0% found this document useful (0 votes)
126 views

Conditions of The Lumbar Spine

1. Low back pain is a common symptom that can be caused by mechanical issues like herniated discs or osteoarthritis, or non-mechanical pathologies like tumors or infections. 2. Red flags indicating a more serious condition include failure to improve with conservative treatment, neurological deficits, or risk factors for conditions like cancer. Imaging is recommended for red flag cases or when a specific diagnosis is suspected. 3. Lumbar radiculopathy is a common mechanical cause of low back pain, often caused by disc herniation or stenosis impinging a nerve root, resulting in pain that radiates down the leg.

Uploaded by

Cherrie Mae
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd
0% found this document useful (0 votes)
126 views

Conditions of The Lumbar Spine

1. Low back pain is a common symptom that can be caused by mechanical issues like herniated discs or osteoarthritis, or non-mechanical pathologies like tumors or infections. 2. Red flags indicating a more serious condition include failure to improve with conservative treatment, neurological deficits, or risk factors for conditions like cancer. Imaging is recommended for red flag cases or when a specific diagnosis is suspected. 3. Lumbar radiculopathy is a common mechanical cause of low back pain, often caused by disc herniation or stenosis impinging a nerve root, resulting in pain that radiates down the leg.

Uploaded by

Cherrie Mae
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd
You are on page 1/ 4

