Pulpal Anatomy and Access Preparations: Appendix A

APPENDIX A
Pulpal Anatomy and Access

Preparations
▲ Lisa R. Wilcox
The illustrations in this appendix depict the size, shape, 2. The size and appearance of the access on anterior
and location of the pulp space within each tooth, as well teeth as viewed from the incisal surface
as the more common morphologic variations. Based on 3. The approximate size of the access opening
this knowledge of the shape of the pulp and its spatial 4. The location of canal orifices and their positions rela-
relationship to the crown and root, the correct outline tive to occlusal landmarks and to each other
form for access preparation is presented from the occlu- 5. The canal curvatures and the location of the apical
sal, lingual, and proximal views. From these illustrations, foramina
the following features can be observed: 6. The configuration of the chamber and cervical portion
of the canals after straight-line access preparation
1. The location of access on posterior teeth relative to
7. The root curvatures that are most common
occlusal landmarks such as marginal ridges and
cusp tips Each illustration gives the following information:
Original canal
orifice
Orifice after Floor of
straight- chamber
Access location line access
from facial
Location
of pulp Access
horns outline
Where dentin shelf

is removed during Root outline,
straight-line access canal location,
and shape in
midroot section
*
*Most common root curvatures
In addition, the percentages of the more common demonstrate relative occurrences. The more common
morphologic variations of the roots and canals are given. root and canal curvatures are included. These are curva-
With many of the tooth groups, the percentages do not tures not readily identified on radiographs (i.e., toward
total 100%. The remaining percentage represents the less the facial and lingual aspects).
common variations not illustrated. Percentages are
approximate to give general information, primarily to Text continued on p. 432
419
420 Appendix A ■
Pulpal Anatomy and Access Preparations
Maxillary Right Central Incisor

B
* Midroot
*
L
Incisal
Cervical view
D M
1 canal: 100%
Maxillary Right Lateral Incisor

B
* Midroot *
L
Incisal
view
D M
1 canal: 100%
Maxillary Right Canine

B
* *
Midroot
L
Incisal
view
1 canal: 100%
D M

Appendix A ■
Pulpal Anatomy and Access Preparations 421
Maxillary Right First Premolar

B
* MESIAL *
* 2 roots
Apical
L
B
Midroot
D M
L B L
B
B
57%
D M Cervical
16%
B
1 root
L
Midroot B
12%
B
L
D M
L
10%

422 Appendix A ■
Maxillary Right Second Premolar

MESIAL
B
*
1 canal:
53%
Midroot
D M B
B
1 root: 88%
2 canals,
1 foramen:
L 22%
D M
2 canals:
13%
Apical
L
D M
B
B
2 roots: 11% Midroot
11%
L
D M B
Cervical
L B L
*Most common root curvatures L

Appendix A ■
Maxillary Right First Molar
*
*
D M D M
D M
3 canals: 40% 4 canals: 60%
* *
B
*
L
Midroot
MESIAL DISTAL
B L
Single
foramen:
80%
L
MESIOBUCCAL
B L B
Midroot section
Two
(mesiobuccal root)
foramina:
20%
2 canals: 60%

424 Appendix A ■
Maxillary Right Second Molar
3 roots: 60%
*
* D M
D M
D M
L
midroot
MESIAL DISTAL
2 mesiobuccal
canals: 38%
Single
foramen: L
15%
B
Two
foramina:
10%

Appendix A ■
Maxillary Right Second Molar
2 roots: 25% L
B
D M Midroot
FACIAL
MESIAL
FACIAL
B
Midroot section
1 root: 10%
L
B
Midroot section

426 Appendix A ■
Mandibular Right Central and Lateral Incisor
D M
Central incisor: 25%
Lateral incisor: 30%
Incisal
view
*
B
L
Midroot
B
Central incisor: 5%
L Lateral incisor: 15%
* Midroot
Mandibular Right Canine
D M
Incisal
view
*
20% B
L
Midroot
B
L
* 70%
Midroot
10%

Appendix A ■
Mandibular Right First Premolar

B MESIAL
L
Midroot
70%
D M
B
*
L
Midroot
B
* 4%
1 foramen:
74%
M D B
L
D M Midroot
B
25%
L
Midroot
2 foramina:
25%

428 Appendix A ■
Mandibular Right Second Premolar

MESIAL
D M B
85%
B L
Midroot
*
*
B
1 foramen:
97%
B
D M
12%
L
L Midroot
D M
3%
2 foramina: L
3%
Midroot

Appendix A ■
Mandibular Right First Molar
D M
M
M D D
* *
MESIAL DISTAL
70%
L
Midroot *
60%
B
20%
L
Midroot
40%
10%

430 Appendix A ■
Mandibular Right Second Molar
D M
D
M M
D
*
*
MESIAL DISTAL
L 92%
40% Midroot
B
5%
35%
L
Midroot
25% 3%

Appendix A ■
Mandibular Right Second Molar

B
D M
C-shaped canal D M
B B B B
Midroot variations
*Most common root curvatures L L L L
Some Uncommon Variations

Mandibular Maxillary Mandibular
two-rooted three-rooted multicanaled
canine premolar premolar
M D D M
D M
D M
Mandibular Maxillary four-rooted

three-rooted molar second molar
D M
D M
D M D M
L
432 Appendix A ■
Examples of access openings prepared in extracted marks (marginal ridge and cusp tips) and (2) the size and
teeth are given here. It is important to recognize: (1) the shape of the access relative to the size and shape of the
location of the access relative to occlusal or lingual land- occlusal or lingual surface.
1. Maxillary central incisor 2. Maxillary canine
3. Maxillary first premolar 4A. Three-canal maxillary molar
4B. Four-canal maxillary molar 5. Mandibular incisor

Appendix A ■
6. Mandibular canine 7. Mandibular premolar
8A. Three-canal mandibular molar 8B. Four-canal mandibular molar

APPENDIX B
Chapter Review Questions

▲ Bruce C. Justman and W. Craig Noblett
Appendix B contains a series of questions for each of the draw on several statements contained within the text.
chapters. These questions have been developed not only The review questions are available in two formats, print
to test the reader’s comprehension of the material, but or electronic on the accompanying DVD. This appendix
also to ensure that the important concepts from each includes a correct answer key. The DVD includes a ratio-
chapter are highlighted. In answering these questions, nale for the correct answer, as well as the page cross refer-
the reader will become acquainted with many of the ence in which those concepts and answers can be found
critical points of information contained within each in the book.
chapter. It is the authors’ intention that by completing this
The questions are set in a multiple choice format and exercise and understanding the answers, the reader will
are drawn from the chapters; however, a specific answer be able to organize the information into a basic under-
may not be immediately evident because a question may standing of endodontics.
434
Appendix B ■
Chapter Review Questions 435
CHAPTER 1 a. Root canal diameter increases in size.

The Dental Pulp and Periradicular Tissues b. Pulp horns grow higher into the cup tips.
c. Overall size of the pulp chamber is reduced.
1. What is the primary function of the dental pulp?
d. Mesiodistal dimension of pulp space is reduced
a. induction
more than apical-occlusal dimension in molars.
b. formation
c. nutrition
10. The cementodentinal junction (CDJ) is which of the
d. defense
following?
a. the area where cementum contacts dentin inside
2. Which of the following is not a stage of tooth the canal
formation? b. located at the same level for each tooth type
a. bud c. usually located 1.0 to 2.0 mm coronal to the apical
b. cap constriction
c. bell d. the widest portion of the canal
d. basal
11. What is the defensive function of the dental pulp?
3. From what cells are odontoblasts derived? a. odontoclast activation for resorption of dentin
a. undifferentiated basal from areas of inflammation
b. undifferentiated ectomesenchymal b. odontoblast formation of enamel to increase
c. undifferentiated cartilage enamel thickness
d. dental papilla c. odontoclast differentiation into macrophages
d. odontoblast formation of dentin in response to
4. Where does deposition of unmineralized dentin injury
matrix begin and in what direction does it proceed?
a. It begins at the cusp tip and progresses laterally. 12. Which of the following is not a major component of
b. It begins at the cusp tip and progresses the odontoblast?
cervically. a. cell body
c. It begins at the cementoenamel junction and b. basally located nucleus
progresses laterally. c. cell process
d. It begins at the cementoenamel junction and d. synaptic junction
progresses coronally.
13. What cell type is primarily related to the immune
5. The cervical loop is the location of which of the system in the dental pulp?
following? a. dendritic
a. the apical constriction b. macrophages
b. formation of the pulp horn c. neutrophils
c. formation of the furcation d. all of the above
d. where inner and outer dental epithelium meet
14. What type of collagen is the most prominent found
6. What is the first thin layer of dentin that is formed? in the dental pulp?
a. predentin a. type I
b. primary dentin b. type II
c. mantle dentin c. type III
d. root sheath d. type IV
7. Epithelial cell rests of Malassez are remnants of 15. Which of the following is not a type of pulp stones?
what? a. free
a. odontoblasts b. attached
b. cementoblasts c. embedded
c. epithelial root sheath d. floating
d. teeth of the deciduous dentition
CHAPTER 2
8. Why are lateral canals clinically significant?
a. Symptoms may persist if not treated.
Protecting the Pulp, Preserving the Apex
b. They allow pulp disease to extend to periodontal 1. What is a “pulp cap”?
tissues. a. an early stage of tooth development
c. They effectively block periodontal disease from b. capping the exposed pulp with a thin layer of
extending to the pulp. lining or base material
d. They contain the greatest amount of pulp tissue. c. capping exposed necrotic tissue by placing a layer
of mineral trioxide aggregate (MTA)
9. What morphological changes occur to the dental d. a method of isolation used during root canal
pulp over time? treatment
436 Appendix B ■
2. What is the effect on blood flow to the pulp when 9. Vital pulp therapies have variable rates of success.
anesthetics with vasoconstrictors are used during What is the most significant determinant of the
restorative procedures? success of vital pulp therapy?
a. reduced by 10% its normal rate a. periapical status before the procedure
b. reduced to less than half its normal rate b. periodontal status before the procedure
c. unchanged c. pulp status before the procedure
d. increased by 25% because of stress on the pulp d. type of restorative material to be used
tissue
10. Which of the following steps are used in step-wise
3. What is dentin “blushing”? evacuation of caries?
a. the color of newly erupted teeth due to large pulp a. removing all the caries in a single visit
chambers b. placing a calcium hydroxide base at the initial
b. use of a masking color during restorative visit
procedures c. placing a glass ionomer base at each visit
c. vascular hemorrhage of pulp tissue, often during d. removing only a superficial layer of caries at the
crown preparation first visit
d. an esthetic concern requiring laminate restorations
11. How does a direct pulp cap for an accidental mechan-
4. Why are deeper carious lesions more injurious to the ical pulp exposure differ from a direct pulp cap for an
dental pulp? exposure caused by caries?
a. increased dentin permeability in deeper areas a. Pulp is likely to be severely inflamed beneath a
b. increased length of the dentinal tubule in deeper deep carious lesion.
areas b. Long-term success for a mechanical exposure is
c. decreased density of dentinal tubules in deeper low.
areas c. Long-term success for a carious exposure is high.
d. decreased diameter of dentin tubules in deeper d. A mechanical exposure should be immediately
areas root canal treated.
5. Why does a blast of compressed air directed at freshly 12. When does apical closure occur in the developing
exposed dentin create a sensation of pain? root?
a. frightens the patient a. at the time of eruption
b. air is cold b. approximately 1 year after eruption
c. rapid outward movement of fluid in patent den- c. approximately 3 years after eruption
tinal tubules d. approximately 5 years after eruption
d. rapid inward movement of fluid in patent dentinal
tubules 13. If the pulp becomes necrotic before root growth is
complete, the resultant root is
6. What is the most important characteristic of any a. short with thick dentin walls
restorative material in determining its effect on the b. short with thin dentin walls
pulp tissue? c. normal length with thick dentin walls
a. heat generated by the material d. normal length with thin dentin walls
b. speed the material sets
c. ability to form a marginal seal 14. What is apexogenesis?
d. life expectancy of the restorative material a. induction of a calcific barrier across an open apex
b. removal of the necrotic pulp
7. What effect does orthodontic tooth movement have c. determination of corrected working length
to the dental pulp? d. continued physiological root formation
a. no clinically significant changes
b. continued reliability to electric pulp testing 15. Which of the following has not been demonstrated
c. extrusion reduces pulpal blood flow for a few with MTA when used in apexification?
minutes a. good biocompatibility
d. intrusive forces have no effect on pulpal blood b. good sealability
flow c. high pH value
d. adjacent zone of necrosis
8. Should bases be used to protect the pulp beneath
metallic restorations?
CHAPTER 3
a. Yes, a thin cement base should be used.
b. Yes, a thick layer of varnish should be used.
Endodontic Microbiology
c. No, a base is only necessary if the tooth is to be 1. Which of the following is not a main portal of entry
crowned. for microorganisms to enter the dental pulp?
d. No, additional thermal insulation is rarely a. dentinal tubules
needed. b. direct pulp exposure
Appendix B ■
c. periodontal disease b. are more resistant to antimicrobial treatments

d. occlusal grooves c. are able to adapt to harsh environmental
conditions
2. Why is there greater dentin permeability near the d. all of the above
pulp?
a. increased thickness of dentin
b. smaller diameter dentinal tubules
CHAPTER 4
c. higher density of dentinal tubules
d. longer length of dentinal tubules Pulp and Periapical Pathosis
1. A direct pulp exposure of a carious lesion is necessary
3. Exposed dentin provides an unimpeded access for to have a pulpal response and inflammation.
bacteria to enter the pulp. a. True
a. True b. False
b. False
2. What factor is the most important in determining if
4. What is anachoresis? pulp tissue becomes necrotic slowly or rapidly after
a. artificial formation of an apical barrier carious pulp exposure and pulpal inflammation?
b. induction of a biologic calcific apical barrier a. virulence of bacteria
c. microorganism transport from blood vessels into b. host resistance
damaged tissue c. amount of circulation
d. systemic infection resulting from infected pulp d. lymph drainage
tissue
3. What is necessary for pulp and periradicular pathosis
5. Root canals can become infected through
to develop?
anachoresis?
a. exposure of pulp tissue
a. true
b. exposure of dentin
b. false
c. presence of bacteria
d. trauma
6. Which of the following is not a category of intrara-
dicular infections?
4. Which of the following statements is true regarding
a. primary
mechanical irritants?
b. secondary
a. Changes to the underlying pulp, such as odonto-
c. tertiary
blast aspiration, are irreversible.
d. persistent
b. Potential for pulp injury decreases as more dentin
7. The most common microorganisms in primary end- is removed.
odontic infections are c. Operative procedures without water coolant cause
a. gram-negative bacteria. more irritation than those performed under water
b. gram-positive bacteria. spray.
c. facultative anaerobes. d. There is decreased permeability and constric-
d. facultative aerobes. tion of blood vessels in the early stages of
pulpitis.
8. Which of the following is not a source of nutrients
for bacteria within the root canal system? 5. What nonspecific inflammatory mediators are not
a. necrotic pulp tissue present when the dental pulp is irritated?
b. inflamed vital pulp tissue a. histamine
c. proteins and glycoproteins seeping into the root b. epinephrine
canal system c. bradykinin
d. components of saliva penetrating into the pulp d. arachidonic acid metabolites
tissue
6. What cell type associated with immune response is
9. Which of the following microorganisms are com- not present in severely inflamed dental pulp?
monly present in large percentages of root canal– a. T lymphocytes
treated teeth that present with persistent apical b. B lymphocytes
periodontitis, indicative of failed treatment? c. macrophages
a. Enterococcus faecalis d. odontoclasts
b. Pseudoramibacter alactolyticus
c. Tannerella forsythia 7. What is the cause of pain during the progression of
d. Dialister invisus pulpal injury?
a. elevation of the sensory nerve threshold
10. Gram-positive bacteria have been demonstrated to b. decrease of arteriole vasodilatation
a. have a higher occurrence in post-instrumentation c. increase of venule vascular permeability
samples d. decrease of pulp tissue pressure
438 Appendix B ■
8. What is reversible pulpitis? CHAPTER 5

a. severe inflammation of pulp tissue Diagnosis and Treatment Planning
b. yields a negative response to electric pulp
1. Which of the following is not one of the five basic
testing
steps in the diagnostic process?
c. yields a positive response to thermal pulp
a. chief complaint
testing
b. medical and dental history
d. requires root canal treatment
c. oral examination
d. review of insurance coverage
9. What is irreversible pulpitis?
a. a severe inflammatory process
2. Are patients that seek endodontic treatment usually
b. precedes reversible pulpitis
younger or older than the general population?
c. resolves when the causative agent is removed
a. Age has not been shown to be a factor.
d. yields a negative response to thermal pulp
b. Patients seeking endodontic treatment are usually
testing
younger.
c. Patients seeking endodontic treatment are usually
10. Which of the following is not a hard tissue change
older.
that may result from pulpal irritation or
inflammation?
a. calcification of pulp tissue spaces 3. During a review of the patient’s healthy history, it is
b. resorption of pulp tissue spaces noted that the patient is on a regimen of intravenous
c. formation of pulp stones bisphosphonate medication. What significance does
d. thickening of periodontal ligament this hold for the patient and their treatment plan?
a. possible side effect of bleeding disorders
11. What are the signs and symptoms associated with b. possible side effect of osteonecrosis of the jaw
symptomatic apical periodontitis (acute apical c. lowered pain threshold
periodontitis)? d. inability to obtain adequate anesthesia
a. normal sensation on mastication
b. normal sensation on finger pressure 4. When pain is one of the patient’s complaints, what
c. marked or excruciating pain on tapping with a question is less relevant regarding their pain and does
mirror handle not need to be asked of the patient?
d. presence of a large periapical lesion a. When did the pain begin?
b. Is the pain always in the same place?
12. What histologic feature differentiates a periapical c. Why did you not seek treatment when the pain
granuloma from a periapical cyst? began?
a. presence of mast cells d. Once initiated, how long does the pain last?
b. presence of lymphocytes
c. presence of plasma cells 5. Why is it important to use control teeth during the
d. presence of an epithelial lined cavity clinical tests?
a. to calibrate the patient’s response
13. Which of the following is not associated with acute b. so the patient can predict which tooth is being
apical abscess? tested
a. moderate to severe discomfort c. so teeth can be tested repeatedly
b. intense and prolonged response to thermal d. to test whether isolation is adequate
stimulus
c. negative response to electric pulp testing 6. A painful response obtained by pressing or by tapping
d. tenderness to percussion and palpation on the crown indicates the presence of which of the
following?
14. What factors may impact and influence whether a. periapical inflammation.
periradicular lesions heal completely or b. pulpal inflammation.
incompletely?
a. size of the lesion 7. What is palpation testing used to determine?
b. blood supply a. pulpal inflammation
c. systemic disease b. periapical inflammation
d. all of the above c. periodontal inflammation
d. periapical histology
15. What is the most important aid in distinguishing
between endodontic and nonendodontic periradicu- 8. Which of the methods of cold testing are preferred
lar lesions? for pulp testing?
a. radiographic location a. regular ice (frozen water)
b. radiographic appearance b. refrigerant spray or CO2
c. pulp vitality testing c. flooding the arch with chilled water
d. patient’s history d. blast of air from the air/water syringe
Appendix B ■
9. How does electrical pulp testing determine the degree CHAPTER 6

