Hydrocephaluss

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Hydrocephalus

• The term hydrocephalus is derived


from the Greek words "hydro"
meaning water and "cephalus"
meaning head.
• Hydrocephalus results from an
imbalance between production and
absorption of CSF.

• The balance between production and


absorption of CSF is critically
important.
• When production is more than
absorption, CSF accumulates within
the ventricular system producing the
dilation of the spaces in the brain
called ventricles.

• The ventricular system is made up of


four ventricles connected by narrow
pathways.
• Normally, CSF flows through the
ventricles, exits into cisterns (closed
spaces that serve as reservoirs) at
the base of the brain, bathes the
surfaces of the brain and spinal cord,
and then is absorbed into the
bloodstream.
• Ideally, the fluid is almost completely
absorbed into the bloodstream as it
circulates; however, there are
circumstances which, when present,
will prevent or disturb the production
or absorption of CSF, or which will
inhibit its normal flow. When this
balance is disturbed, hydrocephalus
is the result.
Flow of CSF
• lateral ventricles--> foramen of
Monro third ventricle -->
aqueduct of Sylvius --> fourth
ventricle --> foramina of
Magendie and Luschka -->
subarachnoid space over brain
and spinal cord --> reabsorption
into venous sinus blood via
arachnoid granulations.
Incidence
• Occurs in 3 - 4 cases in every 1000
births.
• Hydrocephalus may be

Communicating
Non – communicating ( obstructive )
Communicating hydrocephalus
• There is no blockage between the
ventricular system, the basal cisterns
and the spinal subarachnoid space.

• Failure in the absorption system-


unknown cause
• Excessive production of CSF- tumor
or unknown cause
Obstructive or
Non-Communicating hydrocephalus
• The block is at any level in the
ventricular system (source of
production and area of its
reabsorption), commonly at the level
of the aqueduct or foramina of
Luschka and Magendie.
• There are two other forms of
hydrocephalus which do not fit
distinctly into the categories
mentioned above and primarily
affect adults:
1. Hydrocephalus ex-vacuo and
2. Normal pressure hydrocephalus.
• Hydrocephalus ex-vacuo:
Hydrocephalus ex-vacuo occurs
when there is damage to the brain
caused by stroke or traumatic injury.
In these cases, there may be actual
shrinkage (atrophy or wasting) of
brain tissue. In cerebral atrophy
ventricles are dilated but pressure is
not raised .
• Normal pressure hydrocephalus.
Normal pressure hydrocephalus
commonly occurs in the elderly and
is characterized by memory loss,
dementia, gait disorder, urinary
incontinence, and a general slowing
of activity.
Causes
Hydrocephalus can be congenital or
acquired.
• Congenital hydrocephalous:

 Intrauterine infections ( rubella,


cytomegalovirus, toxoplasmosis),
 Intracranial & intraventricular bleed.
 Midline tumors
 Congenital malformations
• Acquired hydrocephalous:

Tuberculosis
Chronic and pyogenic meningitis
Intraventricular hemorrhage
Posterior fossa tumors:
medulloblastoma, astrocytoma,
ependymoma
Arteriovenous malformation
Intracranial hemorrhage
Ruptured aneurysm
Pathophysiology
• The ventricles become greatly
distended

• The increased ventricular pressure


results in thinning of the cerebral
cortex and cranial bones especially
frontal, parietal and temporal areas.
• The floor of the third ventricle commonly
bulges downward, compress the optic
nerves

• The basal ganglia, brain stem and


cerebellum remain relatively normal but
compressed

• The choroid plexus is usually atrophied to


some degree.
Clinical manifestations
• Symptoms of hydrocephalus vary with
age, disease progression, and individual
differences in tolerance to CSF. For
example, an infant's ability to tolerate CSF
pressure differs from an adult's. The infant
skull can expand to accommodate the
buildup of CSF because the sutures (the
fibrous joints that connect the bones of
the skull) have not yet closed.
In infancy
• The most obvious indication of
hydrocephalus is often the rapid
increase in head circumference or an
unusually large head size.

• Delayed closure of the anterior


fontanelle, tense and elevated.
• Spasticity of muscles of extremities
• Later signs: bossing of forehead,
scalp appears shiny with prominent
scalp veins, eyebrows and eyelids
drawn upward, sunset eyes in
infants, difficulty holding head,
strabismus, nystagmus, optic
atrophy, physical and mental
development lag
• Older children
Older children have closed sutures and
present with signs of IICP.
Signs of increased ICP
• Vomiting, sleepiness, restlessness,
irritability, high pitched shrill cry ( infant),
tensed bulging fontanelle ( infant), rapid
increase in head circumference ( infant),

• Changes in vital signs ( increased systolic


pressure, decreased pulse, decreased and
irregular respiration, increased
temperature), pupillary changes,
papilledema, lethargy and stupor, coma,
seizures.
• Older children may also experience;
headache, especially on awakening,
lethargy, fatigue, apathy, problems with
balance, poor coordination, gait
disturbance, urinary incontinence,
personality changes, separation of cranial
sutures ( upto 10 yrs ) visual changes
( double vision )downward deviation of the
eyes (also called "sunsetting").
Diagnostic evaluation
• Physical examination infant’s head
transilluminates, Macewen’s sign
( cracked pot sound on percussion )
• Ophthalmoscopy
• CT scan
• Skull x-ray
• Ventriculography ( rare )
Treatment
• Surgical
Direct operation on the lesion causing obstruction
such as tumor

Intracranial shunts to divert fluid in


noncommunicating hydrocephalus from
ventricular system to the subarachnoid space

Extracranial shunts to divert fluid from ventricular


system to an extracranial compartment
frequently peritoneum or right atrium
Complications
• Complications may include mechanical
failure, infections, obstructions, and the
need to lengthen or replace the catheter.

• Generally, shunt systems require


monitoring and regular medical follow up.
When complications do occur and due to
growth of child, usually the shunt system
will require some type of revision.
• Child with V-A shunt may experience
endocardial contusion, clotting,
leading to bacterial endocarditis,
bacteremia, thromboembolism.
Nursing diagnosis
• Altered cerebral tissue perfusion r/t
IICP
• Altered nutrition ( less ) r/t reduced
oral intake and vomiting
• Risk of impaired skin integrity r/t
alteration in level of conciousness
and enlarged head
• Anxiety r/t surgery
• Risk for fluid volume deficit r/t CSF
drainage, decreased intake
postoperatively
• Risk for injury r/t malfunctioning
shunt, infection
• Ineffective family coping r/t
diagnosis and surgery

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