PREETI and Rahul

Name of Patient : Mrs.
PREETI Test Request ID : 19432202200004

Age/Gender : 26 Yrs/Female Specimen Drawn ON : 20-Feb-2022 08:06PM
Collected AT : CRL COLLECTION POINT Specimen Received ON : 21-Feb-2022 09:10AM
Referred BY : Dr. SELF Report DATE : 21-Feb-2022 11:06AM
Sample Type : Serum - 33633967,Sod.Fluoride - F - 33633969,EDTA Blood - 33633968, -
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CRL SAWSTHYA CARE PLUS(+)

HAEMATOLOGY
Test Description Observed Value Biological Reference Method
Range
HbA1c (ngsp) 5.1 <6:Non diabetic HPLC
6-7:good Control
7-8:weak Control
>8 poor Control %
HbA1c (IFCC) 32.16 mmol/mol HPLC
Estimated Average Glucose 99.67 mg/dl
REMARKS
In vitro quantitative determination of HbA1c in whole blood is utilized in long term monitoring of glycemia.The HbA1c level correlates with the mean
glucose concentration prevailing in the course of the patient's recent history (approx - 6-8 weeks) and therefore provides much more reliable
information for glycemia monitoring than do determinations of blood glucose or urinary glucose. It is recommended that the determination
of HbA1c be performed at intervals of 4-6 weeks during Diabetes Mellitus therapy. Results of HbA1c should be assessed in conjunction with the
patient's medical history, clinical examinations and other findings.
Disclaimer:-For test performed on specimens received or collected from non-CRL locations, it is presumed that the specimen belongs to the patient named or identified as labeled on the container/test request and such verification has been carried Page 1 of 11
out at the point generation of the said specimen by the sender. CRL will be responsible Only for the analytical part of test carried out. All other responsibility will be of referring Laboratory.sample processed and reported at CRL DIAGNOSTICS PVT. LTD , Delhi.
Name of Patient : Mrs. PREETI Test Request ID : 19432202200004
Ref Customer :
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Range
COMPLETE BLOOD COUNT(CBC)23
R.B.C 4.19 4.5-5.5 Millions/cumm Impedance variation
Haemoglobin 12.0 12.0-15.0 g/dl Spectrophotometry
Packed Cell Volume 36.40 40.0-50.0 % Analogical Integration
MCV 86.87 83-101 fL Calculated
MCH 28.64 27.0-32.0 pg Calculated
MCHC 32.97 27.0-48.0 g/dL Calculated
RDW-CV 13.2 11.5-14.0 % Calculated
Platelet Count 162 150-410 1000/uL Impedance Variation
Total WBC Count 7170 4000-10000 /cumm Impedance Variation
NRBC 00 % WBC Histogram and
VCS Technology
MPV 14.40 9.1-11.9 % Calculated
PCT 0.23 0.18-0.39 % Calculated
PDW 33.40 9.0-15.0 % Calculated
Differential Leucocyte Count
Neutrophil 50 40.0-80.0 % flow cytometry/manual
Lymphocyte 36 20.0-40.0 % flow cytometry/manual
Monocytes 10 2-10 % flow cytometry/manual
Eosinophils 04 01-06 % Flow cytometry/manual
Basophils 00 0-1 % Flow cytometry/manual
Absolute Neutrophils 3.59 2.00-7.00 10^3/µL
Absolute Lymphocytes 2.58 1.00-3.00 10^3/µL
Absolute Monocytes 0.72 0.20-1.00 10^3/µL
Absolute Eosinophils 0.29 0.02-0.50 10^3/µL
Absolute Basophils 0.00 0.02-0.10 10^3/µL
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Range
Erythrocyte Sedimentation Rate (ESR) 26 0-20 mm/h Westergren
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Range
BIOCHEMISTRY
GLUCOSE FASTING
Glucose Fasting 70.5 70.0 - 110.0 mg/dL HEXOKINASE
Interpretation (In accordance with the American diabetes association guidelines):
· A fasting plasma glucose level below 110 mg/dL is considered normal.
· A fasting plasma glucose level between 100‑126 mg/dL is considered as glucose intolerant or pre diabetic. A fasting and post‑prandial blood sugar
test (after consumption of 75 gm of glucose) is recommended for all such patients.
· A fasting plasma glucose level of above 126 mg/dL is highly suggestive of a diabetic state. A repeat fasting test is strongly recommended for all such
patients. A fasting plasma glucose level in excess of 126 mg/dL on both the occasions is confirmatory of a diabetic state.
.
Calcium, Serum 9.1 8.6-10.2 mg/dl NM-BAPTA