of pulpal inflammation? Endodontic and Periodontal
a. A shorter response indicates a healthier pulp. Interrelationship
b. A midrange response indicates pulp
inflammation. 1. Which of the following are potential avenues for
c. A midrange response indicates partial necrosis. communication between the dental pulp and
d. It can only be used to determine the presence or periodontium?
absence of vital tissue. a. dentinal tubules
b. apical foramen
c. lateral/accessory canals
10. What are the four characteristics of a periapical lesion
d. all of the above
of endodontic origin?
a. The lamina dura of the tooth socket is intact. 2. What characterizes the diameter of dentinal tubules
b. The lucency remains at the apex in radiographs in radicular dentin?
made at different cone angles. a. The diameter is 1 μm on the root surface, 3 μm at
c. The lucency tends to resemble a round circle. the pulp.
d. It is usually associated with an irreversible b. The diameter is constant.
pulpitis. c. The diameter is 3 μm on the root surface, 1 μm at
the pulp.
11. In which situation is caries removal necessary to d. The diameter increases with age.
obtain a definitive pulpal diagnosis?
a. deep caries with no symptoms and negative pulp 3. Patent accessory canals are characterized by which of
testing the following?
b. deep caries with no symptoms and positive pulp a. They serve as a pathway for microorganisms from
testing pulp to periodontium.
c. shallow caries with mild symptoms and positive b. They can be the result of scaling and root
pulp testing planing.
d. shallow caries with mild symptoms and negative c. In the apical third, they may lead to pulp necrosis
pulp testing if exposed to oral environment.
d. All of the above.
12. How may selective anesthesia be an aid in
diagnosis? 4. A narrow, vertical probing depth associated with pulp
a. It can localize a painful tooth to a specific arch. necrosis, but only mild periodontal disease is
b. It can localize an individual painful tooth in the probably
mandibular arch. a. a vertical root fracture
c. It can confirm the tooth the patient identifies as b. a fistula
the offending tooth. c. a sinus tract
d. It can be used to start posterior and work toward d. a periodontal abscess
the anterior teeth.
5. The effect of periodontal disease on the pulp is
thought to occur by migration of _____________
13. Using the Case Difficulty Assessment system devel-
through dentinal tubules, accessory canals, or the
oped by the American Association of Endodontists,
apical foramen.
cases in which any factors score 3 should be
a. dental plaque
a. treated by a general dentist.
b. saliva
b. treated by an endodontist.
c. microorganisms
d. exudates
14. Which of the following is not a category of external
resorption? 6. Poor endodontic treatment can allow reinfection of
a. inflammatory the canal, leading to treatment failure and subsequent
b. replacement inflammatory response of the periodontal tissues.
c. regenerative a. True
d. surface b. False
15. If a patient is to be referred to an endodontist for 7. Periodontal inflammation resulting from primary
treatment, when is the most appropriate time for the endodontic disease may mimic periodontal disease by
referral? which of the following?
a. before beginning treatment a. generalized increase in probing depths in the
b. during treatment when expected difficulties affected quadrant
arise b. an apical radiolucency
c. before weekends or holidays c. a narrow deep solitary probing defect associated
d. after obturation when separated instruments are with an affected tooth
present d. all of the above
440 Appendix B ■
8. A patient presents with a chief complaint of pain 14. Treatment sequencing for primary endodontic
to cold temperatures. Examination and testing re- disease with secondary periodontal involvement
veal a maxillary left premolar exhibiting severe includes
lingering pain in response to cold. No caries or frac- a. scaling and root planing followed by endodontic
tures are noted. Periodontal probings are 6 to 9 mm treatment
around that specific tooth and 6 to 7 mm around the b. endodontic treatment followed by scaling and
other posterior teeth in the quadrant. The patient root planing
reports having a “deep cleaning” (root planing) three c. endodontic treatment followed by periodontal
times per year. What would the diagnosis in this case surgery
be? d. endodontic treatment followed by reevaluation of
a. primary endodontic disease with secondary peri- periodontal status in 2 to 3 months
odontal involvement
b. primary periodontal disease with secondary end-
odontic involvement
CHAPTER 7
c. separate and unrelated endodontic and periodon-
tal disease
Longitudinal Tooth Fractures
d. a true combined endodontic-periodontic (endo- 1. What category of longitudinal tooth fractures is most
perio) disease process severe?
a. craze lines
9. What will the long-term prognosis for the patient b. fractured cusp
described in question 8 primarily depend on? c. cracked tooth
a. successful periodontal treatment d. vertical root fracture
b. successful endodontic treatment
c. treatment sequencing 2. What clinical conditions or situations are often asso-
d. timing of treatment ciated with cusp fractures?
a. teeth with minimal caries
10. Which of the following best represents similarities b. strong support of the remaining cusps
between endodontic apical and periodontal c. missing marginal ridge
pathosis? d. occlusal composite restorations
a. Both are often the result of traumatic occlusion.
b. Both are usually symptomatic. 3. What is the common direction that fractures extend
c. Both are mediated by microorganisms. in cracked teeth?
d. Both are associated with loss of attachment. a. mesiodistal
b. faciolingual
11. The best description of the effect of moderate peri- c. apical to coronal
odontal disease (loss of attachment in the apical/ d. horizontal
middle third) on the underlying pulp is usually char-
acterized by 4. Which of the following is true as related to pulp tissue
a. no or slight regional inflammation involvement in a cracked tooth?
b. generalized acute inflammation a. The fracture always includes the pulp tissue.
c. generalized chronic inflammation b. The fracture never includes the pulp tissue.
d. necrosis c. The more centered the fracture, the greater chance
e. bacterial invasion through tubules into the pulp for pulp exposure.
d. The more facial or lingual oriented the crack, the
12. Which of the following is characteristic of the true greater the chance for pulp exposure.
combined endo-perio lesion?
a. It occurs much less frequent than the primary 5. Are pulp and periapical tests for cracked teeth consis-
endodontic lesion. tent and reliable?
b. It is usually the end result of a severe endodontic a. Both pulp and periapical testing are consistent.
lesion that causes loss of attachment. b. Pulp testing is consistent, but periapical testing is
c. It is usually the end result of a severe periodontal variable.
lesion. c. Pulp testing is variable, but periapical testing is
d. It is usually successfully managed with both end- consistent.
odontic and periodontal treatment. d. Both pulp and periapical testing are variable.
13. What is the best means of differentiating endodontic 6. How can transillumination be used to distinguish
from periodontal pathosis? between a craze line and a crack line?
a. pulp vitality testing a. It cannot be used to distinguish between the two
b. percussion entities.
c. radiographs b. Transmitted light readily passes through the air
d. probing patterns space of a fracture.
e. location of swelling c. Transilluminated light is blocked by craze lines.
Appendix B ■
d. Transilluminated light is blocked by a cracked 2. Which of the following is not a component of the
tooth. psychologic approach to pain management?
a. control
7. After access preparation of a suspected cracked tooth, b. communication
the crack line is seen to extend through the chamber c. conservation
floor. In this case, what is the prognosis and recom- d. confidence
mended treatment?
a. Prognosis is favorable and continue with root 3. What is the effect of warming the anesthetic solution
canal treatment. on the amount of pain during the injection?
b. Prognosis is questionable, inform patient and con- a. Warmed anesthetic results in less pain during
tinue with treatment. injection.
c. Prognosis is hopeless, and extraction is b. Warmed anesthetic solution results in greater pain
recommended. during injection.
d. It does not change the original prognosis, and the c. There is no difference in pain perception regard-
treatment plan is not altered. less of warming.
8. What is the preferable restoration of a cracked 4. What is a two-stage injection?

tooth? a. Injection of one cartridge of anesthetic, waiting 5
a. post and core and crown minutes followed by injection of a second car-
b. amalgam core tridge of the same anesthetic solution.
c. cast inlay b. Injection of one cartridge of anesthetic, followed
d. full coverage crown by injection of a second cartridge of a different
anesthetic solution.
9. How does a split tooth differ from a cracked c. Injection of a quarter cartridge of anesthetic under
tooth? the mucosal surface, waiting until regional anes-
a. A split tooth precedes a cracked tooth. thesia, then injection of the remainder of the car-
b. A split tooth has an incomplete fracture. tridge to full depth.
c. A split tooth has separable tooth segments. d. Injection of a quarter cartridge of anesthetic under
d. A cracked tooth has the fracture extending the mucosal surface, waiting until regional anes-
faciolingually. thesia, then injection of a cartridge of different
anesthetic solution to full depth.
10. What direction does a vertical root fracture (VRF)
primarily occur? 5. When does the onset of pulpal anesthesia occur after
a. mesiodistal the inferior alveolar injection?
b. faciolingual a. immediately
c. coronal and extending apically b. 0 to 5 minutes
d. no primary direction c. 10 to 15 minutes
d. 30 minutes
11. What is a demonstrated major cause of VRFs?
a. traumatic occlusion 6. Does the direction of the needle bevel affect the effec-
b. occlusal biting habits tiveness of the inferior alveolar nerve block?
c. loss of one or both marginal ridges a. Needle bevel toward the mandibular ramus
d. condensation forces during obturation improves success.
b. Needle bevel away from the mandibular ramus
12. Which is not a possible treatment of a VRF in a mul- improves success.
tirooted tooth? c. Half the cartridge should be injected with the
a. tooth extraction bevel toward the ramus, the needle rotated, and
b. nonsurgical retreatment of the affected root the second half of the cartridge injected with the
c. root amputation of the affected root bevel away from the ramus.
d. hemisection and extraction of the affected root d. Direction of the needle bevel does not affect
success.
CHAPTER 8
Local Anesthesia 7. Is anesthesia of the maxilla commonly more or less
successful than anesthesia in the mandible?
1. What is the allodynia phenomenon? a. more successful
a. Inflamed tissue has an increased threshold of b. less successful
pain. c. comparable success rates
b. Inflamed tissue has a decreased threshold of
pain. 8. Does increasing the volume of anesthetic solution
c. Inflamed tissue is much less sensitive to a mild affect the duration of pulpal anesthesia?
stimulus. a. For mandibular anesthesia, increasing the volume
d. Inflamed tissue responds mildly to a stimulus that improves the success rate with the inferior alveolar
would otherwise be very painful. nerve block.
442 Appendix B ■
b. For maxillary infiltrations, increasing the volume d. Another supplemental injection should not be
increases the depth of pulpal anesthesia. attempted first.
c. For maxillary infiltrations, increasing the volume
increases the duration of pulpal anesthesia. 15. What are the most difficult teeth to anesthetize with
d. For maxillary infiltrations, increasing the volume irreversible pulpitis?
has no effect on the success rate of pulpal a. maxillary molars
anesthesia. b. mandibular molars
c. maxillary anterior teeth
9. What additional anesthesia procedure should be d. maxillary premolars
administered if the classic signs of anesthesia
are present after a standard injection, but the 16. Why should an anesthetic agent not be injected
patient still has sharp pain when the bur enters the directly into a swelling before an incision for
dentin? drainage?
a. repeat the initial injection a. The anesthetic will cause a decreased flow of
b. wait an additional 15 minutes and attempt access exudate following incision.
again b. A direct injection will spread the infection.
c. repeat the injection using a different type of anes- c. There is an increased chance of aspirating
thetic solution blood.
d. use a supplemental injection technique for a d. The swelling has increased blood supply so the
second injection anesthetic is transported quickly into systemic cir-
culation, diminishing the effect.
10. What is a consideration of the intraosseous (IO)
injection?
a. It has not been proved effective. CHAPTER 9
b. It has been recommended as the primary injection Endodontic Emergencies and Therapeutics
technique.
c. It allows the anesthetic solution to be deposited 1. What is the difference between a true endodontic
directly into the pulp tissue of the tooth. emergency and urgency?
d. It allows the anesthetic solution to be deposited a. A true emergency is a condition requiring an
directly into the cancellous bone adjacent to the unscheduled office visit.
tooth. b. A true emergency may be rescheduled for conve-
nience of the patient.
11. What is the best site for an IO injection of a c. An urgency indicates a more severe problem.
premolar? d. An urgency may need to be seen after normal
a. mesial perforation and injection office hours.
b. apical perforation and injection
c. distal perforation and injection 2. How many teeth are usually involved in a true
d. site of injection not important emergency?
a. one tooth
12. What is an important requirement for effectiveness b. two teeth
when using a periodontal ligament (PDL) injection? c. often several teeth throughout the mouth
a. back-pressure during injection d. often teeth in only one quadrant
b. direction of the needle bevel toward the root
surface 3. Which of the following is not an important factor in
c. direction of the needle bevel away from the root assessing the quality and quantity of pain?
surface a. spontaneity
d. all four line angles receive the injection b. intensity
c. time of day of occurrence
13. Can a PDL injection be used for individual tooth d. duration
selective anesthesia as an aid in diagnosis?
a. PDL injection is useful for single tooth 4. Which of the following is not an immediate goal of
anesthesia. the emergency treatment plan?
b. PDL injection is not useful for single tooth a. pharmacotherapeutic management of swelling
anesthesia. b. reducing the irritant
c. reduction of pressure
14. Which of the following is an important consideration d. removal of the inflamed pulp or periradicular
of the intrapulpal injection (IP)? tissue
a. The injection should be given with
back-pressure. 5. What is the most critical factor in a pretreatment
b. It will take several minutes for the injection to take emergency?
effect. a. adequate health history
c. A long-acting anesthetic should be used. b. pain management
Appendix B ■
c. patient management a. more common than flare-ups following cleaning

d. adequate provisional restoration and shaping
b. infrequent and usually resolve spontaneously
6. What is the preferred treatment for an emergency c. approximately 10% to 15% occurrence
appointment with a diagnosis of irreversible pulpitis d. often require nonsurgical root canal retreatment
with symptomatic apical periodontitis?
a. caries excavation with provisional restoration
b. trephination through the mucosa and bone
c. partial or total pulpectomy
d. pharmaceutical management of swelling
CHAPTER 10
7. What is the emergency treatment of choice for pulp

