AAS

The

Anabolic Androgenic Steroid

Cheat-Sheet
HANDBOOK

A Team EvilGSP Publication

 Disclaimer!

This books information has been put together from 4 years of Evil Mentorship, multiple
textbooks, reports, medical and pharmaceutical journals, interviewed medical
professionals, interviewed experts, athletes, and anecdotes from decades of practical
application and experience. Neither the authors nor publishers of this material assume
any liability for the information presented here. It is solely for informational and
entertainment purposes only and should not be considered medical or legal advice.
Readers should be aware many of the substances described in this book may be
dangerous, prohibited and/or used only under the strict supervision of a medical
professional. Always consult with your medical professional and/or the proper legal
authorities before using any drug. The authors and publishers do not condone you break
the law of your land or any other and do not participate in any illegal activities.

Copyright: © 2021 by Team EvilGSP & Hollywood Built. Written by Scott Habermehl &
Team EvilGSP. All rights reserved.

This e-book or any part thereof, may not be reproduced or recorded in any form without
permission in writing from the author or publisher.

For Information Contact:

www.members.teamevilgsp.com
hollywoodbuilt@gmail.com
www.hollywoodbuilt.com

2
“Longer
Lower
Slower”
-B.Chavez

3
 Humbling Thanks
&
Acknowledgements

The AAS Cheat-Sheet E-Book would not be made possible without the contribution
and guidance from my friend, mentor and wise-man B. Chavez, along with Mircea Balaj &
my TeamEvilGSP colleagues, very select individuals, educators, and family who I am
thankful to have by my side.

Thank you.

4
 Purpose

This E-Book is a practical application guide, dedicated to individuals with an

appreciation of the power in pharmacology, its effects on pharmacodynamics,
pharmacokinetics, and the desire to apply these tools in the most effective way.

The information provided in this book is designed as a simple, quick reference guide to
walk the user through individual AAS profiles, providing only the necessary key
components and practical applications, in a very simple straight forward approach.

The “bullet-point” tactics and information presented here are to help minimize your
workload and help you apply the right tool for the job, maximize results and minimize
unwanted effects…with ease.

There is a glossary of key terms used throughout this book listed at the back on pages
73-75, referred to as the “Urban Dictionary.” Please reference this list to ensure a clear
understanding of the language.

Basically, saving you a ton of Time, Money & Pain!!

5
 A Word of Warning

The information provided to you in this e-book is a method of application used to

determine drug compound selection, dosages needed, and duration of use, that most
likely shatter’s the commonly held beliefs on how anabolic androgenic steroids are used
in many sports and athletic circles.

But……you can of course……

6
 Do whatever the
F*@K
you want!!
It’s your body and
your Life!!

7
 Anabolic Androgenic Steroid Profiles

Testosterone Derivatives
• Boldenone (Equipoise) pg. 25
• Methyltestosterone (Android/Metandren) pg. 26
• Methandrostenolone (Dianabol) pg. 27
• Chlorodehyromethyltestosterone (Turinabol) pg. 28
• Fluoxymesterone (Halotestin) pg. 29

Dihydrotestosterone (DHT) Derivatives

• Methenolone (Primobolan) pg. 31
• Drostanolone (Masteron) pg. 32
• Dihydroboldenone (DHB) pg. 33
• Mesterolone (Proviron) pg. 34
• Furazabol (Frazalon) pg. 35
• Oxandrolone (Anavar) pg. 36
• Stanozolol (Winstrol) pg. 37
• Oxymetholone (Anadrol) pg. 38
• Methasterone (Superdrol) pg. 39

Nandrolone (19-Nor) Derivatives

• Nandrolone (Deca-Durabolin) pg. 41
• Trenbolone (Trenabol) pg. 42
• Trestolone (MENT) pg. 43
• Oxabolone (Steranabol Ritardo) pg. 44
• Bolasterone (Myagen) pg. 45

Practical Application
• Risk Adverse example pg. 51
• First Oral only example pg. 52
• First Injectable only example pg. 54
• Female Course example pg. 56

8
 Sex Hormone Synthesis

CHOLESTEROL

PREGNENOLONE

PROGESTERONE

DHEA

ANDROSTENEDIONE

AROMATASE

TESTOSTERONE

ESTRONE (E1) AROMATASE

5-ALPHA REDUCTASE (5AR)

ESTRADIOL (E2)

DHT

ESTRIOL (E3)

9
 Testosterone vs. AAS
Testosterone is an ENDOGENOUS androgen hormone that is anabolic. It is
manufactured mainly by the Leydig cells of the testes in males and ovaries in females,
from a downstream conversion of cholesterol. It is the primary male sex hormone in the
body.

Anabolic Androgenic steroids (AAS) are a class of EXOGENOUS

drugs, defined as synthetic or “man-made” derivatives of testosterone or any related
hormone/metabolite.

There are two main types of AAS:

• 17-alpha alkyl derivatives (orally bioavailable)

• 17-beta ester derivatives (Intramuscular or Subcutaneous injectables)

Anabolic Androgenic steroids (AAS) are created via chemical alterations using one of
these three naturally occurring hormones as their back-bone structure.

The initial back-bone structure is first decided as the starting point for all the different
AAS compounds based off specific characteristics and traits it offers (or doesn’t), to
maximize the benefits of the drugs desired purpose, while minimizing or completely
avoiding undesirable effects.

10
 The Natural Base Structures
Testosterone: 95% is manufactured by the Leydig’s cells in the testes. Testosterone
is a 4-Ring structure and the primary male sex hormone giving the main template that all
AAS compounds are based off. It is synthesized from cholesterol, interacts (binds) with
various receptors in the body, specifically the androgen receptor (AR), to signal the
growth and development of masculine sex organs and secondary sexual characteristics.
Testosterone is the primary substrate for the synthesis of estrogen (aromatization) in the
body and is considered to have an anabolic to androgenic ratio of 100:100. This
anabolic/androgenic ratio is the reference marker all AAS compounds are compared
against, when determining their specific qualities and characteristics. Naturally
occurring testosterone has an elimination half-life of 34 minutes and is excreted in the
urine and feces.

• Drives cell volume

• Drives behavioral changes
• Mean testosterone levels for adults. Males = 630ng/dL Females = 32.6ng/dL
• Men produce 2.5-11mg/day. Females produce ~.25-1mg/day
• Primary substrate for the synthesis of estrogen
• Drives hematology

Nandrolone (19-Nor): Also known as 19-nortestosterone (19-nor), is a naturally

occurring testosterone derivative and endogenous intermediate in the aromatization of
estrogen. Almost identical to testosterone but lacking a carbon atom at the 19th position
which reduces nandrolones androgenic properties, making it suitable for women. A
chemical process known as demethylation (nor), removes the methyl group at the C19
position, replacing it with a hydrogen atom. It is specifically the C-19 demethylated
analogue of testosterone, or “without a carbon group at the 19th position.” Naturally
occurring nandrolone has an elimination half-life of <4.3 hours and is excreted in urine.

• Testosterone derivative
• High anabolic, low androgenic effects – Anabolic:Androgenic Ratio 3-16:1
• Suitable for women and children
• Drives cell volume
• First introduced for medical use in 1959 as Nandrolone phenylpropionate (NPP)
• Clinical use for people in catabolic states (AIDS, Cancer, etc.)
• Benefits muscle growth, anemia, bone density, appetite & RBC production

11
Dihydrotestosterone (DHT): Is an endogenous androgen sex hormone metabolite
derived from the parent hormone, Testosterone. Approximately 5-7% of testosterone is
irreversibly converted to the active metabolite DHT in target tissues by the enzyme 5-
alpha reductase (5AR). The 5AR enzyme reduces testosterone’s double bond at the C4-
C5 position. DHT has an elimination half-life of 53 minutes and is excreted in the urine.

• Testosterone derivative
• Binds with higher affinity to androgen receptors than testosterone
• Activates gene expression more efficiently
• Measures 3-4 times more potent than testosterone with less androgenicity
• Cannot be aromatized into estrogen
• Introduced for medical use as an AAS in 1953
• Anabolic:Androgenic ratio is 1:1

12
 Chemical Structure Modifications
Since its discovery and synthesis in the 1930’s, Testosterone’s base steran structure has
been chemically altered in a variety of ways, thereby changing the anabolic and/or
androgenic properties, creating very different physiological effects. These subtle
alterations have made many anabolic androgenic steroids extremely beneficial in clinical
medicine, but more importantly in physique alterations and athletic performance.

These are the common alterations clever scientists have discovered over the years that
serve a very specific purpose, causing the synthetic hormone to behave in a very specific
way. The unique characteristics each AAS compound offers, is not by coincidence and is
designed for certain situations. It’s up to the athlete and coach to choose the right tool
for the job.

13
 Fun Fact
If you are using ANY exogenous Testosterone as ANY part of your course or bridge, the
only difference you need to consider is the chosen ester and corresponding half-life.

Because…

Testosterone
is

Testosterone
is…

Testosterone

Testosterone Cypionate = Testosterone Enanthate = Testosterone Propionate = Testosterone Suspension =

Testosterone Blends…….

Just equate the half-lives!

14
 Meet The Family
The concept of the anabolic “family tree” originates 30+ years ago with Dan Duchaine
but was first taught to me by Broderick Chavez. It is a fantastic way to simplify the
different AAS compounds and organize them based off their back-bone structure. Since
anabolic androgenic steroids are all synthetic variations of testosterone, the “family
tree” simply categorizes each compound into one of three columns.

• Testosterone direct derivatives (Center column)

• Dihydrotestosterone (DHTs, Left column)
• Nandrolone (19-Nor’s, Right column)
The characteristics and traits from each of the 3 naturally occurring base structures
briefly described above, (testosterone, dihydrotestosterone & nandrolone) should right
away give you some insight as to which column you want to be shopping in when looking
to fill your milligram requirements.

Testosterone

DHT Direct Derivatives 19-Nor

-Methenolone -Boldenone -Nandrolone
-Drostanolone -Methyltestosterone -Trenbolone Acetate
-DHB -Methandrostenolone -Trestolone
-Mesterolone -Chlorodehyromethyltestosterone -Oxabolone
-Furazabol -Fluoxymesterone -Bolasterone
-Oxandrolone
-Stanozolol
-Oxymetholone
-Methasterone

15
 Injectables vs. Orals
After studying the Anabolic Family Tree, you should have an idea of where your drug(s) of
choice belongs, based on the qualities of its root back-bone structure and the general
characteristics common with the compounds in that column.

Now you look at what is needed for you to accomplish the goal (size, focus, strength,
power, speed, hematology, etc.) and which general qualities from the 3 columns would
be best suited.

Once you’ve decided on which of the column(s) you feel will offer the best overall
benefits, you begin to narrow down your compound selection(s) based on the additional
unique and specific characteristics each AAS brings and what would best compliment
the goal.

The three columns in the Family Tree have a wide variety of AAS options to choose from,
each with their own unique qualities and traits. Along with the assortment of compounds
in each column, some consideration should also be made on the type of steroid the user
wishes to use, based on its route of administration.