LUMBAR SPINE

o Metastatic CA – hx of CA, unexplained weight loss,


rest pain, age >50
o Infection – unexplained fever, recent bacterial
Conditions of the Lumbar Spine
infection, immunosuppression, IVDA
Low Back Pain
o Fx – steroids, osteoporosis, recent trauma, age >70
 Is a symptom, not a disease
o Cauda Equina Syndrome – urinary
 80% lifetime prevalence, m/c cause of disability in pts < 45
retention/incontinence, saddle anesthesia, ↓ rectal
y/o (one of the causes of absenteeism)
tone, (B) LE weakness/numbness
 Pain, muscle tension, or stiffness localized below the costal
o Severe/progressive focal neurologic deficit
margin and above the inf gluteal folds, c or s leg pain
o Failure to improve c therapy
 International Association for the Study of Pain (IASP)
o Pain > 4 wks
o Low Back Pain
 Red Flags incidence:
 Lumbar spinal pain
o Compression Fx (4%)
 Sacral spinal pain
 Osteoporosis
 Lumbosacral pain
 Location: thoracolumbar junction, midthoracic
o Gluteal and Loin Pain (not considered as LBP)
spine
 Classification (Etiology):
 Thoracolumbar fx may present c lumbosacral
o Mechanical – biomechanics of the lumbar area,
pain
problems c the structures; HNP, OA, spinal stenosis,
o Metastatic CA (0.7%)
spondylolisthesis, compression fx
 Past hx of CA is single strongest indicator
 Non-radicular
 Metastatic (prostate, lung, breast)
 Radicular – involves nerve root impingement
 Multiple myeloma
o Non-mechanical – pathologic
 Tumor – metastases, MM, lymphoma  Lymphoma
 Infection – osteomyelitis, diskitis  ↑ post-test probability from 0.7% - 9%
 Inflammatory Arthritis – RA, AS  Not including non-melanoma skin CA
o Visceral diseases – Aortic Abdominal Aneurysm, o Cauda Equina Syndrome (0.04%)
nephrolithiasis, pancreatitis, prostatitis  Large central disc herniation (L4-L5)
 Urinary retention initially  overflow
 Classification (Diagnosis):
incontinence
o Complicated – red flag conditions
 Normal post-void residual of urine essentially
o Specific Diagnosis
rules it out
 Lumbar radiculopathy
 Surgical emergency
 Lumbar spinal stenosis
o Infection (0.01%)
 Other conditions such as AS
 Non-spinal LBP
o Uncomplicated (non-specific) – used to be called
o AAA
mechanical LBP, dx of exclusion, > 85% of LBP in
o Nephrolithiasis
primary care
 Acute: < 1 mo, rapid improvement in first o Pancreatitis
month in most pts, recurrence rate up to 1/3, 7- o Prostatitis
10% progress to chronic back pain o Pelvic Inflammatory Disease
 Subacute: 1-3 mos  Possible anatomic sources of LBP – controversial
 Chronic: > 3 mos o Discs (back pain), facet jts (back, buttock, thigh pain),
 Red flags: SI jts, ligaments, muscles (back pain)
 Innervated structures: Pts are highly responsive to tricyclic
o Bone: vertebrae antidepressants
o Jts: Zygoapophyseal  More predictive of outcome than severity of
o Disk: External annulus fibrosus, potentially diseased pain or exam findings
disk o Durations of Symptoms
o Ligaments: ALL, PLL, interspinous  Status of pts @ 2 mos may help predict
o Muscles and fascia outcome @ 12 mos
o Nerve roots  Summary
 Non-innervated structures: Ligamentum flavum, internal o Be aware of red flags
annulus fibrosus, nucleus pulposus o Identify specific dx
 Yellow flags: o Uncomplicated LBP is a dx of exclusion
o Previous hx of disability o MRI for complicated and specific dx
o Inconsistent findings o Diagnostic injections
o Abnormal pain behavior o Don’t forget the yellow flags
o Litigation Lumbar Radiculopathy
o Work dissatisfaction o L4-L5 or L5-S1 (90%), L5 and S1 nerve roots
o Attention-seeking o Inflammation > mechanical compression
o Preference for prolonged bed rest o Phosopholipase A2, TNF-alpha
o Depression o Pain c sitting, bending, coughing, radiates below the
o Chemical dependency knee in a narrow band
o Hx of abuse Nerve Pain Motor Sensory DTR
Root Referral
o Family hx of chronic pain
L4 Medial leg Ankle Medial Knee
 Waddell’s Signs – behavioral response to exam, not a proof of dorsiflexi ankle
malingering, > 3 signs suggests presence of non-organic on
factors L5 Lateral leg, Great toe Web space No
o Superficial tenderness – pain elicited from light dorsal foot dorsiflexi between 1st reliable
touch on skin on and 2nd toe reflex
o Simulation – back pain produced by activities that S1 Post calf, Foot Lateral Ankle
should not be painful e.g. axial loading of the head (1- lateral or plantar aspect of
2 lbs), passive rotation of shoulders and pelvis in same sole of foot flexion the sole of
plane foot
o Distraction – Symptomatic response to a test
changes when test is repeated while pt is distracted, Radicular vs. Referred Pain
adjunct for Cognitive Behavioral Therapy Radicular Pain Somatic-referred Pain
o Regionalization – ratchet-like “giving way” weakness Leg > Back Back > Leg
Shooting, lancinating, Dull, pressure-like, deep
or non-neuroanatomic numbness
cutaneous component
o Overreaction – disproportionate response to routine
Travels along limb in a Extends into limbs across a
exams such as collapsing, grimacing, guarding, narrow band wide region
groans, tremors, etc (+)/(-) neurologic defect (-) neurologic deficit
 Prognosis  Listing away from side of disc herniation (paracentral
o Psychosocial risk factors herniation)
 Yellow flags and Waddell’s Signs  SLR for L5 or S1 root
o Reproduction of radicular pain bet 30-70°  Dx: posterolateral, oblique view demonstrate collar on Scotty
o High sensitivity but low specificity Dog, bone scans (+) 5-7 days and last up to 18 mos