Management of Traumatic Dental Injuries
necrosis without swelling? 1. Why is age a “good news/bad news” situation with
a. trephination for drainage dental trauma?
b. pulpotomy a. Pulps have an incomplete and decreased blood
c. canal débridement to corrected working length supply.
d. single visit root canal treatment b. Pulps are better able to recover and have a better
repair potential.
8. A patient may present with localized swelling; inci- c. Poor development will continue in teeth with
sion for drainage may be indicated. What does drain- damaged necrotic pulps.
age accomplish? d. Dentin has more strength in younger teeth.
a. patient reassurance and management
b. decrease in blood flow to the area 2. Which of the following factors does not need to be
c. allows administration of anesthetic solution to the considered when evaluating a crown fracture with
apex pulp exposure?
d. removal of a very potent irritant—purulence a. extent of fracture
b. stage of root development
9. At an emergency appointment, should teeth be left c. position in the arch
open to drain? d. length of time since the injury
a. Yes, only if there is swelling.
b. Yes, only if there is no swelling. 3. Which of the following is not a step as part of the
c. No, teeth should have an interappointment tem- technique for a shallow (partial) pulpotomy?
porary restoration placed. a. rubber dam isolation
b. pulp tissue removed to about 2 mm below the
10. What is the most important consideration of admin- exposure
istering antibiotics with a localized apical abscess? c. use of a large round carbide bur in the slow-speed
a. The dosage should be for 10 days. handpiece to remove tissue
b. It should be a broad-spectrum antibiotic. d. restoration of the cavity with a hard-setting
c. The dosage levels should be higher than usual. cement
d. Antibiotics are ineffective and should not be
prescribed. 4. How long should horizontal root fractures be splinted
if the coronal section was displaced and
11. What is the incidence of interappointment repositioned?
flare-ups? a. not indicated
a. primarily after obturation at a range of 4.4 to b. 7 to 10 days
6.0% c. 4 to 6 weeks
b. overall at a range of 1.8 to 3.2% d. 3 months
c. overall at a range of 10.7 to 21.3%
d. primarily after vital pulp removal at a range of 7.5 5. Which of the following are types of luxation
to 10.7% injuries?
a. concussion
12. Which of the following has been identified as signifi- b. intrusion
cant factor related flare-ups? c. extrusion
a. teeth with vital pulps d. all of the above
b. teeth without periradicular radiolucency
c. patient presenting with preoperative pain or 6. What is recommended with pulp testing for teeth
swelling with traumatic injuries?
d. completing endodontic treatment in a single a. use of electric pulp testing or carbon dioxide ice
visit to test the injured and adjacent teeth
b. opposing teeth also be tested
13. What is a consideration of postobturation c. retesting is done in 4 to 6 weeks
emergencies? d. all of the above
444 Appendix B ■
7. What information does the color change of the clini- b. The permanent successor is partially erupted.
cal crown provide? c. The intruded tooth appears foreshortened on the
a. There has been a pulp exposure. radiograph.
b. The initial change is gray in color, which always d. The intruded tooth appears elongated on the
indicates pulp necrosis. radiograph.
c. Discoloration may be reversed without
treatment.
CHAPTER 11
d. Calcific metamorphosis discoloration tends to be
yellow to brown and always indicates pulp
Endodontic Radiography
necrosis. 1. Diagnostic radiology is helpful in all of the following
except:
8. What factor should be considered that determines the a. identifying pathosis
treatment of an intrusive luxation injury? b. determining root anatomy
a. depth of intrusion c. determining pulp anatomy
b. stage of root development d. determining pulp responsiveness
c. availability of adjacent teeth for stabilization
d. amount of soft tissue injury 2. What are working length radiographs?
a. Radiographs made by removing the rubber dam.
9. Of the following, what is the best transport medium b. Radiographs placed using an XCP positioning
to use for transporting an avulsed tooth? device.
a. saliva c. Radiographs that help establish an estimated
b. distilled water working length.
c. wrapped in a tissue d. Radiographs determine the distance from the
d. milk radiographic apex to a reference point.
10. Which type of medication is indicated for patients
3. Radiographs are useful to evaluate the following qual-
with avulsed teeth?
ities of an obturation except which of the following?
a. narcotic analgesic
a. length
b. steroid
b. density
c. tetanus booster if more than 5 years since last
c. sealer thickness
administered
d. canal configuration
d. all of the above
11. What additional treatment should be used on the 4. Radiographs are useful in evaluating success and
root surface if an avulsed tooth is replanted after failure at recalls because they do which of the
more than 1 hour after avulsion? following?
a. thorough scrubbing with antimicrobial soap for a. record subjective symptoms
disinfection b. show pulp vitality
b. scaling of the root surface c. may show failures that often occur without adverse
c. soaking the tooth in 2.4% doxycycline for 5 to 20 signs or symptoms
minutes d. accurately diagnose apical pathosis
d. soaking the tooth in a 2.4% solution of sodium
fluoride for 5 to 20 minutes 5. The most accurate radiographs are made by doing
which of the following?
12. Which of the following types of external resorption a. having the patient hold the film in place with
has not been identified with replanted avulsed their index finger
teeth? b. using a paralleling device
a. surface c. increasing/decreasing the vertical angulation to
b. inflammatory move superimposed objects out of the field of
c. refractory vision
d. replacement d. having the rubber dam in place for isolation
13. When is root canal treatment indicated in a mature 6. Use of a paralleling technique may not be feasible
avulsed, replanted tooth? when which of the following occurs?
a. at the time of replantation a. There is a high palatal vault.
b. within 7 to 10 days after replantation b. There are maxillary tori.
c. after 3 months if there is no response to pulp c. A fixed prosthesis is present.
testing d. There are exceptionally short roots.
d. when periapical pathosis is noted
7. F Speed film requires how much less exposure com-
14. A deciduous tooth that has suffered an intrusive luxa- pared to E Speed film?
tion should be extracted if what occurs? a. 10% to 15%
a. The child cries but is compliant. b. 20% to 25%
Appendix B ■
c. 30% to 35% 15. Digital radiography has not been proven to do which
d. 50% of the following?
a. provide superior image quality
8. What does the cone-image shift do? b. reduce radiation to the patient
a. It gives a clear 2-dimensional image. c. increase speed of obtaining an image
b. It superimposes facial and lingual structures. d. accurately and reliably be transmitted between
c. It assists in identifying superimposed canals. computers
d. It moves apical endodontic lesions away from the
root apex.
CHAPTER 12
9. What occurs as the cone position moves away from
parallel?
Endodontic Instruments
a. Objects on the film shift toward the direction of 1. What must an instrument do to completely clean the
the cone. canal space?
b. The facial or buccal object shifts less than the a. be deflected at the canal orifice
lingual object. b. be 2 to 3 mm short of the radiographic apex
c. The lingual object moves relatively in the same c. fit loosely into the canal
direction as the cone. d. contact all walls and surfaces
d. The buccal object moves relatively in the same
direction as the cone. 2. What motion is employed with a hand instrument to
clean and shape canal walls?
10. What is a disadvantage to the cone-image shift? a. pushing
a. Lingual objects become more distorted than buccal b. broaching
objects. c. reaming
b. There is excessive contrast between radiolucent d. vibration
and radiopaque objects.
c. It may superimpose normal anatomic structures 3. Nickel-titanium alloy has increased flexibility over
over the root apices. stainless steel. How does the modulus of elasticity for
d. It does not reveal additional canals within a nickel-titanium alloy compare to that of stainless
root. steel?
a. similar to stainless steel
11. Which of the following is a distinguishing charac- b. one-fourth to one-fifth that of stainless steel
teristic of a radiolucent lesion of endodontic c. half that of stainless steel
pathosis? d. 2 to 3 times that of stainless steel
a. Apical/radicular lamina dura is present and
intact. 4. What is a disadvantage as a result of the increased
b. A round ball shape is characteristic. flexibility of nickel-titanium instruments?
c. The radiolucency stays at the apex regardless of a. difficulty in negotiating curvatures
cone angulation. b. inability to rotate in the canals
d. There is no apparent cause of pulpal necrosis. c. cannot precurve the files to bypass ledges
d. tendency to bind in small canals
12. A radiolucency of endodontic origin is usually present
with what type of pulpal diagnosis? 5. According to ADA Specification No. 28, what is the
a. normal pulp rate of increase in file diameter per running millime-
b. reversible pulpitis ter of length for a K-type file from point D0 to point
c. irreversible pulpitis D16?
d. necrotic pulp a. 0.02 mm per running millimeter of length
b. 0.04 mm per running millimeter of length
13. What is the usual radiographic appearance of con- c. 0.06 mm per running millimeter of length
densing osteitis? d. parallel sided so no increase in diameter
a. diffuse radiopaque appearance
b. uniform smooth borders 6. What is torsional limit?
c. irregular moth-eaten appearance around the a. amount of apical pressure that can be applied to a
apex file to the point of breakage
d. presence of a radiolucent inflammatory lesion b. the beginning of plastic deformation of the
instrument
14. A mesial projection cone adjustment during working c. amount of rotational torque that can be applied
length radiographs is indicated for what? to a “locked” instrument to the point of
a. maxillary anterior teeth breakage
b. maxillary molars with a mesiolingual canal d. amount of force necessary so that a file does not
c. mandibular incisors return to its original shape upon unloading of the
d. mandibular molars with a second distal canal force
446 Appendix B ■
7. Which is a stronger metal alloy: carbon steel or stain- CHAPTER 13

less steel? Internal Anatomy
a. Carbon steel is stronger.
b. Stainless steel is stronger. 1. Lack of knowledge of pulp anatomy is the __________
most common cause of treatment failure.
8. How do Gates-Glidden drills differ from Peeso a. least
reamers? b. second
a. Gates-Glidden drills are a greater length of cutting c. third
surface. d. fourth
b. Gates-Glidden drills are more aggressive cutters.
c. Gates-Glidden drills have an elliptical-shaped 2. Of the following, which is the best technique to
cutting area. determine if a root contains two canals?
d. Gates-Glidden drills are less flexible. a. apex locator
b. viewing access with a microscope
c. searching with an explorer
9. How are broaches intended to be used in the
d. interpreting angled radiographs
canal?
a. planing of canal walls by a push-pull motion
3. The shape of the canal in cross-section is variable but
b. planing of canal walls by a reaming motion
is almost always round in the apical third.
c. placed to the corrected working length around
a. Both parts of the sentence are true.
canal curvatures
b. The first part of the sentence is false, the second
d. entangling and removing canal contents by
part is true.
rotation
c. The first part of the sentence is true, the second
part is false.
10. Which of the following describes the filing motion? d. The entire sentence is false.
a. a single-step motion
b. used only on the furcation side of a molar root 4. Multiple canals in mandibular premolars occur most
canal often in which population?
c. with a 360-degree rotation motion a. Asians
d. circumferential movement around the canal b. African-Americans
walls c. Caucasians
d. No difference by ethnicity
11. Regular inspection of hand files may aid in avoidance
of instrument separation. What file defects should be 5. Alterations in the anatomy of the pulp space occur
looked for on inspection? because of which of the following?
a. unwinding of the flutes a. resorption
b. rolling up or tightening of the flutes b. age
c. distortion of the tip c. calcifications
d. all of the above d. all of the above
12. What are the characteristics of finger spreaders and 6. Calcifications encountered in the pulp space do which
pluggers compared to handled instruments when of the following?
used for lateral condensation? a. represent additional dentin formation
a. They are annealed to give them greater strength. b. can always be detected by radiograph
b. They are best suited for straight canals. c. are always attached to the chamber or canal
c. They are more rigid to access the canal orifice. walls
d. They have greater flexibility. d. often prevent instruments from negotiating
canals
13. Is pressure sterilization superior to dry heat steri-
lization for sterilization of sharp-edged instruments? 7. Which of the following is not associated with the
a. Both are equal and comparable and effective. radicular pulp?
b. Neither should be used for sterilization. a. lateral canals
c. Yes, pressure sterilization is superior. b. apical foramen
d. No, dry heat sterilization is superior. c. pulp horns
d. canal orifices
14. What are the time, temperature, and pressure neces-
sary for sterilization of gauze-wrapped instruments 8. Accessory canals are more common in the apical
using pressure sterilization? third, and more common in posterior teeth.
a. 10 minutes at 121ºC and 15 psi a. The entire sentence is true.
b. 10 minutes at 100ºC and 15 psi b. The first statement is true, the second is false.
c. 20 minutes at 121ºC and 15 psi c. The first statement is false, the second is true.
d. 20 minutes at 100ºC and 15 psi d. The entire sentence is false.
Appendix B ■
9. Which of the following are true regarding the apical c. The rubber dam protects the patient from swallow-
foramen? ing or aspirating instruments and materials.
a. The diameter remains constant throughout d. The rubber dam injures soft tissue from the pres-
life. sure of the clamp.
b. The position of the apical foramen is often visible
on radiograph. 2. What is the recommended rubber dam weight for
c. The foramen is most commonly located 0.5 mm endodontic procedures?
to 1.0 mm away from the anatomic root apex. a. light
d. None of the above. b. medium
c. heavy
d. extra heavy
10. Dens invaginatus (dens in dente) occurs most com-
monly in which teeth?
a. maxillary canines 3. Why are plastic rubber dam frames recommended
b. maxillary lateral incisors over metal frames?
c. maxillary and mandibular lateral incisors a. They are radiolucent.
d. mandibular first premolars b. They are easier to remove during exposure of
interim radiographs.
c. They are more comfortable for the patient.
11. The lingual groove defect is (1) found most frequently d. They are easier to place.
in maxillary central incisors and (2) has a poor prog-
nosis for treatment.
4. Which of the following clamps is designed for an
a. Statements 1 and 2 are true.
anterior tooth?
b. Statement 1 is true, statement 2 is false.
a. No. 8
c. Statement 1 is false, statement 2 is true.
b. No. 212
d. Statements 1 and 2 are false.
c. No. 0
d. No. 24/25
12. A C-shaped canal is characterized by which of the
following? 5. What is an advantage of a provisional crown used to
a. has complex internal anatomy replace missing tooth structure before root canal
b. is most commonly found in Asian populations treatment?
c. usually occurs in mandibular second molars a. It accurately reproduces tooth anatomic
d. should be referred to an endodontist for landmarks.
treatment b. It maintains tooth orientation for access and canal
e. all of the above location.
c. It is easily removed and replaced during root canal
13. Of the following, which tooth or root is the most treatment appointments.
likely to have two canals? d. It increases visibility of the root canal chamber.
a. Maxillary second premolar
b. Mandibular first molar mesial root 6. What is the preferred method for rubber dam place-
c. Mandibular lateral incisor ment on molars?
d. Maxillary first molar mesiobuccal root a. placement as a unit
b. placement of a clamp and rubber dam, followed
by attachment of the frame
14. The lingual root of the maxillary first molar often has
c. placement of a clamp, followed by the dam and
a curvature in the apical third to which of the
then the frame
following?
d. placement of the rubber dam and frame, followed
a. buccal
by placement of the clamp
b. lingual
c. mesial
d. distal 7. What is a major objective of the access opening?
e. none of the above, the root is usually straight a. Locate the primary or largest canal.
b. Achieve unimpeded straight-line access of the
instruments to the first canal curvature or apical
one third.
CHAPTER 14
c. Expose the pulp horns.
Isolation, Endodontic Access, and Length d. Remove all restorative materials.
Determination
1. What is true of the rubber dam? 8. Outline form for access is described best by which of
a. The rubber dam is elective for endodontic the following?
treatment. a. It mimics the shape of the canal or canals.
b. The rubber dam allows irrigating solution to b. It is toward the distal in the occlusal surface in
contact and disinfect surrounding soft tissues. molars.
448 Appendix B ■
c. It is a projection of the internal tooth anatomy CHAPTER 15

onto the external surface. Cleaning and Shaping
d. It is a constant and unchanging shape regardless
1. What is the preferred method to evaluate if a canal
of age.
has been adequately cleaned?
a. The canal is three file sizes larger than the initial
9. What is an advantage of caries removal during
master apical file.
access?
b. The canal walls are “glassy smooth” when explored
a. It enhances the effectiveness of NaOCl.
with a file.
b. It reduces interappointment pain.
c. Dentin shavings obtained are clean and white.
c. It strengthens tooth structure.
d. Irrigant runs clear with no visible debris.
d. It allows assessment of the restorability prior to
the endodontic treatment.
2. The degree of canal enlargement during shaping is
dictated by which of the following?
10. Estimated depth of access is a measurement from
a. method of obturation
b. anatomy of the root
a. incisal edge of anterior teeth to the coronal portion
c. plan for post placement
of the pulp chamber
d. all of the above
b. occlusal reference of posterior teeth to the coronal
portion of the pulp chamber 3. The apical termination point for cleaning and shaping
c. incisal edge of anterior teeth to the radiographic the root canal should be which of the following?
apex of the tooth a. the radiographic apex
d. occlusal reference of posterior teeth to the radio- b. at the major diameter of the apical foramen
graphic floor of the chamber c. within 0 to 2 mm of the radiographic apex
d. 0.5 mm beyond the radiographic apex
11. What is the shape of the access opening of a maxil-
lary central incisor in a young patient? 4. To prevent extrusion of obturating material, clean-
a. round ing and shaping procedures must be confined to
b. triangular the radicular space. Canals filled to the radio-
c. trapezoidal graphic apex would be considered to be the perfect
d. square result.
a. Both statements are true.
12. What is the outline shape of the access for a maxillary b. The first statement is true; the second statement is
first molar? false.
a. round c. The first statement is false; the second statement
b. triangular is true.
c. trapezoidal d. Both statements are false.
d. square
5. For the irrigating solution to effectively reach the
13. What is the outline shape of the access for a man- apical third of the canal, the apical canal should be
dibular first molar with four canals? enlarged to at least a No. _____ file.
a. round a. 25 or 30
b. triangular b. 20 or 25
c. trapezoidal c. 35 or 40
d. square d. 45 or 50
14. To obtain an accurate measurement, how should the 6. Which of the following is the most widely used irrig-
working length radiographs be made? ant solution?
a. They should be made with a loosely fitting file in a. sodium hypochlorite
place. b. ethylenediaminetetraacetic acid (EDTA)
b. They should be made with a minimum of a No. c. MTAD
20 file. d. saline
c. They should be made with a positioning device
and a parallel technique. 7. The best description of a difference between nickel-
d. They should be made with the rubber dam removed titanium and stainless steel instruments is which of
for visibility and access. the following?
a. Nickel-titanium tends to result in better shaping
15. An apex locator is helpful in patients with which of (less transportation) in curved canals.
the following? b. Nickel-titanium usually results in better
a. with high palatal vaults debridement.
b. with implanted cardiac pacemakers c. Nickel-titanium can usually be reused many more
c. with missed canals times than stainless steel.
d. with a strong gag reflex d. Nickel-titanium has sharper cutting edges.
Appendix B ■
8. What is the primary purpose of an irrigant such as a. inadequate cleaning and shaping of the canals
sodium hypochlorite (NaOCl)? b. inadequate obturation of the root canal system
a. kill bacteria c. restorative factors
b. dissolve tissue remnants d. vertical root fracture
c. flush out debris
d. lubricate instruments 2. How does the survival rate for a tooth restored with
cusp protection compare to a tooth without cusp
9. A major advantage to using a lubricant during clean- protection?
ing and shaping is: a. about the same
a. It ensures that canal transportation will not b. a tooth with protected cusps has an enhanced
occur. survival rate
b. It reduces torsional force on the instrument, c. a tooth without protected cusps has an enhanced
decreasing the possibility of fracture. survival rate
c. It minimizes debris production. d. restorations have no effect on tooth survival
d. It reduces operator fatigue. rates
10. Removal of the smear layer after cleaning and shaping 3. Dentin becomes more brittle following the endodon-
does which of the following? tic treatment due to loss of moisture content.
a. promotes coronal leakage a. True
b. decreases dentin permeability b. False
c. allows better adaptation of obturating materials to
canal walls
4. The greatest contributing factor to reduced cuspal
d. forces bacteria into dentinal tubules
stiffness (strength) that predisposes to fracture is
11. EDTA is most effective for which of the following
a. occlusal access opening
uses?
b. loss of one or both marginal ridges
a. decalcifying small canals to allow instruments to
c. an amalgam restoration placed after root canal
negotiate to length
treatment
b. lubricating canals to facilitate instrumentation
d. a bonded composite restoration placed after root
c. bacterial elimination in canals
canal treatment
d. removing smear layer after cleaning and shaping
5. Which of the following describes a definitive restora-
12. How does the “crown-down” technique differ from
tion after root canal treatment?
the “step-back” technique?
a. It should be placed at the time of obturation.
a. It creates a funnel-shaped preparation.
b. It should allow cuspal flexure to absorb occlusal
b. It facilitates tissue removal.
forces.
c. It requires fewer instruments.
c. It should provide a coronal seal.
d. It creates coronal flare early, reducing torsional
d. It should always be a full-coverage crown on pos-
stress on the instruments.
terior teeth.
13. Recapitulation is defined as:
a. The removal of accumulated debris using a small 6. Exposure of obturating materials to oral fluids can be
file at the corrected working length. described by which of the following?
b. Confirmation of working length after completing a. It is not a factor if a sealer is properly used during
cleaning and shaping. obturation.
c. The last irrigation before drying the canal. b. It is a major cause of failure.
d. Verifying the master apical file after cleaning and c. It leads to rapid failure.
shaping. d. It may cause pain to thermal changes.
14. Of the following, which is the most important con- 7. Which of the following describes a definitive
sideration of a temporary restoration? restoration?
a. antimicrobial a. It should be placed as soon as practical.
b. placed over a cotton pellet b. It should be placed at the 6-month recall visit to
c. resistant to acids assure symptoms do not recur.
d. at least 4 mm thick c. It should be placed when radiographic evidence of
healing becomes evident.
d. It should be delayed if there is a questionable
prognosis.
CHAPTER 16
Preparation for Restoration 8. What is the only reason to delay the definitive
1. What is the leading cause for loss of endodontically restoration?
treated teeth? a. to maximize the patient’s insurance benefits
450 Appendix B ■
b. if the patient is unable to pay for the restora- CHAPTER 17

