Abruptio Placenta and Abortion
Abruptio Placenta and Abortion
Abruptio Placenta and Abortion
Pathophysiology
•Small vessel rapture- due to strong vasocontraction
•Bleeding lifts placenta because uterus is unable to contract and control bleeding.- because there is
leakage, the pulling of blood in endometrium
•Due to uterine contractions blood flows through membranes and uterine walls separating them.
•Tissue Damage and blood clots thrombo plastin -escape into maternal circulation through open sinuses
-activate coagulation mechanism - fibrinogen converted to fibrin. – formation of clots
Signs and symptoms of abruptio placenta
• Concealed or visible bleeding
• Painful bleeding
• Dark red bleeding – unoxygenated blood
• Board like abdomen – the uterus is contracted
• Fetal distress – more than 160
• Tetanic contraction – no resting phase
HALLMARK SYMPTOMS:
PAINFUL DARK RED VAGINAL BLEEDING
Placental Grades
A. Grade O - Patient asymptomatic. Small retroperitoneal clot seen after delivery.
B. Grade 1 - Vaginal bleeding, may have abdominal tenderness or slight uterine tetany, mom and baby
not in distress.
C. Grade 2 - Uterine tenderness, tetany with or without evidence of bleeding, baby shows signs of
distress.
D. Grade 3 - Uterine tetany, severe bleeding may not be visible. Baby is dead. Mom often has
coagulopathy.
Diagnostic test
• CBC
• Blood typing
• APTT/PT
• Bleeding time
• Clotting time
• Serum electrolyte
• Serum creatine
• UA
• USD
• NST
• DIC
Consumptive coagulopathy 2° to hypofibrinogenemia along with elevated levels of fibrinogen-
Fibrin degradation products
NURSING CARE:
• Assess and Monitor:
Amount of Vaginal
• I and O
• Measure abdominal girth
• Uterine characteristics and activity EFM-Continuously
• For development of coagulation problems
• Review lab values: CBC, Coagulation studies, PT,PTT
Nursing Diagnosis
• Altered tissue perfusion
• Fluid volume deficit
• Risk for infection
• Anxiety
• Acute pain
• Spontaneous abortion
- threatened abortion
- imminent abortion
- complete abortion
- incomplete abortion
- missed abortion
- recurrent/ habitual abortion
- septic abortion
Pathophysiology/Etiology
1. Cause frequently unknown, but 50% are due to chromosomal anomalies – sa DNA
and RNA structure
2. Exposure or contact with teratogenic agents – example medication
3. Poor maternal nutritional status-
4. Maternal illness with or specific bacterial microorganisms – example UTI
5. History of diabetes, thyroid disease,
6. Smoking or drug abuse or both
7. Immunologic factor
8. Luteal phase defect – in menstruation
9. Postmature sperm or ova
10. Structural defect in the maternal reproductive system (including an incompetent
cervix)
Immune reaction
Bugs ( infection)
Cervical incompetence
Endocrine
Chemiotactic activity – inflammatory response
Induced abortion
1. Therapeutic abortion
2. Eugenic abortion
Types of miscarriage
1. Missed
- no vaginal bleeding
-close cervical os
- no fetal cardiac activity or empty sac
2. Threatened
- vaginal bleeding
- closed cervical os
- fetal cardiac activity
3. Inevitable
- vaginal bleeding
- dilated cervical os
- products of conception may be seen or felt at or above cervical os
4. Incomplete
- vaginal bleeding
- dilated cervical os
- some products of conception expelled and some remain
5. Complete
- vaginal bleeding
- closed cervical os
- products of conception completely expelled
Inevitable Abortion
- Immediate evacuation of pregnancy.
(If duration of pregnancy less than 12 weeks suction evacuation and greater than 12 weeks oxytocin
infusion.)
Threatened Abortion
- Conservative with bed rest and reassurance till bleeding stops .
- Follow up with UL TRASOUND-f2rresence of fetal cardiac activity predicts good outcome in 95%of
cases.
- Hormone therapy -400mg natural progesterone in divided doses orally or vaginally on empirical basis.