UmApi DErmato Qbank
UmApi DErmato Qbank
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EPIDEMIOLOGY 0
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H. The differential diagnosis of bullous pemphigoid does not include linear dermatosis with Ig A
I. In pemphigus vulgaris, the vital prognosis without treatment is good
J. Linear dermatosis with Ig A is a chronic disease of the elderly patient
From the point of view of symptomatology, the following statements are true regarding bullous
dermatoses:
A. In pemphigus vulgaris, the oral mucosa is rarely affected
B. In vegetative pemphigus, the lesions are located in intertriginous areas
C. In pemphigus with Ig A, the lesions tend to coalesce and give a ringed appearance
D. In pemphigus with Ig A, the lesions are "target-like" and resemble erythema multiforme
E. In seborrheic pemphigus, the lesions are located in intertriginous areas
F. When the pemphigus vulgaris lesions are extensive, we can have hyperalbuminemia
G. The subepidermal bulla is found in Duhring Brocq's disease
H. The intraepidermal bulla is found in Duhring Brocq's disease
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From the point of view of symptoms, the following statements are true regarding bullous
dermatoses:
A. Dermatitis herpetiformis is an acute condition
B. The distribution of lesions "in a bouquet" is specific for Duhring Brocq's disease
C. Lesions from bullous pemphigoid are localized with predilection at the level of the flexural area
D. Lesions from bullous pemphigoid are localized with a predilection at the level of the extension areas
E. Among the therapeutic options for pemphigus are plasmapheresis or biological agents
F. The distribution of lesions "in a bouquet" is specific for pemphigus vulgaris
G. In paraneoplastic pemphigus, the lesions are "target-like" and resemble erythema multiforme
H. The therapeutic options for pemphigus do not include plasmapheresis or biological agents
I. Pemphigus with Ig A is a genodermatosis
J. Duhring Brocq's disease is a subepidermal, chronic, intensely pruritus condition
From the point of view of diagnosis, the following statements are true regarding bullous
dermatoses:
A. Vulgar warts are included in the differential diagnosis of bullous pemphigoid
B. Pemphigus vulgaris is produced by auto-antibodies directed against desmoglein 3
C. The cohesion of the dermis is ensured mainly by desmosomes
D. The circulating antibody titer in pemphigus vulgaris always correlates with the activity of the disease
E. The circulating antibodies titer in pemphigus vulgaris correlates with disease activity, but not always
F. Indirect immunofluorescence in pemphigus vulgaris shows anti-intercellular substance antibodies
G. Bullous epidermolysis is included in the differential diagnosis of bullous pemphigoid
H. Medicines cannot induce bullous pemphigoid
I. The cohesion of the dermis is secondarily ensured by desmosomes
J. Pemphigus vulgaris is produced by auto-antibodies directed against desmoglein 13
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From the point of view of diagnosis, the following statements are true regarding bullous
dermatoses:
A. Corticosteroids are the standard treatment in pemphigus
B. Indirect immunofluorescence highlights circulating serum Antibodies in autoimmune bullous
dermatoses
C. In dermatitis herpetiformis we can meet urticaria-like plaques
D. In dermatitis herpetiformis, the lesions are symmetrically distributed on the extension areas
E. Indirect immunofluorescence highlights Antibodies from lesional skin in autoimmune bullous
dermatoses
F. In dermatitis herpetiformis we find small bullae
G. In dermatitis herpetiformis, the lesions are symmetrically distributed on the felxia areas
H. Antibiotics are the standard treatment in pemphigus
I. In pemphigus vulgaris we can find urticaria-like plaques
J. In bullous pemphigoid we can find urticaria-like plaques
From the point of view of diagnosis, the following statements are true regarding bullous
dermatoses:
A. Pemphigus is a chronic condition
B. Cytodiagnosis can reveal autoantibodies
C. Untreated pemphigus vulgaris is fatal in 1-2 years
D. Cytodiagnostic can reveal acantholytic cells of the Tzanck type
E. In linear dermatosis with Ig A, the onset is the most common autoimmune bullous dermatosis
F. Untreated pemphigus vulgaris is marked by repeated bouts of lesions
G. In linear dermatosis with Ig A, the onset is usually after 40 years
H. Pemphigus is an acute condition
I. In vegetative pemphigus, the characteristic lesion is the vesicle
J. Linear dermatosis with Ig A is a rare autoimmune bullous dermatosis
From the point of view of diagnosis, the following statements are true regarding bullous
dermatoses:
A. Cicatricial pemphigoid mainly affects the elderly
B. Sulfones are not used in the treatment of dermatitis herpetiformis
C. In Duhring-Brocq disease, the vital prognosis is good