tion Obturation
c. to allow radiographic healing to become evident
1. What is a possible outcome when there is an overfill
d. if there is a questionable prognosis and failure
of the obturation materials?
would lead to extraction
a. decreased periapical inflammation
b. improved and rapid healing of periapical lesions
9. The practical principles for function and durability c. inadequate apical seal
when designing a definitive restoration include the d. decreased postobturation discomfort
following except which of the following?
a. conservation of tooth structure
2. What is the optimal preparation/obturation
b. retention
length relative to the radiographic apex with pulp
c. placement of a post
necrosis?
d. protection of the remaining tooth structure
a. flush
b. 0.5 to 1.0 mm short
10. What is an indication for use of a direct (amalgam or c. slight extrusion of sealer but not gutta-percha
composite) restoration? d. 1.0 to 3.0 mm short
a. Excessive tooth structure has not been lost.
b. The opposing arch has been restored with full
3. Prognosis and histologic studies show that if there is
coverage crowns.
a length error, there are less problems resulting with
c. Esthetics is not a concern.
d. Only one of the marginal ridges has been lost.
a. overfills
b. underfills
11. Fewer root fractures have been recorded in laboratory
studies when what type of post has been used? 4. Which of the following describes lateral canals?
a. cobalt chromium alloy post a. They connect adjacent canals within the same
b. stainless steel post root.
c. titanium post b. They may allow bacterial and necrotic debris to
d. carbon fiber post leak into the periodontium.
c. They are débrided with copious irrigation.
12. Which of the following describes removal of gutta- d. They are significant in the outcome of most root
percha for post space preparation? canal treatments.
a. immediately after obturation
b. after the sealer has completely set 5. What factors should be considered when deciding the
c. to a depth that allows 2 to 3 mm of remaining timing for obturation?
gutta-percha a. signs and symptoms present
d. completed using solvents b. pulp and periapical status
c. difficulty of the procedure
13. When using a prefabricated post system to restore d. all of the above
a posterior tooth, the most desirable post design
is 6. What pulp and periapical diagnosis may have com-
a. tapered, passively cemented. pleted treatment in a single visit?
b. tapered, threaded screw type. a. symptomatic (acute) apical periodontitis
c. parallel sided, passively cemented. b. asymptomatic apical periodontitis (chronic apical
d. parallel sided, threaded screw type. periodontitis)
c. acute apical abscess
14. Which of the following describes retentive pins? d. painful irreversible pulpitis
a. Retentive pins help strengthen the restoration.
b. Retentive pins minimize stresses to dentin. 7. What material is currently the only universally
c. Retentive pins are the most effective antirotation accepted solid core obturation material?
method for post and cores. a. gutta-percha
d. Retentive pins should not be used because the risks b. synthetic polyester resin-based polymers
outweigh any potential gain. c. silver points
d. solid core (carrier) gutta-percha
15. Placement of a dowel or post through a crown or an
existing restoration is described by which of the 8. Which of the following is a disadvantage of gutta-
following? percha?
a. It adds support for the existing restoration. a. poor adaptation to canal irregularities with
b. It helps maintain integrity of the existing compaction
restoration. b. shrinkage if altered by heat or solvents
c. It improves the seal of the root canal. c. not easily managed and manipulated
d. It is rarely indicated. d. difficult to partially remove from a canal
Appendix B ■
9. Which of the following is an advantage of gutta- b. the possible procedures that may be necessary for
percha? correction
a. adhesiveness to dentin c. how this might affect the outcome
b. slight elasticity and rebound effect d. the root canal treatment will be completed free of
c. expansion on cooling of warmed gutta-percha charge
d. adaptation to canal irregularities with
compaction 2. Which of the following is not a common cause of a
perforation during access preparation?
10. What have recent studies shown synthetic polyester a. mandibular molar with a lingual axial inclination
resin-based polymers to be? of the tooth
a. adhesive to canal walls throughout their length b. searching for canals through an under-prepared
b. inflammatory to tissues access opening
c. mutagenic c. directing the bur parallel to the long axis of the
d. no difference in leakage when compared to gutta- tooth
percha d. presence of a misaligned cast restoration
11. Which of the following have semisolid obtu- 3. What measures are used to prevent perforation during
ration materials (pastes or cements) been shown to access preparation?
do? a. relating tooth angulations independent of the
a. They provide easy control of obturation length. adjacent tooth
b. They exhibit no shrinkage upon setting. b. using only straight-on radiographs
c. They provide an unpredictable and inconsistent c. always having a rubber dam in place before begin-
apical seal. ning access preparation
d. They are biocompatible and nonirritating to peri- d. having a thorough knowledge of both surface and
apical tissues. internal tooth anatomy
4. Which of the following is not an early sign or indica-

12. Which of the following describes the complete setting
tion of a perforation?
of zinc oxide–eugenol (ZnOE)-based sealers?
a. pain during access preparation
a. The setting occurs at approximately 1 hour if left
b. sudden appearance of hemorrhage
exposed to air.
c. burning pain and bad taste during irrigation with
b. The setting depends on contact with dentin.
NaOCl
c. The setting usually requires weeks or months.
d. radiographically malpositioned file
d. The setting usually is approximately 24 hours.
5. If a lateral root perforation does occur, what is the
13. What type of sealer may have a problem with long-
most favorable location for perforation repair?
term solubility?
a. at or above the height of crestal bone
a. ZnOE-based sealers
b. below the crestal bone in the coronal third of the
b. plastic sealers
root
c. glass ionomer sealers
c. on the furcal side of the coronal root surface
d. calcium hydroxide sealers
d. a zipping perforation at the apex of the root
14. Which of the following describes lateral compaction 6. What is the ideal time and material for a nonsurgical
of gutta-percha? repair of a furcation perforation?
a. It is indicated for cases with internal resorption. a. immediate repair with amalgam
b. It involves multiple steps and special b. immediate repair with mineral trioxide aggregate
armamentarium. (MTA)
c. It manages length control well. c. delayed repair with amalgam
d. It is difficult to retreat. d. delayed repair with MTA
15. What is a disadvantage of finger spreaders as com- 7. What is a common cause of ledge formation?
pared to standard long-handled spreaders? a. straight-line access into the canal
a. tactile sensation b. excess irrigating solution
b. instrument length control c. overenlargement of a curved canal with files
c. fracture potential in curved canals d. constant recapitulation and irrigation into the
d. dentin stress during obturation apical portion of the canal
CHAPTER 18 8. What type of canal is most prone to ledge

Procedural Accidents formation?
a. long, small, and curved
1. What should a patient not be told when there has b. incomplete apex formation, curved
been a procedural accident? c. large, long
a. the incident did occur d. short, straight
452 Appendix B ■
9. What is a possible etiology for an apical (zipping) root a. A large well-fitting post and core is present.
perforation? b. There is a separated instrument present that cannot
a. inability to negotiate canals with ledges be retrieved.
b. working length determination with radiographs c. External resorptive root defects are present.
only d. A negotiable canal was not initially treated.
c. trying to locate canals in a small chamber
d. failure to adjust working length after curved canals 2. Which of the following is not a potential contraindi-
are straightened during cleaning and shaping cation for nonsurgical root canal retreatment?
a. post and core restorations
10. What type of perforation has the poorest long-term b. ledges in the root canal walls
prognosis? c. amalgam core restorations into the chamber
a. zipping (apical third) root perforation d. separated root canal instruments
b. stripping perforation in the apical third of the
root 3. Which of the following is not a potential risk associ-
c. stripping perforation in the coronal third of the ated with nonsurgical root canal retreatment?
root below the crest of bone a. thinning and weakening of the root canal
d. direct floor to furcation perforation in a multi- walls
rooted tooth b. inability to remove the initial root canal obtura-
tion material
11. Which of the following is not a common cause of file c. creating a compromised crown-root ratio
separation? d. loosening of a well-fitting fabricated crown
a. limited flexibility
b. manufacturing defects 4. Which of the following describes removal of coronal
c. amount of use restorations before nonsurgical retreatment?
d. amount of force applied a. Removal may prolong retreatment procedures.
b. Removal complicates removal of post and core
12. What approaches may be used to treat a case with a restorations.
separated instrument? c. Removal may be necessary to assess restorability.
a. attempt to remove d. Removal should never be done if the previous
b. attempt to bypass restoration is a full-coverage crown.
c. prepare and obturate to the level of the segment
d. all of the above 5. What steps are involved in retrieval of a prefabricated
post during retreatment?
13. Which of the following scenarios yields the most a. Section and remove the core material and the post
favorable prognosis in cases with a separated at the level of the chamber floor.
instrument? b. An ultrasonic activated tip is used for 3 to
a. a small instrument short of the working length 5 minutes each at several locations circumferen-
b. a small instrument beyond the apical foramen tially around the post, without water for
c. a large instrument at an early stage of visibility.
instrumentation c. Grasp the post with a hemostat or Steiglitz pliers
d. a large instrument close to the working length to rock it back and forth to break the cement
seal.
14. Which of the following causes extrusion of sodium d. Use ultrasonics, then grasp a threaded screw type
hypochlorite (NaOCl) irrigating solution into periapi- post with a hemostat or small-tipped forceps to
cal tissues? unscrew it from the canal.
a. Fitting irrigation needle loosely in the canal
space. 6. The following steps are used to attempt removal of a
b. Wedging irrigation needle in the canal space. canal ledge during nonsurgical retreatment except
c. Using perforated needles during irrigation. which one?
d. Using regular needles during irrigation. a. remove all obstructions coronal to the ledge
b. bypass the ledge with a flexible nickel-titanium
15. Which of the following occurs after minor extrusion hand file
of obturation materials into the periapical tissue? c. file in a circumferential motion after bypassing the
a. It results in significant swelling ledge
b. It results in significant symptoms. d. proceed from small-size files to larger size files
c. It causes some tissue inflammation.
d. It causes more apical leakage. 7. Which of the following is not a factor that may affect
the successful removal of a separated instrument
fragment?
CHAPTER 19
a. size of the instrument separated
Nonsurgical Retreatment b. length of the separated fragment
1. Nonsurgical retreatment should be the first treatment c. location of the fragment within the canal
option for correction when d. length of time the fragment has been in place
Appendix B ■
8. What method or methods have been used to success- c. Flare-ups occur less frequently if irrigation is kept
fully remove gutta-percha from root canals? to a minimum.
a. heat d. High incidence dictates that retreatment should
b. ultrasonics generally be treated in two visits rather than one
c. rotary instruments visit.
d. all of the above
15. The prognosis for nonsurgical root canal retreatment
9. When should Hedstrom hand files or regular hand is which of the following?
reamers be the instruments of choice for gutta-percha a. It is increased with periapical lesions.
removal, without the addition of solvents? b. It has the lowest rate of success without a periapi-
a. The root canal is well sealed with gutta-percha. cal lesion.
b. The gutta-percha is well adapted to canal walls. c. It is similar to initial root canal treatment success
c. A space can be created between the gutta-percha rates.
and the root canal. d. It is best if the etiology of failure can be
d. Gutta-percha fills the root canal chamber. identified.
10. What solvent has been shown to be the most efficient CHAPTER 20
(fastest) in softening gutta-percha? Endodontic Surgery
a. chloroform
b. halothane 1. What is the purpose of incision for drainage?
c. methylchloroform a. to evacuate exudates from a soft tissue swelling
d. xylene b. to obtain a biopsy specimen
c. to prevent a postoperative swelling
11. During the removal of a carrier-based gutta-percha d. to avoid emergency cleaning and shaping
obturator which of the following should occur?
a. The first step is to remove the solid core 2. Profound anesthesia is difficult to attain before inci-
material. sion for drainage. What is a preferred approach for a
b. A combination of techniques for removal of gutta- maxillary cuspid with extensive swelling?
percha, silver cones, and posts is employed. a. Start with an infraorbital block and then infiltrate
c. A small rotary file may be used to engage and at the margins of the swelling.
remove the plastic carrier. b. Start with posterior superior alveolar block and
d. Different solvents are used that would routinely then use refrigerant spray.
be used to remove gutta-percha alone. c. Inject buffer and anesthetic directly into the
swelling.
12. What is the key to success in the retrieval of silver d. Use topical anesthetic and then refrigerant spray.
points? No anesthetic is needed.
a. The key is to engage the silver point with the
ultrasonic tip. 3. Which of the following is not an indication for peri-
b. The key is to remove the silver point and core apical surgery?
material simultaneously. a. a nonnegotiable or blocked canal associated with
c. The key is to retain as much of the coronal extent symptomatic periradicular pathosis
of the point as possible. b. gross overextension of obturating material
d. The key is to remove the core material and silver c. obtain a biopsy
point to the level of canal orifices first. d. to resolve any endodontic treatment failure
13. Which of the following is not true regarding the 4. Which of the following would contraindicate periapi-
removal and retreatment of hard-setting pastes? cal surgery?
a. They are more difficult to remove than a soft 1. anatomical structures in the area
paste. 2. medical complications
b. They may be impossible to remove. 3. lip paresthesia
c. Solvents have been shown to soften hard-setting 4. previous malignancies
pastes. 5. unidentified cause of treatment failure
d. Use of ultrasonics is the most predictable a. 1, 2, and 3
method. b. 1, 3, and 5
c. 1, 2, and 5
14. Which of the following is true regarding interap- d. 2, 3, and 4
pointment flare-ups with nonsurgical root canal e. all of the above
retreatments?
a. Flare-ups occur less frequently when compared to 5. Which of the following is true regarding an incision
initial root canal treatments. over a bony defect?
b. Flare-ups occur frequently, even when debris and a. It should be avoided.
microorganisms are confined to the canals. b. It may cause a postsurgical fenestration.
454 Appendix B ■
c. It may prevent healing of the incision. CHAPTER 21

d. All of the above. Evaluation of Endodontic Outcomes
1. What is the primary determinant for successful end-
6. Which of the following describes a submarginal flap
odontic treatment?
design?
a. selecting the proper obturation technique
a. It is ideal for mandibular posterior teeth.
b. effective elimination of microorganisms from the
b. It causes less scarring.
pulp space
c. It is associated with less gingival recession.
c. using rotary instruments to shape the canals
d. It causes less intraoperative hemorrhage.
d. using an effective irrigation regimen
7. What is the purpose of root end resection?
2. What are the major indicators of successful endodon-
a. removes irritants encased in the apical portion of
tic treatment?
the root
a. lack of discoloration, no tenderness to biting
b. to examine the root
b. no swelling or redness of the gingival area
c. exposes additional canals or fractures
c. absence of symptoms and apical radiolucency
d. all of the above
d. a happy patient who has paid the bill
8. Which of the following is true regarding a root-end
3. A patient presents for a posttreatment examination
cavity preparation?
with no complaint of symptoms except the apical
a. It should be as shallow as possible to preserve
radiolucency that was present before treatment,
tooth structure.
although it appears smaller. Treatment for this patient
b. It should be made to a minimum depth of
would be classified as which of the following?
3 mm.
a. a failure
c. It should only encompass the main portion of the
b. a success
canal.
c. a clinical success but radiographic failure
d. It should be made with a very small bur.
d. a functional tooth with uncertain prognosis
9. An ideal root-end filling material should satisfy all of
the following except which one? 4. Which of the following is not a clinical criterion to
a. The material should be well tolerated by perira- evaluate treatment outcome?
dicular tissues. a. absence of a radiolucency
b. The material should be easily placed. b. no evidence of a sinus tract
c. The material should be absorbable. c. no swelling present
d. The material should be visible radiographically. d. no response to percussion or palpation
10. Which of the following cell types are important in 5. To make valid comparisons between radiographs to
the healing process after periapical surgery? assess healing, which of the following describes how
1. epithelial cells films should be made?
2. macrophages a. in a reproducible manner
3. dendritic cells b. 6 months apart
4. fibroblasts c. at different angles
5. osteocytes d. by the same person to ensure consistency
a. 1, 2, 4, and 5 only
b. 1 and 2 only 6. Which of the following criteria is not considered to
c. 1, 2, and 5 only be a predictor of success or failure?
d. 1, 2, 3, 4, and 5 a. the patient’s medical history
e. 1, 3, and 4 only b. apical pathosis
c. quality of the coronal restoration
11. With root amputation, the factor that most affects d. extent and quality of obturation
success is which of the following?
a. occlusal force patterns 7. The most common preoperative cause of endodontic
b. the type of restoration treatment failure includes all of the following except
c. the length of the root which one?
d. the patient’s oral hygiene a. misdiagnosis
b. leaking coronal restoration
12. All of the following procedures should be referred to c. poor case selection
a specialist with specific training in endodontic d. error in treatment planning
surgery except which one?
a. root-end resection/root-end filling 8. The most common postoperative cause of endodontic
b. incision for drainage treatment failure is which of the following?
c. root amputation a. overextension of obturating material
d. perforation repair b. a separated instrument
Appendix B ■
c. coronal leakage 7. Which of the following is not an indication for inter-