DESCRIPTION
About 50% of the calcium present in circulation is free (also known as ionized calcium); 40% of serum calcium is bound to proteins, especially
albumin (80%) and, secondary, to globulins (20%); and about 10% exists as various small diffusible inorganic and organic anions (eg, bicarbonate,
lactate, citrate).Heart and skeletal muscle contractility are affected by calcium ions; in addition, calcium ions are vital to nervous system function and
are associated with blood clotting and bone mineralization.The concentration of serum calcium is tightly regulated by parathyroid hormone (PTH)
and 1,25-hydroxy vitamin D.
INTERPRETATION-
Serum calcium is decreased (hypocalcemia) in the following conditions:
Hypoparathyroidism , Vitamin D deficiency ,Chronic renal diseases ,Pseudohypoparathyroidism,
Magnesium deficiency (PTH glandular release is magnesium-dependent), Hyperphosphatemia,
Massive transfusion, Hypoalbuminemia, Severe calcium dietary deficiency and Severe pancreatitis (calcium saponification)
Serum calcium is increased(Hypercalcemia) in the following conditions:
Hyperparathyroidism ,Vitamin D excess, Milk-alkali syndrome, Multiple myeloma, owing to bone lesions, Paget disease of bone with prolonged
immobilization, Sarcoidosis, Familial hypocalciuria hypercalcemia,Vitamin A intoxication,Thyrotoxicosis and Addison disease
Ref Customer :

Range
KIDNEY FUNCTION TEST (KFT)
Urea 31.22 15.0-40.0 mg/dl Urease Colorimetric
Creatinine 0.58 0.60-1.20 mg/dL Jaffe Kinetic
Uric Acid 3.62 2.30-6.60 mg/dl Uricase Colorimetric
Sodium (NA+) 136.30 135.0-145.0 mmol/L Ion Selective Electrode
Potassium (K+) 4.03 3.50-5.50 mmol/L Ion Selective Electrode
Chloride 100.00 98-109 mmol/L Ion Selective Electrode
Interpretation:- Kidney blood tests, or Kidney function tests, are used to detect and diagnose disease of the Kidney.
The higher the blood levels of urea and creatinine, the less well the kidneys are working.
The level of creatinine is usually used as a marker as to the severity of kidney failure. (Creatinine in itself is not harmful, but a high level indicates that
the kidneys are not working properly. So, many other waste products will not be cleared out of the bloodstream.) You normally need treatment with
dialysis if the level of creatinine goes higher than a certain value.
Dehydration can also be a come for increases in urea level.
Before and after starting treatment with certain medicines. Some medicines occasionally cause kidney damage (Nephrotoxic Drug) as a side-effect.
Therefore, kidney function is often checked before and after starting treatment with certain medicines.
Ref Customer :

Range
LIVER FUNCTION TEST (LFT)-EXTENDED
Bilirubin Total 0.32 <1.1 mg/dl Diazotized Sulfanilic
Bilirubin Direct 0.10 0-0.3 mg/dl Diazotized Sulfanilic
Bilirubin Indirect 0.22 0.3-1.0 mg/dl Diazotized Sulfanilic
SGOT (AST) 22.7 <31.0 U/L IFCC without pyridoxal
phosphate
SGPT (ALT) 15.8 <33.0 U/L IFCC without pyridoxal
phosphate
Alkaline Phosphatase (ALP) 88.8 35-104 U/L Kinetic Colour
Gamma Glutamyl Transferase (GGT) 13.2 05-40 U/L L-Gamma-glutamyl-3-
carboxy-4-nitroanilide
Substrat
Protein Total 7.28 6.0-8.0 g/dL Biuret
Albumin (Serum) 4.78 3.5-5.5 g/dL Bromo Cresol Green
(BCG)
Globulin 2.5 2.50-3.50 g/dL Calculated
A/G Ratio 1.91 1.5-2.5 Calculated
Interpretation:- Liver blood tests, or liver function tests, are used to detect and diagnose disease or inflammation of the liver. Elevated
aminotransferase (ALT, AST) levels are measured as well as alkaline phosphatase, albumin, and bilirubin. Some diseases that cause abnormal levels
of ALT and AST include hepatitis A, B, and C, cirrhosis, iron overload, and Tylenol liver damage. Medications also cause elevated liver enzymes. There
are less common conditions and diseases that also cause elevated liver enzyme levels.: Liver blood tests, or liver function tests, are used to detect and
diagnose disease or inflammation of the liver. Elevated aminotransferase (ALT, AST) levels are measured as well as alkaline phosphatase, albumin,
and bilirubin. Some diseases that cause abnormal levels of ALT and AST include hepatitis A, B, and C, cirrhosis, iron overload, and Tylenol liver
damage. Medications also cause elevated liver enzymes.There are less common conditions and diseases that also cause elevated liver enzyme levels.
Ref Customer :

