Psychiatric Nursing Review
Psychiatric Nursing Review
Psychiatric Nursing Review
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Chapter One
FOUNDATIONS OF PSYCHIATRIC MENTAL HEALTH NURSING
Mental Health
• The WHO defines health as a state of complete physical, mental, and social wellness, not merely the absence of disease or
infirmity.
• Mental health is influenced by individual factors, including biologic makeup, autonomy, and independence, self-esteem,
capacity for growth, vitality, ability to find meaning in life, resilience or hardiness, sense of belonging, reality orientation,
and coping or stress management abilities; by interpersonal factors, including effective communication, helping others,
intimacy, and maintaining a balance of separateness and connectedness; and by social/cultural factors, including sense of
community, access to resources, intolerance of violence, support of diversity among people, mastery of the environment, and
a positive yet realistic view of the world (damn, that was a mouthful!).
Mental Illness
• The APA (2000) defines a mental disorder as “a clinically significant behavioral or psychological syndrome or pattern that
occurs in an individual and that is associated with present distress or disability or with a significantly increased risk of
suffering death, pain, disability, or an important loss of freedom”.
• Deviant behavior does not necessarily indicate a mental disorder.
Self-awareness issues
• Self-awareness is the process by which the nurse gains recognition of his or her own feelings, beliefs, and attitudes.
Chapter Two
NEUROBIOLOGIC THEORIES AND PSYCHOPHARMACOLOGY
Neurotransmitters
• Neurotransmitters are the chemical substances manufactured in the neuron that aid in the transmission of information
throughout the body.
o They either excite or stimulate an action in the cells (excitatory) or inhibit or stop an action (inhibitory).
o After neurotransmitters are released into the synapse (point of contact between the dendrites and the next neuron)
and relay the message to the receptor cells, they are either transported back from the synapse to the axon to be stored
for later use (reuptake) or are metabolized and inactivated by enzymes, primarily monoamine oxidase (MAO).
• Dopamine, a neurotransmitter located primarily in the brain stem. Dopamine is generally excitatory and is synthesized from
tyrosine, a dietary amino acid.
o Antipsychotic medications work by blocking dopamine receptors and reducing dopamine activity.
• Norepinephrine and Epinephrine
o Norepinephrine, the most prevalent neurotransmitter, is located primarily in the brain stem. It plays a role in mood
regulation.
o Epinephrine is also known as noradrenaline and adrenaline. Epinephrine has limited distribution in the brain but
controls the fight-or-flight response in the peripheral nervous system.
• Serotonin
o A neurotransmitter found only in the brain, is derived from tryptophan, a dietary amino acid.
o The function of serotonin is mostly inhibitory, involved in the control of food intake, sleep and wakefulness,
temperature regulation, pain control, sexual behavior, and regulation of emotions.
o Some antidepressants block serotonin reuptake, thus leaving it available longer in the synapse, which results in
improved mood.
• Histamine
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o The role of histamine in mental illness is under investigation.
• Acetylcholine
o Acetylcholine is a neurotransmitter found in the brain, spinal cord, and peripheral nervous system. It can be
excitatory or inhibitory. It is synthesized from dietary choline found in red meat and vegetables and has been found
to affect the sleep-wake cycle and to signal muscles to become active.
o Studies have shown that people with Alzheimer’s disease have decreased acetylcholine secreting neurons.
• Glutamate
o Glutamate is an excitatory amino acid that at high levels can have major neurotoxic effects.
• Gamma-Aminobutyric Acid (GABA)
o GABA is a major inhibitory neurotransmitter in the brain and has been found to modulate other neurotransmitter
systems rather than to provide a direct stimulus.
o Drugs that increase GABA function such as benzodiazepines are used to treat anxiety and to induce sleep.
Psychopharmacology
• Efficacy refers to the maximal therapeutic effect that a drug can achieve.
• Potency describes the amount of the drug needed to achieve that maximum effect; low-potency drugs require higher doses to
achieve efficacy, whereas high-potency drugs achieve efficacy at lower doses.
• Half Life is the time it takes for half of the drug to be removed from the bloodstream. Drugs with shorter half-life may need
to be given three or four times a day, but drugs with a longer half-life may be given once a day.
• The FDA may issue a black-box warning when a drug is found to have serious or life-threatening side effects. This means
that package inserts must have a highlighted box, separate from the text, which contains a warning about the serious side-
effects.
Antipsychotic drugs
• Also known as neuroleptics, are used to treat the symptoms of psychosis, such as the delusions and the hallucinations seen in
schizophrenia, schizoaffective disorder, and the manic phase of bipolar disorder.
• Antipsychotic’s work by blocking receptors of the neurotransmitter, dopamine.
• Dopamine receptors are classified into subcategories (D1, D2, D3, D4, and D5) and D2, D3, and D4 have been associated
with mental illness.
• The typical antipsychotic drugs are potent antagonists (blockers) of D2, D3, and D4. This makes them effective in treating
target symptoms but also produces many extrapyramidal side effects because of the blocking of the D2 receptors.
• Newer, atypical antipsychotic drugs such as clozapine (Clozaril) are relatively weak blockers of D2, which may account for
the lower incidence of extrapyramidal side effects.
• The newer antipsychotics also inhibit the reuptake of serotonin, increasing their effectiveness in treating the depressive
aspects of schizophrenia.
Extrapyramidal Side Effects
• (EPS) are the major side effects of antipsychotic drugs. They include acute dystonia (prolonged involuntary muscular
contractions that may cause twisting of the body parts, repetitive movements, and increased muscular tone),
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pseudoparkinsonism, and akathisia (intense need to move about). Blockage of the D2 receptors in the midbrain region of the
brain stem is responsible for the development of EPS. Included in the EPS are:
o Torticollis: twisted head and neck
o Opisthotonus: tightness of the entire body with head back and an arched neck.
o Oculogyric crisis: eyes rolled back in a locked position.