o Crossed SLR (Well Leg Raising) is more specific but Spondylolisthesis
less sensitive  Forward or backward slippage of one vertebral body to
 Natural Hx another, commonly seen in the lumbar region
o Favorable  Six categories:
o Conservative and surgical tx are both successful o Isthmic - m/c, stress fx of pars interarticularis
o Large extruded discs are more likely to ↓ in size o Dysplastic – Congenital, dysplasia of facet joints
Spinal Stenosis o Degenerative – older spine, long-standing instability
 Degenerative changes resulting in disc space narrowing, from generation, commonly affects L4-L5 level
vertebral body osteophytosis, jt arthropathy, which narrows o Traumatic – rare, acute fx secondary to trauma
the central canal and lateral recesses (neuroforamina) o Pathologic – medical causes that can cause ↓ bone
 Possible neural compression and ischemia strength
 Usually present at ~50 y/o affecting the L3 and L4 at the o Postsurgical – instability from extensive
lumbar region decompression
 Differential dx of intermittent claudication  Occurrence 2-4x more likely in males
o Intermittent Claudication – relieved by rest Meyerding Grading of Slippage
o Neurogenic intermittent claudication (Pseudo) – Grade 0 0% slippage
relieved by FLEX, use Bicycle Test of van Gelderen
Grade 1 1-25% slippage
 Etiology
o Congenital Grade 2 26-50%
 Hereditary slippage
 Achondroplastic/dwarfism Grade 3 51-75%
o Acquired slippage
 Degenerative (m/c) Grade 4 76-100%
 Spondylotic/spondylolisthetic slippage
 Iatrogenic (postsurgical) Spondyloptosis >100%
 Posttraumatic slippage
 Metabolic (Paget’s disease)
o Traumatic
Lumbar Spinal Stenosis
o Combined
 Congenital, onset in 30s
 Neurogenic claudication – pain in buttocks, thigh, and leg c
 Acquired
standing, relieved by sitting and bending forward
o Degenerative
Spondylolysis
 M/c, late 50s – early 60s, commonly involved
 M/c seen in children and adolescents at the L5 vertebral level
L4-L5, followed by L3-L4
and occurs at the pars interarticularis (pedicle, transverse
 Commonly affects 2-3 spinal segments
process, lamina, and articulator processes)
o Mechanical Compression
 Commonly unilateral
 Compression of microvasculature  nerve root
 Localized back pain exacerbated by motion (HYPEREXT),
ischemia, ↑ microvascular permeability and
standing, lying prone, relieved by FLEX, normal neurologic
edema
exam
 Insidious, chronic LBP that progresses to buttock, thigh, and to restrictive lung disease), plain films typically normal in
leg pain, fatigue, heaviness, pain in the legs c ambulation early stages
(neurogenic claudication) Herniated Nucleus Pulposus
 Intermittent claudication – commonly seen in pts c ASO  A disc injury in which the nucleus pulposus migrates through
(arteriosclerosis obliterans) highest risk factor is smoking, the annulus fibrosus, most commonly occurs at ages 30-40
TAO (thromboangiitis obliterans, Buerger’s disease) and a higher prevalence in the lumbar (L4-L5 or L5-S1) and
cervical (C5-C6) regions
Symptoms Neurogenic Vascular  M/c direction: posterolateral, which compresses the point of
Back Pain Common Uncommon exit of spinal nerve roots, is one of the causes of
Sitting/flexed position Not positional radiculopathy
Standing and resting Pain relief when  Classification:
Pain Relief
usually insufficient standing o Bulging – No annulus defect, disc convexity is beyond
Often slow (>5 mins) Almost immediate vertebral margin
Ambulatory o Prolapsed – Nuclear material protrudes into an annulus
Variable Fixed
tolerance
defect
Uphill vs.
Downhill more painful Uphill more painful o Extruded – Nuclear material extends into the PLL
Downhill
Bicycle ride No pain Pain o Sequestered disc – Nuclear fragment free in the canal
 Common exam findings:  Dx:
o Loss of lumbar lordosis c limited EXT, trunk FLEX o ↓ disc height, vertebral osteophytosis, sclerosis,
forward in standing and walking (Simian posture), no radiography
significant tenderness, (-) SLR, (n) motor exam despite o Disc desiccations, annular tears, HNP – MRI
report of weakness  Tx:
o MRI o Medications (COX2 inhibitors, anti-neuropathic pain)
 Mild – 75-99 o McKenzie Program to centralize extremity pain – LB
 Moderate – 50-74 exercise (EXT) (Williams Program - FLEX)
 Severe - <50 o Lumbar corsets
 Natural hx: o Vertebral distraction may relieve compression
o P 4 yrs:  Cervical – 20-30° of FLEX c 25 lbs of resistance
 70% no change  Lumbar – may require ↑ force (at least ½ of
 15% improved BW) /split table
 15% worsened
o 5-15% may have coexisting cervical spinal stenosis
Ankylosing Spondylitis – rheumatologic, classified as a
seronegative spondyloarthropathy (negative Rheumatoid Factor)
 Seronegative spondyloarthopathies: Psoriasis, AS, Reactive
Arthritis, Inflammatory Bowel Disease
 Crooked vertebra, bamboo spine
 Earliest clinical features:
o Gradual onset in males < 30 y/o, primary
manifestation: morning stiffness, improves c exercise,
not relieved by rest
 Schober’s Test, chest expansion < 2.5 cm (last stage)
(costovertebral and costotransverse jts involvement) (prone

You might also like

pFad - Phonifier reborn

Pfad - The Proxy pFad of © 2024 Garber Painting. All rights reserved.

Note: This service is not intended for secure transactions such as banking, social media, email, or purchasing. Use at your own risk. We assume no liability whatsoever for broken pages.


Alternative Proxies:

Alternative Proxy

pFad Proxy

pFad v3 Proxy

pFad v4 Proxy