The two types of steroids and their route of administration commonly used are
intramuscular (IM) or subcutaneous (SubQ) injections and compounds that are orally
bioavailable.

Two types of Steroids

• 17-alpha alkyl derivatives (oral)

• 17-beta ester derivatives (injectable)

Not all compounds are available in both oral and injectable form, so the route of
administration for each AAS compound can also help make the decision of which steroid
would be the best tool for the job. Ease of use, onset of action, timelines, detection
times, etc. are all factors to consider.

16
 Injectables
17-beta ester derivatives or Injectable steroids are administered either deep into the
muscle tissue (intramuscular) or directly under the skin with a shorter needle
(subcutaneously). The type of ester attached, slows the release of the drug into the
blood and extends the elimination half-life.

Injectables have a slower rate of action and a longer half-life than oral steroids.

Common Androgen Ester Half-lives

Suspension – 1 Day Propionate – 2 Days Acetate – 3 Days
Phenylpropionate – 4.5 Days Butyrate – 6 Days Valerate – 7.5 Days
Hexanoate – 9 Days Enanthate – 10.5 Days Cypionate – 12 Days
Nonanoate – 13.5 Days Decanoate – 15 Days Undecanoate – 16 Days

Orals
17-alpha alkylated derivatives have had the hydrogen atom at the 17th position removed
and replaced with a carbon atom. This alteration allows the steroid to survive first pass
of the liver, conveniently allowing the drug to be orally bioavailable and does not require
intramuscular or subcutaneous injection. A methyl group attached to C-1 can also allow
for oral administration (Methenolone, Mesterolone), but are considered weaker
anabolics.

Oral steroids have a much faster onset of action and a much shorter half-life than
injectable steroids.

Pro’s & Cons

Orals Injectables
-Faster Onset of Action -Slower Onset of Action
-Shorter Action (half-life) -Longer Action (half-life)
-Greater Retention (fluid & CHO’s) -Greater Infection Risk
-Suppress appetite -Less liver toxic

6-8 weeks is the common duration of oral steroid use, but it is recommended here that duration should be
kept to 1 day per kilogram of bodyweight with an equal time “off” as “on” to minimize liver toxicity.

17
 The Anabolic:Androgenic Ratio
First described in 1953 by Hershberger and colleagues as a screening for androgenic and
anabolic effects to help determine a steroids clinical application. The established
anabolic to androgenic ratios were determined using the relative weight changes of the
prostate and size of the levator ani muscle in castrated male RATS. The rats were
treated with Testosterone, and then compared to untreated rats as a baseline reference.

The increase in prostate was indicative of androgenic effects and the increase in the
levator ani muscle was indicative of the anabolic effects. When treated with
Testosterone, the physiological changes were given a score of 100, for both the
androgenic and anabolic effects when compared to the untreated rats. This established
the known AAS ratio of 100:100. All steroids anabolic androgenic effects are rated
against this ratio to indicate the steroids theoretical potency.

In clinical medicine, compounds with a higher anabolic to androgenic ratio are more
commonly used in the treatment of anemia, osteoporosis, wasting diseases (cancer, HIV,
etc.), post trauma, surgery, prolonged immobilization, and improved protein synthesis.

Compounds with a higher androgenic to anabolic ratio are used in hormone replacement
therapy, androgen-deficiency therapy in teenagers, transgender therapy, etc.

Anabolic Rating: How much muscle can the compound build per unit/mg
compared to the reference 100 of Testosterone.

Androgenic Rating: How much masculinizing qualities (body hair, virilization,

etc.) are increased per unit/mg compared to the reference 100 of Testosterone.

All Anabolic Androgenic Steroids use the 100:100 ratio of Testosterone as the baseline
comparison to indicate the theoretical potency and specific anabolic and androgenic
effects. The given ratios for each AAS compound do not indicate the unique
characteristics they potentiate in the human body and are merely a measuring tool to
help determine potency. This may be helpful in assessing/adjusting total milligrams
when switching from one compound to another.

18
 There are NO free rides.
ALL anabolic androgenic steroids can have negative health effects, especially at higher
dosages. The importance of regular blood work cannot be overstated.

• AAS use negatively impacts cardiovascular health markers. There is a general

tendency for steroid use to reduce HDL (“good” cholesterol) and increase LDL
(“bad” cholesterol) as well as serum lipid levels.
• AAS use is toxic to the liver (hepatoxic), especially at higher dosages and/or longer
durations, specifically AAS compounds with a C-17-alpha alkylated alteration.
Blood work and careful monitoring of liver values is good practice.
• All AAS compounds can suppress endogenous testosterone production. Dose,
duration, and compound selection are all relevant considerations in the magnitude
of endogenous suppression. Without pharmacological intervention, the natural
production should (but not always) return to normal within 1-4 months. The
concept of post cycle therapy (PCT) may be a relevant consideration but is not
covered in this book.
• AAS use can increase blood pressure, especially at higher dosages.
Supraphysiological dosages have been shown to increase both systolic and
diastolic blood pressure.
• AAS use effects hematology by stimulating erythropoiesis and driving the
production of red blood cells. Careful attention to RBC, hemoglobin and hematocrit
values on blood work is also good practice.
• AAS use is generally well tolerated by the kidneys at therapeutic dosages, but
supraphysiological dosages can impair kidney function, especially if a degree of
liver toxicity has been experienced.

This is only a small list and brief description of the many possible negative health effects
associated with AAS use. Health status, compound selection, duration and dosages are
the key factors to consider when looking to minimize or even avoid potential negative
health impacts.

Regular screening of health markers is a must!

19
 Attention Females!
The use of Anabolic Androgenic steroids are incredibly powerful tools for women looking
to achieve increased muscularity and/or improved athletic performance. However, the
exogenous supplementation of these hormones can also be equally deleterious to one’s
femininity and overall health. This must be a well thought out and personal decision
before a female considers the use of any AAS compound.

There is NEVER a zero-risk scenario with AAS use.

If you have made the decision to participate in AAS use, understand it is literally
engaging in the process of sexual gender transitioning!! Females are hypersensitive to
the effects of androgen use, therefore, it’s imperative that you pay careful attention to
how your body is responding, if avoiding any unwanted and potentially irreversible
physiological changes is desired.

Biological change is a slow-moving process with un-wanted effects of AAS use being
very insidious for females and extremely difficult to notice if you are not looking for
them…. until it’s too late. Some of the things to watch out for may sound silly and even
quite personal but catching un-wanted effects early may enable the user to prevent
worsening and/or potentially irreversible outcomes.

Virilization is the biological development of adult male characteristics, mostly produced

by androgens. In other words, if avoiding masculinizing effects or maintaining femininity
is important for the female user, then compound selection, dosage and duration are very
important factors to consider.

Anabolic Androgenic Steroids with a lower androgenic ratio are typically the compounds
most females will want to be looking at. It’s the androgenicity effects of the drugs that
pose the greatest risks for females wanting to minimize “male-like” symptoms.

Please remember, there is still ALWAYS a chance of undesirable outcomes.

An Entire book on AAS use for Females is coming soon!!

20
 Virilization in Women
It is strongly recommended that females pay close attention to the following signs of
virilization if masculinizing effects are a concern

• Voice (usually begins with a “scratchy” sensation)

• Female Genitalia
• Receding hairline or thinning of hair
• Access facial and/or body hair
• Oily skin
• Skin thickness and texture
• Decreased breast size

Regular checks with pictures, measurements, self-evaluation, and voice recordings can
help determine if un-wanted changes are approaching an undesirable level. It is best to
STOP immediately if you start to see a problem. Prolonging the use can increase the
chances of the un-wanted effects becoming irreversible.

Duration is the biggest enemy for females to consider when minimizing virilization
symptoms is important and is the reason this book recommends the following drug
course durations.

3 weeks (min) to 9 weeks (max) with an equal time “off” as “on”

During the bridge or “time-off” period, using non-AAS compounds like growth hormone,
insulin, creatine, etc. is an effective strategy in helping to maintain progress while
avoiding further masculinizing effects.

Dosages for females is also an important consideration and is as follows.

Approximately 1-3 milligrams per kilogram of bodyweight

1mg/kg is extremely effective and brings the least amount of risk in terms of un-wanted
effects, while 2mg/kg and 3mg/kg dosages increase both improved outcomes but also
potentially increased problematic issues.

Females should always strive for the minimum effective dosage that brings the desired
results, while exposing themselves to the least overall total drug exposure. This is how
to stand the best chance at avoiding masculinizing effects and maintaining
femininity….IF that is an important aspect of the users end goal.

21
 The Profiles
• The following pages of AAS profiles are separated based on their back-bone
structures of direct testosterone derivatives, DHT derivatives or 19-nor derivatives
and then organized into the 3 columns of the anabolic family tree.
• Many of the Anabolic Androgenic steroids described in this book share their roots
in clinical medicine. Despite the established medicinal benefits anabolic steroids
offer at therapeutic dosage, this book will focus more on how each compound
applies to Improving athletes and sports performance.
• The following pages provide only the key points needed to apply each given
compound, in a practical application scenario to best meet the goal(s) of the
individual.
• Each column of the family tree contains AAS profiles that mimic the same general
characteristics shared by its back-bone structure, and then adds its own
component of specificity. The specific traits of each anabolic need to be
considered when deciding which compound(s) are best suited for the desired
outcome.
• The profile pages were designed intentionally to be brief and straightforward, with
relevance focused on training application and preservation of long-term health.
The responsible athlete should always be in search of the minimum effective
dosage along with the minimum effective duration to achieve the desired results.
• It’s very important to use bloodwork when introducing pharmacology into one’s
lifestyle. I would suggest individuals at least draw bloods before beginning a
course, and then again upon completion. Use this information to help make
intelligent and responsible decisions to maximize the benefits and minimize the
detriments.
• Anabolic courses or “cycles” are typically advised to be between approximately
8-20 weeks for males or 3-9 weeks for females. Outliers exist.
• Depending on many factors, individual dosages range wildly. Anywhere from
TRT/HRT ranges of .5mg/kg to 20+mg/kg is not uncommon. Outliers exist.
• Your AAS course design should consider duration and dosages that compliment a
periodized plan based on training, lifestyle, schedule, goal(s), and health markers.
Ideally designed over the course of the competitive season or calendar year.

22
 Common Drug Dosages

• Hormone Replacement Therapy (HRT) - ~.5-1mg/kg

• Testosterone Replacement Therapy (TRT) - ~.5-1mg/kg
• Sports Hormone Replacement Therapy (Sports HRT) - ~2-3mg/kg
• Sports Testosterone Replacement Therapy (Sports TRT) – ~2-3mg/kg
• Female Athletes - ~1-3mg/kg
• “Average Joe” - ~5-10mg/kg
• State/Provincial Level Athletes - ~10mg/kg
• National Level Athletes - ~15mg/kg
• Professional Athletes - ~20mg/kg

Kg. lbs. Kg. lbs. Kg. lbs.