• Immediate treatment with anticholinergic drugs usually brings rapid relief.
• Pseudoparkinsonism, or drug-induced Parkinsonism if often referred to by the generic label of EPS. Symptoms include a
stiff, stooped posture; mask-like facies; decreased arm swing; a shuffling. festinating gait; drooling; tremor; bradycardia; and
coarse pill rolling movements of the thumb and fingers while at rest.
• Treatment of these symptoms can include adding an anticholinergic agent or amantadine, which is a dopamine agonist that
increases transmission of dopamine blocked by the antipsychotic drug.
Neuroleptic Malignant syndrome
• (NMS) is a potentially fatal idiosyncratic reaction to an antipsychotic. Death rates have been reported at 10% to 20%.
• Symptoms include rigidity, high fever; autonomic instability such as unstable blood pressure, diaphoresis, and pallor;
delirium; and elevated levels of enzymes, particularly creatine and phosphokinase.
• Clients with NMS are confused and often mute; they may fluctuate from agitation to stupor.
• Dehydration, poor nutrition, and concurrent medical illness all increase the risk of NMS.
• Treatment includes immediate discontinuation of the antipsychotic and the institution of supportive medical care to treat
dehydration and hyperthermia.
Tardive Dyskinesia
• (TD) is a syndrome of permanent involuntary movements. This is most commonly caused by the long-term use of
antipsychotic drugs.
• There is no treatment available.
• The symptoms of TD include involuntary movements of the tongue, facial, and neck muscles, upper and lower extremities,
and truncal musculature. Tongue thrusting and protruding, lip smacking, blinking, grimacing, and other excessive
unnecessary facial movements are characteristic.
• One TD has developed, it is irreversible.
Agranulocytosis
• Some antipsychotics produces agranulocytosis. This develops suddenly and is characterized by:
o Fever
o Malaise
o Ulcerative sore throat
o Leucopenia
• The drug must be discontinued immediately if the WBC drops by 50% or to less that 3,000.
Antidepressant drugs
• Although the mechanism of action is not completely understood, antidepressants somehow interact with the two
neurotransmitters, norepinephrine and serotonin.
• Antidepressants are divided into four groups:
o Tricyclic and the related cyclic antidepressants
o Selective serotonin reuptake inhibitors (SSRIs)
o MAO inhibitors (MAOIs)
o Other antidepressants such as venlafaxine (Effexor), bupropion (Wellbutrin), duloxetine (Cymbalta), trazodone
(Desyrel), and nefazodone (Serzone).
• MAOIs have a low incidence of sedation and anticholinergic effects, they must be used with extreme caution for several
reasons:
o A life-threatening side effect, hypertensive crisis, may occur if the client ingests food containing tyramine (an amino
acid) while taking MAOIs.
Mature or aged cheeses
Aged meats (sausage, pepperoni)
Tofu
ALL tap beers and microbrewery beer.
Sauerkraut, soy sauce, or soybean condiments
Yogurt, sour cream, peanuts, MSG
o MAOIs cannot be given in combination with other MAOIs, tricyclic antidepressants, Demerol, CNS depressants,
and hypertensives, or general anesthetics.
o MAOIs are potentially lethal in overdose and pose a potential risk for clients with depression who may be
considering suicide.
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• SSRIs, venlafaxine, nefazodone, and bupropion are often better choices for those who are potentially suicidal or highly
impulsive because they carry no risk of lethal overdose in contrast to the cyclic compounds and the MAOIs. However, SSRIs
are only effective for mild to moderate depression.
• The major actions of antidepressants are with the monoamine neurotransmitter systems in the brain, particularly
norepinephrine and serotonin.
o Norepinephrine, serotonin, and dopamine are removed from the synapses after release by reuptake into presynaptic
neurons. After reuptake, these three neurotransmitters are reloaded for subsequent release or metabolized by the
enzyme MAO.
o The SSRIs block the reuptake of serotonin; the cyclic antidepressants and venlafaxine block the reuptake of
norepinephrine primarily and block serotonin to some degree; and the MAOIs interfere with enzyme metabolism.
Stimulants
• Today, the primary use of stimulants is for ADHD in children and adolescents, residual attention deficit disorder in adults,
and narcolepsy.
• Stimulants are often termed indirectly acting amines because they act by causing release of the neurotransmitters
(norepinephrine, dopamine, and serotonin) from presynaptic nerve terminals as opposed to having direct agonist effects on
the postsynaptic receptors. They also block the reuptake of these neurotransmitters.
• By blocking the reuptake of these neurotransmitters into neurons, they leave more of the neurotransmitter in the synapse to
help convey electrical impulses in the brain.
Cultural considerations
• I’m not going to go much into this. Just know that clients from various cultures may metabolize medication at different rates
and therefore require alterations in standard dosages.
Crisis Intervention
• Maturational crises, sometimes called developmental crises, are predictable events in the normal course of a life, such as
leaving home for the first time, getting married, having children, etc.
• Situational crises are unanticipated or sudden events that threaten an individuals integrity; such as a death of a loved one and
loss of a job.
• Adventitious crises, sometimes called social crises, include natural disasters like floods, earthquakes, or hurricanes; war,
terrorist attacks; riots; and violent crimes such as rape or murder.
Anxiety:
• Increase or change in behavior. Can be anything different from usual behavior (excitement, pacing).
• Nursing interventions:
o Ask “What’s going on?”
o Give supportive care and let the patient know that you’re there.
Defensive:
• Loss of rationality.
• Nursing interventions:
o Direct approach to setting limits.
o Take away privileges.
o Give the patient some control and choices.