d. placement of a post unnecessarily nal bleaching?
a. defective enamel formation
9. The prognosis for nonsurgical retreatment depends b. intrapulpal hemorrhage-induced stain
primarily on which of the following? c. tetracycline-induced stain
a. identification and correction of the cause of d. sealer stain
failure
b. using a different obturation technique 8. One potential complication of internal bleaching is
c. placing the definitive restoration at the obturation external root resorption, which has been associated
appointment with which of the following?
d. all of the above a. high concentration of hydrogen peroxide
b. heat
c. damage to cementum and periodontal tissues
CHAPTER 22 d. all of the above
Bleaching Discolored Teeth: Internal and
9. What is the most common agent employed in exter-
External
nal bleaching?
1. Which of the following is not considered a natural or a. sodium perborate
acquired discoloration source? b. hydrochloric acid
a. tetracycline stain c. carbamide peroxide
b. intrapulpal hemorrhage d. sodium hypochlorite
c. stain from amalgam
d. calcific metamorphosis 10. Which of the following describes the microabrasion
technique?
2. Which of the following is not part of the mechanism a. It is not a true bleaching technique.
of staining caused by fluorosis? b. It uses hydrochloric acid.
a. Hypoplastic defects are produced in enamel by c. It requires meticulous soft tissue isolation.
excess fluoride. d. All of the above.
b. Stain is acquired from chemicals in the oral
cavity.
CHAPTER 23
c. Stain is present in the enamel.
d. Stain is solely caused by fluoride deposits in the Geriatric Endodontics
enamel. 1. Which of the following are changes that occur in the
pulp with age?
3. Which of the following describes tetracycline stain? 1. decreased vascular elements
a. It is classified into three groups based on 2. decreased amount of collagen
severity. 3. increase in numbers of fibroblasts
b. It is often associated with a “banding” pattern. 4. decrease in numbers of odontoblasts
c. It is located in the dentin. 5. increase in occurrence of calcifications
d. All of the above. a. 1, 2, and 3 only
b. 1, 3, and 5 only
4. What is the most common iatrogenic etiology related c. 1, 4, and 5 only
to tooth discoloration? d. 2, 3, and 5 only
a. incomplete removal of pulp tissue e. 1, 2, 3, 4, and 5
b. incomplete removal of obturating material from
the chamber 2. Which of the following statements are true regarding
c. the use of intracanal medicaments calcifications in the pulp space?
d. intracanal irrigants a. Pulp stones are usually found in the radicular
pulp.
5. Which of the following restorative materials can con- b. Pulp stones can increase the incidence of odonto-
tribute to staining? genic pain.
a. amalgam c. Calcifications increase with both age and
b. pins and posts irritation.
c. composite d. Diffuse calcifications are most commonly found in
d. all of the above the pulp chamber.
e. All of the above.
6. What is the most common agent used for internal
bleaching? 3. With age, which of the following describes the pulp
a. carbamide peroxide chamber in molars?
b. sodium perborate a. decreases primarily in a mesiodistal dimension
c. hydrogen peroxide b. decreases primarily in an occlusal-apical
d. sodium peroxyborate monohydrate dimension
456 Appendix B ■
c. remains the same in volume b. increased cementum deposition, modifying the

d. increases in size in response to irritation apical anatomy
c. differences in tissue electrical resistance, making
4. The healing capacity of older patients is significantly apex locators less accurate
less than younger patients because of a decrease in d. patients unable to sit still for radiographs
periradicular vascularity. The vascularity of the
periradicular tissues is a critical determinant in
healing. Chapter Review Questions Answer Key
a. The first statement is false, and the second state-
ment is true. CHAPTER 1
b. The first statement is true, and the second state- 1. b 4. b 7. c 10. a 13. a
ment is false. 2. d 5. d 8. b 11. d 14. a
c. Both statements are true. 3. b 6. c 9. c 12. d 15. d
d. Both statements are false. CHAPTER 2
1. c 4. a 7. a 10. c 13. b
5. Which of the following medical conditions may 2. b 5. c 8. d 11. a 14. d
influence healing in geriatric patients? 3. c 6. c 9. c 12. c 15. d
a. osteoporosis
CHAPTER 3
b. hypertension
c. immunosuppression 1. d 3. b 5. b 7. a 9. a
d. diabetes 2. c 4. c 6. c 8. b 10. d
CHAPTER 4
6. Which of the following describes bisphosphonates? 1. b 4. c 7. c 10. d 13. b
a. They are used in treatment of osteoporosis. 2. d 5. b 8. c 11. c 14. d
b. They are used in treatment of malignancies. 3. c 6. d 9. a 12. d 15. c
c. They are linked to idiopathic osteonecrosis of the
CHAPTER 5
jaws.
1. d 4. c 7. b 10. b 13. b
d. They interfere with osteoclast function.
2. c 5. a 8. b 11. b 14. c
e. All of the above.
3. b 6. a 9. d 12. a 15. a
7. Which of the following is a common finding on CHAPTER 6
examination in geriatric patients? 1. d 4. c 7. c 10. c 13. a
a. extensive restorative experience with multiple 2. a 5. c 8. b 11. a 14. d
large restorations and crowns 3. d 6. a 9. a 12. a
b. lower incidence of periodontal disease
CHAPTER 7
c. exaggerated symptoms associated with pulp
1. d 4. c 7. c 9. c 11. d
pathosis
2. c 5. d 8. d 10. b 12. b
d. excessive salivation
3. a 6. d
8. Which of the following is a consideration between CHAPTER 8
geriatric and younger patients that may affect the 1. b 5. c 8. c 11. c 14. a
ability to make a diagnosis? 2. c 6. d 9. d 12. a 15. b
a. Older patients are more stoic. 3. c 7. a 10. d 13. b 16. d
b. Decreased response to pulp testing is common. 4. c
c. Symptoms of pulpitis are not as acute in older
CHAPTER 9
patients.
1. a 4. a 7. c 10. d 12. c
d. All of the above.
2. a 5. c 8. d 11. b 13. b
3. c 6. c 9. c
9. A common modification in performing root canal
treatment for older patients is which of the CHAPTER 10
following? 1. b 4. c 7. c 10. c 13. b
a. treatment planning for a shorter lifespan 2. c 5. d 8. b 11. d 14. d
b. access cavity without a rubber dam to locate a 3. c 6. d 9. d 12. c
smaller chamber
CHAPTER 11
c. greater need for anesthetic
1. d 4. c 7. b 10. c 13. a
d. larger restorations make isolation easier
2. d 5. b 8. c 11. c 14. b
3. c 6. b 9. c 12. d 15. a
10. Working length determination in elderly patients
may be more difficult because of which of the CHAPTER 12
following? 1. d 4. c 7. b 10. d 13. d
a. increased bone density, making radiographs harder 2. c 5. a 8. c 11. d 14. c
to interpret 3. b 6. c 9. d 12. d
Appendix B ■
CHAPTER 13 CHAPTER 18
1. b 4. b 7. c 10. b 13. b 1. d 4. a 7. c 10. c 13. d
2. d 5. d 8. a 11. c 14. a 2. c 5. a 8. a 11. b 14. b
3. c 6. a 9. c 12. e 3. d 6. b 9. d 12. d 15. c
CHAPTER 19
CHAPTER 14
1. d 4. c 7. d 10. a 13. c
1. c 4. b 7. b 10. b 13. c
2. c 5. d 8. d 11. b 14. d
2. b 5. c 8. c 11. b 14. b
3. c 6. b 9. c 12. c 15. d
3. a 6. a 9. d 12. b 15. d
CHAPTER 20
CHAPTER 15 1. a 4. c 7. d 9. c 11. d
1. b 4. b 7. a 10. c 13. a 2. a 5. d 8. b 10. a 12. b
2. d 5. c 8. c 11. d 14. d 3. d 6. c
3. c 6. a 9. b 12. d
CHAPTER 21
CHAPTER 16 1. b 3. d 5. a 7. b 9. a
1. c 4. b 7. a 10. a 13. c 2. c 4. a 6. a 8. c
2. b 5. c 8. d 11. d 14. d CHAPTER 22
3. b 6. b 9. c 12. a 15. d 1. c 3. d 5. d 7. a 9. c
2. d 4. b 6. b 8. d 10. d
CHAPTER 17
1. c 4. b 7. a 10. d 13. d CHAPTER 23
2. b 5. d 8. b 11. c 14. c 1. c 3. b 5. d 7. a 9. b
3. b 6. d 9. d 12. c 15. c 2. c 4. a 6. e 8. d 10. b
INDEX
A Anatomic difficulties
Abscess and case referral, 90f
apical contraindicating periapical surgery, 362
acute, 41f, 61–62, 84, 158f as indication for periapical surgery, 359–360
chronic, 62–63, 84 Anesthesia
localized fluctuant, 156f conventional, 132
periodontal, 152f difficulty with, 86, 134–135
Access cavities factors affecting, 129–130
overextended, 383 for incision for drainage, 358
perforations during access preparation, 322–328 local. See Local anesthesia
routine, 280–281 in older patients, 412–413
Access errors previously unsuccessful, 130
excessive removal of tooth structure, 250–252 profound, in emergencies, 152
inadequate preparation, 248–250 selective, 82
resulting in stripping perforations, 268f supplemental, 135–142
Access openings when to anesthetize, 131–132
burs, 238f Angled radiographs, 252, 254–255
and canal location, 239–248, 413 Angulation of radiographs, 188, 191f
canal morphologies, 236 Ankylosis-related resorption, 178–179, 180f
general principles, 236 Anterior teeth, retention and core systems, 294
preoperative radiograph assessment, 236 Antibiotic prophylaxis, 71, 279b
prepared in extracted teeth, 432f–433f inappropriate use of, 153–154
removal of restorative materials, 237 for pulp necrosis with diffuse swelling, 157
through crowns, 239 Anticurvature filing, 268, 270, 271f
use of fissure bur, 236–237 Anxiety, 130
Accessory canals, 95, 222 Apex
Accidents anatomy, 222–223
procedural, 90–91, 322–338, 360 closed, replantations, 177–179
with sodium hypochlorite during treatment, 264f locator, 243f, 255–256
Acellular afibrillar cementum, 18 open. See Open apex
Acid etching of dentin, 25 Apexification, 33–34
Actinomycosis, apical, 41f Apexogenesis, 31–32
Adhesive Apical abscess
intraoral, 130–131 acute, 41f, 61–62, 84, 158f
quality of sealer, 306 chronic, 62–63, 84
Adult-onset tetracycline discoloration, 393 Apical canal preparation
Advantages apical patency, 262
of cone-image shift, 191–192 degree of apical enlargement, 261
of lateral compaction, 308 elimination of etiology, 261–262
of pastes, 305 termination of cleaning and shaping, 260–261
of vertical compaction, 314–315 Apical constriction, 6, 223
Afferent blood vessels, 11 Apical foramen
Age, discoloration and, 392–393 anatomy, 222
Age-related changes communication via, 96
in dentin formation, 219 formation of, 4–5
in pulp and dentin, 17 size and location of, 5
in pulp response, 407 variability in, 414f
Aldehydes, 279b Apical patency, 262
Alveolar bone, 4f, 19–20 Apical perforations, 266, 332, 336f
Alveolar nerve block, 134 Apical periodontitis
Alveolar process fracture, 164b, 180, 181 asymptomatic, 59–61, 84
Amalgam microbial causation of, 38, 39f
microleakage with, 27 persistent, 45t
retaining pins for, 25 symptomatic, 58–59, 153–154
Amputation, root, 369–372 Apical seal, 299
Anachoresis, 39–40 Archaea, in endodontic infections, 42
Analgesia, strategy for drug selection, 154f Arterioles, 11
periodontium, 19
Articaine, 133–134, 139
Page numbers followed by f indicate figures; t, tables; b, boxes. Artificial canal creation, 330–331
458
Index 459
Aspiration of instruments, 334 Burs

Asymptomatic apical periodontitis, 59–61, 84 access, 238f
Asymptomatic pulp necrosis, 143 fissure, 236–237
Autotransplantation, 385 Gates-Glidden, 348, 350f
Avulsions, 164b relationship to tooth, 323–324
description, 176 for removing bone, 365f
management, 183 Butterfly clamps, 232
treatment, 176–180
Axons, 15–16 C
Calcification(s), 10–11
B age-related, 407
Back-pressure, with intrapulpal injection, 141 and alterations in anatomy, 219
Bacteria canal, 87
as-yet-uncultivated phylotypes, 42 molar, 246f
correlation with depth of tubular penetration, 261 palatal canal pulp tissue, 239f
in dental emergencies, 149 pulpal, 55–56
in endodontic infections, 42f, 43f removal of, 344–345
gram-negative, 41–42 Calcific metamorphosis, 55f, 81f, 88f
gram-positive, 42 as cause of discoloration, 392, 393f
minimization of, 383 in older patient, 409f, 411f
patterns of colonization, 44 Calcified canals, 192, 194f
role in pulpal pathoses, 50–51 Calcitonin gene-related peptide, 52–53
Bacteriostatic properties of sealer, 306 Calcium hydroxide, 279b
Balanced force technique, 270–271 placement, 280f
Band placement, 233 in pulpotomy, 170
Bases, 27–28 sealers, 307
Bell stage, 2f–3f Canal orifices
Bicuspidization, 369–372, 373f location of, 218b, 252f
Biopsy, 362 MB2, 244, 247–248
Bisphosphonates, 408–409 Canals. See also Apical canal preparation
Bitewing projections, 197 accessory, 95, 222
Biting instruments, 112f anatomy, 218
Biting test, 76f artificial, 330–331
Bleaching blockage, 90
instruments for, 213, 214f glassy smooth walls after cleaning, 259
internal techniques identification of, 218
final restoration, 398, 400 intracanal instrumentation, 210–212
future rediscoloration, 400 lateral, 95
thermocatalytic, 398 formation of, 4
walking bleach, 398 locating, 187, 239–248, 413
materials, 395–398 mesiolingual, 246f
in older patients, 415–416 morphologies, 236, 237f
tetracycline discoloration, 393 number of, 87
vital tooth, 27 obstructions, removal of, 343–344
when to bleach, 400 overenlargement of, 384f
Blockages, as procedural accident, 90 removal of irritants by débridement, 157
Blood dyscrasias, 394 root. See Root canal
Blood flow determination, 78 superimposed, 191
Blood vessels transportation, 266f
afferent, 11 undiscovered, 192, 194f
efferent, 12 wall with and without smear layer, 265f
lymphatics, 12 Canal space preparation, 291–293
vascular physiology, 12–15 Canines
Bonding mandibular, 426f
in crown-root fractures, 171 access to, 247
in teeth with inadequate structure, 233 two-rooted, 431f
Bone loss, 80f maxillary, 420f
root amputation and, 371f access to, 242
in split tooth, 120f severely discolored, 395f
Bone morphogenetic protein, 34 Capillary bed, 12, 13f
Bone resorption, 57f Capping vital pulp, 28
in vertical root fracture, 124f–125f Cap stage, 2f–3f
Bridges Carbamide peroxide, 397
dentin, 8f Carbon dioxide ice testing, 77f
success rates, 381t for luxation injuries, 175–176
Broaches, 209, 210 Carbon fiber posts, 291, 292f
Bud stage, 2f Caries
Bupivacaine, 133 extensive, 234f
460 Index
Caries (Continued) Cleaning accidents (Continued)