Range
LIPID PROFILE BASIC
Total Cholesterol 176.6 <200.00 mg/dL mg/dL Enzymatic Colorimetric
Triglyceride 155.9 0.0-150 :Normal Enzymatic Colorimetric
151-199:Border Line >=200 :
High
200.0-499.0 High
> 500 Very High mg/dl
HDL Cholesterol 56.4 40-60 mg/dL Direct (PVS/PEGME
precipitation & Trinder
reaction)
Non HDL Cholesterol 120.20 Calculated
LDL Cholesterol 89.02 0-130 :Normal Direct (PVS/PEGME
131-155:Borderline precipitation & Trinder
>=160 :High mg/dl reaction)
VLDL Cholesterol 31.18 2.00-30.00 mg/dL Calculated
Cholesterol/HDL Ratio 3.13 <4.00 Calculated
LDL / HDL Cholestrol Ratio 1.58 <3.50 Calculated
HDL/LDL Cholesterol Ratio 0.63 <3.50 Calculated
Total Cholesterol (mg/dL) <200 – Desirable 200‑239 ‑Borderline high <240 – High
HDL Cholesterol (mg/dL), <40 – Low >60 – High
LDL Cholesterol (mg/dL) <100 Optimal
[Primary Target of Therapy] 100-129 Near optimal /above optimal, 130-159 Borderline high,160-189 High,>190 Very high.Serum Triglycerides
(mg/dL) <150 Normal,150-199 Borderline high,200-499 High,>500 Very high
NCEP recommends lowering of LDL Cholesterol as the primary therapeutic target with lipid lowering agents, however, if triglycerides remain >200
mg/dL after LDL goal is Reached, seti secondary goal for non-HDL cholesterol (total minus HDL) 30 mg/dL higher than LDL goal.
Risk Category LDL Goal (mg/dL) Non-HDL Goal (mg/dL)
CHD and CHD Risk Equivalent <100 <130
(10-year risk for CHD>20%)
Multiple (2+) Risk Factors and <130 <160
10-year risk <20%
0-1 Risk Factor <160 <190
Ref Customer :

Range
Iron, Serum 55.0 59-158 ug/dL Colorimetric
Comment-
Iron is an essential trace mineral element which forms an important component of hemoglobin, metallocompounds and Vitamin A. Deficiency of iron,
leads to microcytic hypochromic anemia. The toxic effects of iron are deposition of iron in various organs of the body and hemochromatosis.
Increases in serum iron level are associated with the following:

Idiopathic hemochromatosis, Liver necrosis ( viral hepatitis), Hemosiderosis caused by excessive iron intake (eg, multiple transfusions, excess iron
administration,)Acute iron poisoning (children), Hemolytic anemia, Pernicious anemia, Aplastic or hypoplastic anemia, Lead poisoning, Thalassemia,
Vitamin B6 deficiency, Estrogens, Ethanol, Oral contraceptive
Decreases in the serum iron level are associated with the following:
Iron deficiency anemia, Nephrotic syndrome (loss of iron-binding proteins), Chronic renal failure, Chronic infections, Active hematopoiesis,
Remission of pernicious anemia, Hypothyroidism, Malignancy (carcinoma), Postoperative state, Kwashiorkor
NOTE-If patients with iron deficiency anemia receive iron medication before blood is drawn, normal or high concentrations are typically
noted. Even multivitamins with low (18 mg) elemental iron may produce this result. Thus, 24 hours before a blood draw for serum iron,
all oral iron mediation should be stopped. Parenteral injection of iron dextran may result in high serum iron levels (eg, 500‑1000 µg/dL)
for several weeks.Infusion of sodium ferric gluconate or iron sucrose causes increases in iron levels that last much shorter.
Recent transfusions influence test findings.
Ref Customer :