Acting out person:
• Loss of rational control.
• Nursing interventions:
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o Everything Cathy showed us on non-violent physical crisis intervention
Tension-Reduction:
• Subsiding of energy.
• Nursing interventions:
o Establish therapeutic rapport
o Prime time to talk and teach about preventions of behavior.
What if the patient simply refuses?
• Set limits!
• Make the limits reasonable and enforceable.
Releasing… Venting… Mad as heck!
• Allow the patient to do this!
• Just stay calm as a nurse
• While they’re venting, they’re also releasing. This is a good thing.
Intimidation:
• This is NOT A GOOD THING.
• What if the patient tells you…?
o I know what car you drive.
o I know your last name.
o I know you have 2 dogs and I’m going to kill them.
• Nursing interventions:
o Get a witness! Do not be by yourself with this patient!
Paraverbal communication
• 55% nonverbal
• 7% verbal
• 38% paraverbal it’s not what you say; it’s how you say it!
• TVC (total voice control)
o Tone
o Volume
o Cadence
Incidence
• Major depression occurs at least twice as often in women
• Single and divorced people have the highest rates of depression
Treatments
• Psychotherapy (groups, counselor)
• Psychopharmacology (Meds)
• ECT
Electroconvulsive therapy
• The biggest concern is memory loss.
• Patient is pre-medicated, much like a pre-op patient
• Elders are treated for depression with ECT more frequently than younger persons.
o Elder persons have increased intolerance of side effects of antidepressants
o ECT produces a more rapid response
Suicidal Ideation
• Safety is primary concern
• Watch for overt cues of suicide (Obvious) active
• Covert cues are more subtle—passive
• People who suddenly are happier are of great concern; may have made the suicidal plan are content with their decision.
• People whose meds are finally working—have enough energy to carry out the act
Client’s Affect
• Compare verbal with non-verbal behaviors—do they match up?
• Asocial: Withdrawal from family and friends
• Anhedonic: Lose sense of pleasure
• When confronting these client’s about their behavior, use “I” statements
o “I really wish you’d join the group”
Judgment
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• Feel overwhelmed with normal activities
• Difficulty with task completion
• Always exhausted
Self Concept
• Ruminate: Worry to excess.
• Lack energy for normal activities (always tired)
Interventions
• Assess safety for client (PRIORITY!)
• Perform suicide lethality assessment
• Orient client to new surroundings (they need structure)
• Offer explanations of unit routine (again, need structure)
• Start to promote a therapeutic relationship; schedule short interaction times.
Drug treatment
• Lithium
o Lithium is not metabolized; rather, it is reabsorbed by the proximal tubule and excreted in the urine.
o Thought to work in the synapse to increase destruction of dopamine and norepinephrine; decreases sensitivity to
postsynaptic receptors (Basically- when a person is in a manic phase, they are synapsing super fast. Lithium helps
slow this synapsing down).
o Onset of action is 5-14 days; other drugs must be used during the acute phases to reduce symptoms of mania or
depression.
o Maintenance lithium level is 0.5-1.0 mEq/L.
3 is toxic!
o Lithium is a salt contained in the human body. It not only competes for salt receptor sites but also affects calcium,
potassium, and magnesium ions as well as glucose metabolism.
MUST complete an electrolyte blood panel (focus on Chloride).
o Having too much salt in the diet can cause the lithium level to be too low.
o Not having enough dietary salt can cause the lithium levels to be too high.
o Persistent thirst and diluted urine can indicate the need to call the MD; lithium dosage may need to be reduced.
• Anticonvulsant drugs: mechanism is unclear, but they raise the brains threshold for dealing with stimulation; this prevents the
person from being bombarded with external and internal stimuli.
o Tegretol
Huge concern about agranulocytosis (a decrease in WBC).
Need serum levels monitored 12 hours after last dose.
o Depakote
Need to monitor serum level, CBC with platelets, liver function including ammonia level (ammonia is a by-
product of liver metabolism)
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o Klonopin
Anticonvulsant and benzodiazepine
Drug dependence can occur
Monitor CBC, liver function
Withdrawal drug slowly to prevent GI issues
Cannot be used alone to manage bipolar; must be used in conjunction with lithium or another mood
stabilizer.
Suicide
• 4 out of 5 who actually commit suicide have made at least one prior attempt
• In a majority of cases, there are clear indicators hat the person was very troubled.
• Few than 15% of suicide victims leave suicide notes
• The suicide risk is greatest in the 90 days following a major depressive episode.
• “survivor guilt” happens when 1 or more family members feel guilty that they are still living
• “Separation anxiety” may cause they surviving to “join the beloved deceased”
• Make the patient sign a “contract for life”
• Crisis intervention—may need 1:1 care. The client is no more than 2-3 feet away from a staff member at any time, including
going to the bathroom.
Incidence
• Most common emotional disorder in the U.S.
• Prevalent in women; age <45
Physiologic responses
• Flight or fight responses
• Sympathetic fibers increase the vital signs
• Adrenal glands release adrenalin which causes the body to:
o Take in more oxygen
o Dilate the pupils (brings more light into eyes; better vision)
o Increase the arterial blood pressure and heart rate
o Constrict peripheral vessels (makes skin cool and pale)
o Increase glycogenolysis to free glucose for fuel (glycogen is being broken down in the liver)
o Shunt blood from GI and reproductive organs
Psychological response
• Difficulty with logical thought
• Increased agitation with motor activity
• Increased vital signs
• Client will try to change the feelings of discomfort by:
o Changing behavior by adaptation
o Changing behavior with defense mechanisms
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Anxiety disorders
• Panic disorder
• Phobic disorder
• Agoraphobia
• Obsessive-compulsive
• PTSD
• Generalized anxiety
• Anxiety related to medical conditions
• Substance-induced anxiety disorder
Levels of anxiety
• Mild:
o Psychological: Wide perceptional field, sharpened senses, increased motivation, effective problem solving,
increased learning ability, irritability.
o Physiologic: Restlessness, fidgeting, “butterflies”, difficulty sleeping, hypersensitivity to noise.