45.5 100 70.5 155 95.5 210
47.7 105 72.7 160 97.7 215
50 110 75 165 100 220
52.3 115 77.3 170 102.3 225
54.5 120 79.5 175 104.5 230
56.8 125 81.8 180 106.8 235
59.1 130 84 185 109.1 240
61.4 135 86.4 190 111.4 245
63.6 140 88.6 195 113.6 250
65.9 145 90.1 200 115.9 255
68.2 150 93.2 205 118.2 260

1kg=2.2lbs

Don’t let the “standard” Anabolic Androgenic Steroid dosing protocols, heard through-out
gyms and different athletic circles guide your course design. Anabolic Androgenic
Steroids are drugs, and just like any other drug, they should be dosed based on
bodyweight.

23
Testosterone
Derivatives
Testosterone derivatives create the center-column of the Family Tree.
They are most like endogenous testosterone.

- Good “general” AAS

- Strong Anabolic & Androgenic Effects
- Drives hematology more than any other column
- Average cell volume (more than DHT/less than 19-nor)
- Aromatizes at ~40-60% of Testosterone
- Behavioral changes
- Best used as TRT/HRT

24
Compound: Boldenone
Trade Name: Equipoise (EQ)

Back-Bone Structure: Center Column Behaves like: Testosterone

Theoretical Anabolic Rating: 100 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 50 Reality Androgenic Rating: Equivalent

Half-life: ~14 Days Termination life: ~60-70 Days

Impact on Hematology: Increased Impact on Blood Pressure: Increased

Impact on Kidneys: Moderate Impact on Liver: Low

Impact on Libido: Moderate Impact on Cardiovascular: Moderate

Aromatization Rate: Low Administration: Injectable

Recommended Duration: ~8-20 weeks or HRT Detection Time: ~4-5 months

Retentive Qualities: Mild-Moderate Ester(s): Undecylenate

Anabolic/Androgenic Effects: Boldenone increases nitrogen retention, protein synthesis, appetite, and an
increase in hematology, specifically increasing the production of erythropoietin. Though the androgenic ratio is lower
with boldenone, oily skin, acne and other androgenic side effects are still possible, particularly at higher doses.

Performance/Relevant Effects: The increased hematology from boldenone lends itself well for certain
performance athletes where possible weight gain is not a detriment. Drives appetite and a long steady increase of
lean muscle mass with less water retention than other center column drugs. Anxiety has been reported by some.

Common Usages/Synergy: Commonly used for HRT, “Sports” HRT, strength and quality lean mass accrual as
well as to drive an increase in hematology. Legitimate use is mostly only veterinary, specifically in horses

Female Considerations: Boldenone is a direct derivative of testosterone and should be approached with
caution for females. The lower androgenic rating makes it a slightly better option than other center column drugs,
but typically not recommended.

Description: Boldenone differs from testosterone only with an added double bond between carbons 1 and
2, reducing its aromatization ability and androgenic effects by approximately 40-50% while remaining equally
anabolic. Originally synthesized as an attempt at a long-lasting HRT option from androgen deficiency. Boldenone is
an excellent compound for driving increased hematology and the slow steady accrual of quality lean mass. The
increased hematology effects of Boldenone suggest consistent blood work. Boldenone can also be reduced to a very
potent DHT metabolite dihydroboldenone (DHB), via the 5-alpha reductase enzyme. Center column compounds are
excellent at suppressing endogenous testosterone production. Metabolized by the kidneys & liver and excreted
through urine and feces.

25
Compound: Methyltestosterone
Trade Name: Android/Metandren

Back-Bone Structure: Center Column Behaves like: Testosterone

Theoretical Anabolic Rating: 115-150 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 94-130 Reality Androgenic Rating: Higher

Half-life: 2.5-3 Hours Termination life: ~15-20 Hours

Impact on Hematology: Moderate Impact on Blood Pressure: Moderate

Impact on Kidneys: High Impact on Liver: High

Impact on Libido: High Impact on Cardiovascular: High

Aromatization Rate: High Administration: Oral

Recommended Duration: 1 day/kg-bodyweight Detection Time: ~3-4 Days

Retentive Qualities: High (especially water) Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: Methyltestosterone is an oral version of testosterone. It brings both anabolic

and androgenic effects but with a higher aromatization rate due to the conversion of 17-a methyl-estradiol.
Estrogenic side effects are common.

Performance/Relevant Effects: Drives aggression and increases nitrogen retention, sodium, phosphorus,
potassium, and calcium.

Common Usages/Synergy: Not commonly used for the hypertrophic effect, as the potential adverse health
risks outweigh the benefits with longer durations. However, the use for acute increases in aggression is common in
powerlifters and competitive athletes during an event or particular training session.

Female Considerations: Methyltestosterone is an oral version of testosterone and should be approached with
caution for females, especially when looking to minimize masculinity. Typically, not recommended. During
pregnancy, methyltestosterone has an even greater risk/reward scenario.

Description: Methyltestosterone is an orally available form of testosterone described in 1935. The added
methyl group at the C-17-alpha position for oral bioavailability made methyltestosterone among the first functional
oral steroids to be produced. Methyltestosterone is both anabolic and androgenic with high estrogenic activity due to
its aromatization to 17-alpha methyl-estradiol. The potential risk of adverse health effects is more common with
methyltestosterone and is not often used for the enhancement of athletic performance or physique but may be
beneficial for acute increases of aggression. Methyltestosterone is 90% excreted in the urine and 10% from feces.
Center column compounds are excellent at suppressing endogenous testosterone production. Orals are best taken on
an empty stomach.

26
Compound: Methandrostenolone
Trade Name: Dianabol (D-bol)

Back-Bone Structure: Center Column Behaves like: Testosterone

Theoretical Anabolic Rating: 90-210 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 40-60 Reality Androgenic Rating: Equivalent

Half-life: 3-5 Hours Termination life: 15-20 Hours

Impact on Hematology: Moderate Impact on Blood Pressure: High

Impact on Kidneys: High Impact on Liver: High

Impact on Libido: Moderate-High Impact on Cardiovascular: High

Aromatization Rate: Moderate Administration: Oral

Recommended Duration: 1 day/kg-bodyweight Detection Time: 5-6 Weeks

Retentive Qualities: High Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: Modified to maintain the anabolic effects while reducing androgenic effects.
Gynecomastia is common, despite the lower androgenic rating as well as other common androgenic effects.

Performance/Relevant Effects: Drives the accumulation of weight gain, and strength, potentially beneficial
for athletes in need of sheer size. Retentive qualities in increases of nitrogen, protein synthesis, potassium, etc. in a
short time period.

Common Usages/Synergy: Dbol is commonly used to increase overall mass in athletes and bodybuilders.
When used in conjunction with other compounds, specifically longer acting esters like an enanthate or cypionate, the
results in sheer mass gains are more dramatic with no real further increases in side effects from Dbol alone.

Female Considerations: As with all center column compounds, the use in women should be approached with
caution due to the higher androgenic effects. Dbol does offer a slightly less androgenic rating, making the use in
females possible, but still risky. If used, low doses can be effective for muscle gain, but side effects are still common.

Description: The most commonly used oral anabolic androgenic steroid for physique and performance
enhancements. Dbol was first described in 1955 and released by Ciba Pharmaceuticals in 1958. It is a modified form
of testosterone with the addition of a methyl group at the 17th position, making it orally bioavailable and an added
double bond between carbons C1 & C2, reducing the androgenicity of the drug. Common for physique and
performance enhancement purposes in weightlifters, powerlifters and other athletes. Methandrostenolone is
metabolized by the liver and excreted in the urine.

27
 Chlorodehydro-
Compound:
Methyltestosterone
Trade Name: Oral Turinabol (CDMT)

Back-Bone Structure: Center Column Behaves like: Testosterone

Theoretical Anabolic Rating: >100 - <200 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: <40 Reality Androgenic Rating: Equivalent

Half-life: 16 Hours Termination life: ~80 Days

Impact on Hematology: Moderate Impact on Blood Pressure: Moderate

Impact on Kidneys: Low-Moderate Impact on Liver: Moderate-High

Impact on Libido: Low-Moderate Impact on Cardiovascular: Moderate

Aromatization Rate: None Administration: Oral

Recommended Duration: 1 day/kg-bodyweight Detection Time: 4-12 Months

Retentive Qualities: Low-Moderate Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: A potent derivative of Dianabol, but slightly less anabolic and much less
androgenic. Oral Turninabol has zero estrogenic effects when used in isolation.

Performance/Relevant Effects: New methods of detection can potentially allow Turinabol to be

discoverable for up to 12 months. Can lower SHBG, which can increase other free (unbound) hormones.

Common Usages/Synergy: Mostly bodybuilding, performance & strength communities with synergistic
effects of a “1 + 1 = 3” scenario when used in conjunction with Oxandrolone. For example, 10mg of Turinabol + 10mg
of Oxandrolone offers a potential 30mg anabolic effect.

Female Considerations: The high anabolic low androgenic ratio of CDMT makes it suitable for female use.
Duration should be 3 weeks minimum, 9 weeks maximum. Starting dosage ~1 milligram per kilogram of bodyweight,
with a maximum ~3 milligrams per kilogram of bodyweight. Consider the possible synergistic effects when combined
with Oxandrolone.

Description: CDMT is an East German product from 1961, combining 4-Chlorotestosterone and
Methandrostenolone (Dbol). It differs from testosterone with the added C-17a Methyl Group, a double bond between
carbons 1&2, improving the anabolic:androgenic ratio and an attached chloro-group at the 4-position inhibiting
aromatization. In the 1990’s, CDMT was revealed as a major player in the East German Doping Machine scandal from
1974-1989, which was an aggressive anabolic program specifically designed for cheating the Olympic drug test.
Designed for several medical uses, but mainly in building/preserving lean muscle tissue and bone mass. The low
androgenic ratio makes CDMT suitable for women and children. Metabolized by the liver and excreted through urine.

28
Compound: Fluoxymesterone
Trade Name: Halotestin (Halo)

Back-Bone Structure: Center Column Behaves like: Testosterone

Theoretical Anabolic Rating: 1900 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 850 Reality Androgenic Rating: Higher

Half-life: 9.2 Hours Termination life: 45-50 Hours

Impact on Hematology: Moderate-High Impact on Blood Pressure: High

Impact on Kidneys: High Impact on Liver: High

Impact on Libido: Increased Impact on Cardiovascular: High

Aromatization Rate: None Administration: Oral

Recommended Duration: <6-8 weeks Detection Time: ~2 Months

Retentive Qualities: Low Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: Very high androgenic effects with moderate-high anabolic effects based on
the theoretical anabolic:androgen index. Real world scenarios seem to show much higher anabolic and androgenic
effects. Non estrogenic.

Performance/Relevant Effects: Very powerful drug for driving aggression and strength with minimal weight
gain. Mood changes and possible depression are known side effects. Highly suppressive to endogenous testosterone.

