Open Fractures in Ortho Patients

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OPEN FRACTURES

BY: GASATAYA, CHARISSE ANN G.


01 INTRODUCTION

02 GUSTILLO – ANDERSON CLASSIFICATION

03 LIMITATIONS AND DISADVANTAGES

04 MANAGEMENT
INTRODUCTION
Open Fracture
 An injury where the fracture and the fracture hematoma
communicate with the external environment through a
traumatic defect in the surrounding tissue and overlying skin.
 Generally a result of high energy mechanisms which cause
greater soft tissue disruption that leaves the wound more
susceptible to infection by contaminating bacteria.
 The energy is stored in soft and hard tissues until the strength
of respective material is exceeded. Comminuted pieces may
acquire high velocity after which they propel into the
surrounding soft tissues and cause additional damage.
Compartment Syndrome

Release of
Trauma
mediators of
01 Contamination 02 inflammation
03 “Leaky capillaries”

Intravascular fluid Pressure buildup


04 into the interstitial
space
05 compressing the
microvasculature
06 Cell death
Prognosis
 Fracture pattern
 Local factors
 Amount of foreign debris and contaminant material
 Extent of soft tissue and bone devitalization.
 Systemic factors
 Host nutrition
 Medications
 Nicotine abuse
GUSTILO – ANDERSON
CLASSIFICATION
Gustilo Classification

01 Wound Size

02 Soft Tissue Damage

03 Contamination

04 Fracture Pattern / Degree of


Comminution
I II IIIA IIIB IIIC
Energy Low  Moderate High High High
Wound Size ≤ 1 cm 1-10 cm  usually >10 cm usually >10 cm usually > 10 cm
Soft Tissue
Minimal Moderate  Extensive  Extensive  Extensive 
Damage
Clean Moderate
Contamination  Extensive  Extensive  Extensive 
contamination
Severe Severe Severe
Simple fx pattern
Moderate comminution or comminution or comminution or
Fracture Pattern with minimal
comminution  segmental segmental segmental
comminution
fractures fractures  fractures 
Periosteal
No No Yes Yes Yes
Stripping
Requires free
tissue flap or Typically requires
Skin Coverage Local coverage Local coverage Local coverage
rotational flap flap coverage
coverage

Exposed fracture
Neurovascular with arterial
Normal  Normal Normal  Normal
Injury damage that
requires repair
 Low energy
 < 1 cm long

TYPE I  Minimal soft tissue damage


 Bone injury: minimal comminution
 Adequate soft tissue coverage
OPEN FX  Clean
 Primary closure

1st generation
cephalosporin (e.g.
cefazolin) for 24 hours after
closure
 1 – 10 cm
 No extensive soft tissue damage, flaps or avulsions
(moderate)
TYPE II  Moderate contamination
 Adequate soft tissue coverage
 Primary closure
OPEN FX  Bone injury: moderate comminution
 Soft tissue injury: moderate; some muscle damage

1st generation
cephalosporin (e.g.
cefazolin) for 24 hours after
closure
 Usually >10cm
 Soft tissue injury: severe with crushing
 Adequate soft tissue coverage Despite extensive
TYPE IIIA soft tissue laceration
 Bone injury is usually comminuted; soft tissue
coverage of bone is possible
OPEN FX  Extensive contamination

1st generation
cephalosporin (gram +).
Aminoglycoside (gram -)

cephalosporin/aminoglycosi
de should be continued for
24-72 hours after the last
debridement procedure

Penicillin should be added if


concern for anaerobic
organism (farm injury)
 >10cm
 Extensive soft tissue injury loss, requires flap
TYPE IIIB 

Usually requires soft tissue reconstructive surgery
Periosteal stripping and bone exposure
 Massive contamination
OPEN FX

1st generation
cephalosporin (gram +).
Aminoglycoside (gram -)

cephalosporin/aminoglycosi
de should be continued for
24-72 hours after the last
debridement procedure

Penicillin should be added if


concern for anaerobic
organism (farm injury)
 Usually >10cm
 Soft tissue injury: Very severe loss of cover
 Arterial injury requiring repair
TYPE IIIC  Bone cover is poor
 Usually requires soft tissue reconstructive surgery
OPEN FX  High level of contamination

1st generation
cephalosporin (gram +).
Aminoglycoside (gram -)

cephalosporin/aminoglycosi
de should be continued for
24-72 hours after the last
debridement procedure

Penicillin should be added if


concern for anaerobic
organism (farm injury)
Signs of Vascular Injury
DISADVANTAGES
Limitations & Disadvantages
 Interobserver reliability
 Overlapping categories
 Does not account for tissue viability and tissue necrosis
over time
 Definition has undergone many modifications and does
not have uniformity in application
 Includes wide spectrum of injuries in Type IIIB injuries
 Mainly depends on size of skin wound
 Does not evaluate severity of injury to skin, bone, and
musculotendicous units separately
 Does not address the question of salvage
MANAGEMENT
 DEBRIDEMENT
 All foreign materials and tissues that are contaminated or
suspected to be avascular are systematically removed
 Should be done within 6 hours
 It minimizes the risk factors for infection
 Dead space
 Hematoma
 IMMOBILIZATION
 Skeletal stabilization
 Plaster casts
 Traction
 External skeletal fixation
 Primary internal fixation
 Plate fixation
 Intramedullary nails
 PRIMARY CLOSURE
Grade 1 2 3A 3B 3C

Infection 0-2% 2-7% 10-25% 10-50% 25-50%


Rates

Fracture
Healing 21-28 28-30 30-35 30-35
(weeks)

Amputation 50%
Rate
- END -

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