• Moderate:
o Psychological: perceptual field narrowed to immediate task, selectively attentive, cannot connect thoughts or events
independently, increased use of automatisms
o Physiologic: Muscle tension, diaphoresis, pounding pulse, HA, dry mouth, high voice pitch, faster rate of speech, GI
upset, frequent urination
• Severe:
o Psychological: Perceptual field narrowed to one detail or scattered details; cannot complete tasks; cannot solve
problems or learn effectively; behavior geared toward anxiety relief and is usually ineffective; doesn’t respond to
redirection; feels awe, dread, or horror; cries; ritualistic behavior.
o Physiologic: Severe HA, N/V, diarrhea, rigid stance, vertigo, pale, tachycardia, chest pain.
• Panic:
o Psychological: Perceptual field reduced to focus on self; cannot process any environmental stimuli; distorted
perceptions; loss of rational thought; doesn’t recognize potential danger; can’t communicate verbally; possible
delusions or hallucinations; may be suicidal.
o Physiologic: May bolt and run OR totally immobile and mute; dilated pupils, increased blood pressure and pulse;
flight, fright, or freeze.
Panic disorders
• An episode lasting 15-30 minutes in which a client experiences rapid, intense, escalating anxiety; great emotional discomfort;
and physiologic discomfort.
• Defined as recurrent, unexpected panic attacks followed by a month of persistent concern or worry about having another
attack.
• 75% with panic disorder have spontaneous attacks with no triggers
• Others have attacks stimulated by phobias or chemical changes within the body.
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Treatment
• Psychotherapy
o Positive reframing
o Assertiveness training
• Psychopharmacology
o SSRIs
o Anxiolytics
o Antidepressants
o MAOIs
Phobias
• An illogical, intense, persistent fear of a specific object or social situation that causes extreme distress and interferes with
having a normal life.
• Treatment for phobias:
o Psychopharmacology
Anxiolytics
Benzodiazepines
SSRIs
Beta Blockers
o Psychotherapy
Behavioral therapy
Systemic desensitization
“Flooding” Getting rid of fear all at one time
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Only 46% of parents sought help for their children. KIDS NEED HELP.
• Symptoms of PTSD occur 3 months or more after the trauma.
• Some more signs of PTSD:
o Have issues with authority figures
o Their first emotions are anger, rage, and guilt
o Their guilt comes out as anger (violent behavior)
o Isolate themselves
o Cry
o Don’t want to talk about it
o Drug and alcohol abuse
o Nightmares
o Manifests in physiological symptoms (HA, GI distress)
o Irritable
o Insomnia
• Nursing interventions:
o Have specific staff members assigned to client to facilitate building trust
o Consistency is the key
o Be non-judgmental; encourage client to talk
o Help them acknowledge where grief is coming from
o Involve family
o Give positive feedback
• Goals for PTSD:
o Short term: Safety, decrease insomnia, identify source, grieve!
o Long term: Accept the fact that the experience happened and live healthy.
Substance abuse
• Overdose of alcohol:
o Alcohol is a depressant; decreased respirations and blood pressure, vomiting may cause aspiration.
• Overdose of benzodiazepines require a gastric lavage including instillation of activated charcoal.
• Stimulants
o Cocaine, amphetamines, and Ritalin
o Increases HR and BP; decreases cardiac output and oxygen
o Cocaine specifically causes MI’s
Withdrawal
• Two purposes:
o Safe withdrawal with medication
Suppress symptoms of abstinence
Around the clock schedule and PRN
Never, ever go cold turkey.
o Prevent relapse
May need to go to AA for rest of life.
COGNITIVE DISORDERS
Delirium
• Disturbance of consciousness accompanied by change in cognition; disoriented
o Alert and oriented to person only
o Typically have problems recalling on memory and time.
• Develops over a short period of time
• Easily distracted
• Difficulty concentrating
• Illusions, hallucinations
• Onset is rapid
• Brief duration
• Level of consciousness is impaired
• Slurred speech
• Anxious mood
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Causes of Delirium
• Metabolic
• Infection—UTI
• Low sodium
o Normal is 135-145 mEq/L
o Always check electrolytes!
• Drug related
o Or, withdrawal from drugs and alcohol
o Sedatives and benzodiazepines cause confusion
• Effects of anesthesia
• Adequate nutrition
o Often forget to eat; needs nutritional supplements
• Return to optimal level of functioning
• A goal needs a timeline to make it measurable!
DEMENTIA
Dementia
• More progressive, gradual, and permanent
• Involves multiple cognitive deficits
o Primarily memory impairment
• Involves at least one of the following:
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o Asphasia (deterioration of language function)
o Apraxia (impaired ability to execute motor functions)
o Agnosia (inability to name or recognize objects)
o Disturbance in executive functioning (ability to think abstractly and to plan, initiate, sequence, monitor, and stop
complex behavior)
• May also present:
o Echolalia (echoing what is heard)
o Palilalia (repeating words or sounds over and over)
Causes of Dementia
• Decreased metabolic activity
• Genetic component
• Infection
• Alzheimer’s disease (#1)
• Creutzfeld-Jacob disease (CNS disorder; develops at 40-60 years. Causes by infectious particle that is resistant to boiling)
• Parkinson’s disease
• Huntington’s disease (inherited gene; brain atrophy, demyelination, and enlargement of the brain ventricles. Begins in late
30’s)
• Vascular Dementia (#2)
o Symptoms similar to Alzheimer’s, but more abrupt, followed by rapid changes in functioning; a plateau; more
abrupt changes, another plateau, and so on.
o Caused by decreased blood supply to the brain.
