is an inhibitory neurotransmitter that is often referred to as natures
VALIUM-like substance. When GABA is out of range (high or low excretion values), it
is likely that an excitatory neurotransmitter is firing too often in the brain. GABA will be
sent out to attempt to balance this stimulating over-firing.
DOPAMINE is a special neurotransmitter because it is considered to be both
excitatory and inhibitory. Dopamine helps with depression as well as focus, which you
will read about in the excitatory section.
Excitatory Neurotransmitters
DOPAMINE is our main focus neurotransmitter. When dopamine is either
elevated or low we can have focus issues such as not remembering where we put our
keys, forgetting what a paragraph said when we just finished reading it or simply
daydreaming and not being able to stay on task. Dopamine is also responsible for our
drive or desire to get things done or motivation. Stimulants such as medications for
ADD/ADHD and caffeine cause dopamine to be pushed into the synapse so that focus
is improved. Unfortunately, stimulating dopamine consistently can cause a depletion of
dopamine over time.
NOREPINEPHRINE is an excitatory neurotransmitter that is responsible for
stimulatory processes in the body. Norepinephrine helps to make epinephrine as well.
This neurotransmitter can cause ANXIETY at elevated excretion levels as well as some
MOOD DAMPENING effects. Low levels of norepinephrine are associated with LOW
ENERGY, DECREASED FOCUS ability and sleep cycle problems.
EPINEPHRINE is an excitatory neurotransmitter that is reflective of stress. This
neurotransmitter will often be elevated when ADHD like symptoms are present. Long
Modifiable
Factors:
Non-Modifiable:
Lifestyle
Alcohol
Smoking
Substance
abuse
Failure in
development or
a subsequent
loss of brain
tissue
Diminished
glucose meta,
and oxygen in
frontal cortical
Decrease brain
volume and
abnormal brain
function in
frontal and
temporal lobe
Transmission of signal
requires a complex
series of biochemical
events
Malfunctioning
of transmission
of electrical
impulses
Actions of:
Dopamine
Serotonin
Norepiephrin
e
Acetylcholine
Glumate
Drug increases
dopaminergic
system activity
Drug blocking
post synaptic
dopamine
receptors
Three separate symptoms
complexes/syndromes:
Hallucinations/delusions
Disorganized thoughts and
behavior
Age
o
o
Male (15-25)
Female (2535)
Gender
Enlarged
ventricles
and
cortical
atrophy
Induced
paranoid
psychotic
symptoms
Reduce
psychotic
symptoms
General Survey:
Patient is an adult female appearing as stated age. Of medium height and built.
She is wearing red dress, fairly groomed. Patient is attentive and cooperative and also
maintains good eye contact. Patient seems depressed with appropriate affect. She talks
spontaneously and responds to question. Patient is oriented to time, place and person.
Patient claims that she is not sick.
HEENT: Normocephalic, symmetric short black hair, no visible scalp/lesions, no
cyanosis
Skin: Brown, dry, visible skin lesions on lower extremities, no cyanosis
Neck: Normal in size, symmetrical no mass, normal muscle development and tone, no
palpable lymph nodes
Lung/Chest: Symmetrical chest expansion, clear breath sounds
Heart: Dynamic precordium, normal rate, regular rhythm, no murmur
Abdomen: Flat, symmetrical, Normo-active bowel sounds, soft, non-tender and no
palpable mass
Extremities: No gross deformities, full and equal pulses. No edema
Day 1
a. Aerobic exercise to stretch the muscles and bones (we named the exercise
Laba-dami Dance).
By: Linton Dela Cruz and Kevin Evangelista.
Evaluation: after performing exercise, the patient feels alive, energetic and
cooperative.
b. Then the Recreational therapy (games prepared are Calamansi relay &
Hep-Hep Hooray).
By: Jessica Garcia and Efren Gannaban
Evaluation: the patient is well cooperative and increased her level of functioning.
c. Proper hygiene (facial wash, tooth brushing, cleaning ears, giving them
powder and lotion, cutting nails, etc.)
Evaluation: patient feels comfortable
d. Nutritional Therapy: they ate and we gave them their prizes after the activities.
Assisted by: Floidas Fernando
Evaluation: the patient is happy and feels satisfied.
Day 2
a. Aerobic exercise to stretch the muscles and bones (we named the exercise
Laba-dami Dance).
By: Linton Dela Cruz and Kevin Evangelista.
Evaluation: after performing exercise, the patient feels alive, energetic and
cooperative.
b. Occupational therapy (our group teach them how to make a salted egg)
Evaluation: the patient is cooperative, maintains the daily living and improved her
work skills.
c. Nutritional Therapy: they ate and we gave them their prizes after the activities.
Assisted by: Floidas Fernando
Evaluation: the patient is happy and feels satisfied
Day 3
a. Aerobic exercise to stretch the muscles and bones (we named the exercise
Laba-dami Dance).
By: Linton Dela Cruz and Kevin Evangelista.