mesial, access through, 253f extrusion of irrigant, 334
recurrent, 342f ledge formation, 328–330
removal of, 28, 82, 236 root perforations, 332–333
Carrier-based systems, 317 separated instruments, 333
Carriers with attached gutta-percha, 316f, 348–349 Cleansing of cavity, 24–25
Case difficulty assessment form, 83f Clearing, apical, 271, 276
Case selection Clinical features
crown fractures, 167 cracked tooth, 113–114
decision tree, 31t cusp fracture, 111
for older patients, 411–412 split tooth, 119
system of, 85f vertical root fracture, 122
Cast gold onlay, 291f Clinical findings, with luxation injuries, 175t
Castings, misoriented, 411f Clinical tests
Cast metal post, 294 blood flow determination, 78
Caulking and putty materials, 235f cold and heat tests, 77
Causative factors control teeth, 76–77
for discoloration, 392–394 electrical pulp testing, 77–78, 166, 175–176
for failed root canal treatment, 381–382 significance of thermal tests, 79
for flare-ups, 159 Cocci in endodontic infections, 42t, 44f
for pain, 53 Cold flowable injection system, 318f
for postobturation emergency, 160 Cold stimuli
Cavity pain due to, 54
oral, irritants from, 299 testing with, 77, 175–176
preparation of, 23–25 Collagen
root-end, preparation and filling, 366–367 bundles, 5
routine access, 280–281 pulpal, 10
test Colonization, bacterial, 44
dentin stimulation with, 77 Color coding of file handles, 209
in older patient, 410 Combination technique, 271, 274–276
toilet of, 236 Communication between pulp and periodontium, 94–96
Cell body, odontoblast, 8–9, 11f Compaction
Cellulitis, 156 lateral, 308–311, 312f
Cementation, 25 ultrasonic, 311
Cementodentinal junction, 6, 94–95 vertical, 314–317
Cementoenamel junction, 18 Composite restorations, discoloration and, 395
Cements, 26–27 Computer-controlled injection device, 131f
Cementum, 4f Concussion, luxation injury, 174
cellular, 367f Condensing osteitis, 61, 62f, 80f, 84, 196f
posteruptive deposition of, 5–6 Cone-image shift, 187
types of, 17–18 disadvantages, 192–195
Central core theory, 134f image shift, 190
Central sensitization, 72b indications and advantages, 191–192
Cervical loop, 3 SLOB rule, 190
Chemical irritants, 52 Cones
and discoloration, 401 alignment, in facial projection, 199
Chief complaint, 69, 164 custom, solvent-softened, 311, 313–314
history of, 71–72 gutta-percha, 303f
in older patient, 409 master cone, 189, 308–309, 310f
Chisel-type fracture, 171f silver
Chlorhexidine, 264, 279 exposed, 350f
Chloroform, 348 removal of, 349, 352f
Chronic apical abscess, 62–63, 84 Conservation of tooth structure, 289–290
Chronic apical periodontitis, 59f Constriction, apical, 6, 223
Chronologic hypoplasia, 394 Continuous wave of condensation, 315
Circumferential filing, 267 Contraindications
Clamps for bicuspidization, 370b
designs, 232–233 for hemisection, 370b
placement, 235 medical, for anesthetics, 138
Clarity of cone-image shift, 192–193 for nonsurgical retreatment, 342
Cleaning for periapical surgery, 362–363
apical canal, 260–261 for root amputation, 370b
criteria for evaluating, 278–279 Control teeth testing, 76–77
instruments for, 205 Convenience form, for access, 236
for older patients, 414 Convergence, 16
principles of, 259 Core
Cleaning accidents obturating materials
aspiration or ingestion, 334 pastes, 304–305
creating artificial canal, 330–331 solid, 302–304
Index 461
Core (Continued) Defense, pulp role in, 7

removal of, 344 Deflection of needle, 133
systems, retention and, 294–295 Dendritic cells, 10, 53f
Coronal breakdown, extensive, 281 Dens evaginatus, 224, 225f
Coronal buildups, 233 Dens invaginatus, 224
Coronal fracture, and discoloration, 401 Density of obturation materials, 319
Coronal pulp Dental follicle, 2f, 5
removal of, 153f Dental history, 71, 151
stones in, 12f older patients, 409
Coronal restoration for vertical root fracture, 123
discoloration and, 395 Dental implant success rates, 381t
final, 353 Dental lamina, 2f
Coronal root perforations, 332–333 Dental papilla, 2f–3f
Coronal seal, 289, 299 Dentin
lack of, 383, 384f age-related changes in, 17
in older patients, 416 blushing of, 23
Coronal structure, replacement of, 233 bridge, 8f
Correction of obturation problems, 311 developing, 4f
Corrective surgery, 233–234, 369 etching of, 25
Corticosteroids, 279 formation, 3, 7, 219, 406f
Cracked tooth, 110t, 113–118 hypersensitivity, 16–17
Craze lines, 109, 110t, 111f nerves, 15–16
Cross-innervation, 133 primary and secondary, 6f
Cross-sectional anatomy, 218f reactionary, 8f
Crown regeneration, 34
access through, 239 structural changes in, 288
color changes with luxation injuries, 176 test cavity stimulation, 77
ferrules, 293 type I collagen in, 10
formation of, 3 Dentinal tubules, 24–25
fractures, 164b cocci in, 44f
with pulp exposure, 166–170, 181 communication via, 94–95
without pulp exposure, 166, 181 odontoblast aspiration into, 51f
full, 291f route of root canal infection, 38–39
lengthening, 234, 412f Dentition. See Tooth
preparation of, 23–25 Denture, fixed partial, 385
and odontoblast aspiration, 51f Depth of access
provisional, 233 calculation, 248f, 250f
provisional post, 281–282 measurement, 236
Crown-down technique, 273f Desiccation, by hygroscopy, 26
Crown-root fractures, 164b, 170–172, 181, 241f, 288f Desmosomal junctions, 8
Culturing canal contents, 302 Developmental defects, as cause of discoloration, 393–394
Curettage, periradicular, 365 Diagnosis
Curvatures. See also Anticurvature filing chief complaint, 69
canal, 383 cracked tooth, 114–117
determination of, 191 cusp fracture, 111–112
and prevention of a ledge, 329 differential. See Differential diagnosis
Curved flap, submarginal, 363–364 difficult, 82
Cusp fracture, 110t, 111–113 for emergencies, 150–152
Custom cones, solvent-softened, 311, 313–314 flare-ups, 159
Cyst health history, 69–72
apical, 59, 61f luxation injuries, 175–176
apical radicular, 84f objective examination, 74–79
collapse of cyst wall, 374f in older patients, 409–410
periapical, 99f pain referral phenomenon, 73–74
Cytotoxicity, 26 pulpal status in open apex, 29
radiographic examination, 79–81
D with radiography, 186
Débridement and referral of cases, 85–86
canal, 157 split tooth, 119–120
complete and incomplete, 159 transillumination, 82
inadequate, 301f traumatic injuries, 163–166
Decalcifying agents, 265 vertical root fracture, 123–124
Decompression, 374f Diagnostic radiographs, 188
Deep cavity preparation, 24f Diagnostic terminology, 70t
Deep-reaching clamps, 232–233 Differential diagnosis
Defects combined diseases, 99–101, 102f–103f
developmental, as cause of discoloration, 393–394 in older patients, 410–411
in files, 212f primary endodontic disease, 97–98
lingual groove, 226f primary periodontal disease, 98–99
462 Index
Differential diagnosis (Continued) Emergencies (Continued)

with radiography, 195–197 differentiation from urgency, 149–150
Digital radiography, 186, 201 instruments necessary for, 205
Dilaceration, 225 interappointment, 158–160
in apical third, 89f pain perception and pain reaction, 150
Direct exposure of pulp, 39 postobturation, 160
Direct pulp cap, 28 pretreatment, 152–158
Direct restoration, 290 treatment planning, 152
Disadvantages Enamel
of cone-image shift, 192–195 forming, hypoplasia of, 402
of lateral compaction, 308 fracture, 164b, 166
of pastes, 305 Endemic fluorosis, 393
of vertical compaction, 314–315 Endodontic disease
Disclosing solution, 116f differential diagnosis, 410
Discoloration effect on periodontium, 96
bleaching materials, 395–398 primary, 97–98
causes of, 392–394 Endodontic microbiology
complications of bleaching, 400–401 extraradicular infection, 40
crown, 176 intraradicular infection, 40
due to root canal obturation material, 354f microbial causation of apical periodontitis, 38
endodontically related, 394–395 routes of root canal infection, 38–40
extrinsic, 402–403 Endodontic microbiota, 40–46
following subluxation injury, 182f extraradicular infections, 45–46
intrinsic, 401–402 persistent/secondary endodontic infections, 44–45
nonvital bleaching techniques, 398, 400 primary intraradicular infections, 41–44
after root canal treatment, 314f Endodontic outcomes
Disinfection causes of failed root canal treatments, 381–383
endodontic instruments, 214 evaluation methodology, 377–379
operating field, 236 success and failure
Displacement, 165 definition of, 376–377
Disposable rubber dam systems, 231f predictors of, 380–381
Distal projection, 199 success rates, 379–380
Distolingual root, extra, 90f of treatments after failure of nonsurgical endodontics,
Drainage 383–388
following removal of necrotic debris, 153f when to evaluate, 377
incision for, 143, 155f, 357–359 Endodontics
shapes of rubber drains, 157f geriatric. See Geriatric endodontics
Drills importance of radiography in, 186–187
in access opening, 246–247 nonsurgical, outcomes, 383–388
Gates-Glidden, 209–210 pain issues in, 73b
in intraosseous injection, 137f Endodontic surgery
lentulo spiral, 209 in older patients, 415
Drug information web sites, 154b outcomes, 371–372, 384–385
Drugs, as cause of discoloration, 393, 401–402 recent advances in, 363
Dry cotton pellet, 153 Endodontist, 91–92
Dry heat sterilization, 214 Engine-driven instruments, 205
Drying of cavity, 24–25 Gates-Glidden drills, 209–210
Dye particles, 139f nickel-titanium files, 210
Dyscrasia, blood, 394 Peeso reamers, 210
rotary, 209
E Enlargement, apical
Ecchymosis, postsurgical, 415f degree of, 261
Ecology of endodontic microbiota, 43–44 final, 271, 275f
Ectomesenchyme, 2f Enostosis, 196f
Efferent blood vessels, 12 Eosinophils, 60f
Electrical pulp testing, 77–78, 166, 175–176 Epinephrine, 132–133, 135, 137
Electrodes, 78f–79f Epithelial cell rests of Malassez, 4, 19f
Electronic apex locators, 255–256 Epoxy, 307
Embryology of dental pulp Errors
early development of pulp, 2–3 in access
formation of excessive removal of tooth structure, 250–252
lateral canals and apical foramen, 4–5 inadequate preparation, 248–250
periodontium, 5 resulting in stripping perforations, 268f
root formation, 3–4 procedural, 265–266, 369
Embryonic stem cells, 9 Etching of dentin, 25
Emergencies Ethyl chloride, topical, 358
and case referral, 90 Ethylenediamine tetraacetic acid (EDTA), 265
diagnosis, 150–152 Etidocaine, 133
Index 463
Etiology F film, 189

cracked tooth, 114 Fibroblasts, 10
cusp fracture, 111 Files
elimination of, 261–262 in apical canal space, 260f
overfilling, 335–336 covered with sealer, 307f
split tooth, 119 defects in, 212f
underfilling, 334 Hedstrom, 207f, 347f
vertical root fracture, 122–123 inspected for fluting, 333f
Eugenol, in ZnOE, 26, 304, 306–307 intracanal usage, 210–211
Evaluation master apical, 275–276
of cleaning, criteria for, 278–279 nickel-titanium, 207–208
of endodontic outcomes, methodology, 377–379 engine-driven, 210
of facial skeleton, 165 placed beyond radiographic apex, 262f
of healing, 187 in step-back preparation, 269f–270f
length of follow-up period and, 377 Filing, 266–267
of obturation, 318–319 anticurvature, 268, 270, 271f
pretreatment, 262 Filling, root-end cavity, 366–367
of shaping, criteria for, 278–279 Film-cone placement, 197–199
Examination Final restoration, and internal bleaching, 398,
clinical, 377 400
in emergencies, 151–152 Finger spreaders, 310f
histologic, 379 First aid for avulsed teeth, 177b
instruments for, 204–205 Fixed partial denture, 385
luxation injuries, 175–176 Flap
objective design
clinical tests, 76–79 full mucoperiosteal flap, 364
extra- and intraoral, 74–76 submarginal curved flap, 363–364
periodontal, 79 submarginal triangular and rectangular flap,
for vertical root fracture, 123 364
radiographic. See Radiographic examination reflection, 125f, 143, 346f
traumatic injuries, 163–166 replacement and suturing, 367
Excavation of caries, stepwise, 28 Flare-ups
Explorer, 205f interappointment, 158–160
Exposure posttreatment, 352–353
of pulp Flexibility of instrumentation, 207
crown fractures with, 166–170, 181 Flowable gutta-percha, 317
crown fractures without, 166, 181 Fluctuant abscess, localized, 156f
direct, 39 Fluorosis, endemic, 393
radiographic, 190 Flute design, 348f
External root resorption, 400 Follow-up care, 160
Extraction to nonsurgical retreatment, 353
as alternative, 84 Four-rooted maxillary molars, 431f
socket, 139f Fractures
without replacement, 385 of alveolar process, 164b, 180
Extraoral examination, 74 coronal, and discoloration, 401
Extraoral radiographic technique, 201f crown, 164b, 166–170
Extraradicular infection, 40, 45–46 crown-root, 164b, 170–172, 181, 241, 288f
Extrinsic discolorations, 402–403 cusp, 110t
Extrinsic fiber cementum, primary acellular, 17–18 enamel, 164b, 166
Extrusion horizontal apical, 362
of irrigant, 334 of instrument, 266, 267f
orthodontic, 234 longitudinal. See Longitudinal fractures
Extrusive luxation, 175 originating occlusally, 109f
and referral of cases, 88, 90
F root, 164b, 172–174
Fabrication of instruments, 206 during root canal treatment, 289f
Facial projection, 217 vertical root, 110t, 301, 336, 382f
cone alignment, 199 characteristics of, 124
Facial skeleton evaluation, 165 Fragments
Faciolingual locations, 192 instrument, removal of, 345–347
Failure separated enamel-dentin, reattachment of, 166
coronal leakage and, 378f tooth
definition of, 376–377 bonded temporarily, 172f
of healing, 91 embedded in lip, 165f
of nonsurgical endodontics, 383–388 Full crown, 291f
predictors of, 380–381 Full mucoperiosteal flap, 364
related to deficiencies in obturation, 299–301 Fungi, in endodontic infections, 42
Ferrules, 293 Furcation perforation, 325, 326f
464 Index
G Healing (Continued)
Gates-Glidden burs, 348, 350f failure, 91
Gates-Glidden drills, 209–210, 268, 270, 272f in older adults, 408, 415
Gender differences, in pain, 131 periapical lesions after root canal treatment, 63–64
Geriatric endodontics after periapical surgery, 368–369
bleaching, 415–416 after retreatment, prognosis, 353–354
coronal seal, 416 Health history
diagnostic procedure, 409–410 dental, 71
differential diagnosis, 410–411 health and medical, 69–71
difficult-to-restore dentitions, 406f history of present complaint, 71–72
endodontic surgery, 415 Heat
healing, 408 pain due to, 54
impact of restoration, 414–415 produced during cavity/crown preparation, 23–24
medically compromised patients, 408–409 on setting of luting cements, 26
overdenture abutments, 416 testing with, 77
periradicular response, 408 Heating device for plugger, 316f
pulp response, 407–408 Hedstrom file, 207f, 347f
radiographic findings, 410 Hemisection, 369–372, 372f
retreatment, 415 Hemorrhage, 324–325
root canal treatment, 412–414 intrapulpal, as cause of discoloration, 392
trauma, 416–417 Hemostat, for grasping film, 198f
treatment planning and case selection, 411–412 Hertwig’s epithelial root sheath, 3, 4f
Giant cells, 60f High fever, and tooth discoloration, 394
Gingiva clamping, 233 High pulp horns, 224
Gingivectomy, 233–234 Histologic examination, 379
Glass ionomer History of present illness, 71–72, 164
cements, 27 Horizontal root fractures, 173f, 362
gutta-percha impregnated with, 317–318 Hyaline layer of Hopewell-Smith, 3–4
sealers, 307 Hydrochloric acid-pumice microabrasion, 402–403
Glick No. 1, 205f Hydrogen peroxide, 396–397
Gram-negative bacteria, 41–42 Hygroscopy, desiccation by, 26
Gram-positive bacteria, 42 Hyperplastic pulpitis, 55f
Granulation tissue, 169f Hypersensitivity
Granuloma, periapical, 59 dentin, 16–17
Greenstick fracture, 114f postrestorative, 25
Grossman’s formulation, 307 I
Ground-twisted instruments, 207f
Guide sleeve, in intraosseous injection, 137f Iatrogenic discolorations, 394
Gutta-percha Iatrogenic effects on pulp
advantages of, 303 amalgam, 27
carrier-based systems, 317 cavity/crown preparation, 23–25
composition and shapes, 302–303 dental materials, 25–26
lateral compaction, 308–311 depth of preparation, 26
master cone radiograph, 189 glass ionomer cements, 27
methods of placement, 303–304 local anesthesia, 22–23
new techniques and materials, 317–318 polycarbonate cement, 27
removal of, 292, 347–349 restorative resins, 27
sealability, 303 ZnOE, 26
selection of technique, 308 ZnOP, 26–27
solvent-softened custom cones, 311–314 Ice test. See Carbon dioxide ice testing
vertical compaction, 314–317 Immune complexes in periapical lesions, 58f
Immune system
cells of, 10
H
responses to pulpal pathoses, 52–53
Halides, 279b Immunoglobulins
Hand-operated instruments, 205, 206f IgE, 52
clinical use, 207 IgM, 53f
ground-twisted, 206 in inflamed periapical lesions, 58
intracanal usage, 210–211 Impact injuries, 51
machined, 206 and referral of cases, 88
in removal of silver cone, 351f Impedance apex locator, 255f
Hard pastes, removal of, 349, 352 Implants
Hard tissue endosseous, 387f
changes, caused by pulpal inflammation, 55–56 issue of success for, 388
healing, after periapical surgery, 368–369 single tooth, 385, 388
Healing success rates, 381t
compromised, 340 Impressions, 25
evaluating, 187 Inadequate access preparation, 248–250
Index 465
Incidence Instrumentation (Continued)