Culture
• Native Americans and Eastern countries hold elders in a position of authority, respect, power, and decision making for
family; this does not change despite memory loss or confusion.
• May feel they are being disrespectful and reluctant to make decisions or plans for elders with dementia.
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Nursing process: Assessment
• History
o Remember, interview family
• Motor behavior and general appearance
o Display aphasia
o Conversation repetitive
o Apraxia (such as combing hair)
o Gait disturbance
o Uninhibited behavior; never have displayed these behaviors before.
• Mood and Affect
o Grieve at first
o Emotional outbursts are common
o Pattern of withdrawal; lethargic, apathetic, look dazed and listless.
• Thought process and content
o Executive functioning impaired
o Have to stop working
o Client may accuse others of stealing lost objects
• Sensorium and Intellectual Processes
o First affects recent and immediate memory, eventually impairs the ability to recognize family members and oneself.
o Confabulation: clients make up answers to fill in memory gaps; often inappropriate words or fabricated ideas
(SCREW YOU, ASSHOLE).
o Visual hallucinations are common.
• Judgment and insight
o Underestimate risk
• Self concept
o Initially grieve, and then slowly lose sense of self.
• Roles and Relationships
• Physiologic and self-care considerations
o Altered sleep-wake cycle
o Some clients ignore internal cues such as hunger or thirst
o Neglect bathing and grooming; become incontinent.
SCHIZOPHRENIA
Types of schizophrenia
• Paranoid schizophrenia
o Suspiciousness
o Hostility
o Delusions
o Auditory hallucinations
o Anxiety and anger
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o Aloofness
o Persecutory schemes
o Violence
• Disorganized schizophrenia
o Extreme social withdrawal
o Disorganized speech or behavior
o Flat or inappropriate affect
o Silliness unrelated to speech
o Stereotyped behaviors
o Grimacing mannerisms
o Inability to perform activities of daily living
• Catatonic schizophrenia
o Significant psychomotor disturbances
o Immobility
o Stupor
o Waxy flexibility
o Excessive purposeless motor activity
o Echolalia
o Automatic obedience
o Stereotyped or repetitive behavior
• Undifferentiated schizophrenia
o Undifferentiated schizophrenia does not meet the criteria for paranoid, disorganized, or catatonic schizophrenia
o Delusions and hallucinations
o Disorganized speech
o Disorganized or catatonic behavior
o Flat affect
o Social withdrawal
• Residual schizophrenia
o Diagnosed as schizophrenic in the past
o Time limited between attacks but may last for many years
o The client exhibits considerable social isolation and withdrawal and impaired role functioning
Interventions
• Assess the client’s physical needs
• Set limits on the client’s behaviors when it interferes with others and becomes disruptive
• Maintain a safe environment
• Initiate one-on-one interaction and progress to small groups as tolerated
o Although, reintegrating the client into the milieu as soon as possible is essential
• Spend time with the client even if client is unable to respond
• Monitor for altered thought processes
• Maintain ego boundaries and avoid touching the client
o Touching others without warning or invitation
o Intruding in others’ living spaces
o Talking to or caressing inanimate objects
o Undressing, masturbating, or urinating in public
• Limit the time of interaction with the client
o Initially, the client may only tolerate 5-10 minutes of contact at one time.
• Avoid an overly-warm approach; a neutral approach is less threatening
• Do not make promises to the client that cannot be kept
• Establish daily routines
• Assist the client to improve grooming and to accept responsibility for self-care
• Sit with the client in silence if necessary
• Provide short, brief and frequent contact with the client
• Tell the client when you are leaving
• Tell the client when you do not understand
• Do not “go along” with the clients delusions or hallucinations
• Provide simple concrete activities such as puzzles or word games
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• Reorient the client as necessary
• Help the client establish what is real and unreal
• Stay with the client if he is frightened
• Speak to the client in a simple direct and concise manner
• Reassure the client that the environment is safe
• Remove the client from group situations if the client’s behavior is too bizarre, disturbing, or dangerous to others
o Reassure others that the client’s inappropriate behaviors or comments are not his fault (without violating
confidentiality).
• Set realistic goals
• Initially do not offer choices to the client, and gradually assist the client in making own decisions
• Use canned or packaged food, especially with the paranoid schizophrenic client
• Provide a radio or tape player at night for insomnia
• Explain to the client everything that is being done
• Set limits on the client behavior if the client is unable to do so
• Decrease excessive stimuli in the environment
• Monitor for suicide risk
• Assist the client to use alternative means to express feelings through must or art therapy or writing.
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Abnormal motor behaviors
• Akathisia: Displaying motor restlessness and muscular quivering; the client is unable to sit or lie quietly
• Echopraxia: Repeating the movements of another person
• Waxy flexibility: having one’s arms or legs placed in a certain position and holding that same position for hours
• Dyskinesia: Impairment of the power of voluntary movements
Psychiatric disorders are not diagnosed as easily in children as they are in adults.
• Children lack the abstract cognitive abilities and verbal skills to describe what is happening.
Mental retardation
• Mild retardations: IQ 50-70
• Moderate retardation: IQ 35-50
• Severe retardation: IQ 20-35
• Profound retardation: IQ less than 20.
Adolescent depression
• Some issues are due to background and family issues
• Transition into adulthood often very difficult
• Depression is almost always due to a combination of factors
• Boys are more successful in committing suicide; more violent in attempts
o Acetaminophen affects liver
o Ibuprophen affects kidneys
• Presents as “classic” symptoms in girls
• In boys, depression is more likely to be “acted out” with aggressive behavior such as risk taking, substance abuse,
confrontations with authority.
o Drinking in teenage years (ages 15-17) stops emotional growth. Kids that grow into adults are stuck in this stage
(Identity vs. Role confusion). They learn that drinking is the way to cope. This is not awesome.
