Muscle Imbalance

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Muscle Imbalance

Syndrome
NURWAHIDA PUSPITASARI, M.OR
The Chronic Musculoskeletal Pain
Cycle
Tonic and Phasic Systems
• The tonic system is the first used by the human
body, as it is responsible for maintaining the fetal
posture in newborn infants.
• The phasic system soon is activated as the infant
learns to lift her head for visual orientation.
• The development of normal movement patterns
utilizes reflexive coactivation of the tonic and
phasic systems
Type I muscle fibers in tonic muscles
Type II fibers in phasic muscles
Causes of Muscle Tightness and
Weakness
Upper Crossed Syndrome &
Lower Crossed Syndrome
Upper-Crossed Syndrome
• Upper-crossed syndrome (UCS) is also referred to as
proximal or shoulder girdle crossed syndrome (Janda
1988).
• In UCS, tightness of the upper trapezius and levator
scapula on the dorsal side crosses with tightness of the
pectoralis major and minor.
• Weakness of the deep cervical flexors ventrally crosses
with weakness of the middle and lower trapezius.
• This pattern of imbalance creates joint dysfunction,
particularly at the atlanto-occipital joint, C4-C5
segment, cervicothoracic joint, glenohumeral joint, and
T4-T5 segment
Upper Crossed Syndrome
Specific postural changes are seen in UCS:
• including forward head posture,
• increased cervical lordosis and thoracic kyphosis,
• elevated and protracted shoulders, and
• rotation or abduction and winging of the scapulae
• These postural changes decrease glenohumeral stability as
the glenoid fossa becomes more vertical due to serratus
anterior weakness leading to abduction, rotation, and
winging of the scapulae.
• This loss of stability requires the levator scapula and upper
trapezius to increase activation to maintain glenohumeral
centration (Janda 1988).
Lower Crossed Syndrome
• Lower-crossed syndrome (LCS) is also referred to
as distal or pelvic crossed syndrome (Janda 1987).
• In LCS, tightness of the thoracolumbar extensors
on the dorsal side crosses with tightness of the
iliopsoas and rectus femoris.
• Weakness of the deep abdominal muscles
ventrally crosses with weakness of the gluteus
maximus and medius.
• This pattern of imbalance creates joint
dysfunction, particularly at the : L4-L5 and L5-S1
segments, SI joint, and hip joint.
Specific postural changes seen in LCS
Specific postural changes seen in LCS include:
• anterior pelvic tilt,
• increased lumbar lordosis,
• lateral lumbar shift,
• lateral leg rotation, and
• knee hyperextension.
• If the lordosis is deep and short, then imbalance is
predominantly in the pelvic muscles; if the lordosis is
shallow and extends into the thoracic area, then
imbalance predominates in the trunk muscles (Janda
1987).
Janda identified two subtypes of LCS:
• Janda identified two subtypes of LCS: A and B.
• Patients with LCS type A use more hip flexion and
extension movement for mobility; their standing
posture demonstrates an anterior pelvic tilt with
slight hip flexion and knee flexion.
• These individuals compensate with a
hyperlordosis limited to the lumbar spine and
with a hyperkyphosis in the upper lumbar and
thoracolumbar segments.
LCS Type B
• Janda's LCS type B involves more movement
of the low back and abdominal area.
• There is minimal lumbar lordosis that extends
into the thoracolumbar segments,
compensatory kyphosis in the thoracic area,
and head protraction.
• The COG is shifted backward with the
shoulders behind the axis of the body, and the
knees are in recurvatum.
Stretching
Strengthening
Lower Crossed Syndrome

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