Range
Total Iron Binding Capacity-(TIBC) 432.0 250-400 ug/dL Spectro-photometry
Total iron-binding capacity
The test measures the extent to which iron-binding sites in the serum can be saturated. Because the iron-binding sites in the serum are almost entirely
dependent on circulating transferrin, this is really an indirect measurement of the amount of transferrin in the blood.
Taken together with serum iron and percent transferrin saturation clinicians usually perform this test when they are concerned about anemia, iron
deficiency or iron deficiency anemia. However, because the liver produces transferrin, liver function must be considered when performing this test. It
can also be an indirect test of liver function, but is rarely used for this purpose.
Transferrin Saturation 12.70 16-50 %

COMMENT : 1g of transferrin can carry 1.43g of iron. Normally, iron saturation of transferrin (transferrin saturation) is between 10% and 50%.
Because of its short half-life, transferrin values decrease more quickly in protein malnutrition states and should be taken into consideration while
evaluating iron-deficiency states.
Ref Customer :
Test Description Observed Value Biological Reference Range Method

IMMUNOASSAY
THYROID PROFILE
Triiodothyronine Total (T3) 1.26 0.70-2.04 ng/mL Electrochemiluminescence
immunoassay (ECLIA)
Thyroxine Total (T4) 8.80 4.6-10.5 ug/dL Electrochemiluminescence
immunoassay (ECLIA)
TSH (4th Generation) 3.332 0.40-4.20 uIU/mL Electrochemiluminescence
immunoassay (ECLIA)
PREGNANCY REFERENCE RANGE for TSH IN uIU/mL (As per American Thyroid Association.)
1st Trimester 0.10-2.50 uIU/mL
2nd Trimester 0.20-3.00 uIU/mL
3rd Trimester 0.30-3.00 uIU/mL
INTERPRETATION-
1. Primary hyperthyroidism is accompanied by elevated serum T3 & T4 values along with depressed TSH level.
2 .Primary hypothyroidism is accompanied by depressed serum T3 and T4 values & elevated serum TSH levels.
3. Normal T4 levels accompanied by high T3 levels and low TSH are seen in patients with T3 thyrotoxicosis.
4. Normal or low T3 & high T4 levels indicate T4 thyrotoxicosis ( problem is conversion of T4 to T3)
5. Normal T3 & T4 along with low TSH indicate mild / subclinical HYPERTHYROIDISM .
6. Normal T3 & low T4 along with high TSH is seen in HYPOTHYROIDISM .
7. Normal T3 & T4 levels with high TSH indicate Mild / Subclinical HYPOTHYROIDISM .
8. Slightly elevated T3 levels may be found in pregnancy and in estrogen therapy while depressed levels may be encountered in severe illness , malnutrition ,
renal failure and during therapy with drugs like propanolol.
9. Although elevated TSH levels are nearly always indicative of primary hypothroidism . rarely they can result from TSH secreting pituitary tumours ( seconday
hyperthyroidism )
*TSH IS DONE BY ULTRASENSITIVE 4th GENERATION CHEMIFLEX ASSAY*
COMMENTS:
Assay results should be interpreted in context to the clinical condition and associated results of other investigations. Previous treatment with corticosteroid
therapy may result in lower TSH levels while thyroid hormone levels are normal. Results are invalidated if the client has undergone a radionuclide scan within 7-
14 days before the test. Abnormal thyroid test findings often found in critically ill clients should be repeated after the critical nature of the condition is
resolved.The production, circulation, and disintegration of thyroid hormones are altered throughout the stages of pregnancy.
Disclaimer-
TSH is an important marker for the diagnosis of thyroid dysfunction.Recent studies have shown that the TSH distribution progressively shifts to a higher concentration with age ,and it is debatable
whether this is due to a real change with age or an increasing proportion of unrecognized thyroid diseasein the elderly.
TSH levels are subject to circardian variation,reaching peak levels between 2-4AM and ninimum between 6-10 PM. The variation is the order of 50% hence
time of the day has influence on the measures serum TSH concentration.Dose and time of drug intake also influence the test result. r
Reference ranges are from Teitz fundamental of clinical chemistry 7th ed.
Nabl Scope.
*** End Of Report ***
Name of Patient : Mr. RAHUL Test Request ID : 19432202200005
Age/Gender : 32 Yrs/Male Specimen Drawn ON : 20-Feb-2022 08:07PM
Ref Customer :