• First major episode are during adolescent years; often between the ages of 15-19
• Manic depression
o Teens may be sad and gloomy one day and excited and elevated the next
o Mood stabilizers are important in decreasing mood swings
Lithium (check blood levels!)
Depakote
Tegretol
Neurontin
• In depression, one of the first cues is a large drop in school performance
• Other symptoms disguised:
o Drug/alcohol abuse
o Lack of concentration
o Restlessness or hyperactivity
o Anti-social behavior (conduct disorder)
• Extreme fatigue, sleep all the time but are not rested
• Suicide warning signs…
o Constant insomnia; may be on computer at all hours of the night
o Changes in behavior
o Dropping grades—again, school is a huge issue
• Interventions for suicide
o High risk teens make their decisions after a “disaster” has occurred: break-ups, academic failure, fight with parents,
or run-in with authority
o Alcohol is involved in ½ of all suicides; seriously impairs judgement
• Suicide is not chosen; it happens when pain exceeds resources for pain
• Talk to your kids!
o The best place is in the car when they’re trapped, haha.
Start with the basics; “How are you doing?”
Then, praise
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Then get down and dirty to the real subject
Childhood Schizophrenia
• Group of disorders of thought processes characterized by gradual disintegration of mental function
• Occurs in adolescents or as young adults
• Suicide is the #1 cause of death in young people with schizophrenia
• Treatment and prognosis
o Lifetime of therapy and family support
o Medications
o Struggle for family to stay involved
Often rejected or just can’t take anymore disruption in their lives.
Obsessive-Compulsion disorder
• Symptoms often begin slowly and gradually during their childhood or teenage years and increase in severity as time goes on.
• Though a chronic disease, there will be periods of reduced symptoms followed by “flare-ups”, often stressful times in
person’s life.
• Relief is only temporary; usually both obsessions and compulsions occur together
• Recognize thoughts or behaviors are irrational; but are compelled to continue them “against their will”.
• Treatment:
o Exposure and response prevention
o SSRIs help reduce symptoms of OCD—monitor for side effects
• Compulsions
o Washing, cleaning, constant checking, mental counting rituals
o Touching, ordering, rearranging
o Asking for reassurance or confessing
o Masturbation—especially seen in children who haven’t yet discovered this is socially unacceptable behavior
Autistic disorder
• Most prevalent in boys; identified no later than 3-years of age
• Child has little eye contact, few facial expression, doesn’t use gestures to communicate
• Does not relate to parents or peers, lacks spontaneous enjoyment, apparent absence of mood and emotional affect, can not be
engaged in play or make believe
• Repetitive motor behaviors such as hand-flapping, body twisting, or head banging
• May improve as child acquires language skills
• Short term impatient therapy is used when behaviors such as head banging or tantrums are out of control
o Haldol or Risperadol may be effective (prn, of course)
• Goals of treatment:
o Reduce behavioral symptoms
o Promotes learning and development
o Language skills development
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Manage the environment
Minimal noise and distraction
Face the teacher in the front row and away from window or door
o Ineffective family coping
Will complete tasks
Face the child on his level and use good eye contact
Give the child frequent breaks
Routines are important; child with ADHD do not adjust to changes readily
o Parental support
Listen to parent’s feelings
Because these children often are not diagnosed until the 2nd or 3rd grade, they may have missed much basic
learning for reading and math. Parents should know that it takes time for them to catch up to other children
the same age.
o Evaluation
Medications are often in decreasing hyperactivity and impulsivity relatively quickly.
Improved sociability, peer relations, and academic achievement happen more slowly.
Conduct disorder
• Characterized by persistent antisocial behavior in children and adolescents that significantly impair their ability to function in
social, academic, or occupational area.
o Symptoms are clustered into 4 areas
Aggression to people and animals
Destruction to property
Deceitfulness and theft
Serious violation of rules and the law
o More symptoms
Decreased self-esteem
Poor frustration tolerance
Tempter often out of control
Early onset of sexual behavior, alcohol and substance abuse, smoking, risky behavior
Anti-social
• Types of conduct disorder
o Classified by age of onset
Adolescent-onset type is defined by no behaviors of conduct disorder until after 10 years of age.
Least likely to be aggressive
Have more normal peer relationships
Less likely to have persistent conduct disorder or antisocial personality disorder as adults
Childhood-onset type involves symptoms before 10 years of age
Physically aggressive
Disturbed peer relationships
More likely to have persistent conduct disorder and to develop antisocial personality disorder as
adults
o Can be classified as:
Mild: few conduct problems causing minor harm to others
Lying, truancy, staying out late without permission
Moderate: Number of conduct problems increase as does the amount of harm to others.
Vandalism and theft
Severe: Many conduct problems that cause considerable harm to others.
Forced sex, cruelty to animals, weapons, burglary, robbery.
• Treatment of conduct disorder
o MUST BE GEARED TOWARD DEVELOPMENTAL AGE
o School aged:
Child, family, and school environment are the focus of treatment
Family therapy is essential
o Adolescents
Rely less on their parents, so treatment is based on individual therapy.
Conflict resolution, anger management, social skills
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Try to keep the adolescent in his environment (home)
o Medications have little effect
Antipsychotics for clients who present a clear danger to others
Mood stabilizers for clients with labile moods
• Cultural considerations
o Be careful of diagnosis of Conduct disorder, must know history and circumstances of each child.
