Immersion Death: Dr. Rayyan Al-Ali

Immersion Death
DR. RAYYAN AL- ALI

Bodies retrieved from water may have:
• Died from natural disease before falling into the
water.
• Died from natural disease while already in the
water.
• Died from injury before being thrown into the
water.
• Died from injury while in the water.
• Died from effects of immersion other than
drowning.
• Died from drowning.
Signs of immersion
• Maceration of the skin begins within minutes in warm water, such as
death in a bathtub, but in cold water is visible after a variable time.
The first signs tend to be on areas with an appreciable keratin layer
(such as the fingertips, palms, backs of the hands and later the
soles) where the surface becomes wrinkled, pale and sodden – the
so-called ‘washerwoman’s skin’, for obvious reasons.
After some days in warmer water and up to several weeks in cold,

the thick keratin of hands and feet becomes detached and
eventually peels off in “glove and stocking” fashion.
Problems of obtaining fingerprints in such circumstances are dealt

with later.
• Cutis anserina – or ‘goose-flesh’ – is a common
finding in immersed bodies, but is related to cold
rather than warm water. The erector pilae
muscles attached to each hair follicle can
contract in any type of death.
The distribution of post-mortem hypostasis is
of no value in immersed bodies. It is usual
for most corpses to float or hang in the water
with buttocks uppermost, and the head and
limbs hanging down, but water movements
often roll the body constantly unless in a
placid lake or wide river.
Mud, coal-slurry, oil, silt or sand may be present on or in
the body, as well as other artefacts such as seaweed,
water weed, algae and small aquatic animal life of many
types. Mud may be adherent to the whole body surface
and clothing, or may be retained in the hair, mouth,
nostrils, ears and other more protected sites. In
appropriate circumstances, such deposits should be
retained for scientific examination, if they might help
establish where the body has travelled from in marine or
riverine waters
Where a body is decomposed and partly skeletalized,

molluscs may colonize the bones
ESTIMATION OF DURATION OF
IMMERSION
The over-riding variable factor is water temperature which
has the most effect upon decomposition.
When a body falls into water in average temperate
climates, the following is an approximate guide to timing,
in conjunction with the other usual signs, such as rigor:
• If no wrinkling of the finger-pads is present, less than a
few hours
• Wrinkled fingers, palms and feet, progressively from half
a day to 3 days
• Early decomposition, often first in the dependent head
and neck, abdomen and thighs: 4–10 days
• Bloating of face and abdomen with
marbling of veins and peeling of epidermis
on hands and feet, and slippage of scalp:
2–4 weeks
• Gross skin shedding, muscle loss with
skeletal exposure, partial liquefaction: 1–2
months.
NATURAL DEATH WHILST IN THE
WATER
It may well be impossible to determine whether a person recovered
from the water, with a potentially fatal natural disease, suffered this
before or after immersion.
Cardiovascular disease is the most common reason for death in the

water. A pre-existing condition may be exacerbated either by the
physical exertion of swimming or struggling, or by the effects of cold.
The common belief that swimming after a heavy meal is dangerous
probably has some basis in fact, because of circulatory changes
such as the ‘splanchnic shunt’. When an acute cardiac episode
occurs, such as sudden myocardial insufficiency or an arrhythmia,
death may follow either from the cardiac lesion itself, or from
drowning because of sudden physical incapacity.
INJURIES SUSTAINED BEFORE ENTERING THE
WATER
In marine, air or road transport accidents, the victims may be
injured or killed before entering the water. Persons falling from
docks, bridges or ships may strike masonry or solid obstructions
before hitting the water. Suicides may suffer in the same way or
in exceptional circumstances, may stab themselves or cut their
throats in a position where they fall into water immediately
afterwards.
When the nature of the injuries is such that they could not
have occurred in water, the issue is clear. Burns, missile wounds,
the effect of explosions (other than compression injury) and
patterned injuries may be incompatible with infliction in water,
and must therefore be related to the effects of a previous assault
or some accident on board ship or aircraft.
INJURIES SUSTAINED IN THE WATER
• Trauma in the water is common, as both an ante-mortem
and a postmortem phenomenon.
• The issue usually becomes one of attempting to
distinguish injuries sustained during life from those after
death.
• Water complicates the problem by washing away surface
bleeding from open injuries. A laceration inflicted during
life will, however, usually show some bleeding into the
tissues under the margins of the wound, at least until
postmortem decomposition blurs the appearances.
 Freshwater vs seawater
 Causes of death in drowning
 The autopsy signs of drowning
 Chemical changes in the blood
 Histologic changes
 Diatoms and the diagnosis of drowning
 Estimate time of drowning
Types of drowning:
Drowning is of four types:

Wet drowning: in this , water is inhaled into lungs and the victim has
severe chest pain. If resuscitated, he has no pleasant recollections.
Dry drowning: in this type water does not enter the lungs but deaths
result from immediate sustained laryngeal spasm due to inrush of
water into the nasopharynx or larynx. Thick mucus foam and
frothy may develop, producing a plug. This is seen in 10 to 20%
cases of immersion. Resuscitated victims have panoramic views
past of life and pleasant dreams without distress.
Secondary drowning: (post immersion syndrome or near
drowning): in this type death occurs from within half hour to
several days after resuscitation. Electrolyte disturbances and
metabolic acidosis occur. Death occurs from cerebral anoxia
with irreversible brain damage. Microscopically , the lungs
show hemorrhage desquamative and exudative reaction. Later
change are of inhalation pnemonitis with hyaline membranes
in alveolar ducts and foreign body reaction to inhaled particle,
progressing to bronchopneumonia or abscess formation. The
loss of normal surfactant by the inhaled water can result in
large areas of atelectasis. Death may occur from cerebral
anoxia with irreversible brain damage. Myocardial anoxia may
cause delayed heart failure.
Immersion syndrome(hydrocution or submersion inhibition): death results
from cardiac arrest due to vagal inhibition as result of :
1)Cold water stimulating the nerve ending of the surface of the body.
2)Water striking the epigastria.
3)Cold water entering ear burns , nasal passages, and larynx which cause
stimulation of nerve ending of the mucosa.
Fresh water drowning
 A massive absorption of water through the alveolar membranes.
 Relative anaemia and myocardial hypoxia.
 Hemolysis of red cells release potassium- ventriculer fibrillation.
 Death in freshwater typically took some 4–5 minutes

