Tension Headache
Tension Headache
Tension Headache
1. Definition
Tension-type headache (TTH) is the most common form of headache. It can be categorized into
three subtypes according to the International Classification of Headache Disorders based on
headache frequency: (1) infrequent episodic TTH (<12 headache days/year), (2) frequent episodic
TTH (12-180 days/year), and (3) chronic TTH (>180 days/year).
2. Classification
There are at least 10 episodes of attack in <1 day / month (or <12 days / year), headache ends
within 30 minutes - 7 bilateral days, suppresses binding, no pulsation, mild or moderate, no
nausea / vomiting, may be present phonophobia / photophobia, has absolutely nothing to do with
other headache diseases.
It was found to meet the TTH criteria but less one criterion for TTH was mixed with one of the
probable migrane criteria. Headache lasts> 15 days / month for> 3 months (or> 180 days / year),
headache lasts for several hours or continuously, bilateral, feeling of pressure / binding, mild or
moderate intensity, no severe nausea or vomiting , there may be photopobia / phonopobia, no
relation to other head diseases, at least the last 2 months.
3. Etiology
a. Tension and stress.
b. Tiredness
c. Anxiety
d. Length of reading, typing or concentration (eye strain)
e. Poor posture
f. Neck and spine lesions
g. High blood pressure
h. Physical dan stress emotional
4. Clinical Features
TTH comprises headache attacks with mild to moderate pain intensity and is often described as
having a pressing or tightening (nonpulsating) quality that is not aggravated by routine physical
activity, such as walking or climbing stairs. The pain lasts for at least several hours to days and is
predominantly felt bilaterally.
5. Pathophysiology
TTH is often associated with psychopathological psychological stress disorder, especially anxiety
and depression. Depression is a situation characterized by an unpleasant mood that feels all the
dislike and inability to get pleasure. In sufferers of depression, stress, and disorders (anxiety) in
place there is a lack of serotonin, and noradrenaline levels in the brain. Serotonin and nor-
adrenaline are neurotransmitters that play a role in the healing process and depression, which
regulates mood. Serotonin deficits occur, so vasoconstriction occurs in blood vessels and
transferred to the threshold of headache. Serotonin is degraded by the enzymatic action of
monoamine oxidase and is released through 5-hydroxyindoleacetic acid 5-hydroxyindoleacetic
acid (Mumenthaler and Mattle, 2004).
TTH can be caused by stress, alcohol, and hormonal which will stimulate the sympathetic nervous
system resulting in an increase in nor-epinephrine which is spread to the muscle spindles and
causes vasoconstriction. Nor-epinephrine is also associated with blood vessels so that it is
stimulated by cervical sympathetic ganglia and comfortable neck pain (Wesley, 2001).
6. Therapy