High areas of crime rates
Could be a matter of survival
• Nursing process
o Risk for Other-directed violence
The client will not hurt others or damage property
SET LIMITS
Inform the client of the rule or limit
Explain the consequences if broken
State expected behavior
Behavioral contract
Time out; not a punishment—a place to regain self control
Give client a schedule of daily activities
o Noncompliance
The client will participate in treatment
More likely to participate in treatment and daily routines if they have input concerning the
schedule
o Ineffective coping
The client will learn effective problem-solving and coping skills
Help identify the problem and to solve problems effectively.
o Impaired social interaction
The client will use age-appropriate and acceptable behaviors when interacting with others.
Teach social skills
Discuss the news, sports, or other topics as the client may not know how to have a normal conversation.
o Chronic low self-esteem
The client will verbalize positive, age-appropriate statements about self
TIC disorders
• Sudden, rapid, recurrent, non-rhythmic motor movement or vocalization
• Stress and fatigue exacerbates tics
• Treatment: Risperadol and Zyprexia
• Complex vocal tics
o Coprolalia: Use of socially unacceptable words, often obscene
o Palilalia: Repeating own sounds or words
o Echolalia: Repeating the last heard sound, word, or phrase
Tourette’s syndrome
• Multiple motor tics and one or more vocal tics
• May occur many times a day for over a year
• Usually identified by 7 years of age
Elimination disorders
• Encopresis: repeated passage of feces into inappropriate places such as clothing or floor by a child who is at least 4 years of
age either chronically or developmentally. Often involuntary, but can be intentional (oppositional defiant disorder or conduct
disorder). Associated with constipation that occurs for psychological, not medical reasons.
• Enuresis: Repeated voiding of urine during the day or night into clothing or bed by a child at least 5 years of age.
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• Treated with imipramine (Tofranil), an antidepressant with a side effect of urinary retention.
o Was once treated with vasopressin which decreases circulatory volume.
EATING DISORDERS
The distinguishing factor of anorexia includes an earlier age of onset and below-normal body weight; the person fails to recognize the
eating behavior as a problem. Clients with bulimia have a latter age at onset and a near-normal body weight. They usually are ashamed
and embarrassed by the eating disorder.
Eating disorders appear to be equally common among Hispanic and white women and less common among African American and
Asian women.
Anorexia Nervosa
• A life-threatening eating disorder characterized by the client’s refusal or inability to maintain a minimally normal body
weight, intense fear of gaining weight or becoming fat, significantly disturbed perception of the shape or size of the body,
and steadfast inability or refusal to acknowledge the seriousness of the problem or even that one exists.
• Has experienced amenorrhea for at least 3 consecutive cycles
• Complaints of constipations and abdominal pain
• Cold intolerance
• Hypotension, hypothermia, bradycardia
o Intravascular volume is decreased; less blood to pump through heart, also due to excessive exercise
• Elevated BUN
o Normal levels: 10-20 mg/dl
o Urea is formed in the liver and is the end product of protein metabolism.
o In anorexia, the body has already used fat for energy; it is now breaking down muscles for energy—the reason for
the elevated BUN
• Decreased albumin
o Normal levels: 3.5-5 g/dl
o Measures amount of protein in the body; albumin is a protein formed in the liver.
o Albumin tests are a great indicator of nutritional status
• Leukopenia and mild anemia
o Not enough food and nutrients to replenish cells
• Has a preoccupation with food and food-related activities
• Can be divided into 2 subgroups:
o Restricting subtype: lose weight primarily through dieting, fasting, or excessively exercising.
o Binge eating and purging subtype: engage regularly in binge eating followed by purging.
• Engage in unusual or ritualistic food behaviors
o Refusing to eat around others
o Cutting food into minute pieces
o Not allowing the food they eat to touch their lips
• Excessive exercise is common
• Diagnosed between 14 and 18 years of age
• Pleased with their ability to control their weight and may express this.
• As the illness progresses, depression and lability in mood become more apparent
• Isolate themselves
• Believe peers are jealous of their weight loss and believe family and health care professionals are trying to make them “fat
and ugly”.
• Clients who use laxatives are at a greater risk for medical complications.
• Autonomy may be difficult in families that are overprotective or in with enmeshment (lack of clear boundaries) exists. By
losing weight, these clients have some control in their lives.
• Have body image disturbance
• Can be very difficult to treat because they are often resistant, appear uninterested, and deny their problems.
• Treatment:
o Focusing on weight restoration
o Nutritional rehabilitation
o Rehydration
o Correction of electrolyte imbalances
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o Severely malnourished individuals may require TPN, tube feedings, or hyperalimentation to receive adequate
nutritional intake.
o Access to the bathroom is supervised to prevent purging as clients begin to eat more food.
o Weight gain and adequate food intake are most often the criteria for determining the effectiveness of treatment.
o Amitriptyline (Elavil) and the antihistamine cyproheptadine (Periactin) in high doses (up to 28mg/d) can promote
weight gain in inpatients.
o Olanzapine (Zyprexa) has been used with success because of both its antipsychotic effect (on bizarre body image
distortions) and associated weight gain.
o Fluoxetine (Prozac) has shown some effectiveness in preventing relapse in clients whose weight has been partially
or completely restored; close monitoring is needed because weight loss can be a side effect.
• Family members often describe clients with anorexia as perfectionists with above average intelligence, dependable, eager to
please, and seeking approval before their condition began.
• Clients with anorexia appear slow, lethargic, and fatigued; they may appear emaciated, depending on the amount of weight
loss. May be slow to respond and have difficulty deciding what to say.
• Reluctant to answer questions fully because they do not want to acknowledge any problem.
• Often wear loose clothing in layers
• Seldom smile, laugh, or enjoy any attempts at humor
Bulimia Nervosa
• Characterized by recurrent episodes (at least twice a week for 3 months) of binge eating followed by inappropriate measures
to avoid weight gain such as purging (vomiting, laxatives, diuretics, enemas, or emetics), fasting, or excessively exercising.