cracked tooth, 113 and elimination of etiology, 261–262
cusp fracture, 111 Instruments
interappointment emergency, 158 for bleaching, 213
longitudinal tooth fracture, 108–109 for cleaning and shaping, 205
split tooth, 118–119 for emergencies, 205
vertical root fracture, 122 engine-driven, 209–210
Incision for examination, 204–205
for drainage, 143, 155f, 357–359 fabrication, 206
and reflection, 364–365 fracture of, 266
Incisors fragments, removal of, 345–347
access openings and canal location, 239–242 hand-operated, 206–207
crown-root fracture, 172f intracanal usage
fractured, with pulp exposure, 168f hand instruments, 210–211
immature, trauma to, 164f rotary instruments, 211–212
mandibular central and lateral, 245–247, 426f nomenclature for, 205–206
maxillary, 420f for obturation, 212–213
with necrotic pulp, 30f physical properties, 207–208
Indications. See also Contraindications separated
for bicuspidization, 370b avoidance of, 211–212
for cone-image shift, 191–192 blockage by, 90
for hemisection, 370b prevention and prognosis, 333
for nonsurgical retreatment, 341–342 treatment outcome and, 383
for periapical surgery, 359–362 unretrieved, 342
for root amputation, 370b standardization, 208–209
for supplemental anesthesia, 135 sterilization and disinfection of, 213–214
Indirect restorations, 290–291 ultrasonic. See Ultrasonic instruments
Infection variations, 209
extraradicular, 40, 45–46 Insulating effect of bases, 27–28
intraradicular, 40 Interappointment emergency, 149, 158–160
persistent/secondary endodontic, 44–45 Internal anatomy
primary intraradicular, 41–44 alterations in, 218–220
resorption related to, 178 components of pulp system, 220–223
root canal, 38–40 general considerations, 217–218
Infiltration injections, 133, 358 methods of determining pulp anatomy, 217
Inflammation older patients, 413
irritant-induced, 50f variations of root and pulp anatomy, 224–227
localized, 23f Internal bleaching techniques
pain resulting from, 149 final restoration, 398, 400
periradicular, 51f future rediscoloration, 400
process of, 52 thermocatalytic, 398
pulpal, 22f, 55–56 walking bleach, 398
tissue, 130 when to bleach, 400
vascular changes during, 13, 15 Internal resorption, 56, 220f, 317f
Inflammatory resorption, 86 Interpretation of radiograph, 195
in avulsions, 178, 180f Intracanal medicaments, 279
Infraorbital block, 134 discoloration and, 395
Ingestion of instruments, 334 for older patients, 414
Injection Intracanal resorption, 56
alternative locations, 133 Intraoral examination
intraosseous, 135–138 dentition, 75–76
of obturating pastes, 305 soft tissue, 74–75
painless, 130–131 Intraosseous anesthesia, 135–138
primary, in older patients, 412–413 Intrapulpal hemorrhage, as cause of discoloration, 392
slow, 131, 358 Intrapulpal injection, 141–142
thermoplasticized, 315, 317 Intraradicular infection, 40
Injuries Intrinsic discolorations, 401–402
classification of, 164b Intrinsic fiber cementum
pulp response to, 407–408 primary acellular, 17–18
traumatic. See Traumatic injuries secondary cellular, 18
Injury response, 15 Intrusive luxation, 175, 182–183
pulpal, 81f Irreversible pulpitis, 54–55, 84, 142
Innervation painful, 152–154
accessory, 133 Irrigants
periodontium, 19 extrusion of, 334
pulpal, 15–17 ideal, properties of, 263b
Instrumentation Irrigation
confined to root canal system, 155f with chlorhexidine, 264
466 Index
Irrigation (Continued) Liners, 27

and degree of apical enlargement, 261–262 Lingual groove, 224–225
with sodium hypochlorite, 156, 263–264 defect, 226f
Irritants Lip numbness, 132–133
and alterations in anatomy, 219 Local anesthesia
chemical, 52 conventional, 132
and discoloration, 401 difficulties, 134–135
mechanical, 51 effect on pulp, 22–23
microbial, 49–51 initial management, 130–132
remnants of apical seal in canal, 299 management of
removal by canal débridement, 157 asymptomatic pulp necrosis, 143
Isolation irreversible pulpitis, 142
corrective surgery, 233–234 symptomatic pulp necrosis, 142–143
disinfection of operating field, 236 mandibular, 132–133
in older patients, 413 maxillary, 133–134
replacement of coronal structure, 233 supplemental, 135–142
with rubber dams, 231–233, 235 for surgical procedures, 143
of teeth with inadequate coronal structure, 233 Longevity of root-filled teeth, 287–288
Longitudinal fractures
K categories, 109, 110t
K-type files, 207f, 208–209 cracked tooth, 113–118
craze lines, 109
L fractured cusp, 111–113
Lasers, heat generated by, 23–24 incidence, 108–109
Lateral canals split tooth, 118–122
communication via, 95 vertical root fracture, 122–126
formation of, 4 Lubricants, 264, 330
and length of obturation, 300–301 Luting material, heat on setting, 26
Lateral compaction, with gutta-percha, 308–311, 312f Luxation injuries, 164b, 174–176, 181–183
Lateral condensation, instruments for, 212–213 Lymphatics, 12, 14f
Lateral incisors, 420f Lymphocytes, 60f
access openings and canal location, 239–242
Lateral luxation, 175–176, 182 M
Lateral perforations, 332 Macrophages, 10, 60f
Lateral radiolucency, 337f Magnifier-viewer, 201f
Lateral root perforation, 325, 338 Management
Lateral seal, 299 of artificial canal, 330–331
Leakage of avulsions, 183
coronal, 378f of a ledge, 330
defective restoration, 394f local anesthesia, 130–132
rubber dam, 235 of necrosis, 142–143
Ledge in pretreatment emergency, 152–157
formation, 328–330 of traumatic injuries, 180–183
removal of, 345 Mandible
Ledging, 90 film-cone placement, 200f
Length of obturation, 299–300, 319 radiolucency, 197
Lengths of hand-operated instruments, 208 Mandibular anesthesia, 132–133
Lentulo spiral drills, 209, 210f for irreversible pulpitis, 142
Lesions Mandibular molars
and biopsy, 362 access to, 247–248
periapical non-restorable, 386f
classification of, 58–63 proximal view, 252f
healing after root canal treatment, 63–64 restored, 387f
mediators of, 57–58 three-rooted, 227f, 431f
radiographic examination, 79–80 Mantle dentin, 3
removal of, 343f Mast cells, 52f, 58
persistent, 91 Master apical file, 275–276, 277f
progression of, 53 Master cone, gutta-percha, 189, 308–309, 310f
radiolucent Maxilla
distinguishing characteristics, 195–196 film-cone placement, 200f
periapical, 64f–65f radiographs, 197
pulp necrosis with, 300f Maxillary anesthesia, 133–134
radiopaque, 196f for irreversible pulpitis, 142
resorptive bony, 222f Maxillary canines, 420f
true combined perio-endo, 104f access to, 242
Lidocaine, 132–135, 137 Maxillary central incisors, 420f
Ligation, 233 access openings and canal location, 239–242
Lightspeed instrument, 209, 211f Maxillary zygoma, malar process of, 192f
Index 467
Mechanical irritants, 51 Needles

Median raphe, 200f, 255f insertion for PDL injection, 138f
Mediators of periapical lesions, 57–58 placement
Medical contraindications accuracy of, 133
for anesthetics, 138 for effective irrigation, 264f
for periapical surgery, 362–363 in pulp opening, 141f
Medical history, 69–71, 151, 164, 409 small-gauge, 131
Medically compromised older patients, 408–409 Nerve block
Medicaments, intracanal, 279b incisive, at mental foramen, 133
discoloration and, 395 posterior superior, 134
for older patients, 414 Nerves
Mental foramen, 64f arborization of, 16f
Mepivacaine, 134, 137 in region of pulp core, 15f
Mesial projection, 193f, 199, 217 Neuroanatomy, pulpal and dentinal nerves, 15–16
Mesial radiolucency, 88f Neurons, second-order, 16, 72
Mesial root apex, 223f Neurovascular bundle, 373
Mesiobuccal canal preparation, 273f Nickel-titanium instruments, 206–208
Mesiobuccal root, 89f and elimination of etiology, 261–262
Mesiolingual canal, 246f engine-driven nickel-titanium files, 210
Metallic restorations, discoloration and, 395 rotary files, 276
Metamorphosis, calcific, 55f, 81f, 88f rotary preparation, 271
as cause of discoloration, 392, 393f torsional forces, 263
in older patient, 409f, 411f Nociceptive fibers, 72
Microabrasion, hydrochloric acid-pumice, 402–403 Nomenclature for instruments, 205–206
Microbial irritants, 49–51 Noncollagenous matrix, 10
Microbiota Nonendodontic periradicular pathosis, 64–65
endodontic, 40–46 Nonspecific mediators of periapical lesions, 57–58
in root canal-treated teeth, 45t Nonsteroidal anti-inflammatory drugs (NSAIDs), 154
Micro-computed tomography, 201 Nonsurgical retreatment
Microleakage, 25, 27 contraindications, 342
Microtube removal methods, 347 indications for, 341–342
Midroot perforations, 332 outcomes, 383–384
Mineral trioxide aggregate (MTA), 28, 32, 167, 170, 367, 372 posttreatment considerations, 352–354
Mixed fiber cementum, secondary acellular, 18 procedures for, 343–352
Mixing of sealers, 307 risks and benefits, 342–343
Mobility test, 79 treatment options, 340–341
Molars Number of appointments
mandibular, 429f–431f for older patients, 412
access to, 247–248 treatment planning and, 83
non-restorable, 386f Number of radiographs, 188
proximal view, 252f Numbness, lip, 132–133
restored, 387f Nutrients available to bacteria, 44
three-rooted, 227f, 431f Nutrition, supplied by pulp to dentin, 7
maxillary, 423f–425f
access to, 242, 244 O
four-rooted, 431f Objective examination
retention and core systems, 294–295 clinical tests, 76–79
Morphology extraoral, 74
bowling pin apical, 261f intraoral, 74–76
canals, 236, 237f periodontal, 79
dentin and pulp, 7 for vertical root fracture, 123
MTAD decalcifying agent, 265 Objective tests
Mueller burs, 239f, 245f cracked tooth, 115
Multicanaled mandibular premolar, 431f cusp fracture, 112
Obstructions, canal, removal of, 343–344
N Obturation
Necrotic pulp causes of failure, 299–301
and apical periodontitis, 61f evaluation of, 318–319
as cause of discoloration, 392 with gutta-percha, 307–318, 348–349
due to irritants, 49–51 instruments for, 212–213
incisors with, 30f, 240f materials
previously, with no swelling, 160 core obturating, 302–305
root canal treatment for, 84 and discoloration, 394
symptomatic, 142–143 objectives of, 298–299
symptoms and treatment, 56 in older patients, 414
and timing of obturation, 301–302 radiographs, 189
without and with swelling, 154–157 sealers, 305–307
years after restoration, 295f timing of, 301–302
468 Index
Occlusal load, 288 Patency, apical, 262

Odontoblast layer, 2f, 3 Pathogenesis
capillary within, 14f cracked tooth, 113
Odontoblasts cusp fracture, 111
aspiration of, 51f split tooth, 119
cell body, 11f vertical root fracture, 122
characteristics of, 7–9 Pathogens, in diseased pulp, 97
displacement of, 24f, 25 Patient confidence, 130
pain mediator role, 17f Patient instructions
role in dentin formation, 7 postoperative, 367–368
Old metallic restorations, removing, 25 after traumatic injuries, 183
Open apex Patient symptoms, and timing of obturation, 301
apexification, 32–34 Peeso reamers, 210, 270t
apexogenesis, 31–32 Perception of pain, 150
diagnosis and case assessment, 29 Percussion, 76
replantations, 177–179 in identification of periodontal injury, 165
tissue engineering, 34 sensitivity, 154
treatment planning, 29, 31 Perforation
Operative causes, for failed root canal treatment, 381–382 during access preparation, 322–328
Oral cavity, irritants from, 299 apical, 266, 332, 336f
Orthodontic extrusion, 234 crown, 91
Osteitis, condensing, 61, 62–63, 80f, 84, 196f of post preparation, 193f
Osteoectomy, 365 resorptive, 369
Osteoporosis, 408 root, 91, 332–333
Outcomes site selection, 136–137
causes of failed root canal treatments, 381–383 strip, 96f
endodontic surgery, 371–372 Perforator breakage, 136
evaluation methodology, 377–379 Periapical lesions, 50f
success and failure classification of, 58–63
definition of, 376–377 healing after root canal treatment, 63–64
predictors of, 380–381 mediators of, 57–58
success rates, 379–380 radiographic examination, 79–80
of treatments, after failure of nonsurgical endodontics, removal of, 343f
383–388 Periapical pathosis, 56–58
when to evaluate, 377 Periapical surgery
Outline form, for access, 236, 241f–245f contraindications, 362–363
Overdenture abutments, 416, 417f healing, 368–369
Overfill, 299–300 indications, 359–362
etiology, 335–336 procedures, 363–368
Oxygen tension, 43–44 recent advances in, 363
Periodontal disease, 39
P effect on pulp, 96–97
Pain primary, 98–99
allodynia phenomenon, 130 Periodontal examination, 79, 151
causative factors, 53 Periodontal ligament (PDL), 4f, 18–19
dentinal, 16–17 injection, 138–141
gender differences, 131 perforation into, 324–325
as indication for retreatment, 341 thickened PDL space, 59f
from inflammation, 149 Periodontitis, apical
with intrapulpal injection, 141 asymptomatic, 59–61, 84
of irreversible pulpitis, 54 microbial causation of, 38, 39f
perception of, 150 persistent, 45t
persistent, 91, 360, 362 symptomatic, 58–59, 84, 153–154
psychogenic, 86b Periodontium
pulpal, 7 communication with dental pulp, 94–96
referred, 69, 72b, 73–74, 73b formation of, 5
what it is, 72b safety, with PDL injection, 140–141
Painless injections, 130 vasculature of, 19
Palpation, 76, 151 Peripheral pulp organization, 9f
Paralleling devices, for working films, 198 Periradicular curettage, 365
Parallel radiographs, 252 Periradicular pathosis, nonendodontic, 64–65
Parulis. See Stoma Periradicular surgery, 143
Passive step-back technique, 268, 271f, 330 Periradicular tissues
Pastes alveolar bone, 19–20
advantages and disadvantages, 305 cementoenamel junction, 18
soft and hard, removal of, 349, 352 cementum, 17–18
techniques of placement, 305 periodontal ligament, 18–19
types, 304–305 Persistent intraradicular infection, 40
Index 469
Persistent/secondary endodontic infections, 44–45 Pretreatment evaluation, 262

Phenolics, 279b Prevention
Phylotypes, bacterial, as-yet-uncultivated, 42 of cracked tooth, 118
Physical limitations, 86 of cusp fracture, 113
Pins of flare-ups, 159
effects on pulp, 25 of a ledge, 329–330
retentive, 295 of perforations during access preparation, 323–324
Plastics, 304–305, 307 of separated instruments, 333
Pluggers, 212–213, 315, 316f of split tooth, 122
for gutta-percha, 308 of vertical root fracture, 126
Polishing restorations, 25 Previously treated pulp, 56
Polycarboxylate cement, 27 Prilocaine, 134
Polymorphonuclear leukocytes (PMNs), 49, 60f Primary endodontic disease, 97–98
Porcelain veneer, 400f with secondary periodontal involvement, 99, 102f–103f
fused to metal, 414–415 Primary intraradicular infection, 40
Positioning device, 254f Primary periodontal disease, 98–99
Post with secondary endodontic involvement, 99–101, 103f
carbon fiber, 291, 292f Probing
cemented, 341f for cracked tooth, 117
perforation, 193f periodontal, 75f, 79
prefabricated, 294–295 in primary endodontic disease, 97–98
provisional crowns with, 281–282 Procedural accidents, 90–91
removal system, 344f–345f during cleaning and shaping, 328–334
selection of, 291 as indication for periapical surgery, 360
space preparation, 291–293 during obturation, 334–336
accidents during, 336–337 perforations during access preparation, 322–328
Posterior superior nerve block, 134 during post space preparation, 336–337
Posterior teeth, retention and core systems, 294–295 Profound anesthesia, in emergencies, 152
Postobturation emergency, 149, 160 Prognosis
Postoperative care and instructions, 367–368 artificial canal creation, 331
Predentin, 3, 11f for bicuspidization, 370–371
Predictors of success and failure, 380–381 cracked tooth, 118b
Premolars cusp fracture, 113
mandibular, 427f–428f for healing after retreatment, 353–354
access to, 247 for hemisection, 370–371
multicanaled, 431f for ledge formation, 330
maxillary, 421f–422f primary endodontic disease, 98
access to, 242 with secondary periodontal involvement, 99
three-rooted, 431f primary periodontal disease with endodontic involvement,
receded pulp space, 250f 101
with three canals, 226f–227f for root amputation, 370–371
Preodontoblasts, 9 for separated instruments, 333
Preoperative causes, for failed root canal treatment, 381–382 split tooth, 121–122
Preoperative radiograph, parallel, 254f teeth after perforation repair, 327–328
Preparation techniques, 265–278 treatment in older patients, 411–412
anticurvature filing, 268, 270 true combined diseases, 101
balanced force technique, 270–271 vertical root fracture, 126, 337
circumferential filing, 267 Prophy cup, 78f
combination technique, 271, 274–276 Prophylaxis. See Antibiotic prophylaxis
filing, 266–267 Protection
final apical enlargement and clearing, 271 of pulp, 21, 22t
general considerations, 276–278 of remaining tooth structure, 290
nickel-titanium rotary preparation, 271 with rubber dams, 231
passive step-back technique, 268 Provisional post crown, 281–282
reaming, 266 Psychogenic pain, 86b
recapitulation, 271 Pulp
for root-end cavity, 366–367 age-related changes in, 17
standardized preparation, 267 anatomic regions, 5–7
step-back technique, 267–268 blood vessels, 11–15
step-down technique, 268 capping, 22
watch winding, 266 cavity, 221f
Pressure sterilization, 214 cells of, 7–10
Pretreatment emergency, 149 communication with periodontium, 94–96
management of embryology of, 2–5
painful irreversible pulpitis, 152–154 exposure, 39, 166–170, 181
pulp necrosis with apical pathosis, 154–157 extracellular components, 10–11
postoperative instructions, 157 function and morphology, 7
profound anesthesia, 152 iatrogenic effects on, 22–27
470 Index
Pulp (Continued) Pulp space, 5, 6f, 250f