D. In Duhring-Brocq disease, the vital prognosis is reserved
E. Cicatricial pemphigoid never touches the mucous membranes
F. Cicatricial pemphigoid preferentially affects mucous membranes
G. Sulfones are used in the treatment of dermatitis herpetiformis
H. The autoimmune mechanism is not incriminated in bullous pemphigoid
I. Autoimmune mechanism is suggested in bullous pemphigoid
J. Cicatricial pemphigoid mainly affects children
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C. A rash similar to bullous pemphigoid can develop in patients with lichen planus
D. Rituximab (Antibodies anti CD-20) can be used in the treatment of pemphigus vulgaris
E. In bullous pemphigoid, eosinophils are not present in the liquid in the bulla
F. Rituximab (anti CD-20 antibody) is contraindicated in the treatment of pemphigus vulgaris
G. In pemphigus vulgaris under treatment we can obtain lasting remissions
H. In bullous pemphigoid, neutrophils are present in the fluid in the bulla
I. In bullous pemphigoid, eosinophils are present in the fluid in the bulla
J. In bullous pemphigoid, neutrophils are present in the infiltrate
Regarding treatment and paraclinical diagnosis, the following statements are true for bullous
dermatoses:
A. Mycophenolate mofetil is not a therapeutic option for pemphigus vulgaris
B. In bullous pemphigoid, mast cell degranulation near the basement membrane is excluded
C. In pemphigus vulgaris with only mucosal involvement, the ELISA test is positive for desmoglein 7
D. Mycophenolate mofetil is a therapeutic option for pemphigus vulgaris
E. Prolonged daily baths are contraindicated in pemphigus vulgaris
F. The decision to stop treatment in pemphigus is a strictly clinical one
G. Prolonged daily baths are helpful in removing crusts from pemphigus vulgaris
H. In bullous pemphigoid, mast cell degranulation is noted near the basement membrane
I. In pemphigus vulgaris with only mucosal involvement, the ELISA test is positive for desmoglein 3
J. The decision to stop treatment in pemphigus is clinical and histological
E. In dermatitis herpetiformis, the gluten-free diet can reduce the daily dose of dapsone
F. Bullous pemphigoid lesions tend to spread through the periphery
G. Bullous pemphigoid is a genodermatosis
H. Thyroid disorders are not found in patients with dermatitis herpetiformis
I. Thyroid disorders are more common in patients with dermatitis herpetiformis
J. In the differential diagnosis of dermatitis herpetiformis enters eczema
From a paraclinical point of view, the following statements are True regarding autoimmune
bullous dermatoses:
A. In bullous pemphigoid, the C3 fraction can be detected in almost all patients
B. Tzanck cytodiagnosis is an immunofluorescence method
C. The Tzanck cytodiagnostic shows acantholytic cells in pemphigus vulgaris
D. Cytology or Tzanck cytodiagnosis is done by scraping the bottom of a bulla
E. Complications of systemic corticosteroid therapy in the elderly are very rare and insignificant
F. Complications of systemic corticosteroid therapy in the elderly can be severe
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From a paraclinical point of view, the following statements are True regarding autoimmune
bullous dermatoses:
A. In Duhring-Brocq disease, the lesions are generally asymmetrical
B. Under treatment, relapses of bullous pemphigoid occur rarely
C. In Duhring-Brocq disease, the lesions are generally symmetrical
D. In bullous pemphigoid, Azathioprin is the most used drug after corticosteroids
E. Duhring-Brocq disease lesions are generally intensely asymptomatic
F. Plasmapheresis can be effective in bullous pemphigoid
G. Under treatment, relapses of bullous pemphigoid are common
H. Histology in bullous pemphigoid shows epidermal necrosis
I. The Tzanck cytodiagnostic shows epidermal acatolytic cells in bullous pemphigoid
J. Duhring-Brocq disease lesions are generally intensely itchy
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During the clinical examination of erythema nodosum, we encounter the following except:
A. Hypothermia
B. The location of the skin lesions is usually pretibial
C. Painless erythematous nodules
D. Fever
E. Arthralgias
F. Painful erythematous papules
G. Altered general condition
H. Painful erythematous nodules
I. Itching
J. Good overall condition
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H. Decreased ESR
I. The disease has a chronic evolution
J. Altered general condition
Pyoderma gangrenosum:
A. May be associated with systemic inflammatory diseases, especially inflammatory bowel diseases
B. On histopathological examination, the skin lesions are rich in neutrophils
C. Requires topical and systemic corticosteroid treatment
D. It is a common skin condition
E. Requires topical and systemic antibiotic treatment
F. Clinical evolution depends on the activity of the associated inflammatory bowel disease
G. Ulcers heal without scarring