Range
HAEMATOLOGY
HbA1c (ngsp) 6.3 <6:Non diabetic HPLC
6-7:good Control
7-8:weak Control
>8 poor Control %
HbA1c (IFCC) 45.24 mmol/mol HPLC
Estimated Average Glucose 134.11 mg/dl
REMARKS
In vitro quantitative determination of HbA1c in whole blood is utilized in long term monitoring of glycemia.The HbA1c level correlates with the mean
glucose concentration prevailing in the course of the patient's recent history (approx - 6-8 weeks) and therefore provides much more reliable
information for glycemia monitoring than do determinations of blood glucose or urinary glucose. It is recommended that the determination
of HbA1c be performed at intervals of 4-6 weeks during Diabetes Mellitus therapy. Results of HbA1c should be assessed in conjunction with the
patient's medical history, clinical examinations and other findings.
Ref Customer :
Ref Customer :

Range
COMPLETE BLOOD COUNT(CBC)23
R.B.C 5.36 4.5-5.5 Millions/cumm Impedance variation
Haemoglobin 13.0 13.0-17.0 g/dl Spectrophotometry
Packed Cell Volume 39.60 40.0-50.0 % Analogical Integration
MCV 73.88 83-101 fL Calculated
MCH 24.25 27.0-32.0 pg Calculated
MCHC 32.83 27.0-48.0 g/dL Calculated
RDW-CV 13.7 11.5-14.0 % Calculated
Platelet Count 174 150-410 1000/uL Impedance Variation
Total WBC Count 6060 4000-10000 /cumm Impedance Variation
NRBC 00 % WBC Histogram and
VCS Technology
MPV 12.70 9.1-11.9 % Calculated
PCT 0.22 0.18-0.39 % Calculated
PDW 28.00 9.0-15.0 % Calculated
Differential Leucocyte Count
Neutrophil 65 40.0-80.0 % flow cytometry/manual
Lymphocyte 25 20.0-40.0 % flow cytometry/manual
Monocytes 06 2-10 % flow cytometry/manual
Eosinophils 04 01-06 % Flow cytometry/manual
Basophils 00 0-1 % Flow cytometry/manual
Absolute Neutrophils 3.94 2.00-7.00 10^3/µL
Absolute Lymphocytes 1.52 1.00-3.00 10^3/µL
Absolute Monocytes 0.36 0.20-1.00 10^3/µL
Absolute Eosinophils 0.24 0.02-0.50 10^3/µL
Absolute Basophils 0.00 0.02-0.10 10^3/µL
Ref Customer :

Range
Erythrocyte Sedimentation Rate (ESR) 26 0-20 mm/h Westergren
Ref Customer :

Range
BIOCHEMISTRY
GLUCOSE FASTING
Glucose Fasting 102.0 70.0 - 110.0 mg/dL HEXOKINASE
Interpretation (In accordance with the American diabetes association guidelines):
· A fasting plasma glucose level below 110 mg/dL is considered normal.
· A fasting plasma glucose level between 100‑126 mg/dL is considered as glucose intolerant or pre diabetic. A fasting and post‑prandial blood sugar
test (after consumption of 75 gm of glucose) is recommended for all such patients.
· A fasting plasma glucose level of above 126 mg/dL is highly suggestive of a diabetic state. A repeat fasting test is strongly recommended for all such
patients. A fasting plasma glucose level in excess of 126 mg/dL on both the occasions is confirmatory of a diabetic state.
.
Calcium, Serum 9.4 8.6-10.2 mg/dl NM-BAPTA