Common Usages/Synergy: Due to the increased aggression, Halotestin is more common in sports
performance scenarios. The increased aggression and androgenicity may also offer benefits to a bodybuilder in the
final weeks of their competition prep, keeping intensity up during their training sessions and can help with a hard,
defined stage look if estrogen is controlled. Typical increases in muscle strength, muscle density and definition with
minimal size increases are seen. Common in weight-restricted sports like wrestling, powerlifting, boxing, MMA, etc.

Female Considerations: The high androgenic rate of Halotestin makes it a compound that should be avoided
by women worried about virilization side effects.

Description: Described in 1956, a derivative of testosterone, specifically methyltestosterone (C-17a) with

an addition of 11-beta-hydroxy and 9-alpha-fluoro groups resulting in a more potent orally active non-aromatizable
steroid. Halo offers very high androgenic effects with moderate anabolic effects when compared to Testosterone,
making it a great choice for masculinizing effects and contractile strength, with less potential for significant weight
gain. Originally designed to treat male androgen deficiency, breast cancer in women and anemia. Halo suppresses
endogenous testosterone levels rather quickly, despite the lack of estrogen conversion. Generally, durations of <8
weeks is advised due to the high level of liver toxicity. Metabolized by the liver and excreted in the urine/feces.

29
 DHT’s
Derivatives
DHT derivatives create the left side of the Family Tree

- Good for “Hardness”

- Do not aromatize to estrogen
- Do not 5-Alpha reduce
- Strong Anabolic Effects
- Suitable for women
- Brings the least amount of cell volume (non-retentive)
- Drives C.N.S. and Motor learning

30
Compound: Methenolone
Trade Name: Primobolan

Back-Bone Structure: DHT Column Behaves like: DHT

Theoretical Anabolic Rating: 88 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 44-57 Reality Androgenic Rating: Equivalent

Half-life: 10.5 Days (enanthate) 2-3 Days (acetate) Termination life: 50-60 Days

Impact on Hematology: Low Impact on Blood Pressure: Low

Impact on Kidneys: Low -Moderate Impact on Liver: Low – Moderate

Impact on Libido: Moderate Impact on Cardiovascular: Moderate

Aromatization Rate: None Administration: Oral or Injectable

Recommended Duration: ~8-20 Weeks Detection Time: 4-5 Months/4-5 Weeks

Retentive Qualities: Low Ester(s): Enanthate/C-17b Methyl Group

Anabolic/Androgenic Effects: A relatively strong anabolic with very low androgenic effects and no
estrogenic effects. Even at higher doses. Methenolone acetate (oral) is less anabolic due to liver metabolism, possibly
requiring higher dosages to equate anabolic properties.

Performance/Relevant Effects: Excellent at driving motor pattern learning and central nervous system
development for performance athletes. Drives the accrual of lean contractile tissue with minimal weight gain.

Common Usages/Synergy: Beneficial for athletes looking for increases in lean contractile tissue with minimal
overall weight gain, such as weight restricted or speed sports. Primobolon is a good choice for pre-contest
bodybuilders but also during mass building phases stacked with compounds that bring more retentive qualities such
as Testosterone and its derivatives (center column) or Nandrolone and derivatives (right column).

Female Considerations: The low androgenic effects make both injectable and oral versions viable options for
females with the orals offering greater control on blood hormone levels. The inclusion of stronger anabolic DHT’s like
Stanozolol or Oxandrolone can offer greater increases in anabolic effects.

Description: Introduced in 1962 for medical use, Primobolan is an injectable or orally bioavailable
derivative of Dihydrotestosterone with an additional double bond between carbons 1&2, increasing the anabolic
properties. Oral Primobolan is NOT the typical C-17a alteration, instead the rare C-17b alteration induces minimal
stress on the liver. One of the most “advanced” Anabolic Androgenic Steroids still marketed for medical use in some
countries, but now mostly used for physique and athletic performance improvements. The Injectable version
(enanthate) is much cheaper than its Oral option (acetate). Primobolan is considered one of, if not, the “safest”
anabolic steroids available. Metabolized in the kidneys and liver and excreted through urine and feces.

31
Compound: Drostanolone
Trade Name: Masteron

Back-Bone Structure: DHT column Behaves like: DHT

Theoretical Anabolic Rating: 62-130 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 25-40 Reality Androgenic Rating: Equivalent

Half-life: 2 Days-Propionate/10.5 Days-Enanthate Termination life: ~10 Days

Impact on Hematology: Low Impact on Blood Pressure: Moderate

Impact on Kidneys: Moderate Impact on Liver: Low

Impact on Libido: Moderate Impact on Cardiovascular: Moderate

Aromatization Rate: None Administration: Injectable

Recommended Duration: ~8-20 Weeks Detection Time: ~3 Months

Retentive Qualities: Low Ester(s): Propionate/Enanthate

Anabolic/Androgenic Effects: Strong anabolic from the added 2-a methyl group, with mild androgenic
effects and cannot aromatize to estrogen. In fact, Masteron has estrogenic suppressive qualities, clinically acting like
a selective estrogen receptor modulator (SERM) but also anecdotally as an aromatize inhibitor (AI) in lean individuals.

Performance/Relevant Effects: Excellent at driving motor pattern learning and central nervous system
development for performance athletes. Strong driver of lean contractile tissue accrual with minimal weight gain. Can
cause hypertension via vasoconstriction, not edema like most estrogenic anabolics. Can reduce sex hormone binding
globulin (SHBG).

Common Usages/Synergy: Beneficial for athletes looking for increases in lean muscle and strength, without
overall gains of sheer mass such as weight restricted or speed sports. Bodybuilders enjoy the anti-estrogenic nature of
Masteron leading into a pre-contest phase. During mass building phases, it also stacks well with compounds that
bring more retentive qualities such as Testosterone and its derivatives or Nandrolone and derivatives like Primobolan.

Female Considerations: The low androgenic effects make Masteron a viable option for females, with the orals
offering greater control on blood hormone levels. The inclusion of stronger anabolic DHT’s like Stanozolol or
Oxandrolone can offer greater increases in anabolic effects.

Description: First described in 1959, Masteron was initially used as an inoperable treatment for breast
cancer. It is an injectable derivative of Dihydrotestosterone (DHT) available most commonly in a propionate or
enanthate ester. It has been altered with an introduction of a methyl group at carbon 2 (alpha) to increase its
anabolic properties with the resistance to metabolism by the 3-hydroxysteroid dehydrogenase enzyme in skeletal
muscle tissue. Metabolized by the kidneys and liver and excreted through urine and feces.

32
Compound: 1-Testosterone
Trade Name: Dihydroboldenone (DHB)

Back-Bone Structure: Center Column Behaves like: DHT

Theoretical Anabolic Rating: 200 Reality Anabolic Rating: Higher

Theoretical Androgenic Rating: 100 Reality Androgenic Rating: Lower

Half-life: 10-12 Days Termination life: ~50-60 Days

Impact on Hematology: Low-Moderate Impact on Blood Pressure: Moderate

Impact on Kidneys: Moderate Impact on Liver: Low-Moderate

Impact on Libido: Low Impact on Cardiovascular: Moderate

Aromatization Rate: None Administration: Oral or Injectable

Recommended Duration: ~8-20 Weeks Detection Time: Unknown

Retentive Qualities: Low-Moderate Ester(s): Cypionate

Anabolic/Androgenic Effects: DHB is much more anabolic than androgenic, with no ability to convert to
estrogen due to being 5-alpha reduced. There should be no water retentive qualities, gyno, etc.

Performance/Relevant Effects: Minimal neurological benefits, but an increase in mostly lean contractile
tissue could be beneficial to sports performance. Can cause some people bouts of insomnia and night sweats. DHB
can help improve insulin sensitivity in a relatively linear fashion with the escalation of dosages.

Common Usages/Synergy: A profound ability to build lean tissue, with reports of 10-15lbs of lean muscle
mass gained over 8 weeks. Beneficial for pre-contest or “off-season” blocks as well as athletes looking for lean tissue
with minimal water weight. The inability to convert to estrogen makes the need for an additional aromatizing
compound to be used in conjunction with DHB a consideration.

Female Considerations: The androgenicity of DHB is like testosterone and the sheer power makes virilization
symptoms a real concern with female usage. If chosen, minimal dosages should be considered with immediate
cessation upon any signs of unwanted side-effects. Not recommended.

Description: DHB differs from testosterone with a 1-2 double bond instead of a 4-5 double bond on the A-
ring. It is a 5-alpha reduced form of Boldenone, structurally almost identical to Primobolan but with an approximate
potency 10x’s greater. Available in tablet form (much more liver toxic), transdermal gel or injectable versions. The
injectable version of DHB is the preferred method of administration, attached to a cypionate ester which seems to
provide the most effective method of application, though post injection pain is common. DHB is one of the most
powerful compounds on the market milligram for milligram. A 200mg injection of DHB is equivalent to ~400-600mg
of Testosterone. Metabolized at the liver and excreted thru urine.

33
Compound: Mesterolone
Trade Name: Proviron

Back-Bone Structure: DHT Column Behaves like: DHT

Theoretical Anabolic Rating: 100-150 Reality Anabolic Rating: Lower

Theoretical Androgenic Rating: 30-40 Reality Androgenic Rating: Higher

Half-life: 12-13 Hours Termination life: 2.5-3 Days

Impact on Hematology: Low Impact on Blood Pressure: Low

Impact on Kidneys: Low Impact on Liver: Low

Impact on Libido: Low Impact on Cardiovascular: High

Aromatization Rate: None Administration: Oral

Recommended Duration: 1 day/kg-bodyweight Detection Time: ~8 Days

Retentive Qualities: Low Ester(s): C-1a Methyl Group

Anabolic/Androgenic Effects: Despite the established ratio, Proviron has a much strong androgenic effect
than anabolic effect, due to the rapid reduction to inactive metabolites in muscle tissues where 3-Hydroxysteroid
dehydrogenase enzymes levels are high. The anabolic effects are too weak to be beneficial for muscle building.
Estrogenic effects of Proviron are low.

Performance/Relevant Effects: Proviron has a strong affinity to sex hormone binding globulin (SHBG),
increasing the activity of other steroids by allowing a greater percentage to remain in an unbound state. Proviron
acts as an anti-estrogen with its ability to antagonize the aromatase enzyme and benefits athletes looking for
increased androgen levels. Proviron binds to the aromatase enzyme but cannot alter it, thereby inhibiting its effects.

Common Usages/Synergy: Common with bodybuilders in pre-contest diets as well as athletes looking only for
androgenic effects. The ability to occupy SHBG can potentially increase the effects of other compounds. The anti-
estrogenic properties of Proviron are similar to Masteron but without the anabolic qualities.

Female Considerations: Despite the androgenic rating, Proviron is a strong androgen and should be avoided
by women concerned with virilization effects. However, low doses have been used to increase definition in pre-
contest women.

Description: Schering developed Proviron in 1934, stating it was the first medication in clinical practice to
treat hormone deficiencies in men. It is widespread in clinical use today with a long list of effectiveness and safety,
though its use in performance is very limited, as it is a very weak anabolic compound with some unique androgenic
properties, specifically with its effects of SHBG and anti-estrogenic effects. Metabolized by the liver and excreted
though urine.