H. On histopathological examination, the skin lesions are rich in eosinophils
I. It is a rare skin condition
J. Clinically, it presents as inflammatory nodules and purulent ulcers with purple edges
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SWEET syndrome:
A. It is characterized by hypothermia
B. Causes an acute rash with erythematous-violet plaques on the head, neck and upper trunk
C. The histopathological examination shows that the skin lesions contain numerous lymphocytes
D. It is also called acute febrile neutrophilic dermatosis
E. The histopathological examination shows that the skin lesions contain numerous neutrophils
F. It is also called acute febrile lymphocytic dermatosis
G. It is also called acute afebrile neutrophilic dermatosis
H. It is characterized by fever
I. It is a frequent condition
J. It is a rare condition
Behcet's disease :
A. It cannot affect multiple organs
B. It is an inflammatory condition
C. May cause cutaneous pathergy
D. Does not indicate treatment with colchicine or dapsone for skin lesions
E. Cannot cause cutaneous pathergy
F. It is characterized by recurrent oral vegetations, eye lesions and genital vegetations
G. Does indicate treatment with colchicine or dapsone for skin lesions
H. It is an infectious disease
I. May have skin manifestations such as erythema nodosum, acneiform lesions, thrombophlebitis
J. It is characterized by recurrent oral ulcers, eye lesions and genital ulcers
Regarding the etiopathogenesis of sexually transmitted infections, the following are True:
A. Population groups at risk can be defined in the transmission of syphilis
B. In the transmission of syphilis, we can have the open lesions of other dermatoses as a method
C. Treponema pallidum is very sensitive, survival outside the body is reduced
D. In the transmission of syphilis, we cannot have the open lesions of other dermatoses as a method
E. Syphilis is an infectious disease from the group of dermatophytoses
F. Syphilis transmission is influenced by socioeconomic factors
G. The transmission of syphilis cannot be influenced by socioeconomic factors
H. Treponema pallidum is very resistant, survival outside the body being very frequent
I. In the transmission of syphilis, population groups at risk cannot be defined
J. Syphilis is an infectious disease from the group of treponematoses
From a clinical point of view, we can say the following about sexually transmitted infections:
A. The evolution of syphilis is chronic, undulating
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From a clinical point of view, we can say the following about sexually transmitted infections:
A. In primary syphilis, the incubation period is on average 10-90 weeks
B. Syphilitic chancre is also called hard chancre
C. Syphilitic chancre is painful in most cases
D. Syphilitic chancre is also called inoculation chancre
E. Syphilitic chancre is in most cases painless
F. Syphilitic chancre is also called soft chancre
G. In primary syphilis, the incubation period is on average 3 months
H. In primary syphilis, the incubation period is on average 3 weeks
I. Syphilitic chancre is also called incubation chancre
J. In primary syphilis, the incubation period is on average 10-90 days
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E. The lymph nodes in primary syphilis are adherent to the deep planes
F. Lymph nodes from primary syphilis tend to persist for 2-3 years
G. Secondary syphilis is characterized by generalized eruptions
H. Secondary syphilis begins 6-8 weeks after the appearance of chancre
I. Lymph nodes in primary syphilis are small and painful
J. Lymph nodes from primary syphilis should persist for 2-3 months
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C. Scar
D. Chronic wounds
E. Actinic keratosis
F. Estrogen treatment
G. Smoking
H. Sun exposure (especially UVB radiation)
I. Family history of melanoma
J. Hepatitis C
ABCDE (USA) clinical criteria for the diagnosis of malignant melanoma include:
A. Tumor diameter > 2 cm
B. Color variation
C. Inflammation
D. Nevus asymmetry
E. Irregular border
F. Altered sensation
G. Diameter > 6 mm
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H. Elevation
I. Mild itching
J. Bleeding
A. Older people with dark skin who live in sunny areas are at the highest risk
B. BSCC have minimal metastatic potential
C. Genetic factors are also not involved
D. The exact etiology of BSCC is unknown
E. BSCC typically appear as a rapidly growing pearly or shiny nodule
F. Its incidence is increasing worldwide every year by 15%
G. BSCC arise from cells in the pluripotent cells of the basal epidermis
H. The locations of BSCC are at the level of the cephalic extremity and the upper limbs
I. It is the most common form of skin cancer
J. Mutations in PTCH1 have been detected in sporadic BSCC
B. Smaller but multiple nevi are associated with neurological complications but no increased risk of
melanoma.