DESCRIPTION
About 50% of the calcium present in circulation is free (also known as ionized calcium); 40% of serum calcium is bound to proteins, especially
albumin (80%) and, secondary, to globulins (20%); and about 10% exists as various small diffusible inorganic and organic anions (eg, bicarbonate,
lactate, citrate).Heart and skeletal muscle contractility are affected by calcium ions; in addition, calcium ions are vital to nervous system function and
are associated with blood clotting and bone mineralization.The concentration of serum calcium is tightly regulated by parathyroid hormone (PTH)
and 1,25-hydroxy vitamin D.
INTERPRETATION-
Serum calcium is decreased (hypocalcemia) in the following conditions:
Hypoparathyroidism , Vitamin D deficiency ,Chronic renal diseases ,Pseudohypoparathyroidism,
Magnesium deficiency (PTH glandular release is magnesium-dependent), Hyperphosphatemia,
Massive transfusion, Hypoalbuminemia, Severe calcium dietary deficiency and Severe pancreatitis (calcium saponification)
Serum calcium is increased(Hypercalcemia) in the following conditions:
Hyperparathyroidism ,Vitamin D excess, Milk-alkali syndrome, Multiple myeloma, owing to bone lesions, Paget disease of bone with prolonged
immobilization, Sarcoidosis, Familial hypocalciuria hypercalcemia,Vitamin A intoxication,Thyrotoxicosis and Addison disease
Ref Customer :

Range
KIDNEY FUNCTION TEST (KFT)
Urea 19.35 18.0-45.0 mg/dl Urease Colorimetric
Creatinine 0.81 0.70-1.40 mg/dL Jaffe Kinetic
Uric Acid 5.74 4.40-7.60 mg/dl Uricase Colorimetric
Sodium (NA+) 140.30 135.0-145.0 mmol/L Ion Selective Electrode
Potassium (K+) 4.16 3.50-5.50 mmol/L Ion Selective Electrode
Chloride 100.00 98-109 mmol/L Ion Selective Electrode
Interpretation:- Kidney blood tests, or Kidney function tests, are used to detect and diagnose disease of the Kidney.
The higher the blood levels of urea and creatinine, the less well the kidneys are working.
The level of creatinine is usually used as a marker as to the severity of kidney failure. (Creatinine in itself is not harmful, but a high level indicates that
the kidneys are not working properly. So, many other waste products will not be cleared out of the bloodstream.) You normally need treatment with
dialysis if the level of creatinine goes higher than a certain value.
Dehydration can also be a come for increases in urea level.
Before and after starting treatment with certain medicines. Some medicines occasionally cause kidney damage (Nephrotoxic Drug) as a side-effect.
Therefore, kidney function is often checked before and after starting treatment with certain medicines.
Ref Customer :

Range
LIVER FUNCTION TEST (LFT)-EXTENDED
Bilirubin Total 0.27 <1.1 mg/dl Diazotized Sulfanilic
Bilirubin Direct 0.10 0-0.3 mg/dl Diazotized Sulfanilic
Bilirubin Indirect 0.17 0.3-1.0 mg/dl Diazotized Sulfanilic
SGOT (AST) 21.0 <31.0 U/L IFCC without pyridoxal
phosphate
SGPT (ALT) 23.2 <33.0 U/L IFCC without pyridoxal
phosphate
Alkaline Phosphatase (ALP) 99.5 40-129 U/L Kinetic Colour
Gamma Glutamyl Transferase (GGT) 30.8 15-60 U/L L-Gamma-glutamyl-3-
carboxy-4-nitroanilide
Substrat
Protein Total 7.33 6.0-8.0 g/dL Biuret
Albumin (Serum) 4.89 3.5-5.5 g/dL Bromo Cresol Green
(BCG)
Globulin 2.44 2.50-3.50 g/dL Calculated
A/G Ratio 2.00 1.5-2.5 Calculated
Interpretation:- Liver blood tests, or liver function tests, are used to detect and diagnose disease or inflammation of the liver. Elevated
aminotransferase (ALT, AST) levels are measured as well as alkaline phosphatase, albumin, and bilirubin. Some diseases that cause abnormal levels
of ALT and AST include hepatitis A, B, and C, cirrhosis, iron overload, and Tylenol liver damage. Medications also cause elevated liver enzymes. There
are less common conditions and diseases that also cause elevated liver enzyme levels.: Liver blood tests, or liver function tests, are used to detect and
diagnose disease or inflammation of the liver. Elevated aminotransferase (ALT, AST) levels are measured as well as alkaline phosphatase, albumin,
and bilirubin. Some diseases that cause abnormal levels of ALT and AST include hepatitis A, B, and C, cirrhosis, iron overload, and Tylenol liver
damage. Medications also cause elevated liver enzymes.There are less common conditions and diseases that also cause elevated liver enzyme levels.
Ref Customer :