34
Compound: Furazabol
Trade Name: Frazalon/Miotolan

Back-Bone Structure: DHT Column Behaves like: DHT

Theoretical Anabolic Rating: 270-330 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 73-94 Reality Androgenic Rating: Equivalent

Half-life: 4 Hours Termination life: ~20 Hours

Impact on Hematology: Low Impact on Blood Pressure: Low

Impact on Kidneys: Low-Moderate Impact on Liver: High

Impact on Libido: Low Impact on Cardiovascular: High

Aromatization Rate: None Administration: Oral

Recommended Duration: 1 day/kg-bodyweight Detection Time: ~5-7 Days

Retentive Qualities: Low Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: Relatively strong anabolic with only mild androgenic qualities. Furazabol does
not aromatize in the body, so estrogenic side effects like gynecomastia, water retention, etc. should not be seen.

Performance/Relevant Effects: Performance athletes looking to minimize total weight gain benefit from the
use of Furazabol. It was a popular steroid among Olympic athletes in the 80’s before testing officials had identified
the compound, and therefore not test for it. The controversial thoughts of Oxandrolone’s ability to lower cholesterol
is said to be similar with Furazabol, though this cannot be verified and therefore should not be considered for people
with high cholesterol.

Common Usages/Synergy: Most commonly used with bodybuilders in a pre-contest diet phase where the
additional androgenic properties with no estrogenic activity can bring out that “hardness” quality and dense looking
muscles to a lean physique. Furazabol muscle building qualities could be furthered improved with additional anabolic
compounds like Deca or EQ.

Female Considerations: Though the androgenic ratio is higher than with other DHT’s, Furazabol can still be a
viable option for female use. Virilization side effects are still a concern, so minimal dosage and durations should are
advised.

Description: Furazabol is a Japanese oral anabolic derivative of dihydrotestosterone with an addition a

methyl group at carb on 17-alpha, improving oral bioavailability and the attachment of a furazan group to the A-ring,
replacing the normal 3-keto group. The addition of the furazan group is what gives Furazabol the increased anabolic
properties while reducing the androgenicity. Low dose Furazabol is seemingly less suppressive to natural testosterone
production. It is metabolized mainly in the liver and excreted through urine.

35
Compound: Oxandrolone
Trade Name: Anavar

Back-Bone Structure: DHT Column Behaves like: DHT

Theoretical Anabolic Rating: 322-630 Reality Anabolic Rating: Lower

Theoretical Androgenic Rating: 24 Reality Androgenic Rating: Equivalent

Half-life: 8-9 Hours Termination life: 2-3 Days

Impact on Hematology: Low Impact on Blood Pressure: Low

Impact on Kidneys: Moderate-High Impact on Liver: Low-Moderate

Impact on Libido: Low-Moderate Impact on Cardiovascular: Moderate

Aromatization Rate: None Administration: Oral

Recommended Duration: 1 day/kg-bodyweight Detection Time: ~3 Weeks

Retentive Qualities: Low Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: 6x’s more anabolic than testosterone with significantly lower androgenic
effects. Already 5-alpha reduced, so it cannot aromatize in the body, meaning estrogenic effects aren’t a concern.
Gyno, water retention etc. should not be seen.

Performance/Relevant Effects: Great for increasing lean quality tissue with minimal water weight. Can
increase blood thinning effects of anticoagulant drugs and inhibit the absorption of oral diabetic medication.

Common Usages/Synergy: Bodybuilder use is mostly pre-contest dieting, but mass gaining phases can also
benefit from the added anabolic properties when stacked with center or right column drugs. Anavar works well with
competitive speed and anaerobic performance athletes or in weight restricted sports. Anavar displays a “1 + 1 = 3”
synergy with Oral Turinabol, where 10mg of Anavar + 10mg of Tbol offers a 30mg anabolic response benefit.

Female Considerations: Popular choice for females due to the low androgenic effects and strong ability to
build lean tissue. Minimal dose and duration should still be considered to avoid potential virilization symptoms.

Description: Oxandrolone was first described in 1962 and introduced into medicine a few years later as
Anavar. It is a 5-alpha reduced, 17-alpha alkylated oral steroid derived from dihydrotestosterone with a substitution
of the 2-carbon atom for an oxygen atom, designed to have a strong distinction between its anabolic (increased) and
androgenic properties (decreased). Milligram for milligram, Anavar has approximately 6 times the anabolic ability of
testosterone with a fraction of the androgenic effect, known to increase strength and muscle gains. Clinically proven
effective in lean tissue retention following surgery, trauma, etc. and for women with osteoporosis. It can lower total
cholesterol, but does so by the reduction in HDL levels, skewing the HDL/LDL ratio in the wrong direction. Metabolized
primarily by the kidneys, reducing liver toxicity, and excreted through the urine and feces.

36
Compound: Stanozolol
Trade Name: Winstrol (oral)

Back-Bone Structure: DHT Column Behaves like: DHT

Theoretical Anabolic Rating: 320 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 30 Reality Androgenic Rating: Equivalent

Half-life: ~8-9 Hours Termination life: ~2 Days

Impact on Hematology: Low Impact on Blood Pressure: Low

Impact on Kidneys: Moderate-High Impact on Liver: Moderate

Impact on Libido: Low-Moderate Impact on Cardiovascular: High

Aromatization Rate: None Administration: Oral or Injectable

Recommended Duration: 1 day/kg-bodyweight Detection Time: 10 days - 2 Months

Retentive Qualities: Low Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: Chemical alterations make Winstrol a strong anabolic steroid with low
androgenic qualities. It cannot aromatize in the body due to already being 5-alpha reduced, so estrogenic symptoms
should not be seen.

Performance/Relevant Effects: Drives lean muscle mass without water retention and stimulates appetite.
Prevents the loss of lean tissue following surgery, trauma, etc. and bone mass in osteoporosis. Architectural changes
in ligaments/tendons may cause connective tissue to be stronger but less pliable

Common Usages/Synergy: Bodybuilder use is mostly pre-contest dieting, but mass gaining phases can also
benefit from the strong anabolic properties when stacked with center or right column drugs. Athletes performing
linear movements like powerlifters and sprinters, as well as athletes looking for increases in strength and lean tissue
gain without sheer gains in size or bodyweight like weight restricted sports can all benefit from Winstrol.

Female Considerations: Virilization symptoms are always a concern, but Winstrol is commonly used due to
very low androgenic properties. Dose and duration should still be considered.

Description: Stanazolol was first described in 1959 and released as Winstrol in 1962. Oral Winstrol is a 5-
alpha reduced modified version of dihydrotestosterone (an injectable version is available) that differs with the
addition of a methyl group at carbon 17 for improved oral administration and an attached pyrazol group to the A-
ring, replacing the 3-keto group to increase its anabolic strength and reduce androgenicity. This is a similar alteration
as Furazabol. Winstrol cannot aromatize in the body and has a strong binding affinity to sex hormone binding
globulin (SHBG), which could increase the potency of unbound hormones from other compounds when taken together.

37
Compound: Oxymetholone
Trade Name: Anadrol

Back-Bone Structure: DHT Column Behaves like: 19-Nor

Theoretical Anabolic Rating: 320 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 45 Reality Androgenic Rating: Higher

Half-life: 8-9 Hours Termination life: ~2-3 Days

Impact on Hematology: High Impact on Blood Pressure: High

Impact on Kidneys: High Impact on Liver: High

Impact on Libido: High Impact on Cardiovascular: High

Aromatization Rate: None Administration: Oral

Recommended Duration: 1 day/kg-bodyweight Detection Time: ~8-9 Weeks

Retentive Qualities: High Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: Very potent anabolic steroid with theoretically low androgenic qualities,
though reality shows Anadrol to have much higher androgenic properties. Despite being 5-alpha reduced, Anadrol
can activate the estrogen receptor, therefore a highly estrogenic steroid. Gyno, water retention, etc. are a concern.

Performance/Relevant Effects: Excellent selection for sheer weight gain. Anadrol is also a very strong
driver of hematology, increasing erythropoietin up to 5 times. Water retention is very high, which can also increase
blood pressure.

Common Usages/Synergy: Most common in sports where sheer size is desired. Novice steroid users can gain
upwards of 20-30lbs in 6 weeks. Strength athletes can also benefit from the weight increase, as it can improve
leverages when lifting, and the water retention makes for better muscular contractions and joint lubrication. The
quick weight increase however, can also increase the chance of muscle tears from increased stress on connective
tissue. Commonly used as a “kick-start” for mass building phases when long acting esters are used.

Female Considerations: This is not a drug recommended for female use.

Description: First described in 1959 then made available in 1960, Oxymetholone is a modified form of
Dihydrotestosterone differing with the addition of a methyl group at Carbon 17-alpha, protecting the steroid during
oral administration. An added 2-hydroxymethylene group hinders its metabolism by the 3-hydroxysteroid
dehydrogenase enzyme but also causes an affinity to the estrogen receptor, thus increasing anabolism but also
estrogenic effects of the drug. Anadrol has a strong ability to drive red blood cell production and is considered one of
the strongest compounds commercially available on the market, with dramatic effects. Metabolized by the liver and
kidneys, excreted through urine.

38
Compound: Methasterone
Trade Name: Superdrol/Methyldrostanolone

Back-Bone Structure: DHT Column Behaves like: DHT

Theoretical Anabolic Rating: 20 Reality Anabolic Rating: Higher

Theoretical Androgenic Rating: 400 Reality Androgenic Rating: Lower

Half-life: 8-12 Hours Termination life: ~3-4 Days

Impact on Hematology: Moderate Impact on Blood Pressure: High

Impact on Kidneys: High Impact on Liver: High

Impact on Libido: Moderate Impact on Cardiovascular: High

Aromatization Rate: None Administration: Oral

Recommended Duration: 4-6 weeks max Detection Time: ~10 Days

Retentive Qualities: Low-Moderate Ester(s): C-17a Methyl Group

Anabolic/Androgenic Effects: Very strong anabolic steroid with low androgenic properties, despite the
theoretical rating. The low androgenic effects are similar to Anavar and Winstrol. Superdrol is already 5-alpha
reduced, so aromatization and estrogenic effects shouldn’t be a concern.

Performance/Relevant Effects: Several cases of liver toxicity have been reported. Superdrol causes drastic
increases in 1RM strength, capable of adding significant amounts of lean muscle mass with relatively low increases in
total bodyweight. New potential biomarkers detected in chromatography can detect Superdrol for up to 10 days.
Increases in glucose uptake has been reported, increasing the “pump” experienced during weight training.

Common Usages/Synergy: A high degree of strength gains and lean tissue accrual is common. Bodybuilders
in pre-contest or “off-season” blocks can benefit from the high anabolic potency, as due powerlifters, weightlifters,
etc. Weight gain is higher than other DHT’s making superdrol potentially problematic for weight restricted athletes.

Female Considerations: The low androgenic effects in theory, make this a viable option for women however
the potency of the drug is the concern. The typical Superdrol tablets are far too strong for women, making dosing a
challenge. More user friendly DHT options are likely better choices.