C. Blue nevus is a blue-gray congenital nevus caused by deeper proliferation of melanocytes in the
deep dermis.
D. Atypical nevi are acquired, have regular pigmentation and are larger than common nevi.
E. The risk of malignant transformation of benign nevi is very low.
F. Acquired melanocytic nevi appear at birth, increasing in number and size during childhood and
adolescence.
G. The presence of more than 100 common nevi is associated with a 7-fold increase in the risk of
melanoma.
H. In newborns, congenital melanocytic nevi are present at birth in a percentage of 3-5%
I. Atypical nevi are associated with an increased risk of melanoma.
J. They are the most common benign neoplasms in the human species
J. Dermatofibroma is frequently located on the upper limbs, being more frequent in women.
Malignant melanoma:
A. Risk factors for malignant melanoma include sun exposure, fair skin, multiple melanocytic nevi,
family history of melanoma, and immunosuppression.
B. Superficially spreading malignant melanoma is a pigmented, large, irregular, flat lesion that initially
grows vertically and later develops laterally.
C. Treatment of metastatic melanoma includes oral tyrosine kinase inhibitors – Vemurafenib, Trametinib
and the MEK inhibitor – Dabrafenib.
D. Malignant acral lentiginous melanoma is always linked to sun exposure.
E. About 75% of cutaneous melanomas occur on pre-existing nevi, the rest occur de novo on normal
skin.
F. Although it accounts for only 4% of skin cancer cases, it is the cause of 80% of skin cancer deaths.
G. It is the most serious form of skin cancer.
H. Melanoma on lentigo maligna usually appears as a new nodule.
I. Nodular malignant melanoma is the most aggressive type, presenting as a pigmented, slow-
growing nodule that bleeds or ulcers.
J. Nodular malignant melanoma is rarely amelanotic (non-pigmented) and may mimic pyogenic
granuloma.
Kaposi's sarcoma:
A. In some African countries the seroprevalence of HHV-8 can reach up to 80%.
B. HHV-8 seroprevalence is 10% in the US.
C. The classic or sporadic form presents with fast-growing macules, plaques or nodules, localized on
the upper limbs.
D. The 3 types of Kaposi's sarcoma have an important association with human herpes virus 8 (HHV-8).
E. In the form associated with immunosuppression, the lesions are extensive, involving the oral cavity,
genital and intestinal mucosa.
F. The endemic form has a greater cutaneous extension, with involvement of the lymph nodes.
G. The form associated with immunosuppression is more severe and occurs more frequently in HIV-
positive homosexual men.
H. An important feature of the endemic form is edema.
I. The endemic form occurs in elderly males, especially in Eastern European Jews.
J. It is a tumor of the vascular and lymphatic endothelium that presents itself as nodules and
erythematous plaques.
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D. Topical Imiquimod therapy remains a treatment option in patients with BSCC who do not tolerate
surgery.
E. The recommended treatment for most basal cell carcinomas is wide excision, without requiring
histological examination, if the tumor has been removed with adequate margins.
F. Superficial BSCC can be treated non-surgically with cryotherapy, photodynamic therapy, and topical
Imiquimod.
G. Basal cell carcinomas typically appear as a pearly or shiny nodule, rapidly increasing in size, located
on the head or neck.
H. Basal cell carcinomas have minimal metastatic potential.
I. Mutations in PTCH1, the human counterpart of the Patched gene that regulates the Hedgehog
intracellular signaling pathway, have been detected in sporadic BSCC and Gorlin syndrome.
J. Elderly, fair-skinned people who live in sunny areas have the highest risk of developing basal cell
carcinomas.
Melanocytic nevi:
A. The risk of malignant transformation of benign nevus is very low
B. Benign melanocytic nevi have regular borders
C. Atypical nevi have regular pigmentation
D. Malignant melanocytic nevi have regular borders
E. Congenital melanocytic nevi are present at birth in 5-7% of newborns.
F. They are the rarest benign neoplasms.
G. They are the most common benign neoplasms
H. Congenital melanocytic nevi are present at birth in 1-2% of newborns.
I. The presence of more than 50 common nevi is associated with a 5-fold increase in the risk of
malignant melanoma.