Range
LIPID PROFILE BASIC
Total Cholesterol 123.3 <200.00 mg/dL mg/dL Enzymatic Colorimetric
Triglyceride 213.6 0.0-150 :Normal Enzymatic Colorimetric
151-199:Border Line >=200 :
High
200.0-499.0 High
> 500 Very High mg/dl
HDL Cholesterol 30.7 40-60 mg/dL Direct (PVS/PEGME
precipitation & Trinder
reaction)
Non HDL Cholesterol 92.60 Calculated
LDL Cholesterol 49.88 0-130 :Normal Direct (PVS/PEGME
131-155:Borderline precipitation & Trinder
>=160 :High mg/dl reaction)
VLDL Cholesterol 42.72 2.00-30.00 mg/dL Calculated
Cholesterol/HDL Ratio 4.02 <4.00 Calculated
LDL / HDL Cholestrol Ratio 1.62 <3.50 Calculated
HDL/LDL Cholesterol Ratio 0.62 <3.50 Calculated
Total Cholesterol (mg/dL) <200 – Desirable 200‑239 ‑Borderline high <240 – High
HDL Cholesterol (mg/dL), <40 – Low >60 – High
LDL Cholesterol (mg/dL) <100 Optimal
[Primary Target of Therapy] 100-129 Near optimal /above optimal, 130-159 Borderline high,160-189 High,>190 Very high.Serum Triglycerides
(mg/dL) <150 Normal,150-199 Borderline high,200-499 High,>500 Very high
NCEP recommends lowering of LDL Cholesterol as the primary therapeutic target with lipid lowering agents, however, if triglycerides remain >200
mg/dL after LDL goal is Reached, seti secondary goal for non-HDL cholesterol (total minus HDL) 30 mg/dL higher than LDL goal.
Risk Category LDL Goal (mg/dL) Non-HDL Goal (mg/dL)
CHD and CHD Risk Equivalent <100 <130
(10-year risk for CHD>20%)
Multiple (2+) Risk Factors and <130 <160
10-year risk <20%
0-1 Risk Factor <160 <190
Ref Customer :

Range
Iron, Serum 43.0 59-158 ug/dL Colorimetric
Comment-
Iron is an essential trace mineral element which forms an important component of hemoglobin, metallocompounds and Vitamin A. Deficiency of iron,
leads to microcytic hypochromic anemia. The toxic effects of iron are deposition of iron in various organs of the body and hemochromatosis.
Increases in serum iron level are associated with the following:

Idiopathic hemochromatosis, Liver necrosis ( viral hepatitis), Hemosiderosis caused by excessive iron intake (eg, multiple transfusions, excess iron
administration,)Acute iron poisoning (children), Hemolytic anemia, Pernicious anemia, Aplastic or hypoplastic anemia, Lead poisoning, Thalassemia,
Vitamin B6 deficiency, Estrogens, Ethanol, Oral contraceptive
Decreases in the serum iron level are associated with the following:
Iron deficiency anemia, Nephrotic syndrome (loss of iron-binding proteins), Chronic renal failure, Chronic infections, Active hematopoiesis,
Remission of pernicious anemia, Hypothyroidism, Malignancy (carcinoma), Postoperative state, Kwashiorkor
NOTE-If patients with iron deficiency anemia receive iron medication before blood is drawn, normal or high concentrations are typically
noted. Even multivitamins with low (18 mg) elemental iron may produce this result. Thus, 24 hours before a blood draw for serum iron,
all oral iron mediation should be stopped. Parenteral injection of iron dextran may result in high serum iron levels (eg, 500‑1000 µg/dL)
for several weeks.Infusion of sodium ferric gluconate or iron sucrose causes increases in iron levels that last much shorter.
Recent transfusions influence test findings.
Ref Customer :

Range
Total Iron Binding Capacity-(TIBC) 412.0 250-400 ug/dL Spectro-photometry
Total iron-binding capacity
The test measures the extent to which iron-binding sites in the serum can be saturated. Because the iron-binding sites in the serum are almost entirely
dependent on circulating transferrin, this is really an indirect measurement of the amount of transferrin in the blood.
Taken together with serum iron and percent transferrin saturation clinicians usually perform this test when they are concerned about anemia, iron
deficiency or iron deficiency anemia. However, because the liver produces transferrin, liver function must be considered when performing this test. It
can also be an indirect test of liver function, but is rarely used for this purpose.
Transferrin Saturation 10.40 16-50 %