• Engaging in binge eating secretly
• Binging or purging episodes are often precipitated by strong emotions and followed by guilt, remorse, shame, or self-
contempt.
• Recurrent vomiting destroys tooth enamel, has dental caries and ragged or chipped teeth. Dentists are often the first health
care professionals to recognize this.
• Bulimia is typically diagnosed at 18 or 19.
• Clients with bulimia are aware that their eating behavior is pathologic and go great lengths to hide it from others.
• Clients with a co-morbid personality disorder tend to have poorer outcomes than those without.
• Most are treated on an outpatient basis
• Antidepressants are more effective than the placebos in reducing binge eating
• Clients are often focused on pleasing others and have a history of impulsive behavior such as substance abuse and shoplifting
as well as anxiety, depression, and personality disorders.
• May be underweight, overweight, but are generally close to expected body weight for age and size
• Appear open and willing to talk; initially pleasant and cheerful as though nothing is wrong
Nursing outcomes/interventions
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o Client is weighed daily on awakening and after they have emptied their bladder. Have the client wear a hospital
gown each time they are weighed; they may attempt to place objects in their clothing to give the appearance of
weight gain.
o In bulimia, the clients should sit at a table in a kitchen or dining room.
o Write out a grocery list, it is easier to follow a nutritious eating plan
Ineffective coping
• The client will eliminate use of compensatory behaviors such as excessive exercise and use of laxatives and diuretics
• The client will demonstrate coping mechanisms not related to food
• The client will verbalize feelings of guilt, anger, anxiety, or an excessive need for control
o Help the client recognize emotions such as anxiety or guilt by asking them to describe what they are feeling; allow
adequate time for response. Do not ask “are you anxious? Sad?” because the client may quickly agree rather than
struggle for an answer
o Encourage self-monitoring; a behavior-cognitive approach
SOMATOFORM DISORDERS
Somatization: The transference of mental experiences and states into bodily symptoms.
Somatoform disorders: Characterized as the presence of physical symptoms that suggest a medical condition without demonstrable
organic basis to account fully for them. The three central features of somatoform disorders are as follows:
• Physical complaints suggest major medical illness but have no demonstrable organic basis.
• Psychological factors and conflicts seem important in initiating, exacerbating, and maintaining the symptoms.
• Symptoms or magnified health concerns are not under the client’s conscious control.
Related disorders:
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• Malingering: The intentional production of false or grossly exaggerated physical or psychological symptoms; it is motivated
by external incentives such as avoiding work, evading criminal prosecution, obtaining financial compensation, or obtaining
drugs. Their purpose is some external incentive or outcome that they view as important and results directly from their illness.
People who malinger can stop the physical symptoms as soon as they have gained what they wanted.
• Factitious disorder: This is also known as Munchausen syndrome. Occurs when a person intentionally produces or feigns
physical or psychological symptoms solely to gain attention.
o Munchausen syndrome by proxy occurs when a person inflicts illness or injury to someone else to gain the attention
of emergency medical personnel or to be a “hero” for saving the victim. This occurs most often in people who are in
or familiar with medical professions, such as nurses, physicians, medical technicians, or hospital volunteers.
• Primary gain: Direct external benefits that being sick provides, such as relief of anxiety, conflict, or distress.
• Secondary gains: Internal or personal benefits received from others because one is sick, such as attention from family
members and comfort measures (being brought tea, receiving a back rub).
Treatment:
• Treatment focuses on managing symptoms and improving quality of life.
• A trusting relationship helps to ensure that client’s stay with and receive care from one provider instead of “doctor shopping.”
• SSRIs are commonly used for depression that may accompany somatoform disorders.
Assessment
• The nurse must investigate physical health status thoroughly to ensure there is no underlying pathology requiring treatment. It
is important not to dismiss all future complaints because at any time the client could develop a physical condition that would
require medical attention.
• In many cases, the client’s appearance brightens and they look much better as the assessment interview begins because they
have the nurse’s undivided attention.
• Client’s often have sleep pattern disturbances, lack basic nutrition, and get no exercise.
Nursing diagnoses
• Ineffective coping
o The client will identify the relationship between stress and physical symptoms.
Emotion-focused coping strategies help the clients relax and reduce feelings of stress. This includes
progressive relaxation, deep breathing, guided imagery, and distractions such as music.
Problem-focused coping strategies help to resolve or change a client’s behavior or situation or to manage
life stressors. This includes learning problem solving methods.
The nurse should help the client role play the above situations.
• Ineffective denial
o The client will verbally express emotional feelings
The nurse should not attempt to confront clients about somatic symptoms or attempt to tell them that these
symptoms are not “real.”
Encourage the client to write in a daily journal
Limiting the time that clients can focus on physical complaints alone may be necessary.
The nurse may have to explain to the family about primary and secondary gains; this will encourage
relatives to stop reinforcing the “sick role.”
• Impaired social interactions
o The client will follow an established daily routine
The nurse must help the client to establish this that includes improved health behaviors.
The challenge for the nurse is to validate the client’s feelings while encouraging him to participate in
activities.
The nurse should help the client plan social contact with others, what to talk about (other than the client’s
complaints), and can improve the client’s confidence in making relationships.
• Anxiety
o The client will demonstrate alternative ways to deal with stress, anxiety, and other feelings
• Disturbed sleep pattern
o The client will demonstrate healthier behaviors regarding rest, activity, and nutritional intake.
The nurse explains that inactivity and poor eating habits perpetuate discomfort and that often it is necessary
to engage in behaviors even though one doesn’t feel like it.
• Fatigue
• Pain
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