J. Acquired melanocytic nevi appear in childhood, adolescence and young adulthood
Bowen's disease:
A. Genital Bowen's disease is more common in immunosuppressed people.
B. It typically affects the lower limbs in fair-skinned men.
C. It represents an indolent form of intraepidermal squamous cell carcinoma.
D. Genital Bowen's disease is rare in immunosuppressed people.
E. It represents an intraepithelial carcinoma.
F. A variant of Bowen's disease can affect the oral mucosa.
G. It represents an aggressive form of intraepidermal squamous cell carcinoma.
H. It typically affects the lower limbs in fair-skinned women.
I. A variant of Bowen's disease can affect the genital mucosa.
J. The therapeutic options are similar to those for seborrheic keratosis.
C. Regular border
D. Nevus asymmetry
E. Flat nevi
F. Color variations
G. Irregular border
H. Diameter > 6mm
I. Uniform color
J. Diameter >1cm
Acne vulgaris:
A. It is represented by papules, pustules or erythematous nodules
B. It is associated with the infection produced by Propionibacterium acnes
C. Soaps have a significant effect on the condition
D. It represents the inflammation of hair follicles and sweat glands
E. There is a proven association between acne vulgaris and the consumption of certain foods
F. Acne can cause permanent scarring
G. The severity of acne increases at the end of adolescence
H. It is predominantly located on the face, neck, anterior and posterior chest
I. It is associated with androgenic hormones and follicular obstruction caused by skin exfoliation or
cosmetic products
J. Antibiotics (oral or topical) are recommended as the first line of treatment
F. Before starting treatment with oral isotretinoin, women must have at least one negative pregnancy
test
G. Oral antibiotics can inhibit bacterial growth (second-line therapy, used in combination with a topical
retinoid)
H. Oral isotretinoin requires careful monitoring of liver enzymes and contraception through monthly
hCG testing
I. Oral contraceptives are not useful in the treatment of acne vulgaris
J. Topical benzoyl peroxide has no antimicrobial properties
Acne treatment:
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A. Topical retinoids, azelaic acid, salicylic acid and BPO are: Keratolytic; it blocks the pores, causes skin
dryness, skin peeling
B. Topical isotretinoin is recommended for severe inflammatory acne with scarring
C. Tetracycline and Benzoyl Peroxide are used for the systemic treatment of acne
D. In women, the combination of oral contraceptives and a formula containing Cyproterone acetate
reduces sebum excretion
E. In men, the combination of oral contraceptives and a formula containing Cyproterone acetate
reduces sebum excretion
F. The treatment must target the primary lesion, the vesicle and the associated inflammatory lesions
G. Topical retinoids, azelaic acid, salicylic acid and BPO are: Keratolytic; Helps to unblock pores.
Determines skin dryness, skin flaking
H. The treatment must target the primary lesion, the microcomedone and the associated inflammatory
lesions
I. Systemic isotretinoin is recommended for severe inflammatory acne with scarring
J. Tetracycline and erythromycin are used for the systemic treatment of acne
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The following statements about the anatomy of the skin are true:
A. hair and nails are not appendages of the skin
B. nails grow about 1 cm/day
C. from the depth to the surface, the epidermis is made up of a basal, spinous, granular, lucid,
corneous layer
D. the dermis has 2 areas - a superficial one (papillary dermis) and a deep one (reticulate dermis)
E. the surface of the skin is about 1.5-2 cm 2
F. the hypodermis consists of fat cells
G. melanocytes are located in the epidermis
H. epidermal turnover is achieved in approximately 26-28 days
I. the dermis is the most superficial layer of the skin
J. at palmar and plantar level we find numerous sebaceous glands
A 5-year-old child presents to the doctor for the appearance of an erythematous, scaly plaque,
with an active edge and an attenuated center, on the face for approximately 3 days. Report that
the lesion is pruritic. Vital signs are within normal limits. What is the next step in the management
of this patient?:
A. Systemic corticosteroid.
B. Systemic antibiotic.
C. KOH (potassium hydroxide) examination will show hyphae.
D. The KOH (potassium hydroxide) examination will look like "spaghetti and meatballs".
E. Topical antifungal for several weeks.
F. Topical corticosteroid for several weeks.
G. Oral antifungal for treatment-resistant cases
H. KOH (potassium hydroxide) examination
I. It is most likely a bacterial infection.
J. Systemic antihistamine to relieve symptoms.
Tinea pedis:
A. The KOH (potassium hydroxide) examination will look like "spaghetti and meatballs".
B. It is very common in underweight patients.
C. Treatment consists of topical corticosteroid administration for several weeks.
D. KOH (potassium hydroxide) examination shows hyphae.
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Tinea corporis:
A. It can have as an etiological agent a fungus from the Epidermophyton class
B. It can have as an etiological agent a fungus from the Tricophyton class
C. Treatment consists of topical corticosteroid administration for several weeks.
D. Malassezia furfur may be the etiological agent.
E. It affects the skin of the palms and soles.
F. The skin lesions are flat violet papules.
G. Patients with diabetes frequently associate this infection.
H. The treatment consists of topical antifungal administration for several weeks.
I. Skin lesions are flaccid blisters.
J. It can have as an etiological agent a fungus from the Microsporum class.
Tinea cruris:
A. In case of resistance to topical treatment, an oral antifungal may be recommended.
B. It can have as an etiological agent a fungus from the Microsporum class
C. It can have Staphylococcus aureus as an etiological agent.
D. It affects the skin of the palms and soles.
E. Treatment consists of topical antibiotic administration for several weeks.
F. Skin lesions are bubbles under tension.
G. Patients suffering from obesity frequently associate this infection.
H. The lesions are parenchymal vesicles, arranged on an urticaria-like plaque.
I. It can have as an etiological agent a fungus from the Tricophyton class.
J. Skin lesions are erythematous, scaly, pruritic plaques with active rim .and central attenuation.
Microsporia:
A. It's pilomycosis.
B. It is a bacterial infection.
C. Patients require hospitalization in the intensive care unit.
D. It mainly affects adults.
E. It mainly affects children
F. It is a viral infection.
G. The pathogen most involved is Candida albicans
H. It is contagious.
I. The hairs located on the surface of the plaques are broken 1-3 mm from emergence.
J. The lesions are large, discreetly erythematous plaques, and fine pityriaziform scales are present on
the surface.
A 33-year-old patient presents to the doctor for approximately 2 months of the appearance of a
rash consisting of salmon-colored, light brown or hypopigmented macules, which present easily
removable scales on the surface, located at the level of the posterior chest. The patient states that
the lesions do not tan in the sun like the rest of the skin. What is the next step in the management
of this patient?:
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A 77-year-old patient, known to have type 2 diabetes and obesity, presents to the doctor for the
appearance of approximately 2 weeks of erythematous plaques, with fine scales on the surface,
with active edge and attenuated center, located at the level of the inguinal folds. The patient
reports that the lesions are itchy. What is the next step in the management of this patient?:
A. The pacient has a fungal infection.
B. Direct mycological examination by KOH (potassium hydroxide) examination does not help.
C. We recommend the patient topical treatment with Aciclovir.
D. Mandatory skin biopsy.
E. Oral antibiotic treatment.
F. Oral antifungal in case of resistance to topical therapy.
G. Topical antifungal for several weeks.
H. KOH (potassium hydroxide) examination confirms the diagnosis.
I. The patient has a viral pathology.
J. The patient has tinea cruris.
A. Tinea corporis is a viral skin infection characterized by a scaly rash on the body with a clear center
and raised edges.
B. The bacterial agent involved in the pathogenesis of pityriasis versicolor is Malassezia furfur.
C. Tinea cruris is characterized by the presence of erythematous plaques, located on the scalp.
D. Skin lesions from tinea pedis are located at the plantar level.
E. Skin lesions do not tan in the sun like the rest of the skin in patients with tinea versicolor.
F. Tinea manum is caused by Malassezia furfur.
G. Systemic treatment in Tinea capitis is recommended for cases resistant to topical treatment.
H. In pityriasis versicolor, KOH (potassium hydroxide) examination shows short hyphae and spores.
I. Treatment in Tinea corporis consists of topical antiviral applications.
J. Pityriasis versicolor is caused by a fungus.
A 67-year-old woman was recently diagnosed with onychomycosis (tinea unguium) on the
toenails. Which statements are true?:
A. We recommend topical antifungal treatment to the patient for approximately 7 days.
B. The patient has a fungal infection.
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A 5-year-old boy who goes to kindergarten presents to the doctor with multiple small plaques on
the scalp, covered with little scales, slightly adherent. The hairs are broken at different distances
from the emergence and have diminished tone. Vital signs are within normal limits. What is the
best treatment option?:
A. It is recommended to depilate the hairs before applying topical antifungal treatment.
B. Topical and oral antifungal therapy
C. The patient has an autoimmune condition.
D. The etiological agent involved is Streptococcus pyogenes.
E. The patient has dry scalp trichophytosis.
F. Treatment consists of oral antibiotic administration.
G. The patient has a pilomycosis.
H. It is a bacterial infection.
I. Microscopic examination of hairs confirms the diagnosis.
J. It is recommended to shave the hair before applying topical antifungal treatment.
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A 4-year-old girl who goes to kindergarten presents herself to the doctor for the appearance of
large, discreetly erythematous plaques located on the scalp for approximately 2 weeks. Fine
pityriaziform scales are present on the surface of the lesions. The hairs located on the surface of
the plates are broken 1-3 mm from emergence. What is the best management?:
A. Microscopic examination of hairs confirms the diagnosis.
B. The patient has a viral infection.
C. The patient has a bacterial infection.
D. The girl has a contagious pathology.
E. The patient has pilomycosis.
F. It is recommended to depilate the hairs before applying topical antifungal treatment.
G. We recommend topical and oral antifungal treatment.
H. It is recommended to shave the hair before applying topical antifungal treatment.
I. We recommend oral antibiotic treatment.
J. The pathogen most involved is Candida albicans.
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I. The skin lesions are located away from the entrance gate
J. Patients are afebrile
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Necrotizing fasciitis:
A. It is a viral infection
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Impetigo:
A. It is a fungal infection of the skin
B. It is a contagious skin infection
C. Has facial itching
D. It has golden-yellow crusts
E. It occurs most frequently in children
F. It is often caused by Staphylococcus aureus
G. It is more common in adults
H. It is a viral skin infection
I. It is often caused by Treponema pallidum
J. It is more common in the elderly
Impetigo:
A. It shows erythema
B. It shows bullae
C. It shows necrotic crusts
D. No facial itching
E. Presents petechiae
F. It shows vegetation
G. Has facial itching
H. It is located around mucocutaneous surfaces
I. It has honey-colored crusts
J. It shows lichenification
Impetigo:
A. Linezolid or vancomycin
B. Treatment with oral antibiotics if the evolution is severe
C. Acyclovir treatment
D. Treatment with topical antibiotics
E. Treatment with oral antibiotics if outbreaks occur
F. Treatment with mupirocin
G. Antiviral treatment
H. Dicloxacillin or cephalexin
I. Always treat with oral antibiotics
J. Treatment with oral antibiotics if the patient requests this
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7-year-old patient with sudden onset 3 days ago of erythematous lesions and fragile facial
periorificial blisters that open and become covered with myeliceric crusts.:
A. It is a fungal infection of the skin
B. The diagnosis is impetigo
C. Treatment with oral antibiotics if outbreaks occur
D. Treatment with topical antibiotics
E. It is often caused by HPV
F. It is a contagious skin infection
G. The diagnosis is herpes simplex
H. The diagnosis is gangrene
I. Acyclovir treatment
J. It is often caused by Staphylococcus aureus
55- year-old patient presents for 3 days pain, erythema, edema and a collection, fluctuating in the
central area, located on the upper lip:
A. The diagnosis is gangrene
B. Treatment consists of proper hygiene
C. It can lead to cavernous sinus thrombosis
D. It has high mortality (75% of patients)
E. Cotrimoxazole covers the MRSA spectrum
F. Generalized pruritus is a common complication
G. The diagnosis is skin abscess
H. The pain is usually relieved when the abscess is drained
I. The diagnosis is necrotizing fasciitis
J. Treatment consists of incision and drainage + antibiotic
A 66-year-old patient presents for 3 days of fever, chills, erythema, edema and pain in the left leg,
accompanied by leukocytosis and elevated CRP and ESR.:
A. The diagnosis is tinea corporis
B. It is a bacterial infection
C. The diagnosis is psoriasis
D. The diagnosis is impetigo
E. The treatment lasts 10-14 days
F. It is treated with penicillinase-resistant beta-lactams
G. The diagnosis is scabies
H. It is treated with oral cephalosporins
I. It is a fungal infection
J. The diagnosis is cellulitis
A 71-year-old patient with diabetes presents for 2 days of fever, chills, erythema, edema and
localized pain in the face, accompanied by leukocytosis and elevated CRP and ESR.:
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D. skin ultrasound
E. the presence of severe itching
F. microscopic examination after superficial scraping of the epidermis
G. intense pain at the level of the lesions
H. identification of parasites by optical microscopy
I. absence of itching
J. microscopic examination after deep scraping of the skin
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Which of the following statements regarding the general principles of treatment in scabies are
True?:
A. limiting interpersonal contact
B. avoiding large groups
