ORC and the intra-S-phase checkpoint: a threshold regulates Rad53p activation in S phase

Genes Dev. 2002 Dec 15;16(24):3236-52. doi: 10.1101/gad.239802.

Abstract

The intra-S-phase checkpoint in yeast responds to stalled replication forks by activating the ATM-like kinase Mec1 and the CHK2-related kinase Rad53, which in turn inhibit spindle elongation and late origin firing and lead to a stabilization of DNA polymerases at arrested forks. A mutation that destabilizes the second subunit of the Origin Recognition Complex, orc2-1, reduces the number of functional replication forks by 30% and severely compromises the activation of Rad53 by replication stress or DNA damage in S phase. We show that the restoration of the checkpoint response correlates in a dose-dependent manner with the restoration of pre-replication complex formation in G1. Other forms of DNA damage can compensate for the reduced level of fork-dependent signal in the orc2-1 mutant, yet even in wild-type cells, the amount of damage required for Rad53 activation is higher in S phase than in G2. Our data suggest the existence of an S-phase-specific threshold that may be necessary to allow cells to tolerate damage-like DNA structures present at normal replication forks.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Cycle Proteins*
  • Cell Survival / drug effects
  • Checkpoint Kinase 2
  • Chromosomes, Fungal
  • Cycloheximide / pharmacology
  • DNA Damage
  • DNA Replication
  • DNA, Fungal / biosynthesis*
  • DNA-Binding Proteins / physiology*
  • Dose-Response Relationship, Drug
  • Fungal Proteins / genetics
  • Fungal Proteins / metabolism*
  • Gene Expression Regulation, Fungal
  • Genes, cdc / physiology*
  • Hydroxyurea / pharmacology
  • Methyl Methanesulfonate / pharmacology
  • Mutation
  • Nucleic Acid Conformation
  • Origin Recognition Complex
  • Phosphorylation
  • Protein Serine-Threonine Kinases / genetics
  • Protein Serine-Threonine Kinases / metabolism*
  • Protein Synthesis Inhibitors / pharmacology
  • Replication Origin
  • S Phase / physiology*
  • Saccharomyces cerevisiae / physiology*
  • Saccharomyces cerevisiae Proteins*
  • Ubiquitin / metabolism

Substances

  • Cell Cycle Proteins
  • DNA, Fungal
  • DNA-Binding Proteins
  • Fungal Proteins
  • ORC2 protein, S cerevisiae
  • Origin Recognition Complex
  • Protein Synthesis Inhibitors
  • Saccharomyces cerevisiae Proteins
  • Ubiquitin
  • Cycloheximide
  • Methyl Methanesulfonate
  • Checkpoint Kinase 2
  • Protein Serine-Threonine Kinases
  • RAD53 protein, S cerevisiae
  • Hydroxyurea
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