Description: Superdrol was first described in 1960 but never marketed for clinical use. It is a DHT
derivative most closely resembling Masteron, only much stronger, and with an additional C-17 Alpha methyl group,
making it orally bioavailable. Superdrol has a very high anabolic to androgenic ratio (20:1) and cannot aromatize,
therefore estrogenic side effects should not be a concern. 1 day per kg duration of orals is a great “rule of thumb”
generally recommended here in this book, however Superdrol may require shorter durations due to the potency. It
has been suggested to keep duration 4-6 weeks max. Metabolized in the liver and excreted through urine.

39
 19-Nor
19-nor derivatives create the Right-side column of the Family Tree

- Good for “Fullness”

- Lower Aromatization Rate
- Strong Anabolic Effects
- Weak Androgenic Effects
- Greatest cell volume of all columns (retentive qualities)
- Suitable For Women
- Cause the most amount of weight gain

40
Compound: Nandrolone
Trade Name: Deca-Durabolin

Back-Bone Structure: 19-Nor Column Behaves like: 19-Nor

Theoretical Anabolic Rating: 125 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 37 Reality Androgenic Rating: Equivalent

Half-life: 12-15 days Termination life: 10 Weeks

Impact on Hematology: Moderate Impact on Blood Pressure: Moderate

Impact on Kidneys: Moderate Impact on Liver: Low

Impact on Libido: Low Impact on Cardiovascular: Moderate

Aromatization Rate: Moderate (E1 more so) Administration: Injectable

Recommended Duration: ~8-20 Weeks Detection Time: ~18 Months

Retentive Qualities: High Ester(s): Decanoate

Anabolic/Androgenic Effects: Relatively strong anabolic with weak estrogenic properties. Estrogen
conversion is ~20% of testosterone, occurring mostly in the liver. The 5-a reductase enzyme metabolizes nandrolone
to a much weaker dihydronandrolone (DHN), reducing its androgenic effects.

Performance/Relevant Effects: Estrogen conversion is mostly Estrone (E1) instead of Estradiol (E2).
Increased progesterone effects are common with Nandrolone, causing similar symptoms as elevated estrogen levels,
including “Deca-dick.” There is a potential increase in connective tissue synthesis as well as joint hydration. Deca-
Durabolin is seen as a slow quality muscle builder. Drug tested athletes need to consider detection times.

Common Usages/Synergy: The strong anabolic weak androgenic nature makes this drug popular in all
professional sports, for both men and women. Increased results in muscularity & definition are seen when used in
conjunction with other non-aromatizing steroids. A favorite with pre-contest bodybuilders and “off-season” training
blocks for competitive athletes. Nandrolone can also be found with a shorter half-life propionate ester.

Female Considerations: Low androgenic effects and long ester half-life, allow for less frequent injections.
Dosing with a 1-3mg/kg, bi-weekly is a great protocol for women. Virilization effects are still possible.

Description: First described in 1960, Deca-Durabolin is the brand name for the injectable steroid
nandrolone decanoate. The decanoate ester has a very long half-life, clinically designed for reducing injection
frequency to 3-4 weeks. It is a modified version of nandrolone, structurally like testosterone but with the removal of a
carbon atom at the 19th position and a decanoic acid attached to the 17-beta hydroxyl group, reducing androgenicity.
Deca-Durabolin provides a spike in nandrolone release 24-48 hours following intramuscular injection, which steadily
declines to baseline ~14-21 days later. Deca-Durabolin is metabolized mostly in the liver and excreted through urine.

41
Compound: Trenbolone
Trade Name: Trenabol (enanthate)

Back-Bone Structure: 19-Nor Column Behaves like: 19-Nor

Theoretical Anabolic Rating: 500 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 500 Reality Androgenic Rating: Equivalent

Half-life: ~10.5 Days (enanthate) Termination life: ~50-60 Days

Impact on Hematology: High Impact on Blood Pressure: High

Impact on Kidneys: High Impact on Liver: High

Impact on Libido: Low-High Impact on Cardiovascular: High

Aromatization Rate: None Administration: Oral or Injectable

Recommended Duration: ~8-20 Weeks Detection Time: ~4-5 Months

Retentive Qualities: Moderate Ester(s): Enanthate

Anabolic/Androgenic Effects: Very strong anabolic and androgenic steroid, ~3x’s stronger than
testosterone. Cannot aromatize to estrogen, but progestational activity is high and can cause similar side effects such
as gynecomastia. Cannot be 5-alpha reduced

Performance/Relevant effects: Trenbolone is excellent at driving aggression. Though Trenbolone is not C-

17a alkylated, severe liver toxicity has been seen in higher dosages, along with left ventricular hypertrophy, night
sweats and insomnia. Trenbolone is also known for causing “tren-cough,” over-taxing thyroid production, driving
appetite and massively suppressing endogenous testosterone production.

Common Usages/Synergy: Common in pre-contest dieting phases to help achieve a “harder” look. The lack of
estrogenic activity makes it a good complimentary drug when used with other aromatizing steroids for “off-season” or
mass building phases. Trenbolone is excellent at increasing muscle hardness, definition, and strength with minimal
water retention. Progestins have a strong synergy with estrogen in mammary tissue, making gynecomastia a real
concern even without excessive estrogen.

Female Considerations: This strong androgenic drug is not recommended for female use.

Description: Trenbolone is available with the shorter ester acetate, the longer ester
hexahydrobenzylcarbonate (known as Parabolon) and the more common enanthate. The slow acting esters of
trenbolone have been around since 1967 but was not released for commercial sale until 2004. It is a modified form of
nandrolone with an additional double bond at Carbon 9 and a second double bond at Carbon-11, preventing
aromatization and increasing binding affinity to the androgen receptor. Metabolized in the liver and excreted
through urine.

42
Compound: Trestolone (acetate)
Trade Name: MENT

Back-Bone Structure: 19-Nor Column Behaves like: 19-Nor

Theoretical Anabolic Rating: 2300 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 650 Reality Androgenic Rating: Equivalent

Half-life: Unknown Termination life: Unknown

Impact on Hematology: Low Impact on Blood Pressure: Moderate

Impact on Kidneys: High Impact on Liver: Low

Impact on Libido: High Impact on Cardiovascular: High

Aromatization Rate: Low-Moderate Administration: Injectable

Recommended Duration: ~8-20 Weeks Detection Time: Unknown

Retentive Qualities: High Ester(s): Acetate

Anabolic/Androgenic Effects: Very powerful anabolic, moderate androgenic and estrogenic drug. However,
a potent 7-alpha-methyl-estradiol metabolite is produced, making gynecomastia and water retention problematic.
Progestational activity is considered moderate to high, but limited research in humans is available. Unlike most
nandrolone derivatives, MENT is resistant to 5a reduction due to the C-7 alkylation and cannot be converted to a
“dihydro” metabolite.

Performance/Relevant Effects: Increased androgenicity can cause a feeling of well-being but can also
heighten aggression (anecdotal). MENT has a strong effect on increasing libido but also very powerful in suppressing
natural testosterone production.

Common Usages/Synergy: Building lean contractile tissue in pre-contest dieting and/or “off-season” training
blocks. 10-20mg of MENT is ~equivalent to 75-100mg of Trenbolone.

Female Considerations: MENT is incredibly potent, so effective dosages for women would likely need to be in
micrograms, making this compound inconvenient for female use. The inability to be 5-alpha reduced increases
androgenic effects, making MENT unsuitable for most females. Virilization symptoms are a concern.

Description: MENT is short for methylnortestosterone, discovered in 1963 and studied further in 1990 as
an oral contraceptive and hormonal replacement steroid for men. Similar to nandrolone, but with a methyl group
added to the 7-alpha carbon position, blocking 5-alpha reductase, increasing the anabolic potency and androgenicity
effects. A carboxylic ester is also attached to the 17-beta hydroxy group, extending injection half-life. The C-7a
alkylation allows for moderate oral bioavailability but is not as effective as the common C-17 alteration, making
MENT most often found as an injection or implantation steroid. Metabolized in the liver and excreted through urine.

43
Compound: Oxabolone
Trade Name: Steranabol Ritardo

Back-Bone Structure: 19-Nor Column Behaves like: 19-Nor

Theoretical Anabolic Rating: 50-90 Reality Anabolic Rating: Higher

Theoretical Androgenic Rating: 20-60 Reality Androgenic Rating: Lower

Half-life: ~12-15 Days Termination life: ~8-10 Weeks

Impact on Hematology: Low Impact on Blood Pressure: Low

Impact on Kidneys: Low Impact on Liver: Low

Impact on Libido: Low Impact on Cardiovascular: Low

Aromatization Rate: None Administration: Injectable

Recommended Duration: ~8-20 Weeks Detection Time: Unknown

Retentive Qualities: Low Ester(s): Cypionate

Anabolic/Androgenic Effects: Steranabol Ritardo is a strong anabolic steroid with low androgenic effects.
Already being 5-alpha reduced, aromatization is not a concern, so estrogenic side effects should not be seen.

Performance/Relevant Effects: The addition of the 4-hydroxyl group exhibits the potential in reducing
aromatization of other steroids, reducing their estrogenic effects and potentially interferes with 5-alpha reduction.
Less interference in common libido issues from Deca-Durabolin is also seen with Oxabolone.

Common Usages/Synergy: Being incredibly low dosed at just 12.5mg/ml, Oxabolone was not a very practical
drug for male athletes, especially bodybuilders. However, the weight gain is mostly lean contractile tissue, due to the
low estrogenic and potentially even anti-estrogenic effects of this drug. This could be beneficial in body building pre-
contest diets.

Female Considerations: The low dose availability and androgenicity of the Oxabolone would make it a
suitable drug for women.

Description: Officially becoming a controlled substance in 2005, Oxabolone is an injectable steroid

originally produced in Italy that is no longer available (except UGL). It is derivative of nandrolone (19-nor) with an
addition of a 4-hydroxyl group, creating the inability to convert to estrogen, and the seemingly potential of reducing
aromatization effects of other hormones. Oxabolone only came in 2ml ampules, with 12.5mg/ml concentrations,
making suitable dosages for significant gains in lean tissue growth highly impracticable. Even on the black market,
coming across true Oxabolone would be very difficult to do. It would however, be a potential benefit to both male
and female athletes. Oxabolone is metabolized in the kidneys and liver and excreted through urine.

44
Compound: Bolasterone
Trade Name: Myagen

Back-Bone Structure: Testosterone Behaves like: 19-Nor

Theoretical Anabolic Rating: 575 Reality Anabolic Rating: Equivalent

Theoretical Androgenic Rating: 300 Reality Androgenic Rating: Lower

Half-life: ~6 Hours Termination life: ~1-1.5 Days

Impact on Hematology: Moderate-High Impact on Blood Pressure: High

Impact on Kidneys: Moderate-High Impact on Liver: High

Impact on Libido: Moderate-High Impact on Cardiovascular: High

Aromatization Rate: High Administration: Injectable

Recommended Duration: 1 day/kg-bodyweight Detection Time: ~6 Weeks

Retentive Qualities: High Ester(s): Dual Methyl Groups (C-7&C-17)

Anabolic/Androgenic Effects: Very high anabolic, low androgenic effects with the unique ability to convert
to a potent dimethyl-estradiol, due to the dual methyl groups. This increases potential Estrogenic effects, especially at
higher than therapeutic dosages, making gynecomastia, water retention, etc. a real concern.