C. correct washing of clothes, towels, linen at high temperatures (≈60 o C)
D. close contact with other family members
E. participation in community activities
F. shared use of personal items
G. encouraging prolonged scratching to reduce itching
H. washing clothes and linen at low temperatures
I. sleeping in separate rooms
J. avoiding the shared use of personal items
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G. Ulceration
H. Frequent recurrences
I. Yellow, greasy scales
J. Frequently located on the scalp
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J. Chickenpox-like rash
D. Topical corticosteroids
E. Topical antifungals
F. Removal of the triggering agent
G. Topical antivirals
H. Stopping contact with the allergen
I. Antihistamines
J. Systemic corticosteroids in severe cases
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In the etiology of allergic contact dermatitis, the following are frequently involved:
A. Parfumes
B. Plants
C. Strong acids
D. Topical NSAIDs
E. Heavy metals
F. Topical antibiotics
G. Nickel
H. Topical corticosteroids
I. Gold
J. Strong bases
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Stevens-Johnson syndrome:
A. It affects the mucous membranes
B. There is no skin fragility and skin detachment
C. It does not affect the mucous membranes
D. The peeling of the skin can be quite extensive
E. There is no risk of dehydration
F. It is a light form of polymorphic erhythma
G. It presents an increased risk of dehydration
H. The lesions are limited to the palms and soles
I. It is a severe form of erythema multiforme
J. Patches appear that affect < 10% of the skin surface (BSA)
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G. Antifungals
H. Oral contraceptives
I. Corticosteroids
J. Antivirals
A. Antifungals
B. Pain-killers
C. Systemic antibiotics
D. IV immunoglobulins
E. Treatment in burn wards
F. Chemotherapy
G. Systemic antithermics
H. Stopping the causative agent
I. Hydration IV
J. Aciclovir can be useful in cases caused by HSV
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F. Burn
G. Nodules
H. Fugitive lesions
I. Bullae
J. Pruritus
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E. MRI
F. Serum complement
G. CT
H. CBC
I. AngioCT
J. PET CT
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J. From the point of view of localization, the skin lesions are located on the flexor areas
Regarding the laboratory examination of psoriasis, the following statements are correct :
A. Biopsy of skin lesions shows absence of basal layer
B. Biopsy of skin lesions shows epidermal thickening
C. Skin biopsy shows thickening of the hypodermis
D. Laboratory tests are not necessary for diagnosis
E. Biopsy of skin lesions shows thinning of the epidermis
F. Biopsy of skin lesions shows hyperplasia of the granular layer
G. Skin lesions can be biopsied
H. Biopsy of skin lesions shows absence of granular layer
I. Biopsy of skin lesions shows the presence of nucleated cells in the stratum corneum
J. Biopsy of skin lesions shows absence of nucleated cells in the stratum corneum
The following treatment methods are used in the treatment of vulgar psoriasis:
A. Ciclosporin in mild forms of disease
B. Phototherapy
C. Topical antibiotics as first-line therapy
D. Methotrexate in severe forms of disease
E. Systemic antibiotics as first-line therapy
F. Methotrexate in mild forms of disease
G. Emollients
H. Cyclosporine in severe forms of disease
I. Systemic corticosteroids
J. Topical corticosteroids
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B. The scaly papules are arranged with the long axis parallel to the rib line
C. The skin lesions are arranged on the anterior and posterior thorax, producing the overall Christmas
tree appearance
D. The papules are erythematous
E. Squamous papule lesions cause itching
F. The disease does not occur in children and young adults
G. The scaly papules are arranged with the long axis perpendicular to the line of the ribs
H. Blistering lesions cause itching
I. The papules are brownish
J. Skin lesions do not affect the anterior and posterior thorax
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E. In pityriasis rosea the skin lesions do not affect the anterior and posterior thorax
F. In psoriasis, biopsy of skin lesions indicates the absence of the basal layer
G. In pityriasis rosea, the scaly papules are arranged with their long axis parallel to the rib line
H. Pityriasis rosea begins with heraldic plaque which consists of a single lesion up to 5 cm in diameter
I. From the point of view of localization, skin lesions from psoriasis are arranged on the extensor areas
J. In psoriasis, biopsy of skin lesions indicates the absence of nucleated cells in the stratum corneum
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I. Clinically on the skin, it manifests as pinkish-violet polygonal papules, intensely itchy, with whitish
streaks on the surface
J. Potential triggers include hepatitis B virus and hepatitis C virus
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I. Clinically on the skin, it manifests as pinkish-violet polygonal papules, intensely itchy, with whitish
streaks on the surface
J. Potential triggers include hepatitis B virus and hepatitis C virus
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