injury to, 166 age-related decrease in size, 407
innervation, 15–17 histologic section of, 410f
irritants of, 49–52 visibility of, 416f
lesions, radiographic examination, 80–81 Pulp testing
pathosis, 52–53 with cold, 77, 175–176
anesthetic management of, 142–143 electrical, 77–78, 166, 175–176
periodontal disease effect, 96–97 with heat, 77
previously vital, débridement, 159 in older patient, 409–410
protection of, 21 Pulp therapy. See Vital pulp therapy
from effects of materials, 27–28 Pulp vitality tests, 64, 151
regeneration, 34
safety, with PDL injection, 141 R
selection of appropriate test, 76–77 Radiographic examination
stones, 10–11, 12f–13f, 81, 219 for cracked tooth, 115
vasculature, 13f in emergency, 152
vitality tests, 76 for luxation injuries, 176
Pulpal anesthesia, 132–133 in older patients, 410
Pulpal diseases and outcome evaluation, 377, 379
hard tissue changes caused by pulpal inflammation, 55–56 periapical lesions, 79–80
irreversible pulpitis, 54–55 in pretreatment evaluation, 262
previously treated pulp, 56 pulpal lesions, 80–81
pulpal necrosis, 56 root fracture, 172, 173f
reversible pulpitis, 53–54 for split tooth, 119
Pulpal tissue remnants, and discoloration, 394–395, 397f in traumatic injury, 166
Pulp anatomy for vertical root fracture, 123
alterations in, 218–220 Radiography
components of pulp system, 220–223 cone-image shift, 190–195
general considerations, 217–218 determination of working length, 252, 254–255
methods of determining, 217 differential diagnosis, 195–197
variations in, 224–227 digital, 186, 201
Pulp chamber exposure considerations, 190
anatomy, 86 importance in endodontics, 186–187
calcific metamorphosis, 88f micro-computed tomography, 201
changes in shape, 6f obturation verification radiograph, 311, 319
C-shaped, 226f pre- and postoperative, 359f
disclike configuration, 219f, 407f radiographic anatomy, 195, 217
receded, 242f radiographic sequence, 188–190
shape, 220 special techniques, 197–201
Pulp horns, 5 Radiolucent lesions
anatomy, 220 distinguishing characteristics, 195–196
exposed, 54f periapical, 64f–65f
high, 224 pulp necrosis with, 300f
location, 419f resolution of, 385f–386f
Pulpitis Radiopacity of sealer, 306
hyperplastic, 55f Radiopaque lesions, 196f
irreversible, 54–55, 142 Rapid processing, of working films, 199
reversible, 53–54 Reamers, 207
treatments for, 84 engine-driven, 210
Pulp necrosis intracanal usage, 210–211
with apical pathosis, 154–157 Peeso, 210
and apical periodontitis, 61f Reaming, 266
as cause of discoloration, 392 Reattachment, of separated enamel-dentin fragment, 166
due to irritants, 49–51 Recall evaluation, 379f
incisor with, 30f, 240f Recall radiograph, 187, 188f, 189–190
previous condition of, with and without swelling, 160 Recapitulation, 271
with radiolucent lesions, 300f Recorcinol obturations, 354f
after restoration, 295f Rectangular flap, submarginal, 364
root canal treatment for, 84 Rediscoloration, 400
symptomatic and asymptomatic, 142–143 Redox potential, 43–44
symptoms and treatment, 56 Referral of cases, 84–92
and timing of obturation, 301–302 for bleaching, 403
Pulpotomy, 22t, 28, 153f, 169f for combined diseases, 104
shallow, 170f conditions indicating, 372–373
Pulp polyp, 55f determining difficulty of case, 85
Pulp response general reasons for, 87–90
changes with age, 407 in middle of treatment, 91
to injury, 407–408 procedural accidents, 90–91
Index 471
Referral of cases (Continued) Retreatment (Continued)

procedural difficulties, 86–87 procedures for, 343–352
treatment planning considerations, 85–86 risks and benefits, 342–343
Referred pain, 69, 72b, 73–74, 73b treatment options, 340–341
Reflection, incision and, 364–365 in older patients, 415
Refrigerant, 78f of treated tooth, 88
Removal of tooth structure, excessive, 250–252 Reversible pulpitis, 53–54, 84
Reparative dentin, 8f Risks and benefits, of nonsurgical retreatment, 342–343
Replacement resorption, 86, 178–179, 180f Rods in endodontic infections, 42t
Replantation Root
in avulsions, 177–179 amputation, 369–372
intentional, 385 anatomy, 218
Repositioning, in root fracture, 172, 174 variations in, 224–227
Resection, root-end, 365, 366f after cleaning and shaping, 259f
Resins development after pulpotomy, 29f
cores, 304 extra distolingual, 90f
restorative, 27 extrusion, 100f
Resorption formation, 3–4
ankylosis-related, 178–179, 180f fracture, 164b, 172–174, 181, 301, 382f
cervical external, 406f identification of, 218
external root, 400 maxillary first premolar, 221f
of hard tissue, 56f mesiobuccal, 89f
internal, 220f, 317f perforations, 332–333
in luxation injuries, 176 resorption, external, 400
and referral of cases, 86 Root canal
surface, 178 anatomic regions of, 5f, 220–221
treatment complicated by, 262 curvature, 87
Restoration, preparation for filling stage, bacteria at, 45
coronal seal, 289 irretrievable materials in, 360
design of restoration, 289–291 routes of infection, 38–40
longevity of root-filled teeth, 287–288 Root canal treatment
preparation of canal space and tooth, 291–293 for crown fractures, 170
restoring access through existing restoration, 295 failed, 361f
retention and core systems, 294–295 causes, 381–383
structural and biomechanical considerations, 288–289 following replantation, 179–180
timing of restoration, 289 for older patients, 412–414
Restorations for root fracture, 174
adequate, requirements for, 289 success rates, 381t
after apexification, 33–34 Root-end resection, 365, 366f
coronal Rotary flaring instruments, 210t
discoloration and, 395 diameter of, 270t
final, 353 Rotary instruments, 209
cracked tooth, 118 avoidance of instrument separation, 211–212
cusp fracture associated with, 112f Routine access cavities, 280–281
design of, 289–291 Rubber dams
existing application, 231
and referral of cases, 87–88 leakage, 235
restoring access through, 295 placement, 235
final, and internal bleaching, 398, 400 preparation for placement, 233
final coronal, 353 retainers, 231–233
impact on older patients, 414–415 use of, 323–324
and nonsurgical retreatment, 342 Rushton bodies, 99f
polishing, 25
removal of restorative materials, 237, 239, 343 S
temporary. See Temporary restorations Safety
Retainers, rubber dam, 231–233 of bleaching, 400–401
Retention of microabrasion, 403
and core systems, 294–295 Same lingual, opposite buccal (SLOB), 190, 192
of coronal restoration, 290 Sclerotic bone, 196f
of restorative materials, 343 Sealability of gutta-percha, 303
Retreatment Sealers
due to periodontitis, 315f desirable properties, 305–306
improved techniques of, 97 extrusion of, 96f
nonsurgical mixing and placement, 307
contraindications, 342 types, 306–307
indications for, 341–342 Secondary intraradicular infection, 40
outcomes, 383–384 Second division block, 134
posttreatment considerations, 352–354 Second-order neurons, 16, 72
472 Index
Sectional obturation, 315 Staining

Segments external and internal, 416
separability of, 116, 117f by sealers, 306
of split tooth, 120–121 Standardization, of hand-operated instruments, 208
Selective anesthesia, 82 Standardized preparation, 267
Semisolids, 304–305 Stem cells, 9
Sensitization, central, 72b Step-back preparation, 276
Separated instruments Step-back technique, 267–268, 269f
avoidance of, 211–212 Step-down technique, 268
blockage by, 90 Steps in combination technique, 274b
prevention and prognosis, 333 Steps in obturation
treatment outcome and, 383 with gutta-percha, 309–311
unretrieved, 342 with solvent-softened custom cones, 313–314
Sequelae Sterilization, endodontic instruments, 213–214
to replantation, 178–180 Sterilizers, of dentin, 52
of root fractures, 174 Steroids, 279b
Sequence of diagnosis in emergencies, 151b Stoma, sinus tract, 63f, 75f
Setting time of sealer, 306 Stones, pulpal, 10–11, 12f–13f
Shallow cavity preparation, 24f Stopping, temporary, 236
Shaping Straight-line access, 268f, 272f, 383, 419f
apical canal, 260–261 Stripping perforations, 266, 268f, 325, 327f
criteria for evaluating, 278–279 Subluxation, 174–175
instruments for, 205 Submarginal flap
for older patients, 414 curved, 363–364
principles of, 259–260 triangular and rectangular, 364
Shaping accidents Subodontoblastic capillary plexus, 14f
aspiration or ingestion, 334 Subodontoblastic plexus of Raschkow, 15, 16f
creating artificial canal, 330–331 Substance P, 52
extrusion of irrigant, 334 Success
ledge formation, 328–330 definition of, 376–377
root perforations, 332–333 periodontal lesion, 378f
separated instruments, 333 predictors of, 380–381
Sharpey’s fibers, 5 rates, 379–380
Shrinkage Sulcular flap, 364
of gutta-percha, 303 Superimposed structures, with cone-image shift, 190, 193–194
of sealer, 306 Supplemental anesthesia
Silver cones anesthetic agents, 135
exposed, 350f indications, 135
removal of, 349, 352f intraosseous, 135–138
Silver points, 304 intrapulpal injection, 141–142
Sinus tract PDL injection, 138–141
extraoral, 74f Supporting tissue examination, 165
opening, 341f Surface resorption, 86
Sizing of hand-operated instruments, 208 Surgery
Smear layer removal, 25, 264–265 bicuspidization, 369–372
Sodium hypochlorite, 156, 263–264, 336f, 397–398 conditions indicating referral, 372–374
Sodium perborate, 397 corrective, 233–234, 369
Sodium peroxyborate monohydrate, 397 endodontic
Soft pastes, removal of, 349, 352, 353f in older patients, 415
Soft tissue outcomes, 371–372, 384–385
examination, 74–75, 165 recent advances in, 363
healing, after periapical surgery, 368 hemisection, 369–372
Solubility of sealer, 306 incision for drainage, 357–359
Solvents, for gutta-percha removal, 348 periapical
Solvent-softened custom cones, 311, 313–314 contraindications, 362–363
Sonic instruments, 206, 348 healing, 368–369
Specific mediators of periapical lesions, 58 indications, 359–362
Spirilla in endodontic infections, 42t procedures, 363–368
Splinting recent advances in, 363
luxation injuries, 176 periradicular, 143
for root fracture, 173f–174f postoperative instructions, 158
Split tooth, 110t, 118–122 root amputation, 369–372
Spoon excavator, 205f Suturing, flap, 367, 368f
Spreaders, 212–213, 278f Swelling
finger vs. hand, 309f avoiding injection into, 143
precurved, 308f due to apical abscess, 62f
Stabilization, root fracture, 173f, 174 fluctuant, 358f
Index 473
Swelling (Continued) Tooth (Continued)

progressively spreading, 158f traumatic injury management, 180–183
pulp necrosis without and with, 154–157 root canal-treated, microbiota in, 45
severe mandibular, 150f single tooth implant, 385, 388
submental space, 153f structure
Symptomatic apical periodontitis, 58–59, 84 excessive removal of, 250–252
Symptomatic cases indicating surgery, 360, 362 loss of, 288
Symptomatic endodontic infections, 43 wiggling test, 151
Symptomatic pulp necrosis, 142–143 Topical anesthetic, 130–131
Syringe, for PDL injection, 140f ethyl chloride, 358
Torsional fatigue, 266, 267f
T Torsional limits, 208–209
Taper, 319 nickel-titanium instruments, 263
Techniques Transillumination, 82
for apexification, 33 for cracked tooth, 115
for apexogenesis, 31–32 Transmission pathways, pulp to CNS, 16
bicuspidization, 370 Transportation, apical, 266
for corrective surgery, 369 Traumatic injuries
gutta-percha, 308 alveolar fractures, 180
hemisection, 370 avulsions, 176–180
for placement of as cause of discoloration, 392f
pastes, 305 crown fractures
sealers, 307 with pulp exposure, 166–170
temporary material, 281f without pulp exposure, 166
of preparation, 265–278 crown-root fractures, 170–172
root amputation, 370 enamel fracture, 166
for vertical compaction, 315 examination and diagnosis, 163–166
Temporary restorations, 279–282 luxation, 174–176
crowns, 25 management in primary dentition, 180–183
extensive coronal breakdown, 281 in older patients, 416–417
long-term, 282 and referral of cases, 88
for missing tooth structure, 233 root fractures, 172–174
objectives of temporization, 280 Treatment. See also Retreatment
provisional post crowns, 281–282 cracked tooth, 117–118
routine access cavities, 280–281 cusp fracture, 113
Terminology, diagnostic, 70t flare-ups, 159
Test cavity furcation perforation, 327
dentin stimulation with, 77 luxation injuries, 176
in older patient, 410 options for nonsurgical retreatment, 340–341
Tetracycline, and discoloration, 393, 401f postobturation emergency, 160
Thermal tests, 77, 79 radiography role in, 186–187
Thermocatalytic bleaching, 398 root canal. See Root canal treatment
Thermoplasticized injection, 315, 317 split tooth, 120–121
Three-rooted maxillary premolar, 431f vertical root fracture, 124, 126, 337
Tight junctions, 8 Treatment planning
Timing crown-root fractures, 171–172
of obturation, 301–302 for emergencies, 152
of restoration, 289 following from diagnosis, 82
Tip design, 208 number of appointments, 83
Tissue engineering, 34 for older patients, 411–412
Tissue tolerance, of sealers, 305–306 open apex, 29, 31
T lymphocytes, 10, 58f and referral of cases, 85–86
Toilet of the cavity, 236 for specific conditions, 84
Tooth Triangular flap, submarginal, 364
cracked, 110t, 113–118 Trigeminal nerve, 15
early development, 2f Tubercle, fractured, 225f
examination of, 75–76, 165–166 Two-rooted mandibular canine, 431f
formation, defects in, 394 Two-stage injection, 131
fragments
bonded temporarily, 172f U
embedded in lip, 165f Ultrasonic compaction, 311
with inadequate coronal structure, isolation of, 233 Ultrasonic instruments, 206
location, and referral of cases, 87 for removing
loss, causes of, 287–288 gutta-percha, 348
orthodontic tooth movement, 27 hard pastes, 352
primary tips for preparing root-end cavities, 366f
and PDL injection, 141 used for cleaning and shaping, 263
474 Index
Underfill, 300 Warm vertical compaction, 315

etiology, 334 Watch winding, 266
Unroofing, 252f Web sites, for drug information, 154b
Urgency, differentiation from emergency, 149–150 Weeping canal, 156
Winged clamps, 232
V Working films, 188–189
Varnishes, 27 Working length
Vascular physiology, pulpal, 12–15 determination, 252–256
Vertical compaction, with gutta-percha, 314–317 films, 189
Vertical condensation, instruments for, 213 loss of, 266
Vertical incision for drainage, 358–359 in older patients, 413–414
Vertical root fracture, 110t, 122–126, 301, 336, 382f and prevention of a ledge, 330
Viewers, radiographic, 201
Viruses Z
correlation with apical pathoses, 51
Zinc oxide–eugenol (ZnOE), 26, 304, 306–307
in endodontic infections, 42–43
Zinc phosphate cement (ZnOP), 26–27
Visualization of internal anatomy, 237
Zipping, 266
Vitality tests, pulpal, 76
Zone of Weil, 7
Vital pulp therapy, 21–22, 22t, 28
for crown fractures, 167
Volumes of anesthetic solutions, 134
W
Walking bleach technique, 213, 396f, 398, 399b
Warming of anesthetic solution, 131
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Pulpal Anatomy and Access Preparations: Appendix A

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Pulpal Anatomy and Access Preparations: Appendix A

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APPENDIX A

Pulpal Anatomy and Access

Where dentin shelf

Maxillary Right Central Incisor

*Most common root curvatures

Maxillary Right Lateral Incisor

*Most common root curvatures

Maxillary Right Canine

*Most common root curvatures

Maxillary Right First Premolar

*Most common root curvatures

Maxillary Right Second Premolar

*Most common root curvatures L

Maxillary Right First Molar

3 canals: 40% 4 canals: 60%

*Most common root curvatures

Maxillary Right Second Molar

*Most common root curvatures

Maxillary Right Second Molar

*Most common root curvatures

Mandibular Right Central and Lateral Incisor

*Most common root curvatures

Mandibular Right Canine

*Most common root curvatures

Mandibular Right First Premolar

*Most common root curvatures

Mandibular Right Second Premolar

*Most common root curvatures

Mandibular Right First Molar

*Most common root curvatures

Mandibular Right Second Molar

*Most common root curvatures

Mandibular Right Second Molar

*Most common root curvatures L L L L

Some Uncommon Variations

Mandibular Maxillary four-rooted

1. Maxillary central incisor 2. Maxillary canine

3. Maxillary first premolar 4A. Three-canal maxillary molar

4B. Four-canal maxillary molar 5. Mandibular incisor

6. Mandibular canine 7. Mandibular premolar

8A. Three-canal mandibular molar 8B. Four-canal mandibular molar

Chapter Review Questions

CHAPTER 1 a. Root canal diameter increases in size.

c. periodontal disease b. are more resistant to antimicrobial treatments

8. What is reversible pulpitis? CHAPTER 5

9. How does electrical pulp testing determine the degree CHAPTER 6

8. What is the preferable restoration of a cracked 4. What is a two-stage injection?

c. patient management a. more common than flare-ups following cleaning

7. What is the emergency treatment of choice for pulp

7. Which is a stronger metal alloy: carbon steel or stain- CHAPTER 13

c. It is a projection of the internal tooth anatomy CHAPTER 15

b. if the patient is unable to pay for the restora- CHAPTER 17

4. Which of the following is not an early sign or indica-

CHAPTER 18 8. What type of canal is most prone to ledge

c. It may prevent healing of the incision. CHAPTER 21

c. coronal leakage 7. Which of the following is not an indication for inter-

c. remains the same in volume b. increased cementum deposition, modifying the

Aspiration of instruments, 334 Burs

Caries (Continued) Cleaning accidents (Continued)