Evaluation: after performing exercise, the patient feels alive, energetic and
cooperative.
b. Occupational therapy (our group teach them how to make a salted egg)
Evaluation: the patient is cooperative, maintains the daily living and improved her
work skills.
c. The Remotivation therapy (our group presented a poem about the
environment).
Evaluation: the patient shows willingness to listen and stimulates interest in the
environment.
d. Nutritional Therapy: they ate and we gave them their prizes after the
activities.
Assisted by: Floidas Fernando
Evaluation: the patient is happy and feels satisfied
Day 4
a. Aerobic exercise to stretch the muscles and bones (we named the
exercise Laba-dami Dance).
By: Linton Dela Cruz and Kevin Evangelista.
Evaluation: after performing exercise, the patient feels alive, energetic and
cooperative.
b. Occupational therapy (our group teach them how to make a salted
egg)
Evaluation: the patient is cooperative, maintains the daily living and improved her
work skills.
c.
Evaluation: the patient shows willingness to listen and she learned a lot of things
especially moral lessons.
d. Nutritional Therapy: they ate and we gave them their prizes after the
activities.
Assisted by: Floidas Fernando
Evaluation: the patient is happy and feels satisfied.
Day 5
a. Aerobic exercise to stretch the muscles and bones (we named the
exercise Laba-dami Dance).
By: Linton Dela Cruz and Kevin Evangelista.
Evaluation: after performing exercise, the patient feels alive, energetic and
cooperative.
b. Occupational therapy (our group teach them how to make a salted
egg)
Evaluation: the patient is cooperative, maintains the daily living and improved her
work skills.
c. Music and Art therapy (we instructed our patient to hear and feel the
background music then we asked them to draw in a sheet of paper
about their feelings or ideas that comes in their mind while hearing
the music)
Evaluation: the patient explores her feelings, reduced anxiety, and developed her
social skills.
d. Nutritional Therapy: they ate and we gave them their prizes after the
activities.
Assisted by: Floidas Fernando
Evaluation: the patient is happy and feels satisfied
Evaluation: the patient is well cooperative and increased her level of functioning.
Drug Study:
Drug/Class
Action
Haloperidol
Blocks
postsynaptic
dopamine
receptors in the
brain
Antipsychoti
c
10-20mg/tab
BID
Biperiden
HCL
AntiCholinergic
2mg
PRNxEPS
Risperidon
e
Antipsychotic
4mg
Synthetic
anticholinergic
drug that blocks
cholinergic
response in the
CNS
Blocks
dopamine and
serotonin
receptors in
the brain,
depresses the
RAS;
anticholinergic,
antihistaminic,
and alphaadrenergic
blocking activity
may contribute
to some of its
therapeutic and
adverse actions.
Contraindic
ated
Adverse
Effect
Intervent
ion
Management of
psychotic
symptoms
Sub-cortical
brain
damage
Short-term
treatment of
hyperactivity
u/c epilepsy,
PUD, allergy
to aspirin
Drowsine
ss, EPS,
urinary
retention,
urticarial
Advised
to avoid
prolonged
sun
exposure
Parkinsonian
syndrome especially
to counteract
muscular rigidity
and tremor;
extrapyramidal
symptoms
Untreated
narrow angle
glaucoma,
intestinal
stenosis or
obstruction,
mega colon,
prostatic
hypertrophy
skin
rashes,
dyskinesi
a,
twitching,
impaired
speech,
fatigue
Use
cautiously
with
cardiovascul
ar disease,
pregnancy,
renal or
hepatic
impairment,
hypotension.
Insomnia
, dry
mouth,
rash, dry
skin,
Indication
Treatment for
Schizophrenia
Advised
on
bladder
emptying
before
giving the
drug
Assess
for Parkin
sonism,
EPS
Assess
for allergy
to
risperidon
e,
lactation,
CV
disease,
pregnanc
y, renal or
hepatic
impairme
nt,
hypotensi
on
Assessment
Subjective:
Lagi akong na
ngangati,
nahawa na ako
sa loob
Objective:
(+) dry skin
(+) itchiness
(+) skin rash
(+) disruption of
skin surface
Nursing
Diagnosis
Planning
Impaired Skin
After 3
Integrity related to
hours of nursing
mechanical trauma
intervention the
as manifested by
patient will
patients report of dry
demonstrate
skin, itchiness, skin
understanding of
rash and disruption
plan to heal and
of skin surface
prevent presence
of current skin
condition.
condition/pathology involved to
assess the causative factors.
2. Kept the area clean and dry to
assess client with correcting
condition.
3. Encouraged the client to
maintain clean hands and
shorts fingernails to reduce
disruption of skin when there
is itching.
4. Maintained strict skin hygiene,
using mild non detergent
soap, drying gently and
thoroughly and lubricating with
lotion as indicated to maintain
skin integrity at optimal level.
5. Suggested to use ice or
calamine lotion to decrease
irritable itching.
6. Emphasized the importance of
adequate nutritional/fluid
intake to maintain good health
and skin turgor.
Evaluation
After 3
hours of nursing
intervention the
patient
demonstrated
the
understanding
of plan to heal
and prevent
presence of
current skin
condition.