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    Pathway DM

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    DiahNiati
    DM tipe I and DM tipe II have different etiologies and pathophysiologies: 1) DM tipe I is caused by an autoimmune reaction or environmental factors like viruses that destroy the beta cells in the pancreas, resulting in a lack of insulin production and regulation of blood glucose. 2) DM tipe II is caused by lifestyle and genetic factors like obesity, and results in insulin resistance where cells do not respond properly to insulin despite sufficient insulin production, leading to high blood glucose levels. 3) Both can result in complications affecting many organ systems if not properly managed, such as damage to blood vessels, nerves, eyes, and kidneys.

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    Pathway DM

    Uploaded by

    DiahNiati
    63 views2 pages
    DM tipe I and DM tipe II have different etiologies and pathophysiologies: 1) DM tipe I is caused by an autoimmune reaction or environmental factors like viruses that destroy the beta cells in the pancreas, resulting in a lack of insulin production and regulation of blood glucose. 2) DM tipe II is caused by lifestyle and genetic factors like obesity, and results in insulin resistance where cells do not respond properly to insulin despite sufficient insulin production, leading to high blood glucose levels. 3) Both can result in complications affecting many organ systems if not properly managed, such as damage to blood vessels, nerves, eyes, and kidneys.

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    pathway DM

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    120249475-Pathway-DM

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    DM tipe I and DM tipe II have different etiologies and pathophysiologies: 1) DM tipe I is caused by an autoimmune reaction or environmental factors like viruses that destroy the beta cells in the pancreas, resulting in a lack of insulin production and regulation of blood glucose. 2) DM tipe II is caused by lifestyle and genetic factors like obesity, and results in insulin resistance where cells do not respond properly to insulin despite sufficient insulin production, leading to high blood glucose levels. 3) Both can result in complications affecting many organ systems if not properly managed, such as damage to blood vessels, nerves, eyes, and kidneys.

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    © All Rights Reserved
    Download as DOC, PDF, TXT or read online from Scribd
    Download as doc, pdf, or txt
    63 views2 pages

    Pathway DM

    Uploaded by

    DiahNiati
    DM tipe I and DM tipe II have different etiologies and pathophysiologies: 1) DM tipe I is caused by an autoimmune reaction or environmental factors like viruses that destroy the beta cells in the pancreas, resulting in a lack of insulin production and regulation of blood glucose. 2) DM tipe II is caused by lifestyle and genetic factors like obesity, and results in insulin resistance where cells do not respond properly to insulin despite sufficient insulin production, leading to high blood glucose levels. 3) Both can result in complications affecting many organ systems if not properly managed, such as damage to blood vessels, nerves, eyes, and kidneys.

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    DM tipe I DM tipe II

    Etiologi : -reaksi autoimun etiologi : - Gaya hidup/obesitas

    - Lingkungan (virus Mumps, Rubella, Virus lainnya)

    - Genetik (antigen HLA)

    Autoimun sel B Endapan lipid di vaskular R. Glukosa di sel

    Transport glukosa

    Destruksi sel B di Kelenjar Pankreas sekresi Insulin (-)/ Hiperglikemia

    Gangguan Metabolisme Resistensi insulin

    Sekresi insulin

    Lemak Karbohidrat Protein Hiperinsulinemia

    Hipertrigliserida lipolisis Glikogenesis, Btk glukosa BUN Sel beta terbeban

    Hiperkolesterolema As lemak bebas ke hati Glukoneogenesis GFR Glukosa sel polifagi

    Btk badan keton Hiperglikemia Krsakan Glomerulus scr mekanisme energi


    progresif
    LDL , HDL Ketonuria, ketonemia Glukosuria kompensasi pecah protein Sorbitol

    Gangguan keseimbangan Sekresi air polifagi Gagal ginjal lipolisis,glikogenesis Fruktosa

    asam basa Poliuria BB , lemas, lelah, tenaga


    Kematian
    Endapan lipid di vaskuler Diabetik ketoasidosis T. osmotik koloid plasma
    Dx : perubahan nutrisi kurang
    Dx: Resiko infeksi
    Dx: resiko cidera
    Dx : Kelelahan

    Aterosklerosis Diabetik Ketoasidosis c.intrasel intavaskuler Osmolalitas intravaskuler

    Hipertensi Arteri Koronaria C.intrasel Polidipsi Jejas osmotik pd sel

    Kompensasi jantung Infark Miokard Dehidrasi Sel Schwann rusak Perisit kapiler retina
    Osmoreseptor di
    Atrofi otot Kematian Hiperosmolalitas hipotalamus Hipoksia sel2 perifer Mikroaneurisma retina

    Dekompensasi kordis Hemokonsentrasi Neuropati perifer


    Dx: gangguan
    Kematian Asidosis Metabolik mual, muntah G. motorik+ sensorik penglihatan
    Organ reproduksi Serebro vaskular Koma Hipovolemi Sensasi nyeri
    Dx: risiko cedera
    Impotensi seksual stroke Kematian Trauma tak trasa
    Dx: kurang
    Kematian volume cairan Ulcus Diabetik
    Dx: Gangguan Dx : gangguan
    pola seksualitas Pembedahan integritas kulit

    Kecacatan

    Sekresi insulin
    Dx: Gangguan citra diri
    Hiperglikemi
    Dx: resiko cedera
    Pemberian insulin , Diet ++ palpitasi,berkeringat

    Glukosa k otak Hipoglikemi tremor, ketakutan, rasa lapar dan mual.

    Gangguan fungsi otak Aktivasi pusat autonom Koma

    pusing,penglihatan kabur, ktjman mental Hipotalamus

    ketajaman mental,kejang2,kesadaran Hormon Epinefrin Kematian

    Dx: perubahan
    Mencari informasi Dx: kurang pengetahuan
    Krisis situasi proses keluarga

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