COMMENT : 1g of transferrin can carry 1.43g of iron. Normally, iron saturation of transferrin (transferrin saturation) is between 10% and 50%.
Because of its short half-life, transferrin values decrease more quickly in protein malnutrition states and should be taken into consideration while
evaluating iron-deficiency states.
Ref Customer :
Test Description Observed Value Biological Reference Range Method

IMMUNOASSAY
THYROID PROFILE
Triiodothyronine Total (T3) 1.16 0.70-2.04 ng/mL Electrochemiluminescence
immunoassay (ECLIA)
Thyroxine Total (T4) 6.41 4.6-10.5 ug/dL Electrochemiluminescence
immunoassay (ECLIA)
TSH (4th Generation) 1.113 0.40-4.20 uIU/mL Electrochemiluminescence
immunoassay (ECLIA)
PREGNANCY REFERENCE RANGE for TSH IN uIU/mL (As per American Thyroid Association.)
1st Trimester 0.10-2.50 uIU/mL
2nd Trimester 0.20-3.00 uIU/mL
3rd Trimester 0.30-3.00 uIU/mL
INTERPRETATION-
1. Primary hyperthyroidism is accompanied by elevated serum T3 & T4 values along with depressed TSH level.
2 .Primary hypothyroidism is accompanied by depressed serum T3 and T4 values & elevated serum TSH levels.
3. Normal T4 levels accompanied by high T3 levels and low TSH are seen in patients with T3 thyrotoxicosis.
4. Normal or low T3 & high T4 levels indicate T4 thyrotoxicosis ( problem is conversion of T4 to T3)
5. Normal T3 & T4 along with low TSH indicate mild / subclinical HYPERTHYROIDISM .
6. Normal T3 & low T4 along with high TSH is seen in HYPOTHYROIDISM .
7. Normal T3 & T4 levels with high TSH indicate Mild / Subclinical HYPOTHYROIDISM .
8. Slightly elevated T3 levels may be found in pregnancy and in estrogen therapy while depressed levels may be encountered in severe illness , malnutrition ,
renal failure and during therapy with drugs like propanolol.
9. Although elevated TSH levels are nearly always indicative of primary hypothroidism . rarely they can result from TSH secreting pituitary tumours ( seconday
hyperthyroidism )
*TSH IS DONE BY ULTRASENSITIVE 4th GENERATION CHEMIFLEX ASSAY*
COMMENTS:
Assay results should be interpreted in context to the clinical condition and associated results of other investigations. Previous treatment with corticosteroid
therapy may result in lower TSH levels while thyroid hormone levels are normal. Results are invalidated if the client has undergone a radionuclide scan within 7-
14 days before the test. Abnormal thyroid test findings often found in critically ill clients should be repeated after the critical nature of the condition is
resolved.The production, circulation, and disintegration of thyroid hormones are altered throughout the stages of pregnancy.
Disclaimer-
TSH is an important marker for the diagnosis of thyroid dysfunction.Recent studies have shown that the TSH distribution progressively shifts to a higher concentration with age ,and it is debatable
whether this is due to a real change with age or an increasing proportion of unrecognized thyroid diseasein the elderly.
TSH levels are subject to circardian variation,reaching peak levels between 2-4AM and ninimum between 6-10 PM. The variation is the order of 50% hence
time of the day has influence on the measures serum TSH concentration.Dose and time of drug intake also influence the test result. r
Reference ranges are from Teitz fundamental of clinical chemistry 7th ed.
Nabl Scope.
*** End Of Report ***

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Name of Patient : Mrs.

PREETI Test Request ID : 19432202200004

CRL SAWSTHYA CARE PLUS(+)

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Calcium, Serum 9.1 8.6-10.2 mg/dl NM-BAPTA

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Increases in serum iron level are associated with the following:

Test Description Observed Value Biological Reference Method

Transferrin Saturation 12.70 16-50 %

Test Description Observed Value Biological Reference Range Method

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Calcium, Serum 9.4 8.6-10.2 mg/dl NM-BAPTA

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Test Description Observed Value Biological Reference Method

Increases in serum iron level are associated with the following:

Test Description Observed Value Biological Reference Method

Transferrin Saturation 10.40 16-50 %

Test Description Observed Value Biological Reference Range Method

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