Seawater drowning
 Withdrawal of water from the plasma into the lungs.
 Less deleterious to heart function and helped to explain the longer survival time in
seawater immersion.
 Seawater drowning take 8–12 minutes.

Causes of death in drowning
1) Asphyxia. Inhalation of fluid causes obstruction to the air-passages. Circulatory

and respiratory failure occur simultaneously, due to anoxia of both the
myocardium and the respiratory center.
2) Ventricular fibrillation. In fresh water drowning, arrhythmias of the heart beat,
ventricular tachycardia and fibrillation cause death within three to five minutes.
3) Laryngeal spasm may result from inrush of water into the naso-pharynx or
larynx.
4) Vagal inhibition is due to icy cold water, high emotion or excitement and
unexpected immersion.
5) Exhaustion.
6) Injuries: Fracture of skull and fracture-dislocation of cervical vertebrae may

occur due to the head striking forcibly against some solid object. Concussion
may occur due to striking the head against some hard substance or the water
itself while falling from a height.
The Autopsy Sings of Drowning
Froth in the air passage
 The froth is oedema fluid from the lungs.
 Exudes through the mouth and nostrils.
 pink or red-tinged, because of intrapulmonary bleeding.
 Not all froth around the mouth is from drowning.

Overinflation of the lungs
Overinflation of the lungs (emphysema aquosum)

 The older name for this condition is ‘emphysema aquosum’
 The oedema fluid in the bronchi blocks the passive collapse that normally occurs
at death.
 This may be sufficient to mark the lateral surfaces of the lungs with the
impression of the ribs, leaving visible and palpable grooves after removal of the
organs from the thorax.
Fig. Cut section of lung showing
Pulmonary edema
Sub-pleural heamorrhages (paultauf
spots):
 Reflecting intra-alveolar heamorrhages
Stomach full of water
Haemorrhage into the middle ears and into the temporal
bone
Aqueous liquid inside sinuses
Cadaveric Spasms
Involving the hand (firmly grasping weeds or sands) indicating the victims
was alive when immersed
Chemical changes
 Because of the marked haemodilution that occurs in freshwater drowning and the
electrolyte shifts in saltwater drowning, it is reasonable to expect that chemical
analyses of the plasma should provide reliable evidence of drowning.
 Unfortunately, these theoretical hopes have not been realized in practice, mainly
because of the biochemical chaos that occurs soon after death from any cause, in
which the ability of live cell membranes to partition fluid and electrolytes is
rapidly lost.
 Normally chloride con. In rt. And lt. chambers are equal.
 Changes more than 25mg/100ml : suspect drowning.

Histologic changes
 The reduction of spleen weight and spleen–liver ratios postulated as a diagnostic

marker in drowning when compared with other modes of ‘asphyxia’.
 The skin changes in maceration are also well recorded, and some have been
claimed to have use in dating the period of immersion.
Diatoms
 Diatoms are Unicellular algae with outer siliceous wall.
 Resist heat and acid.
 The basic premise is that when a live person is drowned in water containing
diatoms (microscopic algae with a silicaceous exoskeleton), many diatoms will
penetrate the alveolar walls and be carried to distant target organs such as brain,
kidney, liver and bone marrow. After autopsy, samples of these organs can be
digested with strong acid to dissolve the soft tissue, thus leaving the highly
resistant diatom skeletons to be identified under the microscope.
 When a dead body is deposited in water or when death in the water is not due to
drowning, then, although diatoms may reach the lungs by passive percolation, the
absence of a beating heart prevents circulation of diatoms to distant organs.
Time passed since drowning
Estimated through :
 Rate of body cooling
 Floating of body (occurs after 5-8 days in summer)
 Volume of pleural fluid accumulated

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Immersion Death

DR. RAYYAN AL- ALI

After some days in warmer water and up to several weeks in cold,

Problems of obtaining fingerprints in such circumstances are dealt

Where a body is decomposed and partly skeletalized,

Cardiovascular disease is the most common reason for death in the

Drowning is of four types:

 A massive absorption of water through the alveolar membranes.

 Relative anaemia and myocardial hypoxia.

 Hemolysis of red cells release potassium- ventriculer fibrillation.

 Death in freshwater typically took some 4–5 minutes

 Withdrawal of water from the plasma into the lungs.

 Seawater drowning take 8–12 minutes.

1) Asphyxia. Inhalation of fluid causes obstruction to the air-passages. Circulatory

6) Injuries: Fracture of skull and fracture-dislocation of cervical vertebrae may

 Exudes through the mouth and nostrils.

 pink or red-tinged, because of intrapulmonary bleeding.

 Not all froth around the mouth is from drowning.

Overinflation of the lungs (emphysema aquosum)

 Changes more than 25mg/100ml : suspect drowning.

 The reduction of spleen weight and spleen–liver ratios postulated as a diagnostic

 Diatoms are Unicellular algae with outer siliceous wall.

 Resist heat and acid.

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