Performance/Relevant Effects: Performance enhancing effects are unknown in the literature, but Myagen
would seem to offer many of the same benefits as other derivatives of the 19-nor column but with a stronger ability to
drive hematology. High estrogenic effects are also common with Myagen

Common Usages/Synergy: Myagen has been mostly unavailable since the 1980’s and was rare in athletic
circles. Common usages would likely be similar to other 19-nor compounds, though documented evidence in
unknown. The strong aromatization effects would suggest high retention qualities and likely benefit the goal of sheer
gains in overall size and total bodyweight accumulation.

Female Considerations: Myagen has strong virilization tendencies and is not recommended for women.

Description: Bolasterone was first described in 1959 and sold in the U.S. during the 1960’s as Myagen. It
is structurally like methyltestosterone (C-17 alkylated), technically making it a testosterone derivative, but a slight
chemical alteration makes Myagen behave more like a 19-nor derivative. The difference is a second additional methyl
group attached at carbon 7 (C-7a), which interferes with the 5-a reductase enzyme, preventing the conversion to
dihydro, leaving Myagen much more anabolic than androgenic. The two methyl groups also reduce the affinity to
SHBG, leaving more unbound hormone in the blood, further increasing potency. The only similarity between
methyltestosterone and Myagen seems to be the shared ability of estrogen conversion. Myagen is metabolized in the
liver and kidneys and excreted through urine.

45
 Take-Away
You should now see why typical drug recommendations or “stack” protocols often
suggested, without the specific context of the individual is not the best way to approach
pharmacology. The intricacies of each compound, along with the individuals own
personal goals, lifestyle, and health markers, can make for very unique pharmacological
designs.

A Basic Summary is as Follows.

• KNOW YOUR GOAL! (crucial)
• An HONEST Self-assessment (crucial)
• Draw bloods before you begin the AAS course
• Determine your periodization
• Take your bodyweight in kilograms (kg’s)
• Multiply it by a factor of ~.5-20+/kg.
• Allow ~.5-3g/kg for center column compounds
• Fill the remaining milligrams with the best suited drug(s) for the goal
• Draw bloods at mid-point of the course (peak plasma)
• Consistently check your progress & health markers
• Draw bloods at the end of your course
• Bridge & Recover

As mentioned earlier, when designing your AAS protocols, you must look at the big
picture and periodize everything accordingly. Minimum effective dosage for the minimum
effective duration that brings the desired results is a great strategy to help preserve
health and longevity.

Progress Is Earned Not Given

46
 Family Tree Structures

DHT 19-Nor

Direct Derivatives

47
 Do Steroids Work?
The classic study by Shalender Bhasin, July 4th, 1996

YES they F’n Do!!

48
 Practical Application
Designing an anabolic androgenic steroid course should never be based only on
nonsensical criteria like "I wanna cut" or "I need more size". Instead, to help you be
successful, while simultaneously NOT tanking your health, many things should always be
considered before you can effectively design an AAS protocol.

As a minimum, we suggest the following

• Current Health Status (physiologically, orthopedically, psychologically, etc.)

• Current bloodwork (CBC panel as an absolute minimum)
• Previous bloodwork
• Previous Drug use (you must understand the half-life, esters, etc.)
• Timeline/Periodization
• Finances (Shit aint cheap)
• Accessibility/Legalities
• Risk aversion (health, legal, etc.)
• Personal/Social life (will drug use impede on anything)
• Goal(s)

Just like training, and selecting the appropriate exercise for the goal, a.ka. “Right Tool
for the Job”, AAS use should be thought of in the same way. What do I need
improvement on, and what AAS compound(s) (tools) would be best to accomplish this
task?

Responsible AAS use should be to improve on one's weaknesses!

With all this newly earned knowledge on anabolic

androgenic steroids, it's now time to put it into
practice with a few examples below.

49
WARNING!
The Following
Course Designs
Are Example’s
Only!!
50
 Example 1: The Risk Adverse Individual
A first course for a very risk adverse athlete or individual, seeking improvements in
muscle mass and/or body composition improvements, but also just wanting to "dip his
toes" into the world of sports pharmacology.

Course Protocol
For this scenario, the compound of choice will be Oxandrolone (Anavar).

Block 1 Block 2 Block 3

Week 1 Week 2 Week 3 Week 4 Week 5 Week 6 Week 7 Week 8 Week 9
10mg/day 10mg/day 10mg/day 20/=mg/day 20mg/day 20mg/day 30mg/day 30mg/day 30mg/day

• 9 weeks with three x’s 3-week blocks, each with a different dosing. The reason for
the escalating dosage is because as your body starts to sense the presence of
exogenous androgens, it will slowly start to suppress endogenous production.
• In the first 3 weeks you would have the 10mg/day of Oxandrolone on top of your
own natural production, but by the end, you may be experiencing some
testosterone suppression.
• The incremental increase of Oxandrolone every 3 weeks, starts to compensate for
the lowered endogenous production.
• Oxandrolone could also be swapped out for Stanazolol or Methenolone Acetate

Why Oxandrolone?
Oxandrolone is a DHT derivative (left side of the anabolic family tree) and like many DHT
derivatives, they are usually accessible, convenient for use (oral), cause minimal
estrogen/progesterone elevation, minimal water retention, are well tolerated by most
individuals and take a longer time to suppress natural endogenous production.

Center column Chlorodehydromethyltestosterone (oral Turinabol) could also be

considered because it also has minimal conversion to estrogen and acts more like a DHT
derivative, however, endogenous production would be suppressed much sooner.

19-nor oral AAS compounds are very rare with a higher risk of being fake, for this reason
they have been excluded from this list.

51
 Example 2: The Oral Only Course
This course uses only oral AAS compounds, for the purpose of either lean tissue accrual
or body re-composition. Well suited for a less risk adverse individual/athlete but could
also be a follow-up to the above protocol.

Course Protocol
For this scenario, the compounds of choice will be daily Oxandrolone (Anavar) for the
first 6 weeks, with the addition of daily Methandrostenolone (Dianabol), for the remaining
6 weeks.

Oxandrolone (Anavar) = Top Column Methandrostenolone (Dianbol) = Bottom Column

Block 1 Block 2 Block 3 Block 4

Week Week Week Week Week Week Week Week Week Week Week Week
1 2 3 4 5 6 7 8 9 10 11 12
10mg 10mg 10mg 20mg 20mg 20mg 20mg 20mg 20mg 30mg 30mg 30mg

0mg 0mg 0mg 0mg 0mg 0mg 20mg 20mg 20mg 20mg 20mg 20mg

• 12 weeks with four x’s 3-week blocks. The reason for the escalating dosage is as
your body starts to sense the presence of exogenous androgens, it will begin to
suppress endogenous production.
• In the first 3 weeks you would have the 10mg/day of Oxandrolone on top of your
own natural production, but by the end, you may be experiencing some
testosterone suppression.
• The incremental increase of Oxandrolone starts to compensate for the lowered
endogenous production
• Methandrostenolone is inserted at midpoint to fill in the TRT/HRT gap created by
the endogenous suppression
• Oxandrolone could also be swapped out for Stanazolol or Methenolone Acetate
• Methandrostenolone could be swapped out for Chlorodehydromethyltestosterone
(Oral Turinabol)
• Methandrostenolone aromatizes at ~40-50% rate of testosterone and will add
additional fullness, volume and help with endogenous suppression

19-nor oral AAS compounds are very rare with a higher risk of being fake, for this reason
they have been excluded from this list.

52
 Caution!
Remember, oral anabolic use can be deleterious to your health in many ways as
mentioned earlier in this book. See page 19 for a refresher. More importantly, oral AAS
compounds can be particularly toxic to the liver (hepatoxic), especially at higher
dosages and/or longer durations. Specifically, AAS compounds with a C-17-alpha
alkylated alteration.

Blood work and careful monitoring of liver values is good practice.

To help further reduce the potential burden, the rule of thumb is to NOT exceed your
bodyweight in kilograms, in your total daily dose in milligrams of oral anabolics, and to
not exceed your bodyweight in kilograms in consecutive days of oral use.

For example, a 90kg male should not exceed 90mg’s of oral anabolics in a day
and should not use orals anabolics for longer than 90 consecutive days.

A quick guide to liver health

Aspartate Aminotransferase – AST (SGOT) & Alanine Aminotransferase – ALT (SGPT) ares
liver enzyme that helps metabolize proteins and amino acids in the body. A damaged
liver can cause ALT and AST values to increase.

• ≤100 is just the cost of doing business.

• 100-200 is worth noticing and “You should have a plan”
• 200-300 is concerning and you should be in the very final stages of your plan
• >300 you should shut things down immediately!!

53
 Example 3: Injectable Only Course
This course protocol example is for an 85kg (187lbs) male, looking to improve overall
muscularity. A relatively strong and healthy individual with no issues in his bloodwork.
Hematology is well within range, kidney & liver functions are good, cholesterol values
are good, triglycerides are low, and estrogen is in the middle of the reference range with
no un-wanted estrogenic effects.

This is not his first course, and he fully understands the risks associated with AAS use.

Course Protocol
For this scenario, the compounds of choice will be Testosterone Enanthate and
Methenolone Enanthate (Primobolan)

Testosterone Enanthate = Top Column Methenolone (Primobolan) = Bottom Column

Block 1
Week 1 Week 2 Week 3 Week 4 Week 5 Week 6 Week 7 Week 8
200mg 200mg 200mg 200mg 200mg 200mg 200mg 200mg
100mg 100mg 200mg 200mg 300mg 300mg 400mg 400mg

Block 2
Week 9 Week 10 Week 11 Week 12 Week 13 Week 14 Week 15 Week 16
200mg 200mg 200mg 200mg 200mg 200mg 200mg 200mg
500mg 500mg 500mg 500mg 500mg 500mg 500mg 500mg

• 16-week course with 2 x’s 8-week blocks

• Testosterone stays constant as the Sports TRT backbone for quality of life
• Primobolan escalates in 2-week blocks (weeks 1-8) and levels out from weeks 9-16
• Methenolone is a very refined, sophisticated DHT derivative that is known to
produce very little unwanted side effects, causing very little increases in
aggression, water retention, etc.
• Methenolone is estrogen “neutral”, meaning it will not cause any additional
estrogen burden and being a DHT derivative, it can not 5-alpha reduce back to root
DHT.
• Both compounds use a long ester making for user friendly 1x per week injections

54
 Example 3: Cont’d

Course Considerations
The importance of bloodwork can really guide the athlete or individual into decisions that
could make the difference between a successful course of anabolic androgenic steroid
use and a potential disaster.

Here are a couple variables to consider and examples of modifications that could be
considered, in order to keep health markers where you want them while continuing to
make progress.

Example 1: If the athlete or individual has very low RBC and hemoglobin

• Could swap testosterone for a center column derivative like Boldenone

Undecylenate which is notorious for driving up hematology
• Boldenone converts to estrogen at ~40-50% of testosterone, meaning you might
need to increase the dose by 50-100%.
• Could also use Oxymetholone (Anadrol) instead, if estrogen levels and liver values
allow (remember that Anadrol is not only an oral AAS, but also highly estrogenic).

Example 2: If the athlete or indidividual has high estrogen issues

• Some or all of the Primobolan could be swapped out for Drostanolone (Masteron),
which has an estrogen lowering effect both by acting as an aromatase inhibitor
and by blocking estrogenic actions at the breast tissue level.

Example 3: If the athlete was lacking volume or is more ectomorphic

• Primobolan could be swapped out for a 19Nor compound like Nandrolone. This
compound increases water retention and adds fullness and volume.
• Nandrolone also converts to estrogen at a lower rate and mostly to E1 (estrone)
rather than classic E2 (estradiol)

All the above examples are just that, examples. As previously stated, a lot of data must
go into the process of course design!

55
 Example 4: Female Course
As I went over on pages 20 & 21, AAS use is incredibly powerful for females and extra
caution is always advised. Females produce much lower levels of endogenous
androgens compared to men, in fact only about 10% as much testosterone as males is
endogenously produced by female physiology. This is equivalent to approximately
1mg/day of testosterone.

For that reason, a much smaller dose and duration is needed to achieve exceptional
results.

This course protocol will be based on a 65kg female who is wanting to add some lean
contractile tissue and fullness to her frame but is concerned of virilization effects.

Course Protocol
For this scenario, the compound of choice will be Nandrolone. Both Deca-Durabolin or
Phenyl-propionate could be used.

Nandrolone Deca-Durabolin

Block 1 Block 2 Block 3

Week 1 Week 2 Week 3 Week 4 Week 5 Week 6
62.5mg/week 0mg/week 62.5mg/week 0mg/week 62.5mg/week 0mg/week

• 6 Week duration with 3 x’s 2-week blocks

• Nandrolone (Deca-Durabolin) is administered ONLY on weeks 1, 3 and 5 due to the
very long acting ester half-life. Giving a total of only 3 injections.

Nandrolone Phenyl-propionate (NPP)

Block 1 Block 2 Block 3

Week 1 Week 2 Week 3 Week 4 Week 5 Week 6
50mg/week 50mg/week 75mg/week 75mg/week 100mg/week 100mg/week

• 6 Week duration with 3 x’s 2-week blocks

• NPP is administered weekly, and escalates after each 2 week block
• NPP’s short ester half-life provides the need for more frequent injections but the
ability to stop the escalation of un-wanted effects from accumulating

56
 Example 4: Cont’d
The difference between the two Nandrolone course designs come down to the esters and
their half-life. The long half-life of Deca-Durabolin allows for half the injection frequency
compared to that of NPP. Convenient and preferred for any female that no feel
comfortable with more frequent injections

The short half-life of NPP requires more frequent injections but allows a quicker time the
drug needs to clear the body, if any undesirable effects become a concern.

Why Nandrolone?
Nandrolone is not entirely unknown to female physiology, it is similar to the native
version of testosterone produced by females during gestation, to support the extra
weight of the fetus without causing virilization.

Nandrolone’s anabolic properties are similar to testosterone but with significantly less
androgenic effects, greatly reducing the risk of virilization symptoms. The option of
either a very long or very short half-life gives females the option of the same drug but
with the choice of less frequent injections or more control over the body’s clearance
time of the drug.

What If I Don’t Want to Inject?

If a female user is wanting the convenience of oral anabolic androgenic steroids only,
DHT derivatives are really the only option.

• Methenolone Acetate (oral Primobolan)

• Oxandrolone (Anavar)
• Stanazolol (Winstrol)

The above AAS compounds are all viable options for consideration, but you must still
apply the same rules

• Duration = 3-9 weeks

• Dose = <1-3mg/kg
• Do not exceed your bodyweight in kg’s in terms of daily total milligrams
• Do not exceed your bodyweight in kg’s in terms of consecutive day usage

57
Intramuscular Injection Protocol

58
59
60
 Selecting Site Location
• Carefully select the site for injection, avoiding major blood vessels & nerves
• Use different sites each shot to prevent repeated injections in the same area
• Do not use areas that are bruised, tender, scarred from surgeries or swollen

61
62
63
64
65
66
Subcutaneous Injection Protocol

67
68
69
History of Anabolic Steroid Use

70
 World Anti-Doping Agency’s

Prohibited List – 2021

71
 World Anti-Doping Agency’s

Drug Violations in Sports

The desire for athletes to be the best in their chosen sport or athletic endeavor is
constantly met with ever-mounting pressures and has been around since the very
creation of sport itself. Though considered unethical, its very clear that the use of
performance enhancing substances is very real and exists across essentially all sports
and even age groups.

72
 Urban Dictionary
Alpha: This refers to the location of alterations made to AAS compounds. Alpha being an alteration done to the
outside of the carbon ring structure vs. beta being an alteration made to the inside of the carbon ring structure.

Anabolic-Androgenic Ratio: Each anabolic-androgenic steroid is characterized by an anabolic:androgenic ratio,

testosterone being the reference mark with a ratio of 100:100

Anabolic Androgenic Steroid (AAS): Synthetic man-made derivatives of the male sex hormone Testosterone
or it’s derivatives that offer both anabolic and androgenic effects on the body.

Aromatization: Is the synthesis of Estrogen. An irreversible conversion of Testosterone to estrogen by the

aromatase enzyme, particularly in the liver and adipose tissue, but also occurring in the gonads, central nervous system,
and skeletal muscle tissue.

Beta: This refers to the location of alterations made to AAS compounds. Beta being an alteration made to the inside
of the carbon ring structure vs. alpha being an alteration made to the outside of the carbon ring structure.

Bridge: Commonly referred to as “off” or worse….a “cruise” is the time between drug courses. This is either TRT/HRT
or Sports TRT/HRT but the use of no AAS compounds at all is also an option. A Bridge duration should be equal to the
course duration. Exceptions may exist.

Course: Commonly referred to as a “cycle” or worse…a “blast”. This is the duration of time you are using AAS
compounds, or AAS milligrams are at their highest.

C-17a-Alkylation: An AAS compound, where the hydrogen atom at the 17th alpha position is replaced with a
carbon atom, making the drug more orally bioavailable.

Dihydrotestosterone (DHT): A 5-alpha reduced testosterone metabolite, 3-4 times more potent than
testosterone.

Elimination Life: Is the length of time required for the plasma concentrations of a given drug to fall below a
clinically relevant concentration, considering the drug “eliminated” Typically, an elimination life is equal to 4-5 times the
half-life.

Ester: Carboxylic acids attached to the 17-beta hydroxyl group of the testosterone molecule to increase the active life
span of the steroid. Different esters allow for different half-lives. See page 17.

Family Tree: Comprised of 3 main columns, Testosterone, DHT, 19-Nor’s and their derivatives.

Half-Life: Is the length of time required for the plasma concentration of a given drug to decrease by half of the
starting dosage in the body, or by 50%

Hormone Replacement Therapy (HRT): Therapeutically dosed hormones (NOT testosterone) at

approximately .5-1milligram per kilogram of bodyweight.

73
-Hydroxysteroid Dehydrogenase Enzyme (3-a HSD): An enzyme found in skeletal muscle tissue (and
other tissues of the body) that metabolizes and inhibits the anabolic activity of certain hormones.

Nandrolone: Also known as 19-nortestosterone (19-nor), is a naturally occurring hormone almost identical to
testosterone but lacking a carbon atom at the 19th position.

Peak Plasma: The length of time required for the plasma concentration of a given drug to reach steady-state blood
plasma levels (blood levels) in the body with regular interval administrations. Typically, steady-state levels are reached
with regular administrations after 4-5 times the half-life

Plasma: The yellow-ish fluid part of the blood. Plasma helps maintain blood pressure, pH balance and carries
vitamins, proteins, glucose, electrolytes, hormones, oxygen, etc.

Pharmacodynamics (PD): Describes what the DRUG does to the Body. It is the relationship between the drug
concentration (dose) at the site of action and the biochemical and physiological effects it has on the body. PD is
determined by receptor binding and sensitivity, post-receptor effects and chemical interactions.

Pharmacokinetics (PK): Describes what the BODY does to the Drug. It is the journey the drug takes through the
body. PD is determined by 4 principles, Absorption, Distribution, Metabolism, Excretion (ADME).

Reality Anabolic Rating: Comparison of “real world results” the compound has at driving anabolic qualities vs.
the theoretical anabolic numerical rating. Indicated with a “Lower”, “Equivalent”, or “Higher” score

Reality Androgenic Rating: Comparison of “real world results” the compound has at driving androgenic
qualities vs. the theoretical androgenic numerical rating. Indicated with a “Lower”, “Equivalent”, or “Higher” score.

“Sports” Hormone Replacement Therapy: Athletically dosed hormones (not testosterone) for the “alpha”
individual at approximately 2-3mg/kg of body weight.

“Sports” Testosterone Replacement Therapy: Athletically dosed hormones, specifically testosterone for
the “alpha” individual at approximately 2-3mg/kg of body weight.

Testosterone: The primary endogenous male sex hormone, manufactured by the Leydig’s cells in the testes.

Testosterone Replacement Therapy: Therapeutically dosed testosterone at approximately .5-1milligram

per kilogram of bodyweight.

Theoretical Anabolic Rating: How much muscle can the compound build per unit/mg compared to the
reference 100 of Testosterone.

Theoretical Androgenic Rating: How much masculinizing qualities (body hair, virilization, etc.) are increased
per unit/mg compared to the reference 100 of Testosterone.

74
Virilization: or “Masculinization” is the biological process in the development of adult male characteristics in young
males of females. Most of the changes of virilization are produced by androgens. Women are hyper-sensitive to
virilization effects from AAS use.

5-Alpha Reductase (5AR): An enzyme involved in steroid metabolism, reducing the hormone to its DHT
derivative.

75
 Contact Information & Further Resources

For coaching inquiries, consultations or more information, please visit

hollywoodbuilt@gmail.com
Hollywoodbuilt@teamevilgsp.com
mirceabjr@teamevilgsp.com

For a more comprehensive look at all things anabolic, please visit

www.members.teamevilgsp.com
Or

www.hollywoodbuilt.com

76
 Suggested Reading

Available Only At
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https://hollywoodbuilt.com/shop/

77
 Suggested Reading

Available Only At
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https://hollywoodbuilt.com/shop/

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 Coming Soon….

The Complete AAS Fundamental

Cheat-Sheet for Females Muscular
& What You Need To Anatomy
Know!

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“This Shit’s

Chess,
it ain’t
Checkers”
-A.Harris

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